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Journal articles on the topic "CSMC"

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Frigaard, Niels-Ulrik, Hui Li, Kirstin J. Milks, and Donald A. Bryant. "Nine Mutants of Chlorobium tepidum Each Unable To Synthesize a Different Chlorosome Protein Still Assemble Functional Chlorosomes." Journal of Bacteriology 186, no. 3 (February 1, 2004): 646–53. http://dx.doi.org/10.1128/jb.186.3.646-653.2004.

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ABSTRACT Chlorosomes of the green sulfur bacterium Chlorobium tepidum comprise mostly bacteriochlorophyll c (BChl c), small amounts of BChl a, carotenoids, and quinones surrounded by a lipid-protein envelope. These structures contain 10 different protein species (CsmA, CsmB, CsmC, CsmD, CsmE, CsmF, CsmH, CsmI, CsmJ, and CsmX) but contain relatively little total protein compared to other photosynthetic antenna complexes. Except for CsmA, which has been suggested to bind BChl a, the functions of the chlorosome proteins are not known. Nine mutants in which a single csm gene was inactivated were created; these mutants included genes encoding all chlorosome proteins except CsmA. All mutants had BChl c contents similar to that of the wild-type strain and had growth rates indistinguishable from or within ∼90% (CsmC− and CsmJ−) of those of the wild-type strain. Chlorosomes isolated from the mutants lacked only the protein whose gene had been inactivated and were generally similar to those from the wild-type strain with respect to size, shape, and BChl c, BChl a, and carotenoid contents. However, chlorosomes from the csmC mutant were about 25% shorter than those from the wild-type strain, and the BChl c absorbance maximum was blue-shifted about 8 nm, indicating that the structure of the BChl c aggregates in these chlorosomes is altered. The results of the present study establish that, except with CsmA, when the known chlorosome proteins are eliminated individually, none of them are essential for the biogenesis, light harvesting, or structural organization of BChl c and BChl a within the chlorosome. These results demonstrate that chlorosomes are remarkably robust structures that can tolerate considerable changes in protein composition.
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Zhou, Yong, Lifang Hu, Lunwei Jiang, and Shiqiang Liu. "Genome-wide identification, characterization, and transcriptional analysis of the metacaspase gene family in cucumber (Cucumis sativus)." Genome 61, no. 3 (March 2018): 187–94. http://dx.doi.org/10.1139/gen-2017-0174.

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Metacaspase (MC), a family of caspase-like proteins, plays vital roles in regulating programmed cell death (PCD) during development and in response to stresses in plants. In this study, five MC genes (designated as CsMC1 to CsMC5) were identified in the cucumber (Cucumis sativus) genome. Sequence analysis revealed that CsMC1–CsMC3 belong to type I MC proteins, while CsMC4 and CsMC5 are type II MC proteins. Phylogenetic tree and conserved motif analysis of MC proteins indicated that these proteins can be classified into two groups, which are correlated with the types of these MC proteins. Gene structure analysis demonstrated that type I CsMC genes contain 4–7 introns, while all type II CsMC genes harbor one intron. In addition, many hormone-, stress-, and development-related cis-elements were identified in the promoter regions of CsMC genes. Expression analysis using RNA-seq data revealed that CsMC genes have distinct expression patterns in various tissues and developmental stages. qRT-PCR results showed that the transcript levels of CsMC genes could be regulated by various abiotic stresses such as NaCl, PEG, and cold. These results demonstrate that the cucumber MC gene family may function in tissue development and plant stress responses.
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Li, Hui, and Donald A. Bryant. "Envelope Proteins of the CsmB/CsmF and CsmC/CsmD Motif Families Influence the Size, Shape, and Composition of Chlorosomes in Chlorobaculum tepidum." Journal of Bacteriology 191, no. 22 (September 11, 2009): 7109–20. http://dx.doi.org/10.1128/jb.00707-09.

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ABSTRACT The chlorosome envelope of Chlorobaculum tepidum contains 10 proteins that belong to four structural motif families. A previous mutational study (N.-U. Frigaard, H. Li, K. J. Milks, and D. A. Bryant, J. Bacteriol. 186:646-653, 2004) suggested that some of these proteins might have redundant functions. Six multilocus mutants were constructed to test the effects of eliminating the proteins of the CsmC/CsmD and CsmB/CsmF motif families, and the resulting strains were characterized physiologically and biochemically. Mutants lacking all proteins of either motif family still assembled functional chlorosomes, and as measured by growth rates of the mutant strains, light harvesting was affected only at the lowest light intensities tested (9 and 32 μmol photons m−2 s−1). The size, composition, and biogenesis of the mutant chlorosomes differed from those of wild-type chlorosomes. Mutants lacking proteins of the CsmC/CsmD motif family produced smaller chlorosomes than did the wild type, and the Qy absorbance maximum for the bacteriochlorophyll c aggregates in these chlorosomes was strongly blueshifted. Conversely, the chlorosomes of mutants lacking proteins of the CsmB/CsmF motif family were larger than wild-type chlorosomes, and the Qy absorption for their bacteriochlorophyll c aggregates was redshifted. When CsmH was eliminated in addition to other proteins of either motif family, chlorosomes had smaller diameters. These data show that the chlorosome envelope proteins of the CsmB/CsmF and CsmC/CsmD families play important roles in determining chlorosome size as well as the assembly and supramolecular organization of the bacteriochlorophyll c aggregates within the chlorosome.
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Chung, Soohee, and Donald A. Bryant. "Characterization of the csmD and csmE genes from Chlorobium tepidum. The CsmA, CsmC, CsmD, and CsmE proteins are components of the chlorosome envelope." Photosynthesis Research 50, no. 1 (October 1996): 41–59. http://dx.doi.org/10.1007/bf00018220.

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Bowles, D. K., K. K. Maddali, V. C. Dhulipala, and D. H. Korzick. "PKCδ mediates anti-proliferative, pro-apoptic effects of testosterone on coronary smooth muscle." American Journal of Physiology-Cell Physiology 293, no. 2 (August 2007): C805—C813. http://dx.doi.org/10.1152/ajpcell.00127.2007.

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Sex hormone status has emerged as an important modulator of coronary physiology and cardiovascular disease risk in both males and females. Our previous studies have demonstrated that testosterone increases protein kinase C (PKC) δ expression and activity in coronary smooth muscle (CSMC). Because PKCδ has been implicated in regulation of proliferation and apoptosis in other cell types, we sought to determine if testosterone modulates CSMC proliferation and/or apoptosis through PKCδ. Porcine CSMC cultures (passages 2–6) from castrated males were treated with testosterone for 24 h. Testosterone (20 and 100 nM) decreased [3H]thymidine incorporation in proliferating CSMC to 59 ± 5.3 and 33.1 ± 4.5% of control. Flow cytometric analysis demonstrated that testosterone induced G1arrest in CSMC with a concomitant reduction in the S phase cells. Testosterone reduced protein levels of cyclins D1and E and phosphorylation of retinoblastoma protein while elevating levels of p21cip1and p27kip1. There were no significant differences in the levels of cyclins D3, CDK2, CDK4, or CDK6. Testosterone significantly reduced kinase activity of CDK2 and -6, but not CDK4, -7, or -1. PKCδ small interfering RNA (siRNA) prevented testosterone-mediated G1arrest, p21cip1upregulation, and cyclin D1and E downregulation. Furthermore, testosterone increased CSMC apoptosis in a dose-dependent manner, which was blocked by either PKCδ siRNA or caspase 3 inhibition. These findings demonstrate that the anti-proliferative, pro-apoptotic effects of testosterone on CSMCs are substantially mediated by PKCδ.
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Somara, Sita, Daniela Bashllari, Robert R. Gilmont, and Khalil N. Bitar. "Real-time dynamic movement of caveolin-1 during smooth muscle contraction of human colon and aged rat colon transfected with caveolin-1 cDNA." American Journal of Physiology-Gastrointestinal and Liver Physiology 300, no. 6 (June 2011): G1022—G1032. http://dx.doi.org/10.1152/ajpgi.00301.2010.

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Caveolin-1 (cav-1) plays a key role in PKC-α and RhoA signaling pathways during acetylcholine (ACh)-induced contraction of colonic smooth muscle cells (CSMC). Aged rat CSMC showed sluggish contractility, concomitant with reduced expression of cav-1 with an associated reduction in activation of PKC-α and RhoA signaling pathway. Real-time monitoring of live human CSMC transfected with yellow fluorescent protein-tagged wild-type caveolin 1 cDNA (YFP-wt-cav-1) cDNA in the present study suggests that cav-1 cycles within and along the membrane in a synchronized, highly organized cytoskeletal path. These studies provide, for the first time, the advantages of real-time monitoring of the dynamic movement of caveolin in living cells. Rapid movement of cav-1 in response to ACh suggests its dynamic role in CSMC contraction. Human CSMC transfected with YFP-ΔTFT-cav-1 dominant negative cDNA show fluorescence in the cytosol of the CSMC and no movement of fluorescent cav-1 in response to ACh mimicking the response shown by aged rat CSMC. Transfection of CSMC from aged rat with YFP-wt-cav-1 cDNA restored the physiological contractile response to ACh as well as the dynamic movement of cav-1 along the organized cytoskeletal path observed in normal adult CSMC. To study the force generation by CSMC, three-dimensional colonic rings were bioengineered. Colonic bioengineered rings from aged CSMC showed reduced force generation compared with colonic bioengineered rings from adult CSMC. Colonic bioengineered rings from aged CSMC transfected with wt-cav-1 cDNA showed force generation similar to colonic bioengineered rings from adult rat CSMC. The data suggest that contraction in CSMC is dependent on cav-1 reorganization dynamics, which restores the physiological contractile response in aged CSMC. We hypothesize that dynamic movement of cav-1 is essential for physiological contractile response of colonic smooth muscle.
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Chesebro, James W. "CSMC bookends." Critical Studies in Media Communication 17, no. 2 (June 2000): 232–40. http://dx.doi.org/10.1080/15295030009388392.

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Chesebro, James W. "CSMC bookends." Critical Studies in Media Communication 17, no. 4 (December 2000): 510–13. http://dx.doi.org/10.1080/15295030009388416.

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Crow, Bryan, and Robert Abelman. "CSMC booknotes." Critical Studies in Mass Communication 5, no. 4 (December 1988): 362–64. http://dx.doi.org/10.1080/15295038809366724.

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Crow, Bryan, and Robert Abelman. "CSMC booknotes." Critical Studies in Mass Communication 6, no. 1 (March 1989): 99–101. http://dx.doi.org/10.1080/15295038909366735.

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Dissertations / Theses on the topic "CSMC"

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Talami, Matteo. "Modeling of the Toroidal Field Insert coil for the ITER Project." Master's thesis, Alma Mater Studiorum - Università di Bologna, 2017. http://amslaurea.unibo.it/12916/.

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Il contenuto della tesi riguarda le analisi numeriche e sperimentali effettuate su un campione di cavo superconduttivo del sistema magnetico del reattore sperimentale per la fusione nucleare “ITER”. In particolare, il campione di cavo denominato “Toroidal Field Insert” o “TFI”, appartiene al sistema magnetico toroidale della macchina e viene inserito in un solenoide esterno in modo da replicare le condizioni di campo magnetico tipiche del normale funzionamento di questo conduttore. Le analisi sperimentali effettuate sul campione sono mirate alla caratterizzazione del comportamento durante un ipotetico ciclo di vita del cavo. I parametri principalmente studiati risultano essere: la caratterizzazione dello stato superconduttivo prima e dopo le varie sollecitazioni imposte, l’efficacia idraulica del raffreddamento e la stabilità termica del magnete. In modo complementare alla analisi dei dati sperimentali, due modelli numerici a diverse scale sono stati sviluppati e testati: un primo modello, alla scala di sistema, si occupa dello studio termico e idraulico dell’intera porzione di cavo testata; il secondo, alla scala di componente, si occupa della simulazione elettromagnetica di un riscaldatore induttivo installato sul conduttore volto a misurarne la stabilità. Il confronto tra l’analisi numerica e quella sperimentale ha permesso la comprensione dei principali fenomeni in gioco e la caratterizzazione del conduttore testato.
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Ma, Huiyuan. "A web based compression spring design calculator /." Compression spring design calculator, 2001. http://wwweng.uwyo.edu/commend/Csdc.

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Castiel, Eyal. "Study of QB-CSMA algorithms." Thesis, Toulouse, ISAE, 2019. http://www.theses.fr/2019ESAE0038.

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La performance des réseaux sans fil où les utilisateurs partagent l'air comme moyen de communication est fortement limitée par le phénomène d'interférence électromagnétique. En effet, deux utilisateurs proches qui communiquent sur la même fréquence verront leurs ondes interférer, ce qui peut entraîner la perte de l'information transmise. Ainsi, il est indispensable de mettre en place des protocoles d'accès visant à limiter l'interférence en choisissant de manière efficace les utilisateurs autorisés à émettre à chaque instant. D'un point de vue scientifique, il s'agit d'un problème difficile qui a attiré l'attention de la communauté en informatique et probabilités appliquées depuis plus de 30 ans. Récemment, une nouvelle classe de protocoles d'accès - appelés protocoles CSMA adaptatifs - a émergé, et semble très prometteuse : par exemple, il a été montré que ces nouveaux protocoles possèdent une propriété très attrayante de stabilité maximale. Le but de ce projet est d'approfondir la connaissance que l'on a des protocoles CSMA adaptatifs dits QB (pour l'anglais "Queue-Based") qui à ce jour est encore extrêmement limitée. Concernant ces protocoles, le but de ce projet est de prouver des résultats théoriques permettant de comprendre le compromis réalisable entre débit et délai. Modèle probabiliste - d'un point de vue technique, il s'agit d'étudier le modèle suivant: chaque utilisateur du réseau est représenté par le nœud d'un graphe G, appelé graphe d'interférence, et tel que deux voisins du graphe ne peuvent être actifs simultanément. Des paquets à transmettre arrivent à chaque nœud au cours du temps, et le but est de choisir quels nœuds sont actifs à un moment donné. Le protocole CSMA-QB répond à cette question de la manière suivante : lorsqu'un nœud est actif, il se désactive à taux constant et lorsqu'il est inactif et qu'aucun de ses voisins ne le bloquent, alors il s'active à un taux qui dépend du nombre de paquets en attente de transmission via une fonction ψ appelée fonction d'activation. Le but général de la thèse est de comprendre l'influence de la topologie de G et du choix de ψ sur la performance du protocole. Pour cela, il s'agira d'étudier le temps de mélange de la dynamique de Glauber ainsi qu'un phénomène classique en théorie des probabilités, appelé phénomène de moyennisation stochastique, qui permettent une compréhension fine du comportement dynamique du réseau
Performance of wireless networks, in which users share the air as support for their communications is strongly limited by electromagnetic interference. That is, two users close to each other trying to send a message on the same frequency will experience interference between their messages, eventually leading to the loss of some information. It is then crucial to develop medium access protocols aiming to limit the occurrence of such a phenomena by choosing in an effective (and distributed) manner which station is allowed to transmit. From a scientific point of view, it is a difficult issue which has had some attention from the community in the field of computer science and applied probability in the past 30 years. Recently, a new class of medium access protocols - called adaptive CSMA - emerged and seem quite promising: for example, it has been shown that they exhibit a desirable property: throughput optimality (maximum stability). The goal of this project is to increase the knowledge we have the adaptive CSMA (or CSMA QB, for Queue Based) which is to this day quite limited (notably in the expected waiting time of a request arriving in the system, called delay). Our goal will be to prove theoric results to enhance our understanding of the throughput/delay trade-off
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López, Carballo Gracia Mª. "Caracterización de genes regulados por el ácido retinoico implicados en la diferenciación celular de células SH-SY5Y de neuroblastoma humano." Doctoral thesis, Universitat de València, 2002. http://hdl.handle.net/10803/10017.

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Las señales mediadas por los Receptores Nucleares de Hormonas desempeñan un importante papel en el control de la diferenciación celular. El objetivo de este trabajo es analizar los mecanismos moleculares a través de los cuales se ejerce este control. Como sistema experimental utilizamos células de neuroblastoma humano SH-SY5Y, cuya diferenciación se induce mediante tratamiento con ácido retinoico (RA) que produce la parada en el crecimiento y desencadena la diferenciación celular. Estudiar el mecanismo molecular a través del cual, actúa el RA, también resulta interesante desde un punto de vista biomédico, puesto que los retinoides y sus derivados sintéticos son utilizados en la terapia de neuroblastoma y también de otros tipos de cáncer. Mediante Display Diferencial Ordenado hemos identificando 43 genes regulados (positiva o negativamente) por RA en células SH-SY5Y, entre los que se incluyen factores de transcripción y proteínas relacionadas con el ciclo celular, la transducción de señales y las funciones neuronales. Entre los genes regulados negativamente por RA se encuentra ID3, un factor de transcripción de la familia de los HLH, los cuales desempeñan un papel crucial en la diferenciación celular. ID3 actúa inhibiendo la unión al DNA de ciertos factores de la familia HLH. ID3 bloquea la diferenciación interfiriendo con la actividad de factores HLH neurogénicos. Cuando la expresión de ID3 se reduce por efecto de RA, se activa la cascada de bHLH proneurales, desencadenando la diferenciación celular. La expresión de otros genes bHLH cambió durante la diferenciación inducida por RA: la expresión del gen específico de neuroblastos en proliferación ASCL1 (HASH-1) disminuyó rápidamente tras 6 h de tratamiento con RA, mientras que la expresión de genes promotores de la diferenciación aumentó como es el caso de NEUROD6 y NEUROD1.Los niveles de otros miembros de la familia de los IDs (ID1, ID2) están regulados también negativamente durante la diferenciación inducida por RA en células SH-SY5Y.Mediante Western Blot estudiamos los niveles de proteínas ID1, ID2 e ID3 en células SH-SY5Y tratadas a diferentes tiempos con RA. Los niveles de proteínas disminuyen de manera notoria tras 24 h de tratamiento con RA, en paralelo a lo observado a nivel de ARN mensajero. Los resultados obtenidos indican que el RA produce una regulación negativa coordinada de los genes IDs. El tratamiento con otros inductores de la diferenciación como es el caso del TPA, también dio lugar a una regulación negativa y coordinada de los genes IDs. Esto resalta la importancia de estos genes en los procesos de diferenciación celular. Además hemos visto que la regulación negativa y coordinada de los genes IDs por tratamiento con RA es un mecanismo complejo que implica síntesis de nuevas proteínas y actividad fosfatidil-inositol-3-kinasa (PI3K). El tratamiento con RA activa la vía de señalización intracelular de la PI3K/AKT, resultando en un incremento en la actividad PI3K de extractos de células tratadas con RA y un rápido incremento en la fosforilación de la proteína diana AKT en Serina 473.La activación de la vía PI3K/AKT es necesaria para la diferenciación celular, y probablemente tiene una gran relevancia a nivel fisiológico, puesto que acopla procesos vitales como son la diferenciación y la supervivencia celular. Aunque el mecanismo molecular por el cual el RA activa la vía PI3K/AKT no está todavía definido, los resultados obtenidos indican que, probablemente, podría tratarse de una acción extragenómica del RAR, entre otras razones por la rapidez con la que se produce. Hemos tratado de caracterizar el mecanismo por el cual el RA activa la vía PI3K/AKT. En principio partimos de la hipótesis de que el RA pudiera inducir la expresión de una tirosina-kinasa que fuera capaz de activar la PI3K.Hemos encontrado un buen candidato para esta actividad: RET, el receptor de la familia de neurotrofinas del GDNF, es inducido directa- y fuertemente por RA. En las células SH-SY5Y se expresa uno de los co-receptores que actúa junto a RET, el GFRA2, pero no hemos logrado detectar la expresión de ninguno de los ligandos de la familia del GDNF, que pudiera activar RET actuando de un modo autocrino/paracrino. Por otro lado, no hemos podido observar fosforilación de RET en residuos tirosina, lo que probaría su activación. Estos resultados apoyan la hipótesis que planteamos en la cual el RA desempeñaría una acción de tipo no genómica.Los resultados obtenidos, nos llevan a postular, que el RA desempeña un papel muy importante en la regulación de la supervivencia de las células neurales. La regulación positiva del gen anti-apoptótico BCL2 durante la diferenciación inducida por RA en células de neuroblastoma es un hecho conocido desde hace tiempo, y la diferenciación incrementa la resistencia a la apoptosis inducida por drogas. También se han descrito efectos del RA sobre la supervivencia celular en cultivos primarios de neuroblastos de médula espinal y en neuronas derivadas de células madre neurales. Los resultados presentados aquí proporcionan un mecanismo molecular para explicar estos efectos. Además, pensamos que el fenómeno aquí descrito en células de neuroblastoma pudiera ser relevante en la diferenciación neural en general. La coordinación de las acciones genómicas y extragenómicas del RA da lugar al acoplamiento entre la diferenciación y la supervivencia celular. Por una parte el RA regula la transcripción de genes específicos implicados en diferenciación celular. Por otra parte, a través de una acción extragenómica, el RA activa la vía de PI3K/AKT, que está implicada en la supervivencia celular. Los dos tipos de acciones no se producen de un modo independiente, sino que están entrelazados. La activación extragenómica de la vía PI3K/AKT también está implicada en la regulación transcripcional de genes decisivos para la diferenciación, como hemos demostrado en el caso de la regulación negativa coordinada de los genes IDs. Recíprocamente, el RA puede contribuir a la supervivencia celular a través de la regulación transcripcional positiva de receptores de neurotrofinas, como RET y de trkB. Este acoplamiento entre diferenciación y supervivencia es un fenómeno novedoso.
Nuclear Hormone Receptors (NHR) are key regulators of cell differentiation. We have used the neuroblastoma cell line SH-SY5Y, that undergoes terminal differentiation after addition of Retinoic Acid (RA), as a model system to study the molecular mechanisms through which NHR control this process. By using Ordered Differential Display-PCR we have identified more than 50 genes differentially expressed in RA-treated SH-SY5Y cells. We have found that the expression of ID3, a member of the HLH family of transcription factors, is downregulated during RA-induced differentiation, and run-on transcription experiments demonstrated that this effect was transcriptional. Other ID's, like ID1 and ID2, that were expressed at lower levels in SH-SY5Y cells, were equally downregulated by RA. The levels of ID proteins decreased in parallel to the observed transcriptional repression. The expression of other bHLH genes changed during RA-induced differentiation: the expression of neuroblast-specific ASCL1 (HASH-1) gene was promptly reduced after RA treatment, whereas the expression of differentiation-promoting genes NEUROD6 (NEX-1, HATH-2) and NEUROD1 was increased. Our hypothesis is that ID's block cell differentiation by interfering the action of the neurogenic HLH transcription factors, and that RA released that block by downregulating ID's gene expression. Treatments with TPA, another inducer of neuroblastoma cell differentiation, also resulted in coordinated downregulation of the ID gene expression, underscoring the role of ID genes in differentiation. Downregulation of the ID gene expression by RA involves a complex mechanism, since full transcriptional repression required newly synthesized proteins and signaling by the Phosphatidylinositol-3 kinase. RA treatment activates the Phosphatidylinositol-3 kinase/Akt signaling pathway, resulting in increased Phosphatidylinositol-3 Kinase activity in extracts from RA-treated cells and a rapid increase in phosphorylation of Akt in Ser473. Inhibition of Phosphatidylinositol-3 kinase by LY294002 impaired RA-induced differentiation, as assessed by morphological and biochemical criteria. We propose that RA, by activating the Phosphatidylinositol-3 kinase/Akt signaling pathway, plays an important role in the regulation of the neuronal cell survival.
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Baisden, Joseph M. "AFAP-110 is a cSrc activator." Morgantown, W. Va. : [West Virginia University Libraries], 2003. http://etd.wvu.edu/templates/showETD.cfm?recnum=2766.

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Thesis (Ph. D.)--West Virginia University, 2003.
Title from document title page. Document formatted into pages; contains v, 149 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references.
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Forss, Jonas. "Real-Time Communication over Broadcast Networks." Thesis, University of Skövde, Department of Computer Science, 1999. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-308.

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Today the utilization of all kind of multimedia services in networks increases and due to this demand for real-time communication gets higher for every day. One of the most common protocols used today is Ethernet. It is of interest to find real-time protocols that are useful together with already existing protocols since it is expensive to rebuild the network infrastructure. Several new protocols have been proposed to solve the upcoming problems.

The objective of this project is to make an inventory and a comparison between Ethernet and these new protocols. During this project four different protocols capability to fulfill the demands for real-time communication have been studied and compared with Ethernet. Three of the protocols are built on the same basic technique as Ethernet and these are PCSMA, CSMA-DCR and DOD-CSMA-CD. The fourth protocol is ATM and it transmits in a different way over communication networks.

The analysis of this project is pointing out advantages, similarities and differences between the protocols from a real-time perspective. All four protocols are more suitable for real-time requirements than Ethernet.

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JAIN, NITIN. "MULTICHANNEL CSMA PROTOCOLS FOR AD HOC NETWORKS." University of Cincinnati / OhioLINK, 2001. http://rave.ohiolink.edu/etdc/view?acc_num=ucin995471534.

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Davies, Gwyneth. "The medical culture of the Ovambo of Southern Angola and Northern Namibia." Thesis, Click here to access, 1993. http://lucy.kent.ac.uk/csac/lucy/csacpub/Davies_thesis.

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Lyons, Renee' C. "Contribution as Method: A Book Talk for Foreign-Born American Patriots: Sixteen Volunteer Leaders in the Revolutionary War." Digital Commons@Georgia Southern, 2014. https://digitalcommons.georgiasouthern.edu/cssc/2014/2014/10.

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Constituting a proposal for a book talk associated with the scholarly title Foreign-Born American Patriots: Sixteen Volunteer Leaders of the Revolutionary War, the presenter of this session (and author of the book) will introduce the scholarly work to participants for the purpose of highlighting research based in contribution, rather than interpretation. The author will detail the means by which the investigation of human experience and work product, storylines/patterns, and social cause may provide the context for creative scholarly works. The author will also reveal the unique contribution of Foreign Born American Patriots to historical and Southern Studies discourse, the book serving, up through the date of this proposal, as the only collective work regarding those foreigners who helped the newly formed United States defeat the British Army (many battles fought in the Southern States).
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Tsertou, Athanasia. "Modelling interference in a CSMA/CA wireless network." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/14589.

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Initially, a systematic characterisation of all the possible ways in which two communicating pairs of nodes can interfere with each other is made. Using this as a building block and assuming independence of the stations, an estimate for the network throughput can be derived. The latter proves to be quite accurate for symmetric networks and manages to follow the performance trends in an arbitrary network. Following this, a more detailed Markovian-based mathematical model is proposed for the analysis of the hidden node case. This approach does not rely on common assumptions, such as renewal theory and node synchronisation, and is highly accurate, independently of the system parameters, unlike prior methods. Moreover, the usual decoupling approximation is not adopted; on the contrary, a joint view of the competing stations is taken into consideration. The model is firstly developed based on the assumption that the network stations employ a constant contention window for their backoff process. However, later in the thesis this assumption is relaxed, and performance curves are derived for the case when the stations employ the Binary Exponential Backoff Scheme, as is the case in practice. The Markovian state space is kept relatively small by employing an iterative technique that computes the unknown distributions. The adoption of this technique makes the analysis computationally efficient.
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Books on the topic "CSMC"

1

Kearney, Paul. CSC 364H Notes. Toronto, ON: U. of T. Custom Publishing, 1997.

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(EDT), Chronicle Books. Rhino Csmc Grvs: Virgo. Chronicle Books, 2001.

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(EDT), Chronicle Books. Rhino Csmc Grvs: Sagittarius. Chronicle Books, 2001.

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(EDT), Chronicle Books. Rhino Csmc Grvs: Scorpio. Chronicle Books, 2001.

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(EDT), Chronicle Books. Rhino Csmc Grvs: Capricorn. Chronicle Books, 2001.

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(EDT), Chronicle Books. Rhino Csmc Grvs: Pisces. Chronicle Books, 2001.

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(EDT), Chronicle Books. Rhino Csmc Grvs: Aquarius. Chronicle Books, 2001.

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(EDT), Chronicle Books. Rhino Csmc Grvs: Cancer. Chronicle Books, 2001.

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(EDT), Chronicle Books. Rhino Csmc Grvs: Gemini. Chronicle Books, 2001.

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(EDT), Chronicle Books. Rhino Csmc Grvs: Libra. Chronicle Books, 2001.

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Book chapters on the topic "CSMC"

1

Chung, Soohee, Christiane U. Jakobs, John G. Ormerod, and Donald A. Bryant. "Protein Components of Chlorosomes from Chlorobium Tepidum and Interposon Mutagenesis of csmA and csmC from Chlorobium Vibrioforme 8327D." In Photosynthesis: from Light to Biosphere, 11–16. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0173-5_2.

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Gooch, Jan W. "CSMA." In Encyclopedic Dictionary of Polymers, 185. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_3162.

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Bang, Brandy, Paige L. Baker, Alexis Carpinteri, and Vincent B. Van Hasselt. "CSEC Legislation." In SpringerBriefs in Psychology, 47–50. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-01878-2_9.

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Jenq, Yih-Chyun. "Theoretical Analysis of Slotted ALOHA, CSMA, and CSMA-CD Protocols." In Communications and Networks, 325–46. New York, NY: Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4612-4904-7_14.

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Benslama, Malek, Mohamed Lamine Boucenna, and Hadj Batatia. "Games in CSMA Networks." In Ad Hoc Networks Telecommunications and Game Theory, 93–111. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2015. http://dx.doi.org/10.1002/9781119089377.ch4.

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Thomas, Pradip Ninan, and Elske van de Fliert. "Revisiting CSC Theory." In Interrogating the Theory and Practice of Communication for Social Change, 1–19. London: Palgrave Macmillan UK, 2014. http://dx.doi.org/10.1057/9781137426314_1.

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Thomas, Pradip Ninan, and Elske van de Fliert. "Revisiting CSC Practice." In Interrogating the Theory and Practice of Communication for Social Change, 20–38. London: Palgrave Macmillan UK, 2014. http://dx.doi.org/10.1057/9781137426314_2.

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Pavlovic, Mirjana, and Bela Balint. "Bioengineered CSC Tumors." In Bioengineering and Cancer Stem Cell Concept, 133–37. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-25670-2_11.

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Gotthardt, Claus. "Durchsatz in CSMA-Multihop-Netzen." In Kommunikation in verteilten Systemen, 309–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74570-6_22.

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Giang, Anh Tuan, and Anthony Busson. "Modeling CSMA/CA in VANET." In Analytical and Stochastic Modeling Techniques and Applications, 91–105. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-30782-9_7.

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Conference papers on the topic "CSMC"

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Pervez, Zeeshan, Asad Masood Khattak, Sungyoung Lee, and Young-Koo Lee. "CSMC." In the 5th International Confernece. New York, New York, USA: ACM Press, 2011. http://dx.doi.org/10.1145/1968613.1968650.

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Alim, M. A., M. A. G. Khan, and M. A. Munjer. "CSMC-SPWM modular multilevel converter for HVDC transmission system." In 2016 International Conference on Informatics, Electronics and Vision (ICIEV). IEEE, 2016. http://dx.doi.org/10.1109/iciev.2016.7760105.

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Wang, Xianwei, Qinxian Jiang, and Zhaoliang Wang. "The Mechanical Analysis of CFETR CSMC Non-metallic Components." In 2020 IEEE International Conference on Applied Superconductivity and Electromagnetic Devices (ASEMD). IEEE, 2020. http://dx.doi.org/10.1109/asemd49065.2020.9276059.

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Wang, Xianwei, Xiulian Li, and Zhaoliang Wang. "The Mechanical Performance Analysis of CFETR CSMC Double Current Lead." In 2020 IEEE International Conference on Applied Superconductivity and Electromagnetic Devices (ASEMD). IEEE, 2020. http://dx.doi.org/10.1109/asemd49065.2020.9276355.

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Alim, M. A., M. A. G. Khan, and M. A. Munjer. "Synchronization of different frequency power grids by back-to-back modular multilevel converter with CSMC-SPWM algorithm." In 2016 2nd International Conference on Electrical, Computer & Telecommunication Engineering (ICECTE). IEEE, 2016. http://dx.doi.org/10.1109/icecte.2016.7879564.

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Chen, Guimin, and Shouyin Zhang. "Multistability of Compliant Sarrus Mechanisms." In ASME 2012 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/detc2012-70238.

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Although there are many examples of multistable compliant mechanisms in the literature, most of them are of planar configurations. Considering that a multistable mechanism providing spatial motion could be useful in numerous applications, this paper explores the multistable behavior of the overconstrained spatial Sarrus mechanisms with compliant joints (CSMs). The kinetostatics of CSMs have been formulated based on the pseudo-rigid-body method. The kinetostatic results show that a CSM is capable of exhibiting bistability, tristability, and quadristability. Possible applications of multistable CSMs include deployable structures, static balancing of human/robot bodies and weight compensators.
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Gregory, E. "Lessons Learned on the Development and Manufacture of Internal-Tin Nb3Sn Strand from Work on ITER CSMC and Other Fusion and HEP Applications." In ADVANCES IN CRYOGENIC ENGINEERING: Transactions of the International Cryogenic Materials Conference - ICMC. AIP, 2004. http://dx.doi.org/10.1063/1.1774589.

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Wang Xiaofan, Peter H. J. Chong, and Leong Wai Yie. "Performance comparison of CSMA/CD, CSMA/CA, CSMA/RI, CSMA/PRI and CSMA/PR with BEB." In 2010 5th IEEE Conference on Industrial Electronics and Applications (ICIEA). IEEE, 2010. http://dx.doi.org/10.1109/iciea.2010.5515401.

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Sen, Souvik, Romit Roy Choudhury, and Srihari Nelakuditi. "CSMA/CN." In the sixteenth annual international conference. New York, New York, USA: ACM Press, 2010. http://dx.doi.org/10.1145/1859995.1859999.

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Liu, Ming, Fan Zhang, Kunal Kotian, and Steve Nanney. "Refined Modeling Processes and Compressive Strain Capacity Models." In 2014 10th International Pipeline Conference. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/ipc2014-33202.

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Local buckling generated by excessive bending and/or longitudinal compression is one of the main threats to pipeline integrity. The resistance to local buckling is usually measured by compressive strain capacity (CSC). Extensive work has been performed on the CSC of pipes through both experiments and numerical modeling. Many CSC models have been developed to compute the CSC. The comparison of the existing CSC models often shows (1) large differences in recognized input parameters and their applicable ranges, (2) large differences in computed CSC, especially for pressurized conditions, (3) large differences in recommended safety factors; and (4) inconsistent trends on model conservatism. Refined compressive strain models were developed recently. The development involves comprehensive review of existing CSC models, selecting modeling processes that represent field conditions, sensitivity studies on parameters affecting the CSC, and the model evaluation against experimental data. In this paper, the refined compressive strain models and the key improvement to the modeling processes are summarized.
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Reports on the topic "CSMC"

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Blaskiewicz M. and U. Iriso. How to Use CSEC. Office of Scientific and Technical Information (OSTI), November 2006. http://dx.doi.org/10.2172/1061851.

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Ye, Binglin, Shuling Li, Fengqi Sun, Youfu Fan, Weiguo Chen, and Xiangfu Wang. Effect of full-endoscopic cervical laminectomy and decompression versus anterior cervical decompression with fusion in the treatment of patients with cervical spondylotic myelopathy: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2021. http://dx.doi.org/10.37766/inplasy2021.8.0034.

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Review question / Objective: This systematic review aims to comprehensively assess the efficacy and safety of full-endoscopic cervical laminectomy and decompression versus anterior cervical decompression with fusion in treating cervical spondylotic myelopathy (CSM) patients. Condition being studied: Cervical spondylotic myelopathy (CSM) is a degenerative disease associated with cervical cord compression, which has increased significant health-related social costs and derived disabilities. Anterior cervical discectomy and fusion (ACDF) is the "gold standard" for the treatment of CSM. However, the application of ACDF may cause some complications. Recently, full-endoscopic cervical laminectomy and decompression have shown potential therapeutic effects for CSM. However, no systematic review or meta-analysis has focused on the effects of full-endoscopic cervical laminectomy and decompression in the treatment of CSM. This systematic review aims to comprehensively assess the efficacy and safety of full-endoscopic cervical laminectomy and decompression versus anterior cervical decompression with fusion in treating CSM patients.
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Park, June S., and Keebom Kang. Delay Analysis for Multidimensional Queueing Process in CSMA/CD Local Area Networks. Fort Belvoir, VA: Defense Technical Information Center, September 1991. http://dx.doi.org/10.21236/ada242364.

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Cohen, R. S. CSC Curriculum That Meets the Needs of the FMF. Fort Belvoir, VA: Defense Technical Information Center, January 1991. http://dx.doi.org/10.21236/ada493297.

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Wilson, Stacey. As-Run Thermal Analysis for the CSM-10584 Experiment. Office of Scientific and Technical Information (OSTI), October 2020. http://dx.doi.org/10.2172/1713188.

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Merrill, Albert W., Matthew A. Clark, James Hoffman, Gary L. Gallien, Thomas M. Walsh, Dave Y. Stodden, Sarah A. Lang, and Ragini T. Joshi. Commerce Spectrum Management Advisory Committee (CSMAC) Working Group (WG) 3 Phase 2 Study Summary. Fort Belvoir, VA: Defense Technical Information Center, May 2013. http://dx.doi.org/10.21236/ada590005.

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Miller, D. The reactivity of the thioalkyne, MeSC triple bond CSMe, with ruthenium and tungsten complexes. Office of Scientific and Technical Information (OSTI), June 1990. http://dx.doi.org/10.2172/6971845.

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Ehst, D. A., S. Kim, Y. Gohar, L. Turner, D. L. Smith, and R. Mattas. Application of high temperature ceramic superconductors (CSC) to commercial tokamak reactors. Office of Scientific and Technical Information (OSTI), October 1987. http://dx.doi.org/10.2172/5830528.

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Ghogawala, Zoher, and Melissa R. Dunbar. Comparing Surgical Treatments for Cervical Spondylotic Myelopathy—The CSM-S Trial. Patient-Centered Outcomes Research Institute (PCORI), December 2020. http://dx.doi.org/10.25302/12.2020.ce.13046173.

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Mitchell, Jill. AS-RUN NEUTRONICS EVALUATION FOR THE CSM-10584 EXPERIMENT IN THE ATR. Office of Scientific and Technical Information (OSTI), October 2020. http://dx.doi.org/10.2172/1708851.

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