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Relevant bibliographies by topics / Cucurbitacine E

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Journal articles

Academic literature on the topic 'Cucurbitacine E'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Cucurbitacine E"

1

Jung, Carlo, Benjamin Steuber, and Harald Schwörer. "Lebensmittelvergiftung durch Cucurbitacine." DMW - Deutsche Medizinische Wochenschrift 145, no. 14 (2020): 988–90. http://dx.doi.org/10.1055/a-1163-9741.

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Zusammenfassung Anamnese und klinischer Befund Die 66-jährige Patientin ist wegen einer akuten Lebensmittelvergiftung nach Ingestion von bitteren Zucchini in die Notaufnahme mit Tachykardie, Hypotonie und Somnolenz sowie Exsikkose, Übelkeit, Erbrechen und Hämatochezie eingewiesen worden. Untersuchungen und Diagnose Auffällig waren eine Leukozytose sowie sonografisch eine Kolitis. Die Diagnose einer akuten Lebensmittelvergiftung mit hämorrhagischer Kolitis, ausgelöst durch Cucurbitacin – haltige Zucchini, wurde gestellt. Therapie und Verlauf Nach intravenöser Volumensubstitution stabilisierte s

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2

Vogler, Lars, Andreas Merkenschlager, and Matthias Karl Bernhard. "Cucurbitacin-Vergiftung durch Kürbisse – ein Fallbericht." Kinder- und Jugendmedizin 03, no. 05 (2003): 199–200. http://dx.doi.org/10.1055/s-0037-1617795.

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ZusammenfassungKürbisgewächse können giftige Cucurbitacine enthalten. Bei Cucurbitacinen handelt es sich chemisch um tetrazyklische Triterpene, die in Zier- und Wildkürbissen vorkommen. Durch Kreuzungen oder Rückmutationen können kultivierte Kürbisgewächse erneut die giftige Substanz bilden. Die Vergiftung verursacht vor allem durch Schleimhautreizung akute gastrointestinale Symptome wie Erbrechen, Speichelfluss und Durchfall. Einzelne Todesfälle durch Kreislaufversagen bei Kindern und älteren Menschen wurden beschrieben. Die Therapie erfolgt symptomatisch. Wir berichten von einem 8-jährigen M

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3

Rothenburger, J., and E. Haslinger. "New cucurbitacine glycosides fromGratiola officinalis L." Monatshefte für Chemie - Chemical Monthly 126, no. 12 (1995): 1331–39. http://dx.doi.org/10.1007/bf00807062.

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4

Ku, Jin Mo, Se Hyang Hong, Hyo In Kim, et al. "Cucurbitacin D exhibits its anti-cancer effect in human breast cancer cells by inhibiting Stat3 and Akt signaling." European Journal of Inflammation 16 (January 1, 2018): 1721727X1775180. http://dx.doi.org/10.1177/1721727x17751809.

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Cucurbitacins are triterpenoids commonly found in Cucurbitaceae and Cruciferae and have long been used in traditional medicine. Cucurbitacins demonstrate anti-inflammatory and anti-cancer activities. We investigated whether cucurbitacin D affects viability in breast cancer cells and its mechanism of action. An MTT assay was used to measure the viability of breast cancer cells. Western blot analysis was used to measure the expression of various modulators, such as p-p53, p-Stat3, p-Akt, and p-NF-κB. Doxorubicin and cucurbitacin D affected the viability of MCF7, MDA-MB-231, and SKBR3 cells. Cucu

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5

Cai, Yuee, Xiefan Fang, Chengwei He, et al. "Cucurbitacins: A Systematic Review of the Phytochemistry and Anticancer Activity." American Journal of Chinese Medicine 43, no. 07 (2015): 1331–50. http://dx.doi.org/10.1142/s0192415x15500755.

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Cucurbitacins are highly oxidized tetracyclic triterpenoids that are widely present in traditional Chinese medicines (Cucurbitaceae family), possess strong anticancer activity, and are divided into 12 classes from A to T with over 200 derivatives. The eight most active cucurbitacin components against cancer are cucurbitacin B, D, E, I, IIa, L glucoside, Q, and R. Their mechanisms of action include antiproliferation, inhibition of migration and invasion, proapoptosis, and cell cycle arrest promotion. Cucurbitacins are also found to be the inhibitors of JAK-STAT3, Wnt, PI3K/Akt, and MAPK signali

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6

Schmandke, H. "Cucurbitacine mit Antikrebswirkung in Gurken- und Kürbisgewächsen." Ernährung - Wissenschaft und Praxis 2, no. 5 (2008): 222–26. http://dx.doi.org/10.1007/s12082-008-0179-7.

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7

Wang, Xiaojuan, Mine Tanaka, Herbenya Silva Peixoto, and Michael Wink. "Cucurbitacins: elucidation of their interactions with the cytoskeleton." PeerJ 5 (May 30, 2017): e3357. http://dx.doi.org/10.7717/peerj.3357.

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Cucurbitacins, a class of toxic tetracyclic triterpenoids in Cucurbitaceae, modulate many molecular targets. Here we investigated the interactions of cucurbitacin B, E and I with cytoskeletal proteins such as microtubule and actin filaments. The effects of cucurbitacin B, E and I on microtubules and actin filaments were studied in living cells (Hela and U2OS) and in vitro using GFP markers, immunofluorescence staining and in vitro tubulin polymerization assay. Cucurbitacin B, E and I apparently affected microtubule structures in living cells and cucurbitacin E inhibited tubulin polymerization

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8

Ramezani, Moazzameh, Melika Hasani, Fatemeh Ramezani, and Mahmood Karimi Abdolmaleki. "Cucurbitacins: A Focus on Cucurbitacin E As A Natural Product and Their Biological Activities." Pharmaceutical Sciences 27, no. 1 (2020): 1–13. http://dx.doi.org/10.34172/ps.2020.66.

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For the last years, different types of cucurbitacins have been extracted from various species of Cucurbitaceae family. For this review, all related papers were accumulated by searching electronic databases in the English language, including PubMed, Scopus, and Google Scholar. The keywords of cucurbitacin, cucumber anticancer therapy, cytotoxic effects, chemotherapy, and inhibitor effect were searched until February 2020. According to the result of this review, cucurbitacin E as a tetracyclic triterpenes compound, has been exhibited cell cycle arrest, anti-inflammatory and anticancer activities

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9

Alka Hande, Akhilesh Agrawal, Archana Sonone, Amol Gadbail, Madhuri Gawande, and Swati Patil. "Cucurbitacin: As a candidate against Cytokine Storm in Severe COVID-19 Infection." International Journal of Research in Pharmaceutical Sciences 11, SPL1 (2020): 928–30. http://dx.doi.org/10.26452/ijrps.v11ispl1.3164.

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The severity of SARS-CoV-2 infection is marked by elevated cytokines and chemokines levels like interleukin 6 (IL-6), interferon-gamma (IFN-γ), tumour necrosis factor (TNF), IL-2 and monocyte chemotactic protein 1. This hyperactive pro-inflammatory response identified as the Cytokine Storm (CS) complicates the disease leading to extensive damage of the host tissue, Acute Respiratory Distress Syndrome (ARDS), which further result in multiple organ failures. CS is very critical for the disease progression and is responsible for high death rate in an infected patient. Accordingly, various therape

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10

Sturm. "Analysis of Citrullus colocynthis Cucurbitacine Derivatives with HPLC-SPE-NMR." Scientia Pharmaceutica 77, no. 1 (2009): 254. http://dx.doi.org/10.3797/scipharm.oephg.21.po-55.

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