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Relevant bibliographies by topics / Cyclin C

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Academic literature on the topic 'Cyclin C'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

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Journal articles on the topic "Cyclin C"

1

Ježek, Jan, Daniel G. J. Smethurst, David C. Stieg, et al. "Cyclin C: The Story of a Non-Cycling Cyclin." Biology 8, no. 1 (2019): 3. http://dx.doi.org/10.3390/biology8010003.

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The class I cyclin family is a well-studied group of structurally conserved proteins that interact with their associated cyclin-dependent kinases (Cdks) to regulate different stages of cell cycle progression depending on their oscillating expression levels. However, the role of class II cyclins, which primarily act as transcription factors and whose expression remains constant throughout the cell cycle, is less well understood. As a classic example of a transcriptional cyclin, cyclin C forms a regulatory sub-complex with its partner kinase Cdk8 and two accessory subunits Med12 and Med13 called

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Liu, Zhao-Jun, Takahiro Ueda, Tadaaki Miyazaki, et al. "A Critical Role for Cyclin C in Promotion of the Hematopoietic Cell Cycle by Cooperation with c-Myc." Molecular and Cellular Biology 18, no. 6 (1998): 3445–54. http://dx.doi.org/10.1128/mcb.18.6.3445.

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ABSTRACT Cyclin C, a putative G1 cyclin, was originally isolated through its ability to complement a Saccharomyces cerevisiae strain lacking the G1 cyclin geneCLN1-3. Unlike cyclins D1 and E, the other two G1 cyclins obtained by the same approach and subsequently shown to play important roles during the G1/S transition, there is thus far no evidence to support the hypothesis that cyclin C is indeed critical for the promotion of cell cycle progression. In BAF-B03 cells, an interleukin 3 (IL-3)-dependent murine pro-B-cell line, cyclin C gene mRNA was induced at the G1/S phase upon IL-3 stimulati

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Caldon, C. Elizabeth, C. Marcelo Sergio, Judith Schütte, et al. "Estrogen Regulation of Cyclin E2 Requires Cyclin D1 but Not c-Myc." Molecular and Cellular Biology 29, no. 17 (2009): 4623–39. http://dx.doi.org/10.1128/mcb.00269-09.

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ABSTRACT During estrogen-induced proliferation, c-Myc and cyclin D1 initiate independent pathways that activate cyclin E1-Cdk2 by sequestration and/or downregulation of the CDK inhibitor p21Waf1/Cip1, without significant increases in cyclin E1 protein levels. In contrast, cyclin E2 undergoes a marked increase in expression, which occurs within 9 to 12 h of estrogen treatment of antiestrogen-pretreated MCF-7 breast cancer cells. Both E cyclins are important to estrogen action, as small interfering RNA (siRNA)-mediated knockdown of either cyclin E1 or cyclin E2 attenuated estrogen-mediated proli

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Jänicke, R. U., X. Y. Lin, F. H. Lee, and A. G. Porter. "Cyclin D3 sensitizes tumor cells to tumor necrosis factor-induced, c-Myc-dependent apoptosis." Molecular and Cellular Biology 16, no. 10 (1996): 5245–53. http://dx.doi.org/10.1128/mcb.16.10.5245.

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c-Myc is an important mediator of apoptosis in cytokine- or serum-deprived cells and sensitizes various cell types to tumor necrosis factor alpha (TNF) cytotoxicity. However, downstream mediators of c-Myc-dependent apoptosis are largely unknown. In this study, we investigated whether one or more cyclins which, like c-Myc, are important regulators of the cell cycle are involved in TNF-induced apoptosis downstream of c-Myc. Cyclin D3 and c-Myc levels in HeLa and fibrosarcoma cells correlated with sensitivity of these cells to TNF-induced apoptosis, as both proteins were highly expressed in TNF-s

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Husdal, A., G. Bukholm, and I. R. K. Bukholm. "The Prognostic Value and Overexpression of Cyclin A Is Correlated with Gene Amplification of both Cyclin A and Cyclin E in Breast Cancer Patient." Analytical Cellular Pathology 28, no. 3 (2006): 107–16. http://dx.doi.org/10.1155/2006/721919.

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Deregulation of cell cycle control is a hallmark of cancer. The primary cyclins (A, B1, D1, D3 and E) are crucial for cell cycle progression. Secondary cyclins (C and H) have putative indirect effects on cell cycle propulsion and are not previously evaluated in breast cancer. We have examined protein expression and gene amplification of cyclins in breast carcinomas and correlated the findings with clinical follow-up data. We have previously demonstrated that over-expression of cyclin A is associated with poor prognosis in breast cancer patients. In this study we wanted to evaluate the mechanis

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Sudakin, V., D. Ganoth, A. Dahan, et al. "The cyclosome, a large complex containing cyclin-selective ubiquitin ligase activity, targets cyclins for destruction at the end of mitosis." Molecular Biology of the Cell 6, no. 2 (1995): 185–97. http://dx.doi.org/10.1091/mbc.6.2.185.

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The ubiquitin-mediated degradation of mitotic cyclins is required for cells to exit from mitosis. Previous work with cell-free systems has revealed four components required for cyclin-ubiquitin ligation and proteolysis: a nonspecific ubiquitin-activating enzyme E1, a soluble fraction containing a ubiquitin carrier protein activity called E2-C, a crude particulate fraction containing a ubiquitin ligase (E3) activity that is activated during M-phase, and a constitutively active 26S proteasome that degrades ubiquitinated proteins. Here, we identify a novel approximately 1500-kDa complex, termed t

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Wang, Guo-Li, Xiurong Shi, Elizabeth Salisbury та ін. "Cyclin D3 Maintains Growth-Inhibitory Activity of C/EBPα by Stabilizing C/EBPα-cdk2 and C/EBPα-Brm Complexes". Molecular and Cellular Biology 26, № 7 (2006): 2570–82. http://dx.doi.org/10.1128/mcb.26.7.2570-2582.2006.

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ABSTRACT C/EBPα arrests proliferation of young livers by inhibition of cdk2. In old mice, C/EBPα inhibits growth by repression of E2F-dependent promoters through the C/EBPα-Brm complex. In this paper, we show that cyclin D3-cdk4/cdk6 supports the ability of C/EBPα to inhibit liver proliferation in both age groups. Although cyclin D3-cdk4/cdk6 kinases are involved in the promotion of growth, they are expressed in terminally differentiated cells, suggesting that they have additional functions in these settings. We demonstrate that C/EBPα represents a target for phosphorylation by cyclin D3-cdk4/

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Müller, Carsten, Rong Yang, Gregory Idos, et al. "c-myb Transactivates the Human Cyclin A1 Promoter and Induces Cyclin A1 Gene Expression." Blood 94, no. 12 (1999): 4255–62. http://dx.doi.org/10.1182/blood.v94.12.4255.

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Abstract Cyclin A1 differs from other cyclins in its highly restricted expression pattern. Besides its expression during spermatogenesis, cyclin A1 is also expressed in hematopoietic progenitor cells and in acute myeloid leukemia. We investigated mechanisms that might contribute to cyclin A1 expression in hematopoietic cells. Comparison of cyclin A1 and cyclin A promoter activity in adherent and myeloid leukemia cell lines showed that the cyclin A1 promoter is preferentially active in myeloid cell lines. This preferential activity was present in a small, 335-bp cyclin A1 promoter fragment that

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Müller, Carsten, Rong Yang, Gregory Idos, et al. "c-myb Transactivates the Human Cyclin A1 Promoter and Induces Cyclin A1 Gene Expression." Blood 94, no. 12 (1999): 4255–62. http://dx.doi.org/10.1182/blood.v94.12.4255.424k32_4255_4262.

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Cyclin A1 differs from other cyclins in its highly restricted expression pattern. Besides its expression during spermatogenesis, cyclin A1 is also expressed in hematopoietic progenitor cells and in acute myeloid leukemia. We investigated mechanisms that might contribute to cyclin A1 expression in hematopoietic cells. Comparison of cyclin A1 and cyclin A promoter activity in adherent and myeloid leukemia cell lines showed that the cyclin A1 promoter is preferentially active in myeloid cell lines. This preferential activity was present in a small, 335-bp cyclin A1 promoter fragment that containe

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Leclerc, V., J. P. Tassan, P. H. O'Farrell, E. A. Nigg, and P. Léopold. "Drosophila Cdk8, a kinase partner of cyclin C that interacts with the large subunit of RNA polymerase II." Molecular Biology of the Cell 7, no. 4 (1996): 505–13. http://dx.doi.org/10.1091/mbc.7.4.505.

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A number of cyclins have been described, most of which act together with their catalytic partners, the cyclin-dependent kinases (Cdks), to regulate events in the eukaryotic cell cycle. Cyclin C was originally identified by a genetic screen for human and Drosophila cDNAs that complement a triple knock-out of the CLN genes in Saccharomyces cerevisiae. Unlike other cyclins identified in this complementation screen, there has been no evidence that cyclin C has a cell-cycle role in the cognate organism. Here we report that cyclin C is a nuclear protein present in a multiprotein complex. It interact

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Dissertations / Theses on the topic "Cyclin C"

1

Greene, David. "Cyclin C and Cdk8 in 'Dictyostelium discoideum." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.442460.

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Ponce, Jessica Marie. "Investigating the roles of cyclin C in the mammalian heart." Diss., University of Iowa, 2019. https://ir.uiowa.edu/etd/7015.

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Although pathological alterations in gene expression and mitochondria function in response to cardiac ischemia are well recognized, the mechanisms driving these changes are incompletely understood. Nuclear to mitochondrial communication regulating gene expression and mitochondrial function is a critical process following cardiac ischemic injury. Here we determine that cyclin C, a component of the transcriptional regulator, Mediator complex, directly regulates cardiac and mitochondrial function by modifying mitochondrial fission. We tested the hypothesis that cyclin C has a binary function as a

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Dimova, Nevena Varbinova. "Defining the Ubiquitin and E2-Enzyme Requirements for APC/C-Mediated Degradation of Cyclin B1." Thesis, Harvard University, 2012. http://dissertations.umi.com/gsas.harvard:10401.

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Post-translational modification of proteins with ubiquitin regulates many aspects of cell physiology, including protein degradation. A uniform polyubiquitin chain that is linked through Lys48 has been widely accepted as central for recognition and destruction by the 26S proteasome. Work in more recent years has demonstrated that the repertoire of proteolytic signals may encompass chains of other linkage types, including Lys11-linked ubiquitin chains and short assemblies of mixed linkage. In this dissertation I examine whether catalysis mediated by the Anaphase-Promoting Complex/Cyclosome (APC/

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Höppner, Sabine. "Structure of the Mediator subunit Cyclin C and subunit interaction studies within the Mediator head module." Diss., lmu, 2005. http://nbn-resolving.de/urn:nbn:de:bvb:19-46936.

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Zhang, Suyang. "Mechanism of APC/C activation and substrate specificity in mitosis." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/275479.

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In eukaryotes, cell proliferation and cell cycle transitions are strictly controlled by the anaphase-promoting complex/cyclosome (APC/C). The APC/C is an E3 ubiquitin ligase that regulates chromatid segregation at the metaphase to anaphase transition, exit from mitosis and the establishment and maintenance of G1. The APC/C’s catalytic activity and substrate specificity are controlled by its interactions with two coactivators, Cdc20 and Cdh1. In contrast to Cdh1, APC/C activation by Cdc20 during mitosis requires hyper-phosphorylation of APC/C subunits by cyclin-dependent kinase (Cdk) and polo k

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Schneider, Elisabeth Veronika [Verfasser], Robert [Akademischer Betreuer] Huber, and Adelbert [Akademischer Betreuer] Bacher. "Structural and functional characterization of human protein kinases exemplified on the human cyclin-dependent kinase 8, cyclin C complex / Elisabeth Veronika Schneider. Gutachter: Robert Huber ; Adelbert Bacher. Betreuer: Robert Huber." München : Universitätsbibliothek der TU München, 2012. http://d-nb.info/1035502852/34.

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Schneider, Elisabeth V. [Verfasser], Robert [Akademischer Betreuer] Huber, and Adelbert [Akademischer Betreuer] Bacher. "Structural and functional characterization of human protein kinases exemplified on the human cyclin-dependent kinase 8, cyclin C complex / Elisabeth Veronika Schneider. Gutachter: Robert Huber ; Adelbert Bacher. Betreuer: Robert Huber." München : Universitätsbibliothek der TU München, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:91-diss-20120920-1108725-0-3.

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Desai, Shraddha R. "Role of Protein Kinase C-iota in Glioblastoma." Scholar Commons, 2011. http://scholarcommons.usf.edu/etd/3070.

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The focus of this research was to investigate the role of protein kinase C-iota (PKC-é) in the regulation of Bad function, a pro-apoptotic member of the Bcl-2 family and Cdk7 function, a master cell cycle regulator in glioblastoma. The results were obtained from the human glial tumor derived cell lines, T98G and U87MG. In these cells, PKC-é co-localized and directly associated with Bad as shown by immunofluorescence, immunoprecipitation, and Western blotting. Furthermore, in-vitro kinase activity assay showed that PKC-é directly phosphorylated Bad at phospho specific residues, S112, S136 and

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Silva, Michael Santos. "Flow cytometric of c-FLIPl-mediated regulation of cell cycle and cell population size." Master's thesis, Universidade de Aveiro, 2017. http://hdl.handle.net/10773/22487.

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Mestrado em Bioquímica - Bioquímica Clínica<br>c-FLIP é uma proteína conhecida pela sua capacidade de se ligar ao DISC, onde compete com a procaspase-8 pela interação com FADD. No entanto, existem evidências que a sua isoforma longa consegue regular também o ciclo celular e mecanismos de proliferação. Para além disso, a atividade de c-FLIPL pode ser controlada por fosforilação. Assim, o objetivo deste estudo é perceber como a fosforilação no resíduo de serina 227 nesta proteína afeta a proliferação e ciclo celular. Neste estudo, observamos que a sobre-expressão de c-FLIPL com uma mutação de se

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Roset, i. Huguet Ramon. "Study of the regulation and signalling of cdk2-Cyclin o complexes during apoptosis." Doctoral thesis, Universitat Pompeu Fabra, 2008. http://hdl.handle.net/10803/7199.

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The aim of this thesis is the characterization of a protein involved in apoptosis. Our group has identified an early step common to different forms of intrinsic apoptosis stimuli. This step requires de novo synthesis of a novel Cyclin, Cyclin O, that upon apoptosis induction in lymphoid cells forms active complexes, primarily with Cdk2. Cyclin O expression precedes glucocorticoid and gamma radiation-induced apoptosis in vivo in mouse thymus and its overexpression induces apoptosis in cultured cells. Knocking down the endogenous expression of Cyclin O by shRNA leads to the inhibition of glucoco

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Books on the topic "Cyclin C"

1

Wilson, Amy. Tea, c, d&c: Cycles interrupted. Visual Studies Workshop Press, 1994.

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F, Smith Herbert. Sunday homilies: Cycle C. Alba House, 1991.

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Trust and transform: Cycle C. Shepherds of Christ, 2015.

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Preaching the parables: Cycle C. CSS Pub., 2003.

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Sveriges lantbruksuniversitet. Institutionen fo r ekologi och miljo va rd., ed. Theoretical analyses of C and N cycling in soil. Swedish University of Agricultural Sciences, Dept. of Ecology and Environmental Research, 1987.

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6

Joe, Robinson. Guiding Light: Feed My Soul: Cycle C. Shepherds of Christ, 2010.

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Cowen, Cynthia E. Sermons on the Gospel readings: Cycle C. CSS Pub. co., 2008.

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McCarver, Terry Hazelmai, ed. Cycle C, decorating for Sundays & holy days. Twenty-Third Publications, 1988.

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Kalas, J. Ellsworth. Sermons on the Gospel readings: Cycle C. CSS Pub. Co. Inc., 2003.

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Peterson, Monique. Bike!: Your guide to mountain biking, BMX, road and fast-track racing, C-X racing and more. National Geographic, 2002.

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Book chapters on the topic "Cyclin C"

1

Leclerc, Vincent, and Pierre Léopold. "The cyclin C/Cdk8 kinase." In Progress in Cell Cycle Research. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-5873-6_19.

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Grant, Steven. "Development of Protein Kinase C and Cyclin-Dependent Kinase Inhibitors As Potentiators of Cytotoxic Drug Action in Leukemia." In Combination Cancer Therapy. Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-864-1:061.

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Fulek, Radoslav. "C-Planarity of Embedded Cyclic c-Graphs." In Lecture Notes in Computer Science. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-50106-2_8.

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Du, Xiao-Xia, and Xiao-Dong Su. "Detection of Cyclic Dinucleotides by STING." In c-di-GMP Signaling. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7240-1_6.

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Chambers, Jacob R., and Karin Sauer. "Detection of Cyclic di-GMP Binding Proteins Utilizing a Biotinylated Cyclic di-GMP Pull-Down Assay." In c-di-GMP Signaling. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7240-1_25.

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Campbell, H. E. A. Eddy, and David L. Wehlau. "The Cyclic Group C p." In Modular Invariant Theory. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-17404-9_7.

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Römling, Ute, and Michael Y. Galperin. "Discovery of the Second Messenger Cyclic di-GMP." In c-di-GMP Signaling. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7240-1_1.

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Greaves, T. L., J. D. Cashion, A. L. Benci, N. K. Jaggi, C. Hogan, and A. M. Bond. "Multiple Mössbauer Spectra Data Acquisition with an In-situ Cyclic Voltammetry System." In Hyperfine Interactions (C). Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-010-0281-3_6.

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Borlee, Bradley R., Grace I. Borlee, Kevin H. Martin, and Yasuhiko Irie. "Cyclic di-GMP-Responsive Transcriptional Reporter Bioassays in Pseudomonas aeruginosa." In c-di-GMP Signaling. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7240-1_9.

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Goossens, Nicolas, Sophie Clément, and Francesco Negro. "The HCV particle and its life cycle." In Handbook of Hepatitis C. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-28053-0_2.

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Conference papers on the topic "Cyclin C"

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Ou, Wen-Bin, Christopher D. M. Fletcher, George D. Demetri та Jonathan A. Fletcher. "Abstract 952: Protein kinase C theta (PKCθ) and c-Jun regulate proliferation through cyclin D1 in KIT-independent gastrointestinal stromal tumors". У Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-952.

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Guo, Zheng, Yuning Zhou, Mark Evers, and Qingding Wang. "Abstract 2029: Rictor regulates FBXW7-dependent c-Myc and cyclin E degradation in colorectal cancer." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2029.

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Hongyan, Wang, Li Huan, Qi Yali, Chen Yubing, and Gong Shouliang. "The expressions of β-catenin, c-myc and cyclin D1 in thymoma induced by ionizing radiation." In 2011 International Conference on Human Health and Biomedical Engineering (HHBE). IEEE, 2011. http://dx.doi.org/10.1109/hhbe.2011.6027947.

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Hassan, Sazzad, Niranjan Awasthi, Margaret A. Schwarz, Roderich E. Schwarz, and Urs V. Holzen. "Abstract 1258: Therapeutic potential of the cyclin- dependent kinase inhibitor on c-Myc overexpressing esophageal adenocarcinoma." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1258.

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Hassan, Md Sazzad, Niranjan Awasthi, Jun Li, Margaret A. Schwarz, and Urs von Holzen. "Abstract 975: Antitumor effect of cyclin- dependent kinase inhibitor flavopiridol in c-Myc overexpressing esophageal cancer." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-975.

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Kwan, Suet Ying, Samuel C. Mok, Kwong-Kwok Wong, Rosemarie E. Schmandt, and Karen H. Lu. "Abstract 3559: Ubiquitin C-terminal hydrolase L1 (UCHL1) modulates uterine papillary serous cancer progression through interaction with cyclin B1." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3559.

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Shih-Feng, Yu, and Huang Tze-Sing. "Abstract 3176: Connexin-43 facilitates breast cancer cell proliferation through upregulation of ERK-dependent beta-catenin, c-Myc and cyclin D1 expression." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3176.

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Cai, Tongjian, Zhenghong Zuo, Jin Ding, Dongyun Zhang, Jingxia Li, and Chuanshu Huang. "Abstract 4371: PI3K/Akt/JNK/c-Jun signaling pathway is a mediator for arsenite-induced cyclin D1 expression and cell growth in human bronchial epithelial cells." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-4371.

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Chan, Kwai S., N. Sastry Cheruvu, and Gerald R. Leverant. "Predicting Coating Degradation Under Variable Peak Temperatures." In ASME 1999 International Gas Turbine and Aeroengine Congress and Exhibition. American Society of Mechanical Engineers, 1999. http://dx.doi.org/10.1115/99-gt-381.

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The cyclic oxidation behavior of a CoCrAlY coating on a GTD-111 substrate was characterized by thermal cycling between ambient temperature and a variable peak temperature. The initial peak temperature of the thermal cycle was 954°C (1750°F) and tested for n1 cycles. The peak temperature was then increased to 1010°C (1850°F) for n2 cycles. Finally, the peak temperature was raised again to 1066°C (1950°F) for n3 cycles, where n1, n2, and n3 are in the range of a few hundred to a few thousand cycles. In all three cases, thermal cycles consisting of 55 minutes at the peak temperature and 5 minutes

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Lall, Pradeep, Mahendra Harsha, Jeff Suhling, and Kai Goebel. "Damage Pre-Cursors Based Prognostication of Accrued Damage and Assessment of Operational Readiness of Leadfree Electronics." In ASME 2013 International Technical Conference and Exhibition on Packaging and Integration of Electronic and Photonic Microsystems. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/ipack2013-73251.

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Electronics in high reliability applications may be stored for extended periods of time prior to deployment. Prior studies have shown the elastic modulus and ultimate tensile strength of the SAC leadfree alloys reduces under prolonged exposure to high temperatures [Zhang 2009]. The thermal cycle magnitudes may vary over the lifetime of the product. Long-life systems may be re-deployed several times over the use life of the product. Previously, the authors have identified damage pre-cursors for correlation of the damage progression with the microstructural evolution of damage in second level in

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Reports on the topic "Cyclin C"

1

Strich, Randy. Cyclin C Regulation of the Stress Response and Drug Sensitivity in Breast Cancer. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada382432.

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Dickson, Robert B. Modulation of Cyclin Expression by C-MYC in Malignant and Nonmalignant Mammary Epithelial Cells. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada302398.

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Kandel, Rita A. A Cohert Study of the Relationship Between c-erbB-2 and Cyclin D1 Overexpression, p53 Mutation and/or Protein Accumulation, and Risk of Progression from Benign Breast Disease to Breast Cancer; and Creation of a Bank of Benign Breast Tissue. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada394725.

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Nataro, James P., and David K. Karaolis. Host Immune Response to Bacterial Cyclic Diguanylic Acid (c-di-GMP). Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada533324.

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Fisher, Joshua, Richard Phillips, and Tom Evans. Nutrient Cycle Impacts on Forest Ecosystem Carbon Cycling: Improved Prediction of Climate Feedbacks from Coupled C–Nutrient Dynamics from Ecosystem to Regional Scales. Office of Scientific and Technical Information (OSTI), 2017. http://dx.doi.org/10.2172/1377633.

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Padgett, Richard W. Cell Cycle Regulation by TGFb Signaling in C. elegans. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada398212.

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Padgett, Richard. Cell Cycle Regulation by TGFb Signaling in C. elegans. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada390936.

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Fisher, I. A. Structural stability of 1100{degree}C heated Pd/k during absorption cycling in protium. Office of Scientific and Technical Information (OSTI), 1993. http://dx.doi.org/10.2172/10170331.

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Benninger, L. K. Carbon cycling on the continental margin: Evidence from sediment {sup 14}C and nutrient elements. Final report. Office of Scientific and Technical Information (OSTI), 1998. http://dx.doi.org/10.2172/764715.

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Fisher, I. A. Structural stability of 1100[degree]C heated Pd/k during absorption cycling in protium. [Palladium supported on kieselguhr]. Office of Scientific and Technical Information (OSTI), 1993. http://dx.doi.org/10.2172/6204022.

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