Relevant bibliographies by topics / Cyclomodulin

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'Cyclomodulin'

Author: Grafiati
Published: 6 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "Cyclomodulin"

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Markelova, Natalia N., Elena F. Semenova, Olga N. Sineva, and Vera S. Sadykova. "The Role of Cyclomodulins and Some Microbial Metabolites in Bacterial Microecology and Macroorganism Carcinogenesis." International Journal of Molecular Sciences 23, no. 19 (2022): 11706. http://dx.doi.org/10.3390/ijms231911706.

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A number of bacteria that colonize the human body produce toxins and effectors that cause changes in the eukaryotic cell cycle—cyclomodulins and low-molecular-weight compounds such as butyrate, lactic acid, and secondary bile acids. Cyclomodulins and metabolites are necessary for bacteria as adaptation factors—which are influenced by direct selection—to the ecological niches of the host. In the process of establishing two-way communication with the macroorganism, these compounds cause limited damage to the host, despite their ability to disrupt key processes in eukaryotic cells, which can lead
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Nouri, Roghayeh, Alka Hasani, Kourosh Masnadi Shirazi, et al. "Mucosa-Associated Escherichia coli in Colorectal Cancer Patients and Control Subjects: Variations in the Prevalence and Attributing Features." Canadian Journal of Infectious Diseases and Medical Microbiology 2021 (November 9, 2021): 1–8. http://dx.doi.org/10.1155/2021/2131787.

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Accumulating evidence indicates that specific strains of mucosa-associated Escherichia coli (E. coli) can influence the development of colorectal carcinoma. This study aimed to investigate the prevalence and characterization of mucosa-associated E. coli obtained from the colorectal cancer (CRC) patients and control group. At two referral university-affiliated hospitals in northwest Iran, 100 patients, 50 with CRC and 50 without, were studied over the course of a year. Fresh biopsy specimens were used to identify mucosa-associated E. coli isolates after dithiothreitol mucolysis. To classify the
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Mezerová, Kristýna, Lubomír Starý, Pavel Zbořil, et al. "Cyclomodulins and Hemolysis in E. coli as Potential Low-Cost Non-Invasive Biomarkers for Colorectal Cancer Screening." Life 11, no. 11 (2021): 1165. http://dx.doi.org/10.3390/life11111165.

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The frequent occurrence of E. coli positive for cyclomodulins such as colibactin (CLB), the cytotoxic necrotizing factor (CNF), and the cytolethal distending factor (CDT) in colorectal cancer (CRC) patients published so far provides the opportunity to use them as CRC screening markers. We examined the practicability and performance of a low-cost detection approach that relied on culture followed by simplified DNA extraction and PCR in E. coli isolates recovered from 130 CRC patients and 111 controls. Our results showed a statistically significant association between CRC and the presence of col
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Samba-Louaka, Ascel, Jean-Philippe Nougayrède, Claude Watrin, Eric Oswald, and Frédéric Taieb. "The Enteropathogenic Escherichia coli Effector Cif Induces Delayed Apoptosis in Epithelial Cells." Infection and Immunity 77, no. 12 (2009): 5471–77. http://dx.doi.org/10.1128/iai.00860-09.

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ABSTRACT The cycle inhibiting factor (Cif) belongs to a family of bacterial toxins, the cyclomodulins, which modulate the host cell cycle. Upon injection into the host cell by the type III secretion system of enteropathogenic Escherichia coli (EPEC), Cif induces both G2 and G1 cell cycle arrests. The cell cycle arrests correlate with the accumulation of p21waf1 and p27kip1 proteins that inhibit CDK-cyclin complexes, whose activation is required for G1/S and G2/M transitions. Increases of p21 and p27 levels are independent of p53 transcriptional induction and result from protein stabilization t
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Hsu, Yun, Gregory Jubelin, Frédéric Taieb, Jean-Philippe Nougayrède, Eric Oswald, and C. Erec Stebbins. "Structure of the Cyclomodulin Cif from Pathogenic Escherichia coli." Journal of Molecular Biology 384, no. 2 (2008): 465–77. http://dx.doi.org/10.1016/j.jmb.2008.09.051.

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6

Canizalez-Roman, Adrian, Juan E. Reina-Reyes, Uriel A. Angulo-Zamudio, et al. "Prevalence of Cyclomodulin-Positive E. coli and Klebsiella spp. Strains in Mexican Patients with Colon Diseases and Antimicrobial Resistance." Pathogens 11, no. 1 (2021): 14. http://dx.doi.org/10.3390/pathogens11010014.

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Colon diseases, such as colorectal cancer (CRC), are multifactor diseases that affect more than one million people per year; recently, the microbiota has been associated with an etiologic factor, specifically bacterial cyclomodulin positivity (CM+). Unfortunately, there are no studies from Mexico that detail the presence of bacterial CM+ in patients with colon diseases. We therefore performed a comprehensive study to investigate the associations and prevalence of cyclomodulin-positive Diarrheagenic E. coli (DEC), non-DEC, and Klebsiella spp. strains isolated from Mexican subjects with colon di
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Toro, Tasha B., Julia I. Toth, and Matthew D. Petroski. "The Cyclomodulin Cycle Inhibiting Factor (CIF) Alters Cullin Neddylation Dynamics." Journal of Biological Chemistry 288, no. 21 (2013): 14716–26. http://dx.doi.org/10.1074/jbc.m112.448258.

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8

Buc, Emmanuel, Damien Dubois, Pierre Sauvanet, et al. "High Prevalence of Mucosa-Associated E. coli Producing Cyclomodulin and Genotoxin in Colon Cancer." PLoS ONE 8, no. 2 (2013): e56964. http://dx.doi.org/10.1371/journal.pone.0056964.

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9

Chavez, Carolina Varela, Grégory Jubelin, Gabriel Courties, et al. "The cyclomodulin Cif of Photorhabdus luminescens inhibits insect cell proliferation and triggers host cell death by apoptosis." Microbes and Infection 12, no. 14-15 (2010): 1208–18. http://dx.doi.org/10.1016/j.micinf.2010.09.006.

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Samba-Louaka, Ascel, Jean-Philippe Nougayrède, Claude Watrin, Grégory Jubelin, Eric Oswald, and Frédéric Taieb. "Bacterial cyclomodulin Cif blocks the host cell cycle by stabilizing the cyclin-dependent kinase inhibitors p21waf1and p27kip1." Cellular Microbiology 10, no. 12 (2008): 2496–508. http://dx.doi.org/10.1111/j.1462-5822.2008.01224.x.

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Dissertations / Theses on the topic "Cyclomodulin"

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Mambu, Julien. "Dérégulation de la prolifération cellulaire par Salmonella Typhimurium et implication dans la pathogenèse." Electronic Thesis or Diss., Tours, 2020. http://www.theses.fr/2020TOUR5023.

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L’entéropathogène Salmonella Typhimurium a été mis en cause dans la dérégulation la prolifération cellulaire au niveau de l’intestin après infection. Toutefois, le mécanisme mis en jeu reste peu connu. Salmonella Typhimurium, exprime à sa surface une invasine, Rck, qui interagit et active le récepteur de l’EGF (EpidermalGrowth Factor) à la surface de la cellule cible, conduisant à l’internalisation de la bactérie. Sachant que l’activation du récepteur de EGF régule des processus cellulaires incluant la prolifération-différentiation, ma thèse avait pour objectif de déterminer l’impact de Rck su
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Varela, Chavez Carolina. "Caractérisation fonctionnelle d’une cyclomoduline pro-apoptotique nommée Cif (Cycle Inhibiting Factor) chez les bactéries entomopathogènes du genre Photorhabdus." Montpellier 2, 2009. http://www.theses.fr/2009MON20183.

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Noto, Benjamin [Verfasser], and M. Alexander [Akademischer Betreuer] Schmidt. "Transport des Cyclomodulins Cif in eukaryotische Zellen mit Hilfe von "Cell penetrating peptides" / Benjamin Noto ; Betreuer: M. Alexander Schmidt." Münster : Universitäts- und Landesbibliothek Münster, 2013. http://d-nb.info/1141383748/34.

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Raisch, Jennifer. "Piste infectieuse à Escherichia coli toxinogènes dans le cancer colorectal." Thesis, Clermont-Ferrand 1, 2014. http://www.theses.fr/2014CLF1MM09/document.

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Le cancer colorectal (CCR) est au 3ème rang des cancers les plus répandus dans le monde. Il est bien établi que l'inflammation est un acteur clé de la carcinogenèse colorectale. Parmi les cellules de l'infiltrat inflammatoire tumoral, les macrophages jouent un rôle moteur dans la carcinogenèse en sécrétant un arsenal de cytokines/chimiokines et facteurs protumoraux tels que la prostaglandine E2 (PGE2), produit enzymatique de la cyclo-oxygénase 2 (COX-2). Des dysbioses du microbiote intestinal, en particulier une augmentation d’Escherichia coli (E. coli) appartenant au phylogroupe B2 et product
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