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Books on the topic 'CysLT2'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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1

James, Grant. Dermoid cyst of the ovary. [S.l: s.n., 1987.

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2

A, Zeeb Barbara, Smol J. P, and Wilkinson Anna N, eds. Atlas of chrysophycean cysts. Dordrecht: Kluwer Academic Publishers, 1995.

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3

Orr, Tamra. Ovarian tumors and cysts. New York: Rosen Pub. Group, 2009.

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4

Duff, Katharine E., Barbara A. Zeeb, and John P. Smol. Atlas of Chrysophycean Cysts. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-017-0809-8.

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5

Wilkinson, Anna N., Barbara A. Zeeb, and John P. Smol. Atlas of Chrysophycean Cysts. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-017-0811-1.

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6

NATO Advanced Study Institute on Cyst Nematodes (1985 Martina Franca, Italy). Cyst nematodes. New YorK: Plenum Press, 1986.

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7

NATO Advanced Study Institute on Cyst Nematodes (1985 Martina Franca). Cyst nematodes. New York: Plenum in cooperation with NATO Scientific Affairs Division, 1986.

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8

NATO Advanced Study Institute on Cyst Nematodes (1985 Martina Franca, Italy). Cyst nematodes. New YorK: Plenum Press, 1986.

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9

Lamberti, F., and C. E. Taylor, eds. Cyst Nematodes. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2251-1.

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10

Perry, R. N., M. Moens, and J. T. Jones, eds. Cyst nematodes. Wallingford: CABI, 2018. http://dx.doi.org/10.1079/9781786390837.0000.

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11

Renick, Barry Michael. A survey of jaw cysts. Toronto: B.M. Renick, 1987.

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12

Shiffman, Melvin A., and Mervin Low, eds. Biofilm, Pilonidal Cysts and Sinuses. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-03077-3.

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13

Shear, Mervyn. Cysts of the oral regions. 3rd ed. Oxford: Wright, 1992.

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14

Sciubba, James J. Tumors and cysts of the jaws. Washington, D.C: Armed Forces Institute of Pathology, 2001.

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15

Powell, A. J., ed. A Stratigraphic Index of Dinoflagellate Cysts. Dordrecht: Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2386-0.

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16

Carris, Lori Marie. Fungi colonizing cysts of Heterodera glycines. Urbana, Ill: University of Illinois at Urbana-Champaign, College of Agriculture, Agricultural Experiment Station, U.S. Dept. of Agriculture, 1989.

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17

Sharma, S. B., ed. The Cyst Nematodes. Dordrecht: Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-015-9018-1.

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18

Raimondi, Anthony J., Maurice Choux, and Concezio Di Rocco, eds. Intracranial Cyst Lesions. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-7281-7.

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19

Sporopollenin dinoflagellate cysts: Their morphology and interpretation. Dallas, Tex: Obtain from R.T. Clarke, American Association of Stratigraphic Palynologists Foundation, c/o Mobil Research & Development Corp., 1985.

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20

Shear, Mervyn, and Paul Speight, eds. Cysts of the Oral and Maxillofacial Regions. Oxford, UK: Blackwell Munksgaard, 2007. http://dx.doi.org/10.1002/9780470759769.

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21

Shear, Mervyn. Cysts of the oral and maxillofacial regions. 4th ed. Oxford: Blackwell Pub., 2007.

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22

Shear, Mervyn. Cysts of the oral and maxillofacial regions. 4th ed. Oxford: Blackwell Pub., 2007.

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23

Lindmark, Donald G. Cytochemical methods for assessing Giardia cysts viability. Research Triangle Park, NC: U.S. Environmental Protection Agency, Health Effects Research Laboratory, 1988.

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24

Riding, James B. Jurassic and lowermost Cretaceous dinoflagellate cyst biostratigraphy of the Russian Platform and northern Siberia, Russia. [Dallas, TX]: American Association of Stratigraphic Palynologists Foundation, 1999.

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25

Sharma, Renu. Pathology of cyst nematodes. New Delhi: Malhotra Pub. House, 1988.

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26

Firsova, A. D. Atlas t︠s︡ist khrizofitovykh vodorosleĭ ozera Baĭkal. Novosibirsk: Nauka, 2006.

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27

Hansen, Peer. Chrysophyte stomatocysts in the Azores: Biogeographical implications and 110 new morphotypes. Copenhagen: Council for Nordic Publications in Botany, 2001.

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28

Firsova, A. D. Atlas t︠s︡ist khrizofitovykh vodorosleĭ ozera Baĭkal. Novosibirsk: Nauka, 2006.

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29

Neumann, Christian. Diversitäts- und Häufigkeitsmuster kalkiger Dinoflagellatenzysten aus dem Alb der Bohrung Kirshrode II (zentrales Niedersächsisches Becken, NW-Deutschland) und ihre möglichen Steuerungsmechanismen. Berlin: Selbstverlag Fachbereich Geowissenschaften, FU, 1999.

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30

Shahina, F. Cyst nemotodes of Pakistan: Heteroderidae. Karachi, Pakistan: National Nemotological Research Centre, University of Karachi, 1995.

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31

Inglis, Debbie. Pea cyst nematode: Biology and prevention. [Pullman, Wash.]: Washington State University, Cooperative Extension, 1998.

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32

Poulsen, Niels Erik. Dinoflagellate cysts from marine Jurassic deposits of Denmark and Poland. [Houston, Tex.]: American Association of Stratigraphic Palynologists Foundation, 1996.

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33

Smith, Gay. Biochemical and histochemical studies on odontogenic cysts and their fluids. Birmingham: University of Birmingham, 1986.

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34

Huettel, Robin L. Pest risk analysis for pea cyst nematode. Riverdale, Md.?: APHIS, 1993.

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35

Wilson, Graeme J. Paleocene and Eocene dinoflagellate cysts from Waipawa, Hawkes Bay, New Zealand. Lower Hutt: New Zealand Dept. of Scientific and Industrial Research, 1988.

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36

Wilson, Graeme J. Paleocene and Eocene dinoflagellate cysts from Waipawa, Hawkes Bay, New Zealand. Lower Hutt, N.Z: New Zealand Geological Survey, 1988.

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37

Gabor, Kovacs. A guide to the polycystic ovary: Its effects on health and fertility. Shrewsbury, England: Tfm Publ., 2002.

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38

Moldwin, Robert M. The interstitial cystitis survival guide: Your guide to the latest treatment options and coping strategies. Oakland, Calif: New Harbinger Publications, 2000.

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39

Systematics Of Cyst Nematodes Nematoda Heteroderinae. Brill Academic Publishers, 2010.

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40

Buchanan, Ian A., and Gabriel Zada. Rathke’s Cleft Cysts. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0017.

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Cystic lesions involving the sella have a distinct differential diagnosis including Rathke cleft cyst, cystic pituitary adenoma, craniopharyngioma, arachnoid cyst, and epidermoid among other entities. Workup includes not only cranial imaging but also endocrine evaluation for pituitary dysfunction and ophthalmologic evaluation to assess for visual deficits that may not be immediately apparent to the patient at presentation. Rathke cleft cysts are common and may be found in 20% of autopsy specimens. However, symptomatic Rathke cleft cysts are rare, and surgical decisions should be made judiciously based on preoperative symptoms and workup. Endonasal transphenoidal approach for cyst fenestration is a common surgical management technique for symptomatic lesions. Complete resection of the Rathke cleft cyst wall is not required.

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41

Shah, Ashish H., and Jacques J. Morcos. Dermoid/Epidermoid Tumors. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0018.

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Epidermoid tumors of the central nervous system are typically found in the cerebellopontine angle or parasellar space and comprise approximately 1% of all intracranial tumors. Dermoid cysts tend to occur in midline locations. Both are derived from embryonal tissue and have classic imaging findings on computed tomography and magnetic resonance imaging. Epidermoid tumors and dermoid cysts are benign and grow slowly, although epidermoid cysts can undergo malignant transformation. Surgical decisions and approaches are based on the presenting symptoms and anatomic location of the tumor. Mollaret meningitis is a unique complication of these cysts and may occur if the cyst ruptures preoperatively or during surgery.

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42

B, Sharma S., ed. The cyst nematodes. Dordrecht: Kluwer Academic Publishers, 1998.

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43

Sharma, S. B. The Cyst Nematodes. Sharma S B, 2010.

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44

Arachnoid Cysts. Elsevier, 2018. http://dx.doi.org/10.1016/c2015-0-07024-1.

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45

Arachnoid Cysts. Elsevier, 2018. http://dx.doi.org/10.1016/c2017-0-01879-7.

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46

Falomo, Eniola T. Cyst/Abscess Aspiration. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0053.

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Aspiration is the process of draining a fluid-filled structure, such as a cyst or an abscess, and is typically performed under ultrasound guidance. Cyst aspiration can be done for symptomatic relief (in patients with associated pain or tenderness), and for cases of presumed cysts with sonographic features that are not entirely diagnostic of a cyst. In the latter case, aspiration is performed to help distinguish between a complicated cyst and a solid mass. This chapter, appearing in the section on interventions and surgical procedures, reviews the procedural protocols and pitfalls, clinical management, and imaging follow-up involved in a cyst or abscess aspiration procedure. Topics discussed include indications for performing a cyst or abscess drainage procedure, procedural steps, post-procedure follow-up, and appropriate management of complex masses.

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47

Kettler, Mark D. Circumscribed Mass: Cysts, Complicated Cysts, Clustered Microcysts. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0016.

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Benign cysts can occur in women of all ages, but they have a predilection for women in their 40s and 50s, and are far less common in younger and older women. Most cysts arise at the level of the terminal duct-lobular unit (TDLU); less commonly, cysts are caused by ectasia of central ducts. This chapter, appearing in the section on circumscribed mass, reviews the key clinical and imaging features, differential diagnosis, and management recommendations for primary cystic masses, including simple cysts, complicated cysts, and clustered microcysts. Careful assessment of sonographic features usually allows a definitive diagnosis of these benign entities that do not typically require tissue sampling. Some complex masses containing fluid and/or cystic-appearing components may require tissue sampling for diagnosis.

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48

Dermoid cyst of the ovary. [S.l: s.n., 1987.

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49

Kühn, Wolfgang, and Gerd Walz. The molecular basis of ciliopathies and cyst formation. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0303.

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Abnormalities of the cilium, termed ‘ciliopathies’, are the prime suspect in the pathogenesis of renal cyst formation because the gene products of cystic disease-causing genes localize to them, or near them. However, we only partially understand how cilia maintain the geometry of kidney tubules, and how abnormal cilia lead to renal cysts, and the diverse range of diseases attributed to them. Some non-cystic diseases share pathology of the same structures. Although still incompletely understood, cilia appear to orient cells in response to extracellular cues to maintain the overall geometry of a tissue, thereby intersecting with the planar cell polarity (PCP) pathway and the actin cytoskeleton. The PCP pathway controls two morphogenetic programmes, oriented cell division (OCD) and convergent extension (CE) through cell intercalation that both seem to play a critical role in cyst formation. The two-hit theory of cystogenesis, by which loss of the second normal allele causes tubular epithelial cells to form kidney cysts, has been largely borne out. Additional hits and influences may better explain the rate of cyst formation and inter-individual differences in disease progression. Ciliary defects appear to converge on overlapping signalling modules, including mammalian target of rapamycin and cAMP pathways, which can be targeted to treat human cystic kidney disease irrespective of the underlying gene mutation.

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50

Ong, Albert C. M., and Timothy Ellam. Autosomal dominant polycystic kidney disease. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0307_update_001.

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Autosomal dominant polycystic kidney disease (ADPKD) is responsible for up to 10% of prevalent patients with end-stage renal disease (ESRD). It is characterized by the enlargement of multiple bilateral renal cysts, present in almost all patients by their fifth decade. Loin pain is a common symptom that may be caused by cyst growth, intracyst haemorrhage, nephrolithiasis, or infection. Gross haematuria is also a common feature, but usually settles spontaneously. Excretory impairment develops after extensive cystic change has occurred and progresses to ESRD in half of all affected patients by the age of 60. However, the onset of cystic change and rate of renal functional decline are highly variable between individuals. ADPKD associated with the PKD1 gene has an earlier average age of cyst development and ESRD than PKD2, but the two cannot be distinguished on clinical grounds. Polycystins 1 and 2 are expressed in various organs and extrarenal disease may be the presenting feature. Intracranial aneurysms are five times more common in patients with ADPKD, but rupture is infrequent. Liver cysts are present in most patients and may be complicated by haemorrhage or infection, though liver failure is very rare. Massive hepatic cystic disease is confined to women, reflecting stimulatory effects of oestrogen on hepatic cyst growth. Cardiovascular disease is the leading cause of death in ADPKD and vascular dysfunction is present in many patients even before the development of excretory impairment. However, despite the multisystem manifestations of ADPKD, survival from ESRD is better for patients with ADPKD than for other non-diabetic causes of kidney failure.

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