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Dissertations / Theses on the topic 'Cytochrom bc1'

Author: Grafiati

Published: 24 April 2022

Last updated: 30 July 2025

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1

Ritter, Michaela. "Cytochrom bc1 eine Studie zum Elektronentransfer der bc1-Komplexe des Bakteriums Paracoccus denitrificans und der Hefe Saccharomyces cerevisiae mittels elektrochemisch induzierter Differenzspektroskopie /." [S.l. : s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=971616574.

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Lange, Christian. "Wechselwirkungen des Cytochrom-bc1-Komplexes aus Saccharomyces cerevisiae mit seinen Substraten Cytochrom c und Ubichinon sowie mit Membranlipiden." [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=964744287.

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3

Baer, Kimberly Kay. "Protein Coevolution and Coadaptation in the Vertebrate bc1 Complex." Diss., CLICK HERE for online access, 2007. http://contentdm.lib.byu.edu/ETD/image/etd1994.pdf.

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4

Vallières, Cindy. "Agents antimicrobiens ciblant le complexe III de la chaîne respiratoire mitochondriale : caractérisation de nouveaux inhibiteurs et étude du développement des résistances." Phd thesis, Université Paris Sud - Paris XI, 2012. http://tel.archives-ouvertes.fr/tel-00840029.

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Des inhibiteurs du complexe bc1 de la chaîne respiratoire mitochondriale ont été développés comme agents antimicrobiens pour lutter contre des pathogènes de l'Homme et de plantes. Ces drogues ciblent les poches catalytiques Qo et Qi formées par le cytochrome b. La comparaison de séquences de cette protéine montre que les sites Qo et Qi sont bien conservés entre les organismes mais qu'il existe toutefois des variations qui pourraient expliquer leur différence de sensibilité aux drogues. A l'aide du modèle levure S. cerevisiae, nous avons étudié les déterminants de la résistance/sensibilité natu

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5

Gordon, James Alexander. "Design and synthesis of novel cytochrome bc1 inhibitors for the treatment of toxoplasmosis." Thesis, University of Leeds, 2018. http://etheses.whiterose.ac.uk/22638/.

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Toxoplasmosis is a highly prolific and neglected disease, and current treatments fail to adequately address the challenges it presents. As such development of new more efficacious treatments, tailored to the complex nature of the infection, is required. The causative parasite Toxoplasma gondii is a resilient and resourceful organism, adept at invasion, avoidance and survival within its host. The cytochrome bc1 complex has been proven to be a validated target for the treatment of a number of apicomplexan parasitic diseases, including both malaria and toxoplasmosis. Recent work has also identifi

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6

Pacorel, Benedicte. "Targeting the Hemoglobin Degradation Pathway and the Cytochrome bc1 Complex of Plasmodium Falciparum Malaria." Thesis, University of Liverpool, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.507704.

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7

Garcia, Daniel. "Le cytochrome tétrahémique du centre réactionnel de Rhodopseudomonas viridis, sulfoviridis et de Roseobacter denitrificans : structure, fonction et couplage avec le cytochrome BC1." Aix-Marseille 2, 1994. http://www.theses.fr/1994AIX22062.

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Nous avons determine le potentiel de demi-reduction et l'orientation par rapport a la membrane de chaque heme du cytochrome tetrahemique du centre reactionnel de trois bacteries photosynthetiques: rhodopseudomonas sulfoviridis mesophile et thermophile et roseobacter denitrificans. Les resultats obtenus montrent qu'il existe une forte similitude entre le cytochrome des souches de rhodopseudomonas sulfoviridis (h#1: +390 mv, 75 ; h#2: +310 mv, 56 ; l#1: +72 mv, 58 ; l#2: -40 mv, 72,5) et celui de rhodopseudomonas viridis deja bien caracterise (h#1: +380 mv, 75 ; h#2: +310 mv, 55 ; l#1: +20 mv, 6

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8

Davoudi, Cedric-Farhad [Verfasser]. "Regulation and assembly of the cytochrome bc1-aa3 supercomplex in Corynebacterium glutamicum / Cedric-Farhad Davoudi." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2019. http://d-nb.info/1189416115/34.

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9

Largueze, Jean-Baptiste. "Modèles membranaires biomimétiques pour l'incorporation du cytochrome bc1 des chaînes de transfert d'électrons photosynthétiques de Rhodobacter sphaeroides." Compiègne, 2011. http://www.theses.fr/2011COMP1927.

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La formation de bicouches lipidiques supportées est réalisée sur la surface des pores d’une électrode d’alumine nanoporeuse. Trois types de modèles membranaires ont été étudiés : une membrane reposant directement sur l’alumine et deux modèles découplés du support, soit par un assemblage biotine/streptavidine, soit par des polymères de PEG2000. La formation au sein des pores des différents modèles passe par l’utilisation de liposomes et une fusion déclenchée par l’emploi d’un agent fusogène. La caractérisation de la membrane est alors suivie par la réduction électrochimique d’ubiquinone incorpo

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10

Cedeno, Diana. "Synthesis of UQ10 Analogs, Measurement of their Midpoint Potentials and their Effects on the Activity of WT and T61V bc1 Complexes from Rhodobacter sphaeroides." Diss., The University of Arizona, 2010. http://hdl.handle.net/10150/195424.

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Cytochrome bc1 (Complex III) is an important enzyme that takes part in the respiratory electron transport chain in vertebrates, yeast, and many bacteria. The complex exists as a dimer, in which each monomer contains three catalytic subunits: cytochrome c1, cytochrome b and the Rieske iron-sulfur protein or ISP. Within the inner mitochondrial membranes of eukaryotes, Complex III catalyzes the transfer of two electrons from ubiquinol (UQH2) to cytochrome c, a water-soluble protein, through a process called the modified Q-cycle mechanism. Under very specific conditions, such as mutations within c

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11

Damadeu, Kouemo Laura. "Assessing the in vitro efficacy of in silico designed compounds targeting the malarial Qi site of cytochrome bc1." Diss., University of Pretoria, 2009. http://hdl.handle.net/2263/67749.

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Plasmodium falciparum is the causative agent of the most commonly fatal form of malaria in Africa with annual deaths of more than 300 000. The rapid development and spread of antimalarial drug resistance by the parasite have stimulated research into the development of new drug classes. Target-based drug discovery have been used as a prominent and efficient tool to identify lead drugs. Reports suggest that selectively inhibiting the parasite mitochondrial electron transport could be a potential treatment effective at multi-stages of the parasite life cycle. Inhibitors of cytochrome bc1 (Cyt bc1

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12

Lawrenson, Alexandre. "Antimalarial drug design : targeting the Plasmodium falciparum cytochrome bc1 complex through computational modelling, chemical synthesis and biological testing." Thesis, University of Liverpool, 2012. http://livrepository.liverpool.ac.uk/7719/.

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Malaria is a life-threatening disease which is responsible for roughly one million deaths annually. Previous successes in attempting to eradicate the disease have only been short lived, owing to the increased development of resistance in the parasite. There is a continued need for novel compounds which act at novel therapeutic targets, with the Plasmodium falciparum cytochrome bc1 complex (Pfbc1) representing one such target. Its inhibition halts the biochemical generation of ATP, thus resulting in parasite cell death. Work described in this thesis was concerned with utilising molecular modell

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13

Zeng, Hui [Verfasser], Hartmut [Akademischer Betreuer] Michel, Klaas Martinus [Gutachter] Pos, and Hartmut [Gutachter] Michel. "Structural and functional characterization of cytochrome c oxidase, cytochrome bc1 complex and heme A synthase from Aquifex aeolicus / Hui Zeng ; Gutachter: Klaas Martinus Pos, Hartmut Michel ; Betreuer: Hartmut Michel." Frankfurt am Main : Universitätsbibliothek Johann Christian Senckenberg, 2020. http://d-nb.info/1210555727/34.

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14

Ruffolo, Salvatore C. "Regulation of cytochrome c release from mitochondria during apoptosis by BCL-2 family members." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84319.

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Apoptosis is a stringently regulated programmed cell death pathway by which a cell will kill itself in response to extensive damage, stress or a targeted signal. It is an important process in development, which requires the elimination of superfluous cells, as well as the regulation of mitotically active cells in a fully developed organism. The key regulators at the heart of the apoptotic pathway are a group of proteins known as the BCL-2 family, containing both pro- and anti-apoptotic members. These proteins are intimately involved in mitochondrial and endoplasmic reticulum dysfunction

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15

Gruschke, Steffi. "Early steps in the biogenesis of the bc1 complex in yeast mitochondria : The role of the Cbp3-Cbp6 complex in cytochrome b synthesis and assembly." Doctoral thesis, Stockholms universitet, Institutionen för biokemi och biofysik, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-81033.

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The inner membrane of mitochondria harbors the complexes of the respiratory chain and the ATP synthase, which perform the key metabolic process oxidative phosphorylation. These complexes are composed of subunits from two different genetic origins: the majority of constituents is synthesized on cytosolic ribosomes and imported into mitochondria, but a handful of proteins, which represent core catalytic subunits, are encoded in the organellar DNA and translated on mitochondrial ribosomes. Using yeast as a model organism, I investigated the mitochondrial ribosomal tunnel exit, the region of the r

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16

Castellani, Michela [Verfasser], Bernd [Akademischer Betreuer] Ludwig, and Klaas Martinus [Akademischer Betreuer] Pos. "Substrate binding does not only mean catalysis: internal regulation in the cytochrome bc1 complex from Paracoccus denitrificans / Michela Castellani. Gutachter: Bernd Ludwig ; Klaas Martinus Pos." Frankfurt am Main : Univ.-Bibliothek Frankfurt am Main, 2011. http://d-nb.info/104419510X/34.

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17

Amyot, Suzelle M. "The role of amino acids in the transmembrane and extra-membranous domains of the S. cerevisiae iron sulfur protein in its activity and stability in the cytochrome bc1̳ complex." Morgantown, W. Va. : [West Virginia University Libraries], 1998. http://etd.wvu.edu/templates/showETD.cfm?recnum=379.

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Thesis (M.S.)--West Virginia University, 1998.<br>Title from document title page. On t.p. "1̳" is subscript. Document formatted into pages; contains xi, 49 p. : ill. Includes abstract. Includes bibliographical references (p. 46-48).

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18

Peterson, Sabrina. "Effects of apiaceous vegetable constituents on CYP1A2 activity in humans and a yeast expression system : implications for CYP1A2-activated procarcinogens /." Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/6612.

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19

Brichese, Laetitia. "Phosphorylation de BCL-2 en réponse au traitement par le paclitaxel : mécanismes moléculaires impliqués et conséquences sur sa fonction anti-apoptotique." Toulouse 3, 2002. http://www.theses.fr/2002TOU30083.

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20

Thomenius, Michael James. "B-CELL LYMPHOMA-2 PROTEIN FAMILY, APOPTOSIS AND THE ENDOPLASMIC RETICULUM." Case Western Reserve University School of Graduate Studies / OhioLINK, 2004. http://rave.ohiolink.edu/etdc/view?acc_num=case1080679967.

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21

Corcos, Laurent. "Etude de la regulation de l'expression de genes codant pour des cytochromes p450 dans les cellules d'hepatome de rat." Paris 6, 1988. http://www.theses.fr/1988PA066164.

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Le foie assure la detoxication et, dans certains cas, l'activation des xenobiotiques. L'aflatoxine b1 (afb1) peut etre activee en produits cytotoxiques par certains enzymes hepatiques, les cytochromes p-450. Les cellules differenciees derivees de l'hepatome de rat h35 expriment in vitro un ensemble de fonction hepatiques, dont les cytochromes p-450 specifiques et sont sensibles a l'afb1. Mise au point d'un systeme selectif permettant d'isoler, a partir de celules differenciees d'hepatome, avec une haute efficacite et en une seule etape, des clones de cellules resistantes a afb1. Les genes coda

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22

Meissonnier, Guylaine. "Effets toxiques de l'aflatoxine b1 et de la toxine t-2 sur les systèmes de défenses métaboliques et immunitaires chez le porc, évaluation des effets protecteurs de glucomannanes." Toulouse 3, 2007. http://www.theses.fr/2007TOU30153.

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L'aflatoxine B1 (AFB1) et la toxine T-2 sont des mycotoxines, métabolites secondaires de micromycètes qui peuvent contaminer les denrées alimentaires notamment les céréales. Les objectifs de nos travaux ont été de déterminer chez le porc, une espèce cible et sensible aux mycotoxines, les effets toxiques de l'AFB1 et de la toxine T-2 sur deux systèmes de défense, les enzymes de biotransformation et l'immunité. De plus, nous avons évalué in vivo l'effet protecteur d'un potentiel liant de toxine. Chez le porc, l'exposition à l'AFB1 aux doses testées n'a pas provoqué de manifestations cliniques ma

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23

Lefebvre, Jonathan. "Etude des propriétés apoptotiques du récepteur tyrosine kinase Met." Thesis, Lille 1, 2011. http://www.theses.fr/2011LIL10151/document.

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Le récepteur Met et son ligand l’HGF/SF sont essentiels pour le développement embryonnaire tandis que la dérégulation de la signalisation du récepteur Met est associée à la progression tumorale. Activé par son ligand, Met induit un large panel de réponses biologiques telles que la survie cellulaire, la migration ou la prolifération. En revanche, le fragment p40 Met issu du clivage du récepteur par les caspases participe à l’apoptose. Cette dualité fonctionnelle est caractéristique de la famille des récepteurs à dépendance. Bien que la signalisation positive des RTK soit bien décrite, les mécan

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24

Pietras, Rafał. "Dystrybucja przestrzenna białek opisana przez intermolekularne oddziaływania magnetyczne w układzie cytochrom c_{2} — cytochrom bc_{1}." Praca doktorska, 2015. http://ruj.uj.edu.pl/xmlui/handle/item/42244.

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25

Lange, Christian [Verfasser]. "Wechselwirkungen des Cytochrom-bc1-Komplexes aus Saccharomyces cerevisiae mit seinen Substraten Cytochrom c und Ubichinon sowie mit Membranlipiden / von Christian Lange." 2001. http://d-nb.info/964744287/34.

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26

Anderka, Oliver [Verfasser]. "Strukturelle und funktionelle Untersuchungen am Cytochrom-bc1-Komplex aus Paracoccus denitrificans / von Oliver Anderka." 2005. http://d-nb.info/97778116X/34.

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27

Kleinschroth, Thomas [Verfasser]. "Untersuchung zur Struktur-Funktionsbeziehung des Cytochrom-bc1-Komplexes aus Paracoccus denitrificans / von Thomas Kleinschroth." 2009. http://d-nb.info/992453704/34.

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28

Ritter, Michaela [Verfasser]. "Cytochrom bc1 : eine Studie zum Elektronentransfer der bc1-Komplexe des Bakteriums Paracoccus denitrificans und der Hefe Saccharomyces cerevisiae mittels elektrochemisch induzierter Differenzspektroskopie / von Michaela Ritter." 2004. http://d-nb.info/971616574/34.

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29

Pálsdóttir, Hildur [Verfasser]. "Structure-function relationships in the cytochrome bc1 complex from Saccharomyces cerevisiae / von Hildur Pálsdóttir." 2005. http://d-nb.info/974935263/34.

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30

Yin, Ying. "The role of protein subunits in superoxide generation by the cytochrome bc1 complex from Rhodobacter Sphaeroides." 2009. http://digital.library.okstate.edu/etd/Yin_okstate_0664D_10615.pdf.

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31

Borek, Arkadiusz. "Mechanizm produkcji anionorodnika ponadtlenkowego przez cytochrom $bc_{1}$ badany poprzez modyfikację poszczególnych etapów przepływu elektronów w łańcuchach kofaktorów." Praca doktorska, 2015. http://ruj.uj.edu.pl/xmlui/handle/item/42898.

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Cytochrom $\mathit{bc}_{1}$ jest białkiem łańcucha transportu elektronów. Jego działanie związane jest z budowaniem siły protonomotorycznej służącej do produkcji ATP. Oprócz reakcji katalitycznych (które konserwują energię), w pewnych warunkach mogą zachodzić reakcje uboczne, które prowadzą do dyssypacji energii. W reakcjach katalitycznych energia reakcji utleniania chinolu jest wykorzystywana do translokacji protonów w obrębie cytochromu $\mathit{bc}_{1}$. W tym przypadku, w miejscu katalitycznym Qo następuje rozdział dwóch elektronów dostarczanych przez chinol, które przepływają przez dwa od

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32

Klingen, Astrid [Verfasser]. "Structure based theoretical characterisation of the redox dependent titration behaviour of cytochrome bc1 / vorgelegt von Astrid Klingen." 2007. http://d-nb.info/983252599/34.

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33

Traer, Elie. "Regulation of cytochrome C release in UV-induced apoptosis." 2006. http://edissertations.library.swmed.edu/pdf/TraerE051606/TraerElie.pdf.

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34

Nguyen, Bao Linh Tran. "Structural Driving Factors for the Coupled Electron and Proton Transfer Reactions in Mitochondrial Cytochrome BC1 Complex: Binding Geometries of Substrates and Protonation States of Ionizable Amino Acid Side Chains Near Qi and Qo Sites." 2013. http://digital.library.duq.edu/u?/etd,162288.

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Coupled electron and proton transfer (CEPT) events are fundamental for many bioenergetic conversions that involve redox reactions. Understanding the details underlying CEPT processes will advance our knowledge of (1) how nature regulates energy conversion; (2) our strategies for achieving renewable energy sources; (3) how to cope with CEPT dysfunction diseases. Studies of the detailed mechanism(s) of CEPT in biological systems is challenging due to their complex nature. Consequently, controversies between the concerted and sequential mechanism of CEPT for many systems remain. This dissertation

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35

Chen, Tsung-Chieh, and 陳宗杰. "Morphological Changes and Cytochrome c-, Bcl-2-, and Bax-like Immunoreactivity in Neurons of Heatstroke-induced Rats." Thesis, 2003. http://ndltd.ncl.edu.tw/handle/61654212551207431272.

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碩士<br>國立陽明大學<br>解剖暨細胞生物學研究所<br>91<br>Light microscopy, immunocytochemistry and Western blotting were employed in this study for investigating the morphological changes, the efffect of cytochrome c, Bcl-2 and Bax on the rat neurons induced by the heatstroke. Previous evidence indicate that after the heatstroke the metabolism of the glucose decreasesin the brain of the animals. The heatstroke results from the the increase of the oxidative stress and is followed by the ischemic neuronal injury. Sprague-Dawley rats were used in this study. For the pulsatile arterial pressure and heart rate were m

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36

Cieluch, Ewelina. "Mechanizm zmian dynamicznej dystrybucji elektronów w łańcuchach kofaktorów cytochromu bc_{1} badany przy użyciu impulsowej spektrofotometrii dwuwiązkowej." Praca doktorska, 2014. https://ruj.uj.edu.pl/xmlui/handle/item/61078.

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Nijhawan, Deepak. "Biochemical pathways in apoptosis." 2003. http://edissertations.library.swmed.edu/pdf/NijhawanD050305/NijhawanDeepak.pdf.

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38

Fleury, Isabelle. "Polymorphismes dans des gènes de métabolisme des corticostéroïdes : rôle dans la réponse thérapeutique." Thèse, 2003. http://hdl.handle.net/1866/14201.

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"Metabolic activation of drugs and other xenobiotics in hepatocellular carcinoma." Chinese University of Hong Kong, 1993. http://library.cuhk.edu.hk/record=b5888230.

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Grace S.N. Lau.<br>Thesis (Ph.D.)--Chinese University of Hong Kong, 1994.<br>Includes bibliographical references (leaves 335-362).<br>List of Abbreviations --- p.i<br>Abstract --- p.1<br>Chapter Chapter 1 --- General Introduction and Study Objectives<br>Chapter 1.1 --- Metabolic activation - role in drug toxicity and carcinogenesis --- p.5<br>Chapter 1.2 --- Hepatocellular carcinoma --- p.12<br>Chapter 1.2.1 --- Epidemiology --- p.12<br>Chapter 1.2.2 --- Aetiological factors --- p.17<br>Chapter 1.2.2.1 --- Hepatitis B virus infection --- p.17<br>Chapter 1.2.2.2 --- Cirrhosis --- p.24<b

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Salem, Mohamed M. A., Mohammad Shalbaf, Nick C. Gibbons, Bhavan Chavan, M. Julie Thornton, and Karin U. Schallreuter. "Enhanced DNA binding capacity on up-regulated epidermal wild-type p53 in vitiligo by H2O2-mediated oxidation: a possible repair mechanism for DNA damage." 2009. http://hdl.handle.net/10454/6168.

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Vitiligo is characterized by a patchy loss of inherited skin color affecting approximately 0.5% of individuals of all races. Despite the absence of the protecting pigment and the overwhelming evidence for hydrogen peroxide (H(2)O(2))-induced oxidative stress in the entire epidermis of these patients, there is neither increased photodamage/skin aging nor a higher incidence for sun-induced nonmelanoma skin cancer. Here we demonstrate for the first time increased DNA damage via 8-oxoguanine in the skin and plasma in association with epidermal up-regulated phosphorylated/acetylated p53 and high le

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