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Dissertations / Theses on the topic 'Cytokines'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Webb, Ginette Rachel. "Cytokines and cytokine receptors in osteoarthritis." Thesis, University of Bristol, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.388158.

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2

Deller, Marc Christian. "Structural studies of cytokines and cytokine receptors." Thesis, University of Cambridge, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326028.

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3

Palmblad, Karin. "Cytokines and cytokine-directed intervention in experimental arthritis /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4589-6/.

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4

Affò, Silvia. "Cytokines and Cytokine-Receptors in the Pathogenesis of Alcoholic Hepatitis." Doctoral thesis, Universitat de Barcelona, 2014. http://hdl.handle.net/10803/145435.

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By performing a translational study we identified a specific pattern of genes differentially regulated in patients with severe alcoholic hepatitis (AH). A functional analysis of the gene expression profile showed several pathways deregulated in AH, including cytokines- cytokine receptor interaction. Within cytokine-cytokine receptor interaction pathway, Fn14 has been identified as the only receptor belonging to TNF superfamily to be exclusively up-regulated in patients with AH, and its expression has been shown to be associated with severity of the disease and mortality. We observed that Fn14

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5

Ozaki, Akihiko. "Expression of cytokines and cytokine receptors in human Schwann cells." Kyoto University, 2008. http://hdl.handle.net/2433/135921.

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6

LU, ZHAO-YANG. "Cytokines et antagonistes de cytokines dans le myelome multiple." Nantes, 1993. http://www.theses.fr/1993NANT2079.

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Notre equipe a montre que l'interleukine-6 (il-6) est un facteur essentiel pour la croissance des cellules plasmocytaires malignes et que des traitements par anticorps anti-il-6 reduisaient croissance tumorale chez 50% des malades. La premiere partie de notre travail a consiste a rechercher les raisons pouvant expliquer une non-reponse au traitement. Nous avons ainsi developpe une methodologie nouvelle permettant de quantifier la production journaliere d'il-6 dans tout l'organisme. Nous avons montre que la production d'il-6 chez les malades non repondeurs etait trop forte, superieure au moins

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7

Croxford, J. Ludovic. "Gene therapy for experimental allergic encephalomyelitis by delivery of inhibitory cytokines or cytokine inhibitors." Thesis, University College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.314201.

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8

Ulfgren, Ann-Kristin. "Cytokines in rheumatoid arthritis /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-3823-7/.

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9

Benrick, Anna. "Cytokines in metabolic functions /." Göteborg : Section of Endocrinology, Institute of Neuroscience and Physiology, The Sahlgrenska Academy at University of Gothenburg, 2008. http://hdl.handle.net/2077/9608.

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10

Gilmore, William Hans. "Canine pro-inflammatory cytokines." Thesis, University of Liverpool, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263791.

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11

Baudry, Nathalie. "Cytokines, hypoxie et microcirculation." Paris 5, 1995. http://www.theses.fr/1995PA05CD06.

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Le sepsis constitue une des complications les plus graves pouvant survenir dans le domaine de la réanimation ou dans le traitement des malades immunodéprimés. Il se caractérise par l'apparition de nombreuses complications, tel que l'hypotension associé à une diminution des résistances vasculaires périphériques et le syndrome de défaillance respiratoire (SDRA) responsable une hypoxie systémique. Le but de ce travail est d'étudier d'une part les effets des médiateurs du sepsis (" tumor necrosis factor ", interleukines 1 et 6) sur le tonus et la réactivité à la norépinéphrine (NE) du réseau artér

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12

Ghorayeb, Christine. "The regulation of human B cell effector cytokine profiles by exogenous T helper cell cytokines /." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111556.

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The Bar-Or laboratory recently reported that human B cells from normal subjects can produce either pro-inflammatory (TNF-alpha; LT) or regulatory (IL-10) effector cytokines depending on their context of activation. It was of interest to investigate the change in B cell cytokine profiles from normal subjects when activated in the context of a Th1 pro-inflammatory environment or a Th2 anti-inflammatory environment. It was found that the B cell regulatory network of effector cytokines from normal subjects is significantly modulated depending on the local cytokine milieu. In addition, it was of in

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13

Shimokata, Kaoru. "Cytokines and Local Cellular Immunity." 名古屋大学医学部, 1997. http://hdl.handle.net/2237/6185.

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14

Hill, Andrew Graham. "Metabolic effects of proinflammatory cytokines." Thesis, University of Auckland, 1994. http://hdl.handle.net/2292/5522.

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The metabolic responses to trauma are well characterized and there is growing evidence supporting an important role for cytokines in its pathogenesis. Interleukin-1 (IL-1), Interleukin-6 (IL-6), and Tumour necrosis factor (TNF) are synthesized in the brain, and other tissues, in trauma. In order to test the hypothesis that these cytokines play an important role in the metabolic responses associated with trauma, the effects of chronic cerebroventricular and peripheral infusions of IL-1, IL-6, and TNF on protein metabolism, weight loss, anorexia, and pyrexia in Sprague-Dawley rats were examined.

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15

Wang, Ling Jia. "Cytokines and the human ovary." Title page, contents and abstract only, 1992. http://web4.library.adelaide.edu.au/theses/09PH/09phw2458.pdf.

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Bibliography: leaves 151-179. Examines aspects of the distribution of leukocyte subpopulation in human corpus luteum, cytokine determination in human preovulatory follicular fluid, as well as the effects of cytokines on human granulosalutein cells; with the aim of investigating one of the ovarian regulatory systems, which may be controlled by immune cell-derived cytokines.

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16

Galligan, Carole Lynn. "Inflammatory cytokines in bovine mastitis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0021/NQ47391.pdf.

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17

Zhang, Yan. "Cytokines and skeletal muscle wasting." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/MQ47124.pdf.

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18

Bemelmans, Marcus Henricus Adrianus. "Inflammatory cytokines in obstructive jaundice." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1994. http://arno.unimaas.nl/show.cgi?fid=6812.

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19

Dennison, Jeremy M. T. J. "Cytoadhesion, cytokines and cerebral malaria." Thesis, University of Liverpool, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337149.

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20

Parry, Robin Geoffrey. "Cytokines in minimal change nephropathy." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341511.

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21

Reuter, Simone. "Étude de l’effet de la cytokine TNFa sur la régulation de l’expression de la ?-glutamyl-transférase humaine." Thesis, Nancy 1, 2008. http://www.theses.fr/2008NAN10054/document.

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La??-glutamyltransférase (GGT) est une enzyme membranaire qui catalyse l’hydrolyse du glutathion, le principal antioxydant cellulaire. Cette enzyme est surexprimée dans de nombreux cancers (p.ex. leucémies) et appartient à une famille multigénique d’au moins treize gènes. Le gène I, qui code pour la protéine active, est exprimé en trois ARNm (A, B et C), montrant tous la même phase de lecture ouverte mais différant dans leur région 5’ non traduite. A côté de l’implication de la GGT dans le développement des cancers, elle intervient également dans l’acquisition des résistances aux traitements a

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22

Derouet, Damien. "Activation des récepteurs de cytokines de la famille de l'interleukine-6." Thesis, Angers, 2018. http://www.theses.fr/2018ANGE0006.

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Les cytokines de la famille de l’IL-6 possèdent comme caractéristique commune la capacité à recruter une chaine réceptrice membranaire nommée gp130. Au début de notre étude, cette famille comptait 8 membres : IL-6 et son homologue viral (vIL-6), IL-11, LIF, OSM, CNTF, CT-1 et CLC. Depuis, cette famille s’est enrichie de trois nouveaux membres, NP, IL-27 et IL-31. Ces cytokines sont essentielles au développement (LIF et CT-1) ou sont impliquées dans l’hématopoïèse, les réponses immunitaires et l’inflammation (IL-6, IL-11, IL-27, IL-31 et OSM) alors que d’autres agissent plus particulièrement au

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23

Wong, Hei-kiu. "Functional roles of rarely-used synonymous codon and sequences variation in type I cytokine receptors." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B42609999.

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24

Bonaccorso, Stefania. "Cytokines and depression: a neurochemical hypothesis." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 2005. http://arno.unimaas.nl/show.cgi?fid=6039.

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25

Patrick, Guy M. "Studies of cytokines in alloimmune responses /." Title page, table of contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09php314.pdf.

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26

Flint, Melanie Sarah. "Induction and regulation of epidermal cytokines." Thesis, Imperial College London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322116.

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27

Goncalves, Mario Nuno Penha. "Equine interferon-gamma and associated cytokines." Thesis, University of Glasgow, 2000. http://theses.gla.ac.uk/1064/.

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Cytokines are small proteins or glycoproteins that mediate cellular growth and differentiation and regulate immune responses. Upon encounter with antigen, CD4+ T cells are able to influence the character of the immune response elicited through the expression of distinct types of cytokines. Thl cytokines, especially IFN-γ but also TNF-β and IL-2, constitute one such pattern of expression, promoting cell mediated immune responses. In a broader sense, interleukin-12 and interleukin-18 can also be classified as type I cytokines in as much as they are able to shift the CD4+ cytokine expression patt

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28

McNerlan, Susan E. "Analysis of cytokines in synovial fluid." Thesis, Queen's University Belfast, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333839.

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29

Van, Wely Catherine Ann. "Cytokines, lymphocyte surface molecules and homing." Thesis, University College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298867.

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30

VIGARIO, ANA MARGARIDA. "Cytokines et pathogenese du neuropaludisme murin." Paris 7, 2001. http://www.theses.fr/2001PA077119.

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Le paludisme reste la maladie tropicale la plus importante. Le neuroplaludisme et l'anemie severe sont les deux causes principales de la mort. Une meilleure comprehension des mecanismes impliques dans la pathogenese du paludisme, en particulier du neuropaludisme, est donc des plus important. Differentes etudes, principalement dans les modeles rongeurs, ont demontre l'importance des cytokines pro-inflammatoires dans la pathogenese du neuropaludisme. Nous avons etudie le role de l'ifn- dans le developpement des signes neurologiques a l'aide de souris deficientes pour la chaine du recepteur de l'

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31

Simpson, Kenneth J. "Studies on cytokines in liver pathophysiology." Thesis, University of Edinburgh, 1997. http://hdl.handle.net/1842/21531.

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<I>Aims of the thesis:</I> To study the mechanisms of chemokine production that may occur during hepatic disease, the role of chemokines in hepatic injury and repair following paracetamol poisoning and hepatic expression of chemokines in patients with liver disease. In addition, the roles of tumour necrosis factor alpha (TNF) and stem cell factor (SCF) were also studied following paracetamol poisoning. <I>Results:</I> Both CXC and CC chemokines were produced during monocyte adhesion with human hepatoma cell lines. IL-8 production was dependent on proinflammatory cytokine production. In contras

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32

Schirmer, Stephan Henrik. "Circulating cells and cytokines in arteriogenesis." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2008. http://dare.uva.nl/document/115099.

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33

Al, Turaiki Wael. "Regulatory immune cytokines in RSV infection." Thesis, University of Liverpool, 2014. http://livrepository.liverpool.ac.uk/2008039/.

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Antibody production in the lungs is an essential defence mechanism against respiratory pathogens. However, little is known about the local activation of B cells in the lung. The production of BAFF and APRIL by airway epithelial cells could contribute to local recruitment, activation, class switch recombination and antibody production by B cells in the lung. In vitro, BEAS-2B cells were used to characterize BAFF and APRIL production simulated either by RSV infection or addition of cytokines. RSV and IFN-β significantly induced expression of BAFF mRNA and protein but not APRIL. BAFF mRNA reached

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34

Cebo, Christelle. "Activité lectinique des cytokines humaines : implications dans les voies de transduction lymphocytaires et modélisation de l'interaction cytokine-ligand." Lille 1, 2001. https://pepite-depot.univ-lille.fr/RESTREINT/Th_Num/2001/50376-2001-145.pdf.

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35

Chevalier, Anne Sophie. "Utilisation thérapeutique du G-CSF et du GM-CSF en hématologie." Paris 5, 1993. http://www.theses.fr/1993PA05P248.

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36

Aggad, Dina. "Diversité des interférons et de leurs récepteurs chez le Danio rerio." Montpellier 2, 2009. http://www.theses.fr/2009MON20117.

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Les IFNs sont un groupe de cytokines définis par leur activité antivirale. Chez les mammifères, ils sont divisés en trois groupes. Ils fixent tous des complexes récepteurs distincts et possèdent une structure génique différente. Les types I (principalement α et β) ont un seul exon, le type II (γ) quatre exons et le type III (λ) cinq. Les types I et III constituent un sous-groupe connu sous le nom « d'IFNs induits par les virus », car ils sont directement induits par une infection virale, alors que le type II ne l'est pas. La présence d'IFN induits par les virus et d'IFNγ (possédant 4 exons) on

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37

Goulesque, Bruno. "Remodelage osseux et cytokines dans les lymphopathies malignes : corrélation entre paramètres histomorphométriques et dosage sérique des cytokines." Montpellier 1, 1992. http://www.theses.fr/1992MON11019.

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38

王熙喬 and Hei-kiu Wong. "Functional roles of rarely-used synonymous codon and sequences variation in type I cytokine receptors." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B42609999.

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39

Boukhedouni, Nesrine. "Mécanismes immunologiques impliqués dans la perte des mélanocytes au cours du vitiligo." Thesis, Bordeaux, 2018. http://www.theses.fr/2018BORD0300.

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Le vitiligo est une dermatose inflammatoire caractérisée par une perte progressive des mélanocytes de la lame basale de l’épiderme. Cette pathologie reste à ce jour orpheline de traitement efficace. Toutefois, les mécanismes qui sont liés à la perte des mélanocytes restent débattus et impliquent un détachement des mélanocytes de la lame basale ou leur mort cellulaire par apoptose. Nous avons montré au sein de notre équipe l’implication des lymphocytes T CD8+ effecteurs mémoires dans la pathogénie du vitiligo. Ces populations produisent des niveaux élevés de deux cytokines inflammatoires qui so

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40

Dellacasagrande, Jérôme. "Infection de monocytes humains par Coxiella burnetii : modulation par cytokines." Aix-Marseille 2, 1999. http://theses.univ-amu.fr.lama.univ-amu.fr/1999AIX20672.pdf.

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Coxiella burnetii, l'agent pathogène responsable chez l'homme de la fièvre Q, est une bactérie intracellulaire stricte qui réside dans les monocytes/macrophages. Ces cellules sont douées de puissantes capacités microbicides qui leur confèrent un rôle essentiel dans la lutte anti-bactérienne. Ainsi, paradoxalement, C. Burnetii a pour cible les cellules destinées à la détruire. L'état d'activation des monocytes/macrophages, et par conséquent leur potentiel bactéricide, est contrôlé par les cytokines. Nous avons étudié l'effet des cytokines sur la survie de C. Burnetii dans les monocytes/macropha

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41

Layé, Sophie. "Régulation des cytokines et de leurs récepteurs dans le système nerveux central sous l'effet d'une inflammation périphérique induite par le lipopolysaccharide." Bordeaux 2, 1995. http://www.theses.fr/1995BOR28395.

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42

Fisher, Linda. "Inflammatory cytokines and NFκB in Alzheimer’s disease". Doctoral thesis, Stockholm University, Department of Neurochemistry, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-990.

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<p>Alzheimer’s disease is the most common form of dementia. It is a neurodegenerative disorder characterized by extracellular senile plaques and intracellular neurofibrillary tangles. The main constituent of the senile plaques is the neurotoxic β-amyloid peptide. Surrounding the senile plaques are activated astrocytes and microglia, believed to contribute to neurotoxicity through secretion of proinflammatory cytokines, like interleukin-1β and interleukin-6. For many inflammatory actions, including the cytokine induction in glial cells, the transcription factor NFκB plays a key role. This sugge

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43

Brooks, Lawrence G. "Adipose Tissue Cytokines: Effects of Social Condition." Scholarly Repository, 2009. http://scholarlyrepository.miami.edu/oa_dissertations/428.

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Social support has been demonstrated to reduce cardiovascular disease morbidity and mortality; however, the mechanisms by which social support reduces disease progression are still unclear. Oxytocin (OT) is a neuropeptide commonly associated with positive social interactions. This series of studies investigated the ability of oxytocin to attenuate atherosclerosis and its putative mediators, pro-inflammatory cytokines. Oxytocin receptors were identified by Western Blot on rat adipose tissue and rat adipocytes. OT receptor mRNA was identified in human adipocyte cDNA. In primary culture of r

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44

Yang, Huixin. "Role of cytokines in alveolar macrophage differentiation." Thesis, University of Ottawa (Canada), 1996. http://hdl.handle.net/10393/9918.

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The effects of macrophage colony-stimulating factor (M-CSF) and granulocyte-macrophage colony-stimulating factor (GM-CSF) on rat alveolar macrophage (AM) differentiation in vitro were investigated in this study. Both M-CSF and GM-CSF triggered AM differentiation and multinucleated giant cell (MGC) formation. Morphological analysis further demonstrated that there were two distinct variants of MGC. Type 1 MGC normally contained 3 to 8 nuclei and appeared as a large round cell and type 2 MGC contained a higher number of nuclei (up to 30) and displayed irregular, elongated shapes. We also observed

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45

Giot, Jean-Philippe. "Implication des cytokines inflammatoires dans l'angiodermite nécrotique." Thesis, Poitiers, 2013. http://www.theses.fr/2013POIT1406/document.

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L'angiodermite nécrotique ou "Hypertensive Leg Ulcer" (HLU) est un ulcère de jambe inflammatoire de traitement difficile, associé à une hypertension artérielle chronique. La douleur est très importante et nécessite l'utilisation de la morphine pour soulager les patients. Notre objectif était d'analyser les caractéristiques de l'inflammation et d'étudier son implication dans la physiopathologie de la nécrose cutanée. La peau inflammatoire est le siège d'un infiltrat composé de macrophages et de lymphocytes. L'épiderme et les cellules vasculaires lisses des microvaisseaux présentent des troubles

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46

Rahman, Arman. "Defensins and cytokines in inflammatory bowel disease." Doctoral thesis, Umeå : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1377.

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47

Fisher, Linda. "Inflammatory cytokines and NFkB in Alzheimer's disease /." Stockholm : Department of Neurochemistry, Stockholm University, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-990.

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48

Bueti, Deanna. "Immunomodulatory cytokines regulate intestinal epithelial barrier function /." Title page and abstract only, 2003. http://web4.library.adelaide.edu.au/theses/09SB/09sbb9289.pdf.

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49

Lazzerine, Abigail. "The role of cytokines in avian salmonellosis." Thesis, Imperial College London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.412579.

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50

Anwar, Ghazanfar Ali. "Genetic polymorphism in proinflammatory cytokines in bronchiectasis." Thesis, University of Newcastle Upon Tyne, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.576646.

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Bronchiectasis is a disease characterised by chronic bronchial sepsis and exerts considerable morbidity in those affected. In approximately 50% of cases the aetiology is unknown (idiopathic), raising the possibility of genetic predisposition. Cytokines such as IL-1, IL-6, IL-8 and TNFα are potent neutrophil recruiting molecules and are abundant in bronchiectatic secretions. Cytokine polymorph isms can lead to constitutively high production, and have been associated with a number of chronic inflammatory states. Hypothesis: Gene polymorph isms associated with high cytokine production predispose

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