Relevant bibliographies by topics / Cytoplazma / Books
To see the other types of publications on this topic, follow the link: Cytoplazma.

Books on the topic 'Cytoplazma'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 42 books for your research on the topic 'Cytoplazma.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

Agutter, Paul S. Between nucleus and cytoplasm. London: Chapman and Hall, 1991.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Milani, M. Differential two colour x-ray radiobiology of membrane / cytoplasm yeast cells. Chilton: Rutherford Appleton Laboratory, 1998.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

Schmid, Hans-Peter. Identifizierung und Charakterisierung einer neuen Klasse von Ribonukleoprotein-Partikeln aus dem Cytoplasma der eukaryontischen Zelle: Prosomen. Stuttgart: Biologisches Institut der Universität Stuttgart, 1987.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Ed, Stadelmann, Bushnell William R, and Bushnell Ann H, eds. Pathology of the plant cell. Karachi, Pakistan: Saad Publications, 1996.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

The secretory and endocytic paths: Mechanism and specificity of vesicular traffic in the cell cytoplasm. New York: Wiley, 1987.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
6

Conrad, Gary W. Effects of silver and other metals on the cytoskeleton: Summary of research : 02/15/95-06/30/97. [Washington, DC: National Aeronautics and Space Administration, 1997.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Tse, Wai Yee. Neutrophil Fc[gamma] receptor polymorphisms and pathogenetic mechanisms of neutrophil activation in anti-neutrophil cytoplasm (ANCA)-associated vasculitis. Birmingham: University of Birmingham, 1999.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
8

Hargrove, T. R. Changes in rice breeding in 10 Asian countries: 1965-84 : diffusion of genetic materials, breeding objectives, and cytoplasm. Manila: International Rice Research Institute, 1985.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

Schründer, Jürgen. Cytoplasmatische Vektoren zur heterologen Genexpression bei Hefen: Konstruktion und Analyse linearer Hybridplasmide basierend auf dem Killer-System der Hefe Kluyveromyces lactis. Berlin: J. Cramer, 1996.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
10

Bacterial growth and division: Biochemistry and regulation of prokaryotic and eukaryotic division cycles. San Diego: Academic Press, 1991.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
11

M, Malacinski George, ed. Cytoplasmic organization systems. New York: McGraw-Hill, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
12

The Functioning Cytoplasm. Springer, 2012.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
13

Frey-Wyssling, Albert. Comparative Organellography of the Cytoplasm. Springer, 2012.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
14

Harry, Walter, Brooks Donald E, and Srere Paul A, eds. Microcompartmentation and phase separation in cytoplasm. San Diego, Calif: Academic Press, 2000.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
15

Microcompartmentation and Phase Separation in Cytoplasm. Elsevier, 1999. http://dx.doi.org/10.1016/s0074-7696(08)x6044-9.

Full text
APA, Harvard, Vancouver, ISO, and other styles
16

Allen, Geoffrey. Proteins: Soluble Proteins of the Cytoplasm (Proteins). JAI Press, 1997.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
17

Ph, Jeanteur, ed. Cytoplasmic fate of messenger RNA. Berlin: Springer, 1997.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
18

1943-, Peters R., ed. Nucleo-Cytoplasmic transport. London: Academic Press, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
19

1943-, Peters R., and Trendelenburg M, eds. Nucleocytoplasmic transport. Berlin: Springer-Verlag, 1986.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
20

Dejesus, Carl, and Lourdes Trask. Chloroplasts and Cytoplasm : Structure and Functions: Life Sciences / Biology / Cell Biology. Nova Science Publishers, Incorporated, 2018.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
21

Carton, James. Infectious diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199591633.003.0002.

Full text
Abstract:
Microbes 16Antimicrobial agents 17Human immunodeficiency virus 18Tuberculosis 20Infectious mononucleosis 21Malaria 22Syphilis 24Lyme disease 25Leishmaniasis 26• Single-celled organisms with their double-stranded DNA lying free in cytoplasm surrounded by a cell membrane and cell wall.• Most grow in air (aerobes), but can grow without it (facultative anaerobes). Some only grow in the absence of oxygen (strict anaerobes)....
APA, Harvard, Vancouver, ISO, and other styles
22

Amos, Linda A., and Jan Löwe. Prokaryotic Cytoskeletons: Filamentous Protein Polymers Active in the Cytoplasm of Bacterial and Archaeal Cells. Springer, 2017.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
23

Physiology of rubber tree latex: The laticiferous cell and latex : a model of cytoplasm. Boca Raton, Fla: CRC Press, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
24

Ekiz, Hasan. Nuclear X cytoplasm genetic interactions controlling anther culture response in wheat (Triticum aestivum L.). 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
25

Physiology of Rubber Tree Latex The Laticiferous Cell and Latex- A Modell of Cytoplasm. CRC, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
26

Amos, Linda A., and Jan Löwe. Prokaryotic Cytoskeletons: Filamentous Protein Polymers Active in the Cytoplasm of Bacterial and Archaeal Cells. Springer, 2018.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
27

Bagni, Claudia, and Eric Klann. Molecular Functions of the Mammalian Fragile X Mental Retardation Protein: Insights Into Mental Retardation and Synaptic Plasticity. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199744312.003.0008.

Full text
Abstract:
Chapter 8 discusses how Fragile X syndrome (FXS) is caused by the absence of the RNA-binding protein fragile X mental retardation protein (FMRP). FMRP is highly expressed in the brain and gonads, the two organs mainly affected in patients with the syndrome. Functionally, FMRP belongs to the family of RNA-binding proteins, shuttling from the nucleus to the cytoplasm, and, as shown for other RNA-binding proteins, forms large messenger ribonucleoparticles.
APA, Harvard, Vancouver, ISO, and other styles
28

Leeds, Janet M. DNA precursor compartmentation in mammalian cells: Distribution and rates of equilibration between nucleus and cytoplasm. 1986.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
29

(Editor), Harry Walter, Donald E. Brooks (Editor), Paul A. Srere (Editor), and Kwang W. Jeon (Series Editor), eds. Microcompartmentation and Phase Separation in Cytoplasm (International Review of Cytology, Volume 192) (International Review of Cytology). Academic Press, 1999.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
30

Salama, Alan D. The patient with vasculitis. Edited by Giuseppe Remuzzi. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0159_update_001.

Full text
Abstract:
Primary systemic vasculitis frequently leads to renal involvement and is responsible for significant numbers of patients progressing to end-stage renal disease. Frequently this is due to small vessel vasculitis, in association with antineutrophil cytoplasm antibody, which requires prompt recognition and timely therapeutic intervention to optimize renal and patient outcomes. Other organ systems are often affected. Relapses occur in about 50%.Less commonly medium or larger vessel vasculitis may involve the kidneys and through ischaemia lead to impaired renal function and renovascular hypertension, as in Takayasu’s or Kawasaki disease, and polyarteritis nodosa (PAN).
APA, Harvard, Vancouver, ISO, and other styles
31

Xue, Feiyu. MOLECULAR ANALYSIS OF KELCH, A GENE NECESSARY FOR NURSE CELL CYTOPLASM TRANSPORT IN DROSOPHILA OOGENESIS ( KELCH GENE). 1992.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
32

M, Tazawa, ed. Cell dynamics. Wien: Springer-Verlag, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
33

Peter, Satir, Condeelis John S, Lazarides Elias, University of California, Los Angeles., Genentech Inc, and UCLA Symposium on Signal Tranduction in Cytoplasmic Organization and Cell Motility (1987 : Lake Tahoe, Calif.), eds. Signal transduction in cytoplasmic organization and cell motility: Proceedings of a UCLA Symposium held in Lake Tahoe, California, February 15-21, 1987. New York: Liss, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
34

Mason, Peggy. Synthesis, Packaging, and Termination of Neurotransmitters. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0012.

Full text
Abstract:
The synthesis, packaging, and termination of action of neurotransmitters are detailed. There are far more varieties of peptide neurotransmitters than there are of low-molecular-weight neurotransmitters. Yet low-molecular-weight neurotransmitters are the ubiquitous workhorses of the nervous system. Acetylcholine, the catecholamines norepinephrine and dopamine, serotonin, glutamate, and GABA are examined in some depth. The vesicular transporters that carry low-molecular-weight neurotransmitters from the cytoplasm into synaptic vesicles are covered. The role of monoamines in affect and mood and the psychotropic effects of monoaminergic drugs are discussed. Principles of catecholamine synthesis are applied to understand phenylketonuria. Uptake of monoamines into neurons is discussed in the context of amphetamine, cocaine, and other drugs of abuse. Stiff-person syndrome, which results from an impairment of GABA synthesis, is introduced. The modes of action for peptide and gaseous neurotransmitters are briefly covered.
APA, Harvard, Vancouver, ISO, and other styles
35

Sedel, Frédéric, and Carla E. M. Hollak. Disorders of Thiamine Metabolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0028.

Full text
Abstract:
Thiamine is a water-soluble vitamin acting in the mitochondria as a cofactor for energy metabolism and, in the cytoplasm, in the pentose phosphate biosynthetic pathway. Its transport through the plasma membrane requires two transporters with overlapping functions: THTR1 encoded by SLC19A2, and THTR2 encoded by SLC19A3. Thiamine is transformed into its active form, thiamine pyrophosphate (TPP) by a kinase encoded by the TPK1 gene. Then it may enter the mitochondria through a TPP transporter encoded by SLC25A19. Mutations in SLC19A2 cause thiamine-responsive megaloblastic anemia (TRMA). Mutations in SLC19A3 cause biotin/thiamine–responsive basal ganglia disease. Mutations in SLC25A19 may cause early microcephaly with death in infancy (also called Amish microcephaly) or a later-onset bilateral striatal necrosis with progressive peripheral neuropathy. Recently, mutations in the TPK1 gene have been associated with recurrent encephalopathy with mild lactic acidosis.
APA, Harvard, Vancouver, ISO, and other styles
36

E, Steele Vernon, ed. Cellular and molecular targets for chemoprevention. Boca Raton, Fla: CRC Press, 1992.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
37

Cytoplasmatische Vektoren Zur Heterologen Genexpression Bei Hefen: Konstruktion Und Analyse Linearer Hybridplasmide Basierend Auf Dem Killer - System (Bibliotheca Mycologica). Gebruder Borntraeger Verlagsbuchhandlung, 1996.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
38

Watts, Richard A., and David G. I. Scott. Vasculitis—classification and diagnosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0130.

Full text
Abstract:
The vasculitides are a group of conditions characterized by inflammation and necrosis of blood vessels; they are generally of unknown aetiology. The classification of vasculitides is based on the size of vessel involved and whether there is a known cause (secondary) or not (primary). This approach has stood the test of time. The American College of Rheumatology (ACR) in 1990 produced classification criteria for the major types of vasculitis and in 1994 definitions were promulgated by the Chapel Hill Consensus Conference. These did not include anti-neutrophil cytoplasm antibodies (ANCA) and the ACR scheme did not include microscopic polyangiitis. The definitions have recently been updated to include modern concepts of pathogenesis including ANCA. No validated diagnostic criteria are available for routine clinical practice. The diagnosis of vasculitis requires a high index of suspicion, especially in the systemically unwell patient with multiorgan involvement. The key to diagnosis is a detailed and systematic approach to patient assessment involving all potentially involved organs. In a patient with suspected vasculitis immediate urinalysis is mandatory as the severity of renal involvement at presentation is a major determinant of outcome. Each potentially involved organ should be comprehensively evaluated. Tissue biopsy should be obtained whenever possible, as treatment is potentially toxic using glucocorticoids combined with cytotoxic agents. Biopsy should not, however, delay initiation of treatment. Potential alternative diagnosis should be considered, especially infection and malignancy, and excluded whenever possible.
APA, Harvard, Vancouver, ISO, and other styles
39

Gross, Wolfgang L., and Julia U. Holle. Clinical features of ANCA-associated vasculitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0131.

Full text
Abstract:
The primary ANCA-associated vasculitides are granulomatosis with polyangiitis (Wegener's, GPA), microscopic polyangiitis (MPA), and eosinophilic granulomatosis with polyangiitis (EGPA, Churg-Strauss syndrome, CSS). They predominantly affect small (and medium-sized) vessels and share a variable association with ANCA (anti-neutrophil cytoplasm antibody) directed against neutrophil proteinase 3 (PR3, mainly in GPA) and myeloperoxidase (MPO, mainly in MPA and CSS). Crescentic necrotizing glomerulonephritis and alveolar haemorrhage due to pulmonary capillaritis represent classical (vasculitic) organ manifestations of the ANCA-associated vasculitides (AAV). MPA occurs as a 'pure' small (to medium-size) vessel vasculitis, whereas GPA and CSS are characterized by additional distinct clinical and pathological features. In GPA, granulomatous lesions of the upper and/or lower respiratory tract are a hallmark of the disease. Granulomatous lesions may be large in appearance and occur as space-consuming, infiltrating, and destructive inflammatory masses. GPA is believed to follow a stagewise course with an initial localized form, restricted granulomatous lesions of the upper and/or lower respiratory tract without clinical signs of vasculitis, and a consecutive generalization to systemic vasculitis which may be either non-organ-threatening (early systemic) or organ- and life- threatening (generalized GPA). Rarely, patients arrest in the localized stage and do not progress to systemic disease. In EGPA asthma, hypereosinophilia and eosinophilic organ infiltration (e.g. eosinophilic myocarditis) are typical features of the disease apart from vasculitis. Similarly to GPA, EGPA follows a stagewise course: asthma and eosinophilia may precede full-blown disease for several months or years. Recent cohort studies suggest different phenotypes in EGPA (predominantly vasculitic and MPO-ANCA-positive and predominantly with eosinophilic organ infiltration, usually ANCA-negative). This chapter focuses on the clinical features of the primary AAV and their outcome.
APA, Harvard, Vancouver, ISO, and other styles
40

1941-, Bradshaw Ralph A., McAlister-Henn Lee, Douglas Michael G, E.I. du Pont de Nemours & Company., and University of California, Los Angeles., eds. Molecular biology of intracellular protein sorting and organelle assembly: Proceedings of a DuPont-UCLA symposium held in Taos, New Mexico, January 30-February 5, 1987. New York: Liss, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
41

Krecic, Annette Marie. Nuclear and cytoplasmic functions of a yeast heterogeneous nuclear ribonucleoprotein. 1998.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
42

Jayne, David. Treatment of ANCA-associated vasculitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0132.

Full text
Abstract:
The goals of treatment in anti-neutrophil cytoplasm antibody (ANCA) vasculitis are to stop vasculitic activity, to prevent vasculitis returning, and to address longer-term comorbidities caused by tissue damage, drug toxicity, and increased cardiovascular and malignancy risk. Cyclophosphamide and high-dose glucocorticoids remain the standard induction therapy with alternative immunosuppressives, such as methotrexate or azathioprine, to prevent relapse. Refractory disease resulting from a failure of induction or remission maintenance therapy requires alternative agents and rituximab has been particularly effective. Replacement of cyclophosphamide by rituximab for remission induction is supported by recent evidence. Additional therapy with intravenous methylprednisolone and plasma exchange is employed in severe presentations with failing vital organ function. Drug toxicity contributes to comorbidity and mortality and has led to newer regimens with reduced cyclophosphamide exposure. Glucocorticoid toxicity remains a major problem, with controversy over the rapidity with which glucocorticoids can be reduced or withdrawn. Disease relapse occurs in 50% and requires early detection at a stage when it will not adversely affect outcomes. Rates of cardiovascular disease and malignancy are higher than in control populations but strategies to reduce their risk, apart from cyclophosphamide-sparing regimens, have not been developed. Thromboembolic events occur in 10% and may be linked to the recently identified autoantibodies to plasminogen and tissue plasminogen activator. Outcomes of vasculitis depend heavily on the level of tissue damage at diagnosis, especially renal dysfunction, but are also influenced by patient age, ANCA subtype, disease extent, and response to therapy. Eosinophilic granulomatosis with polyangiitis (Churg-Strauss)is treated along similar principles to granulomatosis with polyangiitis (GPA) and microscopic polyangiitis but the persistence of steroid-dependent asthma in over one-third and differences in pathogenesis has suggested alternative treatment approaches. Chronic morbidity results from tissue damage and is especially common in the upper and lower respiratory tract and kidneys. Tracheobronchial disease is a severe late complication of GPA, while deafness, nasal obstruction, and chronic sinusitis are sequelae of nasal and ear vasculitis. Chronic infection of damaged epithelial surfaces acts as a drive for vasculitic activity and adequate infection control is necessary for stable remission. Chronic kidney disease can stabilize for many years but the risks of endstage renal disease (ESRD) are increased by acute kidney injury at presentation or renal relapse. Renal transplantation is successful, with similar outcomes to other causes of ESRD.
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the bibliographies on the topic 'Cytoplazma' for other source types:
Journal articles Dissertations / Theses
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography