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Dissertations / Theses on the topic 'Cytoskeleton model'

Author: Grafiati

Published: 4 June 2021

Last updated: 14 February 2022

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1

Lane, D. C. "A mathematical investigation of a mechanochemical model for the cytoskeleton." Thesis, University of Oxford, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.379843.

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2

Dewolf, Christine Elizabeth. "Properties of model biological membranes." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244082.

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3

Muddana, Hari Shankar. "Integrated biomechanical model of cells embedded in extracellular matrix." [College Station, Tex. : Texas A&M University, 2006. http://hdl.handle.net/1969.1/ETD-TAMU-1074.

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4

Monreal, Gretel. "Ventricular Remodeling in a Large Animal Model of Heart Failure." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1210007937.

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5

Lewis, Sara Ann. "Functions of Drosophila Pak (p21-activated kinase) in Morphogenesis: A Mechanistic Model based on Cellular, Molecular, and Genetic Studies." Diss., The University of Arizona, 2015. http://hdl.handle.net/10150/594389.

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Intellectual disability (ID) is a common phenotype of brain-development disorders and is heterogeneous in etiology with numerous genetic causes. PAK3 is one gene with multiple mutations causing ID. Affected individuals have microcephaly, and other brain-structure defects have been reported. Additionally, PAK3 is in a genetic network with eighteen other genes whose mutations cause ID, suggesting the molecular mechanisms by which PAK3 regulates of cognitive function may be shared by other genetic ID disorders. Studies in rodent models have shown that the orthologs of PAK3 are important for regul

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6

Bauer, David. "Využití tensegritních struktur pro modelování mechanického chování hladkých svalových buněk." Master's thesis, Vysoké učení technické v Brně. Fakulta strojního inženýrství, 2011. http://www.nusl.cz/ntk/nusl-229836.

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The master’s thesis deals with the computational modelling of the mechanical testing of isolated smooth muscle cells. The main aims are to create computational model of a cell, to simulate single-axis tensile test and to modify the model so that the model reflects real mechanical response. The model of the cell includes cytoplasm, nucleus, cell membrane and cytoskeleton which is modelled as a tensegrite structure. On this model the tensile test was simulated in case of the cell with cytoskeleton and the cell with distributed the cytoskeleton. Force-elongation curves, which were obtained from t

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7

Yadav, Preeti [Verfasser], and Michael [Gutachter] Sendtner. "Studying Neuronal Cytoskeleton Defects and Synaptic Defects in Mouse Model of Amyotrophic Lateral Sclerosis and Spinal Muscular Atrophy / Preeti Yadav. Gutachter: Michael Sendtner." Würzburg : Universität Würzburg, 2016. http://d-nb.info/1113535075/34.

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8

Kovari, Daniel T. "Investigations of the spreading and closure mechanisms of phagocytosis in J774a.1 macrophages." Diss., Georgia Institute of Technology, 2015. http://hdl.handle.net/1853/54882.

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Phagocytosis is the process by which cells engulf foreign bodies. It is the hallmark behavior of white blood cells, being the process through which those cells ingest and degrade pathogens and debris. To date a large amount of research has focused on documenting the existence and role of biochemical components involved with phagocytosis. Scores of signaling molecules have been implicated in the complex signal cascade which drives the process. These molecules are small (typically no larger than 5 nanometers) and operate in a crowded, chemically “noisy,” environment, yet they coordinate the cell

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9

Ganay, Thibault. "Caractérisation du modèle murin de la Neuropathie à Axones Géants : rôle de la gigaxonine dans la survie neuronale et l'organisation du cytosquelette." Thesis, Aix-Marseille 2, 2011. http://www.theses.fr/2011AIX22075.

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La Neuropathie à Axones Géants (NAG) est une maladie neurodégénérative rare et fatale caractérisée par une détérioration du système nerveux central et périphérique, impliquant les fonctions motrices et sensorielles. La détérioration massive du système nerveux est accompagnée d'une désorganisation générale des Filaments Intermédiaires ce qui la différencie de nombreuses maladies neurodégénératives où seuls les neurofilaments(NFs) sont affectés. La protéine déficiente, la gigaxonine, est la sous-unité d'une ubiquitine ligase E3, responsable de la reconnaissance spécifique des substrats MAP1B, MA

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10

Henninger, Nils. "Inhibiting Axon Degeneration in a Mouse Model of Acute Brain Injury Through Deletion of Sarm1." eScholarship@UMMS, 2017. http://escholarship.umassmed.edu/gsbs_diss/900.

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Traumatic brain injury (TBI) is a leading cause of disability worldwide. Annually, 150 to 200/1,000,000 people become disabled as a result of brain trauma. Axonal degeneration is a critical, early event following TBI of all severities but whether axon degeneration is a driver of TBI remains unclear. Molecular pathways underlying the pathology of TBI have not been defined and there is no efficacious treatment for TBI. Despite this significant societal impact, surprisingly little is known about the molecular mechanisms that actively drive axon degeneration in any context and particularly followi

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11

Petrov, Eugene P., Senthil Arumugam, Jens Ehrig, and Petra Schwille. "Cytoskeletal pinning prevents large-scale phase separation in model membranes." Universitätsbibliothek Leipzig, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-182836.

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12

Petrov, Eugene P., Senthil Arumugam, Jens Ehrig, and Petra Schwille. "Cytoskeletal pinning prevents large-scale phase separation in model membranes." Diffusion fundamentals 20 (2013) 40, S. 1, 2013. https://ul.qucosa.de/id/qucosa%3A13607.

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13

Roberts-Pilgrim, Anna M. Phillips Charlotte L. "Glomerulosclerosis in the Col1a2-deficient mouse model homotrimer pathogenesis and MMP expression /." Diss., Columbia, Mo. : University of Missouri--Columbia, 2009. http://hdl.handle.net/10355/6155.

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Title from PDF of title page (University of Missouri--Columbia, viewed on Feb. 20, 2010). The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract appears in the public.pdf file. Dissertation advisor: Charlotte L. Phillips. Vita. Includes bibliographical references.

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14

Kumar, Ravinash Krishna. "Cytoskeletal-like assemblies within model protocells : en route towards synthetic cellular constructs." Thesis, University of Bristol, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.619256.

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The aim of this thesis was to rationally design, construct and test novel protocells equipped with primitive cytoskeletons, which could be produced by integration of simple chemical systems. This was achieved by the non-covalent assembly of a functionalised amino acid, N-fluorenylmethyloxycarbonyl-tyrosine (FMOC-Tyr), to yield highly structured nanofilament morphologies within the aqueous interiors of micron-sized, membrane-bounded compartments. Initially. the formation of the non-covalently assembled FMOC-Tyr nanofilaments was studied in isolation. Significantly, at a critical filament concen

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15

Genty, Christian. "Réponse in vitro des cellules ostéoformatrices aux stimuli mécaniques : étude en microgravité réelle et sur les modèles animaux après surcharge et décharge mécanique." Saint-Etienne, 1993. http://www.theses.fr/1993STET4013.

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Les variations de contraintes mécaniques appliquées sur l'os entrainent des modifications du métabolisme osseux. La diminution des contraintes mécaniques telle qu'elle existe dans le modèle du rat suspendu, induit une perte osseuse rapide dès le début de la suspension qui se stabilise par la suite. L'augmentation des contraintes par un exercice de type dynamique (la course sur tapis roulant) induit un gain osseux. Les mécanismes cellulaires responsables de ces modifications sont encore mal connus. Grâce à la mise au point d'une méthode d'isolement des cellules ostéoformatrices à partir du tibi

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16

Koch, Matthias [Verfasser], and Alexander [Akademischer Betreuer] Rohrbach. "Biomechanics of prokaryotic & eukaryotic cytoskeletal model systems probed by time-multiplexed optical tweezers." Freiburg : Universität, 2015. http://d-nb.info/1119246539/34.

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17

García, Arguinzonis Maísa Inés. "Analysis of signal transduction pathways and the cytoskeleton in VASP-deficient cell lines and mouse models." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=969694741.

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18

Na, Sungsoo. "Effects of mechanical forces on cytoskeletal remodeling and stiffness of cultured smooth muscle cells." Thesis, [College Station, Tex. : Texas A&M University, 2006. http://hdl.handle.net/1969.1/ETD-TAMU-1704.

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19

Roth, Alexander. "Nano-mechanics of biomimetic models of the actin based cytoskeleton from single molecules to complex composite structures /." [S.l. : s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=972309322.

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20

Belbahri, Mohamed Reda. "Rôle des protéines réticulantes de l'actine dans la mécanique de l'endocytose chez la levure." Thesis, Aix-Marseille, 2020. http://www.theses.fr/2020AIXM0209.

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L'objet de cette thèse porte sur l'étude du rôle des protéines réticulantes dans la génération de force des réseaux d'actine branchés lors de l'endocytose. Une première étape consiste en une quantification in-vivo de la dynamique des patchs d'endocytose dans des levures (S. cerevisiae) sauvages et dans des levures mutantes n'exprimant pas une ou plusieurs protéines réticulantes. En mettant ces résultats en parallèle avec des données mécaniques déjà établies par une ancienne doctorante, nous avons pu démontrer une corrélation entre l'élasticité d'un réseau d'actine et l'efficacité de l'endocyto

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21

Palmer, Jeffrey Shane. "Microstructurally-based constitutive models of cytoskeletal networks for simulation of the biomechanical response of biological cells." Thesis, Massachusetts Institute of Technology, 2008. http://hdl.handle.net/1721.1/44752.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Mechanical Engineering, 2008.<br>Includes bibliographical references (v. 2, p. 335-375).<br>The elastic and viscoelastic stress-strain behavior of cytoskeletal networks, important to many cellular functions, is modeled via a microstructurally-informed continuum mechanics approach. The force-extension behavior of the individual filaments is captured with a new analytical expression of the MacKintosh worm-like chain relationship for semiflexible filaments. The filament expression is used in the Arruda-Boyce eight-chain network mode

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22

Wurm, Christine [Verfasser], Andreas [Akademischer Betreuer] Bausch, and Joachim O. [Akademischer Betreuer] Rädler. "Structure, Mechanics and Dynamics of Cytoskeletal Model Systems / Christine Wurm. Gutachter: Andreas Bausch ; Joachim O. Rädler. Betreuer: Andreas Bausch." München : Universitätsbibliothek der TU München, 2011. http://d-nb.info/1012186806/34.

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23

Lombardo, Andrew Thomas. "Cargo Transport By Myosin Va Molecular Motors Within Three-Dimensional In Vitro Models Of The Intracellular Actin Cytoskeletal Network." ScholarWorks @ UVM, 2018. https://scholarworks.uvm.edu/graddis/860.

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Intracellular cargo transport involves the movement of critical cellular components (e.g. vesicles, organelles, mRNA, chromosomes) along cytoskeletal tracks by tiny molecular motors. Myosin Va motors have been demonstrated to play a vital role in the transport of cargos destined for the cell membrane by navigating their cargos through the three-dimensional actin networks of the cell. Transport of cargo through these networks presents many challenges, including directional and physical obstacles which teams of myosin Va-bound to a single cargo must overcome. Specifically, myosin Va motors are p

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24

Kaushik, Azad Kumar. "Etude idiotypique, structurale et analyse du repertoire des autoanticorps dans deux modeles d'autoimmunite naturelle chez la souris : autoanticorps reagissant avec les globules rouges de souris bromelises, autoanticorps reagissant avec l'adn et les." Paris 7, 1987. http://www.theses.fr/1987PA077074.

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25

Haga, Raquel Brandão. "Inibição da migração mediada pelo gene RECK em modelo de glioma humano através de alterações no citoesqueleto e adesão focal." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/9/9136/tde-11092012-134839/.

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Gliomas são tumores altamente invasivos, resistentes aos tratamentos disponíveis atualmente e com alta taxa de mortalidade. A superexpressão de RECK na linhagem de glioma humano T98G comprometeu a capacidade das células de migrar e invadir in vitro, com rearranjo do citoesqueleto e alteração na distribuição espacial de FAK fosforilado. Entretanto, o possível mecanismo envolvido na inibição da migração mediada por RECK não foi desvendado. Para estudarmos os mecanismos envolvidos nesta alteração da capacidade migratória, as células T98G foram transfectadas com o vetor plasmidial pCXN2-hRECK (REC

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26

Maier, Matthias [Verfasser], Andreas [Akademischer Betreuer] Bausch, and Matthias [Akademischer Betreuer] Rief. "Living Cells and Cytoskeletal Model Systems: Mechanics and Responses to Dynamic Shear / Matthias Maier. Gutachter: Andreas Bausch ; Matthias Rief. Betreuer: Andreas Bausch." München : Universitätsbibliothek der TU München, 2014. http://d-nb.info/1052995489/34.

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27

Chamcheu, Jean Christopher. "Disease-causing Keratin Mutations and Cytoskeletal Dysfunction in Human Skin : In vitro Models and new Pharmacologic Strategies for Treating Epidermolytic Genodermatoses." Doctoral thesis, Uppsala universitet, Institutionen för medicinska vetenskaper, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-123071.

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Epidermolysis bullosa simplex (EBS) and epidermolytic ichthyosis (EI) are rare skin fragility diseases characterized by intra-epidermal blistering due to autosomal dominant-negative mutations in basal (KRT5 or KRT14) and suprabasal (KRT1 or KRT10) keratin genes, respectively. Despite vast knowledge in the disease pathogenesis, the pathomechanisms are not fully understood, and no effective remedies exist. The purpose of this work was to search for keratin gene mutations in EBS patients, to develop in vitro models for studying EBS and EI, and to investigate novel pharmacological approaches for

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28

Amaral, Jonatas Bussador do. "Células MCF-7 como modelo 3D no estudo de câncer de mama humano." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-21072011-134443/.

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O diferencial da cultura de células em 3-dimensões é permitir que as células explorem as 3-dimensões do espaço, aumentando assim as interações com o ambiente e entre as células. Em estudos relacionados à biologia do câncer de mama, vem ganhando espaço a utilização de esferóides para estudos que visam à compreensão da morfogênese do espaço luminal. Neste trabalho foi mostrado que as células MCF-7 reorganizam-se em estruturas tubulares e acinares. Em ambas as situações, a formação do lúmen veio acompanhada pelo estabelecimento de uma camada de células polarizadas, arranjo este muito semelhante a

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29

Mefti, Nacim. "Mise en oeuvre d'un modèle mécanique de l'adhésion cellulaire : approche stochastique." Thesis, Vandoeuvre-les-Nancy, INPL, 2006. http://www.theses.fr/2006INPL099N/document.

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L'adhésion cellulaire est un phénomène important en biologie. Le but de ce travail est le développement d'un modèle mécanique décrivant des phénomènes d'adhésion cellulaire à différentes échelles. La première échelle, microscopique, a pour objet la description des phénomènes cinétiques moléculaires durant le rolling. La seconde échelle, mésoscopique, est relative à la modélisation des déformations actives de la cellule durant la motilité. La troisième échelle, dite macroscopique, concerne la description de l'évolution dans le temps de l'adhésion d'une population de cellules. Les simulations ré

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30

Braunger, Julia. "Ezrin activation in vitro: Investigation of ezrin's conformation and the interaction between ezrin and F-actin." Doctoral thesis, Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2013. http://hdl.handle.net/11858/00-1735-0000-0022-609D-5.

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31

Taniguchi, Leandro Utino. "Avaliação imunohistoquímica das alterações do citoesqueleto na parede alveolar em modelo experimental de lesão pulmonar induzida pela ventilação mecânica em ratos." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5159/tde-07122009-180201/.

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INTRODUÇÃO: A ventilação mecânica é uma terapia importante, mas com possíveis complicações. Uma das mais relevantes é a lesão pulmonar induzida pelo ventilador (VILI do inglês Ventilator-induced lung injury). Devido à hiperdistensão alveolar, o pulmão inicia um processo inflamatório, com infiltrado neutrofílico, formação de membrana hialina, fibrogênese e prejuízo de troca gasosa. Nesse processo, a mecanotransdução do estímulo da hiperdistensão celular se faz através do citoesqueleto da célula e de suas interações com a matriz extracelular e com as células vizinhas. Apesar desse papel fundamen

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32

Campos, Luciene Cristina Gastalho. "Identificação e caracterização de proteínas modificadas em enxertos de veias safenas humanas arterializadas no modelo ex vivo." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-16122008-174047/.

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A revascularização cardíaca utilizando a ponte de safena é um procedimento bastante utilizado para restabelecer o fluxo coronariano. Apesar do sucesso deste procedimento, a patência deste enxerto pode chegar a menos de 50% em 10 anos. Atribui-se parte deste insucesso a variações no processo adaptativo à nova condição hemodinâmica, onde o shear stress e o estiramento aumentados podem estar interferindo na função endotelial e vascular. Este processo envolve a participação de diversas proteínas e o estudo de como elas participam conjuntamente é uma importante abordagem para entender as alterações

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33

Lee, Hannah Yun Young. "Calcium homeostasis in lens transparency and the involmement of calpains in cataract." Lincoln University, 2006. http://hdl.handle.net/10182/1897.

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The absolute clarity of the lens of the eye is vital in the visual system. The unique structural and physiological properties of the lens are tightly integrated with highly ordered protein content to allow the lens to remain transparent. Consequently, any alteration or disturbance of these highly ordered proteins can affect the optical properties of the lens. In humans, cataracts are the major cause of blindness, yet the exact aetiology of cataract formation (cataractogenesis) is not fully understood. The purpose of the current research was to investigate whether deregulation of the Ca²⁺-depen

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34

Lomasko, Tatiana. "One-hit Stochastic Decline in a Mechanochemical Model of Cytoskeleton-induced Neuron Death." Thesis, 2008. http://hdl.handle.net/1807/16801.

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Much experimental evidence shows that the cytoskeleton is a downstream target and effector during cell death in numerous neurodegenerative diseases, including Parkinson's, Huntington's, and Alzheimer's diseases. However, recent evidence indicates that cytoskeletal dysfunction can also trigger neuronal death, by mechanisms as yet poorly understood. We studied a mathematical model of cytoskeleton-induced neuron death in which assembly control of the neuronal cytoskeleton interacts with both cellular stress levels and cytosolic free radical concentrations to trigger neurodegeneration. This trigge

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35

Silva, Marina Isabel Oliveira da. "Dissecting the role of cytoskeleton remodelling in a Drosophila model of TTR-induced neurodegeneration." Dissertação, 2015. https://repositorio-aberto.up.pt/handle/10216/82245.

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Silva, Marina Isabel Oliveira da. "Dissecting the role of cytoskeleton remodelling in a Drosophila model of TTR-induced neurodegeneration." Master's thesis, 2015. https://repositorio-aberto.up.pt/handle/10216/82245.

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37

Yadav, Preeti. "Studying Neuronal Cytoskeleton Defects and Synaptic Defects in Mouse Model of Amyotrophic Lateral Sclerosis and Spinal Muscular Atrophy." Doctoral thesis, 2016. https://nbn-resolving.org/urn:nbn:de:bvb:20-opus-138093.

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Amyotrophic lateral sclerosis and spinal muscular atrophy are the two most common motoneuron diseases. Both are characterized by destabilization of axon terminals, axon degeneration and alterations in neuronal cytoskeleton. Accumulation of neurofilaments has been observed in several neurodegenerative diseases but the mechanisms how elevated neurofilament levels destabilize axons are unknown so far. Here, I show that increased neurofilament expression in motor nerves of pmn mutant mice causes disturbed microtubule dynamics. Depletion of neurofilament by Nefl knockout increases the number and re

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38

Saal, Kim Ann. "Modulation of the ROCK pathway in models of Parkinson´s disease." Doctoral thesis, 2015. http://hdl.handle.net/11858/00-1735-0000-0028-8665-9.

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Petr, Martin. "Jaderný myosin 1 a jeho role v regulaci tenze cytoplazmatické membrány." Master's thesis, 2014. http://www.nusl.cz/ntk/nusl-332401.

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Myosin 1c (Myo1c) is a molecular motor involved in regulation of tension-gated ion channels, exocytosis, endocytosis, motility and other membrane-related events. Moreover, it acts as a dynamic linker between the cell membrane and cortical actin network, contributing to the maintenance of plasma membrane tension. In contrast, nuclear myosin 1 (NM1), an isoform of Myo1c, has been described only in the nucleus where it participates in various nuclear processes, including transcription or chromatin remodeling. However, although traditionally regarded as exclusively cytoplasmic or nuclear, all myos

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40

García, Arguinzonis Maísa Inés. "Analysis of signal transduction pathways and the cytoskeleton in VASP-deficient cell lines and mouse models." Doctoral thesis, 2003. https://nbn-resolving.org/urn:nbn:de:bvb:20-opus-6195.

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The mammalian Vasodilator Stimulated Phosphoprotein (VASP) is a founding member of the Ena/VASP family of proteins that includes Drosophila Enabled (ena), the mammalian Ena homologue (Mena) and the Ena-VASP-like protein (Evl). VASP was initially discovered and characterized as a substrate for cGMP- and cAMP-dependent protein kinases (cGKs and cAKs). Ena/VASP proteins are involved in Actin-filament formation, plasma membrane protrusion, acceleration of Actin-based motility of Listeria and the establishment of cell-cell adhesion. Moreover, Ena/VASP proteins have been implicated as inhibitory fac

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41

Roth, Alexander [Verfasser]. "Nano-mechanics of biomimetic models of the actin based cytoskeleton : from single molecules to complex composite structures / Alexander Roth." 2004. http://d-nb.info/972309322/34.

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42

García, Arguinzonis Maísa Inés [Verfasser]. "Analysis of signal transduction pathways and the cytoskeleton in VASP-deficient cell lines and mouse models / vorgelegt von Maísa Inés García Arguinzonis." 2003. http://d-nb.info/969694741/34.

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43

Tsitkov, Stanislav. "Emergent Properties of Biomolecular Organization." Thesis, 2021. https://doi.org/10.7916/d8-dmje-n287.

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The organization of molecules within a cell is central to cellular processes ranging from metabolism and damage repair to migration and replication. Uncovering the emergent properties of this biomolecular organization can improve our understanding of how organisms function and reveal ways to repurpose their components outside of the cell. This dissertation focuses on the role of organization in two widely studied systems: enzyme cascades and active cytoskeletal filaments.Part I of this dissertation studies the emergent properties of the spatial organization of enzyme cascades. Enzyme cascades

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