Relevant bibliographies by topics / D. Herpetiformi

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'D. Herpetiformi'

Author: Grafiati
Published: 1 April 2023
Last updated: 31 July 2025

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Journal articles on the topic "D. Herpetiformi"

1

Otley, Clark C., and Russell P. Hall. "The pathogenesis of dermatitis herpetiformis." Clinics in Dermatology 9, no. 3 (1991): 313–23. http://dx.doi.org/10.1016/0738-081x(91)90022-d.

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ABE, Toshifumi, Michiyoshi KOUNO, Nobuhiro YAMAMOTO, Osamu MORI, Takashi HASHIMOTO, and Masayuki AMAGAI. "A Case of D-penicillamine Induced Herpetiform Pemphigus." Nishi Nihon Hifuka 65, no. 2 (2003): 126–29. http://dx.doi.org/10.2336/nishinihonhifu.65.126.

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3

De Cuyper, C. "Pemphigus herpetiformis in a lung cancer patient." Journal of the European Academy of Dermatology and Venereology 5, no. 1 (1995): S159. http://dx.doi.org/10.1016/0926-9959(95)96420-d.

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4

STIMOU, Sanae, Hafsa TAHERI, Hanane SAADI, and Ahmed MIMOUNI. "IMPETIGO HERPETIFORMIS IN A CASE REPORTPRESENTATION OF A RARE ENTITY." International Journal of Advanced Research 11, no. 01 (2023): 641–43. http://dx.doi.org/10.21474/ijar01/16061.

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Impetigo herpetiformis (IH) is a very rare and specific pustular dermatosis of pregnancy that can be life-threatening for both mother and child. This entity is currently considered as a variant of pustular psoriasis. We report an observation of a 35-year-old female patient who presented with extensive erythematous-pustular plaques for which the diagnosis of impetigo herpetiformiswas made clinically and histologically. The lesions appeared in the third trimester of pregnancy without fetal or maternal impact. The treatment was prednisone, vitamin D supplementation and a macrolide for 10 days.
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5

Hertl, Michael, Andrea Niedermeier, and Luca Borradori. "Bullöse Autoimmundermatosen." Therapeutische Umschau 67, no. 9 (2010): 465–82. http://dx.doi.org/10.1024/0040-5930/a000080.

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Blasenbildende Autoimmunerkrankungen sind seltene, meist schwere Erkrankungen von Haut und Schleimhäuten, die immunologisch durch zirkulierende Antikörper gegen Adhäsionsmoleküle der Epidermis und der dermoepidermalen Junktionszone charakterisiert sind; diese führen zum Adhäsionsverlust von Haut und Schleimhäuten und klinisch zur Ausbildung von Blasen oder Erosionen. Beim Pemphigus findet der Adhäsionsverlust in der Epidermis statt, während beim Pemphigoid, der linearen IgA-Dermatose, der Epidermolysis bullosa acquisita und der Dermatitis herpetiformis Duhring der Adhäsionsverlust subepidermal
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6

NAKAYAMA, Kanichiro, Yoshinori SUENAGA, Teruo UENO, and Seiichiro AONO. "A case of herpetiform pemphigus-like skin lesions induced by D-penicillamine." Nishi Nihon Hifuka 48, no. 6 (1986): 1054–59. http://dx.doi.org/10.2336/nishinihonhifu.48.1054.

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7

Furumura, Minao, Shuhei Imayama, and Yoshiaki Hori. "Three neonatal cases of epidermolysis bullosa herpetiformis (Dowling-Meara type) with severe erosive skin lesions." Journal of the American Academy of Dermatology 28, no. 5 (1993): 859–61. http://dx.doi.org/10.1016/0190-9622(93)70118-d.

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8

De Benedictis-Serrano, Ginno Alessandro, Laura Contreras-Lugo, Génesys Córdova-Rivas, and Carlos Miguel Ríos-González. "El esprúe tropical, una enfermedad olvidada como diagnóstico diferencial de la enfermedad celiaca." Revista de la Facultad de Medicina 66, no. 1 (2018): 129–30. http://dx.doi.org/10.15446/.v66n1.68105.

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Estimado editor, Por medio de la presente nos gustaría traer a discusión la importancia que tiene la enfermedad celiaca (EC), también conocida como esprúe celiaco o enteropatía sensible al gluten, la cual corresponde a una enfermedad autoinmune desencadenada por la ingesta de gluten en individuos predispuestos genéticamente (1). La EC se caracteriza por una amplia variabilidad de manifestaciones clínicas, pero su forma clásica es la más habitual en el adulto; aquí los síntomas guía son de naturaleza gastrointestinal (diarrea, esteatorrea y dolor abdominal inespecífico). Por otro lado, las form
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9

De Benedictis-Serrano, Ginno Alessandro, Laura Contreras-Lugo, Génesys Córdova-Rivas, and Carlos Miguel Ríos-González. "El esprúe tropical, una enfermedad olvidada como diagnóstico diferencial de la enfermedad celiaca." Revista de la Facultad de Medicina 66, no. 1 (2018): 129–30. http://dx.doi.org/10.15446/revfacmed.v66n1.68105.

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Abstract:
Estimado editor, Por medio de la presente nos gustaría traer a discusión la importancia que tiene la enfermedad celiaca (EC), también conocida como esprúe celiaco o enteropatía sensible al gluten, la cual corresponde a una enfermedad autoinmune desencadenada por la ingesta de gluten en individuos predispuestos genéticamente (1). La EC se caracteriza por una amplia variabilidad de manifestaciones clínicas, pero su forma clásica es la más habitual en el adulto; aquí los síntomas guía son de naturaleza gastrointestinal (diarrea, esteatorrea y dolor abdominal inespecífico). Por otro lado, las form
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10

Rizvi, Zainab, Nakhshab Choudhry, Aamir Jamal Gondal, and Nighat Yasmin. "Association of Surfactant Protein D Single Nucleotide Polymorphisms rs721917, rs2243639, rs3088308 with Recurrent Aphthous Stomatitis in Pakistani Population." Genes 14, no. 5 (2023): 1119. http://dx.doi.org/10.3390/genes14051119.

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Recurrent aphthous stomatitis (RAS) is a benign ulcerative condition, defined by the recurrent formation of non-contagious mucosal ulcers. Surfactant protein D (SP-D) is secreted frequently at surfaces exposed directly to body fluids. This study aims to investigate the association of SP-D single nucleotide polymorphisms (SNPs) with the onset of RAS. Blood samples from 212 subjects (106 cases/controls each) were collected during 2019 and genotyped for SP-D SNPs (rs721917, rs2243639, rs3088308) by polymerase chain reaction and restriction fragment length polymorphism followed by 12% polyacrylami
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Dissertations / Theses on the topic "D. Herpetiformi"

1

GAITA, BARTOLOMEA. "Ricerca del sito di origine e del ruolo patogenetico degli anticorpi anti-transglutaminasi epidermica nella Dermatite Erpetiforme." Doctoral thesis, Università degli Studi di Trieste, 2022. http://hdl.handle.net/11368/3015191.

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La Dermatite Erpetiforme (DE) è una malattia infiammatoria cutanea caratterizzata da vescicole papulari pruriginose e da depositi granulari di IgA in corrispondenza delle papille dermiche o lungo la giunzione derma-epiderma. La malattia è considerata la manifestazione cutanea della celiachia, condividendo con quest’ultima l’origine autoimmune, la predisposizione genetica, la presenza di autoanticorpi IgA a livello sierico e la remissione clinica in seguito all’eliminazione del glutine dalla dieta. La transglutaminasi 3 o epidermica (TG-3 or eTG) rappresenta l’antigene verso cui si sviluppa la
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Journal articles
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