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Journal articles on the topic 'D. Herpetiformi'

Author: Grafiati

Published: 1 April 2023

Last updated: 31 July 2025

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1

Otley, Clark C., and Russell P. Hall. "The pathogenesis of dermatitis herpetiformis." Clinics in Dermatology 9, no. 3 (1991): 313–23. http://dx.doi.org/10.1016/0738-081x(91)90022-d.

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2

ABE, Toshifumi, Michiyoshi KOUNO, Nobuhiro YAMAMOTO, Osamu MORI, Takashi HASHIMOTO, and Masayuki AMAGAI. "A Case of D-penicillamine Induced Herpetiform Pemphigus." Nishi Nihon Hifuka 65, no. 2 (2003): 126–29. http://dx.doi.org/10.2336/nishinihonhifu.65.126.

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3

De Cuyper, C. "Pemphigus herpetiformis in a lung cancer patient." Journal of the European Academy of Dermatology and Venereology 5, no. 1 (1995): S159. http://dx.doi.org/10.1016/0926-9959(95)96420-d.

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4

STIMOU, Sanae, Hafsa TAHERI, Hanane SAADI, and Ahmed MIMOUNI. "IMPETIGO HERPETIFORMIS IN A CASE REPORTPRESENTATION OF A RARE ENTITY." International Journal of Advanced Research 11, no. 01 (2023): 641–43. http://dx.doi.org/10.21474/ijar01/16061.

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Impetigo herpetiformis (IH) is a very rare and specific pustular dermatosis of pregnancy that can be life-threatening for both mother and child. This entity is currently considered as a variant of pustular psoriasis. We report an observation of a 35-year-old female patient who presented with extensive erythematous-pustular plaques for which the diagnosis of impetigo herpetiformiswas made clinically and histologically. The lesions appeared in the third trimester of pregnancy without fetal or maternal impact. The treatment was prednisone, vitamin D supplementation and a macrolide for 10 days.

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5

Hertl, Michael, Andrea Niedermeier, and Luca Borradori. "Bullöse Autoimmundermatosen." Therapeutische Umschau 67, no. 9 (2010): 465–82. http://dx.doi.org/10.1024/0040-5930/a000080.

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Blasenbildende Autoimmunerkrankungen sind seltene, meist schwere Erkrankungen von Haut und Schleimhäuten, die immunologisch durch zirkulierende Antikörper gegen Adhäsionsmoleküle der Epidermis und der dermoepidermalen Junktionszone charakterisiert sind; diese führen zum Adhäsionsverlust von Haut und Schleimhäuten und klinisch zur Ausbildung von Blasen oder Erosionen. Beim Pemphigus findet der Adhäsionsverlust in der Epidermis statt, während beim Pemphigoid, der linearen IgA-Dermatose, der Epidermolysis bullosa acquisita und der Dermatitis herpetiformis Duhring der Adhäsionsverlust subepidermal

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6

NAKAYAMA, Kanichiro, Yoshinori SUENAGA, Teruo UENO, and Seiichiro AONO. "A case of herpetiform pemphigus-like skin lesions induced by D-penicillamine." Nishi Nihon Hifuka 48, no. 6 (1986): 1054–59. http://dx.doi.org/10.2336/nishinihonhifu.48.1054.

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7

Furumura, Minao, Shuhei Imayama, and Yoshiaki Hori. "Three neonatal cases of epidermolysis bullosa herpetiformis (Dowling-Meara type) with severe erosive skin lesions." Journal of the American Academy of Dermatology 28, no. 5 (1993): 859–61. http://dx.doi.org/10.1016/0190-9622(93)70118-d.

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8

De Benedictis-Serrano, Ginno Alessandro, Laura Contreras-Lugo, Génesys Córdova-Rivas, and Carlos Miguel Ríos-González. "El esprúe tropical, una enfermedad olvidada como diagnóstico diferencial de la enfermedad celiaca." Revista de la Facultad de Medicina 66, no. 1 (2018): 129–30. http://dx.doi.org/10.15446/.v66n1.68105.

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Estimado editor, Por medio de la presente nos gustaría traer a discusión la importancia que tiene la enfermedad celiaca (EC), también conocida como esprúe celiaco o enteropatía sensible al gluten, la cual corresponde a una enfermedad autoinmune desencadenada por la ingesta de gluten en individuos predispuestos genéticamente (1). La EC se caracteriza por una amplia variabilidad de manifestaciones clínicas, pero su forma clásica es la más habitual en el adulto; aquí los síntomas guía son de naturaleza gastrointestinal (diarrea, esteatorrea y dolor abdominal inespecífico). Por otro lado, las form

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9

De Benedictis-Serrano, Ginno Alessandro, Laura Contreras-Lugo, Génesys Córdova-Rivas, and Carlos Miguel Ríos-González. "El esprúe tropical, una enfermedad olvidada como diagnóstico diferencial de la enfermedad celiaca." Revista de la Facultad de Medicina 66, no. 1 (2018): 129–30. http://dx.doi.org/10.15446/revfacmed.v66n1.68105.

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Estimado editor, Por medio de la presente nos gustaría traer a discusión la importancia que tiene la enfermedad celiaca (EC), también conocida como esprúe celiaco o enteropatía sensible al gluten, la cual corresponde a una enfermedad autoinmune desencadenada por la ingesta de gluten en individuos predispuestos genéticamente (1). La EC se caracteriza por una amplia variabilidad de manifestaciones clínicas, pero su forma clásica es la más habitual en el adulto; aquí los síntomas guía son de naturaleza gastrointestinal (diarrea, esteatorrea y dolor abdominal inespecífico). Por otro lado, las form

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10

Rizvi, Zainab, Nakhshab Choudhry, Aamir Jamal Gondal, and Nighat Yasmin. "Association of Surfactant Protein D Single Nucleotide Polymorphisms rs721917, rs2243639, rs3088308 with Recurrent Aphthous Stomatitis in Pakistani Population." Genes 14, no. 5 (2023): 1119. http://dx.doi.org/10.3390/genes14051119.

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Recurrent aphthous stomatitis (RAS) is a benign ulcerative condition, defined by the recurrent formation of non-contagious mucosal ulcers. Surfactant protein D (SP-D) is secreted frequently at surfaces exposed directly to body fluids. This study aims to investigate the association of SP-D single nucleotide polymorphisms (SNPs) with the onset of RAS. Blood samples from 212 subjects (106 cases/controls each) were collected during 2019 and genotyped for SP-D SNPs (rs721917, rs2243639, rs3088308) by polymerase chain reaction and restriction fragment length polymorphism followed by 12% polyacrylami

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11

Haussmann, Jessica, and Arni Sekar. "Chronic Urticaria: A Cutaneous Manifestation of Celiac Disease." Canadian Journal of Gastroenterology 20, no. 4 (2006): 291–93. http://dx.doi.org/10.1155/2006/871987.

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Celiac disease, or gluten-sensitive enteropathy, is an immune-mediated disease of the small bowel that results in malabsorption. It classically presents with gastrointestinal symptoms including chronic diarrhea, weight loss, abdominal bloating and anorexia. It is becoming more frequently identified in asymptomatic patients with a diagnosis of deficiencies related to malabsorption of iron, folic acid, vitamin B12and vitamin D. It is increasingly identified as a cause for early or refractory osteoporosis. Occasionally, celiac disease presents with cutaneous manifestations alone. Dermatitis herpe

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12

Tukaj, Stefan, Anna Görög, Konrad Kleszczyński, Detlef Zillikens, Sarolta Kárpáti, and Michael Kasperkiewicz. "Autoimmunity to heat shock proteins and vitamin D status in patients with celiac disease without associated dermatitis herpetiformis." Journal of Steroid Biochemistry and Molecular Biology 173 (October 2017): 23–27. http://dx.doi.org/10.1016/j.jsbmb.2016.10.002.

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13

Kajdas, Aleksandra Anna, Agnieszka Żebrowska, Anna Zalewska-Janowska, and Aneta Czerwonogrodzka-Senczyna. "The Role of Nutrition in the Pathogenesis and Treatment of Autoimmune Bullous Diseases—A Narrative Review." Nutrients 16, no. 22 (2024): 3961. http://dx.doi.org/10.3390/nu16223961.

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Autoimmune bullous diseases (AIBDs) are a group of conditions marked by the formation of blisters and erosions on the skin and mucous membranes. It occurs in all age groups, slightly more often affecting women. Several factors may be linked to the development of AIBDs, with nutrition being one of them. The literature mentions various food products and food ingredients acting as disease modifiers. Given the complex relationship between bullous diseases and nutrition, the current literature on AIBDs has been reviewed, with an emphasis on the influence of dietary modifications, various diets, and

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14

Zebrowska, Agnieszka, Malgorzata Wagrowska-Danilewicz, Marian Danilewicz, et al. "Tissue Factor in Dermatitis Herpetiformis and Bullous Pemphigoid: Link between Immune and Coagulation System in Subepidermal Autoimmune Bullous Diseases." Mediators of Inflammation 2015 (2015): 1–9. http://dx.doi.org/10.1155/2015/870428.

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Dermatitis herpetiformis (DH) and bullous pemphigoid (BP) are skin diseases associated with eosinophilic and neutrophilic infiltrations. Although chemokines are critical for the selective accumulation and activation of various leukocyte subsets in the inflammatory process, there are few findings concerning inflammatory cells and production of coagulation factors in blistering diseases. Skin biopsies were taken from 14 patients with DH, 27 with BP, and 20 control subjects. The localization and expression of tissue factor (TF) in skin lesions and perilesional skin were studied by immunohistochem

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15

Gaglani, Bindi, Paola De Mozzi, and Sophia Watts. "CPC11 A case of C3 bullous dermatosis." British Journal of Dermatology 191, Supplement_1 (2024): i13. http://dx.doi.org/10.1093/bjd/ljae090.024.

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Abstract A 44-year-old Asian man was seen as an urgent referral in dermatology for insidious-onset multiple pruritic tense serous-filled blisters. These were of varying sizes and occurred over acral sites and the body, sparing mucosal sites. He has a history of eczema as a child, which was in remission now, and no other medical problems and no regular medications. He denied any bowel problems. A punch biopsy showed subepidermal blistering with few eosinophils and a perivascular lymphocytic infiltrate. A perilesional indirect immunofluorescence was negative for IgG, IgM, IgA and C3 initially, a

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16

Samira, Omar* Fatima Elhawil Salma Sanok Rabea Markus Mohamed Frandah. "Clinical and Biochemical Profile of Libyan Patients with Serology and Biopsy Proven Celiac Disease." Alq J Med App Sci 5, no. 1 (2022): 263–66. https://doi.org/10.5281/zenodo.6562809.

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Background and aims. Celiac disease is a long-term immune disorder that primarily affects the small intestine. Iron deficiency anemia is the most widely experienced anemia in humans, which is a common extra intestinal manifestation of celiac disease. The aim of the current study is to describe the clinical and biochemical profile of adult Libyan celiac disease patients. Method. This was a case series study conducted by reviewing the medical records of the 100 patients diagnosed as celiac disease and followed in gastro-enterology clinic in Tripoli university hospital during the study period. A

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17

Sanae, STIMOU, TAHERI Hafsa, SAADI Hanane, and MIMOUNI Ahmed. "IMPETIGO HERPETIFORMIS IN A CASE REPORTPRESENTATION OF A RARE ENTITY." January 30, 2023. https://doi.org/10.5281/zenodo.7619987.

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Impetigo herpetiformis (IH) is a very rare and specific pustular dermatosis of pregnancy that can be life-threatening for both mother and child. This entity is currently considered as a variant of pustular psoriasis. We report an observation of a 35-year-old female patient who presented with extensive erythematous-pustular plaques for which the diagnosis of impetigo herpetiformiswas made clinically and histologically. The lesions appeared in the third trimester of pregnancy without fetal or maternal impact. The treatment was prednisone, vitamin D supplementation and a macrolide for 10 days.&nb

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18

Melo, Mariana das Neves, and Elisangela Pegas. "Acometimento neonatal do pênfigo vulgar: relato de caso." ARCHIVES OF HEALTH INVESTIGATION 9, no. 2 (2020). http://dx.doi.org/10.21270/archi.v9i2.5057.

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Os pênfigos são dermatoses bolhosas autoimunes, em que há a produção de autoanticorpos direcionados contra moléculas de adesão dos epitélios, levando à perda da coesão celular. As doenças bolhosas intraepidérmicas são compostas pelo grupo dos pênfigos: pênfigo foliáceo clássico, pênfigo foliáceo endêmico, pênfigo eritematoso, pênfigo vulgar, pênfigo vegetante, pênfigo herpetiforme (variante clínica dos pênfigos vulgar e foliáceo), pênfigo por IgA, pênfigo paraneoplásico e pênfigo induzido por medicamentos. Devido à transferência placentária dos anticorpos IgG, existem casos de recém-nascidos c

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19

Liu, Yan, Jie Ma, Haiyun Wang, et al. "P1126PLASMAPHERESIS TREATMENT FOR SEVERE PEMPHIGUS: 10 YEARS' EXPERIENCE OF A SINGLE CENTER IN CHINA." Nephrology Dialysis Transplantation 35, Supplement_3 (2020). http://dx.doi.org/10.1093/ndt/gfaa142.p1126.

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Abstract Background and Aims Pemphigus is a potentially life-threatening autoimmune bullous disorder. The pathogenesis involves with IgG antibodies against desmogleins (Dsg) (Dsg 1 and Dsg 3) on epithelial cell surface. Systemic corticosteroid is considered as the basis of the treatment for pemphigus. As an interventional treatment, plasmapheresis was introduced for treating severe or refractory pemphigus. The literature in this field is limited. This study aimed to review our 10 years’ experience in a single center. Method We retrospectively analyzed 17 patients with severe pemphigus who were

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20

Bobotsis, Robert, Michael Sawchuk, Jenny Shu, and Mariamma G. Joseph. "An Unusual Case of Drug-Induced Linear IgA Bullous Disease." Canadian Journal of General Internal Medicine 12, no. 2 (2017). http://dx.doi.org/10.22374/cjgim.v12i2.239.

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We describe a case of vancomycin-induced linear IgA bullous dermatosis (LABD) with unusual features in a 75-year old woman who received intravenous vancomycin for methicillin-resistant Staphylococcus Aureus infection. Our patient demonstrated an unusual lesional distribution with initial facial involvement and a longer latent period than usual, which to our knowledge has not yet been reported in the literature. A skin punch biopsy including direct immunofluorescence study (IMF) confirmed the diagnosis of LABD. The patient’s cutaneous eruptions resolved over the next 14 days with stoppage of me

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