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Dissertations / Theses on the topic 'D. melanogaster'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 September 2023

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1

Broderick, Kate Elizabeth. "Cyclic GMP - dependent signalling in D. melanogaster Malpighian tubules." Thesis, University of Glasgow, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.252518.

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2

Gutzwiller, Florence. "Evolution and gene expression of Wolbachia in D. melanogaster." Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/evolution-and-gene-expression-of-wolbachia-in-d-melanogaster(621f6995-e64b-40bc-8765-8eb48a8ce0cf).html.

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Wolbachia is a facultative endosymbiont infecting D. melanogaster, among many other arthropod species. In D. melanogaster, Wolbachia is a reproductive manipulator, but also gives some benefits to its host, such as protection against viruses. We used open access libraries of D. melanogaster resequencing data and yet unpublished sequences from our lab, to study the phylogenomics and geographical diversity of Wolbachia in D. melanogaster from five continents. We confirmed the clade structure of Wolbachia infecting D. melanogaster and the vertical transmission of Wolbachia from a single ancestral

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3

Osterried, Lea [Verfasser], Michael [Akademischer Betreuer] Köttgen, and Alexis [Akademischer Betreuer] Hofherr. "Untersuchung der Eigenschaften des Ionenkanals TRPP2 in D. melanogaster." Freiburg : Universität, 2019. http://d-nb.info/1207269670/34.

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4

Vincent, Amanda. "A D. melanogaster parkin mutant larval model of Parkinson's Disease." Thesis, University of York, 2013. http://etheses.whiterose.ac.uk/7535/.

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Parkinson’s Disease (PD) is a neurodegenerative disease, with severely reduced movement in patients. The main effect is the loss of dopaminergic neurons in the central nervous system (CNS). Null mutations of the parkin gene are known to cause PD. I found that Drosophila melanogaster (D. melanogaster) parkin null (dparkin) mutant larvae show neurophysiological abnormalities, a bradykinesia-like locomotory defect and synaptic overgrowth at the neuromuscular junction (NMJ). Neuronal overgrowth is rescued with either muscle or neuronal expression of wild-type dparkin in dparkin mutant larvae. The

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5

Jackson, Catherine. "Gfat1/zeppelin is an essential heterochromatic gene involved in cuticle formation in D. melanogaster /." Burnaby B.C. : Simon Fraser University, 2007. http://ir.lib.sfu.ca/handle/1892/9354.

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Thesis (M.Sc.) - Simon Fraser University, 2007.<br>Theses (Dept. of Molecular Biology and Biochemistry) / Simon Fraser University. Senior supervisor: Dr. Barry M. Honda -- Dept. of Molecular Biology and Biochemistry. Also issued in digital format and available on the World Wide Web.

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6

Masrouha, Nisrine. "Functional analysis of the Drosophila chk2 gene, loki : analysis of novel genetic interactors of Bic-D in Drosophila melanogaster." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=80329.

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Cell cycle checkpoints are signal transduction pathways that control the order and timing of cell cycle transitions, ensuring that critical events are completed before the cell cycle proceeds. The Chk2 family of kinases plays a central role in mediating responses to DNA damage or DNA replication blocks in various organisms. My functional analysis of the Drosophila serine/threonine kinase Loki/Chk2 shows that fly chk2 monitors double-strand breaks caused by irradiation during S and G2 phases and induces cell cycle arrest in embryonic cells around cellularization.<br>loki is also required

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7

Emsley, Jason G. "Computer 3-D reconstruction of the embryonic CNS of Drosophila melanogaster." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/MQ36434.pdf.

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8

Martin, Jennifer. "A Male-killing Bacterium Targets the Neural Tissue in D. melanogaster." Scholarship @ Claremont, 2013. http://scholarship.claremont.edu/scripps_theses/275.

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Bacteria living within the cells of eukaryotic organisms can have profound effects on their hosts. One example is host sex-ratio distortion caused by bacterial endosymbionts, which can be induced through the killing of male progeny during their development. The mechanisms that underlie how bacteria can cause male death are poorly understood. Several previous studies suggested that the neural, and perhaps other, tissues are targeted by the male-killing bacterium, Spiroplasma.In this study, I tested this hypothesis, addressing whether tissues were specifically altered during male development and

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9

Sealfon, Rachel (Rachel Sima). "Predicting enhancer regions and transcription factor binding sites in D. melanogaster." Thesis, Massachusetts Institute of Technology, 2010. http://hdl.handle.net/1721.1/62434.

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Thesis (S.M.)--Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, 2010.<br>Cataloged from PDF version of thesis.<br>Includes bibliographical references (p. 71-75).<br>Identifying regions in the genome that have regulatory function is important to the fundamental biological problem of understanding the mechanisms through which a regulatory sequence drives specific spatial and temporal patterns of gene expression in early development. The modENCODE project aims to comprehensively identify functional elements in the C. elegans and D. melanogaster genomes.

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10

Ma, Hsiu-Ching. "Discovery and characterisation of new miRNAs during embryogenesis of D. melanogaster." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609448.

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11

Balinski, Michael A. "Differential Sexual Survival of D. Melanogaster on Copper Sulfate." Bowling Green State University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1462973269.

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12

Kienitz, Bastian [Verfasser]. "Motorisches Lernen in Drosophila melanogaster / Bastian Kienitz." Mainz : Universitätsbibliothek Mainz, 2012. http://d-nb.info/1021244163/34.

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13

Kienitz, Bastian [Verfasser]. "Motorisches Lernen in Drosophila melanogaster / Bastian Kienitz." Aachen : Shaker, 2010. http://d-nb.info/1081887095/34.

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14

Berg, Christian [Verfasser]. "Kurzzeit-Orientierungsstrategien in Drosophila melanogaster / Christian Berg." Mainz : Universitätsbibliothek Mainz, 2012. http://d-nb.info/1029908745/34.

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15

Praxenthaler, Heiko [Verfasser]. "Molekulare und genetische Analyse des Notch-Repressorkomplexes in D. melanogaster / Heiko Praxenthaler." München : Verlag Dr. Hut, 2015. http://d-nb.info/1075408946/34.

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16

Zonato, Valeria. "Molecular genetic analysis of a seasonal character in D. melanogaster natural populations." Thesis, University of Leicester, 2013. http://hdl.handle.net/2381/27722.

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D. melanogaster has Afrotropical origins, colonising Europe around 10-15 thousand years ago, where they faced the challenges of a variable, seasonal environment. Individuals able to predict oncoming unfavourable winter conditions were at a selective advantage. This may have led to the evolution of seasonal diapause, a physiological response allowing flies to overwinter. In this work I studied the adaptation of the D. melanogaster overwintering strategy to different environmental conditions. Several genes have been found to modulate diapause, and some of these are characterised by two (or more)

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17

Murphy, Kyle Robert. "Nanosilver and CNT-Nanocomposite Toxicology in an In Vivo Model, D. Melanogaster." University of Dayton / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=dayton1429977804.

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18

Courtier-Orgogozo, Virginie. "Formation des organes sensoriels chez D. Melanogaster : lignages cellulaires, apoptose et évolution." Paris 6, 2003. http://www.theses.fr/2003PA066242.

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19

Garcia, Guerreiro Maria del pilar. "Polymorphisme d'insertion de différents éléments transposables dans une population naturelle de D. Melanogaster." Lyon 1, 1994. http://www.theses.fr/1994LYO10303.

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Le but de cette these est d'etudier le polymorphisme d'insertion de trois elements transposables: copia, mdg3 et gypsy dans une population naturelle de drosophila melanogaster. Les differentes methodes de croisement ont ete testees et les criteres de choix explicites. Nos analyses ont montre que la distribution des elements transposables sur l'ensemble des sites ne suit pas une distribution de poisson. Ces resultats suggerent que la distribution de chaque element sur chaque bras est liee a la sequence du site cible, l'accessibilite des differentes regions de l'adn a l'insertion d'un element mo

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20

Agnel, Magali. "Le développement des disques imaginaux chez D. Melanogaster : clonage du gène Rotund et caractérisation de ses deux ARN messagers majeurs." Aix-Marseille 2, 1989. http://www.theses.fr/1989AIX22005.

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21

Heßner, Sabina [Verfasser], and Stephan [Akademischer Betreuer] Schneuwly. "Funktionelle Analyse des ATP13A2 Orthologs in D. melanogaster / Sabina Heßner ; Betreuer: Stephan Schneuwly." Regensburg : Universitätsbibliothek Regensburg, 2016. http://d-nb.info/1137701153/34.

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22

Swan, Andrew. "Initiation of developmental asymmetry by Drosophila Bic-D, DLis-1 and microtubules." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0021/NQ55384.pdf.

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23

Riviere, Nathalie. "Expression du rétrotransposon 412 dans les populations naturelles de Drosophila simulans et D melanogaster." Lyon 1, 2000. http://www.theses.fr/2000LYO10251.

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Les deux espèces jumelles D. Melanogaster et D. Simulans diffèrent pour leur nombre global de copies d'éléments transposables (ETs) et leur génome. D. Simulans a moins de copies que D. Melanogaster et le nombre de copies varie fortement entre populations naturelles. En effet, certains ETs sont absents de la plupart des populations de D. Simulans mais présents en nombre élevé de copies dans quelques-unes. Ces données suggèrent que le génome de l'espèce D. Simulans est en train d'être envahi par de nombreux ETs qui auraient été "mobilisés" récemment. Cette mobilisation serait reliée à la colonis

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24

Buch, Susanne. "Die Signaltransduktionswirkung von Insulin in der adulten Drosophila melanogaster." Karlsruhe : Forschungszentrum Karlsruhe, 2006. http://d-nb.info/983613893/34.

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25

Krause, Tammo [Verfasser]. "Physiologie eines erlernten Körpermodells in Drosophila melanogaster / Tammo Krause." Mainz : Universitätsbibliothek Mainz, 2015. http://d-nb.info/1075167817/34.

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26

Datar, Sanjeev Ashok. "Developmental regulation of growth and cell cycle progression in Drosophila melanogaster : a larval growth arrest screen, and molecular and genetic analysis of the cyclin D/Cdk4 complex /." Thesis, Connect to this title online; UW restricted, 2000. http://hdl.handle.net/1773/5008.

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27

Wester, Jorge Victor Wilfredo Cachay. "Caracterização molecular do módulo regulador TT (Traqueia-Tórax) de >Drosophila melanogaster." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/17/17136/tde-06062017-163006/.

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Estudos funcionais anteriores identificaram um módulo cis-regulador (MCR) de 67 pb (-253/- 187) na região promotora do gene de pufe de DNA BhC4-1 que dirige a expressão do gene repórter na glândula anelar de Drosophila melanogaster. Uma análise bioinformática identificou 67 sequências de D. melanogaster que são similares a sequências contidas no MCR de glândula anelar. Uma das sequências identificadas reside em um fragmento genômico de 657 pb localizado aproximadamente 2500 pb à montante do CG13711, 400 pb à montante do CG12493, em uma região genômica que constitui um dos íntrons do CG32239 (G

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28

Cheng, Zhiyue. "Development of Drosophila melanogaster as a tool for modelling Parkinson disease." Thesis, Griffith University, 2021. http://hdl.handle.net/10072/410476.

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Parkinson’s disease (PD) is a common neurodegenerative disorder that occurs predominantly in people over 50. The patients with PD would exhibit motor symptoms including bradykinesia, muscular rigidity, resting tremor. The typical pathologic features of PD include the formation of Lewy bodies and the loss of dopaminergic neurons in the substantia nigra. Since PD is a late-onset disease, diagnosis before the emergence of the movement disorder is difficult but desirable as early intervention is necessary to minimise the progression of the disease. Defining the underlying genetic components of PD

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29

Garrett, M. J. "Comparative genomic analysis as a tool for locating novel functional elements in D. melanogaster." Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.599326.

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The thesis starts with the examination of a dataset representing the sequences of expressed small RNAs in <i>D. melanogaster. </i>After an examination of the quality of the sequences and what parameters are appropriate for its post-processing, it is used to identify novel tRNA genes which are then validated by comparative analysis. The degree of conservation of a set of putative microRNAs identified using a similar technique is then examined and used to determine the probability that the dataset represents genuine microRNAs. Further investigation is carried out into the conservation of a speci

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30

Frank, Deborah Jean. "Regulation of cell growth in C. elegans and D. melanogaster by ncl-1/brat /." Thesis, Connect to this title online; UW restricted, 2000. http://hdl.handle.net/1773/5029.

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31

Walther, Steffen. "Nutrionomik Analyse nahrungsabhängiger Gen- und Proteinregulation in Drosophila melanogaster (Meigen) /." Karlsruhe : Forschungszentrum Karlsruhe, 2007. http://d-nb.info/985069902/34.

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32

Solanki, Narendra [Verfasser], and Martin [Gutachter] Heisenberg. "Novelty choice in Drosophila melanogaster / Narendra Solanki. Gutachter: Martin Heisenberg." Würzburg : Universität Würzburg, 2014. http://d-nb.info/1102827754/34.

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33

Rahn, Tasja [Verfasser]. "Molecular Characterization of EGFR Signaling in Drosophila melanogaster / Tasja Rahn." Kiel : Universitätsbibliothek Kiel, 2012. http://d-nb.info/106366893X/34.

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34

Sareen, Preeti [Verfasser], and Martin [Akademischer Betreuer] Heisenberg. "Visual attention in Drosophila melanogaster / Preeti Sareen. Betreuer: Martin Heisenberg." Würzburg : Universitätsbibliothek der Universität Würzburg, 2012. http://d-nb.info/102057092X/34.

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35

Walther, Steffen. "Nutrionomik Analyse nahrungsabhängiger Gen- und Proteinregulation in Drosophila melanogaster (Meigen)." Karlsruhe Forschungszentrum Karlsruhe, 2006. http://d-nb.info/985069902/34.

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36

Helmstädter, Martin [Verfasser], and Karl-Friedrich [Akademischer Betreuer] Fischbach. "Funktionelle Untersuchung der Säuger-Neph-Proteine im Modellsystem Drosophila melanogaster." Freiburg : Universität, 2013. http://d-nb.info/1123475431/34.

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37

Solanki, Narendra [Verfasser], and Martin [Akademischer Betreuer] Heisenberg. "Novelty choice in Drosophila melanogaster / Narendra Solanki. Betreuer: Martin Heisenberg." Würzburg : Universitätsbibliothek der Universität Würzburg, 2013. http://d-nb.info/1038151686/34.

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38

Mayer, Lisa [Verfasser]. "cis-Regulation des Drosophila-melanogaster-Gens optomotor-blind / Lisa Mayer." Mainz : Universitätsbibliothek Mainz, 2012. http://d-nb.info/1031361456/34.

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39

Bobkov, Georg Otto Milan [Verfasser], and Patrick [Akademischer Betreuer] Heun. "Identification and characterization of novel centromeric proteins in Drosophila Melanogaster." Freiburg : Universität, 2016. http://d-nb.info/1155097203/34.

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40

Serpe, Rossana. "Identification of clock neurons and downstream circuits that are involved in sleep control in Drosophila melanogaster." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLS257.

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Le moment, la qualité et la quantité de sommeil dépendent de l'interaction fine entre l'horloge circadienne et la machinerie homéostatique (Borbely A. et al., 1982, Daan S. et al., 1984, Borbely et Achermann, 1999). Au cours des dernières années, l'utilisation de divers organismes modèles a fourni de nouvelles perspectives sur les mécanismes neuronaux et moléculaires de la régulation du sommeil (Miyazaki S. et al., 2017). Cependant, les bases moléculaires de l'homéostasie du sommeil et les circuits neuronaux sous-jacents à son interaction avec le réseau circadien n'ont pas été établis en détai

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41

Vieira-Heddi, Cristina. "Comportement de rétrotransposons (copia, mdg1, gypsy et 412) dans les populations de Drosophila melanogaster et D. Simulans." Lyon 1, 1996. http://www.theses.fr/1996LYO10273.

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Les elements transposables des genomes, qui sont responsables d'une fraction importante des mutations, participent a la diversite genetique. Il est donc fondamental de comprendre les mecanismes qui regulent leur nombre de copies dans les populations naturelles. Nous avons compare le comportement de 4 retrotransposons, copia, mdgl, 412 et gypsy, pour leur nombre de copies et leur localisation chromosomique, dans plusieurs populations naturelles de d. Simulans et d. Melanogaster. Les elements n'ont pas le meme comportement chez les deux especes ; certain qui sont fixes chez d. Simulans sont tres

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42

Hidalgo-Downing, Alicia. "Molecular cloning of patched and analysis of its role in intrasegmental patterning in D. melanogaster." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.258158.

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43

Srouji, John. "A Structural and Functional Investigation of Germ Plasm Organization Mediated by D. Melanogaster Oskar Protein." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:23845508.

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Germ cells are the unique source of gametes for multicellular organisms and are specified through different mechanisms. One such specification mechanism is inheritance-mediated, in which the molecular factors necessary and sufficient to impart germ cell fate (collectively referred to as “germ plasm”) are maternally synthesized and deposited during oogenesis or early embryogenesis. Incorporation of this germ plasm into newly formed cells results in primordial germ cells. This mechanism stands in contrast to the predominant metazoan (and likely ancestral) mode of germ cell specification termed i

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44

Protzer, Cornelia. "Einfluss des Notch Antagonisten Hairless auf apoptotische Prozesse in Drosophila melanogaster /." München : Verl. Dr. Hut, 2008. http://d-nb.info/991285255/04.

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45

Buch, Susanne [Verfasser]. "Die Signaltransduktionswirkung von Insulin in der adulten Drosophila melanogaster / Susanne Buch." Karlsruhe : Forschungszentrum Karlsruhe, 2006. http://d-nb.info/983613893/34.

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46

Hoffmann, Inka [Verfasser]. "Identifizierung und Verifizierung von Tbx-Zielgenen in Drosophila melanogaster / Inka Hoffmann." Mainz : Universitätsbibliothek Mainz, 2017. http://d-nb.info/1143987187/34.

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47

Mohr, Carmen [Verfasser], and Britta [Akademischer Betreuer] Hartmann. "Sex-specific transcript complexity in the neural system of Drosophila melanogaster." Freiburg : Universität, 2016. http://d-nb.info/1139639382/34.

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48

Smylla, Thomas [Verfasser]. "Mechanisms of Repression of Notch Signaling in Drosophila melanogaster / Thomas Smylla." München : Verlag Dr. Hut, 2017. http://d-nb.info/1135615446/34.

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49

Peters, Marc [Verfasser]. "Funktionelle neuroanatomische Analyse eines nahrungsabhängigen Schaltkreises in Drosophila melanogaster / Marc Peters." Bonn : Universitäts- und Landesbibliothek Bonn, 2014. http://d-nb.info/1048616118/34.

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50

Fink, Christine [Verfasser]. "Die Architektur des intestinalen Immunsystems der Taufliege Drosophila melanogaster / Christine Fink." Kiel : Universitätsbibliothek Kiel, 2012. http://d-nb.info/1053683421/34.

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