Relevant bibliographies by topics / D1 cells

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'D1 cells'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "D1 cells"

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Feng, Hanping, Yi Zeng, Michael W. Graner, and Emmanuel Katsanis. "Stressed apoptotic tumor cells stimulate dendritic cells and induce specific cytotoxic T cells." Blood 100, no. 12 (2002): 4108–15. http://dx.doi.org/10.1182/blood-2002-05-1389.

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We have previously reported that stressed apoptotic tumor cells are more immunogenic in vivo than nonstressed ones. Using confocal microscopy we have confirmed our previous observation that heat-stressed apoptotic 12B1-D1 leukemia cells(BCR-ABL+) express HSP60 and HSP72 on their surface. To explore how the immune system distinguishes stressed from nonstressed apoptotic tumor cells, we analyzed the responses of dendritic cells to these 2 types of apoptotic cells. We found that nonstressed and heat-stressed apoptotic 12B1-D1 cells were taken up by dendritic cells in a comparable fashion. However
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Muraoka, Rebecca S., Anne E. G. Lenferink, Brian Law, et al. "ErbB2/Neu-Induced, Cyclin D1-Dependent Transformation Is Accelerated in p27-Haploinsufficient Mammary Epithelial Cells but Impaired in p27-Null Cells." Molecular and Cellular Biology 22, no. 7 (2002): 2204–19. http://dx.doi.org/10.1128/mcb.22.7.2204-2219.2002.

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ABSTRACT ErbB2/Neu destabilizes the cyclin-dependent kinase (Cdk) inhibitor p27 and increases expression of cyclin D1. Therefore, we studied the roles of p27 and cyclin D1 in ErbB2-mediated mammary epithelial cell transformation. Overexpression of ErbB2 or cyclin D1 in p27+/− primary murine mammary epithelial cells resulted in increased proliferation, cyclin D1 nuclear localization, and colony formation in soft agar compared to those in p27+/+ cells. In contrast, ErbB2- or cyclin D1-overexpressing p27−/− cells displayed reduced proliferation, anchorage-independent growth, Cdk4 activity, cyclin
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Zhu, Bo, Ashutosh Shrivastava, Cristina Luongo, et al. "Catalysis leads to posttranslational inactivation of the type 1 deiodinase and alters its conformation." Journal of Endocrinology 214, no. 1 (2012): 87–94. http://dx.doi.org/10.1530/joe-11-0459.

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Previously, it was shown that the type 1 deiodinase (D1) is subject to substrate-dependent inactivation that is blocked by pretreatment with the inhibitor of D1 catalysis, propylthiouracil (PTU). Using HepG2 cells with endogenous D1 activity, we found that while considerable D1-mediated catalysis of reverse tri-iodothyronine (rT3) is observed in intact cells, there was a significant loss of D1 activity in sonicates assayed from the same cells in parallel. This rT3-mediated loss of D1 activity occurs despite no change in D1 mRNA levels and is blocked by PTU treatment, suggesting a requirement f
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Rosenwald, I. B., A. Lazaris-Karatzas, N. Sonenberg, and E. V. Schmidt. "Elevated levels of cyclin D1 protein in response to increased expression of eukaryotic initiation factor 4E." Molecular and Cellular Biology 13, no. 12 (1993): 7358–63. http://dx.doi.org/10.1128/mcb.13.12.7358-7363.1993.

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Cyclin D1 is a G1-specific cyclin that has been linked to lymphoid, parathyroid, and breast tumors. Recent studies suggested that high protein levels of cyclin D1 are not always produced when cyclin D1 mRNA is overexpressed in transfected cells, suggesting that posttranscriptional events may be important in cyclin D1 regulation. The mRNA cap-binding protein (eukaryotic initiation factor 4E [eIF-4E]) is a potential regulatory of several posttranscriptional events, and it can itself induce neoplastic transformation. Consequently, we examined eIF-4E as a potential regulator of cyclin D1. Overexpr
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Rosenwald, I. B., A. Lazaris-Karatzas, N. Sonenberg, and E. V. Schmidt. "Elevated levels of cyclin D1 protein in response to increased expression of eukaryotic initiation factor 4E." Molecular and Cellular Biology 13, no. 12 (1993): 7358–63. http://dx.doi.org/10.1128/mcb.13.12.7358.

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Cyclin D1 is a G1-specific cyclin that has been linked to lymphoid, parathyroid, and breast tumors. Recent studies suggested that high protein levels of cyclin D1 are not always produced when cyclin D1 mRNA is overexpressed in transfected cells, suggesting that posttranscriptional events may be important in cyclin D1 regulation. The mRNA cap-binding protein (eukaryotic initiation factor 4E [eIF-4E]) is a potential regulatory of several posttranscriptional events, and it can itself induce neoplastic transformation. Consequently, we examined eIF-4E as a potential regulator of cyclin D1. Overexpr
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Ladha, Mohamed H., Kwang Y. Lee, Todd M. Upton, Michael F. Reed, and Mark E. Ewen. "Regulation of Exit from Quiescence by p27 and Cyclin D1-CDK4." Molecular and Cellular Biology 18, no. 11 (1998): 6605–15. http://dx.doi.org/10.1128/mcb.18.11.6605.

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ABSTRACT The synthesis of cyclin D1 and its assembly with cyclin-dependent kinase 4 (CDK4) to form an active complex is a rate-limiting step in progression through the G1 phase of the cell cycle. Using an activated allele of mitogen-activated protein kinase kinase 1 (MEK1), we show that this kinase plays a significant role in positively regulating the expression of cyclin D1. This was found both in quiescent serum-starved cells and in cells expressing dominant-negative Ras. Despite the observation that cyclin D1 is a target of MEK1, in cycling cells, activated MEK1, but not cyclin D1, is capab
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Epner, Elliot M., Hui Liu, Jing Wang, and Mathew Thayer. "Transvection Mediated by the Translocated Cyclin D1 Gene in Mantle Cell Lymphoma." Blood 110, no. 11 (2007): 4156. http://dx.doi.org/10.1182/blood.v110.11.4156.4156.

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Abstract Cyclin D1 expression in B cells is deregulated by chromosome translocations involving the immunoglobulin heavy chain (IgH) locus in mantle cell lymphoma (MCL). Gene targeting experiments produced MCL cell lines that had lost the translocated t(11;14) and no longer expressed cyclin D1. In these cyclin D1 (−) cells, the nonrearranged cyclin D1 (CCND1) locus reverts from CpG hypomethylated to hypermethylated. Reintroduction of the translocated chromosome by somatic cell hybrid fusion induces loss of methylation at the unrearranged CCND1 locus. Thus, the translocated chromosome exerts a t
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Zwijsen, R. M., R. Klompmaker, E. B. Wientjens, P. M. Kristel, B. van der Burg, and R. J. Michalides. "Cyclin D1 triggers autonomous growth of breast cancer cells by governing cell cycle exit." Molecular and Cellular Biology 16, no. 6 (1996): 2554–60. http://dx.doi.org/10.1128/mcb.16.6.2554.

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Cyclin D1 controls G1-associated processes, including G0-to-G1 and G1-to-S transitions. This study demonstrates a novel aspect of cyclin D1 as a regulator of the transition between G1 and G0. Overexpression of cyclin D1 in MCF7 breast tumor cells resulted in a continued proliferation under low-serum conditions, whereas nonoverexpressing cells ceased to grow. This difference in growth was due to a reduced exit from G1 to G0 in cyclin D1-overexpressing cells. Our data therefore suggest a model in which cyclin D1 overexpression in tumor cells is responsible for hyperproliferation under growth fac
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Flood, Michael D., Johnnie M. Moore-Dotson, and Erika D. Eggers. "Dopamine D1 receptor activation contributes to light-adapted changes in retinal inhibition to rod bipolar cells." Journal of Neurophysiology 120, no. 2 (2018): 867–79. http://dx.doi.org/10.1152/jn.00855.2017.

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Dopamine modulation of retinal signaling has been shown to be an important part of retinal adaptation to increased background light levels, but the role of dopamine modulation of retinal inhibition is not clear. We previously showed that light adaptation causes a large reduction in inhibition to rod bipolar cells, potentially to match the decrease in excitation after rod saturation. In this study, we determined how dopamine D1 receptors in the inner retina contribute to this modulation. We found that D1 receptor activation significantly decreased the magnitude of inhibitory light responses fro
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Mori, Kazunori, Etsuko Hirao, Yosuke Toya, et al. "Competitive Nuclear Export of Cyclin D1 and Hic-5 Regulates Anchorage Dependence of Cell Growth and Survival." Molecular Biology of the Cell 20, no. 1 (2009): 218–32. http://dx.doi.org/10.1091/mbc.e08-04-0428.

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Anchorage dependence of cell growth and survival is a critical trait that distinguishes nontransformed cells from transformed cells. We demonstrate that anchorage dependence is determined by anchorage-dependent nuclear retention of cyclin D1, which is regulated by the focal adhesion protein, Hic-5, whose CRM1-dependent nuclear export counteracts that of cyclin D1. An adaptor protein, PINCH, interacts with cyclin D1 and Hic-5 and potentially serves as an interface for the competition between cyclin D1 and Hic-5 for CRM1. In nonadherent cells, the nuclear export of Hic-5, which is redox-sensitiv
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Dissertations / Theses on the topic "D1 cells"

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Wood, Eleanor Jane. "Phospholipase D1 dynamics in animal cells." Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.614780.

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Kennedy, Christopher R. J. "Signalling pathways of bradykinin-mediated arachidonic acid release in MDCK-D1 cells." Thesis, University of Ottawa (Canada), 1997. http://hdl.handle.net/10393/4074.

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An investigation was undertaken to elucidate the signal transduction pathways involved in bradykinin (BK)-mediated release of arachidonic acid (AA) from the D1 clone of Madin-Darby canine kidney cells (MDCK-D1) which display distal tubule- and cortical collecting duct principal cell-like characteristics. Prostaglandins (PG), generated subsequent to BK stimulation, are known to modulate arginine-vasopressin (AVP)-stimulated water flow across this portion of the nephron. The enzyme immediately responsible for AA release was determined to be the 85 kDa cytosolic phospholipase A$\sb2$ (cPLA$\sb2),
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Kennedy, Chris R. J. "Signalling pathways of bradykinin-mediated arachidonic acid release in MDCK-D1 cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq21003.pdf.

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Benitez-Rajal, Joaquin. "Phospholipase D1 : a possible role in nucleotide-mediated hepatic stellate cell contraction." Thesis, University of Newcastle Upon Tyne, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.366643.

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Matsuo, Koji. "Significance of dopamine D1 receptor signalling for steroidogenic differentiation of human induced pluripotent stem cells." Kyoto University, 2018. http://hdl.handle.net/2433/232124.

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Misiuta, Iwona E. "Characterization of the Dopaminergic Potential of the Human NTera2/D1 (NT2) Cell Line In Vitro." [Tampa, Fla.] : University of South Florida, 2005. http://purl.fcla.edu/fcla/etd/SFE0001184.

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吳毅賢 and Samuel Ng. "Extracellular calcium in dopamine D1-receptor mediated growth hormone release from Chinese grass carp pituitary cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B31219755.

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Ng, Samuel. "Extracellular calcium in dopamine D1-receptor mediated growth hormone release from Chinese grass carp pituitary cells /." Hong Kong : University of Hong Kong, 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B1947104X.

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Kruse, Maria Sol. "Plasticity of the dopamine 1 receptor and its signaling pathway /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-652-9/.

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Verardi, Laura <1988&gt. "The Function of Cyclin D1 in DNA Repair: A New Possible Approach to Increase Sensitivity of Ovarian Cancer Cells to Irradiation." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2017. http://amsdottorato.unibo.it/8070/1/Verardi_Laura_tesi.pdf.

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Cyclin D1 has been recently investigated as an important protein in homologous recombination-directed repair (HDR) mechanism. Cyclin D1 is recruited to damaged foci by BRCA2 and it enhances RAD51 binding to BRCA2. Furthermore, cyclin D1 is essential for RAD51 gene up-regulation in case of double-stranded break (DSB). Meanwhile molecule 3l, an E-3-(2-chloro-3-indolylmethylene)1,3-dihydroindol-2one, has been showed to reduce cyclin D1 level and, for this reason, 3l treatment could be used to study cyclin D1 role in DNA damage repair. Scope of this thesis is to find a new possible combined appro
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Books on the topic "D1 cells"

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Mukherjee, Tinku S. Regulation of the D1 dopamine receptor in rat brain and SK-N-Mc human neuroblastoma cells. National Library of Canada = Bibliothèque nationale du Canada, 1995.

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Penyusunan profil coastal cell (paket PP03-D1): Laporan akhir = final report. Galih Karsa Utama, 2003.

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Kauzman, Adel. Cyclin D1 alterations in giant cell tumor of bone and central giant cell granuloma of the jaw. 2004.

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Book chapters on the topic "D1 cells"

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Minei, Risako, Masato Wakakura, and Kimiyo Mashimo. "Dopamine D1 Receptor on Cultured Müller Cells." In The Neural Basis of Early Vision. Springer Japan, 2003. http://dx.doi.org/10.1007/978-4-431-68447-3_54.

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Insel, Paul A., Rennolds S. Ostrom, Martin C. Michel, and Rainer Büscher. "α1-Adrenergic Receptors of MDCK-D1 Cells Utilize Multiple Signalling Components." In Catecholamine Research. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4757-3538-3_60.

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Soitamo, A., G. Zhou, A. Clarke, G. Öquist, P. Gustafsson, and E. M. Aro. "Over-Production of the D1:2 Protein in Synechococcus sp. PCC 7942 Cells." In Photosynthesis: from Light to Biosphere. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0173-5_745.

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Banes, A. J., M. Sanderson, S. Boitano, et al. "Mechanical Load ± Growth Factors Induce [Ca2+]i Release, Cyclin D1 Expression and DNA Synthesis in Avian Tendon Cells." In Cell Mechanics and Cellular Engineering. Springer New York, 1994. http://dx.doi.org/10.1007/978-1-4613-8425-0_13.

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Baermann, Ben-Niklas, and Sascha Dietrich. "Mantle Cell Lymphoma." In The EBMT Handbook. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-44080-9_87.

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AbstractMantle cell lymphoma (MCL) is an aggressive B-cell lymphoma, which is characterized by the chromosomal translocation t(11;14)(q13;q32) and overexpression of cyclin D1 in the vast majority of cases. Most patients present with advanced stage disease often with extra-nodal dissemination. High Ki67 proliferation index (Hoster et al. 2016), blastoid histologic variant (Bernard et al. 2001), and TP53 mutations represent high-risk features at diagnosis (Eskelund et al. 2017).
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Zer, H., N. Keren, and I. Ohad. "The Regulatory Role of the QB Site in the RCII-D1 Protein Degradation is Gradually Lost Following Exposure of Chlamydomonas Cells to Low Light." In Photosynthesis: from Light to Biosphere. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0173-5_768.

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Malki, Zakaria El, Anass El karkri, Mohamed Bouachrine, and Françoise-Serein Spirau. "Theoretical Study of New Compounds (D1-BT-EDOT-BT-D2-A) Based on 3, 4-Ethylenedioxythiophene (EDOT) and Benzothiadiazole (BT) for Dye Sensitized Solar Cells." In Lecture Notes in Electrical Engineering. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-36475-5_28.

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Wang, Chenguang, Timothy G. Pestell, and Richard G. Pestell. "A New Perspective on Cyclin D1: Beyond Cell Cycle Regulation." In Cell and Molecular Biology of Breast Cancer. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-634-4_13.

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Lavoie, Josée N., Nathalie Rivard, Gilles L’Allemain, and Jacques Pouysségur. "A temporal and biochemical link between growth factor-activated MAP kinases, cyclin D1 induction and cell cycle entry." In Progress in Cell Cycle Research. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-5873-6_5.

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Leila, Ghalmi, and Bensmaine Souhila. "Numerical Simulation of a Solar Cell in CdS/CIGS Using the SCAPS-D1 Software." In Artificial Intelligence and Renewables Towards an Energy Transition. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-63846-7_90.

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Conference papers on the topic "D1 cells"

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Verweij, C. L., M. Hart, and H. Pannekoek. "VON WILLEBRAND FACTOR (vWF) PRO-POLYPEPTIDE IS REQUIRED FOR vWF MULTIMER FORMATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642831.

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The von Willebrand factor (vWF) is a multimeric plasma glycoprotein synthesized in vascular endothelial cells as a pre-pro-polypeptide with a highly repetitive domain structure, symbolized by the formula:(H)-D1-D2-D'-D3-A1-A2-A3-D4-B1-B2-B3-C1-C2-(0H).A heterologous expression system, consisting of a monkey kidney cell line (C0S-1), transfected with full-length vWF cDNA, is shown to mimic the constitutively, secretory pathway of vWF in endothelial cells. The assembly of pro-vWF into multimers and the proteolytic processing of these structures is found to oro-ceed along the following, consecuti
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Zelivianski, S., A. Cooley, and J. Jeruss. "CDK4 Phosphorylation Regulates Smad3 Activity in Cyclin D1 Overexpressing Breast Cancer Cells." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-2152.

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Tsao, George Sai-Wah, C. M. Tsang, Y. L. Yip, et al. "Abstract 2037: Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2037.

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Hsiao, HsiMin, Elizabeth P. Levy, Robert A. Fulton, Thomas H. Thatcher, Richard P. Phipps, and Patricia J. Sime. "Resolvin D1 Attenuates The Inflammatory Response In Primary Human Small Airway Epithelial Cells." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1432.

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Xia, Wei, and Xiao Qi Wang. "Abstract 3055: Potential role of cyclin D1 in regulation of liver cancer stem cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3055.

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Ou, Wenbin, Weicai Chen, Jiaqing Zhu, et al. "Abstract 2707: Downregulation of cyclin D1 sensitizes cancer cells to MDM2 antagonist Nutlin-3." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-2707.

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Kushwaha, Ritu, Venkata Thodima, George J. Bosl, and Raju S. K. Chaganti. "Abstract 5162: MicroRNA regulation of epidermal lineage differentiation of NT2/D1 embryonal carcinoma cells." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5162.

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Mah, Jordina, Melody Duvall, Samuel Kemp, et al. "Resolvin D1 reduces the inflammatory response to rhinovirus infection in COPD bronchial epithelial cells." In ERS Congress 2024 abstracts. European Respiratory Society, 2024. http://dx.doi.org/10.1183/13993003.congress-2024.pa2972.

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Chen, Jun, Karthik V. Giridhar, and Qiming Jane Wang. "Abstract 3127: Protein kinase D1 downregulation facilities PMA-induced apoptosis in LNCaP prostate cancer cells." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3127.

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Jialing Wang and Ziqi Liang. "D2-D1-A random terpolymer based solar cells exhibiting 1 V of open-circuit voltage." In 2016 Progress in Electromagnetic Research Symposium (PIERS). IEEE, 2016. http://dx.doi.org/10.1109/piers.2016.7734784.

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Reports on the topic "D1 cells"

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จันทร์เจ้า, จันทร์เพ็ญ. ฤทธิ์ต้านการเพิ่มจำนวนของเซลล์มะเร็งของซีรูเมนจากชันโรง Tetragonula Laeviceps และพรอพอลิสจากผึ้งพันธุ์ Aris mellifera : รายงานผลการดำเนินงาน. จุฬาลงกรณ์มหาวิทยาลัย, 2015. https://doi.org/10.58837/chula.res.2015.52.

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ความเข้มข้นและระยะเวลาที่เลี้ยงร่วมกับคาร์ดานอลส่งผลต่อ Cytotoxicity ของ BT474 cells เมื่อสังเกตลักษณะทางสัณฐานวิทยาโดยใช้กล้องจุลทรรศน์แบบใช้แสง พบการหดตัวและการหลุดลอยของเซลล์ คาร์ดานอลทำให้เกิด Cell cycle arrest ที่ระยะย่อย G1 ในขณะที่ Doxorubicin ทำให้เกิด Cell cycle arrest ที่ระยะย่อย G2/M พบการตายของเซลล์แบบ Late apoptosis (27.2 ± 1.1%) และ Necrosis (25.4 ± 1.4%) หลังจากการเลี้ยงร่วม 72 ช.ม. ในขณะที่ Doxorubicin ทำให้เกิดการตายของเซลล์แบบ Apoptosis (4.3 ± 0.4%) และ Necrosis (35.8 ± 13.0%) นอกจากนี้ยังพบว่าคาร์ดานอลเปลี่ยนแปลงระดับการแสดงออกของยีนที่เกี่ยวข้องกับ Apoptosis (เพิ่มการแสดงออ
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Ohad, Itzhak, and Himadri Pakrasi. Role of Cytochrome B559 in Photoinhibition. United States Department of Agriculture, 1995. http://dx.doi.org/10.32747/1995.7613031.bard.

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The aim of this research project was to obtain information on the role of the cytochrome b559 in the function of Photosystem-II (PSII) with special emphasis on the light induced photo inactivation of PSII and turnover of the photochemical reaction center II protein subunit RCII-D1. The major goals of this project were: 1) Isolation and sequencing of the Chlamydomonas chloroplast psbE and psbF genes encoding the cytochrome b559 a and b subunits respectively; 2) Generation of site directed mutants and testing the effect of such mutation on the function of PSII under various light conditions; 3)
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