Journal articles: 'Deafness, Noise induced – Namibia'

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Zusho, Hiroyuki. "Perceptive Deafness and Presbycusis. Noise-Induced Deafness." AUDIOLOGY JAPAN 39, no. 2 (1996): 101–8. http://dx.doi.org/10.4295/audiology.39.101.

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Ohhashi, Masami, Nobuko Dota, Nobukiyo Satoh, Yuko Konari, Yuko Tachibana, and Yoshihiko Terayama. "Investigation of noise induced sudden deafness." AUDIOLOGY JAPAN 28, no. 5 (1985): 778–83. http://dx.doi.org/10.4295/audiology.28.778.

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SHITA, TOORU. "Japan Audiological Society and noise-induced deafness." AUDIOLOGY JAPAN 29, no. 3 (1986): 142–46. http://dx.doi.org/10.4295/audiology.29.142.

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HIGUCHI, AKIFUMI. "A clinical study on noise-induced deafness." AUDIOLOGY JAPAN 32, no. 5 (1989): 347–48. http://dx.doi.org/10.4295/audiology.32.347.

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MASAKI, MICHIKI. "Noise-induced deafness and medial high jugular vein bulb." AUDIOLOGY JAPAN 32, no. 5 (1989): 349–50. http://dx.doi.org/10.4295/audiology.32.349.

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Okamoto, Yoshito, Tamiki Daida, Ikuko Nosaka, Takako Namatame, and Hiroyuki Zusho. "Clinical Study of Noise-induced Deafness. PART 15. Statistics of Cases Recognized as Eligible for Worker's Accident Compensation Insurance for Noise-induced Deafness." AUDIOLOGY JAPAN 35, no. 2 (1992): 167–72. http://dx.doi.org/10.4295/audiology.35.167.

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OKAMOTO, KAZUTO. "Results of ABR and SR tests in noise-induced deafness." AUDIOLOGY JAPAN 32, no. 5 (1989): 345–46. http://dx.doi.org/10.4295/audiology.32.345.

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Sincihu, Yudhiakuari, Steven Steven, Mulya Dinata, and Melani Taurusia. "Investigation of Noise Induced Hearing Loss at Shipyard Company, Surabaya." Indonesian Journal of Occupational Safety and Health 8, no. 3 (December 27, 2019): 321. http://dx.doi.org/10.20473/ijosh.v8i3.2019.321-327.

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Introduction: Noise is a health risk that cannot be avoided in production process. Noise has the potential to cause hearing loss for workers. The bad news, noise damage due to noise is permanent. Audiometric screening at shipyard company employees found 81.2% experienced Sensory-Neural Hearing Loss. 61.5% of employees experience deafness in both ears. The research objective was to find the cause of deafness at shipyard company. Methods: A Quantitative research with cross sectional approach in 64 subjects who worked in the ship repair production unit. The sample is chosen with a simple random technique according to inclusion and exclusion criteria. Assessment was carried out using questionnaires, measurement, and direct observations. Result: The bad habit of wearing ear protectors on employees as a cause of deafness (p<.001, Coef .517**). Sound level measurement shows the activity of chipping, welding, cutting and outfitting with noise intensity more than the recommended threshold (85 dBA for 8 hour per day). Noisy sources were found such as blowers, compressors, grinders, cutting mach ines, ringlet machines, hammer blows on plates and generators. Conclusion: Hearing loss in shipyard company employees is a work-related disease caused by the poor culture of wearing ear protectors. The habits does not wear earplug/earmuff tools because its not comfortable in the ear when the appliance is used, the tools too small so its easily lost, forgetten to carry, and tool are not available.

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Sincihu, Yudhiakuari, Steven Steven, Mulya Dinata, and Melani Taurusia. "Investigation of Noise Induced Hearing Loss at Shipyard Company, Surabaya." Indonesian Journal of Occupational Safety and Health 8, no. 3 (December 27, 2019): 321. http://dx.doi.org/10.20473/ijosh.v8i3.2019.325-331.

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Introduction: Noise is a health risk that cannot be avoided in production process. Noise has the potential to cause hearing loss for workers. The bad news, noise damage due to noise is permanent. Audiometric screening at shipyard company employees found 81.2% experienced Sensory-Neural Hearing Loss. 61.5% of employees experience deafness in both ears. The research objective was to find the cause of deafness at shipyard company. Methods: A Quantitative research with cross sectional approach in 64 subjects who worked in the ship repair production unit. The sample is chosen with a simple random technique according to inclusion and exclusion criteria. Assessment was carried out using questionnaires, measurement, and direct observations. Result: The bad habit of wearing ear protectors on employees as a cause of deafness (p<.001, Coef .517**). Sound level measurement shows the activity of chipping, welding, cutting and outfitting with noise intensity more than the recommended threshold (85 dBA for 8 hour per day). Noisy sources were found such as blowers, compressors, grinders, cutting mach ines, ringlet machines, hammer blows on plates and generators. Conclusion: Hearing loss in shipyard company employees is a work-related disease caused by the poor culture of wearing ear protectors. The habits does not wear earplug/earmuff tools because its not comfortable in the ear when the appliance is used, the tools too small so its easily lost, forgetten to carry, and tool are not available.

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FUJITANI, SATOSHI. "Results of examination for cases of noise-induced deafness with tinnitus." AUDIOLOGY JAPAN 29, no. 5 (1986): 343–44. http://dx.doi.org/10.4295/audiology.29.343.

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Lim, Joseph. "Advanced noise-induced deafness among workers in singapore − what has changed?" Noise and Health 20, no. 97 (2018): 217. http://dx.doi.org/10.4103/nah.nah_32_18.

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Sugio, Yuichiro, Takako Namatame, Manabu Matsumoto, Yukiko Iida, Naohiko Watanabe, and Hiroyuki Zusho. "A Clinical Study of Noise-Induced Deafness. Aggravation for Hearing Loss." AUDIOLOGY JAPAN 42, no. 2 (1999): 147–51. http://dx.doi.org/10.4295/audiology.42.147.

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Noorhassim, Ismail, Kimitaka Kaga, and Kousuke Nishimura. "Pure-tone audiometry and auditory brainstem responses in noise-induced deafness." American Journal of Otolaryngology 17, no. 1 (January 1996): 31–35. http://dx.doi.org/10.1016/s0196-0709(96)90040-x.

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Wise, J. "Musicians are at increased risk of noise induced deafness, study finds." BMJ 348, apr30 6 (April 30, 2014): g3014. http://dx.doi.org/10.1136/bmj.g3014.

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French, Andrew S., and Ben Warren. "Gene transcription changes in a locust model of noise-induced deafness." Journal of Neurophysiology 125, no. 6 (June 1, 2021): 2264–78. http://dx.doi.org/10.1152/jn.00119.2021.

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Locust ears have functional and genetic similarities to human ears, including loss of hearing from age or noise exposure. We measured transcript abundances in transcriptomes of noise-exposed and control locust ears. The data indicate remodeling of the ear tympanum and profound reductions in metabolism that may explain reduced sound transduction. These findings advance our understanding of this useful model and suggest further experiments to elucidate mechanisms that ears use to cope with excessive stimulation.

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Ding, Tonghui, Aihui Yan, and Ke Liu. "What is noise-induced hearing loss?" British Journal of Hospital Medicine 80, no. 9 (September 2, 2019): 525–29. http://dx.doi.org/10.12968/hmed.2019.80.9.525.

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Noise-induced hearing loss is sensory deafness caused by long-term exposure of the auditory system to a noisy environment. Auditory fatigue is an early symptom of noise-induced hearing loss, and hearing can gradually recover after people leave a noisy environment. However, if people remain in a noisy environment for a prolonged period of time, their hearing will be permanently impaired. Societal changes mean that people are more likely to be exposed to noise. The hearing loss and tinnitus caused by noise seriously affect people's quality of life and lead to huge economic loss. The pathogenesis of noise-induced hearing loss is complex. Various theories try to explain this, such as the oxidative stress theory, but none perfectly explains the occurrence of noise-induced hearing loss. There is no treatment which can completely reverse the damage. More research is required to explore the pathogenesis and to better guide clinical practice. Preventative strategies, such as educating the public about hearing health, should be adopted to reduce the harm of noise-induced hearing loss.

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Asahina, Norihiko, Gen Nanba, Hajime Kuroda, Ikuko Nosaka, Takako Namatame, and Hiroyuki Zusho. "Clinical Study of Noise-induced Deafness. Part 17: A Study on Cases of Noise-induced Deafness as Viewed from Results of Screening Audiometry in General Health Examination." AUDIOLOGY JAPAN 37, no. 6 (1994): 726–31. http://dx.doi.org/10.4295/audiology.37.726.

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Sugiuchi, Tomoko, Ikuko Nosaka, Takako Namatame, Satoru Fujitani, Kimiko Asano, and Hiroyuki Zusho. "A clinical study on noise-induced deafness. 10. Results of speech audiometry." AUDIOLOGY JAPAN 30, no. 1 (1987): 54–59. http://dx.doi.org/10.4295/audiology.30.54.

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Whittaker, J. D., T. Robinson, A. Acharya, D. Singh, and M. Smith. "Noise-induced hearing loss in small-scale metal industry in Nepal." Journal of Laryngology & Otology 128, no. 10 (September 19, 2014): 871–80. http://dx.doi.org/10.1017/s0022215114001728.

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AbstractBackground:There has been no previous research to demonstrate the risk of noise-induced hearing loss in industry in Nepal. Limited research on occupational noise-induced hearing loss has been conducted within small-scale industry worldwide, despite it being a substantial and growing cause of deafness in the developing world.Method:The study involved a cross-sectional audiometric assessment, with questionnaire-based examinations of noise and occupational history, and workplace noise level assessment.Results:A total of 115 metal workers and 123 hotel workers (control subjects) were recruited. Noise-induced hearing loss prevalence was 30.4 per cent in metal workers and 4.1 per cent in hotel workers, with a significant odds ratio of 10.3. Except for age and time in occupation, none of the demographic factors were significant in predicting outcomes in regression analyses. When adjusted for this finding, and previous noise-exposed occupations, the odds ratio was 13.8. Workplace noise was significantly different between the groups, ranging from 65.3 to 84.7 dBA in metal worker sites, and from 51.4 to 68.6 dBA in the control sites.Conclusion:Metal workers appear to have a greater risk of noise-induced hearing loss than controls. Additional research on occupational noise-induced hearing loss in Nepal and small-scale industry globally is needed.

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Miyakita, Takashi, and Hajime Miura. "A tentative proposal for classification of audiograms in noise-induced deafness: relationship between audiogram and subjective complaints in noise-exposed workers." International Archives of Occupational and Environmental Health 57, no. 3 (March 1986): 173–83. http://dx.doi.org/10.1007/bf00405786.

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CHOSHO, HIROYUKI. "Clinical studies on noise-induced deafness. Changes of auditory acuity for10 years after leaving noisy occupation." AUDIOLOGY JAPAN 29, no. 5 (1986): 311–12. http://dx.doi.org/10.4295/audiology.29.311.

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Zusho, Hiroyuki, Takako Namatame, Ikuko Nosaka, Takeshi Kusumi, and Hitome Kobayashi. "A clinical study on noise-induced deafness. 11 Changes in hearing after desertion from noisy workshop." AUDIOLOGY JAPAN 30, no. 2 (1987): 81–86. http://dx.doi.org/10.4295/audiology.30.81.

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KOGA, KEIJIRO. "Cases of disco-induced deafness in which liabilily to injury was established by noise-load test." AUDIOLOGY JAPAN 32, no. 5 (1989): 353–54. http://dx.doi.org/10.4295/audiology.32.353.

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Yi, Eun Young, Myung Sook Kim, and Myung Jin Bae. "A Study on Imbalanced Sound Sensitivity of BA Earphones Inducing Deafness." Advanced Materials Research 1051 (October 2014): 915–19. http://dx.doi.org/10.4028/www.scientific.net/amr.1051.915.

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One of the popular ways to use smartphones in public places without disturbing others is to use them with earphones. Earphones are used by people of all ages for listening to music, watching sports/television programs, or even talking to someone over the phone. These devices can be generally classified into two types based on their driver unit, dynamic and BA (Balanced Armature). Dynamic type earphones perform relatively poor in withholding all the sound, letting some sound leak out. On the other hand, BA type earphones fit snuggly into the ears and prevent sound leak. Simultaneously, the users are being blocked out from outside noise. This paper will investigate the volume imbalance between the left and the right BA earphones using a coupler, and how users perceive the volume levels differently. The difference in voltage between the left and the right ones was significant, having 0.8mV with 43.9mV on the left and 44.7mV on the right to form 94dBSPL. This imbalance may cause noise induced deafness when continuously using earphones.

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FUJITANI, SATORU. "Clinical study of noise-induced deafness. Part 12: Results of tests on cases of tinnitus and evaluation." Nippon Jibiinkoka Gakkai Kaiho 93, no. 4 (1990): 543–53. http://dx.doi.org/10.3950/jibiinkoka.93.543.

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Zhou, Jiena, Zhihao Shi, Lifang Zhou, Yong Hu, and Meibian Zhang. "Occupational noise-induced hearing loss in China: a systematic review and meta-analysis." BMJ Open 10, no. 9 (September 2020): e039576. http://dx.doi.org/10.1136/bmjopen-2020-039576.

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ObjectiveMost of the Chinese occupational population are becoming at risk of noise-induced hearing loss (NIHL). However, there is a limited number of literature reviews on occupational NIHL in China. This study aimed to analyse the prevalence and characteristics of occupational NIHL in the Chinese population using data from relevant studies.DesignSystematic review and meta-analysis.MethodsFrom December 2019 to February 2020, we searched the literature through databases, including Web of Science, PubMed, MEDLINE, Scopus, the China National Knowledge Internet, Chinese Sci-Tech Journal Database (weip.com), WanFang Database and China United Library Database, for studies on NIHL in China published in 1993–2019 and analysed the correlation between NIHL and occupational exposure to noise, including exposure to complex noise and coexposure to noise and chemicals.ResultsA total of 71 865 workers aged 33.5±8.7 years were occupationally exposed to 98.6±7.2 dB(A) (A-weighted decibels) noise for a duration of 9.9±8.4 years in the transportation, mining and typical manufacturing industries. The prevalence of occupational NIHL in China was 21.3%, of which 30.2% was related to high-frequency NIHL (HFNIHL), 9.0% to speech-frequency NIHL and 5.8% to noise-induced deafness. Among manufacturing workers, complex noise contributed to greater HFNIHL than Gaussian noise (overall weighted OR (OR)=1.95). Coexposure to noise and chemicals such as organic solvents, welding fumes, carbon monoxide and hydrogen sulfide led to greater HFNIHL than noise exposure alone (overall weighted OR=2.36). Male workers were more likely to experience HFNIHL than female workers (overall weighted OR=2.26). Age, noise level and exposure duration were also risk factors for HFNIHL (overall weighted OR=1.35, 5.63 and 1.75, respectively).ConclusionsThe high prevalence of occupational NIHL in China was related to the wide distribution of noise in different industries as well as high-level and long-term noise exposure. The prevalence was further aggravated by exposure to complex noise or coexposure to noise and specific chemicals. Additional efforts are needed to reduce occupational noise exposure in China.

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Xue, Laijun, Yanhong Zhang, and Aichu Yang. "The Effect of a High-Frequency-Hearing-Threshold Weighted Value on the Diagnosis of Occupational-Noise-Induced Deafness." Journal of Biosciences and Medicines 06, no. 12 (2018): 88–99. http://dx.doi.org/10.4236/jbm.2018.612009.

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Zhou, Huaping, Jinpeng Zhou, Hui Li, Changye Hui, and Jing Bi. "Paraoxonase 3 gene polymorphisms are associated with occupational noise-induced deafness: A matched case-control study from China." PLOS ONE 15, no. 10 (October 15, 2020): e0240615. http://dx.doi.org/10.1371/journal.pone.0240615.

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Celaya, Adelaida M., Lourdes Rodríguez-de la Rosa, Jose M. Bermúdez-Muñoz, José M. Zubeldia, Carlos Romá-Mateo, Carlos Avendaño, Federico V. Pallardó, and Isabel Varela-Nieto. "IGF-1 Haploinsufficiency Causes Age-Related Chronic Cochlear Inflammation and Increases Noise-Induced Hearing Loss." Cells 10, no. 7 (July 3, 2021): 1686. http://dx.doi.org/10.3390/cells10071686.

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Insulin-like growth factor 1 (IGF-1) deficiency is an ultrarare syndromic human sensorineural deafness. Accordingly, IGF-1 is essential for the postnatal maturation of the cochlea and the correct wiring of hearing in mice. Less severe decreases in human IGF-1 levels have been associated with other hearing loss rare genetic syndromes, as well as with age-related hearing loss (ARHL). However, the underlying mechanisms linking IGF-1 haploinsufficiency with auditory pathology and ARHL have not been studied. Igf1-heterozygous mice express less Igf1 transcription and have 40% lower IGF-1 serum levels than wild-type mice. Along with ageing, IGF-1 levels decreased concomitantly with the increased expression of inflammatory cytokines, Tgfb1 and Il1b, but there was no associated hearing loss. However, noise exposure of these mice caused increased injury to sensory hair cells and irreversible hearing loss. Concomitantly, there was a significant alteration in the expression ratio of pro- and anti-inflammatory cytokines in Igf1+/− mice. Unbalanced inflammation led to the activation of the stress kinase JNK and the failure to activate AKT. Our data show that IGF-1 haploinsufficiency causes a chronic subclinical proinflammatory age-associated state and, consequently, greater susceptibility to stressors. This work provides the molecular bases to further understand hearing disorders linked to IGF-1 deficiency.

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Shevchenko, Oksana I., D. V. Rusanova, and O. L. Lakhman. "NEUROPHYSIOLOGICAL AND NEUROPSYCHOLOGICAL FEATURES AT PATIENTS FROM OCCUPATIONAL NOISE-INDUCED HEARING LOSS." Hygiene and sanitation 98, no. 10 (October 15, 2019): 1068–73. http://dx.doi.org/10.18821/0016-9900-2019-98-10-1068-1073.

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Introduction. Issues of studying the central origins and development of оccupational pathology in the faces of flight personnel define a need for the application of neurophysiological and neuropsychological methods of research. Material and methods. The levels of constant potential (LCP), characteristics the somatosensory caused potentials (SSVP), neuropsychological features in 95 patients with оccupational neurosensory deafness (ONSD) and 35 cases in a group of comparison are studied. Methods of the statistical analysis with a definition of W-of criterion of Shapiro-Uilka, Mann-Whitney›s U-criterion, coefficient of correlation of Spirmen, the discriminant analysis are applied. Results. in ONSD at patients the quantity of cases with the increased values of average LCP were established to prevail significantly in comparison with those in patients of a group of comparison (39% and 13% respectively, р = 0.03), strengthening of neuropower exchange in right and left frontal, central, left central, left parietal, right temporal assignments, increase in duration of the latent period of N18, N30 components are registered (р < 0.05). The condition of the cognitive sphere was characterized by the easily expressed violations of functions of analytical and synthetic thinking, оral-aural memory, visual, long-term memory, reciprocal coordination, impressive speech. Correlation connection between the index of N13-N20 and indices characterizing a condition of long-term memory, reciprocal coordination, the expressional speech is established (rs=0.45; 0.28; 0.28 at р =0.008; 0.04; 0.03 respectively), an index the between hemispheres of the relations Fd-Fs and latency of P25 and N30 (rs = 0.53 and 0.29 at р =0.009; 0.02 respectively). Diagnostic signs of brain deficiency at patients with ONSD were indices of visual gnosis, reciprocal coordination, LCP in right central, central parietal assignments of a brain, duration of the latent period of N30, N18-N20 (F=9.14; 5.43; 6.08; 4.41 4.77; 4.34 respectively). Conclusions. Violations of power metabolism in the frontal and central and parietal and temporal assignments of a brain, a functional condition of the central carrying-out structures, disorganization of cognitive activity were established to be a consequence of the impact of aviation noise and the reason of brain deficiency.

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Sari, Maesyara Adinda, Adlin Adnan, Delfitri Munir, and Putri Chairani Eyanoer. "Hubungan merokok dengan gangguan pendengaran akibat bising pada pekerja pabrik kelapa sawit X." Oto Rhino Laryngologica Indonesiana 47, no. 2 (January 7, 2018): 95. http://dx.doi.org/10.32637/orli.v47i2.218.

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Latar belakang: Paparan bising dapat mengakibatkan kerusakan pada organ korti, dan menurunkan aliran darah koklea yang mengakibatkan hipoksia koklea. Merokok diketahui sebagai ototoksik langsung dan pemicu iskemia koklea. Kebiasaan merokok dan paparan bising secara sendiri ataupun secara bersama-sama dapat menyebabkan gangguan pendengaran. Tujuan: Mengetahui hubungan merokok terhadap kejadian gangguan pendengaran akibat bising (GPAB). Metode: Penelitian analitik dengan pendekatan potong lintang. Hasil: Dari 122 pekerja, didapati sebanyak 89,3% pekerja mengalami GPAB. Tuli ringan paling banyak ditemukan (68,8%). Didapatkan adanya hubungan yang signifikan antara tipe perokok dengan derajat beratnya GPAB (p=0,000). Kemungkinan pekerja yg merokok untuk mengalami GPAB 1,224 kali lebih besar dibandingkan dengan pekerja yang tidak merokok (PR=1,224, p=0,002, CI 95%=1,077–1,392). Kesimpulan: Terdapat hubungan yang signifikan antara kebiasaan merokok dengan kejadian GPAB.Kata kunci: Merokok, gangguan pendengaran, gangguan pendengaran akibat bising, bising lingkungan kerja ABSTRACT Introduction: Noise exposure can lead to the organ of corti damage, and decrease the blood flow to the cochlea resulting in cochlear hypoxia. Cigarette smoking could be ototoxic to cochlea, and could evoke cochlear ischemia. Cigarette smoking habit and noise exposure, each or together can cause hearing loss. Objective: To identify the correlation between cigarette smoking and noise induced hearing loss (NIHL). Method: Analytic with a cross-sectional study. Results: From the 122 workers, we found that the proportion of NIHL was 89.3%. Mild deafness was most common (68.8%). We found that there was a significant correlation between type of smokers and grade of hearing loss (p=0.000). Workers who smoked likely to undergo NIHL 1.224 times greater than nonsmokers (PR=1.224, p=0.002, CI 95%=1.077–1.392). Conclusion: There was a significant correlation between cigarette smoking habit and noise induced hearing loss.Keywords : Cigarette smoking, hearing loss, noise induced hearing loss, occupational noise exposure

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Conroy, K., and V. Malik. "Hearing loss in the trenches – a hidden morbidity of World War I." Journal of Laryngology & Otology 132, no. 11 (October 29, 2018): 952–55. http://dx.doi.org/10.1017/s0022215118001755.

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AbstractBackgroundOne hundred years ago, millions of British and Allied troops were fighting in the trenches of the Great War. With a tenth of soldiers losing their lives, hearing loss seemed a low priority; however, vast numbers of troops sustained significant hearing loss.MethodA review was conducted of literature published between 1914 and 1925.ResultsSoldiers were exposed to up to 185 dB of sustained noise from new, high-energy weapons, which caused ‘labyrinthine concussion’. Traumatic injuries, non-organic hearing loss and malingering were also common. One source estimated that 2.4 per cent of the army was disabled by hearing loss. However, many British doctors viewed this ‘soldier's deafness’ as a temporary affliction, resulting in soldiers being labelled as malingerers or ‘hysterical’.ConclusionToday, one can recognise that a scant evidence base and misconceptions influenced the mismanagement of hearing loss by otolaryngologists in World War I. However, noise-induced hearing loss is still very much a feature of armed conflict.

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Bista, Madhab Raj, and Pranab Dahal. "Hearing Loss among the Traffic Police in Kathmandu Valley – finding from a pilot cross-sectional study." International Journal of Occupational Safety and Health 6, no. 2 (December 1, 2016): 1–5. http://dx.doi.org/10.3126/ijosh.v6i2.22525.

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Background: Noise induced hearing loss has been increasing rapidly with the advancement of technological, industrial and anthropogenic growth. Hearing loss is identified to be minimizing psychosocial well-being of an individual and reduced economic activities apart from deafness. This study aimed to measure the noise induced health impairment in traffic polices of Kathmandu valley. Methods and Materials: The cross-study was conducted among eighty traffic police in the Kathmandu and Lalitpur districts of Nepal. Purposive stratified sampling method was used for the study. A semi structured interview guide was developed to assess the physical findings and an audiometric test were also conducted with each individual traffic personnel to assess the hearing impairment. Results: The study identified Chabahil in Kathmandu with highest level of noise at 90 A-weighted decibel. Overall, moderate hearing loss in the left ear was reported in 55% of the respondents and mild hearing list in right ear in 46.3% of the respondents. The study found bilateral moderate hearing loss in all the respondent serving more than twenty years in traffic management. The effects of getting tired (80%), difficulties in concentration (76.3%) and increased irritation (72%) were identified as the high ranked health effects. Conclusion: The study reinforces the need of further exploration as this occupational health issue is a growing public health concern.

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Herzog, R. "Lärmminderung an Kistenwaschanlagen." Lärmbekämpfung 14, no. 05 (2019): 146–50. http://dx.doi.org/10.37544/1863-4672-2019-05-12.

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Kistenwaschmaschinen sind in der Lebensmittelindustrie, insbesondere in der Fleischbranche eine der dominanten Schallquellen und mitverantwortlich für das Krankheitsbild der berufsbedingten Lärmschwerhörigkeit (Berufskrankheit Nr. 2301). Entsprechend lärmgeminderte Anlagen werden beim Einkauf zu selten nachgefragt. Hersteller sind nach EG-Maschinenrichtlinie (2006/42/EG) verpflichtet, Schallemissionswerte (sog. Geräuschemissionsangaben) gleichwertig zu sonstigen Leistungsangaben in entsprechenden Verkaufsprospekten, Bedienungsanleitungen als auch auf Internetportalen anzugeben (Tabelle 1). Nur dadurch wird dem Arbeitgeber als Maschinenbetreiber die Möglichkeit gegeben, seiner Pflicht, geräuscharme Produktionsmittel nach dem Stand der Technik einzusetzen, nachzukommen. Die Alternative der nachträglichen Lärmminderung ist zwar möglich, aber immer mit zusätzlichen Kosten und Aufwand verbunden.   Summary Crate washing machines are one of the dominant sound sources in the food industry, especially in the meat industry, and responsible for the clinical picture of occupational noise-induced deafness (occupational disease No. 2301). Accordingly, quiet systems are rarely in demand when shopping. Manufacturers are required by the EC Machinery Directive (2006/42/EC) to specify sound emission values (so-called noise emission data) equivalent to other performance data in corresponding sales prospectuses, operating instructions and on Internet portals (table 1). Only in this way is the employer, as machine operator, given the opportunity to fulfill his duty to use low-noise production equipment according to the state of the art. The alternative of the subsequent noise abatement is possible but partly associated with additional costs and effort.

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Nourbakhsh, Aida, Brett M. Colbert, Eric Nisenbaum, Aziz El-Amraoui, Derek M. Dykxhoorn, Karl Russell Koehler, Zheng-yi Chen, and Xue Z. Liu. "Stem Cells and Gene Therapy in Progressive Hearing Loss: the State of the Art." Journal of the Association for Research in Otolaryngology 22, no. 2 (January 28, 2021): 95–105. http://dx.doi.org/10.1007/s10162-020-00781-0.

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AbstractProgressive non-syndromic sensorineural hearing loss (PNSHL) is the most common cause of sensory impairment, affecting more than a third of individuals over the age of 65. PNSHL includes noise-induced hearing loss (NIHL) and inherited forms of deafness, among which is delayed-onset autosomal dominant hearing loss (AD PNSHL). PNSHL is a prime candidate for genetic therapies due to the fact that PNSHL has been studied extensively, and there is a potentially wide window between identification of the disorder and the onset of hearing loss. Several gene therapy strategies exist that show potential for targeting PNSHL, including viral and non-viral approaches, and gene editing versus gene-modulating approaches. To fully explore the potential of these therapy strategies, a faithful in vitro model of the human inner ear is needed. Such models may come from induced pluripotent stem cells (iPSCs). The development of new treatment modalities by combining iPSC modeling with novel and innovative gene therapy approaches will pave the way for future applications leading to improved quality of life for many affected individuals and their families.

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Dong, Yunpeng, Tao Peng, Weijing Wu, Donghui Tan, Xuezhong Liu, and Dinghua Xie. "Efficient introduction of an isogenic homozygous mutation to induced pluripotent stem cells from a hereditary hearing loss family using CRISPR/Cas9 and single-stranded donor oligonucleotides." Journal of International Medical Research 47, no. 4 (February 28, 2019): 1717–30. http://dx.doi.org/10.1177/0300060519829990.

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Background Heterozygous purinergic receptor p2x gene ( P2RX2) c.178G>T (p.V60L) mutations can lead to progressive hearing loss (HL) and increased susceptibility to noise. However, the underlying mechanisms remain unclear. A combination of human induced pluripotent stem cell (hiPSC) technology with clustered regularly interspaced palindromic repeats (CRISPR)/CRISPR-associated protein (Cas)9-mediated gene editing may provide a promising tool to study gene function and treat hereditary deafness in humans. Methods hiPSC technology and CRISPR/Cas9-mediated gene editing were used to generate heterozygous and homozygous P2RX2 c.178G>T (p.V60L) cell models. Results We generated non-integrative hiPSCs from urine samples derived from three members of a large Chinese family carrying heterozygous P2RX2 c.178G>T mutations (designated P2RX2+/–) as a model to study P2RX2-mediated hereditary HL. Furthermore, we used CRISPR/Cas9 and single-stranded donor oligonucleotides to genetically establish homozygous P2RX2 c.178G>T hiPSCs (designated P2RX2–/–) from heterozygous patient-specific hiPSCs as a control to further study the pathological gene function. Conclusions Heterozygous and homozygous P2RX2-mutated hiPSC lines are good models to investigate the pathological mechanisms of P2RX2 mutations in HL pathogenesis. Our findings confirmed our hypothesis that it is feasible and convenient to introduce precise point mutations into genomic loci of interest to generate gene-mutated hiPSC models.

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Kraus, Frauke, Sarah Tune, Anna Ruhe, Jonas Obleser, and Malte Wöstmann. "Unilateral Acoustic Degradation Delays Attentional Separation of Competing Speech." Trends in Hearing 25 (January 2021): 233121652110132. http://dx.doi.org/10.1177/23312165211013242.

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Hearing loss is often asymmetric such that hearing thresholds differ substantially between the two ears. The extreme case of such asymmetric hearing is single-sided deafness. A unilateral cochlear implant (CI) on the more severely impaired ear is an effective treatment to restore hearing. The interactive effects of unilateral acoustic degradation and spatial attention to one sound source in multitalker situations are at present unclear. Here, we simulated some features of listening with a unilateral CI in young, normal-hearing listeners ( N = 22) who were presented with 8-band noise-vocoded speech to one ear and intact speech to the other ear. Neural responses were recorded in the electroencephalogram to obtain the spectrotemporal response function to speech. Listeners made more mistakes when answering questions about vocoded (vs. intact) attended speech. At the neural level, we asked how unilateral acoustic degradation would impact the attention-induced amplification of tracking target versus distracting speech. Interestingly, unilateral degradation did not per se reduce the attention-induced amplification but instead delayed it in time: Speech encoding accuracy, modelled on the basis of the spectrotemporal response function, was significantly enhanced for attended versus ignored intact speech at earlier neural response latencies (<∼250 ms). This attentional enhancement was not absent but delayed for vocoded speech. These findings suggest that attentional selection of unilateral, degraded speech is feasible but induces delayed neural separation of competing speech, which might explain listening challenges experienced by unilateral CI users.

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Lintong, Fransiska. "GANGGUAN PENDENGARAN AKIBAT BISING." JURNAL BIOMEDIK (JBM) 1, no. 2 (February 6, 2013). http://dx.doi.org/10.35790/jbm.1.2.2009.815.

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Abstract: Noise induced hearing loss is often found among industrial workers all over the world, especially in developing countries, such as Indonesia. The permitted maximum limit of noise for a human being is 80 dB. Noise with a high intensity that occurs for a long time can cause some changes in metabolic processes and the blood supply in the organ of Corti. The impacts of these changes are damage and degeneration of hair cells, and in the long run, the total destruction of this organ and permanent hearing loss. Effects of noise to this sense organ are in the forms of acoustic trauma, noise-induced temporary threshold shift, and noise-induced permanent threshold shift. Noise induced hearing loss is a senso-neural deafness, and is generally bilateral. Key words: noise, organ of Corti, permanent loss, senso-neural deafness Abstrak: Gangguan pendengaran akibat bising sering dijumpai pada pekerja industri di seluruh dunia, terlebih lagi di negara berkembang seperti Indonesia. Ambang batas maksimum aman dari bising bagi manusia adalah 80 dB. Bising dengan intensitas tinggi yang berlang-sung dalam waktu lama akan menyebabkan perubahan metabolisme dan vaskuler. Sebagai akibat terjadi robekan sel-sel rambut organ Corti dan kerusakan degeneratif sel-sel tersebut, yang kemudian berlanjut dengan destruksi total dari organ tersebut dan kehilangan pen-dengaran yang permanen. Efek bising terhadap pendengaran dapat berupa trauma akustik, perubahan ambang pendengaran akibat bising yang berlangsung sementara, dan perubahan ambang pendengaran akibat bising yang berlangsung permanen. Gangguan pendengaran yang terjadi akibat bising adalah berupa tuli senso-neural yang biasanya bilateral. Katakunci: Kebisingan, organ corti, permanen, tuli senso-neural

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Zhang, Andi, Tianyuan Zou, Dongye Guo, Quan Wang, Yilin Shen, Haixia Hu, Bin Ye, and Mingliang Xiang. "The Immune System Can Hear Noise." Frontiers in Immunology 11 (February 18, 2021). http://dx.doi.org/10.3389/fimmu.2020.619189.

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As a stressor widely existing in daily life, noise can cause great alterations to the immune system and result in many physical and mental disorders, including noise-induced deafness, sleep disorders, cardiovascular diseases, endocrine diseases and other problems. The immune system plays a major role in maintaining homeostasis by recognizing and removing harmful substances in the body. Many studies have shown that noise may play vital roles in the occurrence and development of some immune diseases. In humans, both innate immunity and specific immunity can be influenced by noise, and different exposure durations and intensities of noise may exert various effects on the immune system. Short-term or low-intensity noise can enhance immune function, while long-term or high-intensity noise suppresses it. Noise can lead to the occurrence of noise-induced hearing loss (NIHL) through the production of autoantibodies such as anti-Hsp70 and anti-Hsp60 and exert adverse effects related to other immune-related diseases such as some autoimmune diseases and non-Hodgkin lymphoma. The neuroendocrine system, mainly including the hypothalamic-pituitary-adrenal (HPA) axis and the sympathetic-adrenal-medullary (SAM) system, is involved in the mechanisms of immune-related diseases induced by noise and gut microbiota dysfunction. In addition, noise exposure during pregnancy may be harmful to the immune system of the fetus. On the other hand, some studies have shown that music can improve immune function and alleviate the adverse effects caused by noise.

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Vaneva, Marjana. "Cognitive Mapping of the Noun Domains When Zero-deriving Proper from Common Nouns in Macedonian." International Journal of Multidisciplinary and Current Research 6, no. 01 (January 30, 2018). http://dx.doi.org/10.14741/ijmcr.v6i01.10901.

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Noise induced hearing loss NIHL is irreversible sensory neural deafness in one or both ears which develops gradually due to chronic exposure to injurious noise. In this study there were 92 workers chosen under exclusion and inclusion criteria, most of the samples study were male due to hard handling working, 40% of the samples studied were complaining of SNHL (sensory neural hearing loss) are variable from mild to sever one. Although they have normal otoscopic finding, the longer the duration of noise exposure the more damaging effect with SNHL result. There are no preventive measures used in our sample study. NIHL is preventable disease which needs the role of social media in education of peoples.

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Naser, Hussein A., Jaafar M. Khalaf, and Duried A. Mohammed. "Occupational related hearing impairment in Basrah." International Journal of Multidisciplinary and Current Research 6, no. 01 (January 30, 2018). http://dx.doi.org/10.14741/ijmcr.v6i01.10902.

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Noise induced hearing loss NIHL is irreversible sensory neural deafness in one or both ears which develops gradually due to chronic exposure to injurious noise. In this study there were 92 workers chosen under exclusion and inclusion criteria, most of the samples study were male due to hard handling working, 40% of the samples studied were complaining of SNHL (sensory neural hearing loss) are variable from mild to sever one. Although they have normal otoscopic finding, the longer the duration of noise exposure the more damaging effect with SNHL result. There are no preventive measures used in our sample study. NIHL is preventable disease which needs the role of social media in education of peoples.

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Kahan, Elise, and Eleanor Ross. "Knowledge and attitudes of a group of South African mine workers towards noise induced hearing loss and the use of hearing protective devices." South African Journal of Communication Disorders 41, no. 1 (December 31, 1994). http://dx.doi.org/10.4102/sajcd.v41i1.255.

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The study aimed to explore the knowledge and attitudes of a group of mine workers regarding noise induced hearing loss and the use of hearing protective devices. These aims were investigated via a questionnaire administered in a group setting to 55 underground mine workers. The main finding that emerged from the study was that respondents were poorly informed regarding the fact that noise was a health hazard. Furthermore, the knowledge that respondents did possess, appeared to have been derived from the personal experience of working in noisy environments for many years, rather than from educational input. Contrary to expectations, respondents did not view deafness as a status symbol but rather as a negative attribute. Consequently, they were motivated to protect themselves from hearing impairment and to be educated about hearing and the effects of noise. The mine workers complained about discomfort when wearing hearing protective devices as well as feelings of insecurity due to inhibition of communication and the inability to hear warning signals. For these reasons, noise protection was mainly worn in situations perceived as noisy by workers themselves. These results are discussed in terms of their implications for the clinical practice of audiology; hearing conservation in the mining industry; further research; and the training and education of speech-language pathologists and audiologists.

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Sasongko, Sigit, Awan Buana, Dara Fuji Rahayu, and Wildan Kurniawan. "Hearing Function among Squadron 11/Attack Helicopter Pilots in 2019–2020." Asian Journal of Applied Sciences 9, no. 3 (July 1, 2021). http://dx.doi.org/10.24203/ajas.v9i3.6657.

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Noise-Induced Hearing Loss (NIHL) is sensorineural deafness resulting from prolonged exposure to loud noise. In the military environment, personnel with NIHL are often found. One of the professions that are at risk for NIHL is an aviator. Some of the factors that influence the degree of deafness are age and length of work. This research is a descriptive quantitative observational study with a cross-sectional design. The research subjects were Squadron 11/Attack helicopter pilots at Achmad Yani Air Base, Semarang, totaling 32 pilots, which were taken from medical record data. Sampling was done by total sampling. The data obtained were processed using SPSS and grouped into tables accompanied by descriptive explanations of each characteristic. The audiogram results showed that 32 pilots were normal, across all age and length of service categories. This result is due to the appropriate use of hearing protection device (HPD), in the form of a helmet that reduces noise up to 14 dB at 250 Hz, 21 dB at 1000 Hz, 26 dB at 2000 Hz, 37 dB at 4000 Hz, and 42 dB at a frequency of 8000 Hz, which pilots use. The pilot's working time is relatively short with a flight training schedule only 2 times a week and a flight time of around 2-3 hours. The conclusions of the study showed a description of normal hearing function in all Squadron 11/Attack helicopter pilots, based on age and length of service.

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Gu, Xi, Daqi Wang, Zhijiao Xu, Jinghan Wang, Luo Guo, Renjie Chai, Genglin Li, Yilai Shu, and Huawei Li. "Prevention of acquired sensorineural hearing loss in mice by in vivo Htra2 gene editing." Genome Biology 22, no. 1 (March 22, 2021). http://dx.doi.org/10.1186/s13059-021-02311-4.

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Abstract Background Aging, noise, infection, and ototoxic drugs are the major causes of human acquired sensorineural hearing loss, but treatment options are limited. CRISPR/Cas9 technology has tremendous potential to become a new therapeutic modality for acquired non-inherited sensorineural hearing loss. Here, we develop CRISPR/Cas9 strategies to prevent aminoglycoside-induced deafness, a common type of acquired non-inherited sensorineural hearing loss, via disrupting the Htra2 gene in the inner ear which is involved in apoptosis but has not been investigated in cochlear hair cell protection. Results The results indicate that adeno-associated virus (AAV)-mediated delivery of CRISPR/SpCas9 system ameliorates neomycin-induced apoptosis, promotes hair cell survival, and significantly improves hearing function in neomycin-treated mice. The protective effect of the AAV–CRISPR/Cas9 system in vivo is sustained up to 8 weeks after neomycin exposure. For more efficient delivery of the whole CRISPR/Cas9 system, we also explore the AAV–CRISPR/SaCas9 system to prevent neomycin-induced deafness. The in vivo editing efficiency of the SaCas9 system is 1.73% on average. We observed significant improvement in auditory brainstem response thresholds in the injected ears compared with the non-injected ears. At 4 weeks after neomycin exposure, the protective effect of the AAV–CRISPR/SaCas9 system is still obvious, with the improvement in auditory brainstem response threshold up to 50 dB at 8 kHz. Conclusions These findings demonstrate the safe and effective prevention of aminoglycoside-induced deafness via Htra2 gene editing and support further development of the CRISPR/Cas9 technology in the treatment of non-inherited hearing loss as well as other non-inherited diseases.

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Affortit, Corentin, François Casas, Sabine Ladrech, Jean-Charles Ceccato, Jérôme Bourien, Carolanne Coyat, Jean-Luc Puel, Marc Lenoir, and Jing Wang. "Exacerbated age-related hearing loss in mice lacking the p43 mitochondrial T3 receptor." BMC Biology 19, no. 1 (February 1, 2021). http://dx.doi.org/10.1186/s12915-021-00953-1.

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Abstract Background Age-related hearing loss (ARHL), also known as presbycusis, is the most common sensory impairment seen in elderly people. However, the cochlear aging process does not affect people uniformly, suggesting that both genetic and environmental (e.g., noise, ototoxic drugs) factors and their interaction may influence the onset and severity of ARHL. Considering the potential links between thyroid hormone, mitochondrial activity, and hearing, here, we probed the role of p43, a N-terminally truncated and ligand-binding form of the nuclear receptor TRα1, in hearing function and in the maintenance of hearing during aging in p43−/− mice through complementary approaches, including in vivo electrophysiological recording, ultrastructural assessments, biochemistry, and molecular biology. Results We found that the p43−/− mice exhibit no obvious hearing loss in juvenile stages, but that these mice developed a premature, and more severe, ARHL resulting from the loss of cochlear sensory outer and inner hair cells and degeneration of spiral ganglion neurons. Exacerbated ARHL in p43−/− mice was associated with the early occurrence of a drastic fall of SIRT1 expression, together with an imbalance between pro-apoptotic Bax, p53 expression, and anti-apoptotic Bcl2 expression, as well as an increase in mitochondrial dysfunction, oxidative stress, and inflammatory process. Finally, p43−/− mice were also more vulnerable to noise-induced hearing loss. Conclusions These results demonstrate for the first time a requirement for p43 in the maintenance of hearing during aging and highlight the need to probe the potential link between human THRA gene polymorphisms and/or mutations and accelerated age-related deafness or some adult-onset syndromic deafness.

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Holmgren, Melanie, and Lavinia Sheets. "Using the Zebrafish Lateral Line to Understand the Roles of Mitochondria in Sensorineural Hearing Loss." Frontiers in Cell and Developmental Biology 8 (February 5, 2021). http://dx.doi.org/10.3389/fcell.2020.628712.

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Hair cells are the mechanosensory receptors of the inner ear and can be damaged by noise, aging, and ototoxic drugs. This damage often results in permanent sensorineural hearing loss. Hair cells have high energy demands and rely on mitochondria to produce ATP as well as contribute to intracellular calcium homeostasis. In addition to generating ATP, mitochondria produce reactive oxygen species, which can lead to oxidative stress, and regulate cell death pathways. Zebrafish lateral-line hair cells are structurally and functionally analogous to cochlear hair cells but are optically and pharmacologically accessible within an intact specimen, making the zebrafish a good model in which to study hair-cell mitochondrial activity. Moreover, the ease of genetic manipulation of zebrafish embryos allows for the study of mutations implicated in human deafness, as well as the generation of transgenic models to visualize mitochondrial calcium transients and mitochondrial activity in live organisms. Studies of the zebrafish lateral line have shown that variations in mitochondrial activity can predict hair-cell susceptibility to damage by aminoglycosides or noise exposure. In addition, antioxidants have been shown to protect against noise trauma and ototoxic drug–induced hair-cell death. In this review, we discuss the tools and findings of recent investigations into zebrafish hair-cell mitochondria and their involvement in cellular processes, both under homeostatic conditions and in response to noise or ototoxic drugs. The zebrafish lateral line is a valuable model in which to study the roles of mitochondria in hair-cell pathologies and to develop therapeutic strategies to prevent sensorineural hearing loss in humans.

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Tjan, Hardini, Fransiska Lintong, and Wenny Supit. "EFEK BISING MESIN ELEKTRONIKA TERHADAP GANGGUAN FUNGSI PENDENGARAN PADA PEKERJA DI KECAMATAN SARIO KOTA MANADO, SULAWESI UTARA." Jurnal e-Biomedik 1, no. 1 (March 13, 2013). http://dx.doi.org/10.35790/ebm.1.1.2013.1158.

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Abstract: Noise induced hearing loss is caused by noise loud in the long period and a noisy work environment. Noisy work environment is a major problem in occupational health in various countries. The relationship between excessive noise exposure and hearing loss has been recognised since ancient times. Early epidemiological studies of noise induced hearing loss explored the damage risk relationship between occupational noise exposure level and the degree of hearing loss. The purpose of this study is to determine effect of engine noise electronics to auditory disfunction. The research methodeology used is an analytical method with a cross sectional approach. Samples were of 20 person taken from workers at the playground timezone and amazone. Data were obtained through questionnaires and examination of hearing function with the audiometri. Data were analyzed by using the Statistical Product and Service Solutions program (SPSS) and using the Fisher Exact test. Conclusion: The results showed that : There is a 75% hearing loss in all worker. The results of bivariate analysis showed there is no significant association between the hearing loss with the intensity level of noise (p = 0,032). The most common hearing loss is sensorineural deafness which generally occours in both ear. From the result of this study it can be concluded that the workers who work in a place that has the high intensity noise have greater risk of suffening from hearing loss. Keywords: Timezone and Amazone Workers, Noisy, Hearing. Abstrak: Gangguan pendengaran akibat bising ialah gangguan pendengaran yang disebabkan akibat terpajan oleh bising yang cukup keras dalam jangka waktu yang cukup lama dan biasanya disebabkan oleh bising di lingkungan kerja. Bising lingkungan kerja merupakan masalah utama pada kesehatan kerja di berbagai negara. Hubungan antara paparan bising yang berlebihan dan kehilangan pendengaran telah dikenal sejak zaman kuno. Awal studi epidemiologi, gangguan pendengaran yang disebabkan oleh bising mengeksplorasi adanya hubungan atau faktor resiko antara pekerjaan, paparan tingkat kebisingan dan derajat gangguan pendengaran. Tujuan penelitian ini untuk mengetahui efek bising mesin elektronika terhadap gangguan fungsi pendengaran. Metode penelitian yang digunakan yaitu metode analitik dengan menggunakan rancangan cross sectional study. Sampel berjumlah 20 orang yang diambil dari pekerja di tempat bermain timezone dan amazone. Data diperoleh melalui kuisioner dan pemeriksaan fungsi pendengaran dengan menggunakan Audiometri. Data dianalisis dengan menggunakan Statistical Program Product and Service Solution (SPSS) dan menggunakan uji Fisher Exact. Simpulan: Hasil penelitian menunjukkan bahwa : Terdapat gangguan pendengaran sebesar 75 % pada seluruh pekerja. Hasil analisis bivariat menunjukan ada hubungan yang bermakna antara gangguan pendengaran dengan tingkat intensitas bising (p = 0,032). Gangguan pendengaran yang paling banyak diderita oleh pekerja adalah tuli sensorineural (persepsi) yang umumnya terjadi pada kedua telinga. Dari hasil penelitian ini dapat disimpulkan bahwa pekerja yang bekerja pada intensitas bising yang tinggi memiliki resiko lebih besar menderita gangguan pendengaran. Kata Kunci: Pekerja Timezone & Amazone, Bising, Pendengaran

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Manoppo, Fauziah N., Wenny Supit, and Vennetia R. Danes. "HUBUNGAN ANTARA KEBISINGAN DAN FUNGSI PENDENGARAN PADA PETUGAS PT. GAPURA ANGKASA DI BANDAR UDARA SAM RATULANGI MANADO." Jurnal e-Biomedik 2, no. 1 (February 13, 2014). http://dx.doi.org/10.35790/ebm.2.1.2014.3620.

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Abstract: The noise resulted from the advancement of transportation is an issue that cannot be avoided. The Noise Induced Hearing Loss is a decreases of auditory type sensorineural, which is initially not be aware, because there has not disturbed of daily conversation yet. Risk factors that affect the degree of severity of deafness are the intensity of the noise, frequency, long exposure per day, long working period, individual sensitivity, age and other factors that could influence. The aim of this research is to find out whether there is a relationship between noises by function of hearing at the officers of PT. Gapura Angkasa at Sam Ratulangi Airport, Manado. The Method of this research namely the analytic method is a design cross sectional study, and the samples were 20. The Measurement of noise levels is based on a unit of work the Operation which was obtained at the level of 75 dB and 85 dB Loading. From the results obtained from 20 staffs there are only 8 people who work exceed the Threshold Value (NAB) of noise which has been set. Conclusion: The result showed that there is hearing loss about 20 % in all the officers. The result Analysis Bivariat indicates that there is the absence of meaningful relations between noise-induced hearing disorder with the level of intensity noisy (p = 0.591). From the results of this research it can be concluded that the officers who work in on high intensity noise are more at risk of experiencing hearing loss compared to the officers who work on a low intensity noise level. In addition, the factors such as long hours of work, long exposure noise, the use of Ear Protectors Tools (EPT) was also very influential of the onset hearing loss in workers. Keywords: Noise Induced Hearing Loss, Sam Ratulangi Airport, The Officers of PT. Gapura Angkasa. Abstrak: Kebisingan merupakan suatu masalah yang tidak dapat dihindari akibat kemajuan sarana transportasi. Gangguan pendengaran akibat bising adalah penurunan pendengaran tipe sensorineural, yang pada awalnya tidak disadari, karena belum mengganggu percakapan sehari-hari. Faktor risiko yang berpengaruh pada derajat parahnya ketulian ialah intensitas bising, frekwensi, lama pajanan perhari, lama masa kerja, kepekaan individu, umur dan faktor lain yang dapat berpengaruh. Tujuan penelitian ini untuk mengetahui apakah terdapat hubungan antara kebisingan dengan fungsi pendengaran pada petugas PT.Gapura Angkasa di Bandara Sam Ratulangi. Metode penelitian yang digunakan yaitu metode analitik dengan menggunakan rancangan studi cross sectional. Sampel berjumlah 20 orang. Pengukuran tingkat bising di lakukan berdasarkan unit kerja yaitu pada bagian Operation didapatkan 75 dB dan bagian Loading 85 dB. Dari hasil yang didapatkan dari 20 orang petugas hanya 8 orang saja yang bekerja melebihi NAB kebisingan yang telah ditetapkan. Simpulan : Hasil penelitian menunjukkan bahwa terdapat gangguan pendengaran sebesar 20% pada seluruh petugas. Hasil analisis bivariat menunjukkan tidak adanya hubungan yang bermakna antara gangguan pendengaran dengan tingkat intesitas bising (p=0.591). Dari hasil penelitian ini dapat disimpulkan bahwa petugas yang bekerja pada intensitas bising yang tinggi lebih beresiko mengalami gangguan pendengaran dibandingkan dengan petugas yang bekerja pada tingkat intesitas bising yang rendah. Selain itu faktor-faktor seperti lama bekerja, lama pemaparan bising, penggunaan Alat Pelindung Telinga (APT) juga sangat berpengaruh terhadap terjadinya gangguan pendengaran pada petugas. Keywords: Gangguan pendengaran akibat bising , Bandara Sam Ratulangi, Petugas PT. Gapura Angkasa.

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Abrisham, Seyed Mohammad, Mohammad Shafiee, and Mohsen Abediny Sanich. "Evaluation of Hearing Status in Employees of Dental Prosthodontics Laboratories in Yazd." Journal of Tolooebehdasht, September 14, 2020. http://dx.doi.org/10.18502/tbj.v19i3.4172.

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Introduction: Noise Induced Hearing Loss (NIHL) is the first cause of acquired hearing loss. Dentists and dental prosthodontics technicians are exposed to different sounds produced by a variety of devices in their occupations. In this study, we studied the effect of dental prosthesis sounds on the auditory power of the respective staff. Methods: This case-control study was carried out among all technicians of dental prosthesis laboratories in Yazd in 2018 selected by census sampling method. A total of 23 technicians who were willing to cooperate and met the inclusion criteria were included in the experimental group. Furthermore, 23 people who referred to the audiology clinic, were matched with the experimental group in terms of age and gender, and were not working in noisy environment, and had no congenital or acquired deafness were examined as the control group. Finally, the collected data were analyzed by SPSS version 19 using statistical tests. Result: The mean hearing threshold of the right ear at frequencies of 3000 and 4000 Hz and the mean hearing threshold of the left ear at frequencies of 3000, 4000, 6000, and 8000 Hz was significantly higher in the experimental than the control group. The mean hearing threshold of the right ear at the frequency of 4000 Hz and the hearing threshold of the left ear at frequencies of 3000, 4000, and 6000 Hz in participants with job history>4 years was significantly higher. Furthermore, the mean hearing threshold of the right ear in the frequency of 4000 Hz and the hearing threshold of the left ear at frequencies of 3000, 4000, and 6000 Hz years was significantly higher in people with job history >30 years. Conclusion: According to findings, the staffs working in dental prosthodontics laboratories suffer from NIHL at frequencies of 3000 to 8000 Hz. The hearing loss in the left ear was more severe in people with job history of >30 years and in people with job history of > 4 years.Therefore, the use of hearing impaired and hearing protectors is recommended in these people.

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Journal articles on the topic 'Deafness, Noise induced – Namibia'

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