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Journal articles on the topic 'Death of a child, miscarriage, SIDS'

Author: Grafiati

Published: 24 April 2022

Last updated: 30 July 2025

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1

Saanvi, Sharma. "THE EFFECTS OF ALCOHOL AND DRUG CONSUMPTION ON A FETUS." INTERNATIONAL EDUCATIONAL JOURNAL OF SCIENCE AND ENGINEERING - IEJSE 7, no. 3 (2024): 11–13. https://doi.org/10.5281/zenodo.15607443.

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Substance abuse during pregnancy continues to become a rising threat as per increasing statistics and requires global attention to the matter for it to be solved for future proficiency. Alcohol and drug exposure to a fetus within the gestation period, especially the first trimester of pregnancy, can cause severe birth defects and an increased risk of sudden infant death syndrome (SIDS), stillbirth, as well as miscarriage. Through extensive secondary research, the major issues can be seen through abnormalities in the brain structure and components of a child with prenatal alcohol or drug exposu

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2

SHARIPOV, Isroil Latipovich, Shukur Kuylievich PARDAEV, and Bakhodir Kuchkarovich KHOLBEKOV. "SUDDEN INFANT DEATH SYNDROME (literature review)." Journal of biomedicine and practice 7, no. 5 (2022): 5. https://doi.org/10.5281/zenodo.7392900.

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Sudden Infant Death Syndrome (SIDS)] is a sudden death from respiratory arrest in an apparently healthy infant or child under 1 year old, in which an autopsy does not determine the cause of death. Sometimes SIDS is called "cradle death" because it may not be preceded by any symptoms, often the child dies in his sleep. Risk factors in SIDS are divided into 2 main groups: modifiable and non-modifiable. The main hypotheses of thanatogenesis in SIDS are congenital defects and metabolism, immunological incompetence, serotonergic dysfunction, and cardiogenic mechanisms.

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3

Brooks, John G., Ruth E. Gilbert, Peter J. Flemming, Peter J. Berry, and Jean Golding. "Postnatal Growth Preceding Sudden Infant Death Syndrome." Pediatrics 94, no. 4 (1994): 456–61. http://dx.doi.org/10.1542/peds.94.4.456.

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Objective. To compare postnatal growth preceding the sudden infant death syndrome (SIDS) with that of age matched controls. Design. Retrospective case-control study. Each SIDS victim was matched with two controls on date of parental interview, postnatal age, and neighborhood. Clinical and demographic data were collected by parental interview and by review of medical records, and interval body weights were obtained from health visitors' records. Study population. All infants dying of SIDS between 1 May, 1987 and 30 April, 1989 in a geographically defined region consisting of four health distric

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4

BASS, MILLARD, and RITA HASS. "SIDS and Homicide." Pediatrics 92, no. 2 (1993): 302–3. http://dx.doi.org/10.1542/peds.92.2.302.

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To the Editor.— Dr Reece's discussion on differentiating between unexpected death due to the sudden infant death syndrome (SIDS) and one due to homicide1 focuses on the medical literature regarding this topic. May we comment on this diagnostic dilemma based on our experience in pediatric forensic pathology? It is certainly true that the recently expanded definition of SIDS now includes a negative death-scene investigation as an integrated part of this diagnosis.2 However, from a historical perspective, there has been a fundamental misunderstanding about the term "death-scene investigation" in

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REESE, ROBERT M. "SIDS and Homicide." Pediatrics 92, no. 2 (1993): 303. http://dx.doi.org/10.1542/peds.92.2.303.

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In Reply.— Drs Bass and Hass cite the progress being made in the training of scene-investigators and this is desirable. No one would disagree that there is a great need for improved, standardized death-scene investigation by competent and objective personnel in all cases of sudden unexpected death in children. The additional need, as pointed out in the article,1 is for Child Death Review Teams in all jurisdictions,2-5 providing interdisciplinary in-depth investigation of all aspects of the unexpected death.

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6

Ahmed, Sadia, Ian Mitchell, and Gregor Wolbring. "Analysis of sudden infant death syndrome coverage in Canadian newspapers." Journal of Child Health Care 22, no. 4 (2018): 545–62. http://dx.doi.org/10.1177/1367493518763983.

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Sudden infant death syndrome (SIDS; also known as crib death) describes the sudden unexpected death of an infant under one year of age, which remains unexplained after a thorough investigation. SIDS is a public health concern. It is the fourth leading cause of infant death in Canada. Newspapers are a major source of health information for the public, shape public perceptions and can direct the discussion around issues. Despite the potential influence of newspapers, no study has examined the portrayal of SIDS in Canadian newspapers over time. The purpose of our study was to gain an understandin

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7

Carroll, John L., and Gerald M. Loughlin. "Sudden Infant Death Syndrome." Pediatrics In Review 14, no. 3 (1993): 83–93. http://dx.doi.org/10.1542/pir.14.3.83.

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The causes of SIDS are unknown, and there are no tests that predict which infants will die of SIDS. Home monitoring, while it may prevent death in some high-risk infant populations, has not altered the overall incidence of SIDS. Studies have identified several important risk factors, such as prematurity, low birth-weight, inadequate prenatal care, maternal smoking, maternal anemia, and poor socioeconomic conditions. Other recent studies have raised important questions about child care practices, such as infant sleeping position. Still others raise equally important questions about the role of

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8

Sinisa, Franjic. "Unexpected Baby Death - SIDS or Not?" Instant Journal of Forensic Science 3, no. 1 (2021): 17–21. https://doi.org/10.36811/ijfs.2021.110015.

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A two-month-old baby girl died on April 4, 2021, at the Children's Home in Osijek, Croatia. The child was temporarily taken away of her parents because she was born prematurely, she was diagnosed with a risky health condition and it was assessed that the parents were unable to meet the child’s needs. Parents are beneficiaries of social welfare. The mother suffers from diabetes and epilepsy. An autopsy determined that the child had died of pneumonia. The parents last visited the child the day before her death and she was cheerful, happy and in a good mood, but she was coughing, which

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9

Haas, Joel E., James A. Taylor, Abraham B. Bergman, et al. "Relationship Between Epidemiologic Risk Factors and Clinicopathologic Findings in the Sudden Infant Death Syndrome." Pediatrics 91, no. 1 (1993): 106–12. http://dx.doi.org/10.1542/peds.91.1.106.

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The risk of sudden infant death syndrome (SIDS) is said to be enhanced by factors such as prematurity, low birth weight, and perinatal distress. The significance of risk factors for SIDS research was questioned because the majority of SIDS victims seem to lack them. Therefore, postmortem records of 1144 infants who died suddenly and unexpectedly in King County, Washington, over a 25-year period were studied. Deaths were classified as "explained" if a cause was apparent, "classic" SIDS if the history and autopsy were unrevealing or, where the diagnosis of SIDS was doubtful, as "probable" or "po

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Mitchell, E. A., R. P. K. Ford, A. W. Stewart, et al. "Smoking and the Sudden Infant Death Syndrome." Pediatrics 91, no. 5 (1993): 893–96. http://dx.doi.org/10.1542/peds.91.5.893.

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Objective. Maternal smoking has been shown to be a risk factor for sudden infant death syndrome (SIDS). The effect of smoking by the father and other household members has not previously been examined. Methods. A large nationwide case-control study. Four hundred eighty-five SIDS deaths in the postneonatal age group were compared with 1800 control infants. Results. Infants of mothers who smoked during pregnancy had a 4.09 (95% confidence interval [CI] = 3.28, 5.11) greater risk of death than infants of mothers who did not smoke. Infants of mothers who smoked postnatally also had an increased ri

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Hunt, Carl E. "Sudden Infant Death Syndrome and Subsequent Siblings." Pediatrics 95, no. 3 (1995): 430–32. http://dx.doi.org/10.1542/peds.95.3.430.

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Siblings of a prior SIDS victim are at increased risk for SIDS. Documented monitoring is cost-effective in siblings of prior SIDS infants,4 but cost has not yet been studied in any other infant groups. Although the clinical effectiveness of home infant monitoring has not yet been established for any infant group, documented monitoring is a technological advance that will play a critical role in determining whether home monitors can prevent infants from dying of SIDS. The importance of the Steinschneider study4 is therefore related not only to its role in clarifying the cost-effectiveness of do

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Ahn, Young Mee, Kyung-moo Yang, Hong Il Ha, and Jung Ae Cho. "Risk Factors for Sudden Infant Death Syndrome and Sleeping Practices in Korea." Child Health Nursing Research 26, no. 1 (2020): 82–89. http://dx.doi.org/10.4094/chnr.2020.26.1.82.

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Sudden infant death syndrome (SIDS) in Korea remains a poorly-understood subject for both professionals and the public. Recent reports have emphasized ethnic differences in SIDS rates, suggesting that making adjustments in child-rearing practices may contribute substantially to SIDS reduction. Two of the three major risk factors for SIDS-vulnerability of the infant and exogenous factors-need to be understood in particular depth due to their broad scope and sociocultural grounding. This paper presents substantial issues regarding preterm birth and male gender on infants’ vulnerability to SIDS i

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IOV, Tatiana, Cristina FURNICĂ, Sofia Mihaela DAVID, and Diana BULGARU-ILIESCU. "Medical, Forensic and Social Quandaries of Sudden Infant Death Syndrome Today." BRAIN. Broad Research in Artificial Intelligence and Neuroscience 11, no. 3 Sup.1 (2020): 20–30. https://doi.org/10.18662/brain/11.3Sup1/118.

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 Sudden Infant Death Syndrome (SIDS) is described as thesudden, unexplained death (with no attributable cause, during sleep) of aseemingly healthy child before reaching the first year of life. Statistically,SIDS is recognized today as a leading cause of death in infants aged 1 to12 months. In the present article the authors have analyzed known riskfactors, classifications and current standards of forensic investigation whilehighlighting the necessity of detailed clinical history, autopsy, scene ofdeath examination and lab findings (radiology, metabolic anomalies,infectious diseases and to

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Boles, Richard G., Carl Boesel, and Piero Rinaldo. "Sudden Death Beyond Sids." Pediatric Pathology & Laboratory Medicine 16, no. 4 (1996): 691–93. http://dx.doi.org/10.1080/15513819609168705.

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Iov, Tatiana, Cristina Furnică, Sofia Mihaela David, and Diana Bulgaru-Iliescu. "Medical, Forensic and Social Quandaries of Sudden Infant Death Syndrome Today." BRAIN. Broad Research in Artificial Intelligence and Neuroscience 11, no. 3sup1 (2020): 20–30. http://dx.doi.org/10.18662/brain/11.3sup1/118.

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Sudden Infant Death Syndrome (SIDS) is described as the sudden, unexplained death (with no attributable cause, during sleep) of a seemingly healthy child before reaching the first year of life. Statistically, SIDS is recognized today as a leading cause of death in infants aged 1 to 12 months. In the present article the authors have analyzed known risk factors, classifications and current standards of forensic investigation while highlighting the necessity of detailed clinical history, autopsy, scene of death examination and lab findings (radiology, metabolic anomalies, infectious diseases and

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CUMMINS, SUSAN K. "Statistics and Sudden Infant Death Syndrome." Pediatrics 79, no. 3 (1987): 486–87. http://dx.doi.org/10.1542/peds.79.3.486a.

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To the Editor.— The recent cohort study by Oren et al1 examined risk factors for sudden infant death syndrome (SIDS) in infants with prolonged unexplained sleep apnea with color change (severe apnea of infancy). This analysis revealed three possible risk factors for SIDS death: having a subsequent monitored apnea episode needing resuscitation or vigorous stimulation, being a sibling of a SIDS victim, or developing a seizure disorder after monitoring began. Unfortunately, there are two serious problems with the authors' analysis: the misrepresentation of relative risk and the use of the χ2 stat

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Helweg-Larsen, Karin, Lisbeth B. Knudsen, Markil Gregersen, and Jørn Simonsen. "Sudden Infant Death Syndrome (SIDS) in Denmark: Evaluation of the Increasing Incidence of Registered SIDS in the Period 1972 to 1983 and Results of a Prospective Study in 1987 through 1988." Pediatrics 89, no. 5 (1992): 855–59. http://dx.doi.org/10.1542/peds.89.5.855.

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To investigate a reported increase, from 0.4 to 1.3 per thousand live births, in the Danish incidence of sudden infant death syndrome (SIDS), a retrospective analysis of SIDS in Denmark from 1972 to 1983 was carried out. Based on data registered with the National Board of Health, a notable regional difference in SIDS rate between the western and eastern parts of Denmark was found. This difference did not correlate with the overall postneonatal mortality by region. Danish law requires medicolegal investigation in all cases of sudden unexpected death. Medicolegal autopsies are performed only in

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18

Burns, Elizabeth A., J. Daniel House, and Mary R. Ankenbauer. "Sibling Grief in Reaction to Sudden Infant Death Syndrome." Pediatrics 78, no. 3 (1986): 485–87. http://dx.doi.org/10.1542/peds.78.3.485.

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Much of the literature that exists regarding psychologic outcomes of sudden infant death syndrome (SIDS) has focused on parental grief or family response; at least two studies suggest that a SIDS death also affected siblings. It is believed that children who experience the death of a sibling due to SIDS do grieve. Factors related to bereavement are the child's age at the time of the sibling's death, special circumstances of the SIDS death, and explanations and grieving response of the parents. However, no information currently exists that characterizes the course of the grief response of these

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Schechtman, Vicki L., Ronald M. Harper, Adrian J. Wilson, and David P. Southall. "Sleep Apnea in Infants Who Succumb to The Sudden Infant Death Syndrome." Pediatrics 87, no. 6 (1991): 841–46. http://dx.doi.org/10.1542/peds.87.6.841.

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Previous studies have shown the frequency of respiratory pauses to be altered in groups of infants at risk for the sudden infant death syndrome (SIDS). In this study, we assess the frequency of apneic pauses during quiet sleep and rapid eye movement sleep in control infants and infants who subsequently died of SIDS. Sleep states were identified in 12-hour physiological recordings of SIDS victims and matched control infants, and the number of respiratory pauses from 4 to 30 seconds in duration was computed for quiet sleep and rapid eye movement sleep. SIDS victims 40 to 65 days of age showed si

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20

Kahn, A., D. Blum, E. Rebuftat, et al. "Polysomnographic Studies of Infants Who Subsequently Died of Sudden Infant Death Syndrome." Pediatrics 82, no. 5 (1988): 721–27. http://dx.doi.org/10.1542/peds.82.5.721.

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The polygraphic findings from 11 future victims of sudden infant death syndrome (SIDS) are reported and compared with those of matched pairs of control infants. The recordings had been done to alleviate parental anxiety about sleep apnea. Four infants had siblings who were victims of SIDS. Two infants were studied 3.5 to 9.5 weeks before their deaths because of an unexplained apparent life-threatening event that had occurred during sleep. For each victim of SIDS, two control infants were selected from the 2,000 infants who had been tested in the same hospitals. They were matched for sex, gesta

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Corwin, Michael J., Barry M. Lester, Carol Sepkoski, Mark Peucker, Herbert Kayne, and Howard L. Golub. "Newborn Acoustic Cry Characteristics of Infants Subsequently Dying of Sudden Infant Death Syndrome." Pediatrics 96, no. 1 (1995): 73–77. http://dx.doi.org/10.1542/peds.96.1.73.

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Objective. To test the hypothesis that the occurrence of a neonatal cry exhibiting a high first formant is a risk factor for sudden infant death syndrome (SIDS) and to evaluate the association between SIDS and other acoustic cry variables. Method. We recorded cries and obtained medical and demographic data for 21 880 apparently healthy term newborns. Two cries were recorded between days 2 and 7 of life, after a painful stimulus at the time of routine blood drawing. Acoustic variables were measured with an automated computer-based analysis system. Twelve infants died of SIDS. Age at death range

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Boles, Richard, Carl Boesel, and Piero Rinaldo. "Sudden Death Beyond Sids." Fetal and Pediatric Pathology 16, no. 4 (1996): 691–93. http://dx.doi.org/10.3109/15513819609168705.

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23

Schechtman, Vicki L., Ronald M. Harper, Arian J. Wilson, and David P. Southall. "Sleep State Organization in Normal Infants and Victims of the Sudden Infant Death Syndrome." Pediatrics 89, no. 5 (1992): 865–70. http://dx.doi.org/10.1542/peds.89.5.865.

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Infants at increased risk of the sudden infant death syndrome (SIDS) show abnormal patterning of sleep-waking states. It was hypothesized that infants who were to die of SIDS would show abnormalities of sleep state distribution prior to their deaths. Twenty-two 12-hour recordings were obtained from infants who subsequently died of SIDS, and sleep state patterns were compared in these records and 66 records of age-matched control infants. Each 1-minute epoch was classified as quiet sleep, rapid eye movement (REM) sleep, waking, indeterminate state, or artifact-contaminated. Victims of SIDS show

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Mage, David, E. Donner, Mechtild Vennemann, et al. "All sudden unexplained infant respiratory deaths may result from the same underlying mechanism." Scandinavian Journal of Forensic Science 18, no. 1 (2012): 1–9. http://dx.doi.org/10.2478/v10278-012-0001-6.

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All sudden unexplained infant respiratory deaths may result from the same underlying mechanism The Sudden Infant Death Syndrome (SIDS) was defined in 1969 by Beckwith as sudden death of an infant or young child, unexpected by medical history, remaining unexplained after thorough autopsy/death-scene investigation. Recently researchers have used the general terms Sudden Unexplained Death in Infancy (SUDI) and Sudden Unexpected Infant Death (SUID) as "umbrella-terms" covering unexplained deaths (SIDS); sudden deaths for which SIDS risk factors present but insufficient cause is found; and sudden d

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Cutz, Ernest, Donald G. Perrin, Richard Hackman, and Elinor N. Czegledy-Nagy. "Maternal Smoking and Pulmonary Neuroendocrine Cells in Sudden Infant Death Syndrome." Pediatrics 98, no. 4 (1996): 668–72. http://dx.doi.org/10.1542/peds.98.4.668.

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Background. Maternal smoking is a well-recognized risk factor for sudden infant death syndrome (SIDS), but the precise mechanism is unknown. We tested a hypothesis that maternal smoking affects pulmonary neuroendocrine cells (PNECs) and neuroepithelial bodies (NEBs), which are innervated PNEC clusters and presumed airway chemoreceptors. Methods. Lung sections from infants who died of SIDS and whose mothers smoked during pregnancy (n = 22), infants who died of SIDS and whose mothers were nonsmokers (n = 17), and age-matched control infants (n = 15) who died of other causes were immunostained fo

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Pharoah, Peter O. D., and Mary J. Platt. "Sudden Infant Death Syndrome in Twins and Singletons." Twin Research and Human Genetics 10, no. 4 (2007): 644–48. http://dx.doi.org/10.1375/twin.10.4.644.

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AbstractTwins compared with singletons and monozygous (MZ) compared with dizygous (DZ) twins are at increased risk of fetal and infant death, cerebral palsy and many congenital anomalies. The aim of this study is to investigate whether zygosity is a risk factor for the sudden infant death syndrome (SIDS). Birth registration data and draft infant death certificates for all multiple births in England and Wales 1993 to 2003 were provided by the Office for National Statistics. As a partial proxy for zygosity, same-sex was compared with opposite-sex twins for birthweight-specific mortality and mort

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Carroll, Ruth, and Sarah Shaefer. "Similarities and Differences in Spouses Coping with SIDS." OMEGA - Journal of Death and Dying 28, no. 4 (1994): 273–84. http://dx.doi.org/10.2190/6d89-bqju-mfxg-jywu.

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Bereaved parents' marital relationships may be at high risk for problems after a SIDS death because of coping differences between spouses. Five coping patterns developed in prior studies of SIDS parents were used to study differences within thirty-four pairs of parents bereaved by SIDS, three to forty months after their loss. Individual items most frequently used by both genders were examined to explore similarities in coping behaviors. Findings reveal bereaved parents sought support from within the family most frequently and from outside resources the least. Bereaved mothers used these coping

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Martinez, Fernando D. "Sudden Infant Death Syndrome and Small Airway Occlusion: Facts and a Hypothesis." Pediatrics 87, no. 2 (1991): 190–98. http://dx.doi.org/10.1542/peds.87.2.190.

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Respiratory failure is almost certainly the cause of death in the majority of cases of sudden infant death syndrome (SIDS), but the mechanisms leading to it have not been elucidated. SIDS shares many environmental and socioeconomic risk factors with severe forms of bronchiolitis, and the age distribution of incident cases is similar. Present knowledge of lung and airway development during infancy, determinants of peripheral airway patency, changes in lung surface activity in infants with SIDS, and fluid film dynamics in small airways are reviewed. It is hypothesized that many cases of SIDS may

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Beech, Darren J., Paul D. Sibbons, C. Vyvyan Howard, and Dick van Velzen. "Renal Developmental Delay Expressed by Reduced Glomerular Number and Its Association with Growth Retardation in Victims of Sudden Infant Death Syndrome and in “Normal” Infants." Pediatric and Developmental Pathology 3, no. 5 (2000): 450–54. http://dx.doi.org/10.1007/s100240010091.

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In victims of sudden infant death syndrome (SIDS), renal development has been reported to be significantly impaired. In the present study, we used stereological techniques to estimate volume of kidney cortex and total number of glomeruli in a group of human infants. Infants were classified according to cause of death—SIDS or non-SIDS. Cases were further subdivided according to birth weight—normal birth weight (NBW) or low birth weight (LBW) (we were unable to identify any non-SIDS LBW infants for our study). No significant differences were found between NBW and LBW infants (irrespective of cau

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Schoendorf, Kenneth C., and John L. Kiely. "Relationship of Sudden Infant Death Syndrome to Maternal Smoking During and After Pregnancy." Pediatrics 90, no. 6 (1992): 905–8. http://dx.doi.org/10.1542/peds.90.6.905.

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Sudden infant death syndrome (SIDS) is associated with maternal smoking during pregnancy. However, the relationship between tobacco exposure during infancy and SIDS is unknown. The examination of infants whose mothers smoked only after pregnancy will help determine the relationship between passive cigarette exposure during infancy and SIDS risk. This case-control analysis used data on normal birth weight (≥2500 g) infants included in the National Maternal and Infant Health Survey, a nationally representative sample of approximately 10 000 births and 6000 infant deaths. Infants were assigned to

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Vance, J. C., J. M. Najman, M. J. Thearle, G. Embelton, W. J. Foster, and F. M. Boyle. "Psychological Changes in Parents Eight Months After the Loss of an Infant From Stillbirth, Neonatal Death, or Sudden Infant Death Syndrome—A Longitudinal Study." Pediatrics 96, no. 5 (1995): 933–38. http://dx.doi.org/10.1542/peds.96.5.933.

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Objective. We proposed to measure part of the natural history of grief by determining the changes in the psychological symptoms experienced by bereaved parents over the 8 months after the loss of an infant from sudden infant death syndrome (SIDS), neonatal death (NND), or stillbirth (SB). Parents were interviewed twice, at 2 and 8 months after the loss. Methodology. A total of 220 bereaved families (45 SIDS, 93 NND, and 82 SB) were compared with 226 control families who had a live born child. Comparison was based on responses to a standardized measure of anxiety and depression (Delusions-Sympt

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BROOKS, JOHN G. "Are the King County SIDS Data Generalizable?" Pediatrics 92, no. 2 (1993): 304. http://dx.doi.org/10.1542/peds.92.2.304.

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To the Editor.— I wish to applaud Dr Haas and his colleagues for taking advantage of the probably unique, well-validated, longitudinal sudden infant death syndrome (SIDS) database in King County, WA, to try to clarify the interpretation and implications of the multiple reports of consistent associations between certain clinical and demographic characteristics and the occurrence of SIDS.1 I am concerned, however, that most of the differences which they report in prevalence of specific risk factors in each of their three subgroups of SIDS cases ("classic SIDS," "probable SIDS," "ossible SIDS"),

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Beeber, Bruce, and Nicholas Cunningham. "Fatal Child Abuse and Sudden Infant Death Syndrome (SIDS): A Critical Diagnostic Decision." Pediatrics 93, no. 3 (1994): 539–40. http://dx.doi.org/10.1542/peds.93.3.539b.

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We read with interest Dr Robert Reece's thorough article on "Fatal Child Abuse and Sudden Infant Death Syndrome: A Critical Diagnostic Decision," in the February issue of Pediatrics.1 Ever timely is this issue of the interplay between abuse and SIDS, as Reece and those at the Cleveland Child Protection Program have reported. A decade ago, we presented a paper entitled "SIDS—Child Abuse: Diagnostic Interface."2 The findings were based on both written surveys and follow-up telephone contacts.

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Knöbel, Harald H., Chien-Jen Chen, and Kung-Yee Liang. "Sudden Infant Death Syndrome in Relation to Weather and Optimetrically Measured Air Pollution in Taiwan." Pediatrics 96, no. 6 (1995): 1106–10. http://dx.doi.org/10.1542/peds.96.6.1106.

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Objective. To examine the possible role of weather and air pollution in sudden infant death syndrome (SIDS) and suffocation. Methods. Poisson regression analysis was carried out to measure the association between daily rates of SIDS per 1000 live births and daily average values of visibility and temperature in Taiwan between 1981 and 1991. The optimetrical measure of air pollution was used to represent pollution over a whole area rather than at a point source. Results. Mortality from SIDS per 1000 live births was 3.3 times greater in the lowest category of visibility on the day of death than i

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Rognum, TO. "SIDS or not SIDS? Classification problems of sudden infant death syndrome." Acta Paediatrica 85, no. 4 (1996): 401–3. http://dx.doi.org/10.1111/j.1651-2227.1996.tb14049.x.

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Özcanlı Çay, Özlem, and Özlem Kemer Aycan. "Sudden infant death syndrome." Journal of Controversies in Obstetrics & Gynecology and Pediatrics 1, no. 3 (2023): 74–77. http://dx.doi.org/10.51271/jcogp-0016.

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Sudden Infant Death Syndrome (SIDS) is a type of sudden and unexpected infant death, a term that encompasses both deaths from SIDS and ultimately all unexpected infant deaths with a determined cause. 1 Between %27 and % 43 of 3500 sudden unexpected infant death cases in the USA annually are due to SIDS. 2, 3 A number of other terms are used in pediatrics to describe sudden and unexpected deaths. Sudden unexpected death of an infant can be used interchangeably with sudden unexpected infant death, and sudden death in youth (VAS) refers to such death in any child 19 years of age or younger. Sudde

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SHANNON, DANIEL C., and CARL E. HUNT. "SIDS and Sleeping Position." Pediatrics 91, no. 2 (1993): 511–12. http://dx.doi.org/10.1542/peds.91.2.511.

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In Reply.— Guntheroth and Spiers,1 displeased with our commentary in Pediatrics,2 have raised many issues. However, the key question is whether the scientific data incriminating the prone position as a contributor to SIDS warrant a recommendation to change sleeping position at this time. Based on our critical review of the literature, we conclude that this recommendation was premature and unwarranted. Accepting the current definition of SIDS as the sudden death of an infant that remains unexplained even after a thorough postmortem examination, death scene investigation, and review of the clini

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Goldstein, Richard D., Peter S. Blair, Mary Ann Sens, et al. "Inconsistent classification of unexplained sudden deaths in infants and children hinders surveillance, prevention and research: recommendations from The 3rd International Congress on Sudden Infant and Child Death." Forensic Science, Medicine and Pathology 15, no. 4 (2019): 622–28. http://dx.doi.org/10.1007/s12024-019-00156-9.

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Abstract This report details the proceedings and conclusions from the 3rd International Congress on Unexplained Deaths in Infants and Children, held November 26–27, 2018 at the Radcliffe Institute at Harvard University. The Congress was motivated by the increasing rejection of the diagnosis Sudden Infant Death Syndrome (SIDS) in the medical examiner community, leading to falsely depressed reported SIDS rates and undermining the validity and reliability of the diagnosis, which remains a leading cause of infant and child mortality. We describe the diagnostic shift away from SIDS and the practica

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Bingham, Raymond J. "Findings from the National Institute of Nursing Research Related to Neonatal Care: 2008 Update." Neonatal Network 28, no. 1 (2009): e1-e4. http://dx.doi.org/10.1891/0730-0832.28.1.e1.

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A new program designed to help nurses teach parents, family members, and child care providers about risks and protective practices for Sudden Infant Death Syndrome (SIDS) is now available from the National Institutes of Health. The Continuing Education Program on Sudden Infant Death Syndrome (SIDS) Risk Reduction was developed by the Eunice Kennedy Shriver National Institute of Child Health and Human Development (NICHD) and the National Institute of Nursing Research (NINR), in collaboration with national nursing and infant health organizations. (Represented by Jeanette Xaichkin, RNC, MSN, The

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Bulterys, Marc G., Sander Greenland, and Jess F. Kraus. "Chronic Fetal Hypoxia and Sudden Infant Death Syndrome: Interaction Between Maternal Smoking and Low Hematocrit During Pregnancy." Pediatrics 86, no. 4 (1990): 535–40. http://dx.doi.org/10.1542/peds.86.4.535.

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To investigate the hypothesis that chronic fetal hypoxia contributes to the etiology of sudden infant death syndrome (SIDS), a possible interaction between the effect of maternal cigarette smoking and low hematocrit during pregnancy on the risk of SIDS was studied using the US Collaborative Perinatal Project cohort. The 193 SIDS cases identified in the cohort were analyzed with 1930 controls randomly selected from infants who survived the first year of life. After adjustment for maternal age, infants born to mothers who smoked 10 or more cigarettes per day and who were anemic (hematocrit less

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Hoffman, Howard J., Jehu C. Hunter, Karla Damus, et al. "Diphtheria-Tetanus-Pertussis Immunization and Sudden Infant Death: Results of the National Institute of Child Health and Human Development Cooperative Epidemiological Study of Sudden Infant Death Syndrome Risk Factors." Pediatrics 79, no. 4 (1987): 598–611. http://dx.doi.org/10.1542/peds.79.4.598.

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The possible association between diphtheria-pertussis-tetanus (DTP) immunization and the subsequent occurrence of sudden infant death has been examined using data from the National Institute of Child Health and Human Development (NICHD) Sudden Infant Death Syndrome (SIDS) Cooperative Epidemiological Study, a large multicenter, population-based, case-control study. In a preliminary report based on the first 400 eligible singleton SIDS victims and 800 matched living control infants, no temporal association between SIDS and DTP immunization was found. From the final sample of 800 eligible singlet

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BECKWITH, J. BRUCE. "SIDS and Hypoxanthine." Pediatrics 88, no. 5 (1991): 1076–77. http://dx.doi.org/10.1542/peds.88.5.1076b.

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The study by Rognum and Saugstad concerning elevated hypoxanthine levels in the vitreous humor of victims of Sudden Infant Death Syndrome (SIDS) made a laudable attempt to correct for postmortem increases in this marker of hypoxia, but I am concerned that the authors have not sufficiently recognized some characteristics of SIDS that could have confounded their study. Because SIDS usually occurs unobserved during presumed sleep, with the body being discovered an undetermined number of hours later, the true postmortem interval is rarely known.

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Southall, D. P., J. M. Richards, V. Stebbens, A. J. Wilson, V. Taylor, and J. R. Alexander. "Cardiorespiratory Function in 16 Full-Term Infants With Sudden Infant Death Syndrome." Pediatrics 78, no. 5 (1986): 787–96. http://dx.doi.org/10.1542/peds.78.5.787.

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Twenty-four-hour tape recordings of ECG and breathing movements from 16 term infants (≥37 weeks' gestation) who subsequently died of sudden infant death syndrome (SIDS) were compared with recordings from surviving infants from the same populations. Apneic pauses of varying durations, periodic and regular breathing patterns, heart and respiratory rates during regular breathing were measured. Only one of 16 full-term infants with SIDS had findings outside the range of age-matched control infants (an excess of periodic breathing patterns and an absence of regular breathing). When the first record

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Kytir, J., and F. Paky. "Sudden infant death syndrome (SIDS) in Austria. Socioeconomic risk factors for postneonatal SIDS- and non-SIDS-mortality." Monatsschrift Kinderheilkunde 145, no. 6 (1997): 613–18. http://dx.doi.org/10.1007/s001120050161.

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Garstang, Joanna Jane, and Marivjena Menka. "Infant death from accidental suffocation and strangulation in bed in England and Wales: rare or unrecognised events?" BMJ Paediatrics Open 8, no. 1 (2024): e002419. http://dx.doi.org/10.1136/bmjpo-2023-002419.

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BackgroundMandatory joint police and healthcare investigations of sudden unexpected death in infancy (SUDI) have been in place since 2008 in England. These include death scene examination with cause of death determined at multiprofessional case conference. Detailed evidence on sleep arrangements is available for most cases potentially leading to more being identified as due to accidental suffocation. SUDI remaining unexplained following investigation are classified as SIDS (sudden infant death syndrome) or unspecified deaths.Our objective was to determine whether detailed SUDI investigation ha

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Krous, Henry F., Catherine W. Floyd, Julie M. Nadeau, Patricia D. Silva, Brian D. Blackbourne, and Claire Langston. "Medial Smooth Muscle Thickness in Small Pulmonary Arteries in Sudden Infant Death Syndrome Revisited." Pediatric and Developmental Pathology 5, no. 4 (2002): 375–85. http://dx.doi.org/10.1007/s10024-001-0099-x.

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Increased relative medial thickness (RMT) of smooth muscle in small pulmonary arteries, peripheral extension of smooth muscle into the alveolar wall arteries, and right ventricular hypertrophy (RVH), in response to purported prolonged hypoxia, have been reported in sudden infant death syndrome (SIDS). Prone sleep position, an important risk factor for SIDS, predisposes infants to hypoxia from airway obstruction or rebreathing. Since publication of the earlier pulmonary artery studies, the SIDS definition has been expanded, and sudden infant death investigational protocols have been implemented

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Landi, Kristen, Carmen Gutierrez, Barbara Sampson, et al. "Investigation of the Sudden Death of Infants: A Multicenter Analysis." Pediatric and Developmental Pathology 8, no. 6 (2005): 630–38. http://dx.doi.org/10.1007/s10024-005-8095-1.

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The investigation of sudden death of infants varies, and death rates may depend on local practices of death certification. We studied the extent of the investigation and the final cause of death (COD) in 3 regions: New York, New York, USA (NY); King County, Washington, USA (KC); and Montevideo, Uruguay (MU). We conducted a retrospective review of 543 cases (NY 258, KC 56, MU 229) of previously healthy babies who died suddenly without obvious trauma, at ages 0 to 12 months, over a 3-year period (1998 to 2001). All cases included a complete autopsy and histologic examination. Cases were assessed

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Kravtsova, L. A. "Are SIDS, SUDC and SUDEP the different masks of the same great mystery?" Rossiyskiy Vestnik Perinatologii i Pediatrii (Russian Bulletin of Perinatology and Pediatrics) 66, no. 5 (2021): 10–14. http://dx.doi.org/10.21508/1027-4065-2021-66-5-10-14.

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The cases of sudden, unexpected child death against the background of relative clinical well-being, i.e., in the absence of apparent reasons take a special place in the structure of infant mortality. Sudden Infant Death Syndrome (SIDS), which is recognized as one of the leading causes of postnatal infant mortality in most developed countries, is the most common cause of unexpected out-ofhospital death of a child. Today SIDS remains one of the most mysterious problems in medicine. The lack of identifiable mechanisms causing SIDS has led to a large number of theories about the mechanisms respons

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Kahn, A., D. Blum, M. F. Muller, et al. "Sudden Infant Death Syndrome in a Twin: A Comparison of Sibling Histories." Pediatrics 78, no. 1 (1986): 146–50. http://dx.doi.org/10.1542/peds.78.1.146.

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To determine possible characteristics of infant victims of sudden death, we examined 114 items related to the pre- and postnatal histories of 42 pairs of twins one of whom died of sudden infant death syndrome (SIDS) leaving a surviving sibling. Interviews with the parents were conducted after the occurrence of SIDS, and the data were checked with records held by gynecologists and pediatricians. To evaluate the specificity of any factors, we studied a control group of 42 age- and sex-matched pairs of twins, both of whom survived the first year of life. Only 11 of 114 characteristics were signif

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Huang, Lin-Yi, Wan-Ju Chen, Yung-Ning Yang, et al. "Maternal Age, the Disparity across Regions and Their Correlation to Sudden Infant Death Syndrome in Taiwan: A Nationwide Cohort Study." Children 8, no. 9 (2021): 771. http://dx.doi.org/10.3390/children8090771.

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Sudden infant death syndrome (SIDS) has always been a regrettable issue for families. After sleeping in the supine position was proposed, the incidence of SIDS declined dramatically worldwide. However, SIDS still accounts for the top 10 causes of infant deaths in Taiwan. Recognizing the risk factors and attempting to minimize these cases are imperative. We obtained information on cases with SIDS from the National Health Insurance Research Database in Taiwan and interconnected it with the Taiwan Maternal and Child Health Database to acquire infant–maternal basal characteristics between 2004 and

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