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Relevant bibliographies by topics / Dechallenge

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Academic literature on the topic 'Dechallenge'

Author: Grafiati

Published: 3 June 2025

Last updated: 22 June 2025

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Journal articles on the topic "Dechallenge"

1

Stephens, Myles D. "Dechallenge Revisited." Drug Information Journal 29, no. 1 (1995): 335–38. http://dx.doi.org/10.1177/009286159502900139.

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2

V.Vijaya, Prasad* P.Venkata Sravan Kumar Dr R. Siddarama. "DEATH DUE TO STEVENS JOHNSON SYNDROME; A RARE CASE REPORT." Indo American Journal of Pharmaceutical Sciences 04, no. 07 (2017): 2072–76. https://doi.org/10.5281/zenodo.835176.

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Stevens Johnson’s syndrome is a life threatening drug induced hypersensitivity reaction. The drugs that cause SJS commonly are antibacterials (sulfonamides), anticonvulsants (phenytoin, phenobarbital, and carbamazepine), non-steroidal anti-inflammatory drugs (oxicam derivatives) and oxide inhibitors (allopurinol).Clinical symptoms are urticarial skin eruptions, arthralgia or arthritis, lymphadenopathy and fever.Treatment of Stevens Johnsons Syndrome includes systemic steroids, cyclosporine, intravenous immunoglobulin’s and supportive therapy.A male patient of 58 years was admitted in cardiology unit with chief complaints of skin reactions (Erythema andrashes) all over the body and oral erosions. The patient was diagnosed to have drug induced stevens Johnson’s syndromeandthe suspected drug was amoxicillin potassium clavulanate. By using Naranjo scale and WHO-UMC scale it was found to be a probable ADR and it was managed by drug dechallenge, dexamethasone, dew on ultra-lotion, diprovate ointment. During treatment patientdeveloped severe breathlessness and died.Hence there is a need of clinical pharmacy services to detect, manage and prevent adverse drug reactions. Key words: Stevens Johnson’s syndrome, amoxicillin+potassium clavulanate, Naranjo scale, Dechallenge.

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3

Lefebvre, Jean, Luc Poirier, and Yves Lacourcière. "Prospective Trial on Captopril-Related Cough." Annals of Pharmacotherapy 26, no. 2 (1992): 161–64. http://dx.doi.org/10.1177/106002809202600202.

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OBJECTIVE: Quantify and characterize cough as an adverse reaction secondary to captopril. DESIGN: Cohort analytic study: six-month prospective trial including two-week, placebo, run-in period; five-month, single-blind, active treatment; and two-week, dechallenge period. SETTING: Outpatient clinic. PATIENTS: 199 previously treated or newly diagnosed untreated patients with mild to moderate hypertension. INTERVENTIONS: Captopril 50 mg/d during five months. OUTCOME MEASURES: Self-questionnaires, interviews, dechallenge, and rechallenge periods, Naranjo's algorithm. RESULTS AND CONCLUSIONS: (1) Cumulative incidence of captopril-induced cough over a five-month period was 7.1 percent (95 percent C.I., 6.8–7.4 percent). Incidence of drug withdrawal secondary to severe cough was 1.0 percent; this figure is somewhat similar to first reports about the reaction. (2) Cough was a generally mild to moderate symptom, present day and night in 71.4 percent of patients and resistant to antitussive agents. Cough was not statistically related to sex, age, or smoking habit; however, the female gender and the development of a common cold may be predisposing factors.

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4

Romaszko, Jerzy, Ryszard Targonski, Leszek Cichowski, and Janusz Sadowski. "Indapamide-induced hyponatremia or the syndrome of inappropriate antidiuretic hormone secretion: a case report." Open Medicine 6, no. 4 (2011): 407–10. http://dx.doi.org/10.2478/s11536-011-0027-z.

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AbstractWe report a case of an apparently well-documented indapamide-induced hyponatremia. The initial diagnosis was made on the basis of dechallenge and rechallenge performed on two occasions. Further course of the disease, which proved inconsistent with our expectations, prompted us to look for another aetiology leading to the final diagnosis of the syndrome of inappropriate antidiuretic hormone secretion (SIADH) complicated by indapamide treatment.

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5

Zeiner, Evmarie, Lea S. Blaser, Kai Tisljar, Dominik Heim, and Anne Taegtmeyer. "Fatale Agranulozytose nach Metamizol-Reexposition." Praxis 104, no. 3 (2015): 151–54. http://dx.doi.org/10.1024/1661-8157/a001916.

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Wir stellen den Fall eines 63-jährigen Mannes vor, der einen schweren septischen Schock mit letalem Ausgang auf Basis einer Agranulozytose entwickelte. Aufgrund des zeitlichen Zusammenhangs, der Verbesserung nach Dechallenge in der Vorgeschichte und aktuellem Rechallenge ist der Kausalzusammenhang für eine erneute Metamizol-induzierte Agranulozytose sicher. Wir diskutieren die Wichtigkeit, Patienten mit einer verdächtigten Metamizol-induzierten Agranulozytose nicht erneut zu exponieren und Patienten sowie behandelnde Ärzte lückenlos zu informieren.

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6

Schneider, Jenny Sarah, Matteo Montani, and Felix Stickel. "Drug-Induced Autoimmune Hepatitis following Treatment with Zoledronic Acid." Case Reports in Gastroenterology 11, no. 2 (2017): 440–45. http://dx.doi.org/10.1159/000479314.

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Adverse drug reactions are among the most frequent side effects of synthetic and complementary alternative drugs and represent the premier causes of license revocations and acute liver failure. Drug-induced liver injury can resemble literally any other genuine liver disease and usually responds well to drug dechallenge. However, in some cases autoimmune-like hepatitis can evolve, requiring short- and sometimes long-term immunosuppression. Here, we present the hitherto first case of autoimmune-like hepatitis following treatment with zoledronic acid.

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7

Hulisz, Darrell T., Jeffrey R. Welko, and Darell E. Heiselman. "Sinus Arrest Associated With Continuous‐Infusion Cimetidine." Pharmacotherapy: The Journal of Human Pharmacology and Drug Therapy 13, no. 1 (1993): 64–67. http://dx.doi.org/10.1002/j.1875-9114.1993.tb02691.x.

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The administration of intermittent intravenous infusions of cimetidine is infrequently associated with the development of bradyarrhythmias. A 40‐year‐old man with leukemia and no history of cardiac disease developed recurrent, brief episodes of apparent sinus arrest while receiving continuous‐infusion cimetidine 50 mg/hour. The arrhythmias were temporally related to cimetidine administration, disappeared after dechallenge, and did not recur during ranitidine treatment. This is the first reported case of sinus arrest associated with continuous‐infusion cimetidine.

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8

Tandon, Vishal R., Sheikh Hanan Ismail, and Amarjeet Singh. "Fixed drug combination (levosulpiride and rabeprazole)-induced atypical Parkinsonian’s disorders with associated anxiety and low-lying depression." Indian Journal of Pharmacology 56, no. 1 (2024): 52–54. http://dx.doi.org/10.4103/ijp.ijp_929_21.

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We hereby describe a rare case of levosulpiride-induced atypical parkinsonism presenting with sluggish movements, atypical kinetic tremors (tremors with voluntary movement), periorbital tremors, dystonia, difficulty in speech and coordination, postural imbalance, with additional features of difficulty in swallowing and drooling with associated recent onset psychiatric disturbances such as anxiety and low-lying depression. The dechallenge of levosulpiride and medications for associated anxiety and low-lying depression caused a complete remission of the disease within 2 ½ months.

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9

Carlini, Elisaldo Luiz de Araújo, and Solange Aparecida Nappo. "The pharmacovigilance of psychoactive medications in Brazil." Revista Brasileira de Psiquiatria 25, no. 4 (2003): 200–205. http://dx.doi.org/10.1590/s1516-44462003000400004.

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OBJETIVE: The present study aimed to analyze 219 notifications of suspected adverse reactions (AR) produced by psychoactive medicaments (ARPM), notified by Brazilian psychiatrists, during a 3-month period (April 1999 up to September 2001). METHOD: A notifying card for adverse reactions possibly produced by psychoactive medications was quarterly sent to all psychiatrists affiliated to the Brazilian Association of Psychiatry. Once each notification, dully filled in, was received, the possible adverse reaction was analyzed in order to verify the causality RESULTS: The psychiatrists classified as severe 50 of the ARPMs; 150 others were not considered as severe. Among the severe ones there were 3 deaths, 12 life-threatening reactions, 26 ARPMs required or prolonged hospitalization and 9 notifications described temporary disability of the patients. Among the medications, antidepressants ranked first with 122 ARPMs being notified, followed by neuroleptics (46 ARPMs) and antiepiletic medicaments (25 ARPMs). The three main organs and systems affected by the ARs were the Central Nervous System with 102 ARs, skin and mucosa with 44 and gastrointestinal with 21 ARPMs. CONCLUSION: Considering causality, i.e., the association between the medication and the described AR, 24 cases were considered as Definite (with positive dechallenge and rechallenge, i.e., withdrawal and reintroduction of the medication) and 134 other ARPMs were classified as Probable (only dechallenge positive; only with medication withdrawal; rechallenge was not performed).

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10

GAUJOUX-VIALA, CÉCILE, CECILIA GIAMPIETRO, THOMAS GAUJOUX, et al. "Scleritis: A Paradoxical Effect of Etanercept? Etanercept-associated Inflammatory Eye Disease." Journal of Rheumatology 39, no. 2 (2011): 233–39. http://dx.doi.org/10.3899/jrheum.110865.

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Objective.To describe 3 cases of scleritis associated with etanercept use for rheumatoid arthritis (RA) and to review the literature related to inflammatory eye diseases associated with the use of etanercept.Methods.Three cases of severe scleritis during etanercept therapy were analyzed. A systematic review of the literature in PubMed, Embase, and the Cochrane Library was performed, from 1962 to July 2010.Results.Three patients with seropositive RA developed scleritis 7–28 months after initiation of etanercept, for the first time during their long-lasting disease. In all patients the underlying disease had responded well to anti-tumor necrosis factor therapy. Ocular inflammation went into remission after discontinuation of etanercept, and no other relapses were observed. One patient experienced a dechallenge-rechallenge phenomenon (improvement in symptoms following discontinuation of the agent, then reappearance or worsening of symptoms on reexposure to the agent). Forty-two cases of inflammatory eye diseases believed to be associated with the use of etanercept have been reported in the literature: 33 uveitis, 8 scleritis, 1 orbital myositis, concerning 16 patients with RA, 10 with juvenile idiopathic arthritis, 14 with ankylosing spondylitis, and 2 with psoriatic spondyloarthropathy. Dechallenge was performed in 28 patients, leading to resolution of symptoms. Rechallenge was done in 6 cases, with clear exacerbation.Conclusion.Ocular inflammation is paradoxically a potential adverse effect of etanercept, even in previously uninvolved eyes.

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More sources

Book chapters on the topic "Dechallenge"

1

Nahler, Gerhard. "dechallenge." In Dictionary of Pharmaceutical Medicine. Springer Vienna, 2009. http://dx.doi.org/10.1007/978-3-211-89836-9_364.

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