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Journal articles on the topic 'Deep vein thrombosis'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 January 2023

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1

Aziz, F., J. T. Chen, and A. J. Comerota. "Catheter-Directed Thrombolysis of Iliofemoral Deep Vein Thrombosis Reduces Deep Vein Thrombosis Recurrence." Journal of Vascular Surgery 53, no. 1 (January 2011): 252. http://dx.doi.org/10.1016/j.jvs.2010.11.005.

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2

Parsi, Kurosh, Brydon Panozzo, Alison Bull, Anes Yang, Mina Kang, Yana Parsi, and David E. Connor. "Deep vein sclerosis following sclerotherapy: Ultrasonic and d-dimer criteria." Phlebology: The Journal of Venous Disease 35, no. 5 (September 11, 2019): 325–36. http://dx.doi.org/10.1177/0268355519873534.

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Objectives The aim of sclerotherapy is to induce fibrosclerosis of superficial veins. We postulated that inadvertent entry of sclerosants into deep veins can result in sclerotic occlusion, deep vein sclerosis, a non-thrombotic process distinct from spontaneous deep vein thrombosis. The aim of this study was to assess the role of d-dimer in differentiating between deep vein sclerosis and deep vein thrombosis. Methods Proximal trunks of great and small saphenous veins were treated with endovenous laser ablation. Venous tributaries and perforators were treated with foam ultrasound guided sclerotherapy. Ultrasound studies of lower limb deep veins were performed before and one week after the procedures, to detect deep vein occlusions (DVOs). d-dimer levels were measured for DVOs and long-term ultrasound studies monitored the recanalisation rates. Results In a six-year period, 9143 procedures were performed in 1325 patients for bilateral varicose veins. This included 1124 endovenous laser ablation and 8019 foam ultrasound guided sclerotherapy procedures. A total of 259 DVOs (2.83%) were identified on ultrasound which included 251 deep vein sclerosis (2.74%), seven deep vein thrombosis (0.07%) and one endovenous heat-induced thrombosis (EHIT, 0.08%). d-dimer values <0.5 µg/mL excluded deep vein thrombosis s, 0.5–1.0 µg/mL were more likely to be associated with deep vein sclerosis and >1.0 µg/mL were a more likely to be associated with deep vein thrombosis. Lower sclerosant concentrations and higher foam volumes were associated with increased risk of DVO ( p < .0001). No significant relationship was found between DVO and gender or thrombophilia. Deep vein thrombosis and EHIT cases but not deep vein sclerosis patients were anticoagulated. None had thromboembolic complications. Patients were followed up for a median of 299 days (37–1994 days). Recanalisation rates were 71.1% for deep vein sclerosis (92.3% competent) and 71.4% for deep vein thrombosis (60.0% competent). Conclusions Deep vein sclerosis is a relatively benign clinical entity distinct from deep vein thrombosis and does not require anticoagulation. Majority of affected veins on long-term follow-up regain patency and competence. d-dimer can be used to assist in differentiating deep vein sclerosis from deep vein thrombosis.

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3

Gillet, JL, M. Lausecker, M. Sica, JM Guedes, and FA Allaert. "Is the treatment of the small saphenous veins with foam sclerotherapy at risk of deep vein thrombosis?" Phlebology: The Journal of Venous Disease 29, no. 9 (July 17, 2013): 600–607. http://dx.doi.org/10.1177/0268355513497362.

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Objective To assess the deep vein thrombosis risk of the treatment of the small saphenous veins depending on the anatomical pattern of the veins. Method A multicenter, prospective and controlled study was carried out in which small saphenous vein trunks were treated with ultrasound-guided foam sclerotherapy. The anatomical pattern (saphenopopliteal junction, perforators) was assessed by Duplex ultrasound before the treatment. All patients were systematically checked by Duplex ultrasound 8 to 30 days after the procedure to identify a potential deep vein thrombosis. Results Three hundred and thirty-one small saphenous veins were treated in 22 phlebology clinics. No proximal deep vein thrombosis occurred. Two (0.6%) medial gastrocnemius veins thrombosis occurred in symptomatic patients. Five medial gastrocnemius veins thrombosis and four cases of extension of the small saphenous vein sclerosis into the popliteal vein, which all occurred when the small saphenous vein connected directly into the popliteal vein, were identified by systematic Duplex ultrasound examination in asymptomatic patients. Medial gastrocnemius veins thrombosis were more frequent ( p = 0.02) in patients with medial gastrocnemius veins perforator. A common outlet or channel between the small saphenous vein and the medial gastrocnemius veins did not increase the risk of deep vein thrombosis. Conclusion Deep vein thrombosis after foam sclerotherapy of the small saphenous vein are very rare. Only 0.6% medial gastrocnemius veins thrombosis occurred in symptomatic patients. However, the anatomical pattern of the small saphenous vein should be taken into account and patients with medial gastrocnemius veins perforators and the small saphenous vein connected directly into the popliteal vein should be checked by Duplex ultrasound one or two weeks after the procedure. Recommendations based on our everyday practice and the findings of this study are suggested to prevent and treat deep vein thrombosis.

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4

Brandao, Gustavo Mucoucah Sampaio, Marcone Lima Sobreira, and Hamilton Almeida Rollo. "Recanalization after acute deep vein thrombosis." Jornal Vascular Brasileiro 12, no. 4 (October 21, 2013): 296–302. http://dx.doi.org/10.1590/jvb.2013.050.

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The process of recanalization of the veins of the lower limbs after an episode of acute deep venous thrombosis is part of the natural evolution of the remodeling of the venous thrombus in patients on anticoagulation with heparin and vitamin K inhibitors. This remodeling involves the complex process of adhesion of thrombus to the wall of the vein, the inflammatory response of the vessel wall leading to organization and subsequent contraction of the thrombus, neovascularization and spontaneous lysis of areas within the thrombus. The occurrence of spontaneous arterial flow in recanalized thrombosed veins has been described as secondary to neovascularization and is characterized by the development of flow patterns characteristic of arteriovenous fistulae that can be identified by color duplex scanning. In this review, we discuss some controversial aspects of the natural history of deep vein thrombosis to provide a better understanding of its course and its impact on venous disease.

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5

Kaufman, Brian S., and Christopher C. Young. "Deep Vein Thrombosis." Anesthesiology Clinics of North America 10, no. 4 (December 1992): 823–67. http://dx.doi.org/10.1016/s0889-8537(21)00009-2.

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6

Jackson, Mike. "Deep vein thrombosis." Nursing Standard 28, no. 42 (June 18, 2014): 61. http://dx.doi.org/10.7748/ns.28.42.61.s45.

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7

Friera, Alfonsa, Nuria R. Giménez, Paloma Caballero, Pilar S. Moliní, and Carmen Suárez. "Deep Vein Thrombosis." American Journal of Roentgenology 178, no. 4 (April 2002): 1001–5. http://dx.doi.org/10.2214/ajr.178.4.1781001.

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8

Webber, Rachel. "Deep vein thrombosis." Inpharma Weekly &NA;, no. 929 (March 1994): 9–10. http://dx.doi.org/10.2165/00128413-199409290-00015.

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9

Emanuele, Patricia. "Deep Vein Thrombosis." AAOHN Journal 56, no. 9 (September 2008): 389–92. http://dx.doi.org/10.3928/08910162-20080901-02.

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10

Tindale, Rabina. "Deep vein thrombosis." Emergency Nurse 12, no. 8 (December 2004): 8. http://dx.doi.org/10.7748/en.12.8.8.s14.

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Tindale, Rabina. "Deep vein thrombosis." Emergency Nurse 16, no. 4 (July 23, 2008): 6. http://dx.doi.org/10.7748/en.16.4.6.s10.

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12

Thachil, Jecko. "Deep vein thrombosis." Hematology 19, no. 5 (June 18, 2014): 309–10. http://dx.doi.org/10.1179/1024533214z.000000000284.

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13

Walsh, Kieran. "Deep vein thrombosis." BMJ 328, no. 7445 (April 15, 2004): 938. http://dx.doi.org/10.1136/bmj.328.7445.938.

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14

Allen, Lisa, and Andrew Wilkinson. "Deep vein thrombosis." InnovAiT: Education and inspiration for general practice 13, no. 6 (March 31, 2020): 344–51. http://dx.doi.org/10.1177/1755738020911990.

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Deep vein thrombosis (DVT) is a common and serious condition with a substantial risk of morbidity and mortality for patients. A diagnosis of DVT affects both the physical health and psychosocial functioning of patients. It represents a significant cost to the NHS in both expenditure and resource allocation. Management should focus on early recognition, prompt referral and prevention of its associated complications. This article aims to provide relevant background knowledge about DVT with advice on assessment using a clinical risk tool and appropriate referral to secondary care.

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15

Harmon, Ben. "Deep vein thrombosis." Journal of Thoracic Imaging 4, no. 4 (October 1989): 15–19. http://dx.doi.org/10.1097/00005382-198910000-00007.

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16

Weinmann, Eran E., and Edwin W. Salzman. "Deep-Vein Thrombosis." New England Journal of Medicine 331, no. 24 (December 15, 1994): 1630–41. http://dx.doi.org/10.1056/nejm199412153312407.

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17

Emanuele, Patricia. "Deep Vein Thrombosis." AAOHN Journal 56, no. 9 (September 2008): 389–94. http://dx.doi.org/10.1177/216507990805600904.

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18

Darwood, Rosie J., and Frank C. T. Smith. "Deep vein thrombosis." Surgery (Oxford) 31, no. 5 (May 2013): 206–10. http://dx.doi.org/10.1016/j.mpsur.2013.02.001.

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19

Bevis, Paul M., and Frank C. T. Smith. "Deep vein thrombosis." Surgery (Oxford) 34, no. 4 (April 2016): 159–64. http://dx.doi.org/10.1016/j.mpsur.2016.02.001.

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20

Hardy, Thomas J., and Paul M. Bevis. "Deep vein thrombosis." Surgery (Oxford) 37, no. 2 (February 2019): 67–72. http://dx.doi.org/10.1016/j.mpsur.2018.12.002.

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21

Lensing, Anthonie WA, Paolo Prandoni, Martin H. Prins, and HR Büller. "Deep-vein thrombosis." Lancet 353, no. 9151 (February 1999): 479–85. http://dx.doi.org/10.1016/s0140-6736(98)04298-6.

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22

Malhotra, Pankaj. "Deep-vein thrombosis." Lancet 353, no. 9165 (May 1999): 1708. http://dx.doi.org/10.1016/s0140-6736(05)77018-5.

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23

Rodríguez-Fanjul, Javier, Victoria Trenchs, David Muñoz-Santanach, Maria F. de Sevilla, Teresa Toll, Jose Blanch, and Carles Luaces. "Deep Vein Thrombosis." Pediatric Emergency Care 27, no. 5 (May 2011): 417–19. http://dx.doi.org/10.1097/pec.0b013e3182187421.

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24

Simmons, Susan. "Deep vein thrombosis." Nursing 41, no. 8 (August 2011): 33. http://dx.doi.org/10.1097/01.nurse.0000399589.04099.dc.

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25

Hanes, Elizabeth. "Deep vein thrombosis." Nursing 43, no. 8 (August 2013): 43. http://dx.doi.org/10.1097/01.nurse.0000431944.08030.90.

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26

Kyrle, Paul A., and Sabine Eichinger. "Deep vein thrombosis." Lancet 365, no. 9465 (March 2005): 1163–74. http://dx.doi.org/10.1016/s0140-6736(05)71880-8.

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27

Kearon, Clive, Michael J. Kovacs, and Jim A. Julian. "Deep vein thrombosis." Lancet 366, no. 9480 (July 2005): 118. http://dx.doi.org/10.1016/s0140-6736(05)66858-4.

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28

Rossi, Gabriele, and Valeria Rossi. "Deep vein thrombosis." Lancet 366, no. 9480 (July 2005): 118–19. http://dx.doi.org/10.1016/s0140-6736(05)66859-6.

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29

Kyrle, Paul A., and Sabine Eichinger. "Deep vein thrombosis." Lancet 366, no. 9480 (July 2005): 119–20. http://dx.doi.org/10.1016/s0140-6736(05)66860-2.

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30

Bushman, Barbara A. "Deep Vein Thrombosis." ACSMʼs Health & Fitness Journal 23, no. 1 (2019): 4–7. http://dx.doi.org/10.1249/fit.0000000000000442.

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31

Watkins, Jean. "Deep vein thrombosis." Practice Nursing 20, no. 8 (August 2009): 409–10. http://dx.doi.org/10.12968/pnur.2009.20.8.43663.

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32

Connolly, A. A. "Deep vein thrombosis." BMJ 297, no. 6643 (July 23, 1988): 292. http://dx.doi.org/10.1136/bmj.297.6643.292-a.

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33

Al-Zahrani, Hazzaa, Shannon M. Bates, and Jeffrey I. Weitz. "Deep vein thrombosis." Current Treatment Options in Cardiovascular Medicine 1, no. 1 (March 1999): 43–53. http://dx.doi.org/10.1007/s11936-999-0006-8.

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34

Thompson, Amy E. "Deep Vein Thrombosis." JAMA 313, no. 20 (May 26, 2015): 2090. http://dx.doi.org/10.1001/jama.2015.4761.

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35

Shi, Dongquan, Yong Pang, Chen Yao, Feng Wang, Nan Xia, Dongyang Chen, Zhihong Xu, et al. "Deep vein thrombosis after arthroplasty: Nanjing deep vein thrombosis study." Annals of Joint 1 (May 20, 2016): 3. http://dx.doi.org/10.21037/aoj.2016.04.01.

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36

DAVIS, CHRISTOPHER, and ASIM KHWAJA. "Mechanical thrombolysis for deep vein thrombosis." Journal of the American Academy of Physician Assistants 20, no. 12 (December 2007): 53–54. http://dx.doi.org/10.1097/01720610-200712000-00015.

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37

Wicky, Stephan T. "Acute Deep Vein Thrombosis and Thrombolysis." Techniques in Vascular and Interventional Radiology 12, no. 2 (June 2009): 148–53. http://dx.doi.org/10.1053/j.tvir.2009.08.008.

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38

Armon, M. P., and B. R. Hopkinson. "Thrombolysis for acute deep vein thrombosis." British Journal of Surgery 83, no. 5 (May 1996): 580–81. http://dx.doi.org/10.1002/bjs.1800830504.

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39

Shoab, S. S., S. K. Shami, M. P. Armon, and B. R. Hopkinson. "Thrombolysis for acute deep vein thrombosis." British Journal of Surgery 83, no. 10 (October 1996): 1481–82. http://dx.doi.org/10.1002/bjs.1800831057.

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40

Ramadan, SM, EV Kasfiki, CWP Kelly, and I. Ali. "Primary upper extremity deep vein thrombosis (effort thrombosis)." Acute Medicine Journal 20, no. 2 (April 1, 2021): 151–54. http://dx.doi.org/10.52964/amja.0857.

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Primary spontaneous upper extremity deep vein thrombosis is characterised by thrombosis within deep veins draining the upper extremity due to anatomical abnormalities of the thoracic outlet causing axillosubclavian compression and subsequent thrombosis. It is an uncommon condition that typically presents with unilateral arm swelling in a young male following vigorous upper extremity activity. The diagnosis of this condition is usually made by Doppler ultrasound, but other investigations are mandatory to exclude the secondary causes of upper extremity DVT. Different treatment options are available including anticoagulation, thrombolysis, and surgery. We report the case of a young healthy male with athletic physique who presented with pain and swelling of his dominant arm after weightlifting in the gym.

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41

Spentzouris, G., A. Gasparis, RJ Scriven, TK Lee, and N. Labropoulos. "Natural history of deep vein thrombosis in children." Phlebology: The Journal of Venous Disease 30, no. 6 (May 16, 2014): 412–17. http://dx.doi.org/10.1177/0268355514536154.

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Objective To determine the natural history of deep vein thrombosis in children presented with a first episode in the lower extremity veins. Methods Children with objective diagnosis of acute deep vein thrombosis were followed up with ultrasound and clinical examination. Risk factors and clinical presentation were prospectively collected. The prevalence of recurrent deep vein thrombosis and the development of signs and symptoms of chronic venous disease were recorded. Results There were 27 children, 15 males and 12 females, with acute deep vein thrombosis, with a mean age of 4 years, range 0.1–16 years. The median follow-up was 23 months, range 8–62 months. The location of thrombosis involved the iliac and common femoral vein in 18 patients and the femoral and popliteal veins in 9. Only one vein was affected in 7 children, two veins in 14 and more than two veins in 6. Recurrent deep vein thrombosis occurred in two patients, while no patient had a clinically significant pulmonary embolism. Signs and symptoms of chronic venous disease were present at last follow-up in 11 patients. There were nine patients with vein collaterals, but no patient developed varicose veins. Reflux was found in 18 veins of 11 patients. Failure of recanalization was seen in 7 patients and partial recanalization in 11. Iliofemoral thrombosis ( p = 0.012) and failure to recanalize ( p = 0.036) increased significantly the risk for developing signs and symptoms. Conclusions Children with acute proximal deep vein thrombosis develop mild chronic venous disease signs and symptoms at mid-term follow-up and are closely related with iliofemoral thrombosis and failure to recanalization.

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42

Franzeck, U. K., I. Schalch, and A. Bollinger. "On the Relationship between Changes in the Deep Veins Evaluated by Duplex Sonography and the Postthrombotic Syndrome 12 Years after Deep Vein Thrombosis." Thrombosis and Haemostasis 77, no. 06 (1997): 1109–12. http://dx.doi.org/10.1055/s-0038-1656121.

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SummaryIn a prospective study we performed color duplex ultrasonography to evaluate patency and valvular function of previous thrombosed veins 12 years after the acute thrombosis.Normal clinical findings were found in 64% of the patients, mild postthrombotic skin changes in 28%, and marked trophic changes in 5%; only 1 venous ulcer occurred.In 39 patients, 114 initially thrombosed vein segments were evaluated. Thirty-seven of 72 proximal segments were completely recanalized (23 with valvular incompetence) and 21 segments exhibited partial recanalization (19 with valvular incompetence). Superficial femoral vein segments were completely occluded in 19%, however, excellent collateralization was provided via the deep femoral vein. Thromboses of the posterior tibial vein demonstrated a high rate of recanalization with development of valvular incompetence in 52%.Whereas obstruction and valvular incompetence as single factors led to a postthrombotic leg in 8.5% and 33%, respectively, the most frequent cause for the development of the postthrombotic syndrome was the combination of reflux plus obstruction in the deep veins (50%).Compression therapy with elastic compression stockings is recommended for at least 5 years after the acute thrombosis.

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43

Arokiaraj, Mark Christopher. "A Guitaring Technique with 035 Wire and Perforated Coronary Balloon for Thrombolysis in the Treatment of Acute Deep Vein Thrombosis." Journal Of Cardiovascular Emergencies 5, no. 3 (September 1, 2019): 104–7. http://dx.doi.org/10.2478/jce-2019-0012.

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Abstract A patient who underwent thrombolysis and inferior vena cava filter implantation for acute deep vein thrombosis treatment nine years before, presented with deep vein thrombosis on the other limb. The venous angiogram showed deep vein thrombosis in the ilio-femoral vessels. Through left femoral vein puncture, a 6F right Judkins diagnostic catheter was advanced up to the proximal iliac veins, and further advancement was not possible. Hence, a 035 hydrophilic wire was advanced, and through a guitaring technique, the clots were disrupted at the mid-thigh level. Through a 014 coronary wire, a punctured coronary balloon was placed at the distal mid-thigh level, and intravascular thrombolysis was performed for 12 hours, followed by removal of the balloon and the right coronary Judkins catheters. The leg swelling reduced in the first day, and subsequently, the patient was followed up for 7 months with anticoagulation and good resolution of the symptoms.

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44

Konoeda, Hisato, Takashi Yamaki, Atsumori Hamahata, Masakazu Ochi, Atsuyoshi Osada, Yuki Hasegawa, Miho Kirita, and Hiroyuki Sakurai. "Incidence of deep vein thrombosis in patients undergoing breast reconstruction with autologous tissue transfer." Phlebology: The Journal of Venous Disease 32, no. 4 (November 19, 2016): 282–88. http://dx.doi.org/10.1177/0268355516680427.

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Background Breast reconstruction is associated with multiple risk factors for venous thromboembolism. However, the incidence of deep vein thrombosis in patients undergoing breast reconstruction is uncertain. Objective The aim of this study was to prospectively evaluate the incidence of deep vein thrombosis in patients undergoing breast reconstruction using autologous tissue transfer and to identify potential risk factors for deep vein thrombosis. Methods Thirty-five patients undergoing breast reconstruction were enrolled. We measured patients’ preoperative characteristics including age, body mass index (kg/m2), and risk factors for deep vein thrombosis. The preoperative diameter of each venous segment in the deep veins was measured using duplex ultrasound. All patients received intermittent pneumatic pump and elastic compression stockings for postoperative thromboprophylaxis. Results Among the 35 patients evaluated, 11 (31.4%) were found to have deep vein thrombosis postoperatively, and one patient was found to have pulmonary embolism postoperatively. All instances of deep vein thrombosis developed in the calf and were asymptomatic. Ten of 11 patients underwent free flap transfer, and the remaining one patient received a latissimus dorsi pedicled flap. Deep vein thrombosis incidence did not significantly differ between patients with a free flap or pedicled flap (P = 0.13). Documented risk factors for deep vein thrombosis demonstrated no significant differences between patients with and without deep vein thrombosis. The diameter of the common femoral vein was significantly larger in patients who developed postoperative deep vein thrombosis than in those who did not ( P < 0.05). Conclusions The morbidity of deep vein thrombosis in patients who underwent breast reconstruction using autologous tissue transfer was relatively high. Since only the diameter of the common femoral vein was predictive of developing postoperative deep vein thrombosis, postoperative pharmacological thromboprophylaxis should be considered for all patients undergoing breast reconstruction regardless of operative procedure.

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45

Oliveira, Nelson, Emanuel Dias, Ricardo Lima, Fernando Oliveira, and Isabel Cássio. "Primary Iliac Venous Leiomyosarcoma: A Rare Cause of Deep Vein Thrombosis in a Young Patient." Case Reports in Medicine 2011 (2011): 1–5. http://dx.doi.org/10.1155/2011/123041.

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Introduction. Primary venous tumours are a rare cause of deep vein thrombosis. The authors present a case where the definitive diagnosis was delayed by inconclusive complementary imaging.Clinical Case. A thirty-seven-year-old female presented with an iliofemoral venous thrombosis of the right lower limb. The patient had presented with an episode of femoral-popliteal vein thrombosis five months before and was currently under anticoagulation.Phlegmasia alba dolensinstalled progressively, as thrombus rapidly extended to the inferior vena cava despite systemic thrombolysis and anticoagulation. Diagnostic imaging failed to identify the underlying aetiology of the deep vein thrombosis. The definitive diagnosis of primary venous leiomyosarcoma was reached by a subcutaneous abdominal wall nodule biopsy.Conclusion. Primary venous leiomyosarcoma of the iliac vein is a rare cause of deep vein thrombosis, which must be considered in young patients with recurrent or refractory to treatment deep vein thrombosis.

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46

Mewissen, Mark W. "Thrombolysis for Lower-Extremity Deep Vein Thrombosis." Seminars in Vascular Surgery 23, no. 4 (December 2010): 228–34. http://dx.doi.org/10.1053/j.semvascsurg.2010.10.002.

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47

Vedantham, Suresh. "Catheter-directed thrombolysis for deep vein thrombosis." Current Opinion in Hematology 17, no. 5 (September 2010): 464–68. http://dx.doi.org/10.1097/moh.0b013e32833cad98.

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48

Theiss, W. "Thrombolysis or not in deep vein thrombosis." Thrombosis Research 50 (January 1988): 57–59. http://dx.doi.org/10.1016/s0049-3848(88)80039-2.

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49

Somarouthu, Bhanusupriya, Suhny Abbara, and Sanjeeva P. Kalva. "Diagnosing Deep Vein Thrombosis." Postgraduate Medicine 122, no. 2 (March 2010): 66–73. http://dx.doi.org/10.3810/pgm.2010.03.2123.

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50

Denny, Nicholas, Shreshta Musale, Helena Edlin, Ferdinando Serracino-Inglott, and Jecko Thachil. "Chronic deep vein thrombosis." Acute Medicine Journal 17, no. 3 (July 1, 2018): 144–47. http://dx.doi.org/10.52964/amja.0719.

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Deep vein thrombosis (DVT) is an important cause of short-term mortality and long-term morbidity. Although acute DVT is often well managed, there is uncertainty in the management of chronic DVT which is increasingly being noted among patients presenting with similar symptoms to their initial DVT. The presence of a residual venous clot can be a problem for both physicians and patients fearing the risk of emboli to the same extent as the acute DVT. There are also issues in the accurate diagnosis and appropriate management of chronic DVT, which is the focus of the second part of this review.

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