Relevant bibliographies by topics / Deleted in azoospermia-like gene

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  2. Dissertations / Theses
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Academic literature on the topic 'Deleted in azoospermia-like gene'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Deleted in azoospermia-like gene"

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V.A. Ferandra, V. A. Ferandra, and Sukarjati Sukarjati. "DETEKSI DELESI GEN DAZ (Deleted in AZoospermia) PADA PRIA AZOOSPERMIA DENGAN METODE PCR (Polymerase Chain Reaction)." Medical Technology and Public Health Journal 1, no. 1 (2018): 52–62. http://dx.doi.org/10.33086/mtphj.v1i1.298.

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At this time the case of azoospermia is quite common in infertile men. Azoospermia is a condition where the semen does not contain sperm. Many causes azoospermia, including the deletion of a gene at the locus that is located on the Y chromosome long arm (YQ) known as AZF gene (Azoospermia Factor). One of the genes in the AZF region are genes that AZFc DAZ (Deleted in Azoospermia). The purpose of this study was to detect the presence of the DAZ gene deletions in men with azoospermia cases using PCR (Polymerase Chain Reaction). The study design was descriptive. Venous blood samples with EDTA ant
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Ferandra, V. A., and Sukarjati Sukarjati. "DETEKSI DELESI GEN DAZ (Deleted in AZoospermia) PADA PRIA AZOOSPERMIA DENGAN METODE PCR (Polymerase Chain Reaction)." Medical Technology and Public Health Journal 1, no. 1 (2018): 52–58. http://dx.doi.org/10.33086/mtphj.v1i1.759.

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At this time the case of azoospermia is quite common in infertile men. Azoospermia is a condition where the semen does not contain sperm. Many causes azoospermia, including the deletion of a gene at the locus that is located on the Y chromosome long arm (YQ) known as AZF gene (Azoospermia Factor). One of the genes in the AZF region are genes that AZFc DAZ (Deleted in Azoospermia). The purpose of this study was to detect the presence of the DAZ gene deletions in men with azoospermia cases using PCR (Polymerase Chain Reaction). The study design was descriptive. Venous blood samples with EDTA ant
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Zhang, Y. H., S. Q. Mei, X. W. Peng, et al. "Molecular characterization and SNPs analysis of the porcine Deleted in AZoospermia Like (pDAZL) gene." Animal Reproduction Science 112, no. 3-4 (2009): 415–22. http://dx.doi.org/10.1016/j.anireprosci.2008.05.069.

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Choi, HeeJi, and Yong-Pil Cheon. "Anatomy and Function of Deleted in Azoospermia Like (DAZL) Gene in Human and Mouse." Development & Reproduction 29, no. 2 (2025): 19–30. https://doi.org/10.12717/dr.2025.29.2.19.

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Spaine, Deborah M., Pericles Hassun, Agnaldo P. Cedenho, Valdemar Ortiz, and Miguel Srougi. "1512: Threonine 12 Alanine Mutation of the Deleted-In-Azoospermia-Like Gene in Infertile Brazilian Men with Non-Obstructive Azoospermia." Journal of Urology 173, no. 4S (2005): 409–10. http://dx.doi.org/10.1016/s0022-5347(18)35646-5.

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Linher, Katja, Queenie Cheung, Perrin Baker, Gregoy Bedecarrats, Kunio Shiota, and Julang Li. "An epigenetic mechanism regulates germ cell-specific expression of the porcine Deleted in Azoospermia-Like (DAZL) gene." Differentiation 77, no. 4 (2009): 335–49. http://dx.doi.org/10.1016/j.diff.2008.08.001.

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Houston, D. W., J. Zhang, J. Z. Maines, S. A. Wasserman, and M. L. King. "A Xenopus DAZ-like gene encodes an RNA component of germ plasm and is a functional homologue of Drosophila boule." Development 125, no. 2 (1998): 171–80. http://dx.doi.org/10.1242/dev.125.2.171.

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We have identified a localized RNA component of Xenopus germ plasm. This RNA, Xdazl (Xenopus DAZ-like), encodes a protein homologous to human DAZ (Deleted in Azoospermia), vertebrate DAZL and Drosophila Boule proteins. Human males deficient in DAZ have few or no sperm and boule mutant flies exhibit complete azoospermia and male sterility. Xdazl RNA was detected in the mitochondrial cloud and vegetal cortex of oocytes. In early embryos, the RNA was localized exclusively in the germ plasm. Consistent with other organisms, Xdazl RNA was also expressed in the spermatogonia and spermatocytes of fro
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Teng, Yen-Ni, Yung-Ming Lin, Ying-Hung Lin, et al. "Association of a Single-Nucleotide Polymorphism of the Deleted-in-Azoospermia-Like Gene with Susceptibility to Spermatogenic Failure." Journal of Clinical Endocrinology & Metabolism 87, no. 11 (2002): 5258–64. http://dx.doi.org/10.1210/jc.2002-020016.

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Shen, Wei, Bong-Wook Park, Derek Toms, and Julang Li. "Midkine Promotes Proliferation of Primordial Germ Cells by Inhibiting the Expression of the Deleted in Azoospermia-Like Gene." Endocrinology 153, no. 7 (2012): 3482–92. http://dx.doi.org/10.1210/en.2011-1456.

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STEFANIDIS, K., D. LOUTRADIS, L. KOUMBI, et al. "Deleted in Azoospermia-Like (DAZL) gene–expressing cells in human amniotic fluid: a new source for germ cells research?" Fertility and Sterility 90, no. 3 (2008): 798–804. http://dx.doi.org/10.1016/j.fertnstert.2007.06.106.

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Dissertations / Theses on the topic "Deleted in azoospermia-like gene"

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Smith, Joel W. S. "Investigating the mechanism of translational stimulation by Deleted in Azoospermia-like." Thesis, University of Edinburgh, 2008. http://hdl.handle.net/1842/4849.

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The proper expression of a gene to a protein is a complicated process with many steps. One of the major steps is translation, the process of decoding a messenger RNA signal and the building of a protein from its component parts. The control of translation is one of the major steps for the overall control of gene expression and its dysregulation is associated with a wide variety of human diseases including neurological, metabolic and reproductive disorders. Dazl family proteins are germ cell restricted RNA binding proteins that contain a motif characteristic of this family, the DAZ domain. Whil
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Saxena, Richa 1968. "The human Deleted in Azoospermia gene family : structure, function and evolution." Thesis, Massachusetts Institute of Technology, 2000. http://hdl.handle.net/1721.1/9021.

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Thesis (Ph.D.)--Massachusetts Institute of Technology, Dept. of Biology, 2000.<br>Includes bibliographical references.<br>The human Deleted in Azoospermia genes lie within the AZFc region of the Y chromosome that is frequently deleted in men with severe spermatogenic failure and male infertility. This thesis describes the evolution, structure and function of human DAZ genes. First, we determined that Y-linked DAZ genes arose by transposition from an autosomal ancestor during primate evolution, and were repeatedly amplified and pruned by multiple splicing mutations. This was the first example o
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Sousa, Martins Joao Pedro. "Functional analysis of DAZL-mediated translation activation during mammalian gametogenesis." Thesis, University of Edinburgh, 2012. http://hdl.handle.net/1842/9893.

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Gametogenesis is a highly complex process that requires stringent control of gene expression, in which translational regulation plays an essential role. Deleted in Azoospermia-like (DAZL) belongs to the DAZ family of RNA-binding proteins, which are restricted to germ cells, and regulate mRNA translation. Importantly, loss of function of these proteins results in infertility in both males and females in a wide variety of organisms. A model for the mechanism by which DAZL stimulates translation has been proposed based on work in Xenopus laevis (X. laevis) oocytes. In this model, DAZL functions b
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Sprague, David Chase Cameron. "Structure and function of the deleted in azoospermia gene." 2006. http://hdl.handle.net/1969.1/ETD-TAMU-1142.

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A number of genes have been associated with variation in human spermatogenesis related to fertility. One of these, the Deleted in Azoospermia (DAZ) gene, exists as copies on two chromosomes, 3 and Y. The autosomal copy, DAZ-like (DAZL), has one RNA recognition motif (RRM) and is homologous to the DAZL gene found throughout the vertebrate lineage. There are four copies of DAZ on the Y chromosome with a pair at each of two sites. One pair contains a single RRM and the other has three RRMs. Human DAZ is homologous to genes in old world primates and ape Y chromosomes. Both DAZ and DAZL bind messen
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Chiu, Yu-Tien, and 邱于恬. "Functional Analysis of Single-Nucleotide Polymorphism of the Deleted-in-Azoospermia-Like Gene in Taiwanese Men." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/61547006636890129731.

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碩士<br>國立成功大學<br>生物化學研究所<br>95<br>Infertility affects 10–15% of all couples, and impaired sperm production accounts for about half of these cases. Deleted in Azoospermia (DAZ) gene was originally isolated from the AZFc region on the long arm of the human Y chromosome that is frequently deleted in infertile men. DAZ-like (DAZL), an autosomal homolog located on chromosome 3p24, are expressed exclusively in the germ cells, and their protein products contain a highly conserved RNA-binding motif and a unique DAZ repeat. DAZL gene is essential for the differentiation of germ cells. Loss of germ cells
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Lin, Yi-Wen, and 林怡彣. "Characterization of the Human TestisExpressed DAZ (Deleted in Azoospermia) gene." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/22846304514290204637.

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博士<br>國防醫學院<br>生命科學研究所<br>95<br>Abstract PART Ⅰ: gene structure analysis of the DAZ genes Background—The human Y chromosome is unique in that it does not engage in pairing and crossing over during meiosis for most of its length. The azoospermia factor c (AZFc) region of the human Y chromosome consists entirely of very long repeats and is prone to rearrangement. This region is frequently deleted in infertile men with non-obstructive azoospermia or severe oligospermia. Several gene families with testis-specific expression are located within AZFc, including the Deleted in Azoospermia (DAZ) gene
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Chien, Yi-Chieh, and 簡昱杰. "The gene expression of dazl(deleted in azoospermia-like) and gcl(germ cell-less) in the gonad of black porgy, Acanthopagrus schlegeli." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/11215914734962624765.

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碩士<br>國立臺灣海洋大學<br>水產養殖學系<br>100<br>The purpose of this study was to investigate the gene expresstion of deleted in azoospermia-like (dazl) and germ cell-less (gcl) in the gonad of black porgy, Acanthopagrus schlegeli. dazl full length cDNA contains 2359 nucleotides encoding a putative 200 amino acid (aa) protein, and gcl contains 3415 nucleotides encoding a putative 503 aa protein. By RT-PCR, dazl is specificly expressed only in the gonad, and gcl has a high expression in the gonad and low expression level in brain, eyes, heart, liver, kidney and is not detectable in muscle. The gene expressio
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HSU, CHIAO-CHUN, та 徐巧君. "Estrogen Receptorα and β Gene Expression in Primary Human Osteoblast-Like Cells and the Detection of an Exon2-Deleted Estrogen Receptorβ mRNA Variant". Thesis, 2001. http://ndltd.ncl.edu.tw/handle/01716048136598436980.

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Book chapters on the topic "Deleted in azoospermia-like gene"

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Erol, Nihal Öztolan. "Soybean Improvement and the Role of Gene Editing." In A Roadmap for Plant Genome Editing. Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-46150-7_17.

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AbstractSoybean is a major agricultural crop that is used for food, feed, and industrial products. However, soybean production is facing several challenges, including pests, diseases, and environmental factors. In recent years, there has been a growing interest in using gene editing technologies to improve soybean traits. Gene editing technologies offer a promising new approach to improving soybean production and quality.Gene editing technologies can be used to precisely alter the soybean genome. There are a number of different gene editing technologies that can be used to improve soybeans. On
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Selvam, R., and Dr D. Anandhi. "ROLE OF PARKIN GENE IN BREAST CANCER: A MOLECULAR APPROACH." In Futuristic Trends in Biotechnology Volume 3 Book 15. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/v3bjbt15p3ch1.

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Parkin, initially linked to Parkinson's Disease (PD) through its association with autosomal recessive juvenile parkinsonism, encompasses an E3 ubiquitin ligase and represents a member of the RBR (RING, in-between RING, RING) proteins. While predominantly studied in PD, Parkin's altered function has been implicated in various diseases like cerebral ischemia, leprosy, and human cancers, although the precise mechanisms linking Parkin to cancer susceptibility, particularly in breast cancer remain debated. Encoded by a substantial gene, Parkin yields a modest 465 amino acids, housing ubiquitin-like
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Torres, Raul M., and Ralf Kühn. "Nonselectable modifications and removal of selection marker genes." In Laboratory Protocols for Conditional Gene Targeting. Oxford University PressOxford, 1997. http://dx.doi.org/10.1093/oso/9780199636778.003.0004.

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Abstract One of the most straightforward purposes for using Cre/loxP is to remove a loxP-flanked (floxed) selectable marker after homologous recombinants have been identified in a gene targeting experiment. This is certainly important for any experiment designed to modify, but not abrogate, gene function by preventing possible interference of the selectable gene (and its own regulatory elements) on gene function and/or regulation (20, 21, 87). The range of modifications which can be introduced into the target gene is virtually unlimited ranging from alterations in the coding region, like point
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Zbuk, Kevin M., and Charis Eng. "PTEN and the Cowden and Bannayan–Riley–Ruvalcaba Syndromes." In Inborn Errors Of Development. Oxford University PressNew York, NY, 2008. http://dx.doi.org/10.1093/oso/9780195306910.003.0058.

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Abstract The PTEN Hamartoma Tumor Syndrome (PHTS) includes not only the entities Cowden Syndrome (CS; MIM 158350) and Bannayan–Riley–Ruvalcaba syndrome (BRRS; MIM 153480), which are the focus of this chapter, but also Proteus syndrome (PS; MIM 176920), and Proteus-like syndrome (PSL), and perhaps other developmental disorders. These heterogeneous groups of disorders are all, to varying degrees, associated with germline mutations of the PTEN [for phosphatase and tensin homolog, deleted on chromosome 10 (ten)] tumor suppressor gene. PTEN, localized to 10q23.3 is a dual specicity phosphatase. Its
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Torres, Raul M., and Ralf Kühn. "Conditional gene modification." In Laboratory Protocols for Conditional Gene Targeting. Oxford University PressOxford, 1997. http://dx.doi.org/10.1093/oso/9780199636778.003.0009.

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Abstract Following one of the three strategies described in this section, from a single targeting construct, ES clones can be generated which harbor either a loxP-flanked (floxed) allele suitable for conditional gene targeting or which contain the final genetic modification resulting from ere-mediated deletion or inversion. For the floxed allele subsequent ere-mediated recombination will result in gene inactivation or modification, depending on loxP positioning and orientation. The mice derived from ES cells with a floxed allele can be crossed to ere transgenic mice for conditional gene modifi
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