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1

Nori, Graham, and Warner James Dr, eds. Dementia: Alzheimer's and other dementias. 2nd ed. Class, 2002.

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2

Mendez, Mario F. Dementia: A clinical approach. 3rd ed. Butterworth-Heinemann, 2003.

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3

Burns, Alistair, and Raymond Levy, eds. Dementia. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-6805-6.

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4

Edwards, Allen Jack. Dementia. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4757-9963-7.

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5

Krishnamoorthy, Ennapadam S., Martin J. Prince, and Jeffrey L. Cummings, eds. Dementia. Cambridge University Press, 2009. http://dx.doi.org/10.1017/cbo9780511780615.

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6

Maj, Mario, and Norman Sartorius, eds. Dementia. John Wiley & Sons, Ltd, 2002. http://dx.doi.org/10.1002/0470861878.

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7

Mahendra, B. Dementia. Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3183-1.

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8

Quinn, Joseph F. Dementia. John Wiley & Sons Ltd, 2013. http://dx.doi.org/10.1002/9781118656082.

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9

Maj, Mario, and Norman Sartorius, eds. Dementia. John Wiley & Sons, Ltd, 2000. http://dx.doi.org/10.1002/0470842350.

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10

Hickey, Ellen M., and Michelle S. Bourgeois, eds. Dementia. Routledge, 2017. http://dx.doi.org/10.4324/9781315103891.

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11

Keen, Justin. Dementia. Office of Health Economics, 1992.

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12

Hardman, Lizabeth. Dementia. Lucent Books, 2009.

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13

Hardman, Lizabeth. Dementia. Lucent Books, 2009.

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14

Stackpole, Michael A. Dementia. Roc, 1994.

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15

Brice, Pitt, ed. Dementia. Churchill Livingstone, 1987.

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16

1953-, Maj Mario, and Sartorius N, eds. Dementia. 2nd ed. Wiley, 2002.

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17

Executive, NHS Management. Dementia. Health Publications Unit, 1994.

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18

S, Burns Alistair, and Levy Raymond, eds. Dementia. Chapman & Hall Medical, 1994.

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19

S, Burns Alistair, O'Brien John DM, and Ames David 1954-, eds. Dementia. 3rd ed. Hodder Arnold, 2005.

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20

Keen, Justin. Dementia. Office of Health Economics, 1992.

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21

Executive, NHS Management. Dementia. NHS Management Executive, 1994.

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22

D, Bowen James, ed. Dementia. Saunders, 2002.

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23

Patrick, McNamara. Dementia. Praeger, 2011.

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24

Biernacki, Claire. Dementia. John Wiley & Sons, Ltd., 2007.

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25

T, DeKosky Steven, ed. Dementia. W.B. Saunders, 2000.

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26

DM, O'Brien John, Burns Alistair S, and Levy Raymond, eds. Dementia. 2nd ed. Arnold, 2000.

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27

J, Whitehouse Peter, ed. Dementia. F.A. Davis, 1992.

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28

Fernandez, Evelina. Dementia. Alexander Street Press, 2007.

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29

1953-, Maj Mario, and Sartorius N, eds. Dementia. John Wiley, 2000.

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30

McNamara, Patrick, ed. Dementia. Praeger, 2011. http://dx.doi.org/10.5040/9798216964582.

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This three-volume collection of essays provides a comprehensive review of state-of-the-art clinical phenomenology, mechanisms, and treatment strategies for the major dementias—particularly the neuropsychiatric disorders involved. Dementia presents a very significant problem: a recent study by Alzheimer's Disease International estimates that the number of people with dementia worldwide will exceed 35 million by 2050. Neuropsychiatric disorders of the dementias are a major source of stress for the family members of those afflicted and a primary reason for nursing home placement, resulting in the
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31

McNamara, Patrick, ed. Dementia. Praeger, 2011. http://dx.doi.org/10.5040/9798216964575.

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This three-volume collection of essays provides a comprehensive review of state-of-the-art clinical phenomenology, mechanisms, and treatment strategies for the major dementias—particularly the neuropsychiatric disorders involved. Dementia presents a very significant problem: a recent study by Alzheimer's Disease International estimates that the number of people with dementia worldwide will exceed 35 million by 2050. Neuropsychiatric disorders of the dementias are a major source of stress for the family members of those afflicted and a primary reason for nursing home placement, resulting in the
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32

McNamara, Patrick, ed. Dementia. Praeger, 2011. http://dx.doi.org/10.5040/9798216964599.

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This three-volume collection of essays provides a comprehensive review of state-of-the-art clinical phenomenology, mechanisms, and treatment strategies for the major dementias—particularly the neuropsychiatric disorders involved. Dementia presents a very significant problem: a recent study by Alzheimer's Disease International estimates that the number of people with dementia worldwide will exceed 35 million by 2050. Neuropsychiatric disorders of the dementias are a major source of stress for the family members of those afflicted and a primary reason for nursing home placement, resulting in the
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33

Beller, Jerry, and Beller Health. 14 Dementia Types: Alzheimers Lewy Body Dementia Frontotemporal Dementia Vascular Dementia Huntington's Disease Other Dementias. Independently Published, 2019.

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34

Beller, Jerry, and Beller Health. 2019 Dementia Overview: Alzheimers , Lewy Body Dementia , Frontotemporal Dementia , Vascular Dementia , Huntington's Disease , Other Dementias. Independently Published, 2019.

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35

Piggott, Margaret Ann. Neurochemical pathology of dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0007.

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This chapter considers the neurodegenerative disorders Alzheimer’s disease (AD), Lewy body dementias (dementia with Lewy bodies (DLB) and Parkinson’s disease dementia(PDD)), frontotemporal dementia (FTD); and also vascular dementia (VaD) which results from cerebrovascular disease. These different conditions, which give rise to dementia syndromes, each have distinct neurochemical pathologies, with important implications for treatment. As increased age is the common risk factor generally associated with dementing illnesses, neurochemical changes are set in the context of the changes which occur
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36

Brookes, Alpha. Dementia Signs : Frontotemporal Dementia: Dementia Meaning. Independently Published, 2021.

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37

Gan, Li. Cellular Mechanisms of Dementia. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0054.

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Neurodegenerative dementias, including Alzheimer’s disease (AD), Parkinson’s disease (PD) and Frontotemporal dementia (FTD), pose enormous challenges for our aging society. Genetic and mechanistic studies have revealed common molecular and cellular pathways, including imbalanced proteostasis and aberrant innate immune responses. Key pathogens in AD, PD, and FTD accumulate and spread from one brain region to another, resulting in network dysfunction and cognitive decline. These diseases are multifactorial, caused by interactions among multiple genetic, epigenetic, and environmental factors and
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38

Zahn, Roland, and Alistair Burns. Dementia disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198779803.003.0001.

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This chapter provides a brief overview of the different forms of dementia syndromes and provides a simple algorithm for initial differential diagnosis. Rapidly progressive dementias have to be excluded which require specific investigations to detect Creutzfeldt–Jakob as well as inflammatory and autoimmune diseases. A lead symptom-based approach in patients with slowly progressive cognitive and behavioural impairments without neurological symptoms is applied: progressive and primary impairments in recent memory are characteristic of typical Alzheimer’s dementia, primary behavioural changes poin
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39

Engdahl, Sylvia. Dementia. Greenhaven Publishing LLC, 2013.

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40

Hughes, Julian, Stephen Louw, and Steven R. Sabat, eds. Dementia. Oxford University Press, 2005. http://dx.doi.org/10.1093/med/9780198566151.001.0001.

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41

Dickerson, Bradford, and Alireza Atri, eds. Dementia. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199928453.001.0001.

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42

Armstrong, Richard. Dementia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0228.

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Dementia is a syndrome defined by a persistent, progressive decline in multiple cognitive functions to a degree sufficient to detrimentally impact activities of daily living and social function. The syndromic diagnosis of dementia is useful, since the general management and economic burden of these patients remains similar, irrespective of etiology. However, a more precise etiological diagnosis must be sought, since disease-specific treatment is increasingly likely to be appropriate. The term ‘mild cognitive impairment’ (MCI) refers to an objective impairment in a cognitive function (usually m
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43

Hydén, Lars-Christer. Dementia. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199391578.003.0003.

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This chapter focuses on the changing brain of individuals living with dementia and the challenges produced by these changes. It is argued that to better understand what persons with changing brains can do as storytellers, together with other persons, it is necessary to add a social psychological perspective to more traditional psychological knowledge, which often is centered on a decontextualized individual and that person’s diseased brain. The chapter discusses the brain’s role in storytelling. Storytelling is a ubiquitous and important everyday activity, but also a very complex social activi
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44

Quinn, Joseph. Dementia. Wiley & Sons, Incorporated, John, 2013.

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45

Dementia. W.B. Saunders Co., 2002.

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46

Widera, Eric, and Rachelle Bernacki. Dementia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0154.

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Dementia is caused by a variety of disorders that result in a progressive loss of both cognitive and functional abilities. Despite the heterogeneity of disorders, there is a common set of problems that patients and families face living with this syndrome. Symptoms such as pain, eating difficulties, depression, and agitation are all common. As the disease progresses to the advanced stages, the different disorders share a common functional trajectory that includes persistently severe disability with complete dependence on others for basic activities of daily living. Care for individuals with dem
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47

Burrell, James R., and John R. Hodges. Dementia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199658602.003.0010.

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Cognitive neurology has exploded over the last century, and especially over the last 20 years. From the distinction of dementia as a pathological entity, rather than just ‘normal’ ageing, to more sophisticated sub-classification of dementia syndromes, much has been learned, though great challenges remain. From an incredible array of worthy research studies, ten landmark papers in the field of dementia are presented in this chapter. With regard to Alzheimer’s disease, the following are discussed: the initial description of the disease, both clinically and pathologically; the development of mean
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48

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Dementia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0011.

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This chapter discusses the clinical features and evidence-based pharmacological management of dementia disorders (Alzheimer’s disease (AD), vascular dementia, dementia with Lewy bodies (DLB), Parkinson’s disease dementia (PDD), frontotemporal dementia (FTD), mixed dementia, and mild cognitive impairment (MCI)).
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49

Pace, Victor, Adrian Treloar, and Sharon Scott, eds. Dementia. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199237807.001.0001.

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50

Ames, David, Alistair Burns, and John T. O'Brien. Dementia. CRC Press, 2010. http://dx.doi.org/10.1201/b13196.

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