Relevant bibliographies by topics / Demenza frontotemporale

Academic literature on the topic 'Demenza frontotemporale'

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Published: 9 March 2023
Last updated: 10 March 2023

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Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Journal articles on the topic "Demenza frontotemporale":

1

Otto, Markus, Emily Feneberg, and Sarah Anderl-Straub. "Frontotemporale Demenz." Der Klinikarzt 47, no. 10 (October 2018): 472–76. http://dx.doi.org/10.1055/a-0742-4753.

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ZusammenfassungDie behaviorale frontotemporale Demenz (bvFTD) ist bei Patienten vor dem 65. Lebensjahr die zweithäufigste Demenzerkrankung nach der Alzheimer-Erkrankung. Die frontotemporalen Atrophien können sich in Form zweier klinischer Subtypen manifestieren. Entweder stehen bei der behavioralen FTD klinisch Verhaltens-, und Persönlichkeitsveränderungen im Vordergrund oder bei der primär progredienten Aphasie eine Störung von Sprachverständnis und -produktion. Eine sichere Diagnose kann bisher nur post mortem mit Hilfe der Neuropathologie oder durch den Nachweis einer genetischen Mutation erfolgen. Zur Diagnosestellung werden neben der klinischen Einteilung derzeit Bildgebungsverfahren (MRT und FDG-PET) zur Beurteilung des Frontal-, und Temporallappens ergänzt. Die Untersuchung von Tau, Amyloid-ß und der Neurofilamente kann zur Abgrenzung einer Alzheimer-Erkrankung erfolgen.
2

Diehl, Janine, and A. Kurz. "Frontotemporale Demenz." Wiener Medizinische Wochenschrift 152, no. 3-4 (February 2002): 92–97. http://dx.doi.org/10.1046/j.1563-258x.2002.01131.x.

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3

Weih, Markus, Christos Sidiropoulos, and Jens Wiltfang. "Frontotemporale Demenz." Psychiatrie und Psychotherapie up2date 1, no. 2 (March 2007): 89–102. http://dx.doi.org/10.1055/s-2006-951943.

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4

Spatt, J. "Frontotemporale Demenz." Psychopraxis 16, no. 3 (June 2013): 15–18. http://dx.doi.org/10.1007/s00739-013-0071-3.

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5

Woo, Hee-Soon. "Dysphagia and Dietary Changes Cause of Frontotemporal Dementia." Swallowing Rehabilitation 1, no. 1 (March 31, 2018): 7–14. http://dx.doi.org/10.31115/sr.2018.1.1.7.

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6

Antunes Pereira, Daniel, Shara Aline Bueno Dantas, Marco Antônio Orsini Neves, Everton Gonçalves Pinto, Gilberto Canedo Martins Jr, and Antonio Marcos da Silva Catharino. "Frontotemporal dementia: From the clinic to the differential diagnosis." International Journal of Case Reports and Images 14, no. 1 (January 31, 2023): 28–32. http://dx.doi.org/10.5348/101377z01dp2023cr.

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Introduction: Frontotemporal dementia (FTD) is a disease that encompasses several syndromes that differ in their cognitive, behavioral, language signs, and motor phenomena. Only Alzheimer’s disease causes more early-onset dementia cases than FTD. According to World Health Organization (WHO) projections, dementia rates will double every 20 years and reach 115.4 million people in 2050, ranging from 3% to 26%. The FTD spectrum encompasses three variant syndromes, namely the behavioral variant, the semantic variant, and the non-fluent/agrammatical variant. Frontotemporal lobar degeneration is neuropathologically related to the clinical phenotypes of FTD. Therefore, the frontal and temporal lobes suffer from gliosis and selective neuronal loss due to this neurodegenerative condition. Case Report: A 62-year-old doctor with no comorbidities. According to the family, he reports delivering very slowly, which he doesn’t and comes in the processing very efficiently. He claims that he already lost on the street but managed to restore his visuospatial function. He denies changes in his daily life to recent trauma. Neurological examination revealed impaired attention and behavioral changes, impaired short-term memory (mini-mental: 19/30 points), normal laboratory, regular liquor; skull magnetic resonance imaging (MRI) showed mild atrophy in the frontotemporal regions. Decreased activity in areas of the cortex was observed by cerebral perfusion scintigraphy. Conclusion: Therefore, this report is relevant because it correlates a patient with neurological examination and tests with a good indication of FTD; however, the diagnosis can be confirmed with greater accuracy through brain perfusion scintigraphy. It is still possible to observe that although there has been a significant increase in the literature on FTD, its variants and its clinic still need further studies regarding their possible differential diagnoses, mainly related to psychiatric disorders and the behavioral variant of FTD.
7

Mandić-Stojmenović, Gorana, Vladimir Kostić, and Elka Stefanova. "The clinical spectrum of frontotemporal dementia." Medicinski podmladak 69, no. 2 (2018): 9–15. http://dx.doi.org/10.5937/mp69-16325.

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Deramecourt, V., F. Lebert, and F. Pasquier. "Demenze frontotemporali." EMC - Neurologia 8, no. 1 (January 2008): 1–14. http://dx.doi.org/10.1016/s1634-7072(08)70540-3.

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9

Diehl, J., I. R. Mackenzie, H. F�rstl, and A. Kurz. "Die frontotemporale Demenz." Der Nervenarzt 74, no. 9 (September 1, 2003): 785–88. http://dx.doi.org/10.1007/s00115-003-1509-2.

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10

Witt, K., G. Deuschl, and T. Bartsch. "Frontotemporale Demenzen." Der Nervenarzt 84, no. 1 (February 26, 2012): 20–32. http://dx.doi.org/10.1007/s00115-012-3477-x.

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You might also be interested in the extended bibliographies on the topic 'Demenza frontotemporale' for particular source types:
Journal articles Dissertations / Theses Books

Dissertations / Theses on the topic "Demenza frontotemporale":

1

VINCETI, GIULIA. "Personalità premorbosa nello spettro Demenza Frontotemporale – Sclerosi Laterale Amiotrofica (DFT-SLA): correlati comportamentali e di imaging cerebrale." Doctoral thesis, Università degli studi di Modena e Reggio Emilia, 2022. http://hdl.handle.net/11380/1273443.

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Background: la Demenza Frontotemporale (FTD) e la Sclerosi Laterale Amiotrofica (ALS) sono considerati due fenotipi dello stresso continuum neurodegenerativo (lo spettro FTD-ALS) caratterizzato da un substrato patologico e genetico comune. I pazienti possono presentare forme cliniche “pure”, cognitivo/comportamentali (FTD) o motorie (ALS), o forme miste (FTD-ALS). I fattori che determinano lo sviluppo di uno fenotipo piuttosto che un altro sono attualmente sconosciuti. Tuttavia, alcune caratteristiche della personalità dei pazienti sono percepite dai terapeuti come ricorrenti in entrambi i fenotipi: i pazienti ALS tendono a mostrare un comportamento pro-sociale caratterizzato da gentilezza e gradevolezza; al contrario, i pazienti FTD presentano disinibizione, comportamenti antisociali e mancanza di empatia. Questi tratti sono spesso descritti dai congiunti come già presenti e strutturali della personalità del paziente, sebbene attenuati. Obiettivo del presente studio è indagare se pazienti con FTD e ALS siano caratterizzati da differenti profili di personalità nella loro vita premorbosa, con l'ipotesi che la personalità premorbosa rifletta una vulnerabilità specifica dei circuiti cerebrali legati al comportamento sociale e alla funzione motoria. Metodo: sono stati reclutati prospetticamente 46 pazienti (30 FTD, 13 ALS, e 3 FTD-ALS, questi ultimi assegnati al gruppo FTD sulla base del sintomo di presentazione). La personalità dei pazienti è stata valutata attraverso il NEO Personality Inventory 3 (NEO-PI-3), che analizza cinque fattori di personalità (nevroticismo, estroversione, apertura, gradevolezza, coscienziosità). Il NEO-PI-3 è stato somministrato ai familiari dei pazienti, chiedendone la compilazione in duplice copia, con riferimento alla personalità del paziente alla diagnosi e 15 anni prima dell'inizio dei sintomi, rispettivamente. I pazienti sono stati inoltre sottoposti a risonanza magnetica cerebrale (MRI), con acquisizione di sequenze T1 ad alta risoluzione e sequenze di risonanza magnetica funzionale a riposo (rsfMRI) per successive analisi di Voxel based morphometry (VBM) e Probabilistic Independent Component Analysis (ICA), rispettivamente. Risultati: Una differenza significativa nella personalità premorbosa è emersa nel dominio Apertura, evidenziando come i pazienti ALS presentino maggiore apertura alle esperienze, idee ed emozioni rispetto ai pazienti FTD (150 vs 133, p=0.020), così come nel dominio dell'Estroversione, evidenziando come i pazienti ALS si siano caratterizzati, nella vita premorbosa, per maggiore socievolezza, loquacità e ottimismo (150 vs 134, p = 0,006). L'analisi di VBM ha mostrato una correlazione positiva tra il dominio di Nevroticismo premorboso e il volume GM nelle aree dell'ippocampo sinistro e del nucleo accumbens bilaterale. Nell'analisi tra gruppi dei resting state networks (RSNs) precedentemente identificati, il gruppo ALS ha mostrato una maggiore connettività funzionale (FC) rispetto ai soggetti FTD nel RSN sensori-motorio. Nel salience RSN, il gruppo ALS e il gruppo dei controlli hanno mostrato una maggiore FC rispetto alla FTD nel lobo parietale destro e nel cervelletto. Nell'analisi di correlazione tra FC e personalità premorbosa, nel gruppo FTD è emersa una correlazione negativa tra FC nel RSN sensori-motorio ed Estroversione e Apertura, al contrario nel gruppo ALS è emersa una tendenza alla correlazione positiva. Conclusione: il profilo di personalità premorbosa differisce nei pazienti FTD e ALS in due domini, Apertura ed Estroversione, sostenendo l'ipotesi che la personalità premorbosa possa rappresentare un marcatore di vulnerabilità allo sviluppo di disturbi comportamentali o motori.
Background: Frontotemporal dementia (FTD) and amyotrophic lateral sclerosis (ALS) are two clinical expressions of the same neurodegenerative continuum (FTD-ALS spectrum) with common underlying pathology and genetic background. Patients can present with pure behavioural/cognitive (FTD), pure motor (ALS) or mixed (FTD-ALS) forms. What determines the development of one rather than the other phenotype is unknown. However, it is common observation that patients’ personality differs between the phenotypes: ALS patients tend to display a prosocial behavior characterized by kindness and agreeableness; FTD patients tend to present with disinhibition, anti-social behaviors and lack of empathy. These traits are often described by patients’ relatives as having always characterized the patients’ personality. We therefore aimed at testing if FTD and ALS patients had different personality profiles in their premorbid life, with the hypothesis that premorbid personality reflects a specific vulnerability to damage of brain circuits related to social behavior or motor function. Method: We prospectively recruited consecutive eligible FTD and ALS patients presenting to our Neurology Clinics. Patients’ personality was assessed through the NEO Personality Inventory 3 (NEO-PI-3), which analyses the five main personality factors (Neuroticism, Extraversion, Openness, Agreeableness, Conscientiousness). NEO-PI-3 was administered to patients’ caregivers with reference to the patient’s personality at two timepoints: at diagnosis and 15 years prior to symptoms onset. Patients also underwent MRI scan of the brain including High resolution T1-weighted and resting state functional MRI (rsfMRI) sequences. Imaging data were analyzed with FSL tools including voxel-based morphometry (VBM) and probabilistic independent component analysis (ICA). Result: 46 patients (30 FTD, 13 ALS, and 3 FTD-ALS assigned to the FTD group, based on their first symptom) were recruited. A significant difference in premorbid personality emerged in the Openness domain, showing that ALS patients had been more open to new experience, ideas and emotions than FTD patients (150 vs 133, p=0.020), even many years before symptoms onset. Also, a significant difference between groups emerged in the Extraversion domain, showing that ALS patients were characterized, in premorbid life, by higher sociability, loquacity, and optimism (150 vs 134, p = 0.006). The VBM analysis showed a positive correlation between premorbid Neuroticism and GM volume in the areas of left hippocampus-parahippocampal gyrus and left and right nucleus accumbens. In the between-group analysis of the previously identified RSNs, in the sensory-motor RSN ALS showed greater functional connectivity than both FTD and control subjects in the right motor cortex. In the salience RSN, controls and ALS showed increased functional connectivity compared to FTD in the right parietal lobe and in the cerebellum. In the correlation analysis between functional connectivity and premorbid personality scores, a negative correlation between FC in sensory-motor RSN and Extraversion and Openness emerged in FTD, conversely a trend to positive correlation emerged in the ALS group. Conclusion: premorbid personality profile differ in FTD and ALS patients in two domains, Openness and Extraversion, supporting the hypothesis that premorbid personality may represent a vulnerability marker to the development of behavioral or motor disturbances.
2

RIZ, M. A. DE. "FATTORI DI RISCHIO GENETICI NELLA MALATTIA DI ALZHEIMER E NELLA DEMENZA FRONTOTEMPORALE: STUDIO DI ASSOCIAZIONE DI GENI CANDIDATI POSIZIONALI E FUNZIONALI." Doctoral thesis, Università degli Studi di Milano, 2011. http://hdl.handle.net/2434/150554.

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Alzheimer’s disease (AD) and Frontotemporal Lobar Degeneration (FTLD) are two neurodegenerative and multifactorial diseases with complex etiology in which several genes involved in inflammation, oxidative damage and neuronal survival have been proposed to be candidate susceptibility factors. Given these premises, aims of this study have been to further analyze the association of candidate functional and positional genes in a population of 374 patient with AD, 291 with FTLD and 344 age matched controls. The first candidates studied have been the cell-dependent kinase inhibitor (CDKN) 2A and 2B, involved in cell cycle G1 phase progression, because abnormal cell cycle re-entry has been hypothesized to play a role in neurodegenerative disease, primarily AD. Allelic and genotypic frequencies of three tagging Single Nucleotide Polymorphisms (SNPs), namely rs3731239 T/C, rs2811710 C/T and rs3217992 G/A, spanning CDKN2A and CDKN2B, and covering 100% gene variability, were analyzed. According to our data, CDKN2A and CDKN2B don’t likely act as risk factors for AD. Given the potential importance of kinesin KIF24, protein expressed in neurons and involved in axonal transport and neuron development, we studied the distribution of five Single Nucleotide Polymorphism (SNPs) located in the chromosome 9 haplotype identified via linkage analysis, including UBAP1 rs7018487, UBAP2 rs1785506 and rs307658, and KIF24 rs17350674 and rs10814083. A statistically significant increased frequency of the KIF24 rs17350674 AA genotype was observed in FTLD patients compared with controls, particularly in bvFTD and female patients, showing that KIF24 rs17350674 polymorphism may act as a risk factor for sporadic FTLD. BCL2-associated athanogene 1 (BAG1) is an anti-apoptotic factor interacting with tau and regulating its proteasomal degradation. A statistically significant decreased allelic frequency of the BAG-1 rs706118 SNP was observed in patients with FTLD, but not in AD. BAG-1 rs706118 SNP likely acts as protective factor for sporadic FTLD, but not for AD. Finally we studied the Heterogeneous nuclear ribonucleoprotein hnRNP-A1, which may be linked to AD, because it could influence the maturation of the Amyloid Precursor Protein (APP) mRNA, but also to FTLD, because it contains a glycine-rich consensus domain included in TAR DNA binding protein (TDP)43. In our study, hnRNP-A1 rs7967622 C/C genotype acts as risk factor only for FTLD in male population, while hnRNP-A1 relative expression levels were increased in AD patients, togheter with decreased levels of its transcription regulatory factor hsa-miR-590-3p. We confirmed the importance of genes influencing neuronal transport, metabolism and survival in both neurodegenerative diseases AD and FTLD; the observed differences are probably related to specific roles of these genes in pathogenic events occurring in FTLD only.
3

Haussmann, Robert, Marek Wysocki, Moritz D. Brandt, Andreas Hermann, and Markus Donix. "MAPT mutation associated with frontotemporal dementia and parkinsonism (FTDP-17)." Cambridge University Press, 2017. https://tud.qucosa.de/id/qucosa%3A70705.

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We present a 56-year-old patient suffering from frontotemporal dementia with parkinsonism linked to chromosome 17 (FTDP-17). The history included a three-generation pedigree and the patient was found to be a mutation carrier. The diagnosis was hindered by late appearance of the hypokinetic movement disorder. For clinicians, it is important to consider rare neurodegenerative disease variants in early-onset familial dementia syndromes with behavioral, cognitive, and motor symptoms
4

Sampson, Elizabeth Lesley. "Longitudinal studies in frontotemporal dementia." Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406422.

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5

Rascovsky, Katya. "Neuropsychological aspects of frontotemporal dementia /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2005. http://wwwlib.umi.com/cr/ucsd/fullcit?p3167837.

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6

Peters, Frédéric, Daniela Perani, Karl Herholz, Vjera Holthoff, Bettina Beuthien-Baumann, Sandro Sorbi, Alberto Pupi, et al. "Orbitofrontal Dysfunction Related to Both Apathy and Disinhibition in Frontotemporal Dementia." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135825.

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Orbitofrontal metabolic impairment is characteristic of the frontal variant of frontotemporal dementia (fv-FTD), as are early changes in emotional and social conduct. Two main types of behavioral disturbances have been distinguished in fv-FTD patients: apathetic and disinhibited manifestations. In this study, we searched for relationships between brain metabolism and presence of apathetic or disinhibited behavior. Metabolic activity and behavioral data were collected in 41 fv-FTD patients from European PET centers. A conjunction analysis of the PET data showed an expected impairment of metabolic activity in the anterior cingulate, ventromedial and orbital prefrontal cortex, the dorsolateral prefrontal cortex and the left anterior insula in fv-FTD subjects compared to matched controls. A correlation was observed between disinhibition scores on the Neuropsychiatric Inventory scale and a cluster of voxels located in the posterior orbitofrontal cortex (6, 28, –24). Comparison of brain activity between apathetic and nonapathetic fv-FTD patients from two centers also revealed a specific involvement of the posterior orbitofrontal cortex in apathetic subjects (4, 22, –22). The results confirm that the main cerebral metabolic impairment in fv-FTD patients affects areas specializing in emotional evaluation and demonstrate that decreased orbitofrontal activity is related to both disinhibited and apathetic syndromes in fv-FTD
Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich
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Graham, A. J. "Learning and memory in frontotemporal dementia." Thesis, University of Cambridge, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.599590.

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Lindau, Maria. "Clinical differentiation between frontotemporal dementia and Alzheimer's disease : psychometric, behavioral, neuroimaging and neurophysiological information /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-430-5/.

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Vial, Eric. "La démence frontotemporale : analyse clinique de 14 observations." Montpellier 1, 2000. http://www.theses.fr/2000MON11173.

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Yancopoulou, Despina. "Neuropathological and genetic studies on frontotemporal dementia." Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.614974.

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Books on the topic "Demenza frontotemporale":

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R, Hodges John, ed. Frontotemporal dementia syndromes. Cambridge: Cambridge University Press, 2007.

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R, Hodges John, ed. Frontotemporal dementia syndromes. Cambridge: Cambridge University Press, 2007.

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F, Lebert, and Pasquier F, eds. Frontotemporal dementia. Dordrecht: ICG Publications, 1996.

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Dickerson, Bradford C., ed. Hodges' Frontotemporal Dementia. Cambridge: Cambridge University Press, 2016. http://dx.doi.org/10.1017/cbo9781316091586.

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Hodges, John R., ed. Frontotemporal Dementia Syndromes. Cambridge: Cambridge University Press, 2007. http://dx.doi.org/10.1017/cbo9781316135457.

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Roberson, Erik D., ed. Alzheimer's Disease and Frontotemporal Dementia. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-744-0.

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Roberson, Erik D. Alzheimer's disease and frontotemporal dementia: Methods and protocols. New York: Humana Press, 2011.

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Aging, National Institute on. Frontotemporal disorders: Information for patients, families, and caregivers. Bethesda, Md.]: National Institutes of Health, National Institute on Aging, 2010.

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Mates, Andrea W. Language, interaction and frontotemporal dementia: Reverse engineering the social mind. London: Equinox Pub., 2010.

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W, Mates Andrea, Mikesell Lisa, and Smith Michael Sean, eds. Language, interaction and frontotemporal dementia: Reverse engineering the social mind. Oakville, CT: Equinox Pub. Ltd., 2010.

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Book chapters on the topic "Demenza frontotemporale":

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Witt, K. "Frontotemporale Demenz Demenz frontotemporale en." In Gedächtnisstörungen, 204–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-36993-3_15.

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Dinand, Claudia, Martin Berwig, and Margareta Halek. "Menschen mit Frontotemporaler Demenz: Versorgungsbedarfe und Interventionen." In Pflege-Report 2022, 155–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-65204-6_10.

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ZusammenfassungDie Frontotemporale Demenz ist eine von sehr unterschiedlichen früh beginnenden und seltenen Demenzformen, die die betroffenen Menschen und ihre Familien oft unerwartet und radikal vor große, das Leben verändernde Herausforderungen stellt. Trotz zunehmender Forschungsaktivitäten und Aufmerksamkeit in der Fachöffentlichkeit für die Lebenssituation von Menschen mit Demenz jenseits der Alzheimer-Symptomatik gibt es eine Reihe von Versorgungslücken, die es zu schließen gilt. Eine davon ist die Unterstützung und Beratung von Angehörigen in der Bewältigung des gemeinsamen Alltags. Am Beispiel der Machbarkeitsstudie AMEO-FTD wird vorgestellt, welche Potenziale Videofeedback für Menschen mit der verhaltensbetonten Variante der Frontotemporalen Demenz und ihre Bezugspersonen für den Aufbau einer gelingenden Interaktions- und Beziehungsgestaltung haben kann. Anschließend werden literaturbasiert weitere Empfehlungen für Forschung und Praxis gegeben.
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Wider, Christian W., Barbara Jasinska-Myga, Takuya Konno, and Zbigniew K. Wszolek. "Frontotemporal Dementia." In Neurodegeneration, 115–25. Oxford, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781118661895.ch12.

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Pasquier, Florence, Thibaud Lebouvier, and Florence Lebert. "Frontotemporal Dementia." In Neuropsychiatric Symptoms of Cognitive Impairment and Dementia, 279–302. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39138-0_13.

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Doherty, Colin P., Siobhan Hutchinson, Sharon Abrahams, and Robert F. Coen. "Frontotemporal Dementia." In Neurodegenerative Disorders, 115–42. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84996-011-3_6.

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Perry, David C., and Howard J. Rosen. "Frontotemporal dementia." In Non-Alzheimer's and Atypical Dementia, 49–63. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118797662.ch5.

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Léger, Gabriel C. "Frontotemporal Dementia." In Neuro-Geriatrics, 103–24. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56484-5_9.

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Boxer, Adam L. "Frontotemporal Dementia." In The Handbook of Alzheimer's Disease and Other Dementias, 145–78. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444344110.ch4.

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Rascovsky, Katya, and Diana L. Matallana. "Frontotemporal dementia." In International Neurology, 153–56. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118777329.ch43.

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Gustafson, L., and A. Brun. "Frontotemporal Dementia." In Dementias, 151–71. Milano: Springer Milan, 1999. http://dx.doi.org/10.1007/978-88-470-2149-5_7.

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Conference papers on the topic "Demenza frontotemporale":

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MILLER, BRUCE L. "FRONTOTEMPORAL DEMENTIA (FTD): INSIGHTS INTO WISDOM AND ALTRUISM." In Proceedings of the 45th Session of the International Seminars on Nuclear War and Planetary Emergencies. WORLD SCIENTIFIC, 2013. http://dx.doi.org/10.1142/9789814531788_0035.

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Souza, Leonardo de, Maxime Bertoux, Luciano Mariano, Elisa Resende, Antônio Lúcio Teixeira, Francisco Cardoso, Sarah Camargos, Vítor Tumas, and Paulo Caramelli. "MENTALIZING IN FRONTOTEMPORAL DEMENTIA AND PROGRESSIVE SUPRANUCLEAR PALSY." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda015.

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Background: Mentalizing and emotion recognition are impaired in behavioral variant frontotemporal dementia (bvFTD). It is not clear whether these abilities are disturbed in progressive supranuclear palsy (PSP). Objective: To investigate social cognition (SC) between bvFTD and PSP. The neural basis of SC in PSP and bvFTD groups were also investigated by neuroimaging. Methods: Data from the notification sheet were collected and patients were classified according to current clinical and pathological criteria. Results: Groups did not differ on age, schooling and sex. Compared to controls, bvFTD and PSP patients had reduced scores in all tests of SC. bvFTD and PSP did not differ on measures of SC. PSP and bvFTD had cerebral atrophy in critical regions for SC. The cortical correlates of emotion recognition overlapped in bvFTD and PSP, correlating with frontal medial cortex, insula and limbic structures. PSP and bvFTD patients also displayed similar patterns of brain correlations (anterior temporal lobes) for social norms. The neural correlates of mentalizing were associated with frontal and temporal poles bilaterally, in both bvFTD and PSP. Conclusion:PSP patients exhibit impairment in mentalizing. PSP and bvFTD share clinical, cognitive and neuroimaging features.
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Kumari, R. S., P. S. Mathuranath, T. Varghese, and C. Kesavdas. "Segmentation of mr brain images using FCM technique in frontotemporal dementia." In IET Chennai 3rd International Conference on Sustainable Energy and Intelligent Systems (SEISCON 2012). Institution of Engineering and Technology, 2012. http://dx.doi.org/10.1049/cp.2012.2191.

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Jaeger, Antônio, Eduarda Carreira, Natália Gama, Paulo Caramelli, and Leonardo Souza. "CONTROLLED EPISODIC RETRIEVAL IN BEHAVIORAL VARIANT FRONTOTEMPORAL DEMENTIA AND ALZHEIMER DISEASE." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda012.

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Background: Recent studies showed that episodic memory is impaired in behavioral variant of Frontotemporal dementia (bvFTD), but in contrast to Alzheimer disease (AD) it is assumed to be caused primarily by deficits in executive control. Objective: Our goal was to probe this possibility by testing bvFTD and AD patients in a source memory task which manipulated executive control. Methods: We assessed 14 healthy controls (HC), 20 bvFTD patients, and 18 AD patients in a source memory task for spatial location in which objects were first seen at the left or right side of the screen, and at test in the center of the screen, when participants were asked to indicate in which side of the screen each object was studied. Importantly, at test, predictive arrow cues (66.7% valid/33.3% invalid) indicated the likely prior location of each object. Results: BvFTD and AD patients showed indistinguishable overall memory performances, although both showed significantly poorer performances than HC. Furthermore, although both HC and bvFTD participants had their memory judgments affected by cueing, showing poorer memory accuracy after invalid than after valid cues, AD patients showed equivalent performance for both cue types. Conclusion: The current findings support the notion that episodic memory is impaired in bvFTD, and suggests that such impairment can be as severe as in AD. The cause of this impairment, however, was not related to the executive dysfunctions manipulated in the current source memory task, but rather to further mechanisms in the bvFTD memory deficits.
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Dias, Adriano Braga, Vitória Nathaly Espindula de Morais Souza, Jaqueline Pereira Lima, Rafaela Marçal Dobri, and Luan Felipo Botelho. "FATORES DE RISCO PARA DEMÊNCIA LOBAR FRONTOTEMPORAL: REVISÃO DE LITERATURA." In I Congresso Brasileiro de Estudos Patológicos On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/conbesp/55.

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Introdução: A Degeneração Lobar Frontotemporal (DLFT) pode proporcionar um espectro de distúrbios neurodegenerativos com apresentação clínica, patologia e anormalidades genéticas heterogêneas que culminam na atrofia dos lobos frontal e/ou temporal. É considerada uma das causas mais comuns de demência antes dos 65 anos, responsável por até 20% dos casos no pré-senil. Nesse contexto, o peso da genética, bem como fatores ambientais podem estar relacionados ao acometimento de tal patologia. Objetivo: Analisar e descrever através de uma pesquisa bibliográfica os principais fatores de risco para o desenvolvimento da DLFT. Material e métodos: Trata-se de uma revisão de literatura, em que os sites de pesquisa científica “PubMed” e “SciELO” foram as plataformas de busca. Os descritores para a pesquisa foram “frontotemporal dementia” e “frontotemporal dementia and risk factors”, sendo encontrados 1.974 no PubMed e 86 no SciELO, todos gratuitos, completos e entre os anos 2018 e 2022. Para escolha dos artigos, foram utilizados como critérios: ano mais recente e artigo científico que mais se adequa à temática abordada para fins de análise comparativa, a partir do resumo. Assim, utilizou-se 14 artigos do PubMed e 3 do SciELO. Resultados: Pode-se dividir os principais fatores de risco para DLFT em fortes e fracos: os fortes estão ligados a hereditariedade, como mutações genéticas na proteína tau (MAPT), progranulina (GRN) e associadas à expansão de hexanucleotídeos em C9orf72. No entanto, formas mais raras de DLFT têm sido associadas a mutações na proteína contendo valosina (VCP), CHMP2B, ubiquilina 1 (UBQLN1), optineurina (OPTN) e sequestossoma 1 (SQSTM1). Os fatores considerados fracos são os com pouca comprovação científica (como fatores ambientais). Desses, um artigo aponta o diabetes, três colocam o traumatismo cranioencefálico e doenças autoimunes como fatores contribuintes para o risco de desenvolver a DLFT. Conclusão: Dado isso, a análise de literatura recente mostrou que a genética continua sendo o fator de risco mais favorável para doenças neurodegenerativas. Além disso, fatores ambientais e relacionados a traumatismos cranianos também possuem associação com a patologia, mas sem muita comprovação ainda. Sendo assim, não existe conhecimento suficiente para promover mudanças no estilo de vida para prevenir a DLFT em nível populacional.
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Silva, Thais Lima, Tiago Ordonez, Allan Bregola, Valéria Bahia, Mario Cecchini, Henrique Guimarães, Leandro Gambogi, et al. "NEUROPSYCHIATRIC SYMPTOMS IN BEHAVIORAL VARIANT FRONTOTEMPORAL DEMENTIA AND ALZHEIMER’S DISEASE: A 12-MONTH FOLLOW-UP STUDY." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda094.

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Background: Neuropsychiatric symptoms in patients with frontotemporal dementia (FTD) are highly prevalent and may complicate clinical managements. Objective: We tested whether the Neuropsychiatry Inventory (NPI) could detect change in neuropsychiatric symptoms and caregiver´s distress in patients diagnosed with behavioral variant frontotemporal dementia (bvFTD) and Alzheimer’s disease (AD) from baseline to a 12-month follow-up. Methods: The sample consisted of 31 patients diagnosed with bvFTD and 28 patients with AD and their caregivers. A sociodemographic questionnaire, the Mini-Mental State Examination (MMSE), Addenbrooke´s Cognitive Examination Revised (ACE-R), the INECO Frontal Screening (IFS), the Frontal Assessment Battery (FAB), the Executive Interview (EXIT-25) and the Neuropsychiatric Inventory (NPI) were used. Results: Neuropsychiatric symptoms total (NPI Total) and the caregiver Distress score were statistically higher among bvFTD patients at both assessment points. MMSE, ACE-R scores significantly declined and NPI Total and Distress scores significantly increased in both groups. Age was the only independent predictor variable for the NPI Total score in the bvFTD group in the follow up. In the AD group, ACE-R and EXIT-25, conjunctively, were associated with the NPI total score at follow up. Conclusion: Knowing how symptoms evolve over the course of the disease could help the clinician and the caregiver in decisions regarding future management and therapeutic approaches.
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SPILLANTINI, MARIA GRAZIA. "TAU GENE MUTATIONS IN FRONTOTEMPORAL DEMENTIA AND PARKINSONISM LINKED TO CHROMOSOME 17." In Proceedings of the International Seminar on Nuclear War and Planetary Emergencies — 27th Session. WORLD SCIENTIFIC, 2003. http://dx.doi.org/10.1142/9789812705150_0071.

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Horn, Jean-francois, Marie-odile Habert, Alain Giron, and Bernard Fertil. "ALZHEIMER'S DISEASE AND FRONTOTEMPORAL DEMENTIA DIFFERENTIAL AUTOMATIC DIAGNOSIS BASED ON SPECT IMAGES." In 2007 4th IEEE International Symposium on Biomedical Imaging: From Nano to Macro. IEEE, 2007. http://dx.doi.org/10.1109/isbi.2007.357107.

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Mariano, Luciano, Leonardo de Souza, Paulo Caramelli, Antonio Teixeira Júnior, Henrique Guimarães, Elisa Resende, Leandro Gambogi, Vítor Tumas, Francisco Cardoso, and Sarah Camargos. "APATHY IN FRONTOTEMPORAL DEMENTIA AND PROGRESSIVE SUPRANUCLEAR PALSY: AN EXPLORATORY NEUROIMAGING INVESTIGATION." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda018.

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Background: Apathy is a multidimensional syndrome that impairs motivation and mechanisms of behavioural regulation. As apathy is almost ubiquitous in neurodegenerative disorders, understanding the neural basis is required to expand knowledge and ameliorate our instruments. Objective: Verify the feasibility, safety, and adherence of a remote physical and cognitive exercise protocol for elders; and to compare two different protocols and its effects on strength, cognitive functions and well-being. Methods: Use of the PICO strategy, conducted on September 17, on PubMed using “Alzheimer disease” and “diabetes mellitus” as descriptors, identifying 14 articles, selecting 4 after screening. Inclusion criteria: clinical and randomized controlled trials with diabetic and Alzheimer patients, published on the last 5 years. Exclusion criteria: articles focused on medications. Results: Overall adherence to the proposed sessions was 82,36% (sd16,8) in IG and 76,5% (sd24,04) in CG. There were no serious adverse events or drop-outs during the study. There was an improvement in strength and verbal fluency for IG and GDS for both groups (p ≤ 0,05). Conclusion: Both protocols seem a feasible program, reaching an acceptable level of adherence and safety. The technology used can represent a sustainable path for large scale use to promote aging active in developing countries.
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Tiepolt, S., M. Rullmann, M. Patt, TH Jochimsen, KT Hoffmann, ML Schroeter, O. Sabri, and H. Barthel. "Quantitative Susceptibility Mapping (QSM) am PET/MRT bei Patienten mit behavioraler frontotemporaler Demenz (bvFTD)." In NuklearMedizin 2021 – digital. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1726751.

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Reports on the topic "Demenza frontotemporale":

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Wei, Huijie, Xin Mu, Yu Li, Hua Lei, De Yang, Tian Li, and Junwei Ren. Meta-analysis of the association between CHCHD10 Pro34Ser variant and the risk of frontotemporal dementia. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2021. http://dx.doi.org/10.37766/inplasy2021.5.0090.

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To the bibliography