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Books on the topic 'Dendrite development'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Levinson, Laurie H. Flight software development for the Isothermal Dendritic Growth Experiment. National Aeronautics and Space Administration, 1990.

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2

Levinson, Laurie H. Flight software development for the Isothermal Dendritic Growth Experiment. National Aeronautics and Space Administration, 1990.

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3

Bohnenkamp, Hermann Richard. Bioprocess development for the generation of monocycte-derived dendritic cells: Applicability in breast cancer immunotherapy. Forschungszentrum Jülich GmbH, Institut für Biotechnologie, 2004.

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4

Kapfhammer, Josef P. Cellular and molecular control of dendritic growth and development of cerebellar Purkinje cells. Elsevier, 2004.

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5

Voorbij, H. A. M. Dendritic cells and the development of thyroid autoimmune disease and type 1 diabetes mellitus. Thesis, 1989.

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6

Huang, Eric, Kazuo Emoto, Rachel Wong, and Casper Hoogenraad. Dendrites: Development and Disease. Springer, 2018.

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7

Tate, Marcia L. Shouting - Won't Grow Dendrites: A Multimedia Kit for Professional Development. Corwin Press, 2008.

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8

Advanced dendritic web growth development and development of single-crystal silicon dendritic ribbon and high-efficiency solar cell program: Final report. Westinghouse R&D Center, 1986.

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9

Stashwick, Caitlin, Brian J. Czerniecki, and Janos L. Tanyi. Dendritic Cell Vaccine Therapy for Ovarian Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0008.

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Dendritic cell vaccine therapy has emerged as an exciting new field in immunotherapy in ovarian cancer over the past two decades. This chapter reviews the biology of dendritic cells, including dendritic cell subsets, development, activation, and maturation as well as strategies for clinical use of dendritic cell vaccines. While there is encouraging data in preclinical work for dendritic cell vaccine, the outcomes of clinical trials have not shown robust responses. A summary of the clinical trials using dendritic cell vaccines in ovarian cancer is reviewed. Treatment-related toxicities and potential therapies to increase clinical efficacy are also discussed.
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10

Reading And Language Arts Worksheets Dont Grow Dendrites A Multimedia Kit For Professional Development. Corwin Press, 2010.

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11

E, Rosenberger Franz, and United States. National Aeronautics and Space Administration., eds. X-ray transmission microscope development. Center for Microgravity and Materials Research, The University of Alabama in Huntsville, 1995.

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12

Becker, Y. Skin Langerhans (Dendritic) Cells in Virus Infections and AIDS (Developments in Medical Virology). Springer, 1990.

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13

E, Rosenberger Franz, and United States. National Aeronautics and Space Administration., eds. X-ray transmission microscope development: Third semi-annual progress report, NASA contract NAS8-40185. Center for Microgravity and Materials Research, The University of Alabama in Huntsville, 1995.

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14

X-ray transmission microscope development: Final report; NASA contract NAS8-40185; period of performance, 02/28/95-08/31/97. Center for Microgravity and Materials Research, University of Alabama in Huntsville, 1997.

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15

United States. National Aeronautics and Space Administration., ed. X-ray transmission microscope development: Final report; NASA contract NAS8-40185; period of performance, 02/28/95-08/31/97. Center for Microgravity and Materials Research, University of Alabama in Huntsville, 1997.

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16

United States. National Aeronautics and Space Administration., ed. X-ray transmission microscope development: Final report; NASA contract NAS8-40185; period of performance, 02/28/95-08/31/97. Center for Microgravity and Materials Research, University of Alabama in Huntsville, 1997.

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17

Franz, Rosenberger, and United States. National Aeronautics and Space Administration., eds. X-ray transmission microscope development: Third semi-annual progress report, NASA contract NAS8-40185. Center for Microgravity and Materials Research, The University of Alabama in Huntsville, 1995.

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18

Franz, Rosenberger, and United States. National Aeronautics and Space Administration., eds. X-ray transmission microscope development: Third semi-annual progress report, NASA contract NAS8-40185. Center for Microgravity and Materials Research, The University of Alabama in Huntsville, 1995.

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19

van der Vlag, Johan, and Jo H. M. Berden. The patient with systemic lupus erythematosus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0161.

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Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with various clinical manifestations. The hallmark of SLE is the presence of antibodies against nuclear constituents, such as double-stranded (ds)DNA, histones, and nucleosomes. Local deposition of antinuclear antibodies in complex with nuclear autoantigens induces serious inflammatory conditions that can affect several tissues and organs, including the kidney.The levels of antinucleosome and anti-dsDNA antibodies seem to correlate with glomerulonephritis and these autoantibodies can often be detected years before the patient is diagnosed with SLE. Apoptotic debris is present in the extracellular matrix and circulation of patients with SLE due to an aberrant process of apoptosis and/or insufficient clearance of apoptotic cells and apoptotic debris. The non-cleared apoptotic debris in patients with SLE may lead to activation of both the innate (myeloid and plasmacytoid dendritic cells) and adaptive (T and B cells) immune system. In addition to the activation by apoptotic debris and immune complexes, the immune system in SLE may be deregulated at the level of (a) presentation of self-peptides by antigen-presenting cells, (b) selection processes for both B and T cells, and (c) regulatory processes of B- and T-cell responses. Lupus nephritis may be classified in different classes based on histological findings in renal biopsies. The chromatin-containing immune complexes deposit in the capillary filter, most likely due to the interaction of chromatin with the polysaccharide heparan sulphate. A decreased renal expression of the endonuclease DNaseI further contributes to the glomerular persistence of chromatin and the development of glomerulonephritis.Current treatment of lupus nephritis is not specific and aims to reduce the inflammatory response with general immunosuppressive therapies. However, research has revealed novel potential therapeutic candidates at the level of dendritic cells, B cells, and T cells.
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20

Stuart, Philip E., Lam C. Tsoi, Caely A. Hambro, and James T. Elder. Genetics of psoriasis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0005.

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Psoriasis is an immune-mediated inflammatory disease (IMID) characterized by skin inflammation, epidermal hyperplasia, increased risk of arthritis, and cardiovascular morbidity. Substantial evidence indicates that psoriasis is driven by abnormal interactions between cells of the innate and adaptive host defence systems, including keratinocytes, dendritic cells, and T-cells, resulting in a dysregulated immune response and markedly increased epidermal proliferation. The precise aetiology of psoriasis remains unknown. Here, we review how innate and adaptive host defence responses are regulated by genetic factors that modulate the overall risk of psoriasis and dictate whether the disease affects the skin and/or the joints. Specifically, we review the epidemiologic basis for considering psoriasis as a genodermatosis, summarize knowledge derived from linkage and association studies of cutaneous psoriasis (PsC) and psoriatic arthritis (PsA), and attempt to relate genetic and immunologic discoveries in a pathogenetic framework that may eventually allow prediction of the development of PsA in psoriatic individuals.
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21

X-ray transmission microscope development: Final report; NASA contract NAS8-40185; period of performance, 02/28/95-08/31/97. Center for Microgravity and Materials Research, University of Alabama in Huntsville, 1997.

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