Relevant bibliographies by topics / Determinants of myocardial oxygen demand

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Academic literature on the topic 'Determinants of myocardial oxygen demand'

Author: Grafiati
Published: 4 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "Determinants of myocardial oxygen demand"

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Rishu, Segan, Walia Lily, and Mittal Naveen. "EFFECT OF DIABETIC AUTONOMIC NEUROPATHY ON DETERMINANTS OF MYOCARDIAL OXYGEN DEMAND." International Journal of Basic & Applied Physiology 3, no. 1 (2014): 62–66. https://doi.org/10.5281/zenodo.4475995.

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Background: Resting myocardial oxygen demand determinants are less studied in diabetic autonomic neuropathy patients. Objective: An insight into the determinants of myocardial oxygen demand in diabetic autonomic neuropathy patients may explain sudden adverse cardiovascular events in such patients. Methods: Case control study (n=60) was done on forty age matched (40-60 yr) type 2 diabetics with/ without diabetic cardiac autonomic neuropathy and twenty controls for myocardial oxygen demand determinants (heart rate, rate pressure product, systolic blood pressure) at rest. Results: Results demonst
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AKINBOBOYE, O. "Determinants of resting myocardial oxygen demand in the hypertrophied myocardium." American Journal of Hypertension 15, no. 4 (2002): A167. http://dx.doi.org/10.1016/s0895-7061(02)02728-0.

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Deedwania, Prakash C. "Transient myocardial ischemia and its relation to determinants of myocardial oxygen demand." American Journal of Cardiology 66, no. 16 (1990): G25—G27. http://dx.doi.org/10.1016/0002-9149(90)90390-m.

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Schwid, Howard A., Charles W. Buffington, and David P. Strum. "Computer simulation of the hemodynamic determinants of myocardial oxygen supply and demand." Journal of Cardiothoracic Anesthesia 4, no. 1 (1990): 5–18. http://dx.doi.org/10.1016/0888-6296(90)90440-q.

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Asha, Shrivastava, Wasnik Sanjay, and Dave Rashmi. "Correlation Of Glycemic Status With Indicators Of Myocardial Oxygen Usage." International Journal of Basic and Applied Physiology 2, no. 1 (2013): 41–46. https://doi.org/10.5281/zenodo.4479178.

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Present study aimed to investigate the correlation between poor glycemic control determined by glycosylated haemoglobin( A1C) and myocardial oxygen demand. Method: Case-control study comprised of three groups of 50 each age matched (30-45 yrs) normoglycemics, prediadetics and type 2 diabetic mellitus (T2DM) as per American Diabetic Association 2011 (ADA) criteria. The haemodynamic determinants of myocardial oxygen demand measured were heart rate(HR), systolic blood pressure(SBP) and rate pressure product(RPP) Result: The observations revealed significant differences in the fasting plasma gluco
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Pepine, C. J., and W. W. Nichols. "Effects of transient increase in intrathoracic pressure on hemodynamic determinants of myocardial oxygen supply and demand." Clinical Cardiology 11, no. 12 (1988): 831–37. http://dx.doi.org/10.1002/clc.4960111207.

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Chou, Alan F. "Failure of intravenous pindolol to reduce the hemodynamic determinants of myocardial oxygen demand or enzymatically determined infarct size in acute myocardial infarction." Journal of Emergency Medicine 4, no. 5 (1986): 420. http://dx.doi.org/10.1016/0736-4679(86)90232-5.

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Foo, K., N. Sekhri, C. Knight, et al. "The effect of diabetes on heart rate and other determinants of myocardial oxygen demand in acute coronary syndromes." Diabetic Medicine 21, no. 9 (2004): 1025–31. http://dx.doi.org/10.1111/j.1464-5491.2004.01413.x.

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Wieneke, Heinrich, Clemens von Birgelen, Michael Haude, et al. "Determinants of coronary blood flow in humans: quantification by intracoronary Doppler and ultrasound." Journal of Applied Physiology 98, no. 3 (2005): 1076–82. http://dx.doi.org/10.1152/japplphysiol.00724.2004.

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The direct determinants of coronary flow are lumen area and blood flow velocity; however, the precise mechanisms that control these factors are not fully understood. The aim of the present study was to assess by which mechanisms lumen area and coronary flow velocity interact with hemodynamic and morphometric factors, thereby influencing coronary flow. Intracoronary Doppler and ultrasound measurements were performed in 28 patients without coronary lumen irregularities. Flow velocity and lumen cross-sectional area were measured in the proximal segments of all three coronary arteries. Global lume
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Duncker, Dirk J., and Robert J. Bache. "Regulation of Coronary Blood Flow During Exercise." Physiological Reviews 88, no. 3 (2008): 1009–86. http://dx.doi.org/10.1152/physrev.00045.2006.

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Exercise is the most important physiological stimulus for increased myocardial oxygen demand. The requirement of exercising muscle for increased blood flow necessitates an increase in cardiac output that results in increases in the three main determinants of myocardial oxygen demand: heart rate, myocardial contractility, and ventricular work. The approximately sixfold increase in oxygen demands of the left ventricle during heavy exercise is met principally by augmenting coronary blood flow (∼5-fold), as hemoglobin concentration and oxygen extraction (which is already 70–80% at rest) increase o
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Dissertations / Theses on the topic "Determinants of myocardial oxygen demand"

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Lee, Shang Chiun. "Adenosine and Down-Regulation of Myocardial Oxygen Demand." Thesis, University of North Texas, 1991. https://digital.library.unt.edu/ark:/67531/metadc332565/.

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This investigation studied the physiological means by which myocardium can survive and function properly when oxygen supply is limited and cannot initially match oxygen demand. The effects of isoproterenol (ISO) stimulations during low coronary perfusion pressure or hypoxemia on myocardial oxygen demand, work, and oxygen utilization efficiency were investigated.
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Docherty, Annemarie Beth. "Myocardial injury in critically ill patients with co-existing cardiovascular disease." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31247.

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Approximately 30% of people admitted to ICU in the UK have co-existing cardiovascular disease (CVD), and this may rise as life-expectancy increases. Patients with CVD have impaired compensatory mechanisms to enable maximum oxygen delivery to the tissues in the event of critical illness, which itself increases global oxygen demand, further stressing the heart. This is exacerbated by tachycardia and hypotension, which may relatively reduce blood flow to the coronary arteries, and catecholamines which increase myocardial oxygen demand. The myocardium extracts 75% of the oxygen supplied by the cor
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Van, Geluwe Scott Patrick. "Effects of arbutamine stress testing compared to treadmill exercise stress testing on electrocardiographic responses of ischemia and myocardial oxygen demand." Thesis, This resource online, 1995. http://scholar.lib.vt.edu/theses/available/etd-01102009-063427/.

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Books on the topic "Determinants of myocardial oxygen demand"

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Bennett, Jeremy, and Kara Siegrist. Myocardial Ischemia. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0005.

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Coronary artery disease is a prevalent and growing problem in the United States leading to significant morbidity and mortality including myocardial ischemia and infarction. Diagnosis and treatment of myocardial ischemia under general anesthesia can present unique challenges for the anesthesiologist including interpretation of diagnostic monitoring data and options for therapeutic interventions. There are many complex factors that determine myocardial oxygen supply and demand; when these become imbalanced, myocardial ischemia occurs that can progress to infarction. Maintaining a high-degree of
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Evans, Rhys. Cardiac surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198719410.003.0014.

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This chapter discusses the anaesthetic management of cardiac surgery. It begins with a description of myocardial oxygen supply and demand, risk scoring for cardiac surgery, and cardiopulmonary bypass. Surgical procedures covered include coronary artery bypass grafting (CABG) (including emergency and redo CABG), aortic valve replacement (including transcatheter aortic valve implantation), mitral valve replacement, thoracic aortic surgery, pulmonary thromboembolectomy, cardioversion, and implantation of a cardioverter-defibrillator.
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Evans, Rhys. Cardiac surgery. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198719410.003.0014_update_001.

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This chapter discusses the anaesthetic management of cardiac surgery. It begins with a description of myocardial oxygen supply and demand, risk scoring for cardiac surgery, and cardiopulmonary bypass. Surgical procedures covered include coronary artery bypass grafting (CABG) (including emergency and redo CABG), aortic valve replacement (including transcatheter aortic valve implantation), mitral valve replacement, thoracic aortic surgery, pulmonary thromboembolectomy, cardioversion, and implantation of a cardioverter-defibrillator.
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van Lier, Felix, and Robert Jan Stolker. Preoperative assessment and optimization. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0040.

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Perioperative cardiovascular complications (including myocardial ischaemia and myocardial infarction) are the predominant cause of morbidity and mortality in patients undergoing non-cardiac surgery. The pathophysiology of perioperative myocardial infarction is complex. Prolonged myocardial ischaemia due to the stress of surgery in the presence of a haemodynamically significant coronary lesion, leading to subendocardial ischaemia, and acute coronary artery occlusion after plaque rupture and thrombus formation contribute equally to these devastating events. Perioperative management aims at optim
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Combes, Alain, and Nicolas Bréchot. Intra-aortic balloon counterpulsation in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0153.

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The intra-aortic balloon pump (IABP) is a mechanical device consisting of a cylindrical polyethylene balloon that sits in the aorta, approximately 2 cm from the left subclavian artery. A computer-controlled console linked to either an electrocardiogramor a pressure transducer inflates the balloon with helium during diastole (counterpulsation) and actively deflates in systole. This results in an increase in coronary artery blood flow and cardiac output, and reduced left ventricular afterload. These actions combine to decrease myocardial oxygen demand and increase supply. Major complications inc
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Book chapters on the topic "Determinants of myocardial oxygen demand"

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Suga, Hiroyuki, Yoshio Yasumura, Takashi Nozawa, Shiho Futaki, Nobuaki Tanaka, and Masaaki Uenishi. "Determinants of Myocardial Oxygen Demand: Total Mechanical Energy and Contractility." In Microcirculation in Circulatory Disorders. Springer Japan, 1988. http://dx.doi.org/10.1007/978-4-431-68078-9_73.

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Bronicki, Ronald A., and Daniel J. Penny. "Determinants of Myocardial Oxygen Consumption and Delivery." In Simulation in Cardiovascular Critical Care Medicine. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-63557-1_4.

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Suga, Hiroyuki, Yoshio Yasumura, Takashi Nozawa, Shiho Futaki, Nobuaki Tanaka, and Masaaki Uenishi. "Ventricular Systolic Pressure-Volume Area (PVA) and Contractile State (Emax) Determine Myocardial Oxygen Demand." In Oxygen Transport to Tissue X. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4615-9510-6_50.

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Kelly, D. T., S. Ben Freedman, G. S. Roubin, et al. "Nifedipine and Nitroglycerin: A Comparison of Acute Effects on Hemodynamic Determinants of Myocardial Oxygen Consumption and Supply During Exertional Angina." In Mononitrate II. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72689-7_12.

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Salvi, Lucia, and Paolo Salvi. "Hemodynamic determinants of myocardial oxygen demand and supply." In Textbook of Arterial Stiffness and Pulsatile Hemodynamics in Health and Disease. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-323-91391-1.00018-2.

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Woodworth, Glenn E. "Myocardial Oxygen Supply and Demand." In Anesthesiology Review. Elsevier, 2002. http://dx.doi.org/10.1016/b978-0-443-06601-6.50025-6.

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Tobes, Daniel, and Larry Manders. "Myocardial Oxygen Utilization/Consumption." In Basic Anesthesia Review, edited by Alaa Abd-Elsayed. Oxford University PressNew York, 2024. http://dx.doi.org/10.1093/med/9780197584569.003.0262.

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Abstract Myocardial O2 supply and demand exist at all times in a fragile balance. Situations where demand exceeds supply lead to myocardial ischemia. The heart has a relatively high O2 consumption (MVO2) compared with other organs due to its workload. MVO2 can vary greatly based on the current workload of the heart; MVO2 during heavy exercise exceeds that of any other organ in the body. This chapter will summarize myocardial oxygen utilization and consumptions as it relates to ability pump blood and transport oxygen to the rest of our body.
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Crystal, George J. "Myocardial Oxygen Supply--Demand Relations during Isovolemic Hemodilution." In Advances in Pharmacology. Elsevier, 1994. http://dx.doi.org/10.1016/s1054-3589(08)60623-7.

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van Beek-Harmsen, B. J., H. M., and W. J. van der Laarse. "Inadequate Myocardial Oxygen Supply/Demand in Experimental Pulmonary Hypertension." In Pulmonary Hypertension - From Bench Research to Clinical Challenges. InTech, 2011. http://dx.doi.org/10.5772/27192.

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Murphy, Joseph G., and Abhiram Prasad. "Coronary Artery Physiology, Endothelial Dysfunction, and Intracoronary Imaging." In Mayo Clinic Cardiology, 5th ed., edited by Joseph G. Murphy, Nandan S. Anavekar, Barry A. Boilson, Margaret A. Lloyd, Rekha Mankad, and Raymond C. Shields. Oxford University PressNew York, 2024. https://doi.org/10.1093/med/9780197599532.003.0100.

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Abstract The function of the coronary arteries is to provide oxygen and nutrients to the myocardium. In the resting state, myocardial oxygen extraction is near the maximum and coronary sinus oxygen saturation is typically 30% or less. Because myocardial oxygen extraction is already near the maximum, myocardial V.o2 can increase only by an increase in coronary blood flow, and changes in myocardial V.o2 closely parallel changes in coronary blood flow. Important determinants of myocardial V.o2 are heart rate, inotropic state, and intramyocardial wall stress. Myocardial V.o2 can be approximated cl
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Conference papers on the topic "Determinants of myocardial oxygen demand"

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Sugimachi, M., K. Uemura, T. Kawada, T. Shishido, and K. Sunagawa. "Reduction of myocardial oxygen demand by controlling heart rate and hemodynamics simultaneously by novel circulatory model." In 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6091067.

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Nobari, Soroush, Rosaire Mongrain, Richard Leask, and Raymond Cartier. "Effect of Aortic Distensibility on Coronary Flow: A 3D FSI Model of Aortic Valve With the Inclusion of Coronary Arteries." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53487.

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Aortic stiffening and CAS are thought to affect coronary blood flow [1,2]. Pathological conditions such as aortic aneurysm, aortic wall stiffening and calcific aortic stenosis (CAS) will affect the distensibility of the aortic root and therefore the hemodynamics of the region. Reduced aortic distensibility (i.e. increased aortic stiffness) via presence of pathologies such as severe CAS results in a decrease of diastolic backflow. This reduction will cause less flow to enter the coronaries [2,3,4] and therefore reduce the amount of oxygen delivered to myocardium. This reduction of coronary flow
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Braga, Alana Dias Sampaio, Luana do Vale Martins Aguiar, Raphaela Cardoso Pinheiro da Silva, and Pedro Paulo Oliveira Carneiro. "MORBIDITY AND MORTALITY FROM ACUTE MYOCARDIAL INFARCTION IN ELDERLY PEOPLE IN BRAZIL (2018 TO 2022)." In I MED 360: Congresso Regional de Medicina. New Science Publishers, 2024. http://dx.doi.org/10.56238/360med2024-001.

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Introduction: Acute Myocardial Infarction (AMI) is an ischemic condition that causes the death of myocytes due to an imbalance between the supply and demand of oxygen to the myocardium, being the main isolated cause of death among chronic non-communicable diseases in Brazil, especially in the elderly population. Objective: The research aims to analyze the prevalence of morbidity and mortality in the elderly due to AMI, from 2018 to 2022 in Brazil. Methods: This is an ecological time series study, whose data were obtained from the Department of Information Technology of the Unified Health Syste
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Falk, E. A. "UNSTABLE ANGINA PECTORIS: PATHOLOGIC ASPECTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643711.

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Unstable angina pectoris represents a common and important manifestation of acute ischemic heart disease encompassing the broad spectrum of clinical syndromes between stable effort angina and acute myocardial infarction. This group of patientsisfar from uniform concerning underlying pathogenetic mechanisms and prognosis, but generally the risk of infarction or deathis increased during the unstable period. Most patients are presenting with new or worsening effort angina or angina at rest,and especially patients with rest anginaassociated with transient ECG changes seem to constitute a high risk
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ХИЗРИЕВ, М. Д., И. Э. АТАШЕВА, and З. О. ГАРУНОВ. "Drug therapy of patients with stable angina pectoris." In II Международная научно-практическая конференция "Преобразование современного мира: проблемы и возможности". Crossref, 2024. http://dx.doi.org/10.26118/5186.2024.80.69.007.

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Одной из значимых проблем, которые продолжают убивать людей в мировой медицинской практике, являются заболевания системы кровообращения, несмотря на постоянное совершенствование методов лечения. В 2016 году сердечно-сосудистыми заболеваниями страдали, согласно отчету Всемирной организации здравоохранения, 17,9 млн человек, и это составило 85% всех случаев, связанных с инфарктами и инсультами, закончившимися летально [1]. По данным за 2020 год, в Российской Федерации 938 536 смертей вызваны болезнями системы кровообращения, из них 54,2% приходится на ишемическую болезнь сердца. Основными фактор
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