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Journal articles on the topic 'Developmental arrest'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Schor, N. F. "Developmental Arrest." Neurology 71, no. 13 (2008): 1038. http://dx.doi.org/10.1212/01.wnl.0000326580.71352.8c.

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2

Chamiel, Elad, and Sophie D. Walsh. "“House Arrest” or “Developmental Arrest”? A Study of Youth Under House Arrest." International Journal of Offender Therapy and Comparative Criminology 62, no. 14 (2018): 4381–402. http://dx.doi.org/10.1177/0306624x18779183.

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Studies have examined the potential benefits and risks of alternative forms of detention, such as house arrest, for adults but, despite its growing use, little research has examined the implications of house arrest for juveniles. The current research examined the experience of 14 adolescents under house arrest. Six main themes were identified in the narratives of the participants: the experience of detention, daily schedule and utilization of time, emotions and self-reflection, relationships with peers, relation to parents and supervisor(s), and contact with professionals. Findings emphasized

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3

Ihashi, Shunya, Mizuto Hamanaka, Masaya Kaji, et al. "Incomplete activation ofAlyrefandGabpb1leads to preimplantation arrest in cloned mouse embryos." Life Science Alliance 6, no. 11 (2023): e202302296. http://dx.doi.org/10.26508/lsa.202302296.

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Differentiated cell nuclei can be reprogrammed after nuclear transfer (NT) to oocytes and the produced NT embryos can give rise to cloned animals. However, development of NT embryos is often hampered by recurrent reprogramming failures, including the incomplete activation of developmental genes, yet specific genes responsible for the arrest of NT embryos are not well understood. Here, we searched for developmentally important genes among the reprogramming-resistant H3K9me3-repressed genes and identifiedAlyrefandGabpb1by siRNA screening. Gene knockout ofAlyrefandGabpb1by the CRISPR/Cas9 system

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4

Pradillon, Florence, Bruce Shillito, Craig M. Young, and Françoise Gaill. "Developmental arrest in vent worm embryos." Nature 413, no. 6857 (2001): 698–99. http://dx.doi.org/10.1038/35099674.

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5

Koštál, Vladimir, and David L. Denlinger. "Dormancy and developmental arrest in invertebrates." Journal of Insect Physiology 57, no. 5 (2011): 537. http://dx.doi.org/10.1016/j.jinsphys.2011.04.001.

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6

Zhang, Wuwen, Shifeng Li, Kai Li, L. i. Li, Ping Yin, and Guoqing Tong. "The role of protein arginine methyltransferase 7 in human developmentally arrested embryos cultured in vitro." Acta Biochimica et Biophysica Sinica 53, no. 7 (2021): 925–32. http://dx.doi.org/10.1093/abbs/gmab068.

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Abstract Human embryos of in vitro fertilization (IVF) are often susceptible to developmental arrest, which greatly reduces the efficiency of IVF treatment. In recent years, it has been found that protein arginine methyltransferase 7 (PRMT7) plays an important role in the process of early embryonic development. However, not much is known about the relationship between PRMT7 and developmentally arrested embryos. The role of PRMT7 in developmentally arrested embryos was thus investigated in this study. Discarded human embryos from IVF were collected for experimental materials. Quantitative real-

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7

Galis, Frietson, Jacques J. M. van Alphen, and Johan A. J. Metz. "Digit reduction: via repatterning or developmental arrest?" Evolution and Development 4, no. 4 (2002): 249–51. http://dx.doi.org/10.1046/j.1525-142x.2002.02013.x.

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8

Footitt, Steven, and Marc Alan Cohn. "Developmental arrest: from sea urchins to seeds." Seed Science Research 11, no. 01 (2001): 3–16. http://dx.doi.org/10.1079/ssr200055.

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9

Usenko, Crystal Y., David C. Hopkins, Stephen J. Trumble, and Erica D. Bruce. "Hydroxylated PBDEs induce developmental arrest in zebrafish." Toxicology and Applied Pharmacology 262, no. 1 (2012): 43–51. http://dx.doi.org/10.1016/j.taap.2012.04.017.

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10

Page, A. W., and T. L. Orr-Weaver. "The Drosophila genes grauzone and cortex are necessary for proper female meiosis." Journal of Cell Science 109, no. 7 (1996): 1707–15. http://dx.doi.org/10.1242/jcs.109.7.1707.

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In Drosophila, normal female meiosis arrests at metaphase I. After meiotic arrest is released by egg activation, the two meiotic divisions are rapidly completed, even in unfertilized eggs. Since little is known about the regulation of the meiotic cell cycle after the meiotic arrest, we screened for mutants that arrest in meiosis. Here we describe the phenotype of eggs laid by sterile mothers mutant for either grauzone or cortex. These eggs arrest in metaphase of meiosis II, and although they can enter into an aberrant anaphase II, they never exit meiosis. Prolonged sister-chromatid cohesion is

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11

Lin, T. Y., S. Viswanathan, C. Wood, P. G. Wilson, N. Wolf, and M. T. Fuller. "Coordinate developmental control of the meiotic cell cycle and spermatid differentiation in Drosophila males." Development 122, no. 4 (1996): 1331–41. http://dx.doi.org/10.1242/dev.122.4.1331.

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Wild-type function of four Drosophila genes, spermatocyte arrest, cannonball, always early and meiosis I arrest, is required both for cell-cycle progression through the G2/M transition of meiosis I in males and for onset of spermatid differentiation. In males mutant for any one of these meiotic arrest genes, mature primary spermatocytes with partially condensed chromosomes accumulate and postmeiotic cells are lacking. The arrest in cell-cycle progression occurs prior to degradation of cyclin A protein. The block in spermatogenesis in these mutants is not simply a secondary consequence of meiot

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12

Sfakianoudis, Konstantinos, Evangelos Maziotis, Eleni Karantzali, et al. "Molecular Drivers of Developmental Arrest in the Human Preimplantation Embryo: A Systematic Review and Critical Analysis Leading to Mapping Future Research." International Journal of Molecular Sciences 22, no. 15 (2021): 8353. http://dx.doi.org/10.3390/ijms22158353.

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Developmental arrest of the preimplantation embryo is a multifactorial condition, characterized by lack of cellular division for at least 24 hours, hindering the in vitro fertilization cycle outcome. This systematic review aims to present the molecular drivers of developmental arrest, focusing on embryonic and parental factors. A systematic search in PubMed/Medline, Embase and Cochrane-Central-Database was performed in January 2021. A total of 76 studies were included. The identified embryonic factors associated with arrest included gene variations, mitochondrial DNA copy number, methylation p

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13

Fine, Alexander D., Robyn L. Ball, Yasuhiro Fujiwara, Mary Ann Handel, and Gregory W. Carter. "Uncoupling of transcriptomic and cytological differentiation in mouse spermatocytes with impaired meiosis." Molecular Biology of the Cell 30, no. 5 (2019): 717–28. http://dx.doi.org/10.1091/mbc.e18-10-0681.

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Cell differentiation is driven by changes in gene expression that manifest as changes in cellular phenotype or function. Altered cellular phenotypes, stemming from genetic mutations or other perturbations, are widely assumed to directly correspond to changes in the transcriptome and vice versa. Here, we exploited the cytologically well-defined Prdm9 mutant mouse as a model of developmental arrest to test whether parallel programs of cellular differentiation and gene expression are tightly coordinated, or can be disassociated. By comparing cytological phenotype markers and transcriptomes in wil

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14

Raz, V., J. H. Bergervoet, and M. Koornneef. "Sequential steps for developmental arrest in Arabidopsis seeds." Development 128, no. 2 (2001): 243–52. http://dx.doi.org/10.1242/dev.128.2.243.

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The continuous growth of the plant embryo is interrupted during the seed maturation processes which results in a dormant seed. The embryo continues development after germination when it grows into a seedling. The embryo growth phase starts after morphogenesis and ends when the embryo fills the seed sac. Very little is known about the processes regulating this phase. We describe mutants that affect embryo growth in two sequential developmental stages. Firstly, embryo growth arrest is regulated by the FUS3/LEC type genes, as mutations in these genes cause a continuation of growth in immature emb

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15

Hinchliffe, Stephen A., C. Vyvyan Howard, Matthew R. J. Lynch, Paul H. Sargent, Brian A. Judd, and Dick van Velzen. "Renal Developmental Arrest in Sudden Infant Death Syndrome." Pediatric Pathology 13, no. 3 (1993): 333–43. http://dx.doi.org/10.3109/15513819309048221.

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16

Schmiady, Hardi, and Heribert Kentenich. "Cytological studies of human zygotes exhibiting developmental arrest." Human Reproduction 8, no. 5 (1993): 744–51. http://dx.doi.org/10.1093/oxfordjournals.humrep.a138133.

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17

Rivinus, Timothy M. "College Age Substance Abuse as a Developmental Arrest." Journal of College Student Psychotherapy 6, no. 3-4 (1993): 141–66. http://dx.doi.org/10.1300/j035v06n03_08.

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18

Chen, Di, Kally Z. Pan, Julia E. Palter, and Pankaj Kapahi. "Longevity determined by developmental arrest genes inCaenorhabditis elegans." Aging Cell 6, no. 4 (2007): 525–33. http://dx.doi.org/10.1111/j.1474-9726.2007.00305.x.

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19

Filipović, Hrvoje, and Dunja Ilovača. "PERCEPCIJA GRAĐANA O UHIĆENJU KAO MJERI ZA OSIGURANJE PRISUTNOSTI OKRIVLJENIKA." Annual of social work 28, no. 3 (2022): 673–93. http://dx.doi.org/10.3935/ljsr.v28i3.392.

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PERCEPTION OF CITIZENS ON ARREST AS A MEASURE TO ENSURE THE DEFENDANT’S PRESENCE ABSTRACT Arrest is a specific and necessary measure to ensure the defendant’s presence which is made by the police before the beginning of a criminal procedure. Arrest is an “institute” determined by the Constitution of the Republic of Croatia and the most important supranational sources on human rights and freedoms, such as the Universal Declaration of Human Rights, International Covenant on Civil and Political Rights, European Convention for the Protection of Human Rights and Fundamental Freedoms and other sourc

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20

Jaiswal, Sushil K., Sonam Raj, and Melvin L. DePamphilis. "Developmental Acquisition of p53 Functions." Genes 12, no. 11 (2021): 1675. http://dx.doi.org/10.3390/genes12111675.

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Remarkably, the p53 transcription factor, referred to as “the guardian of the genome”, is not essential for mammalian development. Moreover, efforts to identify p53-dependent developmental events have produced contradictory conclusions. Given the importance of pluripotent stem cells as models of mammalian development, and their applications in regenerative medicine and disease, resolving these conflicts is essential. Here we attempt to reconcile disparate data into justifiable conclusions predicated on reports that p53-dependent transcription is first detected in late mouse blastocysts, that p

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21

Crawford, Eugene W., and Lawrence J. Shimkets. "The stringent response in Myxococcus xanthus is regulated by SocE and the CsgA C-signaling protein." Genes & Development 14, no. 4 (2000): 483–92. http://dx.doi.org/10.1101/gad.14.4.483.

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Myxococcus xanthus fruiting body development is induced by amino acid limitation. The decision to grow or develop is established by the RelA-dependent stringent response and A-signaling. We identified two new members of this regulatory hierarchy, socE and the C-signaling gene csgA. SocE depletion arrests growth and induces sporulation under conditions that normally favor growth as well as curtailing DNA and stable RNA synthesis, inhibiting cell elongation, and inducing accumulations of the stringent nucleotides ppGpp and pppGpp [(p)ppGpp]. This system separates C-signaling, which does not occu

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22

Chehab, Nabil H., Asra Malikzay, Michael Appel, and Thanos D. Halazonetis. "Chk2/hCds1 functions as a DNA damage checkpoint in G₁ by stabilizing p53." Genes & Development 14, no. 3 (2000): 278–88. http://dx.doi.org/10.1101/gad.14.3.278.

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Chk2/hcds1, the human homolog of theSaccharomyces cerevisiae RAD53/SPK1 andSchizosaccharomyces pombe cds1 DNA damage checkpoint genes, encodes a protein kinase that is post-translationally modified after DNA damage. Like its yeast homologs, the Chk2/hCds1 protein phosphorylates Cdc25C in vitro, suggesting that it arrests cells in G2 in response to DNA damage. We expressed Chk2/hCds1 in human cells and analyzed their cell cycle profile. Wild-type, but not catalytically inactive, Chk2/hCds1 led to G1 arrest after DNA damage. The arrest was inhibited by cotransfection of a dominant-negative p53 m

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23

Webb, Bruce A., and D. L. Dahlman. "Developmental pathology ofHeliothis virescens larvae parasitized byMicroplitis croceipes: Parasite-mediated host developmental arrest." Archives of Insect Biochemistry and Physiology 2, no. 2 (1985): 131–43. http://dx.doi.org/10.1002/arch.940020203.

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24

Watnick, Randolph S., Stephanie Chiyoko Herring, Arthur G. Palmer, and Max E. Gottesman. "The carboxyl terminus of phage HK022 Nun includes a novel zinc-binding motif and a tryptophan required for transcription termination." Genes & Development 14, no. 6 (2000): 731–39. http://dx.doi.org/10.1101/gad.14.6.731.

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The amino-terminal arginine-rich motif of the phage HK022 Nun protein binds phage λ nascent mRNA transcripts while the carboxy-terminal domain binds RNA polymerase and arrests transcription. The role of specific residues in the carboxy-terminal domain in transcription termination were investigated by mutagenesis, in vitro and in vivo functional assays, and NMR spectroscopy. Coordination of zinc to three histidine residues in the carboxy-terminus inhibited RNA binding by the amino-terminal domain; however, only two of these histidines were required for transcription arrest. These results sugges

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25

Rafferty, Anthony R., and Richard D. Reina. "Arrested embryonic development: a review of strategies to delay hatching in egg-laying reptiles." Proceedings of the Royal Society B: Biological Sciences 279, no. 1737 (2012): 2299–308. http://dx.doi.org/10.1098/rspb.2012.0100.

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Arrested embryonic development involves the downregulation or cessation of active cell division and metabolic activity, and the capability of an animal to arrest embryonic development results in temporal plasticity of the duration of embryonic period. Arrested embryonic development is an important reproductive strategy for egg-laying animals that provide no parental care after oviposition. In this review, we discuss each type of embryonic developmental arrest used by oviparous reptiles. Environmental pressures that might have directed the evolution of arrest are addressed and we present previo

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Sun, Yanmei, Fan Gao, Da Xu, et al. "Wenshen Shengjing Decoction Improves Early Embryo Development by Maintaining Low H3K27me3 Levels in Sperm and Pronuclear Embryos of Spermatogenesis Impaired Mice." Evidence-Based Complementary and Alternative Medicine 2021 (September 27, 2021): 1–11. http://dx.doi.org/10.1155/2021/8035997.

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Many ingredients in Wenshen Shengjing Decoction (WSSJD) can cause epigenetic changes in the development of different types of cells. It is not yet known whether they can cause epigenetic changes in sperms or early embryos. Here, we investigated the role of WSSJD in epigenetic modifications of sperms or early embryos and early embryo development. A mouse model with spermatogenesis disorders was established with cyclophosphamide (CPA). WSSJD was administrated for 30 days. The male model mice after the treatment were mated with the female mice treated with superovulation. The embryo development r

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27

McMahon, William S., Paul C. Gillette, Robert B. Hinton, Jennifer R. Stratton, Fred A. Crawford, and Francis G. Spinale. "Developmental differences in myocyte contractile response after cardioplegic arrest." Journal of Thoracic and Cardiovascular Surgery 111, no. 6 (1996): 1257–66. http://dx.doi.org/10.1016/s0022-5223(96)70229-5.

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28

Crow, T. J., N. Colter, C. D. Frith, E. C. Johnstone, and D. G. C. Owens. "Developmental arrest of cerebral asymmetries in early onset schizophrenia." Psychiatry Research 29, no. 3 (1989): 247–53. http://dx.doi.org/10.1016/0165-1781(89)90053-x.

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29

Miyan, J. A., C. M. Bannister, and F. Mashayekhi. "Developmental arrest in early-onset hydrocephalus: Clues to signalling." Comparative Biochemistry and Physiology Part B: Biochemistry and Molecular Biology 126 (July 2000): S68. http://dx.doi.org/10.1016/s0305-0491(00)80135-2.

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30

Miyan, J. A., C. M. Bannister, and F. Mashayekhi. "Developmental arrest in early-onset hydrocephalus: Clues to signalling." Comparative Biochemistry and Physiology Part B: Biochemistry and Molecular Biology 126 (July 2000): S69. http://dx.doi.org/10.1016/s0305-0491(00)80136-4.

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31

Mohebi, Mehdi, and Soudeh Ghafouri-Fard. "Embryo developmental arrest: Review of genetic factors and pathways." Gene Reports 17 (December 2019): 100479. http://dx.doi.org/10.1016/j.genrep.2019.100479.

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32

Zhang, Deqing, and D. L. Dahlman. "Microplitis croceipes teratocytes cause developmental arrest ofHeliothis virescens larvae." Archives of Insect Biochemistry and Physiology 12, no. 1 (1989): 51–61. http://dx.doi.org/10.1002/arch.940120105.

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33

Frei, Christian, and Susan M. Gasser. "The yeast Sgs1p helicase acts upstream of Rad53p in the DNA replication checkpoint and colocalizes with Rad53p in S-phase-specific foci." Genes & Development 14, no. 1 (2000): 81–96. http://dx.doi.org/10.1101/gad.14.1.81.

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We have examined the cellular function of Sgs1p, a nonessential yeast DNA helicase, homologs of which are implicated in two highly debilitating hereditary human diseases (Werner's and Bloom's syndromes). We show that Sgs1p is an integral component of the S-phase checkpoint response in yeast, which arrests cells due to DNA damage or blocked fork progression during DNA replication. DNA polε and Sgs1p are found in the same epistasis group and act upstream of Rad53p to signal cell cycle arrest when DNA replication is perturbed. Sgs1p is tightly regulated through the cell cycle, accumulates in S ph

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34

Kinoshita, Tsutomu. "Growth factors reverse developmental arrest by zinc in embryos of the sea urchin Hemicentrotus pulcherrimus." Canadian Journal of Zoology 77, no. 3 (1999): 360–67. http://dx.doi.org/10.1139/z98-222.

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During sea urchin development, zinc (Zn) ions produce developmentally arrested embryos called permanent blastulae. In this study, the effects of growth factors on Zn-arrested embryos were examined to elucidate the relation between Zn arrest and morphogenesis that is mediated by growth factors. Embryos treated with Zn maintained their spherical form for a few days in the absence of growth factors. When the culture medium was supplemented with horse serum, gastrulation occurred in the Zn-arrested embryos, some of which developed into symmetrical larvae closely resembling normal plutei. The growt

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35

Tunquist, B. J. "Under arrest: cytostatic factor (CSF)-mediated metaphase arrest in vertebrate eggs." Genes & Development 17, no. 6 (2003): 683–710. http://dx.doi.org/10.1101/gad.1071303.

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36

Wang, Pei, Ji Cui, Chun Zhao, et al. "Differential expression of microRNAs in 2-cell and 4-cell mouse embryos." Zygote 22, no. 4 (2013): 455–61. http://dx.doi.org/10.1017/s0967199413000117.

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SummaryIn vitro fertilized (IVF) human embryos have a high incidence of developmental arrest before the blastocyst stage, therefore characterization of the molecular mechanisms that regulate embryo development is urgently required. Post-transcriptional control by microRNAs (miRNAs) is one of the most investigated RNA control mechanisms, and is hypothesized to be involved actively in developmental arrest in preimplantation embryos. In this study, we extracted total RNA from mouse 2-cell and 4-cell embryos. Using a miRNA microarray, 192 miRNAs were found to be differentially expressed in 4-cell

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37

Shyu, Li-Fang, Jianjun Sun, Hui-Min Chung, Yi-Chun Huang, and Wu-Min Deng. "Notch Signaling and Developmental Cell-Cycle Arrest in Drosophila Polar Follicle Cells." Molecular Biology of the Cell 20, no. 24 (2009): 5064–73. http://dx.doi.org/10.1091/mbc.e09-01-0004.

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Temporal and spatial regulation of cell division is critical for proper development of multicellular organisms. An important aspect of this regulation is cell-cycle arrest, which in many cell types is coupled with differentiated status. Here we report that the polar cells—a group of follicle cells differentiated early during Drosophila oogenesis—are arrested at G2 phase and can serve as a model cell type for investigation of developmental regulation of cell-cycle arrest. On examining the effects of String, a mitosis-promoting phosphatase Cdc25 homolog, and Notch signaling in polar cells, we fo

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38

Mandik-Nayak, Laura, Su-jean Seo, Caroline Sokol, Kathryn M. Potts, Anh Bui, and Jan Erikson. "MRL-lpr/lpr Mice Exhibit a Defect in Maintaining Developmental Arrest and Follicular Exclusion of Anti–double-stranded DNA B Cells." Journal of Experimental Medicine 189, no. 11 (1999): 1799–814. http://dx.doi.org/10.1084/jem.189.11.1799.

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A hallmark of systemic lupus erythematosus and the MRL murine model for lupus is the presence of anti–double-stranded (ds)DNA antibodies (Abs). To identify the steps leading to the production of these Abs in autoimmune mice, we have compared the phenotype and localization of anti-dsDNA B cells in autoimmune (MRL+/+ and lpr/lpr) mice with that in nonautoimmune (BALB/c) mice. Anti-dsDNA B cells are actively regulated in BALB/c mice as indicated by their developmental arrest and accumulation at the T–B interface of the splenic follicle. In the MRL genetic background, anti-dsDNA B cells are no lon

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39

Herrick, Jason R. "Reversible meiotic arrest in feline oocytes." Reproduction, Fertility and Development 26, no. 2 (2014): 258. http://dx.doi.org/10.1071/rd12341.

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Increasing intracellular concentrations of cyclic adenosine monophosphate (cAMP) within the cumulus–oocyte complex (COC) inhibits or delays spontaneous oocyte maturation and improves the developmental competence of the oocyte in many species, but information for carnivores is limited. The objectives of the present study were to describe the effects of isobutyl methylxanthine (IBMX), which decreases cAMP degradation, and forskolin, which increases cAMP production, on spontaneous and induced maturation (by equine chorionic gonadotrophin (eCG) and epidermal growth factor (EGF)) of feline oocytes

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Yasuda, G. K., G. Schubiger, and B. T. Wakimoto. "Genetic characterization of ms (3) K81, a paternal effect gene of Drosophila melanogaster." Genetics 140, no. 1 (1995): 219–29. http://dx.doi.org/10.1093/genetics/140.1.219.

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Abstract The vast majority of known male sterile mutants of Drosophila melanogaster fail to produce mature sperm or mate properly. The ms(3) K81(1) mutation is one of a rare class of male sterile mutations in which sterility is caused by developmental arrest after sperm entry into the egg. Previous studies showed that males homozygous for the K81(1) mutation produce progeny that arrest at either of two developmental stages. Most embryos arrest during early nuclear cycles, whereas the remainder are haploid embryos that arrest at a later stage. This description of the mutant phenotype was based

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41

Sliter, T. J., and L. I. Gilbert. "Developmental arrest and ecdysteroid deficiency resulting from mutations at the dre4 locus of Drosophila." Genetics 130, no. 3 (1992): 555–68. http://dx.doi.org/10.1093/genetics/130.3.555.

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Abstract Loss-of-function mutations of the dre4 gene of Drosophila melanogaster caused stage-specific developmental arrest, the stages of arrest coinciding with periods of ecdysteroid (molting hormone) regulated development. Nonconditional mutations resulted in the arrest of larval development in the first instar; embryogenesis was not impaired, and mutant larvae were behaviorally normal and long-lived. At 31 degrees the temperature-sensitive dre4e55 allele caused the arrest of larval development in the first or second instars. When upshifted to 31 degrees at various times during development,

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42

Shen-Li, Hong, Rónán C. O'Hagan, Harry Hou, James W. Horner, Han-Woong Lee, and Ronald A. DePinho. "Essential role for Max in early embryonic growth and development." Genes & Development 14, no. 1 (2000): 17–22. http://dx.doi.org/10.1101/gad.14.1.17.

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Loss of Max function in the mouse resulted in generalized developmental arrest of both embryonic and extraembryonic tissues at early postimplantation (∼E5.5–6.5), coincident with loss or dilution of maternal Max stores in the expanding embryo in vivo and in blastocyst outgrowths in vitro. Developmentally arrested embryos were reduced in size and exhibited widespread cytological degeneration and feeble BrdU incorporation. Max and, by extension, the Myc superfamily, serve essential roles in early mammalian development and a maternal reservoir of Max exists in sufficient amount to sustain Myc sup

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43

Yan, Shao-Fei, Xin-Yan Liu, Yun-Fei Cheng, et al. "Relationship between Intrauterine Bacterial Infection and Early Embryonic Developmental Arrest." Chinese Medical Journal 129, no. 12 (2016): 1455–58. http://dx.doi.org/10.4103/0366-6999.183411.

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44

Govind, Shubha, Jonathan P. Melk, and Jorge Morales. "DEVELOPMENTAL ARREST AND PHYSICAL ENTRAPMENT ELIMINATES SUPERNUMERARYGANASPIS XANTHOPODAPARASITOIDS INDROSOPHILA MELANOGASTER." Journal of Parasitology 86, no. 3 (2000): 463–70. http://dx.doi.org/10.1645/0022-3395(2000)086[0463:daapee]2.0.co;2.

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45

Calderon, S., I. Kaplan, and G. Gal. "Developmental arrest of tooth bud after correction of mandibular fracture." Dental Traumatology 11, no. 2 (1995): 105–7. http://dx.doi.org/10.1111/j.1600-9657.1995.tb00468.x.

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46

Devès, Mathilde, and Franck Bourrat. "Transcriptional mechanisms of developmental cell cycle arrest: Problems and models." Seminars in Cell & Developmental Biology 23, no. 3 (2012): 290–97. http://dx.doi.org/10.1016/j.semcdb.2012.03.003.

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47

Jordan, Abbie, Melanie Noel, Line Caes, Hannah Connell, and Jeremy Gauntlett-Gilbert. "A developmental arrest? Interruption and identity in adolescent chronic pain." PAIN Reports 3 (September 2018): e678. http://dx.doi.org/10.1097/pr9.0000000000000678.

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Yu, I.-Ting, and Grover M. Hutchins. "Truncus arteriosus malformation: A developmental arrest at Carnegie stage 14." Teratology 53, no. 1 (1996): 31–37. http://dx.doi.org/10.1002/(sici)1096-9926(199601)53:1<31::aid-tera4>3.0.co;2-c.

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Sakurai, Sho, and Hiroshi Imokawa. "Developmental arrest induced by juvenile hormone in larvae ofBombyx mori." Archives of Insect Biochemistry and Physiology 8, no. 4 (1988): 219–28. http://dx.doi.org/10.1002/arch.940080403.

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Poehlmann-Tynan, Julie, Luke Muentner, Kaitlyn Pritzl, et al. "The Health and Development of Young Children Who Witnessed Their Parent’s Arrest Prior to Parental Jail Incarceration." International Journal of Environmental Research and Public Health 18, no. 9 (2021): 4512. http://dx.doi.org/10.3390/ijerph18094512.

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Abstract:

Most U.S. incarceration occurs in jails, with more than 10 million annual admissions, and most individuals in jail are parents of minor children. In this short-term longitudinal study, we examined the health and development of young children who did or did not witness their parent’s arrest prior to parental jail incarceration. 228 individuals in 76 triads (incarcerated parents, children, at-home caregivers) were enrolled from four jails in two states. Jailed parents and caregivers reported on whether the child witnessed the parent’s arrest or crime. Children’s caregivers completed questionnair

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