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Relevant bibliographies by topics / Diabetes Mellitus, Beta Cells, ACE2, SARS-CoV-2

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Journal articles

Academic literature on the topic 'Diabetes Mellitus, Beta Cells, ACE2, SARS-CoV-2'

Author: Grafiati

Published: 14 March 2023

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Journal articles on the topic "Diabetes Mellitus, Beta Cells, ACE2, SARS-CoV-2"

1

Nimje, Prajakta D., Vidya Wasnik (Thatere), and Sumeeta Jain. "MANAGEMENT THROUGH AYURVEDIC MEDICATIONS AND LIFESTYLE MODIFICATION (PATHYASEVANA AND YOGASANA) IN A CASE OF POST-COVID COMPLICATION DIABETES MELLITUS (TYPE 2 DM): A SINGLE CASE STUDY." International Ayurvedic Medical Journal 9, no. 10 (2021): 2619–28. http://dx.doi.org/10.46607/iamj5409102021.

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Diabetes is an asymptomatic disease in most people, so there could be a good number of people who may not be aware of their diabetes before they caught COVID-19. Some studies state that in poorly resourced countries, as many as 50% of people with chronic illnesses, such as diabetes, are undiagnosed. Theoretically, COVID-19 can also cause diabetes as the pancreas have ACE2 receptors, which can enable SARS COV2 to enter the pancreatic beta cells, resulting in structural and functional damage. The present study is carried out to know the traditional Ayurvedic treatment for Diabetes Mellitus. For

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2

Junhai, Gazi Rayeeda, Hasi Rani Saha, and Bidhan Chandra Sarkar. "Prevalence of COVID-19 Among Older People with Type 2 Diabetes Mellitus: A Systematic Review." International Journal of Science and Healthcare Research 7, no. 3 (2022): 273–88. http://dx.doi.org/10.52403/ijshr.20220738.

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Background: On March 11, 2020, the WHO proclaimed the Coronavirus Disease 2019 (COVID-19) pandemic owing to coronavirus-2 (SARS-CoV-2). SARS-CoV-2 is a zoonotic virus that may be spread from bats to humans through airborne droplets and aerosols. SARS-CoV-2 spike protein has a high binding affinity for ACE2 receptors, widely expressed throughout the respiratory system, notably in epithelial lung cells. ACE2 receptors are found in intestinal mucosal, endothelial, heart, renal epithelial as well as cerebral neuronal cells, explaining COVID-19 extrapulmonary symptoms like diarrhea, nausea, vomitin

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3

Kadir, Nursin Abdul, and Ida Parwati. "COVID-19 (Symptomatic Non-Respiratory) with Type 2 Diabetes Mellitus." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 29, no. 1 (2023): 101–6. http://dx.doi.org/10.24293/ijcpml.v29i1.1863.

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COVID-19 is a respiratory infection caused by a new strain of Coronavirus, Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2), which is highly contagious, primarily through respiratory droplets and contact. Typical symptoms include fever, cough, and shortness of breath. Weakness, nausea, and vomiting are often accompanied by respiratory symptoms but are sometimes confusing when these symptoms occur without respiratory symptoms. COVID-19 can affect any age group, are more common in adults and males and increase in patients with comorbidities. One of the most common comorbidities is Di

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4

Casillas Santana, Miguel Angel, Juan Antonio Arreguín Cano, Alejandro Dib Kanán, et al. "Should We Be Concerned about the Association of Diabetes Mellitus and Periodontal Disease in the Risk of Infection by SARS-CoV-2? A Systematic Review and Hypothesis." Medicina 57, no. 5 (2021): 493. http://dx.doi.org/10.3390/medicina57050493.

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The objective of this article was to conduct a systematic review of the literature to contrast the existing evidence regarding the relationship between periodontal disease (PD) and diabetes mellitus (DM) with the possibly increased risk of SARS-CoV-2 infection, as well as to establish a hypothesis that explains the ways in which this interaction could take place. A literature search up from 1 January 2020 to 21 March 2021 was conducted in three electronic databases, namely, PubMed, Web of Science, and Scopus, in order to identify studies on periodontal disease alone or in conjunction with diab

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5

Beliard, Kara A., Mabel Yau, Meredith Wilkes, Christopher Joseph Romero, Elizabeth Wallach, and Robert Rapaport. "SARS-CoV-2 Infection Related Diabetes Mellitus." Journal of the Endocrine Society 5, Supplement_1 (2021): A397. http://dx.doi.org/10.1210/jendso/bvab048.808.

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Abstract Introduction: SARS-Cov-2 (severe acute respiratory distress syndrome- coronavirus 2) viral infection has a predilection for pancreatic beta cells causing insulin deficiency. Studies from the SARS-CoV outbreak in 2003 highlighted the relationship between SARS-CoV and ACE-2 (angiotensin-converting enzyme 2) receptors in pancreatic islet cells. We describe a pediatric patient who developed Diabetes Mellitus after exposure to the Sars-CoV-2 virus. Case Report: A previously healthy 13-year-old female of Mexican descent was found to be hyperglycemic at her annual visit. The patient endorsed

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6

Pramanik, Parthiba, and Purushottam Pramanik. "Diabetes mellitus augments the complications of patients with COVID-19: a review." International Journal of Research in Medical Sciences 8, no. 7 (2020): 2716. http://dx.doi.org/10.18203/2320-6012.ijrms20202925.

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Corona virus disease 2019 (COVID-19) is current pandemic infection caused by RNA virus named severe acute respiratory syndrome coronavirus-2 (SARS Cov-2). The lungs are the organs most affected by COVID-19 and people were died due to severe acute respiratory s syndrome, pneumonia and multi-organs failure. Fatality rate was more, those who suffer in chronic diseases including diabetes mellitus (DM). As COVID-19 pandemic is accelerating , it is important to understand the molecular mechanism through which DM increases the severity related to COVID-19 to able to design more appropriate therapy. T

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7

Wicik, Zofia, Ceren Eyileten, Daniel Jakubik, et al. "ACE2 Interaction Networks in COVID-19: A Physiological Framework for Prediction of Outcome in Patients with Cardiovascular Risk Factors." Journal of Clinical Medicine 9, no. 11 (2020): 3743. http://dx.doi.org/10.3390/jcm9113743.

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Background: Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection (coronavirus disease 2019; COVID-19) is associated with adverse outcomes in patients with cardiovascular disease (CVD). The aim of the study was to characterize the interaction between SARS-CoV-2 and Angiotensin-Converting Enzyme 2 (ACE2) functional networks with a focus on CVD. Methods: Using the network medicine approach and publicly available datasets, we investigated ACE2 tissue expression and described ACE2 interaction networks that could be affected by SARS-CoV-2 infection in the heart, lungs and nervous s

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8

Pelle, Maria Chiara, Isabella Zaffina, Stefania Lucà, et al. "Endothelial Dysfunction in COVID-19: Potential Mechanisms and Possible Therapeutic Options." Life 12, no. 10 (2022): 1605. http://dx.doi.org/10.3390/life12101605.

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SARS-CoV-2, a novel coronavirus found in Wuhan (China) at the end of 2019, is the etiological agent of the current pandemic that is a heterogeneous disease, named coronavirus disease 2019 (COVID-19). SARS-CoV-2 affects primarily the lungs, but it can induce multi-organ involvement such as acute myocardial injury, myocarditis, thromboembolic eventsandrenal failure. Hypertension, chronic kidney disease, diabetes mellitus and obesity increase the risk of severe complications of COVID-19. There is no certain explanation for this systemic COVID-19 involvement, but it could be related to endothelial

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9

Parrey, Ashaq, Abir aijaz, Mohd Ismail, et al. "New Onset Diabetes and Its Incidence in Severe COVID 19 Disease A Single Centre Study From Kashmir." Journal of Endocrinology and Diabetes 8, no. 2 (2012): 1–4. http://dx.doi.org/10.15226/2374-6890/8/2/001152.

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Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the novel coronavirus that causes coronavirus disease 2019 (COVID-19), was first reported in Wuhan, China, in December 2019 and has spread worldwide. SARS-CoV-2 is a positivestranded RNA virus that is enclosed by a protein containing lipid bilayer with a single-stranded RNA genome; SARS-CoV-2 has 82% homology with human SARS-CoV, which causes severe acute respiratory syndrome.SARS-CoV-2, virus binds to angiotensinconverting enzyme 2 (ACE2) receptors, which are expressed in key metabolic organs and tissues, including pancreatic beta

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10

Blîndu, Emanuel, Renata Gerculy, Diana Opincariu, Daniel Cernica, and Imre Benedek. "Cessation of Renin-Angiotensin System Antagonists During the SARS-CoV-2 Pandemic – Do We Have the Evidence?" Journal of Interdisciplinary Medicine 5, no. 3 (2020): 105–9. http://dx.doi.org/10.2478/jim-2020-0022.

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AbstractThe aim of this review is to provide a short update on whether treatment with angiotensin-converting enzyme inhibitors (ACEIs) or angiotensin receptor blockers (ARBs) has beneficial or harmful effects in patients infected with SARS-CoV-2. Epidemiological studies have shown that SARS-CoV-2 infects all age groups, presenting a higher incidence in elderly patients with various comorbidities such as hypertension, diabetes mellitus, and cardiovascular diseases. A large proportion of these patients are treated with ACEIs and ARBs. Since it has been demonstrated that SARS-CoV-2 uses angiotens

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