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Journal articles on the topic 'Diabetes resistance'

Author: Grafiati

Published: 6 July 2024

Last updated: 31 July 2025

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1

Gokalp, Deniz, Alpaslan Tuzcu, Ster Irmak, Mithat Bahceci, and Ozlem Demirpence. "FREQUENCY OF ASPIRIN RESISTANCE IN PATIENTS WITH TYPE 1 AND 2 DIABETES MELLITUS AND ITS ASSOCIATION WITH METABOLIC PARAMETERS." International Journal of Surgery and Medicine 2, no. 3 (2016): 122. http://dx.doi.org/10.5455/ijsm.aspirin-resistance-diabetes.

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2

McClain, D. A., and E. D. Crook. "Hexosamines and insulin resistance." Diabetes 45, no. 8 (1996): 1003–9. http://dx.doi.org/10.2337/diabetes.45.8.1003.

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3

Sceppa, Carmen Castaneda. "Resistance Training and Diabetes." Medicine & Science in Sports & Exercise 36, Supplement (2004): S93—S94. http://dx.doi.org/10.1249/00005768-200405001-00442.

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4

Sceppa, Carmen Castaneda. "Resistance Training and Diabetes." Medicine & Science in Sports & Exercise 36, Supplement (2004): S93???S94. http://dx.doi.org/10.1097/00005768-200405001-00442.

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5

Kraegen, E. W., P. W. Clark, A. B. Jenkins, E. A. Daley, D. J. Chisholm, and L. H. Storlien. "Development of muscle insulin resistance after liver insulin resistance in high-fat-fed rats." Diabetes 40, no. 11 (1991): 1397–403. http://dx.doi.org/10.2337/diabetes.40.11.1397.

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6

Mykkanen, L., D. J. Zaccaro, L. E. Wagenknecht, D. C. Robbins, M. Gabriel, and S. M. Haffner. "Microalbuminuria is associated with insulin resistance in nondiabetic subjects: the insulin resistance atherosclerosis study." Diabetes 47, no. 5 (1998): 793–800. http://dx.doi.org/10.2337/diabetes.47.5.793.

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7

Goran, M. I., and B. A. Gower. "Longitudinal Study on Pubertal Insulin Resistance." Diabetes 50, no. 11 (2001): 2444–50. http://dx.doi.org/10.2337/diabetes.50.11.2444.

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8

Zhan, Yiqiang, Zheng Tang, and Jinming Yu. "Serum ferritin, diabetes, diabetes control, and insulin resistance." Acta Diabetologica 51, no. 6 (2014): 991–98. http://dx.doi.org/10.1007/s00592-014-0656-1.

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9

Wang, J., S. Obici, K. Morgan, N. Barzilai, Z. Feng, and L. Rossetti. "Overfeeding Rapidly Induces Leptin and Insulin Resistance." Diabetes 50, no. 12 (2001): 2786–91. http://dx.doi.org/10.2337/diabetes.50.12.2786.

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10

Massarelli, Laura, Giuseppe Musumeci, Carlo Bussolino, and Valerio Tomaselli. "Diabetes mellitus and clopidogrel “resistance”." Clinical Management Issues 5, no. 2S (2015): 55–58. http://dx.doi.org/10.7175/cmi.v5i2s.1112.

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In our Department arrives a 75-year-old patient with hypertension, diabetes mellitus (DM) treated with hypoglycaemic drugs, dyslipidaemia and ischaemic heart disease post-acute myocardial infarction treated with triple coronary artery bypass surgery and subsequent percutaneous transluminal coronary angioplasty (PTCA). After a new PTCA and positioning of medical stent he is discharged with a double antiplatelet therapy. But after one month two thrombotic events occur in this patients almost simultaneously. Antiplatelet therapy such as clopidogrel and aspirin in combination, is the current gold

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11

Ozsahin, Akatli, Suheyl Asma, Aydana Aksoyek, Cigdem Gereklioglu, and Asli Korur. "Obesity- Insulin Resistance and Diabetes." Turkish Journal of Family Medicine & Primary Care 9, no. 2 (2015): 36. http://dx.doi.org/10.5455/tjfmpc.172673.

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12

Livingstone, C., and G. W. Gould. "Insulin Resistance in Diabetes Mellitus." Scottish Medical Journal 40, no. 2 (1995): 37–39. http://dx.doi.org/10.1177/003693309504000202.

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13

DeFronzo, Ralph A., and Stefano Del Prato. "Insulin resistance and diabetes mellitus." Journal of Diabetes and its Complications 10, no. 5 (1996): 243–45. http://dx.doi.org/10.1016/1056-8727(96)00046-3.

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14

Hadigan, Colleen. "Diabetes, insulin resistance, and HIV." Current Infectious Disease Reports 8, no. 1 (2006): 69–75. http://dx.doi.org/10.1007/s11908-006-0037-1.

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15

Laville, M., and J. A. Nazare. "Diabetes, insulin resistance and sugars." Obesity Reviews 10 (March 2009): 24–33. http://dx.doi.org/10.1111/j.1467-789x.2008.00562.x.

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16

Takano, Chika, Erika Ogawa, and Satoshi Hayakawa. "Insulin Resistance in Mitochondrial Diabetes." Biomolecules 13, no. 1 (2023): 126. http://dx.doi.org/10.3390/biom13010126.

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Mitochondrial diabetes (MD) is generally classified as a genetic defect of β-cells. The main pathophysiology is insulin secretion failure in pancreatic β-cells due to impaired mitochondrial ATP production. However, several reports have mentioned the presence of insulin resistance (IR) as a clinical feature of MD. As mitochondrial dysfunction is one of the important factors causing IR, we need to focus on IR as another pathophysiology of MD. In this special issue, we first briefly summarized the insulin signaling and molecular mechanisms of IR. Second, we overviewed currently confirmed pathogen

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17

Ajjan, R., R. F. Storey, and P. J. Grant. "Aspirin resistance and diabetes mellitus." Diabetologia 51, no. 3 (2008): 385–90. http://dx.doi.org/10.1007/s00125-007-0898-3.

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18

Senn, J. J., P. J. Klover, I. A. Nowak, and R. A. Mooney. "Interleukin-6 Induces Cellular Insulin Resistance in Hepatocytes." Diabetes 51, no. 12 (2002): 3391–99. http://dx.doi.org/10.2337/diabetes.51.12.3391.

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19

Spurlin, B. A., R. M. Thomas, A. K. Nevins, et al. "Insulin Resistance in Tetracycline-Repressible Munc18c Transgenic Mice." Diabetes 52, no. 8 (2003): 1910–17. http://dx.doi.org/10.2337/diabetes.52.8.1910.

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20

Maqbool, Mudasir. "Role of Insulin Resistance in Gestational Diabetes Mellitus: A Literature Review." Chettinad Health City Medical Journal 11, no. 02 (2022): 69–74. http://dx.doi.org/10.24321/2278.2044.202218.

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Several physiologic, hormonal, and molecular processes contribute to the emergence of hyperglycaemia during pregnancy. Increased insulin resistance is seen during the course of a healthy pregnancy. During the early stages of normal pregnancies, the pancreatic β-cells secrete more insulin, which slows the rise in plasma glucose levels. This regulation explains the abnormally modest increases in plasma glucose levels brought on by elevated insulin resistance. The aim of this review was to evaluate the role of insulin resistance in gestational diabetes mellitus (GDM). The authors extensively sear

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21

Pocai, A., K. Morgan, C. Buettner, R. Gutierrez-Juarez, S. Obici, and L. Rossetti. "Central Leptin Acutely Reverses Diet-Induced Hepatic Insulin Resistance." Diabetes 54, no. 11 (2005): 3182–89. http://dx.doi.org/10.2337/diabetes.54.11.3182.

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22

Wang, C. C. L., M. L. Goalstone, and B. Draznin. "Molecular Mechanisms of Insulin Resistance That Impact Cardiovascular Biology." Diabetes 53, no. 11 (2004): 2735–40. http://dx.doi.org/10.2337/diabetes.53.11.2735.

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23

Banks, W. A., A. B. Coon, S. M. Robinson, et al. "Triglycerides Induce Leptin Resistance at the Blood-Brain Barrier." Diabetes 53, no. 5 (2004): 1253–60. http://dx.doi.org/10.2337/diabetes.53.5.1253.

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24

Raymond, R. M., M. P. McLane, W. R. Law, N. F. King, and D. W. Leutz. "Myocardial insulin resistance during acute endotoxin shock in dogs." Diabetes 37, no. 12 (1988): 1684–88. http://dx.doi.org/10.2337/diabetes.37.12.1684.

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25

Veroni, M. C., J. Proietto, and R. G. Larkins. "Evolution of insulin resistance in New Zealand obese mice." Diabetes 40, no. 11 (1991): 1480–87. http://dx.doi.org/10.2337/diabetes.40.11.1480.

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26

Haffner, S. M., R. A. Valdez, H. P. Hazuda, B. D. Mitchell, P. A. Morales, and M. P. Stern. "Prospective analysis of the insulin-resistance syndrome (syndrome X)." Diabetes 41, no. 6 (1992): 715–22. http://dx.doi.org/10.2337/diabetes.41.6.715.

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27

Kohrt, W. M., J. P. Kirwan, M. A. Staten, R. E. Bourey, D. S. King, and J. O. Holloszy. "Insulin resistance in aging is related to abdominal obesity." Diabetes 42, no. 2 (1993): 273–81. http://dx.doi.org/10.2337/diabetes.42.2.273.

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28

Chaiken, R. L., M. A. Banerji, H. Huey, and H. E. Lebovitz. "Do blacks with NIDDM have an insulin-resistance syndrome?" Diabetes 42, no. 3 (1993): 444–49. http://dx.doi.org/10.2337/diabetes.42.3.444.

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29

Hunnicutt, J. W., R. W. Hardy, J. Williford, and J. M. McDonald. "Saturated fatty acid-induced insulin resistance in rat adipocytes." Diabetes 43, no. 4 (1994): 540–45. http://dx.doi.org/10.2337/diabetes.43.4.540.

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30

Barzilai, N., L. She, B. Q. Liu, et al. "Surgical removal of visceral fat reverses hepatic insulin resistance." Diabetes 48, no. 1 (1999): 94–98. http://dx.doi.org/10.2337/diabetes.48.1.94.

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31

Kido, Y., N. Philippe, A. A. Schaffer, and D. Accili. "Genetic modifiers of the insulin resistance phenotype in mice." Diabetes 49, no. 4 (2000): 589–96. http://dx.doi.org/10.2337/diabetes.49.4.589.

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32

Gabriely, I., X. H. Ma, X. M. Yang, L. Rossetti, and N. Barzilai. "Leptin Resistance During Aging Is Independent of Fat Mass." Diabetes 51, no. 4 (2002): 1016–21. http://dx.doi.org/10.2337/diabetes.51.4.1016.

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33

Haffner, S. M., R. D'Agostino, M. F. Saad, et al. "Increased insulin resistance and insulin secretion in nondiabetic African-Americans and Hispanics compared with non-Hispanic whites. The Insulin Resistance Atherosclerosis Study." Diabetes 45, no. 6 (1996): 742–48. http://dx.doi.org/10.2337/diabetes.45.6.742.

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34

Premkumar, Daivasikamani. "The Role of Obesity in Producing Type 2 Diabetes by Insulin Resistance." Diabetes & Obesity International Journal 5, no. 2 (2020): 1–9. http://dx.doi.org/10.23880/doij-16000227.

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Obesity is a common condition due wrong eating habits and less physical activity. But it can produce serious illness like type 2 diabetes. In Malaysia 18 years and above 33.3% (5.4 million) are pre-obese and 27.2% (4.4 million) are obese. 7.2% are known to have diabetes and 8.0% are previously undiagnosed with diabetes. There is a strong relationship between obesity and diabetes mellitus. We must understand the mechanism by which obesity causes diabetes through and insulin resistance. When the fat distribution is more peripheral have more insulin sensitivity than whose fat distribution is more

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35

Perkins, Amanda. "Insulin resistance." Nursing Made Incredibly Easy! 22, no. 3 (2024): 5–13. http://dx.doi.org/10.1097/nme.0000000000000046.

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Insulin resistance is believed to be a precursor to type 2 diabetes mellitus, metabolic syndrome, and nonalcoholic fatty liver disease. Through education, nurses can understand their role in helping patients manage this condition.

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36

Rooney, D. P., and R. P. Robertson. "Hepatic insulin resistance after pancreas transplantation in type I diabetes." Diabetes 45, no. 2 (1996): 134–38. http://dx.doi.org/10.2337/diabetes.45.2.134.

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37

Williams, K. V., J. R. Erbey, D. Becker, S. Arslanian, and T. J. Orchard. "Can clinical factors estimate insulin resistance in type 1 diabetes?" Diabetes 49, no. 4 (2000): 626–32. http://dx.doi.org/10.2337/diabetes.49.4.626.

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38

Bloomgarden, Zachary T. "Developments in Diabetes and Insulin Resistance." Diabetes Care 29, no. 1 (2005): 161–67. http://dx.doi.org/10.2337/diacare.29.01.06.dc06-zb01.

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39

Taylor, R. "Insulin Resistance and Type 2 Diabetes." Diabetes 61, no. 4 (2012): 778–79. http://dx.doi.org/10.2337/db12-0073.

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40

Gedela, Vasavi, and Sree Lakshmi Gosala. "Diabetes a risk for antimicrobial resistance?" International Journal of Basic & Clinical Pharmacology 6, no. 12 (2017): 2811. http://dx.doi.org/10.18203/2319-2003.ijbcp20175103.

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Background: Diabetic foot infections can cause substantial morbidity. The role of Diabetes mellitus in the antimicrobial resistance of pathogens in patients with foot infections is not well clarified. So, we compared the profile of antibiotic resistance in diabetic and non-diabetic foot ulcer infections. Objectives were to compare the antimicrobial resistance pattern in diabetic and non-diabetic lower limb infections.Methods: T Pus was isolated in 50 Diabetic and 50 non-diabetic foot ulcer infections. The organisms were isolated on specific media and antibiotic susceptibility was done by using

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41

Flack, Kyle D., Kevin P. Davy, Matthew W. Hulver, Richard A. Winett, Madlyn I. Frisard, and Brenda M. Davy. "Aging, Resistance Training, and Diabetes Prevention." Journal of Aging Research 2011 (2011): 1–12. http://dx.doi.org/10.4061/2011/127315.

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With the aging of the baby-boom generation and increases in life expectancy, the American population is growing older. Aging is associated with adverse changes in glucose tolerance and increased risk of diabetes; the increasing prevalence of diabetes among older adults suggests a clear need for effective diabetes prevention approaches for this population. The purpose of paper is to review what is known about changes in glucose tolerance with advancing age and the potential utility of resistance training (RT) as an intervention to prevent diabetes among middle-aged and older adults. Age-related

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42

Hannah, Judy S., and Barbara V. Howard. "Dietary fats, insulin resistance, and diabetes." Journal of Cardiovascular Risk 1, no. 1 (1994): 31–37. http://dx.doi.org/10.1097/00043798-199406000-00006.

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43

Reusch, Jane E. B., and Boris B. Draznin. "Atherosclerosis in diabetes and insulin resistance." Diabetes, Obesity and Metabolism 9, no. 4 (2007): 455–63. http://dx.doi.org/10.1111/j.1463-1326.2006.00620.x.

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44

Mestman, MD, Jorge H. "INSULIN RESISTANCE SYNDROME AND GESTATIONAL DIABETES." Endocrine Practice 9, Supplement 2 (2003): 90–92. http://dx.doi.org/10.4158/ep.9.s2.90.

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45

ANG, Christine, and Mary Ann LUMSDEN. "Diabetes and the maternal resistance vasculature." Clinical Science 101, no. 6 (2001): 719. http://dx.doi.org/10.1042/cs20010153.

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46

Cowley, Cheryl. "Insulin resistance in type 2 diabetes." Practice Nursing 11, no. 15 (2000): 27–29. http://dx.doi.org/10.12968/pnur.2000.11.15.13037.

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47

Hannah, J. S., and B. V. Howard. "Dietary Fats, Insulin Resistance, and Diabetes." European Journal of Cardiovascular Prevention & Rehabilitation 1, no. 1 (1994): 31–37. http://dx.doi.org/10.1177/174182679400100106.

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48

ANG, Christine, and Mary Ann LUMSDEN. "Diabetes and the maternal resistance vasculature." Clinical Science 101, no. 6 (2001): 719–29. http://dx.doi.org/10.1042/cs1010719.

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Diabetes is the most common endocrine disorder worldwide, with complications that include the development of both macro- and micro-vascular disease that contribute significantly to patient morbidity and mortality. The severity of diabetic complications is amplified during pregnancy, resulting in a higher incidence of adverse pregnancy outcomes such as pre-eclampsia, placental insufficiency and stillbirth than in non-diabetics. Vascular dysfunction is thought to underlie many of these complications, with the greatest impact occurring at the level of the resistance vasculature, where alterations

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49

Lima, Luís Maurício T. R. "Insulin resistance underlying type 2 diabetes." Lancet Diabetes & Endocrinology 7, no. 6 (2019): 424. http://dx.doi.org/10.1016/s2213-8587(19)30147-0.

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50

Feroe, Aliya G., Roberta Attanasio, and Franco Scinicariello. "Acrolein metabolites, diabetes and insulin resistance." Environmental Research 148 (July 2016): 1–6. http://dx.doi.org/10.1016/j.envres.2016.03.015.

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