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Books on the topic 'Diabetic Chronic Kidney Disease'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Tong, Bin. Comorbidity of cardiovascular disease, diabetes, and chronic kidney disease in Australia. Australian Institute of Health and Welfare, 2007.

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Tong, Bin. Comorbidity of cardiovascular disease, diabetes, and chronic kidney disease in Australia. Australian Institute of Health and Welfare, 2007.

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3

Wada, Takashi, Kengo Furuichi, and Naoki Kashihara, eds. Diabetic Kidney Disease. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-9301-7.

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4

Yang, Junwei, and Weichun He, eds. Chronic Kidney Disease. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-32-9131-7.

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5

S, Cappell Mitchell, ed. Chronic kidney disease. Saunders, 2005.

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6

Kumar, Singh Ajay, ed. Chronic kidney disease. Saunders, 2005.

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7

Piracha, Kashif. Chronic Kidney Disease Complications. Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-49922-7.

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8

McCauley, Jerry, Seyed Mehrdad Hamrahian, and Omar H. Maarouf, eds. Approaches to Chronic Kidney Disease. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-83082-3.

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9

Haddad, Maha N., Erica Winnicki, and Stephanie Nguyen, eds. Adolescents with Chronic Kidney Disease. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-97220-6.

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10

Weir, Matthew R., and Edgar V. Lerma, eds. Chronic Kidney Disease and Hypertension. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1982-6.

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11

Arici, Mustafa, ed. Management of Chronic Kidney Disease. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54637-2.

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12

Lerma, Edgar V., and Matthew R. Weir. Chronic kidney disease and hypertension. Humana Press, 2015.

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13

Arıcı, Mustafa, ed. Management of Chronic Kidney Disease. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-42045-0.

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14

Wolf, Gunter. Diabetes and kidney disease. Wiley-Blackwell, 2013.

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15

Ureña Torres, Pablo A., Mario Cozzolino, and Marc G. Vervloet, eds. Vitamin D in Chronic Kidney Disease. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32507-1.

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16

Covic, Adrian, Mehmet Kanbay, and Edgar V. Lerma, eds. Resistant Hypertension in Chronic Kidney Disease. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56827-0.

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17

Covic, Adrian, David Goldsmith, and Pablo A. Ureña Torres, eds. Parathyroid Glands in Chronic Kidney Disease. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43769-5.

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18

Australian Institute of Health and Welfare. Chronic kidney disease in Australia, 2005. Australian Institute of Health and Welfare, 2005.

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19

Orth, Stephan R. Smoking in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0103.

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Smoking has been acknowledged as the number one preventable cause of death in most countries. The adverse effects of smoking on the kidney are less known. Prospective, population-based, observational studies, and evidence from experimental work indicate that smoking (a) is a relevant risk factor for chronic kidney disease (CKD) in the general population and (b) is associated with an increased risk of deterioration in renal function in CKD patients. The latter is especially true for patients with diabetic nephropathy or hypertensive renal damage. The conclusion is that smoking is an important r

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20

Kidney disease of diabetes. National Institute of Diabetes and Digestive and Kidney Diseases, U.S. Department of Health and Human Services, NIH, National Institute of Diabetes and Digestive and Kidney Diseases, 2014.

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21

Williams, Mark E. Diabetic Chronic Kidney Disease, an Issue of Medical Clinics. Elsevier - Health Sciences Division, 2013.

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22

Goldsmith, David J. Cardiovascular disease and chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0098.

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Even after as full a statistical adjustment as can be made for traditional cardiovascular risk factors has been undertaken, impaired kidney function and raised concentrations of albumin in urine each increase the risk of cardiovascular disease (CVD) by two- to fourfold, the degree increasing with severity. If the patient is also suffering from diabetes (as either the cause of CKD or a complication of it), the risks of CVD increase two- to fourfold again. CKD patients should, therefore, be acknowledged as having perhaps the highest cardiovascular risk of any patient cohort. CVD is underdiagnose

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23

Zhang, Luxia, and Haiyan Wang. Chronic kidney disease in developing countries. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0096_update_001.

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The spread of non-communicable diseases (NCDs) is a barrier to the development of goals including reduction of poverty, health equity, economic stability, and human security. NCDs accounted for 61% of the estimated 58 million deaths and 46% of the global burden of diseases worldwide in 2005. Among NCDs, chronic kidney disease (CKD) is of particular significance. It is recognized that the burden of CKD is not only limited to its impact on demands for renal replacement therapy but has equally major impacts on the health of the overall population. For example, it is now well established that amon

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24

Grams, Morgan E., and Josef Coresh. Chronic kidney disease in the developed world. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0095.

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Chronic kidney disease is common, increasing in prevalence, and associated with significant morbidity and mortality. A disease of multiple and complex aetiologies, chronic kidney disease is more prevalent among elderly, hypertensive, and diabetic persons—all growing segments of the developed world. This chapter discusses trends in and determinants of chronic kidney disease prevalence, incidence, and prognosis. In addition, advances in chronic kidney disease staging and reporting as well as the discovery of a major genetic locus for hypertensive kidney disease in populations of African ancestry

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25

Wiles, Kate, Kate Bramham, and Catherine Nelson-Piercy. Kidney disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0044.

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This chapter describes the physiological adaptations to pregnancy in women with and without renal disease, reports pregnancy outcomes in women with both acute kidney injury and chronic kidney disease, and discusses a management strategy for antenatal and peripartum care. Acute kidney injury (AKI) is difficult to define in pregnancy because of the physiological increase in glomerular filtration. A normal creatinine can mask renal injury in pregnancy. This chapter considers important causes of AKI in pregnancy including pre-eclampsia, HELLP syndrome, thrombotic microangiopathy, acute fatty liver

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26

Jardine, Alan G., and Rajan K. Patel. Lipid disorders of patients with chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0102.

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The risk of developing cardiovascular (CV) disease is increased in patients with chronic kidney disease (CKD) and although dyslipidaemia is a major contributory factor to the development of premature CV disease, the relationship is complex. Changes in lipid fractions are related to glomerular filtration rate and the presence and severity of proteinuria, diabetes, and other confounding factors. The spectrum of CV disease changes from lipid-dependent, atheromatous coronary disease in early CKD to lipid-independent, non-coronary disease, manifesting as heart failure, and sudden cardiac death in a

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27

Prevention of cardiovascular disease, diabetes and chronic kidney disease: Targeting risk factors. Australian Institute of Health and Welfare, 2009.

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28

Chakera, Aron, William G. Herrington, and Christopher A. O’Callaghan. Screening for kidney disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0353.

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Renal disease is common and, with routine reporting of estimated glomerular filtration rates, impairment of renal function is increasingly being recognized. As renal impairment is usually asymptomatic until very advanced, chronic kidney disease (CKD) guidelines have been developed to improve the identification and screening of at-risk populations. Target groups include patients with vascular risk factors (e.g. diabetes mellitus and hypertension); patients with certain multisystem diseases which can cause renal impairment; patients with urological conditions; patients on nephrotoxic medication;

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29

Ferro, Charles J., and Khai Ping Ng. Recommendations for management of high renal risk chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0099.

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Poorer renal function is associated with increasing morbidity and mortality. In the wider population this is mainly as a consequence of cardiovascular disease. Renal patients are more likely to progress to end-stage renal disease, but also have high cardiovascular risk. Aiming to reduce both progression of renal impairment and cardiovascular disease are not contradictory. Focusing on the management of high-risk patients with proteinuria and reduced glomerular filtration rates, it is recommended that blood pressure should be kept below 140/90, or 130/80 if proteinuria is > 1 g/24 h (protein:

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30

Diabetic Nephropathy. Exon Publications, 2024. http://dx.doi.org/10.36255/diabetic-nephropathy.

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Diabetic Nephropathy is a serious complication of diabetes that affects the kidneys and can lead to chronic kidney disease and end-stage renal disease. This article provides information about Diabetic Nephropathy, serving as a resource for patients, their loved ones, and the public. The article is organized into key sections, starting with an introduction to the condition and its risk factors. It discusses the epidemiology, causes, and symptoms of Diabetic Nephropathy, followed by an explanation of the pathophysiology and potential complications. The article covers the methods used for diagnos

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31

Kidney friendly comfort foods, volume II: Eating well for chronic kidney disease patients, including those with diabetes. Shire, 2007.

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32

Glomerulopathies: Cell biology and immunology. Harwood Academic Press, 1996.

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33

Teichroew, Jean Kaplan, ed. Chronic Diseases. Greenwood, 2016. http://dx.doi.org/10.5040/9798400626241.

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This comprehensive two-volume work provides an overview of an area of growing concern, offering readers a one-stop resource for researching the chronic conditions that increasingly plague our society. Chronic diseases and their consequences are among the foremost problems faced by the U.S. health care system, accounting for untold distress and mounting personal and societal costs. Bringing together an unprecedented array of detailed data and facts, this unique two-volume encyclopedia provides information that will help readers understand what they can do to avoid these diseases, as well as how

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34

Winston, Jonathan, Etti Zeldis, John A. Grimaldi, and Esteban Martínez. HIV-Associated Nephropathy, End-Stage Renal Disease, Dialysis, and Kidney Transplant. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0044.

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Antiretroviral therapy has changed the phenotype of HIV-related kidney disease to a more chronic disease model. In addition to HIV-associated nephropathy (HIVAN), patients with HIV may experience kidney dysfunction related to other chronic illnesses, such as diabetes, hypertension, and hepatitis C. Patients with HIV should be monitored for the development of chronic kidney disease and the potential nephrotoxicity of antiretroviral therapy. For patients with HIV who progress to end-stage renal disease, the outcomes on dialysis and management of the dialysis procedure are similar to the outcomes

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35

Lapsia, Vijay, Bernard Jaar, and A. Ahsan Ejaz, eds. Kidney Protection. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190611620.001.0001.

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Kidney disease is a crippling disease that affects approximately ten percent of the population worldwide, with more than 2.6 million individuals estimated to receive renal replace therapy. Chronic kidney disease (CKD) is fast becoming a major public health issue even in resource poor settings, with some estimates predicting a disproportionate increase in countries such as China and India. Consequently, renal protection has become a vital and critical component of prevention. While observational data suggests that awareness remains low, the concept of renal protection is currently under-recogni

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36

Carton, James. Renal pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0010.

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This chapter discusses renal pathology, including acute kidney injury (AKI), chronic kidney disease (CKD), nephrotic syndrome, hereditary renal diseases, Alport’s syndrome and thin basement membrane lesion, hypertensive nephropathy, diabetic nephropathy, minimal change disease (MCD), focal segmental glomerulosclerosis (FSGS), membranous glomerulopathy, glomerulonephritis, IgA nephropathy, post-infectious glomerulonephritis, C3 glomerulopathy, anti-glomerular basement membrane disease, monoclonal gammopathy-associated kidney disease, acute tubular injury, acute tubulointerstitial nephritis, ref

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37

Kinsella, Sinead, and John Holian. The effect of chronic renal failure on critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0218.

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The incidence of chronic kidney disease (CKD) and end-stage kidney disease (ESKD) is increasing, reflecting an increase in the incidence and prevalence of hypertension and type 2 diabetes. Patients with CKD and ESKD frequently experience episodes of critical illness and require treatment in an intensive care unit (ICU)setting. Management requires specific consideration of their renal disease status together with their acute illness. Mortality in critically-ill patients with ESKD is frequently related to their co-morbid conditions, rather than their ESKD status. Illness severity scoring systems

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38

Bramham, Kate, and Catherine Nelson-Piercy. Specific renal conditions in pregnancy. Edited by Norbert Lameire and Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0298.

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Pre-pregnancy glomerular filtration rate, proteinuria, and blood pressure are usually more important in determining the risk of pregnancy in patients with chronic kidney disease, but some diseases may be exacerbated in pregnancy, or appear more liable to complications. This chapter considers immunoglobulin A nephropathy, systemic lupus erythematosus (which may also be associated with some manifestations in the infant), diabetic nephropathy, polycystic kidney disease, reflux nephropathy, single kidney, urological disorders, and angiomyolipomata. Distinguishing underlying renal disease exacerbat

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39

Wald, Ron, and Ziv Harel. The Long-Term Outcomes of Acute Kidney Injury. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0015.

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Recent research has provided important insights on the long-term outcomes of patients who develop acute kidney injury (AKI) in the setting of critical illness. Large epidemiologic studies have demonstrated compelling associations between episodes of AKI and progressive kidney disease and death, respectively, although such studies do not establish causality due to the potential for confounding. Whether AKI is intrinsically toxic or a mere by-product of serious comorbidities (e.g. prior chronic kidney disease, heart failure, diabetes), there is no doubt that AKI survivors are a high-risk group w

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40

Stewart, Douglas, Gaurav Shah, Jeremiah R. Brown, and Peter A. McCullough. Contrast-induced acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0246.

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Contrast-induced acute kidney injury (CI-AKI) occurs because all forms of intravascular contrast contain iodine and their biochemical structures induce immediate changes in systemic and renal vasoreactivity. In the kidneys, contrast induces a transient decrease in renal blood flow. This is more pronounced in patients with chronic kidney disease and diabetes mellitus. The reduction in blood flow allows slowed transit of contrast and reabsorption by the proximal tubular cells where contrast is directly toxic resulting in tubular cell dysfunction and death. When there is considerable damage, a tr

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41

Lameire, Norbert. Renal outcomes of acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0238_update_001.

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This chapter summarizes the accumulating evidence that incomplete or even apparent complete recovery of renal function after acute kidney injury (AKI) may be an important contributor to a growing number of incident chronic kidney disease (CKD) and end-stage renal disease (ESRD) cases, largely in excess of the global growth in CKD prevalence. Evidence based on epidemiologic studies supports the notion that even after adjustment for several important covariates AKI is independently associated with an increased risk for both CKD and ESRD. Several risk factors for the subsequent development of CKD

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42

Treating All Chronic and Regular Diseases with Natural Spices: Removing Excess Weight, Treating Diabetes, Kidney and Heart Disease, Lack of Lactation, and Sexual Dysfunction ... Independently Published, 2021.

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43

Kashani, Kianoush B., and Amy W. Williams. Renal Failure. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0473.

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Renal failure is caused by acute kidney injury or chronic kidney disease. Acute kidney injury (AKI) is a common, devastating complication that increases mortality and morbidity among patients with various medical and surgical illnesses. Also known as acute renal failure, AKI is a rapid deterioration of kidney function that results in the accumulation of nitrogenous metabolites and medications and in electrolyte and acid-base imbalances. This chapter discusses the definition, epidemiology, pathophysiology, and etiology of AKI; the clinical approach to patients with AKI; and the management of AK

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44

Dakshinamurty, K. V. Diabetic Kidney Disease. Elsevier - Health Sciences Division, 2010.

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45

Wada, Takashi, Kengo Furuichi, and Naoki Kashihara. Diabetic Kidney Disease. Springer Singapore Pte. Limited, 2020.

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46

Wada, Takashi, Kengo Furuichi, and Naoki Kashihara. Diabetic Kidney Disease. Springer Singapore Pte. Limited, 2021.

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47

Frsph, Johnson Mbabazi. Chronic Kidney Disease. Independently Published, 2019.

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48

Herrington, William G., Aron Chakera, and Christopher A. O’Callaghan. Chronic kidney disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0163.

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Chronic kidney disease (CKD) is defined as abnormalities of kidney structure or function, where the abnormalities have been present for >3 months and have implications for health. It is characterized by a reduced estimated glomerular filtration rate (eGFR) or other renal abnormalities. CKD is staged according to the eGFR or the degree of albuminuria. The KDIGO (Kidney Disease: Improving Global Outcomes) criteria for CKD is either an eGFR that is <60 ml/min 1.73 m−2 and has been present for >3 months, or one or more markers of kidney damage, when these have been present for >3 month

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49

Upadhyay, Ashish, Lesley A. Inker, and Andrew S. Levey. Chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0094.

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The conceptual model, definition, and classification of chronic kidney disease (CKD) were first described in the National Kidney Foundation’s Kidney Disease Outcomes Quality Initiative (KDOQI) guidelines in 2002 and have had a major impact on patient care and research. Since this publication there has been an increased recognition that the cause of CKD influences progression and complications. In addition, epidemiologic reports from diverse populations have consistently shown graded relations between higher albuminuria and adverse kidney outcomes and complications, in addition to, and independ

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50

El Nahas, Meguid, and Adeera Levin, eds. Chronic Kidney Disease. Oxford University Press, 2009. http://dx.doi.org/10.1093/med/9780199549313.001.0001.

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