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Dissertations / Theses on the topic 'Diet-induced'

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Published: 3 June 2025

Last updated: 31 July 2025

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1

Gao, Yuanqing. "Hypothalamic Glial Cells in Diet Induced Obesity." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1447071648.

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2

Blackman, James R. "Diet-induced thermogenesis in the domestic chicken." Diss., Virginia Polytechnic Institute and State University, 1985. http://hdl.handle.net/10919/54737.

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Four experiments were conducted to examine the energetic responses of chicks from lines divergently selected for 56-day body weight to caloric intake. Caloric overconsumption or calorie-protein imbalances were induced by providing glucose solutions (16% w/v) in lieu of water in the first experiment, and by force-feeding the diet to crop capacity in the subsequent trials. Feed restriction was accomplished by providing 75% of ad libitum intake each day. Both low-weight (LN) and high-weight (HN) chicks reduced their voluntary feed intake when glucose solutions were provided; caloric intake was

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3

Eastgard, Rebecca Lugar. "Diet-induced hyperhomocysteinemia in a mouse model /." Thesis, Connect to this title online; UW restricted, 2002. http://hdl.handle.net/1773/6601.

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4

Soltis, Anthony Robert. "Systems biology of diet-induced hepatic insulin resistance." Thesis, Massachusetts Institute of Technology, 2017. http://hdl.handle.net/1721.1/111272.

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Thesis: Ph. D., Massachusetts Institute of Technology, Department of Biological Engineering, 2017.<br>This electronic version was submitted by the student author. The certified thesis is available in the Institute Archives and Special Collections.<br>Cataloged from student-submitted PDF version of thesis.<br>Includes bibliographical references (pages 192-205).<br>Human obesity is a world-wide health crisis that promotes insulin resistance and type 2 diabetes. Obesity increases intracellular free fatty acid concentrations in peripheral tissues, particularly the liver, which disrupts molecular

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5

Burrage, Lindsay. "Genetic Resistance to Diet-Induced Obesity in Mice." Case Western Reserve University School of Graduate Studies / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1151680415.

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6

Robey, Ian Forrest. "Examining the relationship between diet-induced acidosis and cancer." BioMed Central, 2012. http://hdl.handle.net/10150/610226.

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Increased cancer risk is associated with select dietary factors. Dietary lifestyles can alter systemic acid-base balance over time. Acidogenic diets, which are typically high in animal protein and salt and low in fruits and vegetables, can lead to a sub-clinical or low-grade state of metabolic acidosis. The relationship between diet and cancer risk prompts questions about the role of acidosis in the initiation and progression of cancer. Cancer is triggered by genetic and epigenetic perturbations in the normal cell, but it has become clear that microenvironmental and systemic factors exert modi

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7

Blackmore, Heather Louise. "Programming of cardiovascular disease by maternal diet-induced obesity." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648545.

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8

Yang, Tianxu. "Diet-Induced Obesity in Growth Hormone Receptor Antagonist Mice." Ohio University / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1288987144.

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9

Alexander, Lindsey Ann. "The Role of Inflammation in Diet-Induced Insulin Resistance." University of Toledo / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1260808416.

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10

Kogan, Sophia. "Role of Inflammation in Diet-Induced Obesity: A Dissertation." eScholarship@UMMS, 2013. http://escholarship.umassmed.edu/gsbs_diss/647.

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Obesity results from expansion of white adipose tissue. The inability of white adipose tissue to adequately store lipids leads to ectopic deposition of lipids in non-adipose tissue that can lead to systemic insulin resistance. It is well known that insulin resistance correlates with inflammation of adipose tissue in obese animals and humans. Decreasing inflammation in the adipose tissue has been proven as a therapeutic strategy for improvement of insulin sensitivity in vivo. Numerous factors secreted by immune cells, including macrophages, have been suggested as regulating adipose tissue insul

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11

Junfeng, Shi. "The lipid sensor Ffar4 regulates muscle-based diet-induced thermogenesis." 京都大学 (Kyoto University), 2017. http://hdl.handle.net/2433/225536.

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12

Clements, Fiona. "Factors affecting diet-induced thermogenesis in the marsupial Sminthopsis crassicaudata /." Title page and abstract only, 1997. http://web4.library.adelaide.edu.au/theses/09SB/09sbc626.pdf.

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13

麥丹 and Dan Karen Mai. "Effect of green tea on diet-induced obesity in rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31970059.

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14

梁詠蓮 and Wing-lin Winny Leung. "Feeding behavior and serotonin metabolism in diet-induced obese rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2000. http://hub.hku.hk/bib/B31241980.

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15

Bake, Tina. "Modelling and mechanisms of binge eating and diet-induced obesity." Thesis, University of Aberdeen, 2014. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=203778.

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The studies in this thesis aimed to develop and characterise a rodent model of meal feeding that would mimic aspects of human eating behaviour, leading to the overconsumption of calories and ultimately to obesity. In seeking to identify potential mechanisms that might be involved in the initiation of meals, a palatable scheduled feeding regime was utilised to induce a substantial food intake over short periods of time in rodents. This was done by providing scheduled access to a palatable diet for a 2h-period each day without imposed caloric restriction during the remainder of the day. Initiall

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16

Henderson, Michael. "Glucose and diet-induced obesity modulation of ion channel biophysics." Tallahassee, Fla. : Florida State University, 2008. http://purl.fcla.edu/fsu/lib/digcoll/undergraduate/honors-theses/341773.

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Thesis (Honors paper)--Florida State University, 2008.<br>Advisor: Debra Ann Fadool, Ph.D., Florida State University, College of Arts and Sciences, Dept. of Biological Science. Includes bibliographical references.

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17

Leung, Wing-lin Winny. "Feeding behavior and serotonin metabolism in diet-induced obese rats." Hong Kong : University of Hong Kong, 2000. http://sunzi.lib.hku.hk/hkuto/record.jsp?B22425032.

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18

Mai, Dan Karen. "Effect of green tea on diet-induced obesity in rats." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B23295090.

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19

Peterson, Jonathan M., Marcus M. Seldin, Zhikui Wei, Susan Aja, and G. William Wong. "CTRP3 Attenuates Diet-induced Hepatic Steatosis by Regulating Triglyceride Metabolism." Digital Commons @ East Tennessee State University, 2013. https://dc.etsu.edu/etsu-works/72.

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CTRP3 is a secreted plasma protein of the C1q family that helps regulate hepatic gluconeogenesis and is downregulated in a diet-induced obese state. However, the role of CTRP3 in regulating lipid metabolism has not been established. Here, we used a transgenic mouse model to address the potential function of CTRP3 in ameliorating high-fat diet-induced metabolic stress. Both transgenic and wild-type mice fed a high-fat diet showed similar body weight gain, food intake, and energy expenditure. Despite similar adiposity to wild-type mice upon diet-induced obesity (DIO), CTRP3 transgenic mice were

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20

Ho, Dao Hong. "Cocaine hypophagia and hyperlocomotion in rats before and after exposure to a high-fat diet." Thesis, Texas A&M University, 2003. http://hdl.handle.net/1969.1/1641.

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Relatively few studies have examined the effects of psychostimulants in obese subjects. Using the dietary obese rat model, the present experiments determined the reductions in food intake (hypophagia) and increases in locomotion (hyperlocomotion) induced by cocaine in diet-induced obese prone (DIO-prone) rats and diet resistant prone (DR-prone) rats as well as diet-induced obese (DIO) rats and diet resistant (DR) rats. In Experiment 1, thirty-six male Sprague-Dawley rats were given intra-peritoneal (i.p.) injections of cocaine (0, 10, 20, and 30 mg/kg) immediately prior to placement into loco

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21

Hegrenes, Scott Grayson Juliano Steven A. "Diet-induced phenotypic plasticity of feeding morphology in the genus Lepomis." Normal, Ill. Illinois State University, 1999. http://wwwlib.umi.com/cr/ilstu/fullcit?p9942644.

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Thesis (Ph. D.)--Illinois State University, 1999.<br>Title from title page screen, viewed July 24, 2006. Dissertation Committee: Steven A. Juliano (chair), Wayne A. Riddle, Scott K. Sakaluk, Charles F. Thompson, Douglas W. Whitman. Includes bibliographical references (leaves 126-133) and abstract. Also available in print.

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22

Rector, R. Scott. "The antioxidative effects of exercise training-and diet-induced weight loss." Diss., Columbia, Mo. : University of Missouri-Columbia, 2007. http://hdl.handle.net/10355/6020.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2007.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on October 16, 2007) Vita. Includes bibliographical references.

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23

Narayanaswami, Vidya. "DIET-INDUCED OBESITY: DOPAMINERGIC AND BEHAVIORAL MECHANISMS AS OUTCOMES AND PREDICTORS." UKnowledge, 2013. http://uknowledge.uky.edu/pharmacy_etds/12.

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Obesity and drug abuse share common neural circuitries including the mesocoticolimbic and striatal dopamine reward system. In the current study, a rat model of diet-induced obesity (DIO) was used to determine striatal dopamine function, impulsivity and motivation as neurobehavioral outcomes and predictors of obesity. For the outcome study, rats were randomly assigned a high-fat (HF) or a low-fat (LF) diet for 8 wk. Following the 8-wk HF-diet exposure, rats were segregated into obesity-prone and obesity-resistant groups based on maximum and minimum body weight gain, respectively, and neurobehav

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24

Olorunnisola, Olubukola Sinbad. "Biochemical evaluation of Tulbaghia violacea harv.rhizomes in diet induced hypercholestrolemic rats." Thesis, University of Fort Hare, 2012. http://hdl.handle.net/10353/d1006900.

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Discovery of cheap, nontoxic and readily available antiatherosclerotic drugs is an extraordinary challenge in this modern world. Atherosclerosis and cardiovascular diseases have been predicted to be the leading cause of death by the year 2030. Hence, this thesis was designed to search for plant (s) with anti-atherogenic properties, investigate its possible side effects and extrapolate its likely mechanism(s) of action. An ethnobotanical survey was employed in identification of locally important plants used for the management and treatment of cardiovascular diseases and its predisposing factors

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25

Narayan, Pratyush. "Interleukin-18 as a Therapeutic Target in Western-diet Induced Cardiomyopathy." VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/4789.

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Background: Heart Failure (HF) is characterized by impaired cardiac function with symptoms of dyspnea, fatigue and exercise intolerance. Western diet (WD), a diet high in saturated fats and sugars, causes systemic increase in IL-18, a cytokine elevated in HF, which induces abnormal myocardial structure and diastolic dysfunction. IL-18 binding protein(IL-18BP) binds IL-18, preventing the binding to the IL-18-receptor. We investigated a therapeutic intervention using recombinant-murine IL-18BP to improve the WD-induced cardiomyopathy in C57BL/6J mice. Methods: Echocardiography with pulse-wave Do

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26

Lees, Emma Katherine. "Strategies to reverse diet- and age-induced obesity and insulin resistance." Thesis, University of Aberdeen, 2015. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=229428.

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Ageing and obesogenic diets are two prominent problems in the developed world, as both lead to an increase in body mass and insulin resistance, which can then result in further pathophysiologies, such as type 2 diabetes, cancer and cardiovascular disease. Protein tyrosine phosphatase 1B (PTP1B) contributes to development of body weight gain and insulin resistance through negatively regulating leptin and insulin signalling, respectively. Liver-specific ptp1b deletion from birth improves insulin sensitivity, lipid metabolism and decreases endoplasmic reticulum (ER) stress. However, as a therapy

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27

Masaki, Megumi. "Role of meltrin α (ADAM12) in high-fat diet induced obesity". Kyoto University, 2006. http://hdl.handle.net/2433/143843.

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28

Nonnecke, Eric Brian. "Genistein mediates perturbations in one-carbon metabolism during diet-induced folate deficiency." [Ames, Iowa : Iowa State University], 2010. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:1476333.

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29

Mann, John Clifford. "The effects of diet and ionizing radiation on azoxymethane induced colon carcinogenesis." Thesis, Texas A&M University, 2005. http://hdl.handle.net/1969.1/4250.

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The ability of ionizing radiation to enhance colon carcinogenesis and the role of diet in this process has not been documented. We hypothesized that radiation would enhance the formation of aberrant crypt foci, ACF, known precursor lesions to colon cancer, by suppressing apoptosis and upregulating proliferation in colonocytes. Diets contained a combination of fish oil or corn oil and either pectin or cellulose. We exposed 40 male Sprague-Dawley rats to 1 Gy ionizing radiation (1 GeV Fe) 10 d prior to injection with AOM. Colons were resected at the promotion stage of carcinogenesis (7 wk post i

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30

Dagnone, Jeffrey Damon. "Separate effects of diet- and exercise-induced weight loss on insulin sensitivity." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/mq31194.pdf.

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31

Smith, Scott E. (Scott Edward). "The psychological effects of diet induced lowered tryptophan in normal human males /." Thesis, McGill University, 1985. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=71991.

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Biochemical theories postulate that deficient serotonergic functioning may be etiologically related to affective illness and aggressive behavior. In Study I mood and aggressivity were measured in thirty-six normal male subjects before and after ingestion of a Tryptophan Depleted, Tryptophan Loaded or Balanced amino acid mixture. While no differences in aggressivity were found, the Tryptophan Depleted group scored significantly higher at posttest on the MAACL Depression Scale than the control groups and demonstrated selective attention for dysphoric themes. In Study II a Balanced or Tryptophan

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32

Brownlow, Milene Lara. "Diet-Induced Ketosis and Calorie Restriction in Mouse Models of Alzheimer's Pathology." Scholar Commons, 2013. http://scholarcommons.usf.edu/etd/4870.

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Dietary manipulations and their pharmacological outcomes have been increasingly studied in neurodegenerative diseases. However, a systematic comparison among different methods in validated animal models of Alzheimer's disease is made necessary due to several different approaches applied in recent studies. Moreover, despite the large body of evidence on the effects of calorie restriction (CR) and ketogenic diets (KDs) on amyloid pathology, no consistent data is available on the effects of calorie restriction, ketogenic diet or ketone supplements on tau pathology in transgenic models of AD. More

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33

Sharma, Vishakha. "Aging and Gender Effects in Diet-Induced Obesity and its Metabolic Sequelae." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1522320845867142.

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34

Wasserfurth-Grzybowska, Paulina Melanie [Verfasser]. "Exercise- and diet-induced metabolic and physiological adaptations / Paulina Melanie Wasserfurth-Grzybowska." Hannover : Gottfried Wilhelm Leibniz Universität, 2021. http://d-nb.info/1240449712/34.

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35

Umbaugh, David S. "Neuronal Inflammation| A Potential Contributing Mechanism to High Fat Diet-Induced Neuropathy." Thesis, Southern Illinois University at Edwardsville, 2017. http://pqdtopen.proquest.com/#viewpdf?dispub=10276211.

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<p> Neuropathy, a debilitating complication of diabetes, has primarily been attributed to poor glycemic control, but has recently been associated with obesity and the metabolic syndrome in nondiabetic individuals. A robust body of evidence indicates that a high-fat diet can induce signs of neuropathy in mice but the pathogenesis of high fat diet-induced neuropathy remains unknown. </p><p> <b>PURPOSE:</b> To determine if neuronal inflammation is a potential initiating mechanism for the development of mechanical hypersensitivity and nerve fiber changes (signs of neuropathy) in high fat fed mi

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36

Wu, Pei-Chi Coleman Rosalind A. "Will absence of GPAT1 improve diet-induced atherosclerosis in ApoE heterozygous mice?" Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2008. http://dc.lib.unc.edu/u?/etd,1933.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2008.<br>Title from electronic title page (viewed Dec. 11, 2008). "... in partial fulfillment of the requirement for the degree of Master of Science in the Department of Nutrition." Discipline: Nutrition; Department/School: Public Health.

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37

Shin, Andrew Changhun. "Unveiling diet-induced obesity leptin insensitivity and dysregulation of the HPA axis /." Diss., Connect to online resource - MSU authorized users, 2008.

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38

Pilvi, Taru. "Effects of dairy proteins and calcium on diet-induced obesity in mice." Helsinki : University of Helsinki, 2008. http://urn.fi/URN:ISBN:978-952-10-5020-6.

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39

Kheder, Ramiar Kamal. "Immune modulation in the prevention of pathologies relating to diet-induced obesity." Thesis, University of Leicester, 2017. http://hdl.handle.net/2381/39952.

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Non-alcoholic fatty liver disease (NAFLD) results from accumulation of fat in liver, so-called steatosis. Fatty liver may lead to the development of inflammation (steatohepatitis). This study analysed the role of high fat diet, Vitamin D, and complement properdin in mouse models of high fat diet. Properdin knockout mice, and properdin wild type mice on LDLR-/- and LDLR+/+ background were fed a high fat –high sugar diet or Western (high fat) diet. Body weight, fat pad weight, liver histopathology, immunohistochemistry were analysed. Hepatic expression of candidate genes (TNF-α, srebp-1c, TLR4,

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40

Chen, Ting. "LKB1 Regulation of High-Fat Diet-induced Adaptation in Mouse Skeletal Muscle." BYU ScholarsArchive, 2017. https://scholarsarchive.byu.edu/etd/6682.

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Ad libitum high-fat diet (HFD)-induced obesity leads to insulin resistance in skeletal muscle, altered gene expression, and altered growth signaling, all of which contributes to pathological changes in metabolism. Liver kinase B1 (LKB1) is an important metabolism regulator. The purpose of this dissertation was to understand how knocking out LKB1 influences HFD induced adaptations in mouse skeletal muscle. To do so, control and skeletal muscle LKB1 knock-out (LKB1-KO) mice were put on either standard diet (STD) or HFD for 1 week or 14 weeks, or put on the HFD for 14 weeks and then switched to S

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41

楊達志 and Tat-chi Teddy Yang. "Effect of Chinese green tea on diet-induced hypercholesterolemia and arteriosclerosis in rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1998. http://hub.hku.hk/bib/B31237939.

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42

Mai, Anna. "Oxidative stress in high fat diet-induced metabolic syndrome, hypertension and endothelial dysfunction." Thesis, University of Surrey, 2017. http://epubs.surrey.ac.uk/841704/.

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Oxidative stress attributable to the activation of a Nox2-containing NADPH oxidase is involved in dietary obesity-associated cardiovascular diseases. However, the mechanism of Nox2 activation in dietary obesity remains unclear. In this project age-matched apolipoprotein E knockout (ApoEKO) and Nox2/ApoE double knockout (D-KO) mice were used to investigate high-fat diet (HFD)-induced obesity-related metabolic disorders, Nox2 activation, endothelial and adipose tissue dysfunction. Compared to NCD, HFD ApoEKO mice developed insulin resistance, increased systemic oxidative stress and vascular dysf

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43

Dakin, Rachel Sarah. "Effects of postnatal and maternal diet-induced obesity on physiology and vascular function." Thesis, University of Edinburgh, 2012. http://hdl.handle.net/1842/8256.

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In recent years there has been an explosion in the rates of obesity, defined as a body mass index greater than 30kg/ m2, and associated cardiovascular disease. Alterations in peripheral glucocorticoid metabolism have been suggested to play a role in the development of obesity. Obesity occurs in both sexes, but the risk of associated metabolic disturbance and vascular dysfunction is greater in men. Although there is no accepted definition of obesity in rodents, the term is used to describe animals with a significant increase in fat pad mass often achieved by feeding a high fat diet. Although an

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44

Thapar, Divya. "Osteopontin knockout abates high fat diet-induced insulin resistance and adipose tissue inflammation." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2008. http://wwwlib.umi.com/cr/ucsd/fullcit?p1459910.

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Thesis (M.S.)--University of California, San Diego, 2008.<br>Title from first page of PDF file (viewed January 5, 2009). Available via ProQuest Digital Dissertations. Includes bibliographical references (p. 43-46).

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45

Yang, Tat-chi Teddy. "Effect of Chinese green tea on diet-induced hypercholesterolemia and arteriosclerosis in rats /." Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19657560.

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46

Sideratou, Theodora. "Effect of maternal diet on offspring metabolic programming: changes induced by carbohydrate quality." Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/11987.

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Background Maternal diet and gestational diabetes have important implications for offspring health and disease. The fat mass and obesity associated gene (FTO) has the strongest relationship to increased body fat mass in humans, potentially via its effect on appetite and insulin sensitivity. Leptin plays a key role in regulating energy intake and expenditure, including appetite and hunger. Our hypothesis was that the expression of the Fto and leptin gene is influenced by the quality of carbohydrate in the maternal diet. Aim To compare glucose metabolism and mRNA expression of the Fto, leptin

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47

Culotta, Austin M. "A Change in Grain? Diet Induced Plasticity in the Generalist Grasshopper Melanoplus differentialis." ScholarWorks@UNO, 2018. https://scholarworks.uno.edu/td/2551.

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Phenotypic plasticity is favored in heterogeneous environments in which alternative phenotypes can exploit alternative resources. However, it’s not clear whether phenotypic plasticity is useful in environments that become more homogenous over an organism’s life cycle. I studied a population of grasshopper Melanoplus differentialis that experiences high resource diversity as nymphs but low resource diversity as adults to determine if individuals can undergo diet-induced morphological plasticity in head shape to increase biting ability and ingestion of hard diets. Insects on a soft diet were lar

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48

González, Franquesa Alba. "Assessing metabolic plasticity in diet-induced obese mice upon lifestyle intervention. An integrative approach." Doctoral thesis, Universitat de Barcelona, 2015. http://hdl.handle.net/10803/396201.

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Type 2 Diabetes Mellitus (T2DM) is the metabolic disorder that accounts for the presence of hyperglycemia within insulin resistance (IR). The International Diabetes Federation estimated in 2013 that 382 million people (8.3% of world society ) had diabetes and that this number is set to rise beyond 592 million people in the next 22 years. T2DM accounts for 90% of people with diabetes (WHO 1999). Obesity is considered a major risk factor for developing T2DM over time. The World Health Organization (WHO) stated in 2014 that more than 1.9 billion adults were overweight and of these, over 600 milli

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49

Wüest, Stephan. "Activation of Fas (CD95) in adipocytes contributes to high fat diet-induced insulin-resistance /." [S.l.] : [s.n.], 2009. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000282894.

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50

Yang, Lianxing. "Hypothalamic gene expression profiling in mouse strains susceptible or resistant to diet-induced obesity." [S.l.] : [s.n.], 2005. http://archiv.ub.uni-marburg.de/diss/z2005/0062/.

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