Log in

Relevant bibliographies by topics / Diet-induced / Journal articles

To see the other types of publications on this topic, follow the link: Diet-induced.

Journal articles on the topic 'Diet-induced'

Author: Grafiati

Published: 3 June 2025

Last updated: 22 June 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Diet-induced.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Mercer, Julian G., and Zoë A. Archer. "Putting the diet back into diet-induced obesity: Diet-induced hypothalamic gene expression." European Journal of Pharmacology 585, no. 1 (2008): 31–37. http://dx.doi.org/10.1016/j.ejphar.2007.11.077.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

GE, Chang-rong, Si-zheng GAO, Jun-jing JIA, and Mark Jois. "Diet-Induced Thermogenesis." Agricultural Sciences in China 7, no. 9 (2008): 1133–39. http://dx.doi.org/10.1016/s1671-2927(08)60156-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Mansell, P. I., I. A. Macdonald, and I. W. Fellows. "Diet-induced thermogenesis." Clinical Science 72, no. 2 (1987): 259–60. http://dx.doi.org/10.1042/cs0720259.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Millichap, J. Gordon. "Ketogenic Diet-Induced Cardiomyopathy." Pediatric Neurology Briefs 14, no. 7 (2000): 50. http://dx.doi.org/10.15844/pedneurbriefs-14-7-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Clark, Barbara, Mohammad Wisam Baqdunes, and Gregory M. Kunkel. "Diet-induced oxalate nephropathy." BMJ Case Reports 12, no. 9 (2019): e231284. http://dx.doi.org/10.1136/bcr-2019-231284.

Full text

Abstract:

Oxalate nephropathy is a rare condition and may be overlooked due to lack of recognition and understanding of triggers. An 81-year-old man was sent to nephrologist because of significantly increased creatinine (1.5–1.9 mg/dL) noted for 3 months. He had well-controlled diabetes but no history of kidney disease. He had no chronic diarrhoea or intestinal surgery. He was a health-minded individual who had read extensively about benefit of antioxidants. Initial work-up was unrevealing. Within a few weeks after first visit, he developed acute symptomatic worsening kidney injury with nausea, vomiting and creatinine up to 6.8 mg/dL. Repeat examination of the urine sediment revealed casts containing calcium oxalate crystals. A deeper dietary history revealed widespread oxalate precursor consumption. A kidney biopsy confirmed oxalate nephropathy. Restriction of oxalate consumption combined with adequate hydration, oral calcium acetate resulted in partial renal recovery without need for haemodialysis.

APA, Harvard, Vancouver, ISO, and other styles

6

Adeva, María M., and Gema Souto. "Diet-induced metabolic acidosis." Clinical Nutrition 30, no. 4 (2011): 416–21. http://dx.doi.org/10.1016/j.clnu.2011.03.008.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Galef, Bennett G. "Socially induced diet preference can partially reverse a LiCl-induced diet aversion." Animal Learning & Behavior 13, no. 4 (1985): 415–18. http://dx.doi.org/10.3758/bf03208018.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Briggs, D. I., M. B. Lemus, E. Kua, and Z. B. Andrews. "Diet-Induced Obesity Attenuates Fasting-Induced Hyperphagia." Journal of Neuroendocrinology 23, no. 7 (2011): 620–26. http://dx.doi.org/10.1111/j.1365-2826.2011.02148.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Marcos, Rafael, M. Teresa Macarulla, J. Alfredo Martinez, and Jesús Larralde. "Hormonal diet-induced changes in a pea based diet." International Journal of Food Sciences and Nutrition 45, no. 1 (1994): 41–47. http://dx.doi.org/10.3109/09637489409167016.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Seaman, David R. "The diet-induced proinflammatory state:." Journal of Manipulative and Physiological Therapeutics 25, no. 3 (2002): 168–79. http://dx.doi.org/10.1067/mmt.2002.122324.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

DiFeliceantonio, Alexandra G., and Dana M. Small. "Dopamine and diet-induced obesity." Nature Neuroscience 22, no. 1 (2018): 1–2. http://dx.doi.org/10.1038/s41593-018-0304-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

12

Mickleborough, Timothy D., Martin Lindley, and Ren-Jay Shei. "Diet and Exercise-induced Bronchoconstriction." American Journal of Respiratory and Critical Care Medicine 188, no. 12 (2013): 1469–70. http://dx.doi.org/10.1164/rccm.201309-1598le.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Ming-Chai, Chen. "Diet-Induced Pancreatitis in China." Journal of Clinical Gastroenterology 8, no. 6 (1986): 611–12. http://dx.doi.org/10.1097/00004836-198612000-00003.

Full text

APA, Harvard, Vancouver, ISO, and other styles

14

Weiß, Katharina T., Sigrid Karrer, Michael Landthaler, Philipp Babilas, and Stephan Schreml. "Diet-induced pigmented purpuric dermatosis." Australasian Journal of Dermatology 55, no. 3 (2013): e51-e53. http://dx.doi.org/10.1111/ajd.12038.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Mickleborough, Timothy D., and Martin R. Lindley. "Diet and Exercise-Induced Bronchoconstriction." Chest 130, no. 2 (2006): 623–24. http://dx.doi.org/10.1378/chest.130.2.623-a.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Parsons, Jonathan P., and John G. Mastronarde. "Diet and Exercise-Induced Bronchoconstriction." Chest 130, no. 2 (2006): 624. http://dx.doi.org/10.1378/chest.130.2.623-b.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Lee, Young, May-Yun Wang, Tetsuya Kakuma, et al. "Liporegulation in Diet-induced Obesity." Journal of Biological Chemistry 276, no. 8 (2000): 5629–35. http://dx.doi.org/10.1074/jbc.m008553200.

Full text

APA, Harvard, Vancouver, ISO, and other styles

18

Beck, Y. B., A. Burlet, J. P. Nicolas, and C. Burlet. "Diet-induced overeating and neuropeptide." Appetite 19, no. 2 (1992): 162. http://dx.doi.org/10.1016/0195-6663(92)90025-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

19

Setti, Sharay E., Alyssa M. Littlefield, Samantha W. Johnson, and Rachel A. Kohman. "Diet-induced obesity attenuates endotoxin-induced cognitive deficits." Physiology & Behavior 141 (March 2015): 1–8. http://dx.doi.org/10.1016/j.physbeh.2014.12.036.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Ramadan, M. F. "Physalis peruviana pomace suppresses highcholesterol diet-induced hypercholesterolemia in rats." Grasas y Aceites 63, no. 4 (2012): 411–22. http://dx.doi.org/10.3989/gya.047412.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

Koya-Miyata, Satomi, Norie Arai, Akiko Mizote, et al. "Propolis Prevents Diet-Induced Hyperlipidemia and Mitigates Weight Gain in Diet-Induced Obesity in Mice." Biological & Pharmaceutical Bulletin 32, no. 12 (2009): 2022–28. http://dx.doi.org/10.1248/bpb.32.2022.

Full text

APA, Harvard, Vancouver, ISO, and other styles

22

Sikora, Michael A., Tetsuo Morizono, W. Dixon Ward, Michael M. Paparella, and Kimberly Leslie. "Diet-induced Hyperlipidemia and Auditory Dysfunction." Acta Oto-Laryngologica 102, no. 5-6 (1986): 372–81. http://dx.doi.org/10.3109/00016488609119420.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Lecube, Albert, and Carolina López-Cano. "Obesity, a Diet-Induced Inflammatory Disease." Nutrients 11, no. 10 (2019): 2284. http://dx.doi.org/10.3390/nu11102284.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Thompson, Michael D., and Brian J. DeBosch. "Maternal Fructose Diet-Induced Developmental Programming." Nutrients 13, no. 9 (2021): 3278. http://dx.doi.org/10.3390/nu13093278.

Full text

Abstract:

Developmental programming of chronic diseases by perinatal exposures/events is the basic tenet of the developmental origins hypothesis of adult disease (DOHaD). With consumption of fructose becoming more common in the diet, the effect of fructose exposure during pregnancy and lactation is of increasing relevance. Human studies have identified a clear effect of fructose consumption on maternal health, but little is known of the direct or indirect effects on offspring. Animal models have been utilized to evaluate this concept and an association between maternal fructose and offspring chronic disease, including hypertension and metabolic syndrome. This review will address the mechanisms of developmental programming by maternal fructose and potential options for intervention.

APA, Harvard, Vancouver, ISO, and other styles

25

Koh, Gwanpyo. "Rodent Models of Diet-induced Obesity." Korean Journal of Obesity 25, no. 2 (2016): 45–49. http://dx.doi.org/10.7570/kjo.2016.25.2.45.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Millichap, J. Gordon. "Diet, Carnitine, and Valproate-Induced Ammonemia." Pediatric Neurology Briefs 11, no. 9 (1997): 66. http://dx.doi.org/10.15844/pedneurbriefs-11-9-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Cully, Megan. "Adenosine protects from diet-induced obesity." Nature Reviews Drug Discovery 13, no. 12 (2014): 886–87. http://dx.doi.org/10.1038/nrd4490.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Romon, M., J. L. Edme, C. Boulenguez, J. L. Lescroart, and P. Frimat. "Circadian variation of diet-induced thermogenesis." American Journal of Clinical Nutrition 57, no. 4 (1993): 476–80. http://dx.doi.org/10.1093/ajcn/57.4.476.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

Chang, Richard Cheng-An, Liheng Shi, Cathy Chia-Yu Huang, et al. "High-Fat Diet–Induced Retinal Dysfunction." Investigative Opthalmology & Visual Science 56, no. 4 (2015): 2367. http://dx.doi.org/10.1167/iovs.14-16143.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Rosenberg, Eugene, Ilana Zilber-Rosenberg, Gil Sharon, and Daniel Segal. "Diet-induced mating preference in Drosophila." Proceedings of the National Academy of Sciences 115, no. 10 (2018): E2153. http://dx.doi.org/10.1073/pnas.1721527115.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Chan, Yee Kwan, Mehrbod Estaki, and Deanna L. Gibson. "Clinical Consequences of Diet-Induced Dysbiosis." Annals of Nutrition and Metabolism 63, s2 (2013): 28–40. http://dx.doi.org/10.1159/000354902.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

Retamero, Carolina, Tenesa Rivera, and Kevin Murphy. "“Ephedra-Free” Diet Pill-Induced Psychosis." Psychosomatics 52, no. 6 (2011): 579–82. http://dx.doi.org/10.1016/j.psym.2011.06.003.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

Greenhill, Claire. "New strategy for diet-induced obesity." Nature Reviews Endocrinology 10, no. 2 (2013): 65. http://dx.doi.org/10.1038/nrendo.2013.250.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

Dirkx, Ellen, Robert W. Schwenk, Jan F. C. Glatz, Joost J. F. P. Luiken, and Guillaume J. J. M. van Eys. "High fat diet induced diabetic cardiomyopathy." Prostaglandins, Leukotrienes and Essential Fatty Acids (PLEFA) 85, no. 5 (2011): 219–25. http://dx.doi.org/10.1016/j.plefa.2011.04.018.

Full text

APA, Harvard, Vancouver, ISO, and other styles

35

Mitsutake, Susumu, Hazuki Yokota, Kota Zama, et al. "SMS2 deficiency prevents diet-induced obesity." Chemistry and Physics of Lipids 163 (August 2010): S23. http://dx.doi.org/10.1016/j.chemphyslip.2010.05.069.

Full text

APA, Harvard, Vancouver, ISO, and other styles

36

Carbone, Salvatore, Adolfo G. Mauro, Stefano Toldo, and Antonio Abbate. "Diet-Induced Obesity HFpEF Murine Models." JACC: Basic to Translational Science 3, no. 1 (2018): 157. http://dx.doi.org/10.1016/j.jacbts.2018.01.004.

Full text

APA, Harvard, Vancouver, ISO, and other styles

37

STORLIEN, L. H., D. A. PAN, A. D. KRIKETOS, and L. A. BAUR. "High Fat Diet-Induced Insulin Resistance." Annals of the New York Academy of Sciences 683, no. 1 Dietary Lipid (1993): 82–90. http://dx.doi.org/10.1111/j.1749-6632.1993.tb35694.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

38

Kirk, Claudia A., J. Lee Beverly, Robert C. Ritter, et al. "Diet-Induced Cholecystokinin Release in Cats." Journal of Nutrition 124, suppl_12 (1994): 2670S—2671S. http://dx.doi.org/10.1093/jn/124.suppl_12.2670s.

Full text

APA, Harvard, Vancouver, ISO, and other styles

39

de Gritz, B. G., and T. Rahko. "DIET-INDUCED RESIDUAL FORMATION IN PIGS." Gerontology 41, no. 2 (1995): 305–18. http://dx.doi.org/10.1159/000213752.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Poizot-Martin, Isabelle, Kaled Benourine, Patrick Philibert, et al. "Diet-induced thermogenesis in HIV infection." AIDS 8, no. 4 (1994): 501–4. http://dx.doi.org/10.1097/00002030-199404000-00013.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Della Guardia, Lucio, Carla Roggi, and Hellas Cena. "Diet-induced acidosis and alkali supplementation." International Journal of Food Sciences and Nutrition 67, no. 7 (2016): 754–61. http://dx.doi.org/10.1080/09637486.2016.1198889.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Hoorn, Ewout J., Dominique M. Bovée, Daniël A. Geerse, and Wesley J. Visser. "Diet-Exercise-Induced Hypokalemic Metabolic Alkalosis." American Journal of Medicine 133, no. 11 (2020): e667-e669. http://dx.doi.org/10.1016/j.amjmed.2020.04.019.

Full text

APA, Harvard, Vancouver, ISO, and other styles

43

Weisinger, R. S., L. Stahl, D. P. Begg, M. Jois, A. Desai, and J. Smythe. "Molasses extract decreases diet-induced obesity." Appetite 57 (July 2011): S46. http://dx.doi.org/10.1016/j.appet.2011.05.291.

Full text

APA, Harvard, Vancouver, ISO, and other styles

44

Bauer, John E. "Diet-induced alterations of lipoprotein metabolism." Journal of the American Veterinary Medical Association 201, no. 11 (1992): 1691–94. http://dx.doi.org/10.2460/javma.1992.201.11.1691.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

Goraya, Nimrit, and Donald E. Wesson. "Pathophysiology of Diet-Induced Acid Stress." International Journal of Molecular Sciences 25, no. 4 (2024): 2336. http://dx.doi.org/10.3390/ijms25042336.

Full text

Abstract:

Diets can influence the body’s acid–base status because specific food components yield acids, bases, or neither when metabolized. Animal-sourced foods yield acids and plant-sourced food, particularly fruits and vegetables, generally yield bases when metabolized. Modern diets proportionately contain more animal-sourced than plant-sourced foods, are, thereby, generally net acid-producing, and so constitute an ongoing acid challenge. Acid accumulation severe enough to reduce serum bicarbonate concentration, i.e., manifesting as chronic metabolic acidosis, the most extreme end of the continuum of “acid stress”, harms bones and muscles and appears to enhance the progression of chronic kidney disease (CKD). Progressive acid accumulation that does not achieve the threshold amount necessary to cause chronic metabolic acidosis also appears to have deleterious effects. Specifically, identifiable acid retention without reduced serum bicarbonate concentration, which, in this review, we will call “covert acidosis”, appears to cause kidney injury and exacerbate CKD progression. Furthermore, the chronic engagement of mechanisms to mitigate the ongoing acid challenge of modern diets also appears to threaten health, including kidney health. This review describes the full continuum of “acid stress” to which modern diets contribute and the mechanisms by which acid stress challenges health. Ongoing research will develop clinically useful tools to identify stages of acid stress earlier than metabolic acidosis and determine if dietary acid reduction lowers or eliminates the threats to health that these diets appear to cause.

APA, Harvard, Vancouver, ISO, and other styles

46

L'Abbé, Mary R., Keith D. Trick, and Bartholomeus Belonje. "Diet-Induced Nephrocalcinosis in Female Rats." Journal of Nutrition 126, no. 11 (1996): 2940. https://doi.org/10.1093/jn/126.11.2940.

Full text

APA, Harvard, Vancouver, ISO, and other styles

47

Cheng, J., L. Chen, S. Han, L. Qin, N. Chen, and Zhongxiao Wan. "Treadmill running and rutin reverse high fat diet induced cognitive impairment in diet induced obese mice." Journal of nutrition, health & aging 20, no. 5 (2015): 503–8. http://dx.doi.org/10.1007/s12603-015-0616-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Langhi, C., Y. F. Otero, F. Le Joubioux, B. Guigas, S. Peltier, and P. Sirvent. "TOTUM-070 prevents diet-induced hypercholesterolemia in Western diet fed mice." Atherosclerosis 355 (August 2022): 82. http://dx.doi.org/10.1016/j.atherosclerosis.2022.06.471.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Sakaguchi, Yusuke, Takayuki Hamano, Isao Matsui, et al. "Low magnesium diet aggravates phosphate-induced kidney injury." Nephrology Dialysis Transplantation 34, no. 8 (2018): 1310–19. http://dx.doi.org/10.1093/ndt/gfy358.

Full text

Abstract:

Abstract Background Magnesium is known to protect against phosphate-induced tubular cell injuries in vitro. We investigated in vivo effects of magnesium on kidney injuries and phosphate metabolism in mice exposed to a high phosphate diet. Methods Heminephrectomized mice were maintained on a high phosphate/normal magnesium diet or a high phosphate/low magnesium diet for 6 weeks. We compared renal histology, phosphaturic hormones and renal α-Klotho expression between the two diet groups. Results High phosphate diet–induced tubular injuries and interstitial fibrosis were remarkably aggravated by the low-magnesium diet. At 1 week after high phosphate feeding when serum creatinine levels were similar between the two groups, the low magnesium diet suppressed not only fecal phosphate excretion but also urinary phosphate excretion, resulting in increased serum phosphate levels. Parathyroid hormone (PTH) levels were not appropriately elevated in the low magnesium diet group despite lower 1,25-dihydroxyvitamin D and serum calcium levels compared with the normal magnesium diet group. Although fibroblast growth factor 23 (FGF23) levels were lower in the low magnesium diet group, calcitriol-induced upregulation of FGF23 could not restore the impaired urinary phosphate excretion. The low magnesium diet markedly downregulated α-Klotho expression in the kidney. This downregulation of α-Klotho occurred even when mice were fed the low phosphate diet. Conclusions A low magnesium diet aggravated high phosphate diet–induced kidney injuries. Impaired PTH secretion and downregulation of renal α-Klotho were likely to be involved in the blunted urinary phosphate excretion by the low magnesium diet. Increasing dietary magnesium may be useful to attenuate phosphate-induced kidney injury.

APA, Harvard, Vancouver, ISO, and other styles

50

Zitnan, R., S. Kuhla, P. Sanftleben, et al. "Diet induced ruminal papillae development in neonatal calves not correlating with rumen butyrate." Veterinární Medicína 50, No. 11 (2012): 472–79. http://dx.doi.org/10.17221/5651-vetmed.

Full text

Abstract:

The objective of this study was to investigate the development of rumen mucosa and the level of plasma IGF-1 in calves induced by different amounts and types of milk replacers and solid diet. Forty-five male Holsteincalves 7 days of age were assigned to three groups: group I milk free replacer, late weaned; group II milk free replacer, early weaned, and group III milk replacer, early weaned. All animals received additional concentrate, water and maize silage were offered ad libitum. In each group, three calves were slaughtered at 41 days of age. The concentration of ruminal total SCFA and the molar proportion of butyrate did not differ between the groups, but the molar proportion of acetate was lower (P = 0.01) and the proportion of propionate was higher (P = 0.02) in early weaned calves. Compared to the late weaned calves (group I) the length, width and surface of the papillae of atrium ruminis, the length and width of the papillae of ventral ruminal sac and the length of the papillae of ventral blind sac were greater (P &lt; 0.05) in the early weaned calves fed low amounts of milk and high amounts of concentrate (group III). Furthermore, there was a tendency of plasma IGF-1 concentration to be increased (P&nbsp;= 0.1) in early weaned calves. The plasma levels of glucose and insulin were decreased (P &lt; 0.01, and P = 0.03, respectively). Positive correlations existed between papillae length and plasma IGF-1 concentrations (P &lt; 0.10). Insulin and glucose concentrations were negatively correlated with parameters of papillae development (P &lt; 0.1). In conclusion, the development of rumen papillae was stimulated in calves consuming increased amounts of concentrate. The effect was not correlated with the molar proportion of butyrate, but with the molar propionate proportion in the rumen and with the plasma IGF-1 concentration

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!