Relevant bibliographies by topics / Dietary compounds

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Dietary compounds'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Dietary compounds"

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Banikazemi, Zarrin, Hemat Aghagolzadeh Haji, Mohsen Mohammadi, Gholamreza Taheripak, Elmira Iranifar, Mohsen Poursadeghiyan, Abdullah Moridikia, Bahman Rashidi, Mohsen Taghizadeh, and Hamed Mirzaei. "Diet and cancer prevention: Dietary compounds, dietary MicroRNAs, and dietary exosomes." Journal of Cellular Biochemistry 119, no. 1 (October 4, 2017): 185–96. http://dx.doi.org/10.1002/jcb.26244.

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Kuhnle, G. G. C., and S. A. Bingham. "Dietary meat, endogenous nitrosation and colorectal cancer." Biochemical Society Transactions 35, no. 5 (October 25, 2007): 1355–57. http://dx.doi.org/10.1042/bst0351355.

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Colorectal cancer is the third most common cancer in developed countries such as the U.K., but incidence rates around the world vary approx. 20-fold. Diet is thought to be a key factor determining risk: red and processed meat, but not white meat or fish, are associated with an increased risk of colorectal cancer. The endogenous formation of N-nitroso compounds is a possible explanation because red and processed meat, but not white meat or fish, cause a dose-dependent increase in faecal ATNCs (apparent total N-nitroso compounds) and the formation of nitroso-compound-specific DNA adducts in humans. Red meat is particularly rich in haem which has been found to promote the endogenous formation of ATNC. Nitrosyl haem and nitroso thiols have been identified as major constituents of both faecal and ileal ATNC with a significant increase in the formation of these compounds following a diet rich in red meat. In vitro incubations show that, under simulated gastric conditions, nitroso thiols are the main species of nitroso compound formed, suggesting that acid-catalysed thionitrosation is the initial step in the endogenous formation of nitroso compounds. Nitrosyl haem and other nitroso compounds can then form under the alkaline and reductive conditions of the small and large bowel.
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Smith, Annalise, and Samita Andreansky. "Antitumor Immunity and Dietary Compounds." Medical Sciences 2, no. 1 (December 27, 2013): 1–22. http://dx.doi.org/10.3390/medsci2010001.

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Lapčík, O. "Endocrinological aspects of dietary habits." Czech Journal of Food Sciences 22, No. 1 (November 16, 2011): 29–38. http://dx.doi.org/10.17221/3404-cjfs.

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Dietary habits reflect both the recent economic possibilities and the cultural history of individual human populations. They may influence endocrine systems and thus affect the health of the respective populations in several manners: (1) People consuming exclusively local products may lack certain micronutrients. This is important especially in areas with low levels of iodine and/or selenium in the environment. Thyroid gland insufficiency resulting from the iodine deficiency was widespread in many areas of Central Europe until the introduction of iodine supplementation in the second half of 20&lt;sup&gt;<sup>th</sup> &lt;/sup&gt;century. Iodine deficiency is still a serious problem in many areas of Africa and Asia. (2) Numerous cultural plants contain compounds able to influence important metabolic pathways. Iodine deficiency is usually worsened by thyroidal peroxidase inhibitors, so-called goitrogens. Phenolic and terpenoid compounds may interfere in the metabolism of steroid hormones. Glycyrrhetinic acid from licorice is a potent inhibitor of 11-beta-hydroxysteroid dehydrogenase. Isoflavonoids from legumes (e.g. genistein and daidzein) and their metabolites (e.g. equol) were found to inhibit the following enzymes: aromatase, 5alfa-reductase, 7alfa-hydroxylase, 3beta-hydroxysteroid and 17beta-hydroxysteroid dehydrogenases, etc. Isoflavonoid sulphates influence local availability of steroids by inhibiting sterol sulphatases. (3) Plant-derived compounds are able to interact with nuclear receptors and act either as hormone agonists or as antagonists. Recently, the attention has been paid namely to the phenolic substances interacting with oestrogen receptors so-called phyto-oestrogens. &nbsp;
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Hughes, Claude L., and Tilak R. Dhiman. "Dietary Compounds in Relation to Dietary Diversity and Human Health." Journal of Medicinal Food 5, no. 2 (June 2002): 51–68. http://dx.doi.org/10.1089/109662002760178131.

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Noh, Choi, Hwang, Jung, Kim, and Kim. "Dietary Compounds for Targeting Prostate Cancer." Nutrients 11, no. 10 (October 8, 2019): 2401. http://dx.doi.org/10.3390/nu11102401.

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Prostate cancer is the third most common cancer worldwide, and the burden of the disease is increased. Although several chemotherapies have been used, concerns about the side effects have been raised, and development of alternative therapy is inevitable. The purpose of this study is to prove the efficacy of dietary substances as a source of anti-tumor drugs by identifying their carcinostatic activities in specific pathological mechanisms. According to numerous studies, dietary substances were effective through following five mechanisms; apoptosis, anti-angiogenesis, anti-metastasis, microRNA (miRNA) regulation, and anti-multi-drug-resistance (MDR). About seventy dietary substances showed the anti-prostate cancer activities. Most of the substances induced the apoptosis, especially acting on the mechanism of caspase and poly adenosine diphosphate ribose polymerase (PARP) cleavage. These findings support that dietary compounds have potential to be used as anticancer agents as both food supplements and direct clinical drugs.
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Langner, Ewa, and Wojciech Rzeski. "Dietary derived compounds in cancer chemoprevention." Współczesna Onkologia 5 (2012): 394–400. http://dx.doi.org/10.5114/wo.2012.31767.

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Williamson, Gary, Andrea J. Day, Geoff W. Plumb, and Delphine Couteau. "Human metabolism of dietary chemopreventive compounds." Biochemical Society Transactions 28, no. 1 (February 1, 2000): A7. http://dx.doi.org/10.1042/bst028a007b.

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Vitaglione, Paola, Filomena Morisco, Nicola Caporaso, and Vincenzo Fogliano. "Dietary Antioxidant Compounds and Liver Health." Critical Reviews in Food Science and Nutrition 44, no. 7-8 (February 10, 2005): 575–86. http://dx.doi.org/10.1080/10408690490911701.

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Tako, Elad. "Dietary Plant-Origin Bio-Active Compounds, Intestinal Functionality, and Microbiome." Nutrients 12, no. 11 (October 22, 2020): 3223. http://dx.doi.org/10.3390/nu12113223.

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In recent years, plant-origin bio-active compounds in foods (staple crops, fruit, vegetables, and others) have been gaining interest, and processes to consider them for public health recommendations are being presented and discussed in the literature. However, at times, it may be challenging to demonstrate causality, and there often is not a single compound–single effect relationship. Furthermore, it was suggested that health benefits may be due to metabolites produced by the host or gut microbiome rather than the food constituent per se. Over the years, compounds that were investigated were shown to increase gut microbial diversity, improve endothelial function, improve cognitive function, reduce bone loss, and many others. More recently, an additional and significant body of evidence further demonstrated the nutritional role and potential effects that plant-origin bio-active compounds might have on intestinal functionality (specifically the duodenal brush border membrane, morphology, and the abundance of health-promoting bacterial populations). Hence, the special issue “Dietary Plant-Origin Bio-Active Compounds, Intestinal Functionality, and Microbiome” comprises 11 peer-reviewed papers on the most recent evidence regarding the potential dietary intake and effects of plant-origin bio-active compounds on intestinal functionality, primarily in the context of brush border functional proteins (enzymes and transporters), mineral (and other nutrients) dietary bioavailability, and the intestinal microbiome. Original contributions and literature reviews further demonstrated the potential dietary relevance that plant bio-active compounds hold in human health and development. This editorial provides a brief and concise overview that addresses and summarizes the content of the Dietary Plant-Origin Bio-Active Compounds, Intestinal Functionality, and Microbiome special issue.
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Dissertations / Theses on the topic "Dietary compounds"

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Ostertag, Sonja. "Estimated dietary exposure to perfluorinated compounds in Canada." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112549.

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Perfluorinated carboxylates (PFCAs), sulfonates (PFSs) and perfluoroalkylsulfonamides (PFOSAs) have been detected in whole blood and serum of non-occupationally exposed humans, yet sources of exposure have not been fully elucidated. The objectives of this study were to estimate dietary exposure to perfluorooctane sulfonate (PFOS), PFCAs (C7--C 11) and fluorotelomer unsaturated carboxylates (FTUCAs) for the general Canadian and Inuit populations prior to the phase-out of perfluorooctyl-sulfonyl production by 3M and voluntary reductions in PFOA emissions under the PFOA stewardship program. PFCs were measured in 65 archived composite food samples prepared for the 1998 Canadian Total Diet Study (TDS) and 68 archived traditional foods from Nunavut using a newly developed methanol extraction combined with a solid phase extraction clean up. Dietary exposure was estimated using food intake data available from studies carried out between 1997 and 1998 in southern Canada and Nunavut.
PFCs were detected in eight composite food samples from the Canadian TDS and in 61 traditional food samples. Elevated concentrations of PFCs were found in caribou liver (6.2+/-5.5 ng/g), ringed seal liver (7.7, 10.2 ng/g), polar bear meat (7.0 ng/g), beluga meat (7.0, 5.8 ng/g), luncheon meats (5.02 ng/g), cookies (2.7 ng/g), processed cheese (2.1 ng/g) and peppers (1.8 ng/g). Low levels of total PFCs (<1.5 ng/g) were measured in 41 traditional foods including: meat (caribou, ptarmigan, snow goose, bearded seal, walrus, black duck), berries, and fish (lake trout, arctic char). PFCs were not detected in beverages, unprocessed meats, breads, cereals and fruits from the TDS composite samples analyzed.
The ranges of estimated daily exposure to PFCs were between 2 and 59 ng-person-1 and 210 to 610 ng-person-1 for average Canadians and Inuit in Nunavut respectively. There were no statistically significant differences in mean PFC exposure levels for different age and gender groups in the general Canadian population. Inuit men in the 41 to 60 year old age group had statistically significantly higher estimated daily exposure to PFCs (p<0.05) than younger men and women from the same age group. This higher exposure was associated with the consumption of beluga muktuk, caribou liver and bearded seal intestine.
Traditional foods contributed a higher percentage to PFC exposure than market foods in all age and gender groups for the Inuit population. In general, caribou meat, arctic char meat and cookies contributed most to dietary exposure for Inuit, with caribou flesh contributing 43 to 75 percent to daily PFC dietary exposure. Dietary exposure for the general Canadian population was associated with the consumption of cakes and cookies, processed cheese, and regular cheese.
Levels of dietary exposure to PFCs estimated in these studies do not pose any significant health risk to either population based on current toxicological information.
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Geraets, Liesbeth. "Dietary PARP-1 inhibitors as anti-inflammatory compounds." Maastricht : Maastricht : Universitaire Pers ; University Library, Universiteit Maastricht [host], 2008. http://arno.unimaas.nl/show.cgi?fid=14252.

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Rich, Clive George. "Morphine-like compounds in brain tissue and dietary materials." Thesis, University of Sunderland, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.328983.

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Schwartzman, Judy Susan. "Dietary N-nitroso compounds and the risk of brain tumours in adults." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq28753.pdf.

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Morton, Lincoln William. "The role of dietary phenolic compounds in the detoxification of reactive nitrogen species." University of Western Australia. Dept. of Medicine, 2003. http://theses.library.uwa.edu.au/adt-WU2003.0026.

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[Truncated abstract. Please see the pdf format for the complete text.] Interest in the role of peroxynitrite in the pathogenesis of atherosclerosis has increased due to many in vitro studies which have demonstrated its potent oxidising and nitrating capability and immunohistochemical staining studies which demonstrate nitration of tyrosine in vivo. It is frequently suggested that the production of nitric oxide and superoxide at sites of inflammation implicates peroxynitrite as the major damaging reactive nitrogen species in vivo. Evidence for a role for peroxynitrite is often demonstrated by measurement of 3-nitrotyrosine yet even this cannot distinguish peroxynitrite from other nitrating species. Clearly, however, if peroxynitrite is important in atherogenesis, then identification of mechanisms for its detoxification could provide a means of preventing such effects. Therefore, this Thesis has sought to determine whether phenolic compounds of dietary origin can be preferentially nitrated by reactive nitrogen species thereby protecting endogenous structures, such as low density lipoproteins, from atherogenic modifications. This Thesis focuses upon phenolic acids as they have received relatively less attention than other classes of phenolic compounds, such as flavonoids, yet they are quite abundant in socially important beverages such as red wine. In order to complete the required analyses, the development of methods to detect phenolic acids and their nitration products together with 3-nitrotyrosine, dityrosine and 5-nitro-γ-tocopherol was necessary. The initial in vitro experiments described herein sought to determine the products of reaction of peroxynitrite with phenolic acids of the 4-hydroxy and 3,4-dihydroxy type and then to examine whether these products could account for a protective effect upon tyrosine, lipids and endogenous anti-oxidants, if any was observed, when isolated LDL was treated with SIN-1, which releases peroxynitrite through the simultaneous generation of nitric oxide and superoxide. A concurrent minor focus was to examine the relationship between structure and activity of these phenolic acids under various regimes of oxidative insult. These experiments indicate that, at least in this in vitro model, oxidation is a dominant mechanism over nitration. Peroxynitrite was shown to nitrate coumaric acid in moderate yields but exclusive oxidation of caffeic acid appeared to occur. Although a potential role for γ-tocopherol as an anti-nitration agent was inferred, all types of chemical treatment of LDL in the presence of phenolic acids yielded oxidation as the primary end point. In fact, nitration of tyrosine was not detected and nitration of coumaric acid was at the limit of detection. Since nitration of tyrosine is generally regarded as important in many disease states, a more physiological nitrating mechanism involving artificially stimulated neutrophils was used. This system demonstrated that although physiologically relevant reactive nitrogen species can result in nitration of phenolic compounds, in a complex system including biological structures (LDL) and phenolic compounds, oxidation but not nitration of all species appears to occur. As a consequence of the results above, an examination of carotid plaque was undertaken to determine to what extent nitration occurred relative to oxidation in atherosclerotic tissue. These studies applied methods developed herein to detect 3-nitrotyrosine and dityrosine in complex biological matrices as markers of nitration and oxidation respectively. The data obtained demonstrated that nitration was a minor modification of protein (0.01%) compared to oxidation (0.3%) even in a highly diseased tissue such as carotid artery plaque. A secondary study examining plasma revealed that dityrosine, which has been implicated in irreversible albumin aggregation in chronic renal failure and more recently in heart disease, is elevated in chronic renal failure subjects compared to well matched controls. A separate examination of plasma from healthy subjects revealed that in both the fasting and post prandial state 3-nitrotyrosine could not be detected and, in fact, interfering species could be problematic in the GC-MS analysis of 3-nitrotyrosine. The lack of nitration of any substrate observed in vitro using reactive nitrogen species generated in the aqueous phase, the relative lack of nitration of tyrosine in plaque proteins and the lipophilicity of nitric oxide, the precursor of all reactive nitrogen species, suggested that nitration could be more closely associated with lipid structures. The known ability of γ-tocopherol to form 5-nitro-γ-tocopherol was used to probe this concept. The 5-nitro-γ-tocopherol content of lipid extracts obtained from carotid artery plaques was very high (30%). This indicated that nitration is predominantly a lipid phase phenomenon and that nitrating species are present in much greater abundance than oxidising species in vivo.
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Frank, Jan. "Dietary phenolic compounds and vitamin E bioavailability : model studies in rats and humans /." Uppsala : Dept. of Microbiology, Swedish Univ. of Agricultural Sciences, 2004. http://epsilon.slu.se/a446.pdf.

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Rylander, Lars. "Dietary exposure to persistent organochlorine compounds and health effects in women and their infants epidemiological studies on birthweight, cancer incidence and mortality /." Lund : Dept. of Occupational and Environmental Medicine, Institute of Laboratory Medicine, Lund University, 1997. http://books.google.com/books?id=rgFrAAAAMAAJ.

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Sgwane, Thulile Seipone. "Effect of essential oil compounds on metabolism of dietary polyunsaturated fatty acids by ruminal microorganisms in vitro." Thesis, University of Aberdeen, 2015. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227111.

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Ruminant products, meat and milk, were long ago discovered to contain a high content of saturated fat associated with various chronic diseases in human, including coronary heart disease. Increased consumption of n-3 fatty acids, linolenic (LNA; 18:3n-3), eicosapentaenoic acid (EPA; 20:5n-3) and docosahexaenoic acid (DHA; 22:6n-3) by man has been linked with reduced incidences of these diseases. Therefore, in vitro experiments carried out in this thesis investigated the possibility that the dietary inclusion of essential oil compounds (EOC) might modify ruminal biohydrogenation of polyunsaturated fatty acids (PUFA), and thereby improve the fatty acid profiles of ruminant derived foods. In the first experiment, twenty EOC were screened, of which carvacrol, thymol, cis/trans-citral, geraniol, nerol, (+)-α-pinene, l-menthone, linalyl acetate and (R)-(+)-pulegone were most effective at protecting the PUFA from biohydrogenation. The reduced accumulation of stearic acid with concomitant increases in vaccenic acid following the addition of the EOC confirmed their capacity to inhibit biohydrogenation of both LA and LNA. However, the observed decreases in total volatile fatty acids concentrations suggested that the inclusion of these compounds at 300 mg l-1 also suppressed microbial fermentation. Of these EOC, carvacrol and thymol were the most potent inhibitors of fermentation as they decreased total VFA concentrations by ≥60% from those of the control. In the second experiment, selected EOC, namely linalyl acetate, l-menthone and (+)-α-pinene, were tested at lower doses. It was observed that these EOC when added at 100 and 200 mg l-1 also inhibited biohydrogenation with minimal effect on VFA concentration. In experiment 3, pure culture work was undertaken to assess the effects of EOC on three key Butyrivibrio spp., involved in the biohydrogenation process, namely B. fibrisolvens, B. hungatei and B. proteoclasticus. The EOC inhibited biohydrogenation through their bacteriostatic effects on these bacteria. In conclusion, these studies revealed that EOC have the potential to modify ruminal biohydrogenation to varying extents depending on the type of compound used and the dose applied. Consequently, they might be useful at improving the healthiness of ruminant products, however their effects have to be confirmed in vivo and ensure that they do not cause significant inhibition of fibre digestion.
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Zhang, Zhenxian. "ASSESSMENT OF THE FEASIBILITY OF CO-ADMINISTRATION OF PHENOLIC DIETARY COMPOUNDS WITH PHENYLEPHRINE TO INCREASE ITS BIOAVAILABILITY." VCU Scholars Compass, 2013. http://scholarscompass.vcu.edu/etd/539.

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R-(-)-Phenylephrine (PE) is the most commonly used nonprescription oral nasal decongestant in the United States. It is a selective α1-adrenergic receptor agonist and has many years of safe usage. However, the efficacy of PE is controversial, due to its extensive pre-systemic metabolism, which leads to low and variable oral bioavailability (38 ± 9%, mean ± SD). Sulfation plays a very important role in pre-systemic metabolism of PE. The sulfation of PE occurs at its phenolic group, which is the preferred structural feature of many sulfotransferase (SULT) substrates. Compounds with phenolic groups have similar structures to PE, which may share the same SULT isoforms with PE and have the potential to inhibit PE sulfation. Co-administration of the phenolic compounds from the Food and Drug Administration’s (FDA) “Generally Recognized as Safe” (GRAS) list, Everything Added to Food in the United States (EAFUS), or dietary supplements along with PE could be an effective strategy to inhibit the pre-systemic sulfation of PE. The primary side effect of PE is hypertension. Since monoamine oxidase (MAO) inhibitors may increase the risk of hypertension, they should not be taken with PE. In order to increase the oral bioavailability and eventually improve the efficacy of PE, this research project aimed to investigate the feasibility of inhibiting the pre-systemic sulfation of PE with phenolic dietary compounds. Considering the safety issue, this research project also aimed to investigate whether these phenolic dietary compounds have inhibitory effects on MAO-A/B. A human colon adenocarcinoma epithelial cell line (LS180), which shows sulfation activity, was used as a model to test the effect of these phenolic compounds on the sulfation of PE. The extent of disappearance of PE was significantly (p < 0.05) decreased to the following (mean ± SEM, as % of control) when incubated with phenolic dietary compounds in LS180 cells for 14 - 19 hrs: curcumin 24.5 ± 14.0%, guaiacol 51.3 ± 8.0%, isoeugenol 73.9 ± 4.3%, pterostilbene 70.6 ± 4.2%, resveratrol 14.2 ± 28.0%, zingerone 52.4 ± 14.6%, and the combinations eugenol + propylparaben 42.6 ± 8.4%, vanillin + propylparaben 37.0 ± 11.2%, eugenol + propylparaben + vanillin + ascorbic acid 31.1 ± 10.9%, eugenol + vanillin 57.5 ± 20.6%, and pterostilbene + zingerone 36.5 ± 7.0%. The combinations of curcumin + resveratrol and curcumin + pterostilbene + resveratrol + zingerone almost completely inhibited PE disappearance. PE sulfate formation was inhibited 67.0 ± 4.2% (mean ± SEM, as % of control) by guaiacol and 71.7 ± 2.6% by pterostilbene + zingerone. The combinations of curcumin + resveratrol and curcumin + pterostilbene + resveratrol + zingerone inhibited ≥ 99% of PE sulfate formation. These results were consistent with those from analysis of the disappearance of PE in LS180 cells. These phenolic inhibitors for sulfation were also tested to see whether they have any inhibitory effects on MAO-A or B. Significant inhibition was found with curcumin, guaiacol, isoeugenol, pterostilbene, resveratrol, and zingerone on both MAO-A and B. Further kinetic studies were conducted to investigate the concentration of an inhibitor at which the enzyme activity is reduced by half (IC50) (mean ± SEM) of these inhibitors. The most potent inhibitor for MAO-A was resveratrol (0.313 ± 0.008 μM) followed by isoeugenol (3.72 ± 0.20 μM), curcumin (12.9 ± 1.3 μM), pterostilbene (13.4 ± 1.5 μM), zingerone (16.3 ± 1.1 μM), and guaiacol (131 ± 6 μM). The most potent inhibitor for MAO-B was pterostilbene (0.138 ± 0.013 μM), followed by curcumin (6.30 ± 0.11 μM), resveratrol (15.8 ± 1.3 μM), isoeugenol (102 ± 5 μM), and guaiacol (322 ± 27 μM). Since these phenolic compounds all have relatively low oral bioavailability, any MAO inhibition which could occur systemically is expected to be limited. Most inhibitory effects on MAO-A and B if any would be limited to the GI tract and liver. In conclusion, several compounds and combinations showed inhibition on PE sulfation in LS180 cell model, which may have potential to inhibit the pre-systemic sulfation of PE to improve its oral bioavailability. These compounds also showed the unexpected inhibition on human MAO-A and B with different potency, which could guide the selection of phenolic dietary compounds for further studies, along with the sulfation inhibition results and their pharmacokinetic (PK) properties such as bioavailability.
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Amer, Dena Ahmed Mohamed. "Estrogen Receptor-Beta Dependent Activities of Dietary Compounds in a Genetically Modified Rat Raphe Nuclei-Derived Cell Line." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2011. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-70312.

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Estrogens greatly affect the activity and connectivity of serotonergic neural cell populations, which extend from clusters of nuclei in the brainstem, termed the raphe nuclei, where estrogen receptor β is the most abundantly expressed estrogen receptor subtype. Estrogenic effects on the raphe nuclei are primarily important for influencing various neuropsychological behaviors, including depression, mood swings and anxiety behaviors. Because of this connection, phases of intense hormone fluctuations for instance during menopause are often associated with several mood disturbances that often reduce the quality of life of menopausal women. Accordingly, long-term use of hormone replacement therapy appeared to be the method of choice for many menopausal women to help alleviate vasomotor symptoms, which may include neuropsychological changes such as depression. However, given the limitations and number of serious health risks attributed to hormone replacement therapy, natural compounds such as phytoestrogens are receiving widespread awareness due to their occurrence in medicinal plant extracts and a wide variety of food items including dietary supplements with respective health claims. Flavonoids, particularly the isoflavones and the naringenin-type flavanones, belong to a group of polyphenolic plant-derived secondary metabolites known to possess estrogen-like bioactivities. Nevertheless, little is known about their transactivational activity and their potential to regulate endogenous gene expression of estrogen responsive genes in the raphe nuclei due to the lack of suitable cellular models expressing sufficient amounts of functional estrogen receptor β. Hence, a raphe nuclei-derived cell line that expresses a functional estrogen receptor β was sought as a model to investigate effects of flavonoids in vitro. In this regard, RN46A-B14 cells derived from embryonic day 13 rat medullary raphe nuclei were primarily used in this study as the main cellular model. Nonetheless, expression of endogenous estrogen receptor β in these cells was not sufficient to pursue downstream investigations of estrogen-dependent activities. To overcome this deficit, a rat raphe nuclei-derived in vitro model that overexpresses a functional estrogen receptor β was initially established (herein termed RNDA cells) by stably transducing its parent cell line, RN46A-B14 cells, with a suitable lentiviral expression vector encoding a human estrogen receptor β gene. The stable expression and the functional characterization of the transgenic receptor was confirmed by Western blot analysis and luciferase reporter gene assays, respectively. The same reporter gene assay was used to scrutinize the transactivational activity of the flavonoids in RNDA cells. Key results revealed that Genistein, Daidzein, Equol, Naringenin and 8-Prenylnaringenin demonstrated high transactivational activity in a concentration-dependent manner by stimulating luciferase expression from an estrogen responsive element-regulated reporter gene construct transiently transfected in RNDA cells. Low transactivational activity was observed in RNDA cells in response to increasing concentrations of 7-(O-prenyl)naringenin-4'-acetate. However, no transactivational activity was noticed in response to 6-(1,1-Dimethylallyl)naringenin in the studied cell model. All effects elicited by the flavonoids were antagonized by the pure estrogen receptor antagonist, Fulvestrant, indicating that all substances act by binding to and activating the transgenic ERβ. Additional effects were observed in RNDA cells in response to a co-treatment of 1 µM of either Genistein or Daidzein, but not Equol, with 10 nM 17β-Estradiol. Slight antagonistic effects were observed in the same studied cell line when either 8-Prenylnaringenin or 7-(O-prenyl)naringenin-4'-acetate, but not Naringenin or 6-(1,1-Dimethylallyl)naringenin, were co-added with 17β-Estradiol. Results from the reporter gene assays were validated on the basis of regulation of mRNA expression of estrogen responsive genes following the global assessment of 17β-Estradiol-induced gene expression in this cell line using a DNA microarray technique. Out of 212 estrogen-regulated genes with at least two-fold change of expression, six were selected according to specific features of estrogenic regulation of expression. The expression of the six selected 17β-Estradiol-regulated genes was validated using quantitative real-time PCR analysis. The regulation of mRNA expression of the selected genes in response to the tested flavonoids was then investigated in RNDA cells. Additionally, because RNDA cells encode a temperature-sensitive mutant of the Simian Virus 40 large T-antigen, their neuronal differentiation is constitutive upon shifting them from conditions promoting proliferation (permissive temperature) to differentiation (non permissive temperature). Hence, the regulation of mRNA expression of the selected genes in response to the tested flavonoids was additionally investigated as RNDA cells differentiate. In RNDA cells grown under proliferative conditions, 17β-Estradiol up-regulated mRNA expression of camello-like 5, sex determining region Y-box 18 and keratin type I cytoskeletal 19. Similar effects were observed in response to 8-Prenylnaringenin, Genistein, Daidzein and Equol. In addition, 17β-Estradiol down-regulated mRNA expression of neurofilament medium polypeptide and zinc finger DHHC-type containing 2. Similar effects were observed in response to 8-Prenylnaringenin, Naringenin, Genistein, Daidzein and Equol. Yet, no effect was observed on the regulation of mRNA expression of solute carrier family 6 member 4 in response to 17β-Estradiol or the flavonoids in RNDA cells grown under proliferative conditions. When RNDA cells were shifted to conditions promoting differentiation, changes in cell morphology, in mRNA expression levels and in responsiveness towards 17β-Estradiol or the flavonoids were observed. These expression studies additionally highlighted some of the genes as indicator genes for RNDA cellular differentiation. The newly established RNDA cell line should prove useful to elucidate basic physiological properties of estrogen receptor β in the raphe nuclei. The present study should serve as the basis to help shed light on molecular and cellular mechanisms following the action of phytoestrogens, endocrine disruptors or other exogenous estrogen receptor ligands in neural cell populations, particularly the raphe nuclei, for further applications within the brain.
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Books on the topic "Dietary compounds"

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Aura, Anna-Marja. In vitro digestion models for dietary phenolic compounds. [Espoo, Finland]: VTT Technical Research Centre of Finland, 2005.

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2

Schwartzman, Judy Susan. Dietary N-nitroso compounds and the risk of brain tumours in adults. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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Venugopal, V. Marine products for healthcare: Functional and bioactive nutraceutical compounds from the ocean. Boca Raton: Taylor & Francis, 2009.

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Dolara, Piero, ed. TOX: lezioni di tossicologia. Florence: Firenze University Press, 2006. http://dx.doi.org/10.36253/88-8453-412-7.

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TOX is a manual aimed primarily at the Toxicology courses of the Faculty of Pharmacy and Science, that can also be profitably utilised by students following different degree courses in other Faculties (Medicine, Agriculture, Engineering)and by anyone else looking for a thorough but succinct overview of toxicological questions. TOX covers the principal sectors of general toxicology (acute and chronic toxicity, mutagenesis, teratogenesis, carcinogenesis, reproductive toxicity, oxidative damage, epidemiological methods), specialist toxicology(dietary toxicity, tobacco smoke, pesticides, N-nitroso compounds, heterocyclic amines and aromatic amines toxicity) and environmental toxicology (environmental estrogens, PAH, heavy metals, dioxins and polychlorinated di-benzo-furans, water and air pollution).
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Dietary reference intakes: Proposed definition and plan for review of dietary antioxidants and related compounds. Washington, D.C: National Academy Press, 1998.

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Dietary Reference Intakes: Proposed Definition and Plan for Review of Dietary Antioxidants and Related Compounds. National Academies Press, 1998.

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Dietary Plant Origin Bio-Active Compounds, Intestinal Functionality and Microbiome. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03943-866-2.

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Nollet, Leo M. L., and Se-Kwon Kim. Marine Microorganisms: Extraction and Analysis of Bioactive Compounds. Taylor & Francis Group, 2016.

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Institute of Medicine (U. S.). Dietary Reference Intakes: Proposed Definition and Plan for Review of Dietary Antioxidants and Related Compounds (Compass Series). National Academies Press, 1999.

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Medicine, Institute Of, and Iom. Dietary Reference Intakes for Vitamin C, Vitamin E, Selenium, and Carotenoids: A Report of the Panel on Dietary Antioxidants and Related Compounds, Subcommittees ... Anduses (Dietary Reference Intakes). National Academy Press, 2000.

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Book chapters on the topic "Dietary compounds"

1

Zhao, Ming Ming, Shi Hang Xiong, Guan Ding Zhao, and Hua Yu. "Organosulfur Compounds in Food." In Handbook of Dietary Phytochemicals, 1–21. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-1745-3_41-1.

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Zhao, Ming Ming, Shi Hang Xiong, Guan Ding Zhao, and Hua Yu. "Organosulfur Compounds in Food." In Handbook of Dietary Phytochemicals, 1741–61. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-4148-3_41.

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Sagdic, Osman, and Fatih Tornuk. "Antimicrobial Properties of Organosulfur Compounds." In Dietary Phytochemicals and Microbes, 127–56. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-3926-0_4.

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Zhao, Hefei, and Changmou Xu. "Natural Phenolic Compounds as Anti-obesity and Anti-cardiovascular Disease Agent." In Dietary Phytochemicals, 205–21. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72999-8_11.

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Vongsawasdi, Punchira, and Athapol Noomhorm. "Bioactive Compounds in Meat and their Functions." In Functional Foods and Dietary Supplements, 113–38. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118227800.ch5.

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Vasta, Valentina, and Rui J. B. Bessa. "Manipulating Ruminal Biohydrogenation by the Use of Plants Bioactive Compounds." In Dietary Phytochemicals and Microbes, 263–84. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-3926-0_9.

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Hursting, Stephen D., Sarah M. Smith, Leticia Nogueira, Rebecca DeAngel, Laura Lashinger, and Susan N. Perkins. "Dietary Energy Balance, Calorie Restriction, and Cancer Prevention." In Bioactive Compounds and Cancer, 147–64. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-627-6_7.

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Repo-Carrasco-Valencia, Ritva. "Dietary Fibre and Bioactive Compounds of Kernels." In Pseudocereals, 71–93. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781118938256.ch4.

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Supic, Gordana, Darja Wagner, and Zvonko Magic. "Epigenetic Impact of Bioactive Dietary Compounds in Cancer Chemoprevention." In Critical Dietary Factors in Cancer Chemoprevention, 153–81. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-21461-0_7.

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Nakornpanom, Nantarat Na, and Porntip Sirisoontaralak. "The Effect of Irradiation on Bioactive Compounds in Plant and Plant Products." In Functional Foods and Dietary Supplements, 387–403. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118227800.ch14.

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Conference papers on the topic "Dietary compounds"

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Manig, F., M. Hellwig, F. Pietz, and T. Henle. "Saliva as a tool to monitor endogenous and dietary glycation compounds." In Diabetes Kongress 2019 – 54. Jahrestagung der DDG. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1688155.

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Xia, Xinghui, Haotian Wang, and Derek C. g. Muir. "Dietary Uptake Pattern Affects Bioaccumulation and Biomagnification of Hydrophobic Organic Compounds in Fish." In Goldschmidt2020. Geochemical Society, 2020. http://dx.doi.org/10.46427/gold2020.2919.

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Pal, Purab, Karen Hales, and Dale Buchanan Hales. "Abstract B35: Dietary flaxseed supplementation in ovarian cancer: Elucidating the molecular actions of its biologically derived active compounds." In Abstracts: AACR Special Conference on Advances in Ovarian Cancer Research; September 13-16, 2019; Atlanta, GA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3265.ovca19-b35.

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Mares-Mares, Everardo, Paola Elizabeth Iwinski-Hern�ndez, Ignacio Alberto Vallejo-Gonzalez, Luis Armando Garcia de la Rosa, Ma Cristina Del Rinc�n-Castro, and Maria Azucena Rocha-Mendoza. "Microencapsulation of bioactive compounds obtained from pitahaya and soya for possible use as a dietary supplement in menopause." In 2021 ASABE Annual International Virtual Meeting, July 12-16, 2021. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2021. http://dx.doi.org/10.13031/aim.202100209.

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Hardy, Tabitha M., Olayode O. Babatunde, and Trygve O. Tollofsbol. "Abstract 2598: Dietary compounds EGCG and sulforaphane differentially reactivate ER-α in African American and Caucasian breast cancer cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2598.

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Pajari, Anne-Maria, Essi Päivärinta, Vesa Lapinkero, Jessica Manngård, Tiina Pellinen, Suvi Itkonen, and Anne Salonen. "Abstract 965: Effects of replacing dietary animal protein with plant-based protein on the formation of intestinal N-nitroso compounds (NOCs) and biomarkers of colon cancer in healthy adults–a 12-week intervention study." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-965.

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Pajari, Anne-Maria, Essi Päivärinta, Vesa Lapinkero, Jessica Manngård, Tiina Pellinen, Suvi Itkonen, and Anne Salonen. "Abstract 965: Effects of replacing dietary animal protein with plant-based protein on the formation of intestinal N-nitroso compounds (NOCs) and biomarkers of colon cancer in healthy adults–a 12-week intervention study." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-965.

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Calinescu, Ioan, Alin Vintila, Aurel Diacon, Mircea Vinatoru, Ana Maria Galan, and Sanda Velea. "GROWTH OF NANNOCHLORIS ALGAE IN THE PRESENCE OF MICROWAVES (CONTINUOUS REACTOR)." In Ampere 2019. Valencia: Universitat Politècnica de València, 2019. http://dx.doi.org/10.4995/ampere2019.2019.9820.

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Algae are very effective in capturing the sun's energy, carbon dioxide from the atmosphere, and nutrients to turn them into useful substances (carbohydrates, oils, proteins, etc.). Besides the main metabolites, there are also secondary metabolites, such as carotenoids (astaxanthin, β-carotene, lutein, lycopene, and canthaxanthin [1]). Both major and compounds existing in small amounts in algae are useful. Oils and carbohydrates could provide biofuels, proteins can get products with nutritional value and from carotenoids can be prepared food supplements. Obtaining biofuel from algae has not yet proved to be economically viable [2, 3]. A much higher interest might be getting food supplements from algae. To increase their value as ingredients for food supplements, algal oils should have a higher degree of unsaturation (rich in omega 3) and an increased carotenoid content to be an important additional benefit in over all processing of algae. There are studies that refer to the influence of environmental factors on algae composition [2], but the microwave influence on algae growth, especially algal metabolites composition change is very poor studied. In this paper, besides the experiments for the activation of algal growth in discontinuous reactors [4] additional work was conducted in a continuous photobioreactor. The goal was checking not only the growth of microalgae but also their content in polyunsaturated oil and in carotenoids. By microwave-controlled irradiation of the nutrient and algae flux, which is recirculated through the photobioreactor and through a glass reactor located in a TE-type monomod cavity, the lipid content of the algae increased, but only, the modification of the lipid fraction content was significantly increased in the concentration of polyunsaturated acids with 16 and 18 carbon atoms. As far as carotenoids are concerned, the algae nannochloris has a higher carotenoid content over many known vegetables holding carotene or lycopene (carrots or tomatoes). Besides oil increasing microwave treatment produced a significant increase in carotenoid content of algae. They can be extracted together with omega-3-rich algal oil and are the basis of very valuable dietary supplements.
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Chakraborty, Amlan, Jennifer Boer, Simon Royce, Kirsty Wilson, Cordelia Selomulya, and Magdalena Plebanski. "Effect of a small natural dietary compound on lung pathology in airway inflammation." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa1045.

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Mishra, Manoj K., James A. Stokes, Amanee Salaam, Elijah Nyairo, Udai Singh, and Derrick Dean. "Abstract 4107: Efficient delivery of dietary compound modulates mcp-1 in murine prostate cancer cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4107.

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Reports on the topic "Dietary compounds"

1

Wu, Guangyu. Preventive Role of Specific Dietary Factors and Natural Compounds Against DNA Damage and Oxidative Stress. Fort Belvoir, VA: Defense Technical Information Center, August 1999. http://dx.doi.org/10.21236/ada377925.

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