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Journal articles on the topic 'Dietary uptake'

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Published: 3 June 2025

Last updated: 31 July 2025

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1

Toloza, E. M., and J. Diamond. "Ontogenetic development of nutrient transporters in rat intestine." American Journal of Physiology-Gastrointestinal and Liver Physiology 263, no. 5 (1992): G593—G604. http://dx.doi.org/10.1152/ajpgi.1992.263.5.g593.

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We measured intestinal brush-border uptakes of three sugars and three amino acids, plus intestinal morphometric parameters, in rats from the day of birth until adulthood. Rates of body weight gain had pronounced peaks in the suckling phase and again during weaning, separated by a dip at the onset of weaning. These two peaks coincided with peaks or plateaus in intestinal growth and in glucose (Glc) and proline (Pro) uptake capacities, which may provide the basis for high rates of body growth. Pro uptake declined relative to Glc uptake upon weaning, reflecting decreasing protein needs for growth

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2

Southon, Adam, Mark A. Greenough, George Ganio, Ashley I. Bush, Richard Burke, and James Camakaris. "Presenilin Promotes Dietary Copper Uptake." PLoS ONE 8, no. 5 (2013): e62811. http://dx.doi.org/10.1371/journal.pone.0062811.

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3

BARDOCZ, SUSAN, ANN WHITE, GEORGE GRANT, DAVID S. BROWN, TRACEY J. DUGUID, and ARPAD PUSZTAI. "Uptake and bioavailability of dietary polyamines." Biochemical Society Transactions 24, no. 2 (1996): 226S. http://dx.doi.org/10.1042/bst024226s.

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4

Wood, Chris M., Natasha M. Franklin, and Som Niyogi. "The Protective Role of Dietary Calcium Against Cadmium Uptake and Toxicity in Freshwater Fish: an Important Role for the Stomach." Environmental Chemistry 3, no. 6 (2006): 389. http://dx.doi.org/10.1071/en06056.

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Environmental Context. Contamination of freshwater ecosystems by cadmium is of increasing concern with accumulation and toxicity in aquatic animals occurring through both waterborne and dietary routes. Increases in water calcium (‘hardness’) levels protect against waterborne uptake. Physiological research on freshwater fish has demonstrated that this occurs because cadmium moves through the calcium uptake pathway at the gills. Surprisingly, elevated dietary calcium also protects against waterborne exposure by down-regulating the calcium uptake pathway at the gills, and against dietary exposure

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5

Thomson, A. B. R. "Ileal resection and dietary content of sucrose influence the intestinal uptake of monosaccharides." Canadian Journal of Physiology and Pharmacology 65, no. 11 (1987): 2281–86. http://dx.doi.org/10.1139/y87-361.

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The intestinal uptake of 0.5 and 40 mM glucose, galactose, and 3-O-methyl glucose (3-O-MG) was examined in vitro in rabbits fed a high (HS) or a low (LS) sucrose diet. In animals with an intact intestinal tract, the jejunal uptake of 0.5 mM 3-O-MG was unaffected by the dietary content of sucrose, whereas the uptake of 40 mM 3-O-MG was lower in LS than HS. The uptake of 40 mM galactose was higher in LS than HS and the uptake of 0.5 mM galactose was similar in HS and LS, whereas the uptake of 0.5 mM but not 40 mM glucose was lower in LS than HS. In animals subjected 6 weeks previously to an ilea

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6

Karasov, W. H., B. W. Darken, and M. C. Bottum. "Dietary regulation of intestinal ascorbate uptake in guinea pigs." American Journal of Physiology-Gastrointestinal and Liver Physiology 260, no. 1 (1991): G108—G118. http://dx.doi.org/10.1152/ajpgi.1991.260.1.g108.

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We measured ascorbic acid (AA) uptake across the intestinal brush border in vitro in intact tissue from guinea pigs fed maintenance AA (200 mg/kg diet) or made hypervitaminotic (5,000 mg/kg diet) or hypovitaminotic (chronically and acutely). Total uptake per centimeter ileum was 25-50% lower in hypervitaminotic juvenile, adult male, and lactating guinea pigs compared with their respective controls, whereas carrier-mediated D-glucose uptake and Na(+)-independent AA uptake were similar. High dietary ascorbate specifically reduced the Vmax for carrier-mediated AA uptake. Hypovitaminosis had no si

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7

Kjoss, V. A., C. N. Kamunde, S. Niyogi, M. Grosell, and C. M. Wood. "Dietary Na does not reduce dietary Cu uptake by juvenile rainbow trout." Journal of Fish Biology 66, no. 2 (2005): 468–84. http://dx.doi.org/10.1111/j.0022-1112.2005.00612.x.

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8

Ferraris, R. P., J. Diamond, and W. W. Kwan. "Dietary regulation of intestinal transport of the dipeptide carnosine." American Journal of Physiology-Gastrointestinal and Liver Physiology 255, no. 2 (1988): G143—G150. http://dx.doi.org/10.1152/ajpgi.1988.255.2.g143.

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Uptake of the dipeptide L-carnosine was measured in everted intestinal sleeves of mice whose dietary protein level or else proportion of protein in the form of free amino acids was varied experimentally. Carnosine uptake was highest in the jejunum, regardless of ration. Compared with a low-protein (18%) ration, a high-protein (72%) ration stimulated carnosine uptake by 30-70% in duodenum and jejunum (but not in ileum). This stimulation was observed even in the presence of peptidase inhibitors that inhibit cell surface hydrolysis of dipeptides. Measured carnosine hydrolysis was low or negligibl

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9

Jarocka-Cyrta, E., N. Perin, M. Keelan, et al. "Early dietary experience influences ontogeny of intestine in response to dietary lipid changes in later life." American Journal of Physiology-Gastrointestinal and Liver Physiology 275, no. 2 (1998): G250—G258. http://dx.doi.org/10.1152/ajpgi.1998.275.2.g250.

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This study was undertaken to test the hypothesis that a change in the mother’s diet at the time of birth and continued during suckling modifies the intestinal transport of nutrients in the suckling offspring. Pregnant rat dams were fed one of four semisynthetic diets during pregnancy [high or low n-6/n-3 diet or a diet enriched with arachidonic acid (AA) or docosahexaenoic acid (DHA)] and were fed the same diet at the time of birth or switched to another diet. The greatest body weight gain was in the suckling rats (15–16 days of age) fed a low n-6/n-3 diet. Switching from this diet caused weig

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10

Shu, R., E. S. David, and R. P. Ferraris. "Dietary fructose enhances intestinal fructose transport and GLUT5 expression in weaning rats." American Journal of Physiology-Gastrointestinal and Liver Physiology 272, no. 3 (1997): G446—G453. http://dx.doi.org/10.1152/ajpgi.1997.272.3.g446.

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Rates of fructose uptake by the small intestine of neonatal rats are typically very low from parturition through weaning but undergo a dramatic increase immediately after weaning is completed. In this study, we used intestinal fructose transport as a model to determine whether nutrient transport, normally enhanced only after completion of weaning, can be enhanced earlier during development. We found that ontogenetic changes in levels of GLUT5 mRNA correlate well with already known ontogenetic changes in rates of intestinal fructose transport: low levels and rates during suckling and weaning, a

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11

Toloza, E. M., and J. M. Diamond. "Ontogenetic development of transporter regulation in bullfrog intestine." American Journal of Physiology-Gastrointestinal and Liver Physiology 258, no. 5 (1990): G770—G773. http://dx.doi.org/10.1152/ajpgi.1990.258.5.g770.

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Intestinal nutrient transporter activity is adapted to dietary substrate levels on three time scales: reversibly, within an adult individual, to rapid dietary changes; developmentally, to normal ontogenetic changes in diet; and evolutionarily, among carnivores, omnivores, and herbivores, to a species' natural diet. Does the capacity for rapid reversible adaptation itself vary adaptively during development? Substrate-dependent regulation would make functional sense in herbivorous/omnivorous tadpoles in which dietary substrate levels fluctuate unpredictably, but would serve no purpose in strictl

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12

Thomson, A. B. R., M. Keelan, M. Garg та M. T. Clandinin. "Dietary effects of ω3-fatty acids on intestinal transport function". Canadian Journal of Physiology and Pharmacology 66, № 7 (1988): 985–92. http://dx.doi.org/10.1139/y88-162.

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Animals were fed for 2 weeks on one of four isocaloric and isocholesterolic semisynthetic diets: high 18:3ω3, low 18:3ω3, high 20:5ω3, or low 20:5ω3. The weight of the intestine and the percentage of the wall consisting of mucosa was greater in high 20:5ω3 than in high 18:3ω3, and greater in low 20:5ω3 than in low 18:3ω3, although the mucosal surface area was 26% lower in high 20:5ω3 than high 18:3ω3. The jejunal uptake of 40 mM glucose and ileal uptake of 40 mM galactose was greater in high 18:3ω3 than in high 20:5ω3; jejunal uptake of fatty acid 12:0 was higher, but 18:0 was lower in high 18

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13

Liu, S., V. E. Baracos, H. A. Quinney, and M. T. Clandinin. "Dietary fat modifies exercise-dependent glucose transport in skeletal muscle." Journal of Applied Physiology 80, no. 4 (1996): 1219–24. http://dx.doi.org/10.1152/jappl.1996.80.4.1219.

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Exercise stimulates muscle glucose uptake both directly and by increasing the sensitivity of this process to insulin. This study was designed to investigate whether the level of dietary fat would interact with the action of acute exercise in the presence or absence of insulin. Weanling female Sprague-Dawley rats were fed two levels of dietary fat (5 vs. 20%; wt/wt) for 6 wk. Rats then remained sedentary or were exercised by a single bout of swimming for a total of 2 h with 5-min rest intervals each 0.5 h. Basal (insulin-independent) and insulin-stimulated glucose uptake rates were determined i

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14

Pierson, Hannah, Haojun Yang, and Svetlana Lutsenko. "Copper Transport and Disease: What Can We Learn from Organoids?" Annual Review of Nutrition 39, no. 1 (2019): 75–94. http://dx.doi.org/10.1146/annurev-nutr-082018-124242.

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Many metals have biological functions and play important roles in human health. Copper (Cu) is an essential metal that supports normal cellular physiology. Significant research efforts have focused on identifying the molecules and pathways involved in dietary Cu uptake in the digestive tract. The lack of an adequate in vitro model for assessing Cu transport processes in the gut has led to contradictory data and gaps in our understanding of the mechanisms involved in dietary Cu acquisition. The recent development of organoid technology has provided a tractable model system for assessing the det

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15

IKEDA, Saiko, Takako NIWA, and Kanae YAMASHITA. "Selective Uptake of Dietary Tocotrienols into Rat Skin." Journal of Nutritional Science and Vitaminology 46, no. 3 (2000): 141–43. http://dx.doi.org/10.3177/jnsv.46.141.

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16

Peck, Michael D., Paul B. Spalding, Frederick L. Moffat, Tieran Han, and Wenche Jy. "Dietary Olive Oil Enhances Murine Lymphocyte Calcium Uptake." Journal of Trauma: Injury, Infection, and Critical Care 49, no. 1 (2000): 109–14. http://dx.doi.org/10.1097/00005373-200007000-00017.

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17

Drozdowski, Laurie A., Miyoung Suh, Eekjoong Park, M. Tom Clandinin, and Alan B. R. Thomson. "Dietary Gangliosides EnhanceIn VitroGlucose Uptake in Weanling Rats." Journal of Parenteral and Enteral Nutrition 31, no. 5 (2007): 423–29. http://dx.doi.org/10.1177/0148607107031005423.

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18

Johnson, J. R., and E. S. Lamothe. "A Review of the Dietary Uptake of Th." Health Physics 56, no. 2 (1989): 165–68. http://dx.doi.org/10.1097/00004032-198902000-00003.

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19

Stewart-Knox, B., J. Hamilton, H. Parr, and B. Bunting. "Dietary strategies and uptake of reduced fat foods." Journal of Human Nutrition and Dietetics 18, no. 2 (2005): 121–28. http://dx.doi.org/10.1111/j.1365-277x.2005.00595.x.

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20

Ferraris, R. P., and J. Diamond. "Crypt-villus site of glucose transporter induction by dietary carbohydrate in mouse intestine." American Journal of Physiology-Gastrointestinal and Liver Physiology 262, no. 6 (1992): G1069—G1073. http://dx.doi.org/10.1152/ajpgi.1992.262.6.g1069.

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Intestinal brush-border glucose uptake rate is regulated by dietary carbohydrate level. However, this uptake response takes a day or more after a change in dietary carbohydrate level. Is this dietary signal perceived in the crypts, and is the glucose transporter activity of enterocytes irreversibly programmed there? If so, this time lag could reflect cell migration times along the crypt-villus axis, since glucose transport is not fully expressed until cells reach the midvillus. Alternatively, however, the time lag could arise from the induction process itself, if glucose transporter activity i

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21

Kamunde, Collins, Cheryl Clayton, and Chris M. Wood. "Waterborne vs. dietary copper uptake in rainbow trout and the effects of previous waterborne copper exposure." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 283, no. 1 (2002): R69—R78. http://dx.doi.org/10.1152/ajpregu.00016.2002.

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Juvenile rainbow trout ( Oncorhynchus mykiss) were exposed to waterborne Cu (22 μg/l) in moderately hard water for up to 28 days. Relative to control fish kept at background Cu levels (2 μg/l), Cu-preexposed fish displayed decreased uptake rates of waterborne Cu via the gills but not of dietary Cu via the gut during 48-h exposures to64Cu-radiolabeled water and diet, respectively. At normal dietary and waterborne Cu levels, the uptake rates of dietary Cu into the whole body without the gut were 0.40–0.90 ng · g−1 · h−1, >10-fold higher than uptake rates of waterborne Cu into the whole body w

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22

Kamunde, Collins, Martin Grosell, Dave Higgs, and Chris M. Wood. "Copper metabolism in actively growing rainbow trout (Oncorhynchus mykiss): interactions between dietary and waterborne copper uptake." Journal of Experimental Biology 205, no. 2 (2002): 279–90. http://dx.doi.org/10.1242/jeb.205.2.279.

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SUMMARY Juvenile rainbow trout Oncorhynchus mykiss were exposed to diets with low (12.6 nmol g–1), normal (50.4 nmol g–1) or elevated (4437.5 nmol g–1) Cu concentrations in combination with either low (5.8 nmol l–1) or normal (48.5 nmol l–1) waterborne Cu levels over a 50-day period, during which body mass increased up to fivefold. A nutritional requirement for Cu was demonstrated based on growth response and whole body and tissue Cu status. Simultaneous low Cu levels in both the water and the diet depressed growth by 31 % over 7 weeks. There were reductions in both specific growth rate (SGR,

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23

Thomson, A. B. R., M. Keelan, M. Garg, and M. T. Clandinin. "Spectrum of effects of dietary long-chain fatty acids on rat intestinal glucose and lipid uptake." Canadian Journal of Physiology and Pharmacology 65, no. 12 (1987): 2459–65. http://dx.doi.org/10.1139/y87-390.

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Isocaloric modification in the ratio of dietary polyunsaturated-to-saturated fatty acids influences intestinal uptake of actively and passively transported nutrients. This study was undertaken to determine which dietary fatty acid was responsible for these alterations in absorption. Adult female rats were fed isocaloric semisynthetic diets high in palmitic and stearic acids (SFA), oleic acid (OA), linoleic acid (LA), or linolenic acid (LNA). An in vitro technique was used to measure the uptake of varying concentrations of glucose as well as a series of fatty acids and cholesterol. Jejunal upta

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24

Zewde, Tewabech, Feng Wu, and David L. Mattson. "Influence of dietary NaCl on L-arginine transport in the renal medulla." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 286, no. 1 (2004): R89—R93. http://dx.doi.org/10.1152/ajpregu.00309.2003.

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Previous work demonstrated that l-arginine, the substrate for nitric oxide (NO) synthase, is carried into inner medullary collecting duct (IMCD) cells via system y+, that the major system y+ gene product in IMCD is the cationic amino acid transporter 1 (CAT1), and that blockade of l-arginine uptake in the renal medulla decreases NO and leads to systemic hypertension. The present study determined the influence of dietary sodium intake on l-arginine uptake in IMCD, on CAT1 immunoreactive protein in the renal medulla, and on the hypertensive response to blockade of l-arginine uptake in the renal

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25

Said, Hamid M., Nabendu Chatterjee, Riaz Ul Haq, et al. "Adaptive regulation of intestinal folate uptake: effect of dietary folate deficiency." American Journal of Physiology-Cell Physiology 279, no. 6 (2000): C1889—C1895. http://dx.doi.org/10.1152/ajpcell.2000.279.6.c1889.

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Folate is an essential micronutrient that, in mammals, must be obtained from exogenous sources via intestinal absorption. Previous studies have characterized different aspects of the mechanism of the intestinal folate uptake process. Much less, however, is known about regulation of this process. In this study, we examined the effect of dietary folate deficiency on intestinal folate uptake using the rat as an animal model. The results showed that dietary folate deficiency leads to a significant ( P < 0.01) and specific upregulation in the transepithelial transport of folic acid. The upregula

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26

Solberg, D. H., and J. M. Diamond. "Comparison of different dietary sugars as inducers of intestinal sugar transporters." American Journal of Physiology-Gastrointestinal and Liver Physiology 252, no. 4 (1987): G574—G584. http://dx.doi.org/10.1152/ajpgi.1987.252.4.g574.

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Intestinal sugar transport increases with dietary carbohydrate levels, but the specific regulatory signals involved have been little studied. Hence we compared rations containing one of five sugars [D-glucose, D-galactose, 3-O-methyl-D-glucose (3-O-MG), D-fructose, and maltose] in their effects on brush-border uptake of five transported solutes (D-glucose, D-galactose, 3-O-MG, D-fructose, and L-proline) by everted sleeves of mouse small intestine. As confirmed by transepithelial potential difference (PD) measurements, there is a distinct fructose transporter that does not evoke a PD, along wit

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27

Perin, Nilza, Elizabeth Jarocka‐Cyrta, Monika Keelan, Tom Clandinin, and Alan Thomson. "Dietary Lipid Composition Modifies Intestinal Morphology and Nutrient Transport in Young Rats." Journal of Pediatric Gastroenterology and Nutrition 28, no. 1 (1999): 46–53. http://dx.doi.org/10.1002/j.1536-4801.1999.tb02003.x.

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ABSTRACTBackground:Varying lipid content of the diet of pregnant and nursing dams results in alterations in sugar and lipid uptake into the intestine of their suckling offspring. In this study, we wished to determine whether the same alterations in dietary lipid result in adaptation of intestinal transport in postweaning rats.Methods:During nursing, the dams were fed the same diet that their offspring were fed for 3 more weeks after weaning. These semipurified diets contained: 1) 15.8% of total fatty acids (w/w) as 18:2n‐6 and an n6/n3 ratio of 7.3:1; 2) a diet with 17.6% of total fatty acids

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28

Van Den Berg, Gerrit J., and Anton C. Beynen. "Influence of ascorbic acid supplementation on copper metabolism in rats." British Journal of Nutrition 68, no. 3 (1992): 701–15. http://dx.doi.org/10.1079/bjn19920127.

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An attempt was made to unravel further the mechanism by which high dietary concentrations of ascorbic acid influence copper metabolism. The addition of ascorbic acid to the diet of rats caused about a twofold increase in plasma ascorbate concentrations and reduced group mean plasma and tissue concentrations of Cu. The effect of 10 g ascorbic acid/kg diet was greater than that of 1 g/kg. Ascorbic acid feeding reduced blood haemoglobin concentrations and packed cell volume values. Dietary ascorbic acid caused a significant decrease in apparent Cu absorption from the intestine. Ascorbate, intrave

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29

Drozdowski, L., T. Woudstra, G. Wild, M. T. Clandinin, and A. B. R. Thomson. "Dietary lipids modify the age-associated changes in intestinal uptake of fructose in rats." American Journal of Physiology-Gastrointestinal and Liver Physiology 288, no. 1 (2005): G125—G134. http://dx.doi.org/10.1152/ajpgi.00311.2003.

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Because reduced nutrient absorption may contribute to malnourishment in the elderly, age and diet modulate fructose uptake in mice, and alterations in fructose uptake may be paralleled by changes in the abundance of fructose transporters, the objectives of this study were to determine 1) the effects of aging on fructose absorption in rats, 2) the effect of feeding diets enriched with saturated fatty acids (SFA) vs. polyunsaturated fatty acids (PUFA), and 3) the mechanisms of these age-and diet-associated changes. Male Fischer 344 rats aged 1, 9, and 24 mo received isocaloric diets enriched wit

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30

Boki, Keito, Shigeaki Kadota, Masae Takahashi, and Manabu Kitakouji. "Uptake of Polycyclic Aromatic Hydrocarbons by Insoluble Dietary Fiber." JOURNAL OF HEALTH SCIENCE 53, no. 1 (2007): 99–106. http://dx.doi.org/10.1248/jhs.53.99.

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31

Miller, W. J., W. M. Sherman, H. Dodd, and J. L. Ivy. "Influence of dietary carbohydrate on skeletal muscle glucose uptake." American Journal of Clinical Nutrition 41, no. 3 (1985): 526–32. http://dx.doi.org/10.1093/ajcn/41.3.526.

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32

Stavric, B., and R. Klassen. "Dietary effects on the uptake of benzo[a]pyrene." Food and Chemical Toxicology 32, no. 8 (1994): 727–34. http://dx.doi.org/10.1016/s0278-6915(09)80005-7.

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33

Carline, Robert, Patrick Barry, and H. George Ketola. "Dietary Uptake of Polychlorinated Biphenyls (PCBs) by Rainbow Trout." North American Journal of Aquaculture 66, no. 2 (2004): 91–99. http://dx.doi.org/10.1577/a03-028.1.

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Wassef, Lesley, and Loredana Quadro. "Uptake of Dietary Retinoids at the Maternal-Fetal Barrier." Journal of Biological Chemistry 286, no. 37 (2011): 32198–207. http://dx.doi.org/10.1074/jbc.m111.253070.

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Belfroid, A., J. Meiling, H. J. Drenth, J. Hermens, W. Seinen, and K. Vangestel. "Dietary Uptake of Superlipophilic Compounds by Earthworms (Eisenia andrei)." Ecotoxicology and Environmental Safety 31, no. 3 (1995): 185–91. http://dx.doi.org/10.1006/eesa.1995.1061.

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Dupre, Rebecca A., Ryan Ardoin, Jesse Trushenski, Chris Jackson, Casey Grimm, and Brennan Smith. "Dietary uptake of geosmin in rainbow trout (Oncorhynchus mykiss)." Aquaculture 571 (June 2023): 739458. http://dx.doi.org/10.1016/j.aquaculture.2023.739458.

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37

Caviedes-Vidal, E., and W. H. Karasov. "Glucose and amino acid absorption in house sparrow intestine and its dietary modulation." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 271, no. 3 (1996): R561—R568. http://dx.doi.org/10.1152/ajpregu.1996.271.3.r561.

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We acclimated house sparrows (Passer domesticus; 26 g) to high-starch (HS), high-protein (HP), and high-lipid (HL) diets and tested the predictions that uptake of D-glucose and amino acids will be increased with increased levels of dietary carbohydrate and protein, respectively. HS birds had lower mediated D-glucose uptake rate than HP birds. Total uptake of L-leucine at low concentration (0.01 mM), but not of L-proline at 50mM, was increased by dietary protein. Measures of D-glucose maximal mediated uptake (1.2 +/- 0.2 nmol.min-1.mg-1) and intestinal mass (1 g) indicated that the intestine's

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38

Elimrani, Ihsan, Karim Lahjouji, Ernest Seidman, Marie-Josée Roy, Grant A. Mitchell, and Ijaz Qureshi. "Expression and localization of organic cation/carnitine transporter OCTN2 in Caco-2 cells." American Journal of Physiology-Gastrointestinal and Liver Physiology 284, no. 5 (2003): G863—G871. http://dx.doi.org/10.1152/ajpgi.00220.2002.

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l-Carnitine is derived both from dietary sources and biosynthesis. Dietary carnitine is absorbed in the small intestine and then distributed to other organs. Previous studies using Caco-2 cells demonstrated that the transport ofl-carnitine in the intestine involves a carrier-mediated system. The purpose of this study was to determine whether the uptake of l-carnitine in Caco-2 cells is mediated by the recently identified organic cation/carnitine transporter (OCTN2). Kinetics ofl-[3H]carnitine uptake were investigated with or without specific inhibitors. l-Carnitine uptake in mature cells was s

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Milman, Nils Thorm. "A Review of Nutrients and Compounds, Which Promote or Inhibit Intestinal Iron Absorption: Making a Platform for Dietary Measures That Can Reduce Iron Uptake in Patients with Genetic Haemochromatosis." Journal of Nutrition and Metabolism 2020 (September 14, 2020): 1–15. http://dx.doi.org/10.1155/2020/7373498.

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Objective. To provide an overview of nutrients and compounds, which influence human intestinal iron absorption, thereby making a platform for elaboration of dietary recommendations that can reduce iron uptake in patients with genetic haemochromatosis. Design. Review. Setting. A literature search in PubMed and Google Scholar of papers dealing with iron absorption. Results. The most important promoters of iron absorption in foods are ascorbic acid, lactic acid (produced by fermentation), meat factors in animal meat, the presence of heme iron, and alcohol which stimulate iron uptake by inhibition

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40

Spital, Aaron, and Richard H. Sterns. "Extrarenal potassium adaptation: the role of aldosterone." Clinical Science 76, no. 2 (1989): 213–19. http://dx.doi.org/10.1042/cs0760213.

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1. Prior adaptation to a high potassium (HK) diet reduces the increment in plasma potassium after nephrectomy and acute potassium loading. Previous work has suggested that this ‘extrarenal potassium adaptation’ is due to direct stimulation of cellular potassium uptake by chronic hyperaldosteronism. 2. In contrast, we have shown that when dietary potassium is withdrawn from HK rats, large urinary potassium losses persist, resulting in ‘paradoxical potassium depletion’. This potassium depletion facilitates cellular potassium uptake and is, at least in part, responsible for extrarenal potassium a

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41

Titus, E., W. H. Karasov, and G. A. Ahearn. "Dietary modulation of intestinal nutrient transport in the teleost fish tilapia." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 261, no. 6 (1991): R1568—R1574. http://dx.doi.org/10.1152/ajpregu.1991.261.6.r1568.

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Tilapia (Oreochromis mossambicus) were fed a diet with either 60% carbohydrate (70% grain-4% fish meal) or 17% carbohydrate (11% grain-65% fish meal) for greater than or equal to 4 wk. Intestinal uptake of radiolabeled acetate, D-glucose, and L-proline was measured in brush-border membrane vesicles. As expected, fish fed high carbohydrate had significantly higher D-glucose uptake than those fed low carbohydrate [maximal uptake rate (Vmax), respectively, 84.2 +/- 18.2 vs. 37.4 +/- 10.9 pmol.mg protein-1.s-1; n = 4 batches of vesicles in each case; t test, P less than 0.025]. The change in gluco

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Cheema, Muhammad Arslan, Muhammad Abdullah Zain, Khadija Cheema, and Waqas Ullah. "Thyroxine-induced periodic paralysis: a rare complication of nutritional supplements." BMJ Case Reports 11, no. 1 (2018): e227946. http://dx.doi.org/10.1136/bcr-2018-227946.

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The consumption of daily nutritional supplements has risen dramatically all over the world. Many people believe that dietary supplements, if not useful, are at least safe to fulfil small dietary gaps. Many nutritional supplements have not been approved by Federal Drug Administration due to their unregulated active ingredients, but they are available as over the counter. One of the active ingredients, exogenous triiodothyronine (T3), has been reported in dietary supplements. We present a case of sudden onset of tetraparesis. Laboratory workup showed hypokalaemia, low thyroid-stimulating hormone

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Timmermans, Klaas R., Elly Spijkerman, Marcel Tonkes, and Harrie Govers. "Cadmium and Zinc Uptake by Two Species of Aquatic Invertebrate Predators from Dietary and Aqueous Sources." Canadian Journal of Fisheries and Aquatic Sciences 49, no. 4 (1992): 655–62. http://dx.doi.org/10.1139/f92-074.

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Cadmium and Zn uptake rates via food and water were determined under laboratory conditions for two species of freshwater invertebrate predators. Water mites (Limnesia maculata) and caddisfly larvae (Mystacides spp.) were exposed for 4 wk to either contaminated chironomid larvae (Chironomus riparius, 288–639 μg Cd∙g−1 or 778–1152 μg Zn∙g−1) or contaminated water (0.1 mg Cd∙L−1 or 1.0 mg Zn∙L−1). Cadmium was readily accumulated in the two species from both dietary and aqueous sources. A clear difference between exposed and untreated organisms was established. Zinc uptake was generally lower than

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Martín-Tereso, Javier, and Jean-Baptiste Daniel. "28 Integrating homeostasis in practical mineral supplementation recommendations." Journal of Animal Science 102, Supplement_3 (2024): 80–81. http://dx.doi.org/10.1093/jas/skae234.089.

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Abstract A long-established objective in ruminant nutrition is an accurate supply of the 4 main trace metal nutrients Zn, Cu, Mn, and Fe, respecting bioavailability, nutritional interactions, and nutrient tolerance. Although, current dietary supplementation practices, justified by the multiple uncertainties of trace metal supply, results in dietary levels that widely exceed nutritional guidelines. Additionally, reference feeding recommendations are defined assuming fixed and highly conservative uptake coefficients, an approach that disregards the opportunity, and limitations, of absorptive reg

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Vialat, Marine, Elissa Baabdaty, Amalia Trousson, et al. "Cholesterol Dietary Intake and Tumor Cell Homeostasis Drive Early Epithelial Tumorigenesis: A Potential Modelization of Early Prostate Tumorigenesis." Cancers 16, no. 11 (2024): 2153. http://dx.doi.org/10.3390/cancers16112153.

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Epidemiological studies point to cholesterol as a possible key factor for both prostate cancer incidence and progression. It could represent a targetable metabolite as the most aggressive tumors also appear to be sensitive to therapies designed to decrease hypercholesterolemia, such as statins. However, it remains unknown whether and how cholesterol, through its dietary uptake and its metabolism, could be important for early tumorigenesis. Oncogene clonal induction in the Drosophila melanogaster accessory gland allows us to reproduce tumorigenesis from initiation to early progression, where tu

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Wolffram, S., C. Hagemann, and E. Scharrer. "Regression of high-affinity carrier-mediated intestinal transport of taurine in adult cats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 261, no. 5 (1991): R1089—R1095. http://dx.doi.org/10.1152/ajpregu.1991.261.5.r1089.

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The sulfur-containing beta-amino acid taurine is an essential nutrient for cats. Owing to some metabolic peculiarities, cats are more dependent on dietary sources of taurine than other mammals. We therefore studied taurine uptake by intestinal brush-border membrane vesicles (BBMV) isolated from cat small intestine. Taurine uptake by feline BBMV was not influenced by a transmembrane Na+ gradient compared with Na(+)-free conditions. Kinetic analysis of initial taurine uptake yielded no evidence for a carrier-mediated transport process. These findings cannot be attributed to an overall inability

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Plichta, Veronika, Johann Steinwider, Nina Vogel, et al. "Risk Assessment of Dietary Exposure to Organophosphorus Flame Retardants in Children by Using HBM-Data." Toxics 10, no. 5 (2022): 234. http://dx.doi.org/10.3390/toxics10050234.

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Due to their extensive usage, organophosphorus flame retardants (OPFRs) have been detected in humans and in the environment. Human are exposed to OPFRs via inhalation of indoor air, dust uptake or dietary uptake through contaminated food and drinking water. Only recently, few studies addressing dietary exposure to OPFRs were published. In this study, we used human biomonitoring (HBM) data of OPFRs to estimate how much the dietary intake may contribute to the total exposure. We estimated by reverse dosimetry, the daily intake of tris (2-chloroethyl) phosphate (TCEP), tris (1-chloro-2-propyl) ph

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Uranga, Ana Paola, James Levine, and Michael Jensen. "Isotope tracer measures of meal fatty acid metabolism: reproducibility and effects of the menstrual cycle." American Journal of Physiology-Endocrinology and Metabolism 288, no. 3 (2005): E547—E555. http://dx.doi.org/10.1152/ajpendo.00340.2004.

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Oxidation and adipose tissue uptake of dietary fat can be measured by adding fatty acid tracers to meals. These studies were conducted to measure between-study variability of these types of experiments and assess whether dietary fatty acids are handled differently in the follicular vs. luteal phase of the menstrual cycle. Healthy normal-weight men ( n = 12) and women ( n = 12) participated in these studies, which were block randomized to control for study order, isotope ([3H]triolein vs. [14C]triolein), and menstrual cycle. Energy expenditure (indirect calorimetry), meal fatty acid oxidation,

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Liu, Gege, Lixia Chen, Haining Tian, et al. "Adult Triploid Rainbow Trout Can Adapt to Various Dietary Lipid Levels by Coordinating Metabolism in Different Tissues." Metabolites 13, no. 3 (2023): 396. http://dx.doi.org/10.3390/metabo13030396.

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Triploid rainbow trout can adapt to various dietary lipid levels; however, the mechanisms of systematic adaptation are not well understood. To investigate how adult triploid rainbow trout maintains lipid hemostasis under different exogenous lipid intake, a 77-day feeding trial was conducted. Diets with lipid contents of 20%, 25%, and 30% were formulated and fed to triploid rainbow trout with an initial weight of 3 ± 0.02 kg, and they were named L20, L25, and L30 group, respectively. Results showed that the condition factor, hepatosomatic index, liver color, and plasma triglyceride were compara

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Cermak, Rainer, Sandra Landgraf, and Siegfried Wolffram. "Quercetin glucosides inhibit glucose uptake into brush-border-membrane vesicles of porcine jejunum." British Journal of Nutrition 91, no. 6 (2004): 849–55. http://dx.doi.org/10.1079/bjn20041128.

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Recent experimental data point to an interaction of dietary flavonol monoglucosides with the intestinal Na-dependent glucose transporter 1 (SGLT1). To investigate this interaction in more detail, we performed experiments with SGLT1-containing brush-border-membrane vesicles (BBMV) from pig jejunum. The flavonol quercetin-3-O-glucoside (Q3G) concentration-dependently inhibited Na-dependent uptake of radioactively labelled d-glucose into BBMV. Uptake of l-leucine was not inhibited by Q3G, indicating a specific interaction of the glucoside with SGLT1. Whereas the maximal transport rate of concentr

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