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Journal articles on the topic 'Diffuse myocardial fibrosis'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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1

Spinale, Francis G. "Diffuse Myocardial Fibrosis." JACC: Basic to Translational Science 10, no. 6 (2025): 860–61. https://doi.org/10.1016/j.jacbts.2025.03.011.

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2

Katbeh, Asim, Tomas Ondrus, Emanuele Barbato, et al. "Imaging of Myocardial Fibrosis and Its Functional Correlates in Aortic Stenosis: A Review and Clinical Potential." Cardiology 141, no. 3 (2018): 141–49. http://dx.doi.org/10.1159/000493164.

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Patients with severe aortic stenosis (AS) show progressive fibrotic changes in the myocardium, which may impair cardiac function and patient outcomes even after successful aortic valve replacement. Detection of patients who need an early operation remains a diagnostic challenge as myocardial functional changes may be subtle. In recent years, speckle tracking echocardiography (STE) and cardiac magnetic resonance mapping have been shown to provide complementary information for the assessment of left ventricular mechanics and identification of subtle damage by focal or diffuse myocardial fibrosis
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3

Shen, Jianhong, Zanfei Li, and Xianliang Ren. "Clinical Application of Information Medicine Cardiac Magnetic Resonance Quantitative Imaging in Central Nervous System Cardiology Cardiomyopathy." Journal of Medical Imaging and Health Informatics 11, no. 2 (2021): 623–29. http://dx.doi.org/10.1166/jmihi.2021.3369.

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Objective: To study the left ventricular systolic function in patients with central nervous system endocarditis myocarditis by using information-based cardiac magnetic resonance quantitative imaging technique. LGE and T1 mapping techniques were used to evaluate myocardial localized fibrosis and diffuse fibrosis, respectively. The situation is to explore the limitations and the relationship between diffuse cardiomyopathy and cardiac function. Methods: A total of 44 patients with dilated cardiomyopathy and 23 healthy volunteers were scanned with a magnetic resonance imager to determine the occur
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Screever, Elles M., Thomas M. Gorter, Tineke P. Willems, et al. "Diffuse Myocardial Fibrosis on Cardiac Magnetic Resonance Imaging Is Related to Galectin-3 and Predicts Outcome in Heart Failure." Biomolecules 13, no. 3 (2023): 410. http://dx.doi.org/10.3390/biom13030410.

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Aims: Ongoing adverse remodeling is a hallmark of heart failure (HF), which might be reflected by either focal or diffuse myocardial fibrosis. Therefore, in (pre)clinical settings, we used immunohistochemistry or cardiac magnetic resonance imaging (CMR) to investigate the association of (focal or diffuse) fibrosis with cardiac biomarkers and adverse events in HF. Methods and results: In C57Bl/6J mice, we determined the presence and extent of myocardial fibrosis 6 weeks post-myocardial infarction (MI). Furthermore, we studied 159 outpatient HF patients who underwent CMR, and determined focal an
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5

Niss, Omar, Robert Fleck, Fowe Makue, et al. "Association between diffuse myocardial fibrosis and diastolic dysfunction in sickle cell anemia." Blood 130, no. 2 (2017): 205–13. http://dx.doi.org/10.1182/blood-2017-02-767624.

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Key Points Diffuse myocardial fibrosis is a common and novel mechanism of heart disease in SCA that can be detected noninvasively. Diffuse myocardial fibrosis is strongly associated with diastolic dysfunction in individuals with SCA.
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Plehn, Jonathan F., and Robert W. W. Biederman. "Diffuse Myocardial Fibrosis in Dilated Cardiomyopathy." JACC: Cardiovascular Imaging 15, no. 4 (2022): 591–93. http://dx.doi.org/10.1016/j.jcmg.2022.01.013.

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7

Karamitsos, Theodoros D., and Stefan Neubauer. "Detecting Diffuse Myocardial Fibrosis With CMR." JACC: Cardiovascular Imaging 6, no. 6 (2013): 684–86. http://dx.doi.org/10.1016/j.jcmg.2012.10.030.

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8

Dweck, Marc, and Rong Bing. "Diffuse Myocardial Fibrosis in Aortic Stenosis." JACC: Cardiovascular Imaging 12, no. 1 (2019): 120–22. http://dx.doi.org/10.1016/j.jcmg.2018.06.026.

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9

Moon, James C., Thomas A. Treibel, and Erik B. Schelbert. "T1 Mapping for Diffuse Myocardial Fibrosis." Journal of the American College of Cardiology 62, no. 14 (2013): 1288–89. http://dx.doi.org/10.1016/j.jacc.2013.05.077.

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10

Lao, Qun, Wenping Xia, Jing Jin, Yuzhu Jia, and Jianju Feng. "Modified Look-Locker Inverse-Recovery (MOLLI) Sequence of Quantitative Imaging in Dirty Magnetic Resonance Longitudinal Relaxation Time Diagnostic Value of GE Combined with Longitudinal Relaxation Time Quantitative Imaging for Myocardial Amyloidosis." Journal of Healthcare Engineering 2021 (October 19, 2021): 1–12. http://dx.doi.org/10.1155/2021/2800891.

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The pathological changes of myocarditis include degeneration and necrosis of myocardial cells and infiltration of inflammatory cells in the myocardial interstitium, accompanied by obvious myocardial fibrosis. Myocardial fibrosis is a determinant of ventricular remodeling and an important indicator of the classification of clinical risk factors and has an important value in evaluating the prognosis of heart disease. Cardiac magnetic resonance (CMR) is the “gold standard” for evaluating the shape and function of the heart, and it can show the characteristic pathological changes of myocardial tis
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11

Sado, Daniel M., Andrew S. Flett, and James C. Moon. "Novel imaging techniques for diffuse myocardial fibrosis." Future Cardiology 7, no. 5 (2011): 643–50. http://dx.doi.org/10.2217/fca.11.45.

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12

Thein, Swee Lay, and Vandana Sachdev. "Diffuse myocardial fibrosis as an SCD biomarker." Blood 140, no. 11 (2022): 1191–92. http://dx.doi.org/10.1182/blood.2022017725.

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13

Xie, Jiajun, Xuhua Jian, Qiyang Lu, et al. "T1 Mapping for Noninvasively Detecting Diffuse Fibrosis in Severe Aortic Stenosis." Journal of Medical Imaging and Health Informatics 10, no. 7 (2020): 1534–39. http://dx.doi.org/10.1166/jmihi.2020.3076.

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Purpose: To evaluate myocardial diffuse fibrosis in severe aortic stenosis (SAS) with cardiac magnetic resonance imaging (MRI) T1 mapping technique. Methods: Twenty-seven SAS patients and 15 controls were enrolled and performed cardiac MRI. Left ventricular (LV) structure, function and T1-derived parameters were measured to compare between SAS group and the controls. Correlation between T1-derived parameters and the extent of histologic fibrosis was performed in 15 patients who underwent aortic valve replacement surgery and myocardial biopsy. Results: The SAS group had LV remodeling with ventr
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14

López, Begoña, Susana Ravassa, María U. Moreno, et al. "Diffuse myocardial fibrosis: mechanisms, diagnosis and therapeutic approaches." Nature Reviews Cardiology 18, no. 7 (2021): 479–98. http://dx.doi.org/10.1038/s41569-020-00504-1.

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15

Riesenkampff, Eugénie, Ching Kit Chen, Paul F. Kantor, et al. "Diffuse Myocardial Fibrosis in Children After Heart Transplantations." Transplantation 99, no. 12 (2015): 2656–62. http://dx.doi.org/10.1097/tp.0000000000000769.

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16

Osório, Joana. "New CMR approach to measure diffuse myocardial fibrosis." Nature Reviews Cardiology 7, no. 10 (2010): 542. http://dx.doi.org/10.1038/nrcardio.2010.135.

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17

Ntusi, Ntobeko A. B., Stefan K. Piechnik, Jane M. Francis, et al. "Diffuse Myocardial Fibrosis and Inflammation in Rheumatoid Arthritis." JACC: Cardiovascular Imaging 8, no. 5 (2015): 526–36. http://dx.doi.org/10.1016/j.jcmg.2014.12.025.

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18

von Stumm, Maria, Johannes Petersen, Martin Sinn, et al. "Correlation of Myocardial Native T1 and Left Ventricular Reverse Remodeling after Valvular Surgery." Journal of Clinical Medicine 12, no. 7 (2023): 2649. http://dx.doi.org/10.3390/jcm12072649.

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Myocardial native T1 is a known cardiovascular magnetic resonance (CMR) imaging biomarker to quantify diffuse myocardial fibrosis in valvular cardiomyopathy. We hypothesized that diffuse myocardial fibrosis assessed by preoperative T1 mapping might correlate with LV reverse remodeling after valvular surgery. A prospective monocentric cohort study was conducted including 79 consecutive patients with valvular cardiomyopathy referred for surgical treatment of severe aortic or severe functional mitral regurgitation. Native T1 values were assessed by CMR before surgery. LV geometry parameters (i.e.
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19

Iyer, Nithin R., Thu-Thao Le, Michelle S. L. Kui, et al. "Markers of Focal and Diffuse Nonischemic Myocardial Fibrosis Are Associated With Adverse Cardiac Remodeling and Prognosis in Patients With Hypertension: The REMODEL Study." Hypertension 79, no. 8 (2022): 1804–13. http://dx.doi.org/10.1161/hypertensionaha.122.19225.

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Background: The prognostic significance of focal and diffuse myocardial fibrosis in patients with cardiovascular risk factors is unclear. Methods: REMODEL (Response of the Myocardium to Hypertrophic Conditions in the Adult Population) is an observational cohort of asymptomatic patients with essential hypertension. All participants underwent cardiovascular magnetic resonance to assess for myocardial fibrosis: nonischemic late gadolinium enhancement (LGE), native myocardial T1, postcontrast myocardial T1, extracellular volume fraction including/excluding LGE regions, interstitial volume (extrace
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20

Lee, Daniel C., Monique E. Hinchcliff, Roberto Sarnari, et al. "Diffuse cardiac fibrosis quantification in early systemic sclerosis by magnetic resonance imaging and correlation with skin fibrosis." Journal of Scleroderma and Related Disorders 3, no. 2 (2018): 159–69. http://dx.doi.org/10.1177/2397198318762888.

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Purpose: To evaluate the utility of cardiac magnetic resonance T1 mapping in early systemic sclerosis and its association with skin score. Methods: In total, 24 consecutive patients with early systemic sclerosis referred for cardiovascular evaluation and 12 controls without systemic sclerosis were evaluated. All patients underwent cine, T1 mapping, and late gadolinium–enhanced cardiac magnetic resonance imaging. T1 mapping indices were compared between systemic sclerosis patients and controls (extracellular volume fraction, gadolinium partition coefficient (λ), pre-contrast T1, and post-contra
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21

Mavrogeni, Sophie I., and Alessia Pepe. "The emerging role of cardiovascular magnetic resonance in the evaluation of cardiac involvement in systemic sclerosis." Rheumatology and Immunology Research 5, no. 2 (2024): 93–98. http://dx.doi.org/10.1515/rir-2024-0012.

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Abstract Systemic sclerosis (SSc) is an autoimmune rheumatic disease, characterized by vascular, inflammatory and fibrotic alterations. Cardiac involvement is the « fatal tip of the iceberg» in SSc, as it leads to high morbidity/mortality. Cardiovascular imaging modalities play an important role in the early diagnosis and treatment assessment of cardiac involvement. Echocardiography is the corner stone for evaluation of cardiac involvement, providing information about function, wall motion, pulmonary pressure, pericardium and valvular disease. It is a low-cost modality, widely available, witho
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22

Gotschy, Alexander, Suzana Jordan, Christian T. Stoeck, et al. "Diffuse myocardial fibrosis precedes subclinical functional myocardial impairment and provides prognostic information in systemic sclerosis." European Heart Journal - Cardiovascular Imaging 24, no. 3 (2022): 373–82. http://dx.doi.org/10.1093/ehjci/jeac094.

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Abstract Aims Myocardial involvement is common in patients with systemic sclerosis (SSc) and causes myocardial fibrosis and subtle ventricular dysfunction. However, the temporal onset of myocardial involvement during the progression of the disease and its prognostic value are yet unknown. We used cardiovascular magnetic resonance (CMR) to investigate subclinical functional impairment and diffuse myocardial fibrosis in patients with very early diagnosis of SSc (VEDOSS) and established SSc and examined whether this was associated with mortality. Methods and results One hundred and ten SSc patien
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23

Nielsen, Signe H., Naja D. Mygind, Marie M. Michelsen, et al. "Accelerated collagen turnover in women with angina pectoris without obstructive coronary artery disease: An iPOWER substudy." European Journal of Preventive Cardiology 25, no. 7 (2018): 719–27. http://dx.doi.org/10.1177/2047487318758750.

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Aim Collagens are major cardiac extracellular matrix components, known to be actively remodelled and accumulated during diffuse myocardial fibrosis. We evaluated whether accelerated collagen turnover described by neo-epitope biomarkers reflecting collagen formation and degradation separates patients with diffuse myocardial fibrosis from asymptomatic controls. Methods and results Seventy-one women with angina pectoris without significant coronary artery disease assessed by invasive coronary angiogram were included. Competitive enzyme-linked immunosorbent assays (ELISAs) measuring circulating pr
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24

Meloni, Antonella, Laura Pistoia, Vincenzo Positano, et al. "Association between Myocardial Iron Overload and Diffuse Myocardial Fibrosis in Thalassemia Major." Blood 140, Supplement 1 (2022): 2489–90. http://dx.doi.org/10.1182/blood-2022-155866.

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25

乔, 斌杰. "Progress in Diagnosis and Treatment of Diffuse Myocardial Fibrosis." Advances in Clinical Medicine 13, no. 01 (2023): 356–61. http://dx.doi.org/10.12677/acm.2023.131054.

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26

Lee, Hyun-Jung, Heesun Lee, Sung Mok Kim, et al. "Diffuse Myocardial Fibrosis and Diastolic Function in Aortic Stenosis." JACC: Cardiovascular Imaging 13, no. 12 (2020): 2561–72. http://dx.doi.org/10.1016/j.jcmg.2020.07.007.

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27

Cochet, Hubert, Xavier Iriart, Antoine Allain-Nicolaï, et al. "Focal scar and diffuse myocardial fibrosis are independent imaging markers in repaired tetralogy of Fallot." European Heart Journal - Cardiovascular Imaging 20, no. 9 (2019): 990–1003. http://dx.doi.org/10.1093/ehjci/jez068.

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Abstract Aims To identify the correlates of focal scar and diffuse fibrosis in patients with history of tetralogy of Fallot (TOF) repair. Methods and results Consecutive patients with prior TOF repair underwent electrocardiogram, 24-h Holter, transthoracic echocardiography, exercise testing, and cardiac magnetic resonance (CMR) including cine imaging to assess ventricular volumes and ejection fraction, T1 mapping to assess left ventricular (LV) and right ventricular (RV) diffuse fibrosis, and free-breathing late gadolinium-enhanced imaging to quantify scar area at high spatial resolution. Stru
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28

Engele, Leo J., Roel L. F. van der Palen, Anastasia D. Egorova, et al. "Cardiac Fibrosis and Innervation State in Uncorrected and Corrected Transposition of the Great Arteries: A Postmortem Histological Analysis and Systematic Review." Journal of Cardiovascular Development and Disease 10, no. 4 (2023): 180. http://dx.doi.org/10.3390/jcdd10040180.

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In the transposition of the great arteries (TGA), alterations in hemodynamics and oxygen saturation could result in fibrotic remodeling, but histological studies are scarce. We aimed to investigate fibrosis and innervation state in the full spectrum of TGA and correlate findings to clinical literature. Twenty-two human postmortem TGA hearts, including TGA without surgical correction (n = 8), after Mustard/Senning (n = 6), and arterial switch operation (ASO, n = 8), were studied. In newborn uncorrected TGA specimens (1 day–1.5 months), significantly more interstitial fibrosis (8.6% ± 3.0) was o
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Mavrogeni, Sophie, Dimitris Apostolou, Panayiotis Argyriou, et al. "T1 and T2 Mapping in Cardiology: “Mapping the Obscure Object of Desire”." Cardiology 138, no. 4 (2017): 207–17. http://dx.doi.org/10.1159/000478901.

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The increasing use of cardiovascular magnetic resonance (CMR) is based on its capability to perform biventricular function assessment and tissue characterization without radiation and with high reproducibility. The use of late gadolinium enhancement (LGE) gave the potential of non-invasive biopsy for fibrosis quantification. However, LGE is unable to detect diffuse myocardial disease. Native T1 mapping and extracellular volume fraction (ECV) provide knowledge about pathologies affecting both the myocardium and interstitium that is otherwise difficult to identify. Changes of myocardial native T
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30

Guglielmo, Marco, and Gianluca Pontone. "Clinical implications of cardiac magnetic resonance imaging fibrosis." European Heart Journal Supplements 24, Supplement_I (2022): I123—I126. http://dx.doi.org/10.1093/eurheartjsupp/suac085.

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Abstract Cardiac magnetic resonance (CMR) is a non-invasive imaging method that allows to characterize myocardial tissue. In particular, using the late gadolinium enhancement technique, it is possible to identify areas of focal fibrosis. Specific distribution patterns of this fibrosis allow us to distinguish ischaemic cardiomyopathy (iCMP) from non-ischaemic cardiomyopathy (nCMP) and sometimes to identify the aetiology of the latter. Diffuse fibrosis can also be identified using the parametric T1 mapping sequences. For this purpose, the native T1 of the tissue is measured before the administra
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31

Niss, Omar, Michael D. Taylor, Robert Fleck, et al. "Diffuse Myocardial Fibrosis Is a Common Feature of Sickle Cell Anemia That Is Associated with Diastolic Dysfunction and Restrictive Cardiac Physiology." Blood 128, no. 22 (2016): 8. http://dx.doi.org/10.1182/blood.v128.22.8.8.

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Abstract Background: We have recently shown that the cardiomyopathy of sickle cell anemia (SCA) is characterized by restrictive physiology (diastolic dysfunction, left atrial [LA] enlargement and normal systolic function) superimposed on hyperdynamic features (left ventricular [LV] enlargement and eccentric hypertrophy) (JACC Cardiovasc Imaging 9:244-253;2016; PNAS 2016 in press). Similar to other restrictive cardiomyopathies, SCA-related cardiomyopathy may lead to mild, secondary pulmonary hypertension (PH) with elevated tricuspid regurgitant jet velocity (TRV), and can be complicated by arrh
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32

Rajah, Megan R., Erna Marais, Gerald J. Maarman, Emma Doubell, Anton F. Doubell, and Philip G. Herbst. "Utility of Serum Biomarkers of Myocardial Fibrosis in High-Gradient Severe Aortic Stenosis: An Explorative Cardiovascular Magnetic Resonance Imaging-Based Study." Diagnostics 15, no. 9 (2025): 1143. https://doi.org/10.3390/diagnostics15091143.

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Background: Myocardial fibrosis in aortic stenosis (AS) is associated with a significant risk of poor clinical outcomes. Myocardial fibrosis can be evaluated using cardiovascular magnetic resonance (CMR) imaging and may be useful for risk-stratifying patients at high risk for poorer outcomes. A circulating biomarker of fibrosis may be a cheaper, more accessible alternative to CMR in lower-to-middle-income countries. This study evaluated the correlation between serum biomarkers of myocardial fibrosis (TGF-β1, PICP, and PIIINP) with CMR markers of myocardial fibrosis (T1 mapping, extracellular v
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33

Sharma, Akshay, Anthony Merlocco, Cara Morin, et al. "Myocardial Fibrosis Improves in Young Patients with Sickle Cell Disease after Hematopoietic Cell Transplantation." Blood 142, Supplement 1 (2023): 2518. http://dx.doi.org/10.1182/blood-2023-178267.

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Introduction: Cardiovascular complications are a leading cause of early mortality in patients with sickle cell disease (SCD). Chronic anemia, iron overload and repeated microvascular ischemic insults with reperfusion injury often result in progressive myocardial injury and maladaptive remodeling in SCD patients. Patients with SCD develop diffuse myocardial fibrosis, which can be detected by quantifying the myocardial extracellular volume (ECV) using cardiovascular magnetic resonance (CMR) imaging. Our group and others have previously shown that diffuse myocardial fibrosis is highly prevalent i
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34

Hanneman, Kate, and Rachel Wald. "Cardiac MRI T1 mapping in unrepaired anomalous left coronary artery from the pulmonary artery." Cardiology in the Young 27, no. 9 (2017): 1832–35. http://dx.doi.org/10.1017/s1047951117000932.

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AbstractA 29-year-old gravida1para1woman presented with increasing fatigue. Multi-modality imaging demonstrated the left coronary artery arising from the main pulmonary artery with large collateral vessels in the interventricular septum, in keeping with unrepaired anomalous left coronary artery from the pulmonary artery. Cardiac MRI T1 mapping demonstrated globally elevated non-contrast T1 and extracellular volume fraction values, which suggested the presence of diffuse interstitial myocardial fibrosis. Cardiac MRI T1 mapping allows for a new dimension of myocardial characterisation, providing
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35

Evdokimova, Larisa S., Irina E. Itskovich, Tatiana N. Novikova, and Tatiana V. Garpinchenko. "Atrial fibrillation in a patient with diffuse myocardial fibrosis and mitral annular disjunction." Cardiac Arrhythmias 3, no. 3 (2024): 19–26. http://dx.doi.org/10.17816/cardar567797.

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A case of atrial fibrillation and premature ventricular complexes (PVC) in a patient with mitral valve prolapse and mitral annular disjunction is described. Rhythm disturbances occurred after a new coronavirus infection. Also, the patient has a history of combined treatment of left breast cancer, which contributed to the appearance of myocardial fibrosis as an arrhythmogenic substrate. Due to the ineffectiveness of conservative antiarrhythmic therapy, a radiofrequency catheter procedure was performed, which proved unsuccessful. The purpose of the article is to present the possible causes of ca
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36

Hwang, In-Chang, Hyung-Kwan Kim, Jun-Bean Park, et al. "Aortic valve replacement-induced changes in native T1 are related to prognosis in severe aortic stenosis: T1 mapping cardiac magnetic resonance imaging study." European Heart Journal - Cardiovascular Imaging 21, no. 6 (2019): 653–63. http://dx.doi.org/10.1093/ehjci/jez201.

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Abstract Aims Native T1 times from T1 mapping cardiac magnetic resonance (CMR) are associated with myocardial fibrosis in aortic stenosis (AS). We investigated whether changing patterns in native T1 predict clinical outcomes after aortic valve replacement (AVR) in severe AS patients. Methods and results Forty-three patients with severe AS (65.9 ± 8.1 years; 24 men) who underwent T1 mapping CMR at baseline and 1 year after AVR were prospectively enrolled. Upper limit of native T1 from healthy volunteers was used to define normal myocardium and diffuse fibrosis (native T1 < 1208.4 and ≥12
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Ali, Nahla D., Noha Behairy, Ahmed Kharabish, Wesam Elmozy, Ahmed Yahya Hegab, and Samira Saraya. "Cardiac MRI T1 mapping and extracellular volume application in hypertrophic cardiomyopathy." Egyptian Journal of Radiology and Nuclear Medicine 52, no. 1 (2021). http://dx.doi.org/10.1186/s43055-021-00419-y.

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Abstract Background Hypertrophic cardiomyopathy (HCM) is one of the commonest inheritable cardiac disorders. Being a global disease with diffuse myocardial fibrosis, it has a wide range of adverse outcomes ending with sudden cardiac death. Cardiac magnetic resonance (CMR) with late gadolinium enhancement (LGE) has become a reference standard for visualization of focal myocardial fibrosis. In the setting of less severe or more diffuse fibrosis, LGE is unlikely to reveal the presence of abnormal tissue given the lack of normal myocardium as a reference. Direct measurement of myocardial T1 time (
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Katbeh, Asim, Tomas Ondrus, Emanuele Barbato, et al. "Imaging of Myocardial Fibrosis and Its Functional Correlates in Aortic Stenosis: A Review and Clinical Potential." December 5, 2018. https://doi.org/10.1159/000493164.

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Patients with severe aortic stenosis (AS) show progressive fibrotic changes in the myocardium, which may impair cardiac function and patient outcomes even after successful aortic valve replacement. Detection of patients who need an early operation remains a diagnostic challenge as myocardial functional changes may be subtle. In recent years, speckle tracking echocardiography (STE) and cardiac magnetic resonance mapping have been shown to provide complementary information for the assessment of left ventricular mechanics and identification of subtle damage by focal or diffuse myocardial fibrosis
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39

Sun, Jianhong, Yuanwei Xu, Yang Wu, et al. "The diagnostic value of sST2 for myocardial fibrosis in idiopathic inflammatory myopathies in subclinical stage of cardiac involvement." Rheumatology, April 24, 2023. http://dx.doi.org/10.1093/rheumatology/kead182.

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Abstract Objective Myocardial fibrosis occurs in the early subclinical stage of cardiac involvement in idiopathic inflammatory myopathies (IIM). Soluble suppression of tumorigenicity 2 (sST2) is known to have an immunomodulatory impact during autoimmune disease development. The current study investigated the diagnostic value of sST2 for myocardial fibrosis during early stage of cardiac involvement in IIM. Methods A total of 44 IIM patients with normal heart function and 32 age- and gender-matched healthy controls (HCs) were enrolled. Serum sST2 levels were measured by ELISA and cardiac magneti
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40

Weiner, Jeremy, Corinna Heinisch, Salome Oeri, et al. "Focal and diffuse myocardial fibrosis both contribute to regional hypoperfusion assessed by post-processing quantitative-perfusion MRI techniques." Frontiers in Cardiovascular Medicine 10 (September 19, 2023). http://dx.doi.org/10.3389/fcvm.2023.1260156.

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IntroductionIndications for stress-cardiovascular magnetic resonance imaging (CMR) to assess myocardial ischemia and viability are growing. First pass perfusion and late gadolinium enhancement (LGE) have limited value in balanced ischemia and diffuse fibrosis. Quantitative perfusion (QP) to assess absolute pixelwise myocardial blood flow (MBF) and extracellular volume (ECV) as a measure of diffuse fibrosis can overcome these limitations. We investigated the use of post-processing techniques for quantifying both pixelwise MBF and diffuse fibrosis in patients with clinically indicated CMR stress
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Andresen, K., LG Klaeboe, ØH Lie, et al. "No signs of diffuse myocardial fibrosis by T1 mapping in male elite endurance athletes." European Heart Journal - Cardiovascular Imaging 23, Supplement_1 (2022). http://dx.doi.org/10.1093/ehjci/jeab289.423.

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Abstract Funding Acknowledgements Type of funding sources: Public hospital(s). Main funding source(s): Oslo University Hospital Rikshospitalet, Department of Cardiology, Oslo, Norway. Background/Introduction: Observational data indicating increased prevalence of focal myocardial fibrosis in endurance athletes have raised concerns regarding the potential detrimental cardiac consequences of long-term athleticism. Cumulative exercise and competitive exposure has been associated with focal myocardial fibrosis visualised as late gadolinium enhancement (LGE) on cardiac magnetic resonance (CMR) imagi
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42

Demolder, Anthony, Dan Devos, Julie De Backer, and Laura Muiño‐Mosquera. "Assessment of Myocardial Fibrosis in Marfan Syndrome Using Cardiac Magnetic Resonance Imaging." Molecular Genetics & Genomic Medicine 12, no. 11 (2024). http://dx.doi.org/10.1002/mgg3.70024.

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ABSTRACTBackgroundImpaired myocardial function and arrhythmia are important manifestations of Marfan syndrome (MFS). Studies assessing myocardial fibrosis in relation to these manifestations are scarce.MethodsThis cross‐sectional, single‐center study assessed ventricular volumes, ventricular function, and myocardial fibrosis by cardiac magnetic resonance imaging (CMR) in patients with MFS harboring a (likely) pathogenic FBN1 variant. The presence and extent of fibrosis were assessed by late gadolinium enhancement (LGE) and extracellular volume measurement (ECV). Data on 24‐h Holter monitoring
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43

Dusenbery, Susan M., Jane W. Newburger, Kimberlee Gauvreau, Annette Baker, and Andrew J. Powell. "Abstract O.56: Myocardial Fibrosis in Patients with a History of Kawasaki Disease: a Pilot Study." Circulation 131, suppl_2 (2015). http://dx.doi.org/10.1161/circ.131.suppl_2.o56.

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Background: Myocarditis occurs in acute Kawasaki disease (KD), and case series with endomyocardial biopsy report that KD patients (pts) may have myocardial fibrosis. The new cardiac magnetic resonance (CMR) technique of myocardial T1 measurement is a noninvasive means of measuring diffuse myocardial fibrosis. We sought to assess the prevalence and risk factors for diffuse myocardial fibrosis in KD pts using CMR. Methods: In this retrospective study, all pts with KD who had a CMR with extracellular volume fraction (ECV) measurement at Boston Children’s Hospital were included. The ECV, a measure
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Petrescu, ANIELA, M. Cvijic, S. Bezy, et al. "Can shear wave imaging distinguish between diffuse interstitial and replacement myocardial fibrosis?" European Heart Journal - Cardiovascular Imaging 22, Supplement_1 (2021). http://dx.doi.org/10.1093/ehjci/jeaa356.009.

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Abstract Funding Acknowledgements Type of funding sources: None. Background Diffuse interstitial or myocardial replacement fibrosis are common features of a large variety of cardiomyopathies. These alterations contribute to functional changes, particularly to an increased myocardial stiffness (MS). Histological examination is the gold standard for myocardial fibrosis quantification, however, it requires endomyocardial biopsy which is invasive and not without risks. Cardiac magnetic resonance (CMR) can characterize the extent of both diffuse and replacement fibrosis and may have prognostic valu
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Andresen, Kristoffer, Lars Gunnar Klæboe, Øyvind Haugen Lie, et al. "No adverse association between exercise exposure and diffuse myocardial fibrosis in male endurance athletes." Scientific Reports 14, no. 1 (2024). http://dx.doi.org/10.1038/s41598-024-57233-5.

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AbstractThe potential association between endurance exercise and myocardial fibrosis is controversial. Data on exercise exposure and diffuse myocardial fibrosis in endurance athletes are scarce and conflicting. We aimed to investigate the association between exercise exposure and markers of diffuse myocardial fibrosis by cardiovascular magnetic resonance imaging (CMR) in endurance athletes. We examined 27 healthy adult male competitive endurance athletes aged 41 ± 9 years and 16 healthy controls in a cross sectional study using 3 Tesla CMR including late gadolinium enhancement and T1 mapping.
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Li, Song-Nan, Lei Zhao, Lu Wang, et al. "Abstract 13451: Assessment of Diffuse Ventricular Fibrosis in Atrial Fibrillation Using Extracellular Volume Fraction: Initial Study." Circulation 132, suppl_3 (2015). http://dx.doi.org/10.1161/circ.132.suppl_3.13451.

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Objectives: In atrial fibrillation (AF), diffuse myocardial fibrosis may be induced by arrhythmia or reflect pre-existing cardiomyopathy. The presence of concurrent AF and diffuse myocardial fibrosis has been associated with an increased risk of heart failure progression.Unlike focal fibrosis, diffuse myocardial fibrosis is not visualized on delayed enhancement magnetic resonance imaging (MRI), but can be quantified with ECV fraction. Hypothesis: The extracellular volume (ECV) fraction may provide new insights to evaluate diffuse myocardial fibrosis of the left ventricle (LV) in patients with
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47

Ross, Laura, Benedict Costello, Zoe Brown, et al. "Myocardial fibrosis and arrhythmic burden in systemic sclerosis." Rheumatology, February 8, 2022. http://dx.doi.org/10.1093/rheumatology/keac065.

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Abstract Objectives Cardiac complications of SSc are a leading cause of SSc-associated death. Cardiac imaging for identifying substrate abnormality may be useful in predicting risk of cardiac arrhythmias or future cardiac failure. The aim of this study was to quantify the burden of asymptomatic fibro-inflammatory myocardial disease using cardiac magnetic resonance imaging (CMR) and assess the relationship between asymptomatic myocardial fibrosis and cardiac arrhythmias in SSc. Methods Thirty-two patients with SSc with no documented history of pulmonary vascular or heart disease underwent CMR w
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Ragab, Haissam, Gunnar K. Lund, Lynn Breitsprecher, et al. "Prevalence and pattern of focal and potential diffuse myocardial fibrosis in male and female marathon runners using contrast-enhanced cardiac magnetic resonance." European Radiology, January 23, 2023. http://dx.doi.org/10.1007/s00330-023-09416-3.

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Abstract Objectives This study analyzed the prevalence and pattern of focal and potential diffuse myocardial fibrosis detected by late gadolinium enhancement (LGE) and extracellular volume (ECV) imaging in male and female marathon runners using cardiac magnetic resonance (CMR). Methods Seventy-four marathon runners were studied including 55 males (44 ± 8 years) and 19 females (36 ± 7 years) and compared to 36 controls with similar age and sex using contrast-enhanced CMR, exercise testing, and blood samples. Results Contrast-enhanced CMR revealed focal myocardial fibrosis in 8 of 74 runners (11
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Joao AC Lima, Sirisha Donekal. "Diffuse Interstitial Myocardial Fibrosis by T1 Myocardial Mapping: Review." Translational Medicine 03, no. 01 (2013). http://dx.doi.org/10.4172/2161-1025.1000111.

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Kwak, Soongu, and Seung-Pyo Lee. "Prognostic role of myocardial fibrosis markers in aortic stenosis: implications for clinical decision-making." Journal of the Heart Valve Society 1, no. 1 (2024). https://doi.org/10.1177/30494826241296412.

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Aortic stenosis (AS) causes chronic pressure overload in the left ventricle (LV) that ultimately results in myocardial fibrosis. This fibrosis is associated with advanced symptoms, LV decompensation, and poor prognosis. Cardiovascular magnetic resonance (CMR) offers a non-invasive method to assess myocardial fibrosis, correlating well with histology and predicting adverse events in AS patients. Histologic studies have confirmed the prevalence of myocardial fibrosis in AS and its association with symptoms and LV dysfunction. CMR techniques, including late gadolinium enhancement (LGE) and T1-map
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