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1

Pohjanvirta, Raimo. The AH receptor in biology and toxicology. Hoboken, N.J: Wiley, 2011.

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2

NATO Advanced Study Institute on Carbon Dioxide: Chemical and Biochemical Uses as a Source of Carbon (1986 Pugnochiuso, Italy). Carbon dioxide as a source of carbon: Biochemical and chemical uses. Dordrecht: D. Reidel Pub. Co., 1987.

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3

The slender thread: Web of life, the story of carbon dioxide. Corpus Christi: Helix Press, 1985.

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4

McKinney, Aubrey R. The slender thread: Web of life, the story of carbon dioxide. Corpus Christi, Tex: Helix Press, 1995.

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5

Thoene, Barbara. Untersuchungen zur Aufnahme und Metabolisierung atmosphärischen Stickstoffdioxyds in oberindischen Organen der Fichte (Picea abies (L.) Karst.). Frankfurt/M: Wissenschafts-Verlag W. Maraun, 1991.

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6

W, Ludden Paul, Burris John E, and Burris Robert H. 1914-, eds. Nitrogen fixation and CO₂ metabolism: Proceedings of the Fourteenth Steenbock Symposium held 17-22 June 1984 at the University of Wisconsin--Madison, Madison, Wisconsin, U.S.A. New York: Elsevier, 1985.

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7

Ludden, P. W., and J. E. Burris. Nitrogen Fixation and Carbon Dioxide Metabolism. Elsevier, 1985.

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8

Nahas, G. Carbon Dioxide and Metabolic Regulations. Springer, 2011.

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9

Pohjanvirta, Raimo. AH Receptor in Biology and Toxicology. Wiley & Sons, Incorporated, John, 2011.

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10

Pohjanvirta, Raimo. AH Receptor in Biology and Toxicology. Wiley & Sons, Incorporated, John, 2011.

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11

Pohjanvirta, Raimo. AH Receptor in Biology and Toxicology. Wiley & Sons, Incorporated, John, 2011.

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12

Pohjanvirta, Raimo. Ah Receptor in Biology and Toxicology. Wiley & Sons, Incorporated, John, 2012.

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13

1919-, Tolbert N. E., and Preiss Jack 1932-, eds. Regulation of atmospheric C0₂ and 0₂ by photosynthetic carbon metabolism. New York: Oxford University Press, 1994.

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14

(Editor), M. Aresta, and G. Forti (Editor), eds. Carbon Dioxide as a Source of Carbon: Biochemical and Chemical Use (NATO Science Series C:). Springer, 1987.

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15

1934-, Brändén Carl-Ivar, Schneider Gunter, and Kungl Svenska vetenskapsakademien, eds. Carbon dioxide fixation and reduction in biological and model systems: Proceedings of the Royal Swedish Academy of Sciences Nobel symposium, 1991. Oxford: Oxford University Press, 1994.

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16

Chakera, Aron, William G. Herrington, and Christopher A. O’Callaghan. Disorders of acid–base balance. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0178.

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Normal metabolism results in a net acid production of approximately 1 mmol/kg day−1. Physiological pH is regulated by excretion of this acid load (as carbon dioxide) by the kidneys and the lungs. A series of buffers in the body reduces the effects of metabolic acids on body and urine pH. For acid–base disorders to occur, there must be excessive intake (or loss) of acid (or base) or, alternatively, an inability to excrete acid. For these changes to result in a substantially abnormal pH, the various buffer systems must been overwhelmed. The pH scale is logarithmic, so relatively small changes in
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17

Ho, Kwok M. Kidney and acid–base physiology in anaesthetic practice. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0005.

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Anatomically the kidney consists of the cortex, medulla, and renal pelvis. The kidneys have approximately 2 million nephrons and receive 20% of the resting cardiac output making the kidneys the richest blood flow per gram of tissue in the body. A high blood and plasma flow to the kidneys is essential for the generation of a large amount of glomerular filtrate, up to 125 ml min−1, to regulate the fluid and electrolyte balance of the body. The kidneys also have many other important physiological functions, including excretion of metabolic wastes or toxins, regulation of blood volume and pressure
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18

Burton, Derek, and Margaret Burton. Excretion. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198785552.003.0008.

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Excretion is the removal of metabolic wastes such as ammonia, carbon dioxide, ions and water as well as toxic xenobiotics and metals. The process involves the gills, kidney, liver and rectal gland (elasmobranchs and coelacanth). In the liver, amino acids, haemoglobin, steroids and molecules resulting from human activities are transformed to excretable products. The rectal gland excretes ions, notably Na+ and Cl−. The kidney in teleosts has a distinction between an anterior head-kidney containing haematopoietic tissue and endocrine tissue and the posterior region with nephrons (kidney tubules).
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19

U, Sliwka, and United States. National Aeronautics and Space Administration., eds. Effects of sustained low-level elevations of carbon dioxide on cerebral blood flow and autoregulation of the intracerebral arteries in humans. [Washington, DC: National Aeronautics and Space Administration, 1996.

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20

Jacobsen, Dean, and Olivier Dangles. Strategies and adaptations to aquatic life at high altitude. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198736868.003.0005.

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Chapter 5 is focused on how organisms cope with the environmental conditions that are a direct result of high altitude. Organisms reveal a number of fascinating ways of dealing with a life at high altitude; for example, avoidance and pigmentation as protection against damaging high levels of ultraviolet radiation, accumulation of antifreeze proteins, and metabolic cold adaptation among species encountering low temperatures with the risk of freezing, oxy-regulatory capacity in animals due to low availability of oxygen, and root uptake from the sediment of inorganic carbon by plants living in wa
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21

Joynt, Gavin M., and Gordon Y. S. Choi. Blood gas analysis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0072.

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Arterial blood gases allow the assessment of patient oxygenation, ventilation, and acid-base status. Blood gas machines directly measure pH, and the partial pressures of carbon dioxide (PaCO2) and oxygen (PaO2) dissolved in arterial blood. Oxygenation is assessed by measuring PaO2 and arterial blood oxygen saturation (SaO2) in the context of the inspired oxygen and haemoglobin concentration, and the oxyhaemoglobin dissociation curve. Causes of arterial hypoxaemia may often be elucidated by determining the alveolar–arterial oxygen gradient. Ventilation is assessed by measuring the PaCO2 in the
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22

The Arterial Chemoreceptors. Springer, 2006.

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23

(Editor), Yoshiaki Hayashida, Constancio Gonzalez (Editor), and Hisatake Kondo (Editor), eds. The Arterial Chemoreceptors (Advances in Experimental Medicine and Biology). Springer, 2006.

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24

Kirchman, David L. Degradation of organic matter. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789406.003.0007.

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The aerobic oxidation of organic material by microbes is the focus of this chapter. Microbes account for about 50% of primary production in the biosphere, but they probably account for more than 50% of organic material oxidization and respiration (oxygen use). The traditional role of microbes is to degrade organic material and to release plant nutrients such as phosphate and ammonium as well as carbon dioxide. Microbes are responsible for more than half of soil respiration, while size fractionation experiments show that bacteria are also responsible for about half of respiration in aquatic hab
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25

G, O'Regan R., and International Symposium on Arterial Chemoreceptors (12th : 1993 : Dublin, Ireland), eds. Arterial chemoreceptors: Cell to system. New York: Plenum Press, 1994.

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