Academic literature on the topic 'Disciform scar'

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Journal articles on the topic "Disciform scar"

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Yoon, Won Tae, Jong Woo Kim, Chul Gu Kim, and Jae Hui Kim. "Proportion and Reasons for Ineligibility to Re-register for Extended Health Insurance in Neovascular Age-related Macular Degeneration." Journal of the Korean Ophthalmological Society 62, no. 7 (July 15, 2021): 948–56. http://dx.doi.org/10.3341/jkos.2021.62.7.948.

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Purpose: To evaluate the proportion and reasons for ineligibility to re-register for extended health insurance at 5 years in patients diagnosed with neovascular age-related macular degeneration (AMD) and registered for extended health insurance. Methods: This retrospective study was performed in patients diagnosed with neovascular AMD and registered for extended health insurance with follow-up for at least 5 years. The criteria for re-registration for extended health insurance were determined along with the ineligibility for re-registration. Results: In total, 263 patients were included in the
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Chuah, Chin Tek, and Caroline Chee. "Idiopathic polypoidal choroidal vasculopathy as a cause of a disciform macular scar." Clinical and Experimental Ophthalmology 31, no. 2 (April 2003): 163–65. http://dx.doi.org/10.1046/j.1442-9071.2003.00627.x.

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Kurysheva, N. I., O. A. Pererva, and A. A. Ivanova. "Clinical cases of the formation of outer retinal tubulations after aflibercept injections in exudative and disciform age-related macular degeneration." POINT OF VIEW. EAST – WEST, no. 4 (November 2, 2021): 71–73. http://dx.doi.org/10.25276/2410-1257-2021-4-71-73.

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Outer retinal tubulation (ORT) is a common finding in optical coherence tomography, which is usually associated with age-related macular degeneration. It is not clear are ORT appear as a sign of a degenerative process in the outer retinal layers or a way of photoreceptors survival. There are no data on the dynamics of ORT associated with angiogenesis inhibitors injections. This paper presents two clinical cases of dynamic observation of tubulation formation in age-related macular degeneration in the disciform scar stage and in the exudation stage after Aflibercept injections based on optical c
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Lee, Junyeop, You Na Kim, and June-Gone Kim. "Monthly Alternating Injections of Aflibercept and Bevacizumab for Neovascular Age-Related Macular Degeneration." Journal of Clinical Medicine 11, no. 6 (March 11, 2022): 1543. http://dx.doi.org/10.3390/jcm11061543.

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We investigated the efficacy of monthly alternating injections of aflibercept and bevacizumab (MAAB) for maintenance treatment in patients with neovascular age-related macular degeneration (AMD) who showed improvement with the initial monthly injections but presented with rapid worsening after conversion to bimonthly injections. We included 72 patients with neovascular AMD who showed improvement with loading injections of aflibercept. For maintenance treatment, bevacizumab was administered every alternate month between the bimonthly aflibercept injections in 24 (33.3%) eyes showing worsening (
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Savastano, Maria Cristina, Claudia Fossataro, Matteo Mario Carlà, Chiara Fantozzi, Benedetto Falsini, Alfonso Savastano, Clara Rizzo, Raphael Kilian, and Stanislao Rizzo. "OCT angiography analysis of choriocapillaris vascular density in different stages of age-related macular degeneration." Frontiers in Ophthalmology 2 (September 22, 2022). http://dx.doi.org/10.3389/fopht.2022.985262.

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ObjectivesTo analyze the choriocapillaris vessel density (CVD) of eyes at different stages of Age-related Macular Degeneration (AMD) with Optical Coherence Tomography Angiography (OCTA).MethodsThis is a prospective observational cross-sectional study on 21 age-matched healthy eyes and 84 eyes with AMD (i.e., early AMD, late AMD, Geographic Atrophy [GA], and disciform scar AMD). OCTA was used to automatically measure the CVD (%), on both the whole macula and the foveal area, in a layer going from 9 µm above to 30 µm below the Bruch’s membrane. Furthermore, in the GA subgroup, the extension of t
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"Polypoidal Choroidal Vasculopathy and Treatments." Güncel Retina Dergisi (Current Retina Journal), July 1, 2017, 262–67. http://dx.doi.org/10.37783/crj-0043.

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Polypoidal choroidal vasculopathy (PCV), characterized by aneurysmal polypoidal lesions in the choroidal vasculature, is a subtype of exudative age-related macular degeneration (AMD) which can cause permanent vision loss due to hemorrhage, exudation, macular edema, and disciform scar formation. Besides anti-vascular endothelial growth factor (anti-VEGF) therapy, photodynamic therapy (FDT) and combination therapies are currently being used in the treatment of PCV.
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"Classification and Pathogenesis in Dry-Form (Non-Neovascular) Age-Related Macular Degeneration." Güncel Retina Dergisi (Current Retina Journal), April 1, 2017, 105–11. http://dx.doi.org/10.37783/crj-0018.

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Age-related macular degeneration is basically evaluated as non-neovascular (dry) and neovascular (wet) type. Initial stages start with atrophy, drusen development in RPE-Bruch membrane level and pigmentary changes and in final stages may result in CNVM, disciform scar, and geographic atrophic changes. Visual acuity loss is more severe and faster in neovascular ARMD, late stages of geographic atrophy involving foveal center may also cause significant visual loss.
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"Choroidal Neovascularization in Retinal Telangiectasia." Güncel Retina Dergisi (Current Retina Journal), October 1, 2017, 296–99. http://dx.doi.org/10.37783/crj-0049.

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Retinal telangiectasia is a neuro-degenerative macular disease that is limited to juxtafoveal region in one or both eyes with capillary telangiectasia and focal retinal gliosis. Choroidal neovascularization (CNV) is one of the main complications that cause visual deterioration in retinal telangiectasia. When patients develop CNV, rapid visual deterioration with subretinal hemorrhage, cystoid macular edema, hard exudates, disciform scar, and retinocoroidal anastomosis may occur. In our review article we discussed CNV in retinal telangiectasia and current treatment approaches.
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Kumawat, Devesh, Srikanta K. Padhy, and Vinod Kumar. "Clinical and Multimodal Imaging Features of Subretinal Drusenoid Deposits." Journal of Ophthalmic and Vision Research, April 29, 2021. http://dx.doi.org/10.18502/jovr.v16i2.9082.

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Purpose: To describe the multimodal imaging (MMI) features of subretinal drusenoid deposits (SDD) in Indian population.
 Methods: Patients diagnosed to have SDD from January 2016 to December 2018 at our tertiary care center were recruited. The diagnosis of SDD was made on the basis of MMI consisting of a combination of color fundus photography (CFP), optical coherence tomography (OCT), red-free (RF) imaging, blue autofluorescence (BAF), and near-infrared reflectance (NIR) imaging. The morphological type and distribution of SDD and the associated retinal lesions were reviewed.
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Dissertations / Theses on the topic "Disciform scar"

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Cherepanoff, Svetlana. "Age-related macular degeneration: histopathological and serum autoantibody studies." University of Sydney, 2008. http://hdl.handle.net/2123/2464.

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Doctor of Philosophy (PhD)<br>BACKGROUND: The accumulation of abnormal extracellular deposits beneath the retinal pigment epithelium characterises the pathology of early age-related macular degeneration. However, the histopathological threshold at which age-related changes become early AMD is not defined, and the effect of each of the deposits (basal laminar deposit and membranous debris) on disease progression is poorly understood. Evidence suggests that macrophages play a key role in the development of AMD lesions, but the influence of basal laminar deposit (BLamD) and membranous debris on t
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Cherepanoff, Svetlana. "Age-related macular degeneration: histopathological and serum autoantibody studies." Thesis, The University of Sydney, 2007. http://hdl.handle.net/2123/2464.

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BACKGROUND: The accumulation of abnormal extracellular deposits beneath the retinal pigment epithelium characterises the pathology of early age-related macular degeneration. However, the histopathological threshold at which age-related changes become early AMD is not defined, and the effect of each of the deposits (basal laminar deposit and membranous debris) on disease progression is poorly understood. Evidence suggests that macrophages play a key role in the development of AMD lesions, but the influence of basal laminar deposit (BLamD) and membranous debris on the recruitment and programming
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Book chapters on the topic "Disciform scar"

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Azem, Nur, and Michaella Goldstein. "Disciform Scar." In Encyclopedia of Ophthalmology, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-35951-4_1019-1.

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Azem, Nur, and Michaella Goldstein. "Disciform Scar." In Encyclopedia of Ophthalmology, 647–48. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_1019.

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Harrie, Roger P., and Cynthia J. Kendall. "Case Study 154 Subretinal Disciform Scar." In Clinical Ophthalmic Echography, 353–54. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7082-3_154.

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Alibhai, A. Yasin, and Daniela Ferrara. "Disciform Scar." In Atlas of Retinal OCT: Optical Coherence Tomography, 36. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-46121-4.00016-9.

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"Subretinal Disciform Scar." In Clinical Ophthalmic Echography, 359–60. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-75244-0_160.

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Adelman, Ron A., and Patricia Pahk. "Visual Field Defects in Chorioretinal Disorders." In Visual Fields. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780195389685.003.0012.

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Pathologic processes involving the retina or choroid can present with a wide variety of visual field defects. Usually visual field defects of retinal diseases directly correlate with the fundus findings. Visual field changes are often the result of damage to the retina or scarring but, in conjunction with other clinical findings, they may help narrow the differential diagnosis. Most of the macular lesions result in visual field defects that do not respect the vertical or horizontal midline. Occasionally inflammatory disorders result in visual field defects that do not directly correlate with the retinal findings. For example, patients with multiple evanescent white dot syndrome (MEWDS) may have an enlarged blind spot. Macular disorders can cause central or paracentral scotomas depending on the location of the lesion. Causes of macular pathology include drusen, atrophy from dry age-related macular degeneration (AMD), retinal hemorrhage, choroidal neovascular membrane, macular edema, macular hole, macular scar, pathologic myopia, and macular dystrophies of the retina or choroid. Central serous chorioretinopathy (CSCR) can show a relative defect that is anatomically correlated with the area of subretinal or sub RPE (retinal pigment epithelium) fluid accumulation. Residual pigmentary changes in inactive CSCR can also cause a relative depression in the corresponding visual field. Pathologic myopia can present with a variety of visual field defects depending on the retinal findings, such as posterior staphyloma or choroidal neovascular membrane. AMD may show nonspecific changes in the central or paracentral visual field that correlate with the structural damage to the retina and choroid. Geographic atrophy in dry AMD can cause a dense scotoma correlated with the pattern of the atrophy. Choroidal neovascular membranes can cause a depression in the correlating visual field due to edema or hemorrhage. Disciform scars in endstage AMD can also cause a dense scotoma. Macular holes may cause a small central scotoma. Pattern dystrophies are a family of disorders with a common pathology at the level of the RPE. Butterfly dystrophy, an autosomal dominant disorder, and Sjögren reticular dystrophy, an autosomal recessive disorder, are two examples of pattern dystrophies.
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