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Books on the topic 'Disorders cerebral circulation'

Author: Grafiati
Published: 28 May 2022
Last updated: 30 July 2025

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1

International Symposium of Applied Physiology of the Peripheral Circulation (5th 2000 Pittsburgh, Pa.). Cerebral blood flow: Mechanisms of ischemia, diagnosis and therapy. Springer, 2002.

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2

Mitagvariia, N. P. Cerebral blood flow regulation. Nova Science Publishers, 2009.

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3

1943-, Hartmann A., and Hoyer S. 1933-, eds. Cerebral blood flow and metabolism measurement. Springer-Verlag, 1985.

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4

Edvinsson, Lars. Cerebral blood flow and metabolism. Raven Press, 1993.

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5

1924-, Wüllenweber R., Klinger M. 1943-, and Brock M. 1938-, eds. Regulation of cerebral blood flow and metabolism ; Neurosurgical treatment of epilepsy ; Rehabilitation in neurosurgery. Springer-Verlag, 1987.

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6

A, Unterberg, Schneider G. H. 1965-, and Lanksch W. 1938-, eds. Monitoring of cerebral blood flow and metabolism in intensive care. Springer-Verlag, 1993.

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7

Howard, Yonas, and International Conference on Xe/CT CBF (1st : 1990 : Orlando, Fla.), eds. Cerebral blood flow measurement with stable xenon-enhanced computed tomography. Raven Press, 1992.

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8

Jesse, Weinberger, ed. Noninvasive imaging of cerebrovascular disease. A.R. Liss, 1989.

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9

M, Valdueza José, ed. Neurosonology and neuroimaging of stroke: A teaching atlas. Thieme, 2008.

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10

Auer, L. M. The Cerebral Veins. Springer, 2013.

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11

Krause, Diana N., and Lars Edvinsson. Cerebral Blood Flow and Metabolism (Periodicals). 2nd ed. Lippincott Williams & Wilkins, 2001.

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12

Blood-Brain Barrier in Health and Disease: Morphology, Biology and Immune Function. Taylor & Francis Group, 2015.

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13

Blood-Brain Barrier in Health and Disease: Pathophysiology and Pathology. Taylor & Francis Group, 2015.

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14

Wullenweber and Klinger. Regulation of Cerebral Blood Flow and Metabolism/Neurosurgical Treatment of Epilepsy/Rehabilitation in Neurosurgery (Deutsche Gesellschaft Fur Neurochirurgie // Proceedings of the Annual Congress). Springer-Verlag, 1987.

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15

Xe, International Conference on, and Japan) Ct Cbf 1992 (Fukuoka-Shi. Quantitative Cerebral Blood Flow Measurements Using Stable Xenon/Ct: Clinical Applications. Blackwell/Futura, 1995.

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16

(Editor), G. H. Schneider, A. W. Unterberg (Editor), and Wolfgang R. Lanksch (Editor), eds. Monitoring of Cerebral Blood Flow and Metabolism in Intensive Care: Proceedings of an International Symposium, Berlin, October 1992 (Acta Neurochirurgica Supplementum). Springer-Verlag Telos, 1994.

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17

Chadwick, David, Alastair Compston, Michael Donaghy, et al. Investigations. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0100.

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This chapter describes the many methods that can be used to investigate neurological disorders. The application and suitability for specific disorder types are outlined, as are contraindications for use. Methods of imaging the central nervous system include computed tomography (CT) imaging, several magnetic resonance (MR) scanning methods, Single photon emission computed tomography (SPECT) and Positron Emission Tomography (PET). Invasive (angiography) and non-invasive methods of imaging the cerebral circulation are also outlined.The standard method of recording electrical activity of the brain
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18

Borden, Neil M. 3D Angiographic Atlas of Neurovascular Anatomy and Pathology. Cambridge University Press, 2006.

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19

3D Angiographic Atlas of Neurovascular Anatomy and Pathology. Cambridge University Press, 2007.

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20

Borden, Neil M. 3D Angiographic Atlas of Neurovascular Anatomy and Pathology. Cambridge University Press, 2006.

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21

Borden, Neil M. 3D Angiographic Atlas of Neurovascular Anatomy and Pathology. Cambridge University Press, 2006.

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22

Borden, Neil M. 3D Angiographic Atlas of Neurovascular Anatomy and Pathology. Cambridge University Press, 2012.

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23

Sprigings, David. Coma. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0040.

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Coma is a pathological state of unconsciousness from which a patient cannot be roused to wakefulness by stimuli, and reflects dysfunction of the brainstem reticular system and its thalamic projections (the neuronal basis of wakefulness), or diffuse injury of both cerebral hemispheres. A unilateral lesion of a cerebral hemisphere (e.g. haemorrhagic stroke) will not cause coma unless there is secondary compression of the contralateral hemisphere or brainstem. Coma is a medical emergency, because a comatose patient is at high risk of permanent brain injury or death, caused either by the underlyin
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