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Journal articles on the topic 'Disseny lo gic'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 July 2025

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1

Krol, Ambassador George, and Anoushka Ramesh. "Russia, China, and Protests: Caught Between Two Powers, Kazakhstan Navigates Internal Dissent." Georgetown Journal of International Affairs 23, no. 1 (2022): 65–71. http://dx.doi.org/10.1353/gia.2022.0011.

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2

Nagle, John. "LGBTQ+ Activism Under Attack: The Legitimation Crisis of Lebanon's Power-Sharing." Georgetown Journal of International Affairs 25, no. 1 (2024): 200–207. http://dx.doi.org/10.1353/gia.2024.a934904.

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Abstract: This article examines the current wave of harassment of Lebanese LGBTQ+ activism in the context of the power-sharing government undergoing a "legitimation crisis." It argues that as Lebanon's power-sharing system has increasingly become more semi-authoritarian, state and non-state actors have responded to popular dissent by framing LGBTQ+ activism as a security threat. The article concludes by questioning the role of international actors as supporters of Lebanese LGBTQ+ rights.

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Busnelli, Marta, Erika Peverelli, Giovanna Mantovani, Anna Spada та Bice Chini. "Deciphering the specific role of Gαi/o isoforms: functional selective oxytocin ligands and somatostatin SST5 receptor mutants". Biochemical Society Transactions 41, № 1 (2013): 166–71. http://dx.doi.org/10.1042/bst20120306.

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Receptor coupling to different G-proteins and β-arrestins has been described for a number of GPCRs (G-protein-coupled receptors), suggesting a multi-state model of receptor activation in which each receptor can assume a number of different active conformations, each capable of promoting the coupling to a specific effector. Consistently, functional-selective ligands and biased agonists have been described to be able to induce and/or stabilize only a subset of specific active conformations. Furthermore, GPCR mutants deficient in selective coupling have been reported. Functional selective ligands

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4

Nettles-Barcelón, Kimberly D., Gillian Clark, Courtney Thorsson, Jessica Kenyatta Walker, and Psyche Williams-Forson. "Black Women’s Food Work as Critical Space." Gastronomica 15, no. 4 (2015): 34–49. http://dx.doi.org/10.1525/gfc.2015.15.4.34.

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Black American women have long sustained a complex relationship to food—its production, consumption, and distribution within families, communities, and the nation. Black women, often represented in American culture as “natural” good cooks on the one hand and beset by obesity on the other, straddle an uncomfortable divide that is at the heart of contemporary debate about the nature of our food system. Yet, Black women as authorities in the kitchen and elsewhere in matters of food—culturally, politically, and socially—are largely absent, made invisible by the continued salience of intersecting v

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Al-Jizawi, Noura, Siena Anstis, and Gözde Böcü. "Targeted Across Borders: Digital Transnational Repression, Gender Dimensions, and the Role of Host States." Georgetown Journal of International Affairs 26, no. 1 (2025): 109–16. https://doi.org/10.1353/gia.2025.a965783.

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Abstract: Transnational repression (TNR)—the practice through which authoritarian regimes target critical voices in exile—has been emboldened and expanded by digital technologies. Despite having fled their home states, human rights defenders, activists, and dissidents abroad are not immune to the repressive practices of authoritarian regimes that use digital technologies to intimidate and repress dissent. The impacts of both digital and physical forms of TNR are far-reaching, with detrimental effects on individuals' social, psychological, and physical well-being. This article examines the prac

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Fang, Kuan-Te, Hsin Hung, Nga Yin Sadonna Lau, Jou-Hsi Chi, Deng-Chyang Wu, and Kuang-Hung Cheng. "Development of a Genetically Engineered Mouse Model Recapitulating LKB1 and PTEN Deficiency in Gastric Cancer Pathogenesis." Cancers 15, no. 24 (2023): 5893. http://dx.doi.org/10.3390/cancers15245893.

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The LKB1 and PTEN genes are critical in gastric cancer (G.C.) development. LKB1, a robust tumor suppressor gene, encodes a serine/threonine kinase that directly triggers the activation of AMPK—an integral cellular metabolic kinase. The role of the LKB1 pathway extends to maintaining the stability of epithelial junctions by regulating E-cadherin expression. Conversely, PTEN, a frequently mutated tumor suppressor gene in various human cancers, emerges as a pivotal negative regulator of the phosphoinositide 3-kinase (PI3K) signaling pathway. This study is set to leverage the H+/K+ ATPase Cre tran

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7

Ajani, Jaffer A., Yan Xu, Longfei Huo, et al. "YAP1 mediates gastric adenocarcinoma peritoneal metastases that are attenuated by YAP1 inhibition." Gut 70, no. 1 (2020): 55–66. http://dx.doi.org/10.1136/gutjnl-2019-319748.

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ObjectivePeritoneal carcinomatosis (PC; malignant ascites or implants) occurs in approximately 45% of advanced gastric adenocarcinoma (GAC) patients and associated with a poor survival. The molecular events leading to PC are unknown. The yes-associated protein 1 (YAP1) oncogene has emerged in many tumour types, but its clinical significance in PC is unclear. Here, we investigated the role of YAP1 in PC and its potential as a therapeutic target.MethodsPatient-derived PC cells, patient-derived xenograft (PDX) and patient-derived orthotopic (PDO) models were used to study the function of YAP1 in

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8

Wang, Ruiping, Shumei Song, Kazuto Harada, et al. "Multiplex profiling of peritoneal metastases from gastric adenocarcinoma identified novel targets and molecular subtypes that predict treatment response." Gut 69, no. 1 (2019): 18–31. http://dx.doi.org/10.1136/gutjnl-2018-318070.

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ObjectivePeritoneal carcinomatosis (PC) occurs frequently in patients with gastric adenocarcinoma (GAC) and confers a poor prognosis. Multiplex profiling of primary GACs has been insightful but the underpinnings of PC’s development/progression remain largely unknown. We characterised exome/transcriptome/immune landscapes of PC cells from patients with GAC aiming to identify novel therapeutic targets.DesignWe performed whole-exome sequencing (WES) and whole transcriptome sequencing (RNA-seq) on 44 PC specimens (43 patients with PC) including an integrative analysis of WES, RNA-seq, immune profi

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9

Masresha, Bezanesh Melese, Kasim Mohammed Yesuf, Yikeber Abebaw Moyehodie, Hailegebrael Birhan Biresaw, Solomon Sisay Mulugeta, and Gedam Derbew Addisia. "Determinant factors of leprosy-related disability; comparison of acceleration failure time and parametric shared frailty models." PLOS ONE 18, no. 4 (2023): e0271883. http://dx.doi.org/10.1371/journal.pone.0271883.

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Background Leprosy is an illness persisting for a long time or constantly recurring brought about by Mycobacterium leprae. The collusion of the causing agent with Schwann cells leads to incapable of being changed loss of fringe nerve tissue; followed by incapacity and that is not restricted to actual powerlessness yet additionally makes a negative picture, prompting segregation and social disgrace against the altered people also, their families. Methods The analysis of this study comprises 205 samples of patients at All African TB and Leprosy Rehabilitation and Training Centre from January 201

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10

Covey, Todd M., Michael Gulrajani, Heiko Becker, et al. "Single Cell Network Profiling as a Platform to Reveal Leukemia-Specific Signaling Signatures and Sensitivity to Kinase Inhibitor Therapies." Blood 116, no. 21 (2010): 2753. http://dx.doi.org/10.1182/blood.v116.21.2753.2753.

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Abstract Abstract 2753 Aberration in kinase activity by either the gain-of-function mutations or overexpression of the encoding genes plays a pivotal role in myeloid leukemogenesis. An increasing number of kinase inhibitors are being developed as “targeted therapies” for the treatment of acute myeloid leukemia (AML) and other myeloproliferative disorders. However, given the biologic and clinical heterogeneity inherent to these diseases, an unmet medical need exists for tools to guide the choice of inhibitor(s) most relevant for individual patients. With the aim of developing a platform for the

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11

Fan, Yibo, Yuan Li, Xiaodan Yao, et al. "Epithelial SOX9 drives progression and metastases of gastric adenocarcinoma by promoting immunosuppressive tumour microenvironment." Gut, August 24, 2022, gutjnl—2021–326581. http://dx.doi.org/10.1136/gutjnl-2021-326581.

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ObjectiveMany cancers engage embryonic genes for rapid growth and evading the immune system. SOX9 has been upregulated in many tumours, yet the role of SOX9 in mediating immunosuppressive tumour microenvironment is unclear. Here, we aim to dissect the role of SOX9-mediated cancer stemness attributes and immunosuppressive microenvironment in advanced gastric adenocarcinoma (GAC) for novel therapeutic discoveries.MethodsBulk RNAseq/scRNA-seq, patient-derived cells/models and extensive functional studies were used to identify the expression and functions of SOX9 and its target genes in vitro and

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12

Läger, Frederic, Yassin Denis Bouzzine, and Rainer Lueg. "Carbon performance and corporate financial performance during crises: Evidence from the COVID‐19 pandemic and the Global Financial Crisis." Journal of Industrial Ecology, December 29, 2024. https://doi.org/10.1111/jiec.13603.

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AbstractEconomic crises offer a valuable perspective on the relationship between carbon performance and corporate financial performance (CFP). Crises test corporate performance and challenge the allegedly universal synergies that developed under “normal” circumstances. However, the current research in this area is limited and has often yielded insignificant results. Drawing from a global dataset spanning 15 years, we investigate the moderating influences of two distinct crises on the interplay between carbon emission intensity and CFP. Employing fixed‐effects regression analysis, we dissect th

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13

Kayani, Umar, Fakhrul Hasan, Tonmoy Choudhury, and Farrukh Nawaz. "Unveiling WCM and firm performance relationship: evidence from Shariah compliance United Kingdom firms." International Journal of Islamic and Middle Eastern Finance and Management, January 6, 2025. https://doi.org/10.1108/imefm-04-2024-0168.

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Purpose The purpose of this study is to investigate the realtionship between the strategic maneuvers in working capital management (WCM) and the ensuing corporate performance, chiefly within the purview of companies aligned with Shariah-compliant financial practices during epochs of fiscal distress. Design/methodology/approach This study deploys a fixed effect regression model to dissect the WCM-performance nexus for Shariah and non-Shariah compliance firms in the UK. Here, the authors used FTSE 350 index data from 1990 till 2022. The authors used return on assets, return on equity and net pro

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14

Cao, Yuncan, Wenjing Shu, Peng Jin, et al. "NAD metabolism-related genes provide prognostic value and potential therapeutic insights for acute myeloid leukemia." Frontiers in Immunology 15 (June 20, 2024). http://dx.doi.org/10.3389/fimmu.2024.1417398.

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IntroductionAcute myeloid leukemia (AML) is an aggressive blood cancer with high heterogeneity and poor prognosis. Although the metabolic reprogramming of nicotinamide adenine dinucleotide (NAD) has been reported to play a pivotal role in the pathogenesis of acute myeloid leukemia (AML), the prognostic value of NAD metabolism and its correlation with the immune microenvironment in AML remains unclear.MethodsWe utilized our large-scale RNA-seq data on 655 patients with AML and the NAD metabolism-related genes to establish a prognostic NAD metabolism score based on the sparse regression analysis

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15

Whiting, Sam, Tully Barnett, and Justin O'Connor. "‘Creative City’ R.I.P.?" M/C Journal 25, no. 3 (2022). http://dx.doi.org/10.5204/mcj.2901.

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The Creative City Unlike the terms ‘creative industries’, which nobody ever quite understood, and ‘creative class’, about which actual ‘creatives’ were always ambiguous, the ‘creative city’ has been an incredibly successful global policy meme, to which cities across the world continue to aspire. From the early 1990s, faced with de-industrialisation, rising unemployment, and the increased global mobility of capital, professionals, and consumer-tourists, the ‘creative city’ became an essential part of the new urban imaginary for politicians, planners, local growth coalitions, and advocates and p

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