Relevant bibliographies by topics / Diversion colitis

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Diversion colitis'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 February 2022

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Journal articles on the topic "Diversion colitis"

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Tang, S.-J., and F. Bhaijee. "Diversion Colitis." Video Journal and Encyclopedia of GI Endoscopy 1, no. 2 (2013): 318–19. http://dx.doi.org/10.1016/s2212-0971(13)70138-9.

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Eggenberger, John C., and Asim Farid. "Diversion colitis." Current Treatment Options in Gastroenterology 4, no. 3 (2001): 255–59. http://dx.doi.org/10.1007/s11938-001-0037-z.

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Whelan, R. L., D. Abramson, D. S. Kim, and H. F. Hashmi. "Diversion colitis." Surgical Endoscopy 8, no. 1 (1994): 19–24. http://dx.doi.org/10.1007/bf02909487.

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Murray, Frank E., Michael J. O'Brien, Desmond H. Birkett, Susan M. Kennedy, and J. Thomas Lamont. "Diversion colitis." Gastroenterology 93, no. 6 (1987): 1404–8. http://dx.doi.org/10.1016/0016-5085(87)90272-1.

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Chetty, R., S. Hafezi, and E. Montgomery. "An incidental enterocolic lymphocytic phlebitis pattern is seen commonly in the rectal stump of patients with diversion colitis superimposed on inflammatory bowel disease." Journal of Clinical Pathology 62, no. 5 (2009): 464–67. http://dx.doi.org/10.1136/jcp.2008.063917.

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Aims:Enterocolic lymphocytic phlebitis (ELP) is an uncommon cause of bowel pathology and most frequently results in ischaemia. It is characterised by an artery-sparing, venulocentric lymphoid infiltrate that causes a phlebitis and vascular compromise. Rare cases of ELP have been encountered with lymphocytic colitis in the absence of ischaemic bowel change. The present study examined the occurrence of ELP in the setting of diversion colitis and inflammatory bowel disease, as well as in random colectomy specimens.Methods:The study cohort comprised the following: 26 completion proctectomy specime
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Thorsen, Amy. "Noninfectious Colitides: Collagenous Colitis, Lymphocytic Colitis, Diversion Colitis, and Chemically Induced Colitis." Clinics in Colon and Rectal Surgery 20, no. 1 (2007): 047–57. http://dx.doi.org/10.1055/s-2007-970200.

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JOWETT, S. L. "Diversion colitis as a trigger for ulcerative colitis." Gut 46, no. 2 (2000): 293b—293. http://dx.doi.org/10.1136/gut.46.2.293b.

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Komorowski, Richard A. "Histologic Spectrum of Diversion Colitis." American Journal of Surgical Pathology 14, no. 6 (1990): 548–54. http://dx.doi.org/10.1097/00000478-199006000-00005.

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Williams, G. T., and J. Rhodes. "Collagenous colitis: disease or diversion?" BMJ 294, no. 6576 (1987): 855–56. http://dx.doi.org/10.1136/bmj.294.6576.855.

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GERAGHTY, J. M., and A. K. CHARLES. "Aphthoid ulceration in diversion colitis." Histopathology 24, no. 4 (1994): 395–97. http://dx.doi.org/10.1111/j.1365-2559.1994.tb00545.x.

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Dissertations / Theses on the topic "Diversion colitis"

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Oliveira, Ariano Jose Freitas de. "Emprego dos ?cidos graxos de cadeia curta na colite de deriva??o fecal : estudo em ratos Wistar." Universidade Federal do Rio Grande do Norte, 2011. http://repositorio.ufrn.br:8080/jspui/handle/123456789/13233.

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Made available in DSpace on 2014-12-17T14:13:40Z (GMT). No. of bitstreams: 1 ArianoJFO_TESE.pdf: 4702952 bytes, checksum: 4c768ba0623fffd6e9b8beb9419b70d5 (MD5) Previous issue date: 2011-09-05<br>Diversion colitis is a chronic inflammatory process affecting the dysfunctional colon, after a colostomy. It is postulated that nutritional deficiency of the colonic epithelium by the absence of short-chain fatty acids (SCFA) is one of the factors responsible for the appearance of DC and that their employment could reverse the morphological changes of the mucosa. The treatment of choice for fecal di
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SEPEHRI, SHADI. "Microbial etiology of Inflammatory Bowel Disease: Microbial diversity and the role of Escherichia coli." 2010. http://hdl.handle.net/1993/3977.

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Inflammatory bowel disease (IBD), comprises Crohn’s disease (CD) and ulcerative colitis (UC), and is a chronic relapsing inflammation of gastrointestinal tract without any known cause or cure. Currently, it is accepted that IBD is a result of a dysfunctional immune response to commensal bacteria in a genetically susceptible host, and that environmental factors can trigger the onset or reactivation of the disease. This thesis considers the possibility of a specific pathogenic agent as well as an imbalance in the composition of the normal microflora in the pathogenesis of IBD. Gut biopsy tissues
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Books on the topic "Diversion colitis"

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Sykes, Nigel P. Constipation and diarrhoea. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0203.

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Constipation is far more common in palliative care than diarrhoea and results not just from the use of opioids but also from the multifactorial effects of debility secondary to disease. Most palliative care patients will require regular administration of an oral laxative in a dose titrated against response, with the aim of avoiding the use of suppositories or enemas if possible as these are less liked. The lack of clear differences in laxative efficacy means that cost and patient choice are key factors in guiding treatment. Diarrhoea in palliative care most often results from excess laxative o
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Book chapters on the topic "Diversion colitis"

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Soergel, Konrad H. "Diversion colitis." In Inflammatory Bowel Disease: From Bench to Bedside. Springer Netherlands, 2003. http://dx.doi.org/10.1007/978-1-4020-3477-0_45.

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De Hertogh, Gert. "Diversion Colitis." In Encyclopedia of Pathology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-40560-5_1436.

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Alexander-Williams, J. "Bypass, Diversion and Dilatation." In Crohn’s Disease and Ulcerative Colitis. Springer London, 1993. http://dx.doi.org/10.1007/978-1-4471-3296-7_11.

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"Diversion Colitis." In Diagnostic Pathology: Gastrointestinal. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-37673-0.50113-0.

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"Diversion Colitis and Pouchitis." In Diseases of the Colon. CRC Press, 2016. http://dx.doi.org/10.3109/9781420018578-32.

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Shen, Jing-Jing, and Baochuan Guo. "Novel Approaches for Pouchitis and Colitis With or Without Diversion." In Pouchitis and Ileal Pouch Disorders. Elsevier, 2019. http://dx.doi.org/10.1016/b978-0-12-809402-0.00043-5.

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Sanderson, Jeremy, and Peter Irving. "Ulcerative colitis." In Oxford Textbook of Medicine, edited by Jack Satsangi. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0307.

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Ulcerative colitis is a chronic relapsing and remitting disease in which chronic inflammation affects the rectum and extends proximally to a variable extent. The precise aetiology remains unknown but involves an interplay between reduced diversity in the gut microbiota and a genetically dysregulated gut immune system and epithelial barrier. Typical presentation of mild or moderate disease is with a gradual onset of symptoms including diarrhoea, rectal bleeding, and the passage of mucus. Severe disease is characterized by anorexia, nausea, weight loss, and severe diarrhoea, with the patient likely to look unwell with fever, tachycardia, and other signs of volume depletion, and the abdomen may be distended and tympanitic, with reduced bowel sounds and marked colonic tenderness. Diagnosis is usually made on the basis of exclusion of infective colitis by stool culture and the finding of typical diffuse inflammation in the rectum and above at sigmoidoscopy. Management requires rapid control of symptoms with induction therapy followed by maintenance of remission. Mild disease is typically treated with 5-aminosalicyclic acid delivered both orally and by enema, and moderate disease by 5-aminosalicyclic acid and steroids. Patients with severe disease require hospital admission, intravenous steroids, and daily review by both a physician and a surgeon experienced in the management of ulcerative colitis. Ciclosporin or infliximab are used as rescue therapies for steroid-resistant acute severe ulcerative colitis, but colectomy should not be delayed when this is required. Maintenance therapy with immunomodulators and biological therapies are both effective at maintaining remission, and several new biologicals are in clinical trials.
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Priyamvada, Priyamvada. "Dysbiosis in Microbiome Leading to Colitis-Associated Cancer." In Diagnostic and Treatment Methods for Ulcerative Colitis and Colitis-Associated Cancer. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-3580-6.ch007.

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Colitis-associated cancers are a metastatic form of inflammatory bowel disease considered a vital health associated risk factor causing the death of approximately five lacs people every year throughout the world. There are trillions of bacteria that are associated with our gut as a part of our healthy microbiome. The microbiota plays a plethora of important role in determining the normal physiological processes of the cells and, subsequently, the body. The imbalance in microbiome diversity (dysbiosis) due to abnormal dietary habitats, hectic lifestyle, and other factors thus alters the normal physiological processes of the body, thereby causing several chronic diseases. Therefore, it is essential to maintain the homeostasis between the host and their gut microbiome. So, based on the facts mentioned above, this chapter is entirely devoted to providing an overview of colitis-associated cancer and their relation with the dysbiosis of a healthy microbiome. Moreover, the mechanism involved in the development of colorectal cancer and its preventive insights has also been addressed.
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Rogler, Gerhard. "Inflammatory bowel disease: basic mechanisms." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0274.

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Inflammatory bowel disease (IBD) affects 1 in 200 people in Western countries with the two main forms being Crohn’s disease and ulcerative colitis. Clinically, IBD is characterized by chronic diarrhoea, bloody bowel movements, abdominal pain, weight loss, fatigue, fever, and extraintestinal manifestations such as arthralgia or skin affections. In rare cases, pericarditis or myocardial involvement is found. Genome-wide association studies have identified more than 250 genetic susceptibility factors. Additionally, so-far unidentified environmental factors play an important role in IBD pathophysiology. They may directly act on the intestinal mucosa or mediate their effects via the intestinal microbiota which is altered in its diversity. In patients with IBD, a ‘leaky’ intestinal barrier and impaired intestinal defence leads to increased bacterial invasion into the epithelial mucus layer and to bacterial translocation into the bloodstream. Bacterial wall products such as lipopolysaccharides are found in higher concentrations in IBD patients.
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Conference papers on the topic "Diversion colitis"

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Pacheco, Rodrigo, Christiano Esposito, Alberto Schanaider, et al. "Comparative study between ultrasound biomicroscopy and histopathology of diversion colitis on rats." In 2008 IEEE Ultrasonics Symposium (IUS). IEEE, 2008. http://dx.doi.org/10.1109/ultsym.2008.0421.

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