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Journal articles on the topic 'Diversion colitis'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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1

Tang, S.-J., and F. Bhaijee. "Diversion Colitis." Video Journal and Encyclopedia of GI Endoscopy 1, no. 2 (2013): 318–19. http://dx.doi.org/10.1016/s2212-0971(13)70138-9.

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2

Eggenberger, John C., and Asim Farid. "Diversion colitis." Current Treatment Options in Gastroenterology 4, no. 3 (2001): 255–59. http://dx.doi.org/10.1007/s11938-001-0037-z.

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3

Whelan, R. L., D. Abramson, D. S. Kim, and H. F. Hashmi. "Diversion colitis." Surgical Endoscopy 8, no. 1 (1994): 19–24. http://dx.doi.org/10.1007/bf02909487.

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4

Murray, Frank E., Michael J. O'Brien, Desmond H. Birkett, Susan M. Kennedy, and J. Thomas Lamont. "Diversion colitis." Gastroenterology 93, no. 6 (1987): 1404–8. http://dx.doi.org/10.1016/0016-5085(87)90272-1.

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5

Chetty, R., S. Hafezi, and E. Montgomery. "An incidental enterocolic lymphocytic phlebitis pattern is seen commonly in the rectal stump of patients with diversion colitis superimposed on inflammatory bowel disease." Journal of Clinical Pathology 62, no. 5 (2009): 464–67. http://dx.doi.org/10.1136/jcp.2008.063917.

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Aims:Enterocolic lymphocytic phlebitis (ELP) is an uncommon cause of bowel pathology and most frequently results in ischaemia. It is characterised by an artery-sparing, venulocentric lymphoid infiltrate that causes a phlebitis and vascular compromise. Rare cases of ELP have been encountered with lymphocytic colitis in the absence of ischaemic bowel change. The present study examined the occurrence of ELP in the setting of diversion colitis and inflammatory bowel disease, as well as in random colectomy specimens.Methods:The study cohort comprised the following: 26 completion proctectomy specime

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6

Thorsen, Amy. "Noninfectious Colitides: Collagenous Colitis, Lymphocytic Colitis, Diversion Colitis, and Chemically Induced Colitis." Clinics in Colon and Rectal Surgery 20, no. 1 (2007): 047–57. http://dx.doi.org/10.1055/s-2007-970200.

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7

JOWETT, S. L. "Diversion colitis as a trigger for ulcerative colitis." Gut 46, no. 2 (2000): 293b—293. http://dx.doi.org/10.1136/gut.46.2.293b.

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8

Komorowski, Richard A. "Histologic Spectrum of Diversion Colitis." American Journal of Surgical Pathology 14, no. 6 (1990): 548–54. http://dx.doi.org/10.1097/00000478-199006000-00005.

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9

Williams, G. T., and J. Rhodes. "Collagenous colitis: disease or diversion?" BMJ 294, no. 6576 (1987): 855–56. http://dx.doi.org/10.1136/bmj.294.6576.855.

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10

GERAGHTY, J. M., and A. K. CHARLES. "Aphthoid ulceration in diversion colitis." Histopathology 24, no. 4 (1994): 395–97. http://dx.doi.org/10.1111/j.1365-2559.1994.tb00545.x.

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11

&NA;. "An update on diversion colitis." Advances in Anatomic Pathology 7, no. 3 (2000): 192–93. http://dx.doi.org/10.1097/00125480-200007030-00012.

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12

Abbasakoor, F., C. Mahon, and P. B. Boulos. "Diversion colitis in sigmoid neovagina." Colorectal Disease 6, no. 4 (2004): 290–91. http://dx.doi.org/10.1111/j.1463-1318.2004.00653.x.

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13

Rhatigan, Ronald M. "Colonic mucosa in diversion colitis." Human Pathology 24, no. 10 (1993): 1150. http://dx.doi.org/10.1016/0046-8177(93)90202-r.

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14

Matsumoto, Satohiro, and Hirosato Mashima. "Efficacy of Combined Mesalazine Plus Corticosteroid Enemas for Diversion Colitis after Subtotal Colectomy for Ulcerative Colitis." Case Reports in Gastroenterology 10, no. 1 (2016): 162–65. http://dx.doi.org/10.1159/000445868.

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Diversion colitis is a benign inflammatory process that occurs in any part of the large bowel excluded from the fecal stream by a diverting colostomy. While most of the patients with diversion colitis usually are asymptomatic, a minority has abdominal pain and rectal discharge of blood or mucus. A 65-year-old Japanese man was diagnosed as having diversion colitis with ulcerative colitis at 4 months after subtotal colectomy. Corticosteroid and mesalazine enemas were started nonsynchronously. A proctoscopy after 2 months showed no response. Prednisolone injections were started at 1.0 mg/kg daily

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15

Lim, A. G., F. L. Langmead, R. M. Feakins, and D. S. Rampton. "Diversion colitis: a trigger for ulcerative colitis in the in-stream colon?" Gut 44, no. 2 (1999): 279–82. http://dx.doi.org/10.1136/gut.44.2.279.

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The aetiology of ulcerative colitis is unknown. Two patients without pre-existing inflammatory bowel disease in whom end colostomy for faecal incontinence was complicated by diversion colitis in the defunctioned rectosigmoid colon, are described. In both instances, colitis with the clinical, colonoscopic, and microscopic features of ulcerative colitis developed about a year later in the previously normal in-stream colon proximal to the colostomy. These cases suggest that diversion colitis may be a risk factor for ulcerative colitis in predisposed individuals and that ulcerative colitis can be

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16

Yamaguchi, Keizo, Masahiko Tanigawa, Goichi Nakayama, et al. "A Case of Diversion Colitis Successfully Treated with Prednisolone Enema." Nippon Daicho Komonbyo Gakkai Zasshi 68, no. 8 (2015): 552–57. http://dx.doi.org/10.3862/jcoloproctology.68.552.

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17

Agarwal, Vijay P., and Elihu M. Schimmel. "Diversion Colitis: A Nutritional Deficiency Syndrome?" Nutrition Reviews 47, no. 9 (2009): 257–61. http://dx.doi.org/10.1111/j.1753-4887.1989.tb02857.x.

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18

&NA;. "Diversion Colitis—20 Years A-Growing." Journal of Clinical Gastroenterology 15, no. 4 (1992): 281–83. http://dx.doi.org/10.1097/00004836-199212000-00003.

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19

Canning, Douglas A. "Diversion Colitis in Children With Colovaginoplasty." Journal of Urology 168, no. 1 (2002): 389. http://dx.doi.org/10.1016/s0022-5347(05)64952-x.

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20

Rice, A. J., C. R. Abbott, and N. M. Mapstone. "Granulomatous vasculitis in diversion procto‐colitis." Histopathology 34, no. 3 (1999): 276–77. http://dx.doi.org/10.1111/j.1365-2559.1999.tb00055.x.

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21

Syed, H. A., P. S. J. Malone, and R. J. Hitchcock. "Diversion colitis in children with colovaginoplasty." BJU International 87, no. 9 (2001): 857–60. http://dx.doi.org/10.1046/j.1464-410x.2001.02180.x.

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22

Järnerot, Gunnar, Curt Tysk, Johan Bohr, and Sune Eriksson. "Collagenous colitis and fecal stream diversion." Gastroenterology 109, no. 2 (1995): 449–55. http://dx.doi.org/10.1016/0016-5085(95)90332-1.

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23

Keli, Elie, Michel Bouchoucha, Ghislain Devroede, Françoise Carnot, Thierry Ohrant, and Paul-Henri Cugnenc. "Diversion-related experimental colitis in rats." Diseases of the Colon & Rectum 40, no. 2 (1997): 222–28. http://dx.doi.org/10.1007/bf02054992.

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24

Castro, Leonaldson dos Santos, Alberto Schanaider, and Bettina Wolff Castro. "Colitis following fecal diversion: still a challenge." Acta Cirurgica Brasileira 15, no. 4 (2000): 195–200. http://dx.doi.org/10.1590/s0102-86502000000400002.

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After fecal diversion, nonspecific colitis may be seen in the defunctionalized colon. The purpose of this prospective study is to identify specific findings that could help in the differential diagnosis between diversion colitis and other inflammatory bowel diseases in order to avoid inappropriate diagnosis and therapy. It was studied, prospectively, thirteen consecutive patients from two public hospitals of Rio de Janeiro who had undergone temporary colostomy for indications other than inflammatory bowel disease. They were submitted to endoscopy with biopsy of both proximal and distal colorec

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25

Ishikawa, Akihisa, Kazuhiro Enomoto, Yasushi Akita, et al. "A Case of Crohn's Disease with Diversion Colitis." Progress of Digestive Endoscopy(1972) 48 (1996): 206–7. http://dx.doi.org/10.11641/pdensks.48.0_206.

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26

Kiernan, Miranda G., J. Calvin Coffey, Shaheel M. Sahebally, et al. "Systemic Molecular Mediators of Inflammation Differentiate Between Crohn’s Disease and Ulcerative Colitis, Implicating Threshold Levels of IL-10 and Relative Ratios of Pro-inflammatory Cytokines in Therapy." Journal of Crohn's and Colitis 14, no. 1 (2019): 118–29. http://dx.doi.org/10.1093/ecco-jcc/jjz117.

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Abstract Background and Aims Faecal diversion is associated with improvements in Crohn’s disease but not ulcerative colitis, indicating that differing mechanisms mediate the diseases. This study aimed to investigate levels of systemic mediators of inflammation, including fibrocytes and cytokines, [1] in patients with Crohn’s disease and ulcerative colitis preoperatively compared with healthy controls and [2] in patients with Crohn’s disease and ulcerative colitis prior to and following faecal diversion. Methods Blood samples were obtained from healthy individuals and patients with Crohn’s dise

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27

LIM, A. G. "Diversion colitis: a trigger for ulcerative colitis in the instream colon." Gut 46, no. 3 (2000): 440a—440. http://dx.doi.org/10.1136/gut.46.3.440a.

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28

Okamura, T., H. Minaguchi, K. Toriya, et al. "Diversion Colitis and Proctitis. A Case Report." Nippon Daicho Komonbyo Gakkai Zasshi 45, no. 1 (1992): 79–82. http://dx.doi.org/10.3862/jcoloproctology.45.79.

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29

Tominaga, Kentaro, Kenya Kamimura, Kazuya Takahashi, Junji Yokoyama, Satoshi Yamagiwa, and Shuji Terai. "Diversion colitis and pouchitis: A mini-review." World Journal of Gastroenterology 24, no. 16 (2018): 1734–47. http://dx.doi.org/10.3748/wjg.v24.i16.1734.

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30

Edwards, George, and Warren. "Diversion colitis - new light through old windows." Histopathology 34, no. 1 (1999): 1–5. http://dx.doi.org/10.1046/j.1365-2559.1999.00624.x.

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31

Dixon, M. F. "Diversion colitis: new light through old windows." Histopathology 35, no. 1 (1999): 86. http://dx.doi.org/10.1046/j.1365-2559.1999.00728.x.

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32

&NA;. "Short chain fatty acids improve diversion colitis." Inpharma Weekly &NA;, no. 726 (1990): 12. http://dx.doi.org/10.2165/00128413-199007260-00027.

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33

Villanacci, V., I. C. Talbot, E. Rossi, and G. Bassotti. "Ischaemia: a pathogenetic clue in diversion colitis?" Colorectal Disease 9, no. 7 (2007): 601–5. http://dx.doi.org/10.1111/j.1463-1318.2006.01182.x.

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34

Dixon. "Diversion colitis: new light through old windows." Histopathology 35, no. 1 (1999): 86. http://dx.doi.org/10.1046/j.1365-2559.1999.0728a.x.

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35

Edwards, George, and Warren. "Diversion colitis: new light through old windows." Histopathology 35, no. 1 (1999): 86–87. http://dx.doi.org/10.1046/j.1365-2559.1999.0728b.x.

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36

Rodríguez-Padilla, Ángela, Germán Morales-Martín, Rocío Pérez-Quintero, Juan Gómez-Salgado, Ricardo Rada-Morgades, and Carlos Ruiz-Frutos. "Diversion Colitis: Macro and Microscopic Findings after Probiotics Stimulation." Biology 10, no. 4 (2021): 303. http://dx.doi.org/10.3390/biology10040303.

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The use of a loop ileostomy as the defunctioning procedure of choice to protect a distal colonic anastomosis causes histological and endoscopic changes in the intestinal mucosal architecture, which have been related to chronic inflammation and changes in the microflora that consequently impact the intestinal structure and function following fecal stream diversion. The aim of this study was to evaluate the histological and endoscopic changes on the colonic mucosa in patients with diversion colitis after stimulation of the efferent loop with probiotics prior to closure of the protective ileostom

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37

Almeida, Marcos Gonçalves de, José Aires Pereira, Canila Morais Gonçalves da Silva, et al. "Evaluation of topical n-acetylcysteine in diversion colitis." Journal of Coloproctology (Rio de Janeiro) 32, no. 3 (2012): 223–31. http://dx.doi.org/10.1590/s2237-93632012000300004.

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38

Jarnerot, G., J. Bohr, C. Tysk, and S. Eriksson. "Faecal stream diversion in patients with collagenous colitis." Gut 38, no. 1 (1996): 154–55. http://dx.doi.org/10.1136/gut.38.1.154-b.

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39

Roe, A. M., B. F. Warren, A. J. Brodribb, and C. Brown. "Diversion colitis and involution of the defunctioned anorectum." Gut 34, no. 3 (1993): 382–85. http://dx.doi.org/10.1136/gut.34.3.382.

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40

Pereira, José Aires, Murilo Rocha Rodrigues, Daniela Tiemi Sato, et al. "Evaluation of sucralfate enema in experimental diversion colitis." Journal of Coloproctology 33, no. 4 (2013): 182–90. http://dx.doi.org/10.1016/j.jcol.2013.08.005.

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41

Ma, C. K., C. Gottlieb, and P. A. Haas. "Diversion colitis: A clinicopathologic study of 21 cases." Human Pathology 21, no. 4 (1990): 429–36. http://dx.doi.org/10.1016/0046-8177(90)90206-k.

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42

Gundling, F., M. Tiller, A. Agha, W. Schepp, and I. Iesalnieks. "Successful autologous fecal transplantation for chronic diversion colitis." Techniques in Coloproctology 19, no. 1 (2014): 51–52. http://dx.doi.org/10.1007/s10151-014-1220-2.

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43

Vujanic and Dojcinov. "Diversion colitis in children: an iatrogenic appendix vermiformis?" Histopathology 36, no. 1 (2000): 41–46. http://dx.doi.org/10.1046/j.1365-2559.2000.00803.x.

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44

Orsay, Charles P., Dong O. Kim, Russell K. Pearl, and Herand Abcarian. "Diversion colitis in patients scheduled for colostomy closure." Diseases of the Colon & Rectum 36, no. 4 (1993): 366–67. http://dx.doi.org/10.1007/bf02053940.

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45

van der Holst, A. M., T. Otten, A. R. P. K. M. van Renterghem, et al. "P184 Temporary defunctioning ileostomy in therapy refractory Crohn’s colitis: A viable alternative to immediate colorectal resection?" Journal of Crohn's and Colitis 14, Supplement_1 (2020): S230—S231. http://dx.doi.org/10.1093/ecco-jcc/jjz203.313.

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Abstract Background The faecal stream is one of many factors influencing the pathogenesis of Crohn’s disease. Previous research demonstrated some Crohn’s patients benefit from temporary faecal diversion. This study aims to determine the role of temporary faecal diversion by means of a defunctioning ileostomy in treating patients with therapy refractory Crohn’s colitis. Methods Data were retrieved by retrospectively assessing patient records. All patients receiving a temporary ileostomy for therapy refractory Crohn’s colitis from three Dutch hospitals between 2010 and 2018 were included. Patien

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46

Rodríguez-Padilla, Ángela, Germán Morales-Martín, Rocío Pérez-Quintero, Juan Gómez-Salgado, and Carlos Ruiz-Frutos. "Serological Biomarkers and Diversion Colitis: Changes after Stimulation with Probiotics." Biomolecules 11, no. 5 (2021): 684. http://dx.doi.org/10.3390/biom11050684.

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Diversion colitis is a non-specific inflammation of a defunctionalised segment of the colon after a temporary stoma has been performed. This inflammation is associated with an alteration of certain inflammatory serum markers. The aims of this study were, firstly, to evaluate the modification of inflammatory biomarkers after stimulation with probiotics prior to closure of the protective ileostomy. Secondly, to identify if a relationship could be established between the severity of diversion colitis and the alteration of inflammatory biomarkers in the blood. A prospective, randomized, double-bli

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47

Kagawa, Tetsuya, Shin Nakatani, Hiroyuki Kishimoto, et al. "An Extended Retroperitoneal Emphysema with Diversion Colitis after Colonoscopy." Japanese Journal of Gastroenterological Surgery 49, no. 8 (2016): 804–11. http://dx.doi.org/10.5833/jjgs.2015.0141.

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48

Komuro, Y. "Diversion colitis with a mucosal tear on endoscopic insufflation." Gut 52, no. 9 (2003): 1388–89. http://dx.doi.org/10.1136/gut.52.9.1388.

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49

Sorrentini, I., G. D'Argenio, C. Iannace, et al. "Butyrate enemas in diversion colitis enhanches tissue transglutaminase activity." Gastroenterology 114 (April 1998): A1088—A1089. http://dx.doi.org/10.1016/s0016-5085(98)84427-2.

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50

YEONG, M. L., P. B. BETHWAITE, J. PRASAD, and W. H. ISBISTER. "Lymphoid follicular hyperplasia?a distinctive feature of diversion colitis." Histopathology 19, no. 1 (1991): 55–62. http://dx.doi.org/10.1111/j.1365-2559.1991.tb00894.x.

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