Relevant bibliographies by topics / DMBA induced Carcinogenesis

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'DMBA induced Carcinogenesis'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "DMBA induced Carcinogenesis"

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Schneider, Joanne, David Warshawsky, Kent Mitchell, et al. "Effects of DMBA Preparation on DMBA-Induced Rat Mammary Carcinogenesis." Polycyclic Aromatic Compounds 22, no. 3-4 (2002): 831–39. http://dx.doi.org/10.1080/10406630290103988.

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Modi, Badri G., Jason Neustadter, Elisa Binda, et al. "Langerhans Cells Facilitate Epithelial DNA Damage and Squamous Cell Carcinoma." Science 335, no. 6064 (2012): 104–8. http://dx.doi.org/10.1126/science.1211600.

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Polyaromatic hydrocarbons (PAHs) are prevalent, potent carcinogens, and 7,12-dimethylbenz[a]anthracene (DMBA) is a model PAH widely used to study tumorigenesis. Mice lacking Langerhans cells (LCs), a signatory epidermal dendritic cell (DC), are protected from cutaneous chemical carcinogenesis, independent of T cell immunity. Investigation of the underlying mechanism revealed that LC-deficient skin was relatively resistant to DMBA-induced DNA damage. LCs efficiently metabolized DMBA to DMBA-trans-3,4-diol, an intermediate proximal to oncogenic Hras mutation, and DMBA-treated LC-deficient skin c
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Anandhi Nallu, Suresh Kathiresan, Sivakumar Kathiresan, and Ilanchit chenni. "Tumour preventive potential of sclareol on 7, 12 dimethylbenz [a] anthracene (DMBA) induced hamster buccal pouch carcinogenesis." International Journal of Research in Pharmaceutical Sciences 11, no. 1 (2020): 1182–91. http://dx.doi.org/10.26452/ijrps.v11i1.1956.

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Sclareol has demonstrated a broad variety of biological activities that belong to the antioxidant anti-inflammatory and anticancer activities that are used in traditional medicine. In the current study, we intended to explore the antitumor possible of sclareol along 7, 12-dimethylbenz (a) anthracene (DMBA) evoked golden Syrian hamster’s buccal pouch carcinogenesis. Lipid peroxidation and antioxidants were assessed by the buccal tissue, and plasma of DMBA evoked golden Syrian hamster buccal pouch carcinogenesis of observational animals. We observed 100% of tumor establishment and noted defects
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Rajasekaran, D., S. Manoharan, MM Prabhakar, and A. Manimaran. "Enicostemma littorale prevents tumor formation in 7,12-dimethylbenz(a)anthracene-induced hamster buccal pouch carcinogenesis." Human & Experimental Toxicology 34, no. 9 (2015): 911–21. http://dx.doi.org/10.1177/0960327114562033.

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Oral cancer is one of the most common malignancies worldwide, and India has recorded the highest annual incidence of oral cancer in comparison with other countries. Altered lipid peroxidation and antioxidant status along with defect in detoxification cascade have been implicated in the pathogenesis of several cancers including oral cancer. The aim of this study was to investigate the chemopreventive potential of ethanolic extract of Enicostemma littorale leaves (ElELet) in 7,12-dimethylbenz(a)anthracene (DMBA)-induced hamster buccal pouch carcinogenesis. Oral tumor was developed in the buccal
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Akrom, Akrom, Titiek Hidayati, Sagiran Sagiran та Indrayanti Indrayanti. "Black Cumin Seeds Extract Increase Lymphocyte Activity in IFN-γ Secretion in Sprague Dawley Rat (SD) Induced by Dimethylbenzantracene". Indonesian Journal of Cancer Chemoprevention 10, № 3 (2019): 140. http://dx.doi.org/10.14499/indonesianjcanchemoprev10iss3pp140-148.

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Interferon-gamma (IFN-γ) is one of the central cytokines in the anti-carcinogenesis immune response. Black cumin seeds (BCS) have an active content of thymoquinone and unsaturated fatty acids with biological activity as immunomodulators. This study aimed to determine the effect of administration of BCS extract on IFN-γ secretion activity by DMBA-induced SD rat lymphocytes. In vivo experimental study on DMBA-induced SD rats, BCS extract was given with three doses for two weeks before being induced and five weeks during DMBA induction. IFN-γ levels in lymphocyte culture supernatants were determi
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Tsai, Ruei-Lan, Bing-Ying Ho, and Tzu-Ming Pan. "Red Mold Rice Mitigates Oral Carcinogenesis in 7,12-Dimethyl-1,2-Benz[a]anthracene-Induced Oral Carcinogenesis in Hamster." Evidence-Based Complementary and Alternative Medicine 2011 (2011): 1–8. http://dx.doi.org/10.1093/ecam/nep215.

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The prevalence of oral tumor has exponentially increased in recent years; however, the effective therapies or prevention strategies are not sufficient. Red mold rice is a traditional Chinese food, and several reports have demonstrated that red mold rice had an anti-tumor effect. However, the possible anti-tumor mechanisms of the red mold rice are unclear. In this study, we examined the anti-tumor effect of red mold rice on 7,12-dimethyl-1,2-benz[a]anthracene (DMBA)-induced oral tumor in hamster. The ethanol extract of red mold rice (RMRE) treatment significantly decreases the levels of DMBA-in
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KONDO, Masanori, Kohzo TSUCHIKAWA, and Joji KATO. "Two-phase carcinogenesis on the DMBA induced carcinoma." Japanese Journal of Oral & Maxillofacial Surgery 34, no. 12 (1988): 2537–43. http://dx.doi.org/10.5794/jjoms.34.2537.

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Davis, Leemol, and Girija Kuttan. "Effect of Withania somnifera on DMBA induced carcinogenesis." Journal of Ethnopharmacology 75, no. 2-3 (2001): 165–68. http://dx.doi.org/10.1016/s0378-8741(00)00404-9.

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Hudson, Tamaro S., Bradley A. Carlson, Mark J. Hoeneroff, et al. "Selenoproteins reduce susceptibility to DMBA-induced mammary carcinogenesis." Carcinogenesis 33, no. 6 (2012): 1225–30. http://dx.doi.org/10.1093/carcin/bgs129.

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Anindyajati, Anindyajati, Andita Pra Darma, Ika Nurjizah, Dita Brenna Septhea, and Agung Endro Nugroho. "Ficus septica Burm. f. Leaves Ethanolic Extract Triggered Apoptosis on 7,12-Dimethylbenz[a]anthracene-Induced Rat Mammary Carcinogenesis Qualitatively." Indonesian Journal of Cancer Chemoprevention 3, no. 1 (2012): 334. http://dx.doi.org/10.14499/indonesianjcanchemoprev3iss1pp334-338.

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Ficus septica Burm. f. ethanolic extract (FEE) shows cytotoxic effects on several cancer cell lines. Our research aimed to investigate the effect of FEE on apoptosis induction and p53 expression against carcinogenesis of 7,12-Dimethylbenz[a]anthracene (DMBA)-induced rat mammary.The research was conducted by comparing both apoptosis induction and p53 expression in DMBA-induced rats that were treated with FEE against control groups. Cells that undergo apoptosis were visualized by Double Staining method with acridine orange and ethidium bromide, while p53 expression was detected by IHC staining.
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Dissertations / Theses on the topic "DMBA induced Carcinogenesis"

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Tsai, Ruei-Lan, and 蔡瑞蘭. "Effects of Red Mold Rice on Oral Carcinogenesis in DMBA-Induced Hamster Animal Model." Thesis, 2008. http://ndltd.ncl.edu.tw/handle/24653544454282122703.

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碩士<br>國立臺灣大學<br>微生物與生化學研究所<br>96<br>The ethanol extract of red mold rice (RMRE) can inhibit the growth of squamous cell carcinoma-4 (SCC-4) cells (IC50 = 116 μg/mL), and monacolin K is the main component in RMRE to exert the inhibition of cancer cell growth. Moreover, RMRE inhibited lipopolysaccharide-induced oxidative stress and inflammation in RAW264.7 cells. And the anti-oxidation and anti-inflammatory effects were not only due to monacolin K, but supposed to be a synergic effect among monacolin K, citrinin, monacin, ankaflavinn, and others. Furthermore, DMBA induced oral cancer in hamster
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Chen, Chi-Juen, and 陳致均. "The study of sequential expression of placental glutathione S- transferase(GST-P)during DMBA-induced hamster buccal pouch squamous cell carcinogenesis." Thesis, 1996. http://ndltd.ncl.edu.tw/handle/21788672615398277605.

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碩士<br>高雄醫學院<br>牙醫學研究所<br>84<br>To date, GST isoenzymes appear to be potential markers in oral mucosa.In the first part of our study(1992), assessments of the expression and locallization of the GST isoenzymes in biopsies from the normal oral mucosa, benign fibroma, potentially premalignant(leukoplakia, submucous fibrosis,verrucous hyperplasia and malignant(squamous cell carcinoma and verrucouscarcinoma) oral lesions were made immunohistochemically with specific polyclomal antibodies
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Nadenau, Eva. "Expression von SDF-1/CXCL12 und CXCR4 in der sequenziellen DMBA-induzierten Karzinogenese des Hamsters." Doctoral thesis, 2015. http://hdl.handle.net/11858/00-1735-0000-0022-5F9D-7.

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Book chapters on the topic "DMBA induced Carcinogenesis"

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Miller, Edward G., Francisco Rivera-Hidalgo, and William H. Binnie. "3-Methoxybenzamide, A Possible Initiator for DMBA-Induced Carcinogenesis." In ADP-Ribose Transfer Reactions. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4615-8507-7_54.

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Dolezalova, V., A. Rejthar, R. Neunutil, et al. "Biochemical Markers of Cell Heterogeneity in Rat Mammary Tumors Induced by 7,12-Dimethylbenzanthracene (DMBA) and N-Methyl-N-Nitrosuorea (MNU)." In Biochemistry of Chemical Carcinogenesis. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0539-2_26.

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José Soto-Castillo, Juan, and Isicio Ortega-Medina. "Experimental Carcinogenesis with 7,12-Dimethylbenz(a)Anthrazene (DMBA) and Its Inhibition with Extra Virgin Olive Oil and a Diet of Mature Olives (Picual Variety)." In Olive Oil - New Perspectives and Applications [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96514.

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7,12-Dimethylbenz(a)anthrazene (DMBA) is a carcinogen that induces carcinomas within a few weeks of application. Forty-four male hamsters were divided into four groups: DMBA dissolved in paraffin oil (DMBA-PO), DMBA dissolved in olive oil (DMBA-OO), paraffin oil and olive oil. Their mouths were swabbed daily with paraffin oil or extra virgin olive oil alternatively for the first two weeks, during the biweekly application of DMBA at 0.5% diluted in paraffin oil or olive oil for five weeks and daily until the twentieth week. The animals in the DMBA-OO and olive oil groups received an additional diet of mature Picual olives. The DMBA-PO carcinogen effect (35 carcinomas) is 100% and the inhibitory effect 0. The use of olive oil as DMBA solvent and the ad libitum diet with Picual olive has an inhibitory effect of 80%, with only three intraepithelial carcinomas and four verrucous carcinomas occurring and no invasive carcinoma.
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Conference papers on the topic "DMBA induced Carcinogenesis"

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Naseemuddin, Mohammed, Tahseen H. Nasti, and Nabiha Yusuf. "Abstract 3459: Loss ofp16INK4agene renders mice more susceptible to DMBA-induced mammary carcinogenesis." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3459.

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Das, Ila, Asha Acharya, Archana Sengupta, Shukta Das, Sudin Bhattacharya, and Tapas Saha. "Abstract 2880: Spices suppress oxidative stress during DMBA induced skin carcinogenesis in mice, mimicking human non-melanoma skin cancer." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2880.

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Diagaradjane, Parmeswaran, Mohammed A. Yaseen, Jie Yu, Michael S. Wong, and Bahman Anvari. "Autofluorescence characterization of DMBA-TPA-induced two-stage carcinogenesis in mouse skin for the early detection of tissue transformation." In Biomedical Optics 2005, edited by Kenneth E. Bartels, Lawrence S. Bass, Werner T. W. de Riese, et al. SPIE, 2005. http://dx.doi.org/10.1117/12.589328.

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Zhang, Xinyan, Xin Chen, Ye Lu, et al. "Abstract 1600: Chemoprevention of 7,12-dimethylbenz[a]anthracene (DMBA)-induced hamster cheek pouch carcinogenesis by topical garcinol through inhibition of 5-lipoxygenase." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1600.

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Ramadoss, Hemavardhini, Suresh Kathiresan, and Sanjay Gupta. "Abstract 129: Apoptosis & anti-proliferative nature of Aescin on 7,12 dimethylbenz[a]anthracene (DMBA) induced hamster buccal pouch carcinogenesis via downregulating Notch signaling." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-129.

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You might also be interested in the extended bibliographies on the topic 'DMBA induced Carcinogenesis' for particular source types:
Journal articles
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