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Journal articles on the topic 'Dmp53'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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1

Chen, Su, Hui-Min Wei, Wen-Wen Lv, Da-Liang Wang, and Fang-Lin Sun. "E2 Ligase dRad6 Regulates DMP53 Turnover inDrosophila." Journal of Biological Chemistry 286, no. 11 (January 4, 2011): 9020–30. http://dx.doi.org/10.1074/jbc.m110.190314.

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2

Bodai, László, Norbert Pardi, Zsuzsanna Újfaludi, Orsolya Bereczki, Orbán Komonyi, Eva Balint, and Imre M. Boros. "Daxx-like Protein ofDrosophilaInteracts with Dmp53 and Affects Longevity andArkmRNA Level." Journal of Biological Chemistry 282, no. 50 (October 12, 2007): 36386–93. http://dx.doi.org/10.1074/jbc.m705547200.

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3

Monk, Adrian C., Helen E. Abud, and Gary R. Hime. "Dmp53 is sequestered to nuclear bodies in spermatogonia of Drosophila melanogaster." Cell and Tissue Research 350, no. 2 (August 12, 2012): 385–94. http://dx.doi.org/10.1007/s00441-012-1479-4.

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4

Jassim, O. W. "Dmp53 protects the Drosophila retina during a developmentally regulated DNA damage response." EMBO Journal 22, no. 20 (October 15, 2003): 5622–32. http://dx.doi.org/10.1093/emboj/cdg543.

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5

Bauer, Johannes H., Chengyi Chang, Gina Bae, Siti Nur Sarah Morris, and Stephen L. Helfand. "Dominant-negative Dmp53 extends life span through the dTOR pathway in D. melanogaster." Mechanisms of Ageing and Development 131, no. 3 (March 2010): 193–201. http://dx.doi.org/10.1016/j.mad.2010.01.007.

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6

Bauer, J. H., C. Chang, S. N. S. Morris, S. Hozier, S. Andersen, J. S. Waitzman, and S. L. Helfand. "Expression of dominant-negative Dmp53 in the adult fly brain inhibits insulin signaling." Proceedings of the National Academy of Sciences 104, no. 33 (August 8, 2007): 13355–60. http://dx.doi.org/10.1073/pnas.0706121104.

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7

Colombani, Julien, Cédric Polesello, Filipe Josué, and Nicolas Tapon. "Dmp53 Activates the Hippo Pathway to Promote Cell Death in Response to DNA Damage." Current Biology 16, no. 14 (July 2006): 1453–58. http://dx.doi.org/10.1016/j.cub.2006.05.059.

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8

Ibarra, E. Piedra, J. T. Torres Gregorio, B. G. Ramírez Cruz, M. E. Heres Pulido, L. F. Santos Cruz, and A. Ponciano Gomez. "Transcription levels of Cyp450s, H99, Hsps and Dmp53 in Drosophila melanogaster larvae treated with zearalenona." Toxicology Letters 258 (September 2016): S76—S77. http://dx.doi.org/10.1016/j.toxlet.2016.06.1357.

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9

Merino, Carlos, Enrique Reynaud, Martha Vázquez, and Mario Zurita. "DNA Repair and Transcriptional Effects of Mutations in TFIIH inDrosophila Development." Molecular Biology of the Cell 13, no. 9 (September 2002): 3246–56. http://dx.doi.org/10.1091/mbc.e02-02-0087.

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Mutations in XPB and XPD TFIIH helicases have been related with three hereditary human disorders: xeroderma pigmentosum, Cockayne syndrome, and trichothiodystrophy. The dual role of TFIIH in DNA repair and transcription makes it difficult to discern which of the mutant TFIIH phenotypes is due to defects in any of these different processes. We used haywire (hay), theDrosophila XPB homolog, to dissect this problem. Our results show that when hay dosage is affected, the fly shows defects in structures that require high levels of transcription. We found a genetic interaction between hay andcdk7, and we propose that some of these phenotypes are due to transcriptional deficiencies. We also found more apoptotic cells in imaginal discs and in the CNS of hay mutant flies than in wild-type flies. Because this abnormal level of apoptosis was not detected in cdk7 flies, this phenotype could be related to defects in DNA repair. In addition the apoptosis induced by p53 Drosophila homolog (Dmp53) is suppressed in heterozygous hay flies.

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10

Bauer, Johannes H., Siti Nur Sarah Morris, Chengyi Chang, Thomas Flatt, Jason G. Wood, and Stephen L. Helfand. "dSir2 and Dmp53 interact to mediate aspects of CR-dependent life span extension in D. melanogaster." Aging 1, no. 1 (November 6, 2008): 38–48. http://dx.doi.org/10.18632/aging.100001.

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11

Yanku, Yifat, Eliya Bitman-Lotan, Yaniv Zohar, Estee Kurant, Norman Zilke, Martin Eilers, and Amir Orian. "Drosophila HUWE1 Ubiquitin Ligase Regulates Endoreplication and Antagonizes JNK Signaling During Salivary Gland Development." Cells 7, no. 10 (September 26, 2018): 151. http://dx.doi.org/10.3390/cells7100151.

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The HECT-type ubiquitin ligase HECT, UBA and WWE Domain Containing 1, (HUWE1) regulates key cancer-related pathways, including the Myc oncogene. It affects cell proliferation, stress and immune signaling, mitochondria homeostasis, and cell death. HUWE1 is evolutionarily conserved from Caenorhabditis elegance to Drosophila melanogaster and Humans. Here, we report that the Drosophila ortholog, dHUWE1 (CG8184), is an essential gene whose loss results in embryonic lethality and whose tissue-specific disruption establishes its regulatory role in larval salivary gland development. dHUWE1 is essential for endoreplication of salivary gland cells and its knockdown results in the inability of these cells to replicate DNA. Remarkably, dHUWE1 is a survival factor that prevents premature activation of JNK signaling, thus preventing the disintegration of the salivary gland, which occurs physiologically during pupal stages. This function of dHUWE1 is general, as its inhibitory effect is observed also during eye development and at the organismal level. Epistatic studies revealed that the loss of dHUWE1 is compensated by dMyc proeitn expression or the loss of dmP53. dHUWE1 is therefore a conserved survival factor that regulates organ formation during Drosophila development.

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12

Ortega-Arellano, Hector Flavio, Marlene Jimenez-Del-Rio, and Carlos Velez-Pardo. "Dmp53, basket and drICE gene knockdown and polyphenol gallic acid increase life span and locomotor activity in a Drosophila Parkinson's disease model." Genetics and Molecular Biology 36, no. 4 (2013): 608–15. http://dx.doi.org/10.1590/s1415-47572013000400020.

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13

Pankotai, Tibor, Orbán Komonyi, László Bodai, Zsuzsanna Újfaludi, Selen Muratoglu, Anita Ciurciu, László Tora, János Szabad, and Imre Boros. "The Homologous Drosophila Transcriptional Adaptors ADA2a and ADA2b Are both Required for Normal Development but Have Different Functions." Molecular and Cellular Biology 25, no. 18 (September 15, 2005): 8215–27. http://dx.doi.org/10.1128/mcb.25.18.8215-8227.2005.

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ABSTRACT In Drosophila and several other metazoan organisms, there are two genes that encode related but distinct homologs of ADA2-type transcriptional adaptors. Here we describe mutations of the two Ada2 genes of Drosophila melanogaster. By using mutant Drosophila lines, which allow the functional study of individual ADA2s, we demonstrate that both Drosophila Ada2 genes are essential. Ada2a and Ada2b null homozygotes are late-larva and late-pupa lethal, respectively. Double mutants have a phenotype identical to that of the Ada2a mutant. The overproduction of ADA2a protein from transgenes cannot rescue the defects resulting from the loss of Ada2b, nor does complementation work vice versa, indicating that the two Ada2 genes of Drosophila have different functions. An analysis of germ line mosaics generated by pole-cell transplantation revealed that the Ada2a function (similar to that reported for Ada2b) is required in the female germ line. A loss of the function of either of the Ada2 genes interferes with cell proliferation. Interestingly, the Ada2b null mutation reduces histone H3 K14 and H3 K9 acetylation and changes TAF10 localization, while the Ada2a null mutation does not. Moreover, the two ADA2s are differently required for the expression of the rosy gene, involved in eye pigment production, and for Dmp53-mediated apoptosis. The data presented here demonstrate that the two genes encoding homologous transcriptional adaptor ADA2 proteins in Drosophila are both essential but are functionally distinct.

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14

Fregoso, Mariana, Jean-Philippe Lainé, Javier Aguilar-Fuentes, Vincent Mocquet, Enrique Reynaud, Frédéric Coin, Jean-Marc Egly, and Mario Zurita. "DNA Repair and Transcriptional Deficiencies Caused by Mutations in the Drosophila p52 Subunit of TFIIH Generate Developmental Defects and Chromosome Fragility." Molecular and Cellular Biology 27, no. 10 (March 5, 2007): 3640–50. http://dx.doi.org/10.1128/mcb.00030-07.

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ABSTRACT The transcription and DNA repair factor TFIIH is composed of 10 subunits. Mutations in the XPB, XPD, and p8 subunits are genetically linked to human diseases, including cancer. However, no reports of mutations in other TFIIH subunits have been reported in higher eukaryotes. Here, we analyze at genetic, molecular, and biochemical levels the Drosophila melanogaster p52 (DMP52) subunit of TFIIH. We found that DMP52 is encoded by the gene marionette in Drosophila and that a defective DMP52 produces UV light-sensitive flies and specific phenotypes during development: organisms are smaller than their wild-type siblings and present tumors and chromosomal instability. The human homologue of DMP52 partially rescues some of these phenotypes. Some of the defects observed in the fly caused by mutations in DMP52 generate trichothiodystrophy and cancer-like phenotypes. Biochemical analysis of DMP52 point mutations introduced in human p52 at positions homologous to those of defects in DMP52 destabilize the interaction between p52 and XPB, another TFIIH subunit, thus compromising the assembly of the complex. This study significantly extends the role of p52 in regulating XPB ATPase activity and, consequently, both its transcriptional and nucleotide excision repair functions.

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15

de Bustros, A., R. Y. Lee, D. Compton, T. Y. Tsong, S. B. Baylin, and B. D. Nelkin. "Differential utilization of calcitonin gene regulatory DNA sequences in cultured lines of medullary thyroid carcinoma and small-cell lung carcinoma." Molecular and Cellular Biology 10, no. 4 (April 1990): 1773–78. http://dx.doi.org/10.1128/mcb.10.4.1773.

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Regulation of expression of the human calcitonin gene was found to differ between two tumor lines of different tissue origin, medullary thyroid carcinoma (TT line) and small-cell lung carcinoma (DMS53 line). Distal 5' DNA elements between -750 and -2000 exhibited a stronger basal activity in DMS53 than in TT cells, whereas proximal DNA sequences between -132 and -252 mediated a dramatic cyclic AMP response in TT but not DMS53 cells.

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16

de Bustros, A., R. Y. Lee, D. Compton, T. Y. Tsong, S. B. Baylin, and B. D. Nelkin. "Differential utilization of calcitonin gene regulatory DNA sequences in cultured lines of medullary thyroid carcinoma and small-cell lung carcinoma." Molecular and Cellular Biology 10, no. 4 (April 1990): 1773–78. http://dx.doi.org/10.1128/mcb.10.4.1773-1778.1990.

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Regulation of expression of the human calcitonin gene was found to differ between two tumor lines of different tissue origin, medullary thyroid carcinoma (TT line) and small-cell lung carcinoma (DMS53 line). Distal 5' DNA elements between -750 and -2000 exhibited a stronger basal activity in DMS53 than in TT cells, whereas proximal DNA sequences between -132 and -252 mediated a dramatic cyclic AMP response in TT but not DMS53 cells.

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17

ANDERSEN, TIMOTHY L., and TONY R. MARTINEZ. "DMP3: A DYNAMIC MULTILAYER PERCEPTRON CONSTRUCTION ALGORITHM." International Journal of Neural Systems 11, no. 02 (April 2001): 145–65. http://dx.doi.org/10.1142/s0129065701000576.

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This paper presents DMP3 (Dynamic Multilayer Perceptron 3), a multilayer perceptron (MLP) constructive training method that constructs MLPs by incrementally adding network elements of varying complexity to the network. DMP3 differs from other MLP construction techniques in several important ways, and the motivation for these differences are given. Information gain rather than error minimization is used to guide the growth of the network, which increases the utility of newly added network elements and decreases the likelihood that a premature dead end in the growth of the network will occur. The generalization performance of DMP3 is compared with that of several other well-known machine learning and neural network learning algorithms on nine real world data sets. Simulation results show that DMP3 performs better (on average) than any of the other algorithms on the data sets tested. The main reasons for this result are discussed in detail.

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18

Карпунин, Григорий Анатольевич, and Grigorii Anatol'evich Karpunin. "О ключевом пространстве криптосистемы Мак-Элиса на основе двоичных кодов Рида - Маллера." Diskretnaya Matematika 16, no. 2 (2004): 79–84. http://dx.doi.org/10.4213/dm153.

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19

Камозина, О. В., and O. V. Kamozina. "Булевы решетки $n$-кратно $Omega$-биканонических классов Фиттинга." Diskretnaya Matematika 14, no. 3 (2002): 47–53. http://dx.doi.org/10.4213/dm253.

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20

Гонсалес, С., Santos Gonzalez, Е. Коусело, Elena Couselo, Виктор Марков, Viktor Markov, Александр Нечаев, and Aleksandr Nechaev. "Параметры рекурсивных МДР-кодов." Diskretnaya Matematika 12, no. 4 (2000): 3–24. http://dx.doi.org/10.4213/dm353.

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21

Дьяконова, Елена Евгеньевна, and Elena Evgen'evna D'yakonova. "Ветвящийся процесс с миграцией в случайной среде." Diskretnaya Matematika 9, no. 1 (1997): 30–42. http://dx.doi.org/10.4213/dm453.

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22

Козлов, Вадим Никитович, and Vadim Nikitovich Kozlov. "О кодировании дискретных фигур." Diskretnaya Matematika 8, no. 4 (1996): 57–61. http://dx.doi.org/10.4213/dm553.

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23

Pyo, Ayoung, Dong-Yeon Kim, Heejung Kim, Daejin Lim, Seong Young Kwon, Sae-Ryung Kang, Hyung-Seok Kim, Hee-Seung Bom, and Jung-Joon Min. "Ultrasensitive detection of malignant melanoma using PET molecular imaging probes." Proceedings of the National Academy of Sciences 117, no. 23 (May 21, 2020): 12991–99. http://dx.doi.org/10.1073/pnas.1922313117.

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Malignant melanoma has one of the highest mortality rates of any cancer because of its aggressive nature and high metastatic potential. Clinical staging of the disease at the time of diagnosis is very important for the prognosis and outcome of melanoma treatment. In this study, we designed and synthesized the18F-labeled pyridine-based benzamide derivativesN-(2-(dimethylamino)ethyl)-5-[18F]fluoropicolinamide ([18F]DMPY2) andN-(2-(dimethylamino)ethyl)-6-[18F]fluoronicotinamide ([18F]DMPY3) to detect primary and metastatic melanoma at an early stage and evaluated their performance in this task. [18F]DMPY2 and [18F]DMPY3 were synthesized by direct radiofluorination of the bromo precursor, and radiochemical yields were ∼15–20%. Cell uptakes of [18F]DMPY2 and [18F]DMPY3 were >103-fold and 18-fold higher, respectively, in B16F10 (mouse melanoma) cells than in negative control cells. Biodistribution studies revealed strong tumor uptake and retention of [18F]DMPY2 (24.8% injected dose per gram of tissue [ID/g] at 60 min) and [18F]DMPY3 (11.7%ID/g at 60 min) in B16F10 xenografts. MicroPET imaging of both agents demonstrated strong tumoral uptake/retention and rapid washout, resulting in excellent tumor-to-background contrast in B16F10 xenografts. In particular, [18F]DMPY2 clearly visualized almost all metastatic lesions in lung and lymph nodes, with excellent image quality. [18F]DMPY2 demonstrated a significantly higher tumor-to-liver ratio than [18F]fluorodeoxyglucose ([18F]FDG) and the previously reported benzamide tracersN-[2-(diethylamino)-ethyl]-5-[18F]fluoropicolinamide ([18F]P3BZA) andN-[2-(diethylamino)-ethyl]-4-[18F]fluorobenzamide ([18F]FBZA) in B16F10-bearing or SK-MEL-3 (human melanoma)-bearing mice. In conclusion, [18F]DMPY2 might have strong potential for the diagnosis of early stage primary and metastatic melanoma using positron emission tomography (PET).

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24

Камозина, О. В., and O. V. Kamozina. "Алгебраические решетки кратно $Omega$-расслоенных классов Фиттинга." Diskretnaya Matematika 18, no. 2 (2006): 139–45. http://dx.doi.org/10.4213/dm53.

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25

Wang, Liyun, Danyu Xia, Jianbin Chao, Junjie Zhang, Xuehong Wei, and Pi Wang. "A dimethoxypillar[5]arene/azastilbene host–guest recognition motif and its applications in the fabrication of polypseudorotaxanes." Organic & Biomolecular Chemistry 17, no. 24 (2019): 6038–42. http://dx.doi.org/10.1039/c9ob00862d.

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A host–guest recognition motif based on 1,4-dimethoxylpillar[5]arene (DMP5) and guest trans-4,4′-vinylenedipyridine (trans-G1) was fabricated and applied to construct metallosupramolecular polypseudorotaxanes.

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26

Koning, A. M. H., W. K. H. Kuchenbecker, H. Groen, A. Hoek, J. A. Land, K. S. Khan, and B. W. J. Mol. "Economic consequences of overweight and obesity in infertility: a framework for evaluating the costs and outcomes of fertility care." Human Reproduction Update 16, no. 3 (January 1, 2010): 246–54. http://dx.doi.org/10.1093/humupd/dmp053.

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27

Fons, C., A. Ormazabal, R. Artuch, E. Fernandez, J. Campistol, and A. Garćla. "DMP03 Pontocerebellar hypoplasia type 2: clinical and biochemical findings." European Journal of Paediatric Neurology 11 (September 2007): 77. http://dx.doi.org/10.1016/s1090-3798(08)70542-7.

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28

Winter, Steffen. "Curvature measures and fractals." Dissertationes Mathematicae 453 (2008): 1–66. http://dx.doi.org/10.4064/dm453-0-1.

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29

Ravi, Rajani K., Arunthathi Thiagalingam, Erich Weber, Martin McMahon, Barry D. Nelkin, and Mack Mabry. "Raf-1 Causes Growth Suppression and Alteration of Neuroendocrine Markers in DMS53 Human Small-Cell Lung Cancer Cells." American Journal of Respiratory Cell and Molecular Biology 20, no. 4 (April 1999): 543–49. http://dx.doi.org/10.1165/ajrcmb.20.4.3406.

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