Relevant bibliographies by topics / DNA-damaged

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'DNA-damaged'

Author: Grafiati
Published: 4 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "DNA-damaged"

1

Lehmann, Alan R. "Replication of Damaged DNA." Cell Cycle 2, no. 4 (2003): 299–301. http://dx.doi.org/10.4161/cc.2.4.407.

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Caetano, R. A., and P. A. Schulz. "Delocalized states in damaged DNA." Brazilian Journal of Physics 36, no. 2a (2006): 459–61. http://dx.doi.org/10.1590/s0103-97332006000300061.

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Chatterjee, N., and W. Siede. "Replicating Damaged DNA in Eukaryotes." Cold Spring Harbor Perspectives in Biology 5, no. 12 (2013): a019836. http://dx.doi.org/10.1101/cshperspect.a019836.

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Lukin, Mark, and Carlos de los Santos. "NMR Structures of Damaged DNA." Chemical Reviews 106, no. 2 (2006): 607–86. http://dx.doi.org/10.1021/cr0404646.

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Inoki, Taeko, Hitoshi Endo, Yutaka Inoki, et al. "Damaged DNA-binding protein 2 accelerates UV-damaged DNA repair in human corneal endothelium." Experimental Eye Research 79, no. 3 (2004): 367–76. http://dx.doi.org/10.1016/j.exer.2004.06.010.

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Yeh, J. I., A. S. Levine, S. Du, et al. "Damaged DNA induced UV-damaged DNA-binding protein (UV-DDB) dimerization and its roles in chromatinized DNA repair." Proceedings of the National Academy of Sciences 109, no. 41 (2012): E2737—E2746. http://dx.doi.org/10.1073/pnas.1110067109.

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Lanuszewska, J., A. Cudak, J. Rzeszowska-Wolny, and P. Widłak. "Detection of damage-recognition proteins from human lymphocytes." Acta Biochimica Polonica 47, no. 2 (2000): 443–50. http://dx.doi.org/10.18388/abp.2000_4024.

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Proteins recognizing and binding to damaged DNA (DDB-proteins) were analyzed in human lymphocytes obtained from healthy donors. Using an electrophoretic mobility shift assay several complexes between nuclear extract proteins and damaged DNA were detected: a complex specific for DNA damaged by N-acetoxy-N-acetylaminofluorene, another complex specific for UV-irradiated DNA, and two complexes specific for DNA damaged by cis-dichlorodiammine platinum. All the detected complexes differed in electrophoretic mobility and possibly contained different proteins. Complexes specific for free DNA ends were
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Rzeszowska-Wolny, J., and P. Widłak. "Damaged DNA-binding proteins: recognition of N-acetoxy-acetylaminofluorene-induced DNA adducts." Acta Biochimica Polonica 46, no. 1 (1999): 173–80. http://dx.doi.org/10.18388/abp.1999_4195.

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Proteins which bind to the DNA damaged by genotoxic agents can be detected in all living organisms. Damage-recognition proteins are thought to be generally involved in DNA repair mechanisms. On the other hand, the relevance to DNA repair of some other proteins which show elevated affinity to damaged DNA (e.g. HMG-box containing proteins or histone H1) has not been established. Using the electrophoretic mobility-shift assay we have investigated damage-recognition proteins in nuclei from rat hepatocytes. We detected two different protein complexes which preferentially bound the DNA damaged by N-
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Strickland, Paul T., and Cristl Gentile. "Separation of carcinogen-damaged DNA fragments from undamaged DNA." Nucleic Acids Research 19, no. 24 (1991): 6955. http://dx.doi.org/10.1093/nar/19.24.6955.

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Williams, Loren Dean, and Qi Gao. "DNA-ditercalinium interactions: implications for recognition of damaged DNA." Biochemistry 31, no. 17 (1992): 4315–24. http://dx.doi.org/10.1021/bi00132a024.

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Dissertations / Theses on the topic "DNA-damaged"

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Upton, Amy Louise. "Replication of damaged DNA." Thesis, University of Nottingham, 2009. http://eprints.nottingham.ac.uk/11332/.

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DNA is under constant attack from numerous damaging agents and our cells deal with thousands of lesions every day. With such constant damage it is inevitable that the template will not be completely cleared of lesions before the replication complex arrives. The consequences of the replisome meeting an obstacle will depend upon the nature of the obstacle. I have focussed upon replication in Escherichia coli and the effect of UV-induced lesions, which would block synthesis by the replicative polymerases. It is accepted that a UV lesion in the lagging strand template can be bypassed by the replis
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Brown, Stephanie Marie. "Replication of damaged DNA in mammalian cells." Thesis, University of Sussex, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.445620.

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Turner, Jayne Susan. "Molecular recognition of damaged DNA using synthetic affinity reagents." Thesis, Open University, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.418471.

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Potentially carcinogenic DNA damage that may be derived from dietary sources can be quantified using a number of methods. Some of the best of these methods are based on the use of antibodies that have high affinity and specificity for modified DNA bases. However, given the sometimes limited availability of antibodies there is a requirement for novel reagents that mimic the properties of the best antibodies. From a chemical perspective antibodies are `over engineered' and synthetic affinity reagents would recreate the optimal properties of the binding site with respect to selectivity and affini
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Zamble, Deborah B. (Deborah Beth) 1971. "The responses of cellular proteins to cisplatin-damaged DNA." Thesis, Massachusetts Institute of Technology, 1999. http://hdl.handle.net/1721.1/73344.

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Thesis (Ph.D.)--Massachusetts Institute of Technology, Dept. of Chemistry, 1999.<br>Vita.<br>Includes bibliographical references.<br>The anticancer drug cisplatin is a small inorganic molecule that forms several types of covalent adducts on DNA. There is evidence that at least some of the consequences of this genetic damage are mediated by proteins that bind to the cisplatin-DNA cross-links or influence cellular pathways in response to the genotoxic stress. In either case, such factors can regulate the processing of the cisplatin lesions and thereby affect cellular sensitivity to the drug. Ide
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Jung, Yongwon 1977. "Cellular responses against DNA damaged by platinum anticancer drugs." Thesis, Massachusetts Institute of Technology, 2005. http://hdl.handle.net/1721.1/33746.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Chemistry, 2005.<br>Vita.<br>Includes bibliographical references.<br>The anticancer activity of platinum-based drugs such as cisplatin, carboplatin, and oxaliplatin is mediated by their ability to attack DNA such that generated adducts trigger numerous cellular responses. A better understanding of these processes is critical for developing more effective therapeutic approaches, which can increase the anti-cancer activity of the drugs while minimizing side effects and extending successful treatment to a wider range of human cancer
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Baynton, Kathy J. "Replication of damaged dna in the yeast saccharomyces cerevisiae." Université Louis Pasteur (Strasbourg) (1971-2008), 1998. http://www.theses.fr/1998STR13123.

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Les adduits formes par le cancerogene n-2-acetylaminofluorene (aaf) sur le c8 de la guanine sont tres bloquants lors de la replication et induisent principalement des mutations par decalage du cadre du lecture. Un systeme a ete developpe permettant de mesurer le niveau de synthese en face d'une lesion aaf (stl), une des strategies de tolerance des lesions dans la levure saccharomyces cerevisiae. Des vecteurs heteroduplexes contenant sur un brin la sequence cible (5'-ggg#a#a#f-3' ou 5'-ggcg#a#a#fcc-3') et sur le brin complementaire une heterologie de sequence constituee d'une boucle de 4 nucleo
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Hatton, David H. "DNA repair and replication of ultraviolet-damaged human melanoma cell lines." Thesis, University of Cambridge, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.386324.

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Zainol, Murizal. "Assessment of phytochemicals in preventing oxidatively damaged DNA in bladder cancer." Thesis, University of Leicester, 2011. http://hdl.handle.net/2381/10158.

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Oxidatively damaged DNA is thought to be important in both the initiation and development of bladder carcinoma. Phytochemical compounds are thought to promote optimal health, partly via their antioxidant effects in protecting cellular components against damaging free radicals. The objective of this study is to assess the effect of a standardised bilberry extract, mirtoselect, on the level of endogenous and induced oxidatively damaged DNA in bladder cancer cells, as assessed by the Comet assay. The Comet assay, also known as single-cell gel electrophoresis, represents a simple method for measur
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Hall, Ashley. "ASSESSMENT AND IN VITRO REPAIR OF DAMAGED DNA TEMPLATES FROM FORENSIC STAINS." Doctoral diss., University of Central Florida, 2005. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/2621.

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DNA extracted from biological stains is often intractable to analysis. This may due to a number of factors including a low copy number (LCN) of starting molecules, the presence of soluble inhibitors or damaged DNA templates. Remedies may be available to the forensic scientist to deal with LCN templates and soluble inhibitors but none presently exist for damaged DNA. In fact, only recently has the biochemical nature, the extent of DNA damage in physiological stains and the point at which the damage inflicted upon a particular sample precludes the ability to obtain a genetic profile for purposes
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Hurley, Eldon Kenneth Jr. "Photolyase: Its Damaged DNA Substrate and Amino Acid Radical Formation During Photorepair." Thesis, Virginia Tech, 2005. http://hdl.handle.net/10919/31084.

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Ultraviolet light damages genomic material by inducing the formation of covalent bonds between adjacent pyrimidines. Cis-syn cyclobutane pyrimidine dimers (CPD)constitute the most abundant primary lesion in DNA. Photolyase, a light-activated enzyme, catalytically repairs these lesions. Although many steps in the photolyase-mediated repair process have been mapped, details of the mechanism remain cryptic. Difference FT-IR spectroscopy was employed to obtain new mechanistic information about photorepair. Purified oligonucleotides, containing a central diuracil, dithymidine, or cyclobutane thymid
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Books on the topic "DNA-damaged"

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Stiffler, Kristen. Repair of damaged DNA samples for identification at crime scenes. National University, 2017.

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Damaged DNA. Winword Publishing, 2006.

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Belden, K. R., and Georgie Belden. Growing Up Belden: A Case of Damaged DNA. Independently published, 2019.

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Marcionelli, Rosanna. Repair of UV-damaged DNA by photolyase in silent chromatin of yeast. 2004.

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Sabatinos, Sarah Anne. Investigation of the function of UV damaged DNA binding protein 1 (DDB1) in mammalian systems. 2005.

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Anderson, Maureen. Damaged DNA: Get Free from Family Addictions - the Secret of Getting Free from Your Past. BookPatch LLC, The, 2017.

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Gray, Doug, Carole Proctor, and Tom Kirkwood. Biological aspects of human ageing. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0001.

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At the molecular and cellular levels human ageing is characterized by the accumulation of unrepaired random damage, and an accompanying loss of function. A major source of damage is oxidative stress caused by the generation of reactive oxygen species as a by-product of respiration. DNA and proteins are both susceptible to damage but whereas DNA damage repair systems exist, faulty proteins are generally removed by protein degradation systems. During ageing these systems become less efficient and the subsequent accumulation of damaged protein promotes protein aggregation, a process which is espe
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Douglas, Kenneth. Bioprinting. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780190943547.001.0001.

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Abstract: This book describes how bioprinting emerged from 3D printing and details the accomplishments and challenges in bioprinting tissues of cartilage, skin, bone, muscle, neuromuscular junctions, liver, heart, lung, and kidney. It explains how scientists are attempting to provide these bioprinted tissues with a blood supply and the ability to carry nerve signals so that the tissues might be used for transplantation into persons with diseased or damaged organs. The book presents all the common terms in the bioprinting field and clarifies their meaning using plain language. Readers will lear
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Wills, Christopher. Why Ecosystems Matter. Oxford University PressOxford, 2024. http://dx.doi.org/10.1093/oso/9780192887573.001.0001.

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Abstract The ecosystems of the world, the complex and resilient communities of interacting species that surround us, play a central role in our planet’s health and in our own survival. This book shows how recent insights into the many capabilities of ecosystems will enable us to understand and protect them. We will travel to remote deserts, thriving rainforests, coral reefs, and the open ocean to discover how each of their groups of fiercely competing and often cooperating species evolve and adapt to change. Laboratory experiments are now revealing the central roles played by communities of ba
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Book chapters on the topic "DNA-damaged"

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Guengerich, F. Peter. "Analysis of Damaged DNA." In Molecular Life Sciences. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-6436-5_295-1.

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Guengerich, F. Peter. "Spectroscopy of Damaged DNA." In Molecular Life Sciences. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-6436-5_410-1.

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Guengerich, Frederick Peter. "Damaged DNA, Analysis of." In Molecular Life Sciences. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-1531-2_295.

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Guengerich, Frederick Peter. "Spectroscopy of Damaged DNA." In Molecular Life Sciences. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-1531-2_410.

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Dedon, Peter C., and Marita C. Barth. "Oxidatively Damaged DNA and Inflammation." In Oxidative Damage to Nucleic Acids. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-72974-9_15.

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Reardon, J. T., and A. Sancar. "The Repair of UV-Damaged DNA." In Nucleic Acids and Molecular Biology. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84292-4_4.

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Zuo, Li, Tingyang Zhou, and Chia-Chen Chuang. "The Consequences of Damaged Mitochondrial DNA." In Mitochondrial Mechanisms of Degeneration and Repair in Parkinson's Disease. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42139-1_3.

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Ayling, Charlotte. "TA Cloning Approaches to Cloning DNA with Damaged Ends DNA." In Methods in Molecular Biology. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3004-4_5.

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Mitchell, David L. "Radioimmunoassay of DNA Damaged by Ultraviolet Light." In Technologies for Detection of DNA Damage and Mutations. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-0301-3_6.

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Laval, Jacques, Cécile Bauche, Juan Jurado, Franck Paillard, Murat Saparbaev, and Olga Sidorkina. "Repair of DNA Damaged by Free Radicals." In Advances in DNA Damage and Repair. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4865-2_19.

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Conference papers on the topic "DNA-damaged"

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Heider, Margaret R., Lixin Chen, Chen Song, et al. "Abstract 3522: Enzymatic DNA repair enables high quality library preparation and accurate sequencing from highly damaged DNA inputs." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-3522.

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"PARP1 activation directs RNA binding proteins to DNA damages to form PARG reversible compartments enriched in damaged DNA." In Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-359.

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Heider, Margaret R., Lixin Chen, Chen Song, et al. "Abstract 3522: Enzymatic DNA repair enables high quality library preparation and accurate sequencing from highly damaged DNA inputs." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-3522.

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Kunina, E. I., A. V. Shernyukov, A. V. Yudkina, D. O. Zharkov, and E. G. Bagryanskya. "DETERMINATION OF THE SPATIAL STRUCTURE OF A DNA DUPLEX WITH A NON-COMPLEMENTARY CU PAIR BY NMR SPECTROSCOPY*." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-131.

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DNA molecules are constantly being damaged. In human cells the amount of damage can reach several thousand per cell per day. This damage includes changes in the chemical structure of DNA, chain breakage or the presence of incorrect bases or their fragments. However, cells have repair systems to help repair these damages. The study of processes modeling DNA repair is an urgent task, since DNA damage is the cause of many diseases. DNA structures with AP sites can be used to develop drugs to treat tumors and other conditions.
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Hirono, Yuriko, Ayaka Kawazoe, Eri Shigeyoshi, et al. "The Inhibitory Mechanism Of Apoptosis In DNA Damaged-Alveolar Macrophages By Cigarette Smoke." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4539.

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"Nucleotide excision repair proteins and PARP1/PAR interplay regulats protein assembly on damaged DNA." In Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-366.

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Cooper, S., R. Jankowiak, and G. J. Small. "Fluorescence line narrowing and high resolution analysis of DNA and globin damage from carcinogens." In International Laser Science Conference. Optica Publishing Group, 1986. http://dx.doi.org/10.1364/ils.1986.tue7.

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Approximately 70% of cancers are believed to be initiated by genotoxic agents. As enzymatic repair of damaged DNA is very body specific, body burden and fundamental studies of chemical carcinogenesis require the development of new bioanalytical techniques which can directly determine damage of intact DNA from carcinogenic matabolites. Assessment of damage at a level of ~1:109 base pairs is required for ~10-100 μg of DNA. To be most useful the technique should possess the selectivity to distinguish between structurally very similar DNA-metabolite adducts. In both regards, laser-excited fluoresc
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Bernardini, Michele, Alessandro Ferri, Lucia Migliorelli, et al. "Augmented Microscopy for DNA Damage Quantification: A Machine Learning Tool for Environmental, Medical and Health Sciences." In ASME 2019 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/detc2019-97902.

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Abstract The Comet Assay is a well-known procedure employed to investigate the DNA damage and can be applied to several research areas such as environmental, medical and health sciences. User dependency and computation time effort represent some of the major drawbacks of the Comet Assay. Starting from this motivation, we applied a Machine Learning (ML) tool for discriminating DNA damage using a standard hand-crafted feature set. The experimental results demonstrate how the ML tool is able to objectively replicate human experts scoring (accuracy detection up to 92%) by solving the related binar
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Lowe, Dana J. Ruminski, Deepika Philkana, Catherine Huang, Matthew G. Butler, Omoshile Clement, and Bharathi Anekella. "Abstract 1322: Development and performance of a formalin-damaged multiplexed DNA tumor mutation FFPE reference material." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-1322.

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Petrović, Sandra, Bojana Laban, Tatjana Momić, and Andreja Leskovac. "EFFECTS OF Phytolacca dioica EXTRACT ON IONIZING RADIATION-INDUCED DAMAGE IN VITRO." In 8th Workshop Food and Drug Safety and Qualit. Vinča Institute of Nuclear Sciences - National Institute of the Republic of Serbia, 2024. http://dx.doi.org/10.46793/8fdsq.pc5sp.

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This pilot study aimed to examine the impact of Phytolacca dioica on ionizing radiation-induced DNA damage and lipid peroxidation using human peripheral blood lymphocytes as a model system. The results showed that P. dioica possesses neither antioxidant nor genoprotective effects in radiolytically damaged tissue. Its potential use as a radiosensitizer should be further examined.
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Reports on the topic "DNA-damaged"

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Prakash, L. Repair of uv damaged DNA: Genes and proteins of yeast and human. Office of Scientific and Technical Information (OSTI), 1992. http://dx.doi.org/10.2172/5400958.

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Louisek Prakash. Repair of DNA damaged by ionizing radiation and other oxidative agents in yeast and human. Office of Scientific and Technical Information (OSTI), 2000. http://dx.doi.org/10.2172/807355.

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Louise Prakash. Repair of DNA damaged by ionizing radiation and other oxidative agents in yeast and human. Office of Scientific and Technical Information (OSTI), 2000. http://dx.doi.org/10.2172/807356.

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Prakash, L. Repair of uv damaged DNA: Genes and proteins of yeast and human. Progress report, November 1, 1991--April 15, 1992. Office of Scientific and Technical Information (OSTI), 1992. http://dx.doi.org/10.2172/10141131.

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Christopher, David A., and Avihai Danon. Plant Adaptation to Light Stress: Genetic Regulatory Mechanisms. United States Department of Agriculture, 2004. http://dx.doi.org/10.32747/2004.7586534.bard.

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Original Objectives: 1. Purify and biochemically characterize RB60 orthologs in higher plant chloroplasts; 2. Clone the gene(s) encoding plant RB60 orthologs and determine their structure and expression; 3. Manipulate the expression of RB60; 4. Assay the effects of altered RB60 expression on thylakoid biogenesis and photosynthetic function in plants exposed to different light conditions. In addition, we also examined the gene structure and expression of RB60 orthologs in the non-vascular plant, Physcomitrella patens and cloned the poly(A)-binding protein orthologue (43 kDa RB47-like protein).
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