Log in

Relevant bibliographies by topics / DNA-damaged / Journal articles

To see the other types of publications on this topic, follow the link: DNA-damaged.

Journal articles on the topic 'DNA-damaged'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'DNA-damaged.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Lehmann, Alan R. "Replication of Damaged DNA." Cell Cycle 2, no. 4 (2003): 299–301. http://dx.doi.org/10.4161/cc.2.4.407.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Caetano, R. A., and P. A. Schulz. "Delocalized states in damaged DNA." Brazilian Journal of Physics 36, no. 2a (2006): 459–61. http://dx.doi.org/10.1590/s0103-97332006000300061.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Chatterjee, N., and W. Siede. "Replicating Damaged DNA in Eukaryotes." Cold Spring Harbor Perspectives in Biology 5, no. 12 (2013): a019836. http://dx.doi.org/10.1101/cshperspect.a019836.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Lukin, Mark, and Carlos de los Santos. "NMR Structures of Damaged DNA." Chemical Reviews 106, no. 2 (2006): 607–86. http://dx.doi.org/10.1021/cr0404646.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Inoki, Taeko, Hitoshi Endo, Yutaka Inoki, et al. "Damaged DNA-binding protein 2 accelerates UV-damaged DNA repair in human corneal endothelium." Experimental Eye Research 79, no. 3 (2004): 367–76. http://dx.doi.org/10.1016/j.exer.2004.06.010.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Yeh, J. I., A. S. Levine, S. Du, et al. "Damaged DNA induced UV-damaged DNA-binding protein (UV-DDB) dimerization and its roles in chromatinized DNA repair." Proceedings of the National Academy of Sciences 109, no. 41 (2012): E2737—E2746. http://dx.doi.org/10.1073/pnas.1110067109.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Lanuszewska, J., A. Cudak, J. Rzeszowska-Wolny, and P. Widłak. "Detection of damage-recognition proteins from human lymphocytes." Acta Biochimica Polonica 47, no. 2 (2000): 443–50. http://dx.doi.org/10.18388/abp.2000_4024.

Full text

Abstract:

Proteins recognizing and binding to damaged DNA (DDB-proteins) were analyzed in human lymphocytes obtained from healthy donors. Using an electrophoretic mobility shift assay several complexes between nuclear extract proteins and damaged DNA were detected: a complex specific for DNA damaged by N-acetoxy-N-acetylaminofluorene, another complex specific for UV-irradiated DNA, and two complexes specific for DNA damaged by cis-dichlorodiammine platinum. All the detected complexes differed in electrophoretic mobility and possibly contained different proteins. Complexes specific for free DNA ends were

APA, Harvard, Vancouver, ISO, and other styles

8

Rzeszowska-Wolny, J., and P. Widłak. "Damaged DNA-binding proteins: recognition of N-acetoxy-acetylaminofluorene-induced DNA adducts." Acta Biochimica Polonica 46, no. 1 (1999): 173–80. http://dx.doi.org/10.18388/abp.1999_4195.

Full text

Abstract:

Proteins which bind to the DNA damaged by genotoxic agents can be detected in all living organisms. Damage-recognition proteins are thought to be generally involved in DNA repair mechanisms. On the other hand, the relevance to DNA repair of some other proteins which show elevated affinity to damaged DNA (e.g. HMG-box containing proteins or histone H1) has not been established. Using the electrophoretic mobility-shift assay we have investigated damage-recognition proteins in nuclei from rat hepatocytes. We detected two different protein complexes which preferentially bound the DNA damaged by N-

APA, Harvard, Vancouver, ISO, and other styles

9

Strickland, Paul T., and Cristl Gentile. "Separation of carcinogen-damaged DNA fragments from undamaged DNA." Nucleic Acids Research 19, no. 24 (1991): 6955. http://dx.doi.org/10.1093/nar/19.24.6955.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Williams, Loren Dean, and Qi Gao. "DNA-ditercalinium interactions: implications for recognition of damaged DNA." Biochemistry 31, no. 17 (1992): 4315–24. http://dx.doi.org/10.1021/bi00132a024.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Friedman, Joshua I., and James T. Stivers. "Detection of Damaged DNA Bases by DNA Glycosylase Enzymes." Biochemistry 49, no. 24 (2010): 4957–67. http://dx.doi.org/10.1021/bi100593a.

Full text

APA, Harvard, Vancouver, ISO, and other styles

12

Lindahl, T. "Recognition and processing of damaged DNA." Journal of Cell Science 1995, Supplement 19 (1995): 73–77. http://dx.doi.org/10.1242/jcs.1995.supplement_19.10.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Wetzel, Cynthia C., and Steven J. Berberich. "p53 binds to cisplatin-damaged DNA." Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression 1517, no. 3 (2001): 392–97. http://dx.doi.org/10.1016/s0167-4781(00)00305-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

14

Rossner, Pavel, and Radim J. Sram. "Immunochemical detection of oxidatively damaged DNA." Free Radical Research 46, no. 4 (2011): 492–522. http://dx.doi.org/10.3109/10715762.2011.632415.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Carell, Thomas. "Sunlight-Damaged DNA Repaired with Sunlight." Angewandte Chemie International Edition in English 34, no. 22 (1995): 2491–94. http://dx.doi.org/10.1002/anie.199524911.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Ahmadi, Ali, and Soon-Chye Ng. "Fertilizing ability of DNA-damaged spermatozoa." Journal of Experimental Zoology 284, no. 6 (1999): 696–704. http://dx.doi.org/10.1002/(sici)1097-010x(19991101)284:6<696::aid-jez11>3.0.co;2-e.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Bagchi, Srilata, and Pradip Raychaudhuri. "Damaged-DNA Binding Protein-2 Drives Apoptosis Following DNA Damage." Cell Division 5, no. 1 (2010): 3. http://dx.doi.org/10.1186/1747-1028-5-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

18

Koehler, D. R., and P. C. Hanawalt. "Digestion of damaged DNA by the T7 DNA polymerase-exonuclease." Biochemical Journal 293, no. 2 (1993): 451–53. http://dx.doi.org/10.1042/bj2930451.

Full text

Abstract:

We have investigated the 3′-5′-exonuclease activity of phage T7 DNA polymerase for its usefulness as an approach for the detection of lesions in DNA. Unlike the T4 DNA polymerase-exonuclease, which is commonly used to map the position and frequency of lesions in very small DNA fragments, T7 DNA polymerase-exonuclease is able to hydrolyse almost completely the large fragments from KpnI-restricted mammalian DNA. However, we found that the exonuclease was also able to hydrolyse DNA containing several kinds of lesions: cyclobutane pyrimidine dimers, thymine glycols, and mono-adducts of 4′-hydroxym

APA, Harvard, Vancouver, ISO, and other styles

19

Ramadan, Kristijan, Igor V. Shevelev, Giovanni Maga та Ulrich Hübscher. "DNA Polymerase λ from Calf Thymus Preferentially Replicates Damaged DNA". Journal of Biological Chemistry 277, № 21 (2002): 18454–58. http://dx.doi.org/10.1074/jbc.m200421200.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Villani, Giuseppe, and Nicolas Tanguy Le Gac. "Interactions of DNA Helicases with Damaged DNA: Possible Biological Consequences." Journal of Biological Chemistry 275, no. 43 (2000): 33185–88. http://dx.doi.org/10.1074/jbc.r000011200.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

Hoelzer, M. A., and R. E. Michod. "DNA repair and the evolution of transformation in Bacillus subtilis. III. Sex with damaged DNA." Genetics 128, no. 2 (1991): 215–23. http://dx.doi.org/10.1093/genetics/128.2.215.

Full text

Abstract:

Abstract Natural genetic transformation in the bacterium Bacillus subtilis provides an experimental system for studying the evolutionary function of sexual recombination. The repair hypothesis proposes that during transformation the exogenous DNA taken up by cells is used as template for recombinational repair of damages in the recipient cell's genome. Earlier results demonstrated that the population density of transformed cells (i.e., sexual cells) increases, relative to nontransformed cells (primarily asexual cells), with increasing dosage of ultraviolet irradiation, provided that the cells

APA, Harvard, Vancouver, ISO, and other styles

22

Pietrowska, Monika, and Piotr Widłak. "Characterization of a novel protein that specifically binds to DNA modified by N-acetoxy-acetylaminofluorene and cis-diamminedichloroplatinum." Acta Biochimica Polonica 52, no. 4 (2005): 867–74. http://dx.doi.org/10.18388/abp.2005_3400.

Full text

Abstract:

Proteins recognizing DNA damaged by the chemical carcinogen N-acetoxy-acetylaminofluorene (AAAF) were analyzed in nuclear extracts from rat tissues, using a 36 bp oligonucleotide as a substrate and electrophoretic mobility shift and Southwestern blot assays. One major damage-recognizing protein was detected, whose amount was estimated as at least 10(5) copies per cell. Levels of this protein were similar in extracts from brain, kidney and liver, but much lower in extracts from testis. The affinity of the detected protein for DNA damaged by AAAF was about 70-fold higher than for undamaged DNA.

APA, Harvard, Vancouver, ISO, and other styles

23

Gebel, Jakob, Marcel Tuppi, Nicole Sänger, Björn Schumacher, and Volker Dötsch. "DNA Damaged Induced Cell Death in Oocytes." Molecules 25, no. 23 (2020): 5714. http://dx.doi.org/10.3390/molecules25235714.

Full text

Abstract:

The production of haploid gametes through meiosis is central to the principle of sexual reproduction. The genetic diversity is further enhanced by exchange of genetic material between homologous chromosomes by the crossover mechanism. This mechanism not only requires correct pairing of homologous chromosomes but also efficient repair of the induced DNA double-strand breaks. Oocytes have evolved a unique quality control system that eliminates cells if chromosomes do not correctly align or if DNA repair is not possible. Central to this monitoring system that is conserved from nematodes and fruit

APA, Harvard, Vancouver, ISO, and other styles

24

Pearlman, D., Holbrook, D. Pirkle, and S. Kim. "Molecular models for DNA damaged by photoreaction." Science 227, no. 4692 (1985): 1304–8. http://dx.doi.org/10.1126/science.3975615.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Tsuge, Maasa, Yusuke Masuda, Hidenori Kaneoka, Shunsuke Kidani, Katsuhide Miyake, and Shinji Iijima. "SUMOylation of damaged DNA-binding protein DDB2." Biochemical and Biophysical Research Communications 438, no. 1 (2013): 26–31. http://dx.doi.org/10.1016/j.bbrc.2013.07.013.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Sharova, N. P. "How does a cell repair damaged DNA?" Biochemistry (Moscow) 70, no. 3 (2005): 275–91. http://dx.doi.org/10.1007/s10541-005-0113-4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Hacohen, Nir, and Yuk Yuen Lan. "Damaged DNA marching out of aging nucleus." Aging 11, no. 19 (2019): 8039–40. http://dx.doi.org/10.18632/aging.102340.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Georgiades, Emily, Nicola Crosetto, and Magda Bienko. "Compartmentalizing damaged DNA: A double-edged sword." Molecular Cell 84, no. 1 (2024): 12–13. http://dx.doi.org/10.1016/j.molcel.2023.12.001.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

Widłak, P. "Reconstitution of UV-damaged DNA into chromatin using Xenopus oocyte extracts." Acta Biochimica Polonica 45, no. 2 (1998): 595–603. http://dx.doi.org/10.18388/abp.1998_4252.

Full text

Abstract:

Chromatin was reconstituted in vitro using Xenopus oocyte extracts and plasmid DNA containing UV radiation-induced damage. Damaged DNA was assembled into minichromosomes with an efficiency similar to that of control, non-irradiated DNA. Oocyte extracts were competent to carry out DNA repair, which was elicited by nicking damaged templates followed by DNA synthesis during chromatin assembly. Newly synthesized DNA was efficiently reconstituted into nucleosomes.

APA, Harvard, Vancouver, ISO, and other styles

30

Ide, Hiroshi, and Mitsuharu Kotera. "Human DNA Glycosylases Involved in the Repair of Oxidatively Damaged DNA." Biological & Pharmaceutical Bulletin 27, no. 4 (2004): 480–85. http://dx.doi.org/10.1248/bpb.27.480.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Shiels, Jerome C., Bozidar Jerkovic, Anne M. Baranger, and Philip H. Bolton. "RNA–DNA Hybrids Containing Damaged DNA are Substrates for RNase H." Bioorganic & Medicinal Chemistry Letters 11, no. 19 (2001): 2623–26. http://dx.doi.org/10.1016/s0960-894x(01)00527-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

PROTIĆ, MIROSLAVA. "Eukaryotic Damaged DNA-Binding Proteins: DNA Repair Proteins or Transcription Factors?" Annals of the New York Academy of Sciences 726, no. 1 DNA Damage (1994): 333–35. http://dx.doi.org/10.1111/j.1749-6632.1994.tb52843.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

Gordienko, I. "UvrAB activity at a damaged DNA site: is unpaired DNA present?" EMBO Journal 16, no. 4 (1997): 880–88. http://dx.doi.org/10.1093/emboj/16.4.880.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

Raja, Sripriya J., and Bennett Van Houten. "UV-DDB as a General Sensor of DNA Damage in Chromatin: Multifaceted Approaches to Assess Its Direct Role in Base Excision Repair." International Journal of Molecular Sciences 24, no. 12 (2023): 10168. http://dx.doi.org/10.3390/ijms241210168.

Full text

Abstract:

Base excision repair (BER) is a cellular process that removes damaged bases arising from exogenous and endogenous sources including reactive oxygen species, alkylation agents, and ionizing radiation. BER is mediated by the actions of multiple proteins which work in a highly concerted manner to resolve DNA damage efficiently to prevent toxic repair intermediates. During the initiation of BER, the damaged base is removed by one of 11 mammalian DNA glycosylases, resulting in abasic sites. Many DNA glycosylases are product-inhibited by binding to the abasic site more avidly than the damaged base.

APA, Harvard, Vancouver, ISO, and other styles

35

Helland, D. E., P. W. Doetsch, and W. A. Haseltine. "Substrate specificity of a mammalian DNA repair endonuclease that recognizes oxidative base damage." Molecular and Cellular Biology 6, no. 6 (1986): 1983–90. http://dx.doi.org/10.1128/mcb.6.6.1983-1990.1986.

Full text

Abstract:

The substrate specificity of a calf thymus endonuclease on DNA damaged by UV ligh, ionizing radiation, and oxidizing agents was investigated. End-labeled DNA fragments of defined sequence were used as substrates, and the enzyme-generated scission products were analyzed by using DNA sequencing methodologies. The enzyme was shown to incise damaged DNA at pyrimidine sites. The enzyme incised DNA damaged with UV light, ionizing radiation, osmium tetroxide, potassium permanganate, and hydrogen peroxide at cytosine and thymine sites. The substrate specificity of the calf thymus endonuclease was comp

APA, Harvard, Vancouver, ISO, and other styles

36

Helland, D. E., P. W. Doetsch, and W. A. Haseltine. "Substrate specificity of a mammalian DNA repair endonuclease that recognizes oxidative base damage." Molecular and Cellular Biology 6, no. 6 (1986): 1983–90. http://dx.doi.org/10.1128/mcb.6.6.1983.

Full text

Abstract:

The substrate specificity of a calf thymus endonuclease on DNA damaged by UV ligh, ionizing radiation, and oxidizing agents was investigated. End-labeled DNA fragments of defined sequence were used as substrates, and the enzyme-generated scission products were analyzed by using DNA sequencing methodologies. The enzyme was shown to incise damaged DNA at pyrimidine sites. The enzyme incised DNA damaged with UV light, ionizing radiation, osmium tetroxide, potassium permanganate, and hydrogen peroxide at cytosine and thymine sites. The substrate specificity of the calf thymus endonuclease was comp

APA, Harvard, Vancouver, ISO, and other styles

37

Mitra, Manu. "DNA Repairing and its Mechanism in the Cell." ACTA Scientific Medical Sciences 3, no. 8 (2019): 116–19. https://doi.org/10.5281/zenodo.3338556.

Full text

Abstract:

DNA (Deoxyribonucleic Acid) repair denotes collection of processes by which a cell identifies and corrects the damage to the DNA molecules that encode its genome. Several incisions causes structural damage to the DNA molecule and may alter or eliminate the cell’s ability to transcribe the gene that are affected DNA encodes. There are many techniques and methods to repair DNA, however, in this paper few methods are reviewed. For instance – Mechanism for repairing damaged DNA, Novel technique to repair damaged DNA, Scientist confirm DNA repair, Repairing faulty genes to cure diseases

APA, Harvard, Vancouver, ISO, and other styles

38

Peng, Aimin, Andrea L. Lewellyn, and James L. Maller. "Undamaged DNA Transmits and Enhances DNA Damage Checkpoint Signals in Early Embryos." Molecular and Cellular Biology 27, no. 19 (2007): 6852–62. http://dx.doi.org/10.1128/mcb.00195-07.

Full text

Abstract:

ABSTRACT In Xenopus laevis embryos, the midblastula transition (MBT) at the 12th cell division marks initiation of critical developmental events, including zygotic transcription and the abrupt inclusion of gap phases into the cell cycle. Interestingly, although an ionizing radiation-induced checkpoint response is absent in pre-MBT embryos, introduction of a threshold amount of undamaged plasmid or sperm DNA allows a DNA damage checkpoint response to be activated. We show here that undamaged threshold DNA directly participates in checkpoint signaling, as judged by several dynamic changes, inclu

APA, Harvard, Vancouver, ISO, and other styles

39

Hayes, Steven, Pavel Shiyanov, Xiaoqun Chen, and Pradip Raychaudhuri. "DDB, a Putative DNA Repair Protein, Can Function as a Transcriptional Partner of E2F1." Molecular and Cellular Biology 18, no. 1 (1998): 240–49. http://dx.doi.org/10.1128/mcb.18.1.240.

Full text

Abstract:

ABSTRACT The transcription factor E2F1 is believed to be involved in the regulated expression of the DNA replication genes. To gain insights into the transcriptional activation function of E2F1, we looked for proteins in HeLa nuclear extracts that bind to the activation domain of E2F1. Here we show that DDB, a putative DNA repair protein, associates with the activation domain of E2F1. DDB was identified as a heterodimeric protein (48 and 127 kDa) that binds to UV-damaged DNA. We show that the UV-damaged-DNA binding activity from HeLa nuclear extracts can associate with the activation domain of

APA, Harvard, Vancouver, ISO, and other styles

40

Stokes, Matthew P., Ruth Van Hatten, Howard D. Lindsay, and W. Matthew Michael. "DNA replication is required for the checkpoint response to damaged DNA in Xenopus egg extracts." Journal of Cell Biology 158, no. 5 (2002): 863–72. http://dx.doi.org/10.1083/jcb.200204127.

Full text

Abstract:

Alkylating agents, such as methyl methanesulfonate (MMS), damage DNA and activate the DNA damage checkpoint. Although many of the checkpoint proteins that transduce damage signals have been identified and characterized, the mechanism that senses the damage and activates the checkpoint is not yet understood. To address this issue for alkylation damage, we have reconstituted the checkpoint response to MMS in Xenopus egg extracts. Using four different indicators for checkpoint activation (delay on entrance into mitosis, slowing of DNA replication, phosphorylation of the Chk1 protein, and physical

APA, Harvard, Vancouver, ISO, and other styles

41

Doherty, Rachel, and Srinivasan Madhusudan. "DNA Repair Endonucleases." Journal of Biomolecular Screening 20, no. 7 (2015): 829–41. http://dx.doi.org/10.1177/1087057115581581.

Full text

Abstract:

Genomic DNA is constantly exposed to endogenous and exogenous damaging agents. To overcome these damaging effects and maintain genomic stability, cells have robust coping mechanisms in place, including repair of the damaged DNA. There are a number of DNA repair pathways available to cells dependent on the type of damage induced. The removal of damaged DNA is essential to allow successful repair. Removal of DNA strands is achieved by nucleases. Exonucleases are those that progressively cut from DNA ends, and endonucleases make single incisions within strands of DNA. This review focuses on the g

APA, Harvard, Vancouver, ISO, and other styles

42

Baran, Vladimír, and Jozef Pisko. "Cleavage of Early Mouse Embryo with Damaged DNA." International Journal of Molecular Sciences 23, no. 7 (2022): 3516. http://dx.doi.org/10.3390/ijms23073516.

Full text

Abstract:

The preimplantation period of embryogenesis is crucial during mammalian ontogenesis. During this period, the mitotic cycles are initiated, the embryonic genome is activated, and the primary differentiation of embryonic cells occurs. All cellular abnormalities occurring in this period are the primary cause of fetal developmental disorders. DNA damage is a serious cause of developmental failure. In the context of DNA damage response on the cellular level, we analyzed the course of embryogenesis and phenotypic changes during the cleavage of a preimplantation embryo. Our results document that DNA

APA, Harvard, Vancouver, ISO, and other styles

43

Yasuda, Gentaro, Ryotaro Nishi, Eriko Watanabe, et al. "In Vivo Destabilization and Functional Defects of the Xeroderma Pigmentosum C Protein Caused by a Pathogenic Missense Mutation." Molecular and Cellular Biology 27, no. 19 (2007): 6606–14. http://dx.doi.org/10.1128/mcb.02166-06.

Full text

Abstract:

ABSTRACT Xeroderma pigmentosum group C (XPC) protein plays an essential role in DNA damage recognition in mammalian global genome nucleotide excision repair (NER). Here, we analyze the functional basis of NER inactivation caused by a single amino acid substitution (Trp to Ser at position 690) in XPC, previously identified in the XPC patient XP13PV. The Trp690Ser change dramatically affects the in vivo stability of the XPC protein, thereby causing a significant reduction of its steady-state level in XP13PV fibroblasts. Despite normal heterotrimeric complex formation and physical interactions wi

APA, Harvard, Vancouver, ISO, and other styles

44

Rachofsky, Edward, J. B. Ross, and Roman Osman. "Conformation and Dynamics of Normal and Damaged DNA." Combinatorial Chemistry & High Throughput Screening 4, no. 8 (2001): 675–706. http://dx.doi.org/10.2174/1386207013330706.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

Møller, Peter, Pernille Høgh Danielsen, Kim Jantzen, Martin Roursgaard, and Steffen Loft. "Oxidatively damaged DNA in animals exposed to particles." Critical Reviews in Toxicology 43, no. 2 (2013): 96–118. http://dx.doi.org/10.3109/10408444.2012.756456.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Zitka, Ondrej, Sona Krizkova, Sylvie Skalickova, et al. "Electrochemical Study of DNA Damaged by Oxidation Stress." Combinatorial Chemistry & High Throughput Screening 16, no. 2 (2013): 130–41. http://dx.doi.org/10.2174/138620713804806274.

Full text

APA, Harvard, Vancouver, ISO, and other styles

47

Zitka, Ondrej, Sona Krizkova, Sylvie Skalickova, et al. "Electrochemical Study of DNA Damaged by Oxidation Stress." Combinatorial Chemistry & High Throughput Screening 16, no. 2 (2013): 130–41. http://dx.doi.org/10.2174/1386207311316020007.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Hirai, T., T. Torizawa, K. Kato, O. Nikaido, E. Otsuka, and I. Shimada. "Nobel antibody with UV-damaged DNA cleaving activity." Seibutsu Butsuri 41, supplement (2001): S99. http://dx.doi.org/10.2142/biophys.41.s99_2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Damsma, Gerke E., Aaron Alt, Florian Brueckner, Thomas Carell, and Patrick Cramer. "Mechanism of transcriptional stalling at cisplatin-damaged DNA." Nature Structural & Molecular Biology 14, no. 12 (2007): 1127–33. http://dx.doi.org/10.1038/nsmb1314.

Full text

APA, Harvard, Vancouver, ISO, and other styles

50

Jung, Yongwon, and Stephen J. Lippard. "RNA Polymerase II Blockage by Cisplatin-damaged DNA." Journal of Biological Chemistry 281, no. 3 (2005): 1361–70. http://dx.doi.org/10.1074/jbc.m509688200.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!