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Journal articles on the topic 'Doberman pinschers'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 January 2023

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1

Thornburg, L. P. "Histomorphological and Immunohistochemical Studies of Chronic Active Hepatitis in Doberman Pinschers." Veterinary Pathology 35, no. 5 (September 1998): 380–85. http://dx.doi.org/10.1177/030098589803500507.

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Liver tissue samples were reviewed from 35 Doberman Pinschers with chronic active hepatitis in the precirrhotic stage. Thirty dogs had elevated hepatic copper concentrations, and five had normal liver copper concentrations. The earliest changes were inflammation and scar tissue deposition around the small hepatic vein branches. There was also apoptosis of scattered hepatocytes in zone 3. Inflammation consisted of macrophages, lymphocytes, and plasma cells. As the disease progressed, collagen deposition increased around the hepatic veins; in some liver specimens, thin scar tissue septa radiated from the hepatic vein branches, and inflammation spread to include the portal tracts. The sinusoids adjacent to the scar tissue were converted to endothelial-lined, thin-walled vessels. Chronic active hepatitis (commonly referred to as Doberman hepatitis or chronic active hepatitis of Dobermans) is a progressive fibrosis, inflammation and hepatocyte loss beginning among zone 3 hepatocytes around the terminal hepatic vein branches. The histomorphologic changes were the same among those Dobermans with elevated hepatic copper and those with normal hepatic copper. The cause was not determined, but these morphologic studies support the idea of immune-mediated disease.

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2

Ramsey, DT, KL Ketring, MB Glaze, B. Knight, and JA Render. "Ligneous conjunctivitis in four Doberman pinschers." Journal of the American Animal Hospital Association 32, no. 5 (September 1, 1996): 439–47. http://dx.doi.org/10.5326/15473317-32-5-439.

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Ligneous conjunctivitis (LC) was diagnosed in four unrelated Doberman pinschers. Thick, opaque membranes of the palpebral conjunctivae and nictitating membranes were present bilaterally. Three dogs had concurrent signs of multisystemic disease. A thick, amorphous, eosinophilic, hyaline-like material in the substantia propria of the conjunctiva--containing a moderate, mononuclear cell infiltrate--was evident on histological examination. A predominance of T lymphocytes, few macrophages, and weak positive staining for immunoglobulin G (IgG) and immunoglobulin A (IgA) were evident by immunohistochemical staining. The clinical and histological appearance of LC in Doberman pinschers and humans is similar.

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3

Vollmar, Andrea, Philip R. Fox, Kathryn M. Meurs, and Si-Kwang Liu. "Dilated Cardiomyopathy in Juvenile Doberman Pinschers." Journal of Veterinary Cardiology 5, no. 1 (May 2003): 23–27. http://dx.doi.org/10.1016/s1760-2734(06)70041-8.

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4

Lynne O'Sullivan, M., Michael R. O'Grady, and Sandra L. Minors. "Assessment of Diastolic Function by Doppler Echocardiography in Normal Doberman Pinschers and Doberman Pinschers with Dilated Cardiomyopathy." Journal of Veterinary Internal Medicine 21, no. 1 (2007): 81. http://dx.doi.org/10.1892/0891-6640(2007)21[81:aodfbd]2.0.co;2.

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5

O'Sullivan, M. Lynne, Michael R. O'Grady, and Sandra L. Minors. "Assessment of Diastolic Function by Doppler Echocardiography in Normal Doberman Pinschers and Doberman Pinschers with Dilated Cardiomyopathy." Journal of Veterinary Internal Medicine 21, no. 1 (January 2007): 81–91. http://dx.doi.org/10.1111/j.1939-1676.2007.tb02932.x.

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6

Crespi, Julian A., Laura S. Barrientos, and Guillermo Giovambattista. "von Willebrand disease type 1 in Doberman Pinscher dogs: genotyping and prevalence of the mutation in the Buenos Aires region, Argentina." Journal of Veterinary Diagnostic Investigation 30, no. 2 (December 22, 2017): 310–14. http://dx.doi.org/10.1177/1040638717750429.

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von Willebrand disease (vWD) is the most common inherited coagulopathy in dogs, particularly in Doberman Pinschers. We developed a pyrosequencing-based assay to estimate the frequency of the c.7437G>A mutation associated with vWD type 1 in the Doberman Pinscher population of Buenos Aires, Argentina. We found a 0.41 frequency for the mutated allele, which varied significantly within families (family 1 = 0.43, family 2 = 0.58, unrelated animals = 0.35). The use of a popular founder male carrier of mutant allele A increased vWD incidence within a family and in the general population. The mode of inheritance was confirmed as autosomal dominant with incomplete penetrance. No differences were found between sexes and coat colors. Pyrosequencing was a good complement to clinical and coagulation tests for vWD type 1 diagnosis and a useful alternative for detecting the c.7437G>A mutation.

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7

Moon-Fanelli, Alice A., Nicholas H. Dodman, and Nicole Cottam. "Blanket and flank sucking in Doberman Pinschers." Journal of the American Veterinary Medical Association 231, no. 6 (September 2007): 907–12. http://dx.doi.org/10.2460/javma.231.6.907.

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8

Mandigers, P. J. J., T. S. G. A. M. van den Ingh, B. Spee, L. C. Penning, P. Bode, and J. Rothuizen. "Chronic hepatitis in Doberman pinschers. A review." Veterinary Quarterly 26, no. 3 (September 2004): 98–106. http://dx.doi.org/10.1080/01652176.2004.9695173.

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9

Calvert, Clay A., Gilbert J. Jacobs, Marc Kraus, and John Brown. "Signal-Averaged Electrocardiograms in Normal Doberman Pinschers." Journal of Veterinary Internal Medicine 12, no. 5 (September 1998): 355–64. http://dx.doi.org/10.1111/j.1939-1676.1998.tb02135.x.

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10

Jeffery, N. D. "Femoral Head and Neck Excision Complicated by Ischiatic Nerve Entrapment in Two Dogs." Veterinary and Comparative Orthopaedics and Traumatology 06, no. 04 (1993): 215–18. http://dx.doi.org/10.1055/s-0038-1633062.

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SummaryTwo dogs, both Doberman n pinschers, developed ischiatic nerve palsy following femoral head and neck excisions. Each had undergone surgery entailing the incorporation of a muscle flap (in one case deep gluteal, in the other biceps femoris) to reduce bone-on-bone contact after ostectomy. At later exploratory surgery, the ischiatic nerve was found to be entrapped by muscle in the ischiatic fossa, caudal to the greater trochanter. Partial excision of the ischium relieved the compression, allowing rapid recovery of function. The complication of ischiatic nerve entrapment may warrant consideration when contemplating femoral head and neck excision in cases in which the greater trochanter and ischial tuberosity are abnormally close.Ischiatic nerve palsy developed several weeks after femoral head and neck excision in two Dobermann pinschers. Both dogs had undergone surgery involving muscle sling placement. The ischiatic nerve was found to be entrapped in muscle in each case. Resection of portions of the ischium allowed rapid recovery of function.

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11

Wess, G., J. Simak, M. Mahling, and K. Hartmann. "Cardiac Troponin I in Doberman Pinschers with Cardiomyopathy." Journal of Veterinary Internal Medicine 24, no. 4 (April 16, 2010): 843–49. http://dx.doi.org/10.1111/j.1939-1676.2010.0516.x.

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12

Poppenga, Robert H., Allan L. Trapp, W. Emmett Braselton, Calvert G. Louden, John M. Gumbs, and James B. Dalley. "Hexachlorophene Toxicosis in a Litter of Doberman Pinschers." Journal of Veterinary Diagnostic Investigation 2, no. 2 (April 1990): 129–31. http://dx.doi.org/10.1177/104063879000200209.

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13

Lynne O'Sullivan, M., Michael R. O'Grady, and Sandra L. Minors. "Plasma Big Endothelin-1, Atrial Natriuretic Peptide, Aldosterone, and Norepinephrine Concentrations in Normal Doberman Pinschers and Doberman Pinschers with dilated Cardiomyopathy." Journal of Veterinary Internal Medicine 21, no. 1 (2007): 92. http://dx.doi.org/10.1892/0891-6640(2007)21[92:pbeanp]2.0.co;2.

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14

O'Sullivan, M. Lynne, Michael R. O'Grady, and Sandra L. Minors. "Plasma Big Endothelin-1, Atrial Natriuretic Peptide, Aldosterone, and Norepinephrine Concentrations in Normal Doberman Pinschers and Doberman Pinschers with Dilated Cardiomyopathy." Journal of Veterinary Internal Medicine 21, no. 1 (January 2007): 92–99. http://dx.doi.org/10.1111/j.1939-1676.2007.tb02933.x.

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15

Smith, Caren E. "Plasma fatty acid concentrations in Boxers and Doberman Pinschers." Journal of the American Veterinary Medical Association 232, no. 4 (February 15, 2008): 563. http://dx.doi.org/10.2460/javma.232.4.563.

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16

Smith, Caren E., Lisa M. Freeman, Kathryn M. Meurs, John E. Rush, and Allison Lamb. "Plasma fatty acid concentrations in Boxers and Doberman Pinschers." American Journal of Veterinary Research 69, no. 2 (February 2008): 195–98. http://dx.doi.org/10.2460/ajvr.69.2.195.

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17

MILLER, WILLIAM H. "Follicular Dysplasia in Adult Black and Red Doberman Pinschers." Veterinary Dermatology 1, no. 4 (December 1990): 181–87. http://dx.doi.org/10.1111/j.1365-3164.1990.tb00098.x.

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18

Calvert, Clay A., and John Brown. "Influence of Antiarrhythmia Therapy on Survival Times of 19 Clinically Healthy Doberman Pinschers With Dilated Cardiomyopathy That Experienced Syncope, Ventricular Tachycardia, and Sudden Death (1985–1998)." Journal of the American Animal Hospital Association 40, no. 1 (January 1, 2004): 24–28. http://dx.doi.org/10.5326/0400024.

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Overtly healthy Doberman pinschers, having moderate to severe myocardial failure secondary to dilated cardiomyopathy, which experienced ventricular tachycardia, syncope or collapse, and sudden death were studied to determine the effect of antiarrhythmic medication on their clinical outcome. Antiarrhythmia drug therapy may have retarded sudden death in 13 treated dogs compared to the six dogs not administered antiarrhythmia drugs.

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19

Ogata, Niwako, Timothy E. Gillis, Xiaoxu Liu, Suzanne M. Cunningham, Steven B. Lowen, Bonnie L. Adams, James Sutherland-Smith, et al. "Brain structural abnormalities in Doberman pinschers with canine compulsive disorder." Progress in Neuro-Psychopharmacology and Biological Psychiatry 45 (August 2013): 1–6. http://dx.doi.org/10.1016/j.pnpbp.2013.04.002.

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20

Riehl, Joyce, Mutsumi Okura, Emmanuel Mignot, and Seiji Nishino. "Inheritance of von Willebrand's disease in a colony of Doberman Pinschers." American Journal of Veterinary Research 61, no. 2 (February 2000): 115–20. http://dx.doi.org/10.2460/ajvr.2000.61.115.

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21

Klüser, L., P. J. Holler, J. Simak, G. Tater, P. Smets, D. Rügamer, H. Küchenhoff, and G. Wess. "Predictors of Sudden Cardiac Death in Doberman Pinschers with Dilated Cardiomyopathy." Journal of Veterinary Internal Medicine 30, no. 3 (May 2016): 722–32. http://dx.doi.org/10.1111/jvim.13941.

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22

Mandigers, P. J. J., T. S. G. A. M. Ingh, P. Bode, E. Teske, and J. Rothuizen. "Association between Liver Copper Concentration and Subclinical Hepatitis in Doberman Pinschers." Journal of Veterinary Internal Medicine 18, no. 5 (September 2004): 647–50. http://dx.doi.org/10.1111/j.1939-1676.2004.tb02600.x.

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23

Wess, G., A. Schulze, V. Butz, J. Simak, M. Killich, L. J. M. Keller, J. Maeurer, and K. Hartmann. "Prevalence of Dilated Cardiomyopathy in Doberman Pinschers in Various Age Groups." Journal of Veterinary Internal Medicine 24, no. 3 (February 23, 2010): 533–38. http://dx.doi.org/10.1111/j.1939-1676.2010.0479.x.

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24

Drost, Wm Tod, Terry W. Lehenbauer, and Jeff Reeves. "MENSURATION OF CERVICAL VERTEBRAL RATIOS IN DOBERMAN PINSCHERS AND GREAT DANES." Veterinary Radiology Ultrasound 43, no. 2 (March 2002): 124–31. http://dx.doi.org/10.1111/j.1740-8261.2002.tb01659.x.

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25

López-Alvarez, Jordi, Sonja Fonfara, Brigite Pedro, Hannah Stephenson, Peter J. Cripps, and Joanna Dukes-McEwan. "Assessment of mechanical ventricular synchrony in Doberman Pinschers with dilated cardiomyopathy." Journal of Veterinary Cardiology 13, no. 3 (September 2011): 183–95. http://dx.doi.org/10.1016/j.jvc.2011.05.003.

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26

Sykes, K. T., A. B. Saunders, A. R. Hoffmann, and B. F. Porter. "Histologic comparison in two Doberman pinschers with a dilated cardiomyopathy phenotype." Journal of Veterinary Cardiology 33 (February 2021): 1–5. http://dx.doi.org/10.1016/j.jvc.2020.10.001.

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27

De Decker, Steven, Ingrid M. V. L. Gielen, Luc Duchateau, Henri J. J. van Bree, Tim Waelbers, Valérie Bavegems, and Luc M. L. Van Ham. "Morphometric dimensions of the caudal cervical vertebral column in clinically normal Doberman Pinschers, English Foxhounds and Doberman Pinschers with clinical signs of disk-associated cervical spondylomyelopathy." Veterinary Journal 191, no. 1 (January 2012): 52–57. http://dx.doi.org/10.1016/j.tvjl.2010.12.017.

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28

Calvert, CA, GJ Jacobs, and CW Pickus. "Unfavorable influence of anesthesia and surgery on Doberman pinschers with occult cardiomyopathy." Journal of the American Animal Hospital Association 32, no. 1 (January 1, 1996): 57–62. http://dx.doi.org/10.5326/15473317-32-1-57.

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Exacerbation of heart rhythm disturbances or deterioration of left-ventricular function occurred in six Doberman pinschers with occult cardiomyopathy following anesthesia and surgery. These abnormalities improved over time in surviving dogs, supporting a causal relationship between anesthesia/surgery and exacerbation. Moderate myocardial failure and ventricular tachyarrhythmias of variable severity existed prior to anesthesia and surgery in each dog. The incidence of these adverse effects is uncertain but probably significant. Exacerbation of arrhythmias may be life-threatening or lethal. Embarrassment of left-ventricular function may be inapparent or, if function already is marginal, may precipitate congestive heart failure.

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29

Tidholm, A., and L. Jonsson. "A retrospective study of canine dilated cardiomyopathy (189 cases)." Journal of the American Animal Hospital Association 33, no. 6 (November 1, 1997): 544–50. http://dx.doi.org/10.5326/15473317-33-6-544.

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The case records of 189 dogs (including 38 breeds) with congestive heart failure caused by dilated cardiomyopathy were studied retrospectively. Airedale terriers, boxers, Doberman pinschers, English cocker spaniels, Newfoundlands, St. Bernards, and standard poodles were over-represented. German shepherd dogs were under-represented. A male predominance was observed. Systolic murmurs were detected in 25% of the cases. Atrial fibrillation was the most common arrhythmia. Mild hyperglycemia and mild-to-moderate hypercholesterolemia were found in 38% and 33% of cases, respectively. Histopathological changes consisted of attenuated wavy fibers and interstitial fibrosis.

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30

Blum, Binyamin. "The Hounds of Empire: Forensic Dog Tracking in Britain and its Colonies, 1888–1953." Law and History Review 35, no. 3 (August 2017): 621–65. http://dx.doi.org/10.1017/s0738248017000232.

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On February 23, 1938, a Jerusalem Military Court convicted Mustafa Mansour of the unlawful possession of a weapon and for opening fire at a bus. The prosecution's key witness tying Mansour to the shooting was, however, not human but canine. Due to darkness the police could not pursue the “brigands” immediately following the incident. They returned at dawn accompanied by Doberman Pinschers. The dogs tracked footprints from the crime scene to the defendant's village, and then to his house, where the police discovered a few rounds of ammunition, some of which were spent.

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31

Meurs, Kathryn M., Alex L. Magnon, Alan W. Spier, Mathew W. Miller, Linda B. Lehmkuhl, and Jeffrey A. Towbin. "Evaluation of the cardiac actin gene in Doberman Pinschers with dilated cardiomyopathy." American Journal of Veterinary Research 62, no. 1 (January 2001): 33–36. http://dx.doi.org/10.2460/ajvr.2001.62.33.

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32

Meurs, Kathryn M., Kristina P. Hendrix, and Michelle M. Norgard. "Molecular evaluation of five cardiac genes in Doberman Pinschers with dilated cardiomyopathy." American Journal of Veterinary Research 69, no. 8 (August 2008): 1050–53. http://dx.doi.org/10.2460/ajvr.69.8.1050.

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33

Foss, K., R. C. da Costa, P. J. Rajala-Shultz, and M. J. Allen. "Force Plate Gait Analysis in Doberman Pinschers with and without Cervical Spondylomyelopathy." Journal of Veterinary Internal Medicine 27, no. 1 (December 26, 2012): 106–11. http://dx.doi.org/10.1111/jvim.12025.

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34

Calvert, Clay A., Cynthia W. Pickus, Gilbert J. Jacobs, and Jack Brown. "Signalment, Survival, and Prognostic Factors in Doberman Pinschers With End-Stage Cardiomyopathy." Journal of Veterinary Internal Medicine 11, no. 6 (November 1997): 323–26. http://dx.doi.org/10.1111/j.1939-1676.1997.tb00474.x.

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35

DeMarco, JoAnn, Sharon A. Center, Nathan Dykes, Amy E. Yeager, Bruce Kornreich, Ed Gschrey, Kelly A. Credille, Magali Guffroy, Fabio Piero, and Beth A. Valentine. "A Syndrome Resembling Idiopathic Noncirrhotic Portal Hypertension in 4 Young Doberman Pinschers." Journal of Veterinary Internal Medicine 12, no. 3 (May 1998): 147–56. http://dx.doi.org/10.1111/j.1939-1676.1998.tb02110.x.

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36

Calvert, Clay A., Gilbert J. Jacobs, Linda Medleau, Cynthia W. Pickus, John Brown, and Michelle McDermott. "Thyroid-Stimulating Hormone Stimulation Tests in Cardiomyopathic Doberman Pinschers: A Retrospective Study." Journal of Veterinary Internal Medicine 12, no. 5 (September 1998): 343–48. http://dx.doi.org/10.1111/j.1939-1676.1998.tb02133.x.

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37

Calvert, Clay A., Gilbert Jacobs, Cynthia W. Pickus, and David D. Smith. "Results of ambulatory electrocardiography in overtly healthy Doberman Pinschers with echocardiographic abnormalities." Journal of the American Veterinary Medical Association 217, no. 9 (November 2000): 1328–32. http://dx.doi.org/10.2460/javma.2000.217.1328.

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38

CARNABUCI, Cristina, Rosalba TOGNETTI, Tommaso VEZZOSI, Federica MARCHESOTTI, Valentina PATATA, and Oriol DOMENECH. "Left shift of the ventricular mean electrical axis in healthy Doberman Pinschers." Journal of Veterinary Medical Science 81, no. 4 (2019): 620–25. http://dx.doi.org/10.1292/jvms.18-0699.

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39

Speeti, M., J. Eriksson, S. Saari, and E. Westermarck. "Lesions of Subclinical Doberman Hepatitis." Veterinary Pathology 35, no. 5 (September 1998): 361–69. http://dx.doi.org/10.1177/030098589803500505.

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This investigation describes histologic lesions in the livers of 18 Doberman Pinschers suffering from subclinical doberman hepatitis (DH). The dogs' ages ranged from 2.5 to 7 years; 15 were females and 3 were males. At the time of liver biopsy, the dogs had no clinical signs of liver disease, although serum alanine aminotransferase (ALT) values had been elevated in two samples in successive months. In the histologic examination, all biopsies revealed parenchymal and portal mononuclear inflammation. In the parenchyma, the inflammation was diffuse, with multifocal clusters of inflammatory cells. The periportal reaction was usually mild to moderate. Bridging necrosis (3/18) and bile duct proliferation (2/18) were rare. Excessive copper was detected by rubeinic acid stain in every specimen. Postmortem liver samples were obtained from nine dogs 3.5–65 months after the initial biopsy specimen; five of these dogs had been euthanatized for reasons other than DH, and liver specimens revealed piecemeal necrosis (5/5), bridging necrosis (3/5), and bile duct proliferation (2/5). Four of them had been euthanatized because of DH. Liver lesions of these dogs were typical for chronic active hepatitis, with bridging and piecemeal necrosis (4/4), portal expansion (4/4), bile duct proliferation (4/4), and fibrosis (4/4). A scoring system was used to evaluate changes numerically from biopsy to postmortem samples. Lesions in all dogs had progressed. The most important histologic changes were expansion of portal areas ( P = 0.008), increased periportal and bridging necrosis ( P = 0.008), increased fibrosis ( P = 0.016), and proliferation of the bile ducts ( P = 0.063).

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40

Meneses, Rebeca de Sousa, Mariana Lima Duarte, Karine Azevedo Fernandes, Ana Claudia do Nascimento Oliveira, Sabrina Lorena Virginio Araújo, Anne Gabrielle Moura Ferreira, Sueane Filipe Aguiar, Victória Araújo Brito, Ályd Paloma Mendes Barros, and Hewellin Jacinto Melo. "Espondilomielopatia cervical ou Síndrome de Wobbler em cães: revisão de literatura." Research, Society and Development 11, no. 13 (October 17, 2022): e90111334920. http://dx.doi.org/10.33448/rsd-v11i13.34920.

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A Espondilomielopatia cervical ou Síndrome de Wobbler se caracteriza por compressões dinâmicas e estáticas da medula espinhal e das raízes nervosas que causam sinais neurológicos e musculares, mais comumente a dor cervical. O presente estudo teve como discorrer sobre a etiologia, diagnóstico, sintomas, fisiopatologia, tratamento e prognóstico da espondilomielopatia cervical em cães. Google Acadêmica, PubMed e livros de clínica médica e cirúrgica de pequenos animais. A coleta de dados foi realizada entre os meses de setembro de 2021 a fevereiro de 2022, e utilizou os seguintes termos para pesquisa: “espondilomielopatia cervical em cães”, “wobblers syndrome”, e “espondilomielopatia”, “tratamento espondilomielopatia cervical” abrangendo as modalidades de: relatos de caso e revisão de literatura, selecionando os estudos de 1989 a 2022, nos idiomas português e inglês. A Síndrome de Wobbler canina é um distúrbio neurológico recorrente em animais de raças grandes ou gigantes, principalmente Dobermanns pinschers, representantes de 80% dos casos, além da existência de relatos em outras raças. Conclui-se então que apesar das incertezas relacionadas à etiologia da Síndrome de Wobbler, é relevante a relação entre essa patologia e fatores genéticos de raças de cães de grande porte, como Doberman pinscher, e a conformação corporal de determinadas raças, como Basset Hound. A ressonância magnética, embora seja o exame de eleição, é raramente usado para o diagnóstico, o que implica na adoção de mielografia associada ao exame prévio do líquido cefalorraquidiano como método mais utilizado diante de suspeitas clínicas, uma vez que a análise de radiografias não apresenta informações suficientes.

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41

Brooks, Marjory B., Hollis N. Erb, Polly A. Foureman, and Kunal Ray. "von Willebrand disease phenotype and von Willebrand factor marker genotype in Doberman Pinschers." American Journal of Veterinary Research 62, no. 3 (March 2001): 364–69. http://dx.doi.org/10.2460/ajvr.2001.62.364.

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42

Mausberg, Theresa-Bernadette, Gerhard Wess, Julia Simak, Lisa Keller, Michaela Drögemüller, Cord Drögemüller, Matthew T. Webster, Hannah Stephenson, Joanna Dukes-McEwan, and Tosso Leeb. "A Locus on Chromosome 5 Is Associated with Dilated Cardiomyopathy in Doberman Pinschers." PLoS ONE 6, no. 5 (May 20, 2011): e20042. http://dx.doi.org/10.1371/journal.pone.0020042.

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43

Foss, K., R. C. da Costa, and S. Moore. "Three-Dimensional Kinematic Gait Analysis of Doberman Pinschers with and without Cervical Spondylomyelopathy." Journal of Veterinary Internal Medicine 27, no. 1 (November 29, 2012): 112–19. http://dx.doi.org/10.1111/jvim.12012.

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44

Everett, R. M., J. McGann, H. C. Wimberly, and J. Althoff. "Dilated Cardiomyopathy of Doberman Pinschers: Retrospective Histomorphologic Evaluation of Heart from 32 Cases." Veterinary Pathology 36, no. 3 (May 1999): 221–27. http://dx.doi.org/10.1354/vp.36-3-221.

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45

Calvert, Clay A., Gilbert J. Jacobs, and Cynthia W. Pickus. "Bradycardia-Associated Episodic Weakness, Syncope, and Aborted Sudden Death in Cardiomyopathic Doberman Pinschers." Journal of Veterinary Internal Medicine 10, no. 2 (March 1996): 88–93. http://dx.doi.org/10.1111/j.1939-1676.1996.tb02034.x.

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46

Wess, G., O. Domenech, J. Dukes-McEwan, J. Häggström, and S. Gordon. "European Society of Veterinary Cardiology screening guidelines for dilated cardiomyopathy in Doberman Pinschers." Journal of Veterinary Cardiology 19, no. 5 (October 2017): 405–15. http://dx.doi.org/10.1016/j.jvc.2017.08.006.

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47

O'Sullivan, M. Lynne, Michael R. O'Grady, W. Glen Pyle, and John F. Dawson. "Evaluation of 10 genes encoding cardiac proteins in Doberman Pinschers with dilated cardiomyopathy." American Journal of Veterinary Research 72, no. 7 (July 2011): 932–39. http://dx.doi.org/10.2460/ajvr.72.7.932.

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Lima, Carolina G. D., Ronaldo C. da Costa, Kari D. Foss, and Matthew J. Allen. "Temporospatial and kinetic gait variables of Doberman Pinschers with and without cervical spondylomyelopathy." American Journal of Veterinary Research 76, no. 10 (October 2015): 848–52. http://dx.doi.org/10.2460/ajvr.76.10.848.

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Meurs, Kathryn M., Joshua A. Stern, Darcy Adin, Bruce W. Keene, Theresa C. De Francesco, and Sandra P. Tou. "Assessment of PDK4 and TTN gene variants in 48 Doberman Pinschers with dilated cardiomyopathy." Journal of the American Veterinary Medical Association 257, no. 10 (November 15, 2020): 1041–44. http://dx.doi.org/10.2460/javma.2020.257.10.1041.

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Calvert, Clay A., and Gilbert J. Jacobs. "Heart rate variability in Doberman Pinschers with and without echocardiographic evidence of dilated cardiomyopathy." American Journal of Veterinary Research 61, no. 5 (May 2000): 506–11. http://dx.doi.org/10.2460/ajvr.2000.61.506.

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