Academic literature on the topic 'Dorsal anterior cingulate cortex'

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Journal articles on the topic "Dorsal anterior cingulate cortex"

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Chen, Hui Juan, Li Zhang, Jun Ke, Rongfeng Qi, Qiang Xu, Yuan Zhong, Mengjie Pan, Jianjun Li, Guang Ming Lu, and Feng Chen. "Altered resting-state dorsal anterior cingulate cortex functional connectivity in patients with post-traumatic stress disorder." Australian & New Zealand Journal of Psychiatry 53, no. 1 (November 19, 2018): 68–79. http://dx.doi.org/10.1177/0004867418812674.

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Objective: The brain functional alterations at regional and network levels in post-traumatic stress disorder patients are still unclear. This study explored brain functional alterations at regional and network levels in post-traumatic stress disorder patients with resting-state functional magnetic resonance imaging and evaluated the relationship between brain function and clinical indices in post-traumatic stress disorder. Methods: Amplitude of low-frequency fluctuation and seed-based functional connectivity analyses were conducted among typhoon survivors with ( n = 27) and without post-traumatic stress disorder ( n = 33) and healthy controls ( n = 30) to assess the spontaneous brain activity and network-level brain function. Pearson correlation analyses were performed to examine the association of brain function with clinical symptom and social support. Results: Both the post-traumatic stress disorder group and the trauma-exposed control group showed decreased amplitude of low-frequency fluctuation in the dorsal anterior cingulate cortex relative to the healthy control group. The post-traumatic stress disorder group showed increased dorsal anterior cingulate cortex functional connectivity with the right paracentral lobule and bilateral precentral gyrus/postcentral gyrus relative to both control groups. Both traumatized groups exhibited decreased dorsal anterior cingulate cortex functional connectivity with the right hippocampus and left cerebellum relative to the healthy control group. More decreased dorsal anterior cingulate cortex functional connectivity with the right hippocampus was found in the post-traumatic stress disorder group. The Checklist-Civilian Version score positively correlated with functional connectivity between the dorsal anterior cingulate cortex and the right paracentral lobule as well as between the dorsal anterior cingulate cortex and the right precentral gyrus/postcentral gyrus. The social support was associated with functional connectivity between the dorsal anterior cingulate cortex and the bilateral precentral gyrus/postcentral gyrus as well as the dorsal anterior cingulate cortex and the left middle frontal gyrus. Conclusion: Trauma exposure may result in aberrant local and network-level functional connectivity in individuals with or without post-traumatic stress disorder. Altered amplitude of low-frequency fluctuation in the dorsal anterior cingulate cortex may be a predisposing risk factor for post-traumatic stress disorder development following trauma exposure. More prominent decreased dorsal anterior cingulate cortex functional connectivity with the right hippocampus might be specific in the post-traumatic stress disorder group. Improvement of social support might possibly be significant for post-traumatic stress disorder patients.
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Schulte, MHJ, AE Goudriaan, AM Kaag, DP Kooi, W. van den Brink, RW Wiers, and L. Schmaal. "The effect of N-acetylcysteine on brain glutamate and gamma-aminobutyric acid concentrations and on smoking cessation: A randomized, double-blind, placebo-controlled trial." Journal of Psychopharmacology 31, no. 10 (September 19, 2017): 1377–79. http://dx.doi.org/10.1177/0269881117730660.

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Using data form a 14-day double-blind trial with 48 smokers randomized to either N-acetylcysteine (2400 mg) or placebo, we tested the effect of N-acetylcysteine on glutamate and gamma-aminobutyric acid concentrations in the dorsal anterior cingulate cortex and on smoking cessation. Smoking related behaviors and neurotransmitter concentrations in the dorsal anterior cingulate cortex were assessed before and after treatment. Forty-seven non-smoking males served as baseline controls. Smokers showed higher baseline glutamate but similar gamma-aminobutyric acid concentrations than non-smokers. There were no treatment effects on dorsal anterior cingulate cortex neurotransmitter concentrations, smoking cessation, craving, or withdrawal symptoms. These results confirm glutamate disbalance in smokers, but not efficacy of N-acetylcysteine.
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Samuel, Nir, Eilat Kahana, Aryeh Taub, Tamar Reitich-Stolero, Rony Paz, and Aeyal Raz. "Neurons in the Nonhuman Primate Amygdala and Dorsal Anterior Cingulate Cortex Signal Aversive Memory Formation under Sedation." Anesthesiology 134, no. 5 (March 8, 2021): 734–47. http://dx.doi.org/10.1097/aln.0000000000003732.

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Background Anesthetics aim to prevent memory of unpleasant experiences. The amygdala and dorsal anterior cingulate cortex participate in forging emotional and valence-driven memory formation. It was hypothesized that this circuitry maintains its role under sedation. Methods Two nonhuman primates underwent aversive tone–odor conditioning under sedative states induced by ketamine or midazolam (1 to 8 and 0.1 to 0.8 mg/kg, respectively). The primary outcome was behavioral and neural evidence suggesting memory formation. This study simultaneously measured conditioned inspiratory changes and changes in firing rate of single neurons in the amygdala and the dorsal anterior cingulate cortex in response to an expected aversive olfactory stimulus appearing during acquisition and tested their retention after recovery. Results Aversive memory formation occurred in 26 of 59 sessions under anesthetics (16 of 29 and 10 of 30, 5 of 30 and 21 of 29 for midazolam and ketamine at low and high doses, respectively). Single-neuron responses in the amygdala and dorsal anterior cingulate cortex were positively correlated between acquisition and retention (amygdala, n = 101, r = 0.51, P < 0.001; dorsal anterior cingulate cortex, n = 121, r = 0.32, P < 0.001). Neural responses during acquisition under anesthetics were stronger in sessions exhibiting memory formation than those that did not (amygdala median response ratio, 0.52 versus 0.33, n = 101, P = 0.021; dorsal anterior cingulate cortex median response ratio, 0.48 versus 0.32, n = 121, P = 0.012). The change in firing rate of amygdala neurons during acquisition was correlated with the size of stimuli-conditioned inspiratory response during retention (n = 101, r = 0.22 P = 0.026). Thus, amygdala and dorsal anterior cingulate cortex responses during acquisition under anesthetics predicted retention. Respiratory unconditioned responses to the aversive odor anesthetics did not differ from saline controls. Conclusions These results suggest that the amygdala–dorsal anterior cingulate cortex circuit maintains its role in acquisition and maintenance of aversive memories in nonhuman primates under sedation with ketamine and midazolam and that the stimulus valence is sufficient to drive memory formation. Editor’s Perspective What We Already Know about This Topic What This Article Tells Us That Is New
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Heilbronner, Sarah R., and Benjamin Y. Hayden. "Dorsal Anterior Cingulate Cortex: A Bottom-Up View." Annual Review of Neuroscience 39, no. 1 (July 8, 2016): 149–70. http://dx.doi.org/10.1146/annurev-neuro-070815-013952.

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Fujiwara, Juri, Philippe N. Tobler, Masato Taira, Toshio Iijima, and Ken-Ichiro Tsutsui. "Segregated and Integrated Coding of Reward and Punishment in the Cingulate Cortex." Journal of Neurophysiology 101, no. 6 (June 2009): 3284–93. http://dx.doi.org/10.1152/jn.90909.2008.

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Reward and punishment have opposite affective value but are both processed by the cingulate cortex. However, it is unclear whether the positive and negative affective values of monetary reward and punishment are processed by separate or common subregions of the cingulate cortex. We performed a functional magnetic resonance imaging study using a free-choice task and compared cingulate activations for different levels of monetary gain and loss. Gain-specific activation (increasing activation for increasing gain, but no activation change in relation to loss) occurred mainly in the anterior part of the anterior cingulate and in the posterior cingulate cortex. Conversely, loss-specific activation (increasing activation for increasing loss, but no activation change in relation to gain) occurred between these areas, in the middle and posterior part of the anterior cingulate. Integrated coding of gain and loss (increasing activation throughout the full range, from biggest loss to biggest gain) occurred in the dorsal part of the anterior cingulate, at the border with the medial prefrontal cortex. Finally, unspecific activation increases to both gains and losses (increasing activation to increasing gains and increasing losses, possibly reflecting attention) occurred in dorsal and middle regions of the cingulate cortex. Together, these results suggest separate and common coding of monetary reward and punishment in distinct subregions of the cingulate cortex. Further meta-analysis suggested that the presently found reward- and punishment-specific areas overlapped with those processing positive and negative emotions, respectively.
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Shenhav, Amitai, Jonathan D. Cohen, and Matthew M. Botvinick. "Dorsal anterior cingulate cortex and the value of control." Nature Neuroscience 19, no. 10 (September 27, 2016): 1286–91. http://dx.doi.org/10.1038/nn.4384.

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Srinivasan, Lakshminarayan, Wael F. Asaad, Daniel T. Ginat, John T. Gale, Darin D. Dougherty, Ziv M. Williams, Terrence J. Sejnowski, and Emad N. Eskandar. "Action Initiation in the Human Dorsal Anterior Cingulate Cortex." PLoS ONE 8, no. 2 (February 27, 2013): e55247. http://dx.doi.org/10.1371/journal.pone.0055247.

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Wang, Jue, Ning Yang, Wei Liao, Han Zhang, Chao-Gan Yan, Yu-Feng Zang, and Xi-Nian Zuo. "Dorsal anterior cingulate cortex in typically developing children: Laterality analysis." Developmental Cognitive Neuroscience 15 (October 2015): 117–29. http://dx.doi.org/10.1016/j.dcn.2015.10.002.

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Sheth, Sameer A., Matthew K. Mian, Shaun R. Patel, Wael F. Asaad, Ziv M. Williams, Darin D. Dougherty, George Bush, and Emad N. Eskandar. "Human dorsal anterior cingulate cortex neurons mediate ongoing behavioural adaptation." Nature 488, no. 7410 (June 24, 2012): 218–21. http://dx.doi.org/10.1038/nature11239.

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Azab, Habiba, and Benjamin Y. Hayden. "Correlates of decisional dynamics in the dorsal anterior cingulate cortex." PLOS Biology 15, no. 11 (November 15, 2017): e2003091. http://dx.doi.org/10.1371/journal.pbio.2003091.

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Dissertations / Theses on the topic "Dorsal anterior cingulate cortex"

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Adkins, Jordan M. "Anterior Cingulate Cortex and Dorsal Hippocampal Glutamate Receptors Mediate Generalized Fear in Female Rats." Kent State University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=kent1544527648769848.

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Siegel-Ramsay, Jennifer Eileen. "Dorsal anterior cingulate cortex glutamate concentrations and their relationships in adults with autism spectrum disorder." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/33096.

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Previous studies have reported altered glutamate (Glu) concentrations in the blood and brain of individuals with autism spectrum disorder (ASD) compared to neurotypical controls (NC), but the direction (increased or decreased) of metabolite differences is still unclear. Moreover, the relationship between Glu and both brain function and clinical manifestations of the disorder require further investigation. Within this study, we investigated metabolite concentrations within the dorsal anterior cingulate cortex (dACC), a brain region functionally associated with inhibitory executive control tasks and also part of the salience network. There were 19 participants with ASD and 20 NCs between the ages of 23 and 58 years who participated in this study. A study clinician administered the Autism Diagnostic Observation Schedule (ADOS) to individuals with ASD to further confirm their diagnosis. In addition, all participants in this study completed assessments of general intelligence and attention, which included an inhibitory executive control task. Researchers also acquired in vivo single-voxel proton magnetic resonance spectroscopy (1H-MRS) in the dACC to quantify both Glu and combined Glu and glutamine (Glx) concentrations. We hypothesised that these metabolite concentrations would be altered (decreased or increased) in adult participants with ASD compared to NCs and would correlate with inhibitory performance and ASD severity in individuals with ASD. Participants also underwent a resting-state functional magnetic resonance imaging (fMRI) scan to assess the relationship between functional connectivity and Glu and Glx concentrations. We also hypothesised that there would be an altered relationship between local Glu and Glx concentrations and seed-based functional connectivity in adults with ASD compared to NCs. There were no significant group differences in Glu or Glx concentrations between individuals with ASD and NCs. Furthermore, we did not find any relationship between metabolite concentrations and either inhibitory performance or clinical symptoms of the disorder. This evidence suggests that increased or decreased Glu and Glx concentrations were not a core marker of altered brain function in the dACC in this group of adult individuals with ASD. When individuals taking psychotropic medications were excluded from the analysis, there was a significant interaction between age and group for Glx concentrations. This evidence weakly suggests disease-specific variations in Glx concentrations over the lifespan of an individual with ASD. Nevertheless, this result did not survive correction for multiple comparisons and requires further replication. In our final experiment, we reported that Glu concentrations were negatively correlated with right and left dACC seed-based resting-state functional connectivity to the left medial temporal lobe only in individuals with ASD. We also reported an interaction between groups in the association between Glx concentrations and both left and right dACC functional connectivity to other salience network regions including the insular cortex. This evidence suggests that local Glu and Glx concentrations were incongruent with long-distance functional connectivity in individuals with ASD. This analysis was largely exploratory, but further investigation and replication of these relationships may further explain the pathophysiology of the disorder as well as provide a useful marker for therapeutic intervention.
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Rahman, Sufia Saburan. "Differential contributions of subregions of the dorsal anterior cingulate cortex to negative emotion in the common marmoset." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/277913.

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The dorsal anterior cingulate cortex (dACC) has been implicated in a broad range of cognitive and emotional functions, including the processing of negative emotion. Furthermore, abnormalities in dACC activity have been associated with anxiety and depression, disorders in which negative emotion is dysregulated. Thus, a better understanding of the precise contributions of the dACC to negative emotion could give us important insights into the neurobiological mechanisms underlying these debilitating neuropsychiatric disorders. However, despite extensive study of the dACC, its precise role in negative emotion is unclear. Instead there is mounting evidence that rather than being one functionally homogeneous region, subregions of the dACC may have distinct functional roles. This evidence is largely correlational, and interventional studies in experimental animals are required to address this. Accordingly, the work in this thesis causally assessed the contributions of two spatially distinct subregions of the dACC (rostral and caudal) to the regulation of the behavioural and cardiovascular correlates of negative emotion in the common marmoset (Callithrix jacchus). These dACC subregions were targeted with indwelling cannulae to enable pharmacological manipulations to be carried out in a range of tasks, used to assess distinct components of negative emotion, such as conditioned fear and anxiety. The findings suggest that the rostral dACC and the caudal dACC do indeed have distinct contributions to the expression of negative emotion and the regulation of anxiety, respectively. Furthermore, an assessment of the anterograde projections of these subregions provides anatomical support for the observed functional differences.
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Hussain, Muhammad Zubair. "Molecular Adaptations in the Endogenous Opioid System in Human and Rodent Brain." Doctoral thesis, Uppsala universitet, Institutionen för farmaceutisk biovetenskap, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-205133.

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The aims of the thesis were to examine i) whether the endogenous opioid system (EOS) is lateralized in human brain areas involved in processing of emotions and pain; ii) whether EOS responses to unilateral brain injury depend on side of lesion, and iii) whether in human alcoholics, this system is involved in molecular adaptations in brain areas relevant for cognitive control of addictive behavior and habit formation. The main findings were that (1) opioid peptides but not opioid receptors and classic neurotransmitters are markedly lateralized in the anterior cingulate cortex involved in processing of  positive and negative emotions and affective component of pain. The region-specific lateralization of neuronal networks expressing opioid peptides may underlie in part lateralization of higher functions in the human brain including emotions and pain. (2) Analysis of the effects of traumatic brain injury (TBI) demonstrated predominant alteration of dynorphin levels in the hippocampus ipsilateral to the injury, while injury to the right hemisphere affected dynorphin levels in the striatum and frontal cortex to a greater extent than that to the left hemisphere. Thus, trauma reveals a lateralization in the mechanisms mediating the response of dynorphin expressing neuronal networks in the brain. These networks may differentially mediate effects of left or right brain injury on lateralized brain functions. (3) In human alcoholics, the enkephalin and dynorphin systems were found to be downregulated in the caudate nucleus and / or putamen that may underlie in part changes in goal directed behavior and formation of a compulsive habit in alcoholics. In contrast to downregulation in these areas, PDYN mRNA and dynorphins in dorsolateral prefrontal cortex, k-opioid receptor mRNA in orbitofrontal cortex, and dynorphins in hippocampus were upregulated in alcoholics. Activation of the k-opioid receptor by upregulated dynorphins may underlie in part neurocognitive dysfunctions relevant for addiction and disrupted inhibitory control. We conclude that the EOS exhibits region-specific lateralization in human brain and brain-area specific lateralized response after unilateral TBI in mice; and that the EOS is involved in adaptive processes associated with specific aspects of alcohol dependence.
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Feroz, Farah Shahnaz [Verfasser], and Christoph [Akademischer Betreuer] Mulert. "Time Course of the Dorsal and Rostral-Ventral Anterior Cingulate Cortex Reveals the Influence of Emotional Valence and Arousal on Cognitive Control in Healthy Subjects and Patients With Schizophrenia / Farah Shahnaz Feroz ; Betreuer: Christoph Mulert." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2018. http://d-nb.info/1168380928/34.

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Syme, Catriona. "Cardiovascular reactivity and the anterior cingulate cortex." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=98504.

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Relationships between cardiovascular and neural activities were explored in 11 patients with intractable epilepsy. Using depth electrodes, we examined the cardiovascular response to direct electrical stimulation, and monitored EEG activity during simple physical and mental challenges known to elicit a cardiovascular response. Stimulation was most commonly performed in four regions: the hippocampus, amygdala, and the supra- and sub-callosal anterior cingulate cortex (ACC). While increases in systolic blood pressure (SBP) in response to electrical stimulation of these brain regions were observed with equal frequency, a striking cardiovascular response was observed in one patient when stimulating the supracallosal ACC (an increase in SBP of almost 50 mmHg). Cross-correlation analysis revealed significant relationships between EEG power and SBP during physical but not mental challenges, and the pattern of these relationships was only observed for power of EEG data recorded from the ACC. The observed relative timing of the correlations was consistent in the three patients whose SBP increased upon ACC stimulation. These results support the role of the ACC in cardiovascular regulation, as revealed in previous studies in experimental animals.
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Apps, Matthew. "Anterior cingulate cortex : contributions to social cognition." Thesis, Royal Holloway, University of London, 2012. http://repository.royalholloway.ac.uk/items/ed1f2ffb-d93b-e7fa-1121-846d7f34efd8/9/.

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It has been suggested that the Anterior Cingulate Cortex (ACC) plays an important role in decision-making. Activity in this area reflects processing related to two principles of Reinforcement Learning Theory (RLT): (i) signalling the predicted value of actions at the time they are instructed and (ii) signalling prediction errors at the time of the outcomes of actions. It has been suggested that neurons in the gyrus of the ACC (ACCg) process information about others' decisions and not one's own. An important aim of this thesis is to investigate whether the ACCg processes others' decisions in a manner that conforms to the principles of RLT. Four fMRI experiments investigate activity in the ACCg at the time of cues that signal either the predicted value of others' actions or that signal another's predictions are erroneous. • Experiment 1: Activity in the ACCg occurred when the outcome of another's decision was unexpectedly positive. • Experiment 2: Activity in the ACCg varied parametrically with the discrepancy between another's prediction of an outcome and the actual outcome known by the subject, in a manner that conformed to the computational principles of RLT. • Experiment 3: Activity in the ACCg varied with the predicted value of a reward, discounted by the amount of effort required to obtain it. • Experiment 4: Activity in the ACCg varied with the value of delayed rewards that were discounted in a manner that conformed to a social norm. These results support the hypothesis that the ACCg processes the predicted value of others' actions and also signals when others' predictions about the value of their actions are erroneous, in a manner that conforms to the principles of RLT.
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Ma, Liya. "Coding of sequential behaviors by anterior cingulate cortex ensembles." Thesis, University of British Columbia, 2014. http://hdl.handle.net/2429/48442.

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The anterior cingulate cortex (ACC) has been implicated in a myriad of different functions. Converging evidence suggests that the ACC continuously monitors and evaluates actions and their consequences. Such functions are essential in representing action sequences which are the building blocks of all complex behaviors. This dissertation seeks to delineate how ACC neuronal ensembles represent different types of information with special emphasis on action sequences. Chapter 2 shows that the ACC ensembles represents different action sequences via unique activity patterns that change if the order of the actions are altered or if the locations of the actions is changed. Interestingly such shifts are achieved when overall levels of activity remain fixed. Chapter 3 reveals a very different arrangement in which progression through a sequence of actions towards a goal is associated with a change in the overall level of neural activity without a significant change in the patterns of activity. Specifically, ACC ensembles display a smooth progressive change in overall activity over three lever press actions that culminate in a reward. In contrast, the dorsal striatal (DS) ensembles recorded simultaneously from the same animals display fluctuations in activity level that are tightly linked to each action. Together these two chapters show that the ACC may use two different firing rate-related codes to convey categorical versus continuous forms of information. Chapter 4 provides a further examination of the mechanisms which allows the ACC ensembles to encode multiple types of categorical information. While the DS neurons encode both the sequence and the location of the levers in a somewhat synchronized fashion, ACC neurons encoded both of these types of information but kept them functionally segregated. As a result, even though ACC single neurons were no better than the DS in sequence decoding, sequence decoding by ACC ensembles was far superior to DS ensembles. The last chapter attempts to produce a unified theory of ACC function based on its coding properties. I will argue that the ACC monitors many aspects of experience while evaluating the current state with reference to a goal. Its multiple coding schemes efficiently serve both monitoring and evaluating functions.
Medicine, Faculty of
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Shinozaki, Jun. "Heterospecific and conspecific social cognition in the anterior cingulate cortex." Kyoto University, 2008. http://hdl.handle.net/2433/135801.

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Walton, Mark. "The role of the anterior cingulate cortex in making and changing decisions." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.400291.

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Books on the topic "Dorsal anterior cingulate cortex"

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Haber, Suzanne N. Neurocircuitry Underlying OCD. Edited by Christopher Pittenger. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228163.003.0020.

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Structural and functional imaging studies have identified abnormalities in the brains of individuals with OCD. The most consistent findings point to pathology in the circuitry connecting the prefrontal cortex with the basal ganglia, and especially to abnormalities in the orbitofrontal cortex (OFC), ventromedial prefrontal cortex (vmPFC), dorsal anterior cingulate cortex (dACC), and striatum. This chapter describes the detailed anatomy and interconnectivity of these structures, together with its functional correlates, to provide context for the more detailed treatment of abnormalities seen in OCD provided in the chapters that follow. These corticostriatal circuits are critical for reward processing, reward learning, and action selection, and so disruption in these circuitries in OCD may underlie abnormalities in these domains. Precisely defining the anatomy of these circuits and how it is disrupted in OCD, at both the group and individual level, is increasingly important, as it may help us to optimize anatomically targeted treatment strategies.
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Ding, Hoi-Ki. Involvement of the anterior cingulate cortex in remote conditioned taste aversion memory recall. 2006.

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Griffin, Amy L. Anterior cingulate cortex modulation of conditioned jaw movement and learning-related hippocampal activity in rabbit. 2003.

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Taylor, Véronique A., and Pierre Rainville. Endogenous opioids in placebo-induced analgesia. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0011.

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Placebos achieve scientifically proven pain-relieving effects yet are inactive substances for the treatment of pain. Levine, Gordon, and Fields were the first to demonstrate the role of endogenous opioids in placebo-induced analgesia during dental post-operative pain. Several studies using pharmacological manipulations and/or neuroimaging techniques confirmed their findings that placebo analgesia is reversible by naloxone, and also identified brain pathways involved in opioidergic neurotransmission during placebo analgesia (prefrontal regions rich in opioid receptors such as the anterior cingulate cortex, presumably initiating descending pain modulation through downstream projections to the brainstem). Fifty years of research in pharmacology and neurobiology have contributed to the identification of physical as well as psychological determinants of placebo analgesia. Expectations of pain relief are maintained by conditioned learning and reward-related processes, reflected by interactions between different neurotransmitters (opioids, dopamine, endocannabinoids) in a variety of brain circuits related to executive/cognitive processes as well as affect and reward.
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Book chapters on the topic "Dorsal anterior cingulate cortex"

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Cassaday, H. J. "Anterior Cingulate Cortex." In Encyclopedia of Personality and Individual Differences, 188–91. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-24612-3_727.

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Cassaday, H. J. "Anterior Cingulate Cortex." In Encyclopedia of Personality and Individual Differences, 1–4. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-28099-8_727-1.

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Cohen, Ronald A., and Anna MacKay-Brandt. "Anterior Cingulate Cortex." In Encyclopedia of Clinical Neuropsychology, 257–60. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1265.

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Cohen, Ronald A., and Anna MacKay-Brandt. "Anterior Cingulate Cortex." In Encyclopedia of Clinical Neuropsychology, 184–86. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_1265.

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Cohen, Ronald, and Anna MacKay-Brandt. "Anterior Cingulate Cortex." In Encyclopedia of Clinical Neuropsychology, 1–3. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-56782-2_1265-2.

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Benga, Oana. "Intentional communication and the anterior cingulate cortex." In Vocalize to Localize, 159–78. Amsterdam: John Benjamins Publishing Company, 2009. http://dx.doi.org/10.1075/bct.13.10ben.

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Clark, Danielle, Irina Dedova, and Izuru Matsumoto. "Proteomics of the Anterior Cingulate Cortex in Schizophrenia." In Genomics, Proteomics, and the Nervous System, 381–98. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-7197-5_14.

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Vogt, Brent A., Robert W. Sikes, and Leslie J. Vogt. "Anterior Cingulate Cortex and the Medial Pain System." In Neurobiology of Cingulate Cortex and Limbic Thalamus, 313–44. Boston, MA: Birkhäuser Boston, 1993. http://dx.doi.org/10.1007/978-1-4899-6704-6_11.

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Brown, Joshua W. "Models of Anterior Cingulate Cortex Function in Cognitive Control." In The Wiley Handbook of Cognitive Control, 259–73. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781118920497.ch15.

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Neafsey, E. J., R. R. Terreberry, K. M. Hurley, K. G. Ruit, and R. J. Frysztak. "Anterior Cingulate Cortex in Rodents: Connections, Visceral Control Functions, and Implications for Emotion." In Neurobiology of Cingulate Cortex and Limbic Thalamus, 206–23. Boston, MA: Birkhäuser Boston, 1993. http://dx.doi.org/10.1007/978-1-4899-6704-6_7.

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Conference papers on the topic "Dorsal anterior cingulate cortex"

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Wang, R., Q. Fan, Z. Zhang, Y. Chen, S. Tong, and Y. Li. "White matter integrity correlates with choline level in dorsal anterior cingulate cortex of obsessive compulsive disorder patients: A combined DTI-MRS study." In 2017 39th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2017. http://dx.doi.org/10.1109/embc.2017.8037616.

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Gasecka, Alicja, Pierre-Eric Lutz, Arnaud Tanti, Naguib Mechawar, Gustavo Turecki, and Daniel C. Côté. "Early Life Adversity Leads to Demyelination in the Anterior Cingulate Cortex." In Bio-Optics: Design and Application. Washington, D.C.: OSA, 2019. http://dx.doi.org/10.1364/boda.2019.jt4a.15.

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Wen, Linfei, Congyu Liao, Meng Chen, Darong Zhu, Xu Yan, Jianhui Zhong, and Song Chen. "Detection and Evaluation of GABA Concentration in Anterior Cingulate Cortex and Occipital Cortex by MEGA-PRESS." In 2015 7th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2015. http://dx.doi.org/10.1109/itme.2015.33.

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Fanhua, Meng, Ma Meifang, and Tian Xin. "Neuronal Ensemble Score Coding in the Anterior Cingulate Cortex of Rats under Fear Conditioning." In 2008 Second International Symposium on Intelligent Information Technology Application (IITA). IEEE, 2008. http://dx.doi.org/10.1109/iita.2008.478.

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Wenwen, Bai, Ma Meifang, and Tian Xin. "Neuronal Ensemble Entropy Coding in the Anterior Cingulate Cortex of Rats under Fear Conditioning." In 2008 International Workshop on Geoscience and Remote Sensing (ETT and GRS). IEEE, 2008. http://dx.doi.org/10.1109/ettandgrs.2008.348.

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Russell, M., F. Howe, T. Barrick, and N. Sofat. "SAT0530 Central sensitisation in hand osteoarthritis: the anterior cingulate cortex is involved in pain processing." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.3537.

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Sklar, Samuel, Matthew Walmer, Pierre Sacre, Catherine A. Schevon, Shraddha Srinivasan, Garrett P. Banks, Mark J. Yates, et al. "Neuronal activity in human anterior cingulate cortex modulates with internal cognitive state during multi-source interference task." In 2017 39th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2017. http://dx.doi.org/10.1109/embc.2017.8036985.

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Schmitt, S., T. Sauder, F. Meier, J. Engelen, H. Bröhl, B. Dietsche, J. Heinen, et al. "The Impact of Polygenic Risk for Schizophrenia on Memory-related Activation in the Anterior Cingulate Cortex (ACC)." In Abstracts of the 30th Symposium of the AGNP. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1606424.

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Iacovella, Vittorio, and Uri Hasson. "Magnitude of task-induced deactivation of insula and anterior cingulate cortex is related to inter-individual differences in RMSSD." In 2014 8th Conference of the European Study Group on Cardiovascular Oscillations (ESGCO). IEEE, 2014. http://dx.doi.org/10.1109/esgco.2014.6847565.

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Yuming, Zhuang, and Tian Xin. "Phase Synchronization Analysis of 14-Channel LFPs in the Anterior Cingulate Cortex of Rats Under Fear Condition via Equal-Time Correlation Matrix." In 2008 Second International Symposium on Intelligent Information Technology Application (IITA). IEEE, 2008. http://dx.doi.org/10.1109/iita.2008.494.

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