Academic literature on the topic 'Drosophila Hematopoiesis'

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Journal articles on the topic "Drosophila Hematopoiesis"

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Lanot, René, Daniel Zachary, François Holder, and Marie Meister. "Postembryonic Hematopoiesis in Drosophila." Developmental Biology 230, no. 2 (2001): 243–57. http://dx.doi.org/10.1006/dbio.2000.0123.

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Lan, Wenwen, Sumin Liu, Long Zhao, and Ying Su. "Regulation of Drosophila Hematopoiesis in Lymph Gland: From a Developmental Signaling Point of View." International Journal of Molecular Sciences 21, no. 15 (2020): 5246. http://dx.doi.org/10.3390/ijms21155246.

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The Drosophila hematopoietic system is becoming increasingly attractive for its simple blood cell lineage and its developmental and functional parallels with the vertebrate system. As the dedicated organ for Drosophila larval hematopoiesis, the lymph gland harbors both multipotent stem-like progenitor cells and differentiated blood cells. The balance between progenitor maintenance and differentiation in the lymph gland must be precisely and tightly controlled. Multiple developmental signaling pathways, such as Notch, Hedgehog, and Wnt/Wingless, have been demonstrated to regulate the hematopoie
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Remillieux-Leschelle, Nathalie, Pedro Santamaria, and Neel B. Randsholt. "Regulation of Larval Hematopoiesis in Drosophila melanogaster: A Role for the multi sex combs Gene." Genetics 162, no. 3 (2002): 1259–74. http://dx.doi.org/10.1093/genetics/162.3.1259.

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Abstract Drosophila larval hematopoietic organs produce circulating hemocytes that ensure the cellular host defense by recognizing and neutralizing non-self or noxious objects through phagocytosis or encapsulation and melanization. Hematopoietic lineage specification as well as blood cell proliferation and differentiation are tightly controlled. Mutations in genes that regulate lymph gland cell proliferation and hemocyte numbers in the body cavity cause hematopoietic organ overgrowth and hemocyte overproliferation. Occasionally, mutant hemocytes invade self-tissues, behaving like neoplastic ma
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Evans, Cory J., and Utpal Banerjee. "Transcriptional regulation of hematopoiesis in Drosophila." Blood Cells, Molecules, and Diseases 30, no. 2 (2003): 223–28. http://dx.doi.org/10.1016/s1079-9796(03)00028-7.

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Varga, Gergely I. B., Gábor Csordás, Gyöngyi Cinege, et al. "Headcase is a Repressor of Lamellocyte Fate in Drosophila melanogaster." Genes 10, no. 3 (2019): 173. http://dx.doi.org/10.3390/genes10030173.

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Due to the evolutionary conservation of the regulation of hematopoiesis, Drosophila provides an excellent model organism to study blood cell differentiation and hematopoietic stem cell (HSC) maintenance. The larvae of Drosophila melanogaster respond to immune induction with the production of special effector blood cells, the lamellocytes, which encapsulate and subsequently kill the invader. Lamellocytes differentiate as a result of a concerted action of all three hematopoietic compartments of the larva: the lymph gland, the circulating hemocytes, and the sessile tissue. Within the lymph gland,
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Owusus-Ansah, Edward, Tina Mukherjee, William Kim, and Utpal Banerjee. "Maintaining Stem Cell Fate During Drosophila Hematopoiesis." Free Radical Biology and Medicine 49 (January 2010): S7. http://dx.doi.org/10.1016/j.freeradbiomed.2010.10.675.

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Lin, Xionghui, and Irene Söderhäll. "Crustacean hematopoiesis and the astakine cytokines." Blood 117, no. 24 (2011): 6417–24. http://dx.doi.org/10.1182/blood-2010-11-320614.

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Abstract Major contributions to research in hematopoiesis in invertebrate animals have come from studies in the fruit fly, Drosophila melanogaster, and the freshwater crayfish, Pacifastacus leniusculus. These animals lack oxygen-carrying erythrocytes and blood cells of the lymphoid lineage, which participate in adaptive immune defense, thus making them suitable model animals to study the regulation of blood cells of the innate immune system. This review presents an overview of crustacean blood cell formation, the role of these cells in innate immunity, and how their synthesis is regulated by t
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Lebestky, T. "A Serrate-expressing signaling center controls Drosophila hematopoiesis." Genes & Development 17, no. 3 (2003): 348–53. http://dx.doi.org/10.1101/gad.1052803.

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Ramond, Elodie, Bianca Petrignani, Jan Paul Dudzic, et al. "The adipokine NimrodB5 regulates peripheral hematopoiesis in Drosophila." FEBS Journal 287, no. 16 (2020): 3399–426. http://dx.doi.org/10.1111/febs.15237.

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Milton, Claire C., Felix A. Grusche, Joffrey L. Degoutin, et al. "The Hippo Pathway Regulates Hematopoiesis in Drosophila melanogaster." Current Biology 24, no. 22 (2014): 2673–80. http://dx.doi.org/10.1016/j.cub.2014.10.031.

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Dissertations / Theses on the topic "Drosophila Hematopoiesis"

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Miller, Marion. "Caractérisation du rôle et du mode d'action de MLF au cours de l'hématopoïèse chez la drosophile." Thesis, Toulouse 3, 2015. http://www.theses.fr/2015TOU30282/document.

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L'hématopoïèse est le processus développemental qui permet la formation des cellules qui composent le sang. Au niveau moléculaire, de nombreux facteurs de transcription permettent une régulation fine de ce processus et la dérégulation de leur activité, en affectant la différenciation ou la prolifération des cellules sanguines, peut conduire à l'apparition d'hémopathies telles que les leucémies. De manière intéressante, de nombreux gènes contrôlant l'hématopoïèse sont conservés entre la Drosophile et l'homme. Ces dernières années, cet insecte a donc émergé en tant que modèle pour l'étude du dév
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Vyas, Aditi. "Identification of Novel Stat92E Target Genes in Drosophila Hematopoiesis." Ohio University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1450868635.

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Louradour, Isabelle. "Réponse au parasitisme par des guêpes chez la drosophile : rôle de la voie de signalisation Toll/NFkB." Thesis, Toulouse 3, 2015. http://www.theses.fr/2015TOU30256/document.

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Dans tous les organismes animaux la réponse immunitaire est divisée en deux composantes : la réponse humorale, qui consiste en la production d'un grand nombre de molécules toxiques pour le pathogène, et la réponse cellulaire, qui met en jeu des cellules immunitaires produites lors de l'hématopoïèse. Chez les mammifères adultes, l'hématopoïèse se déroule dans la moelle osseuse, où un microenvironnement particulier appelé " niche hématopoïétique " contrôle l'auto-renouvèlement, la prolifération et la différenciation des Cellules Souches Hématopoïétiques (CSH) à l'origine de l'ensemble des cellul
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Shen, Ying. "The JAK/STAT pathway in Drosophila hematopoiesis: function and regulatory mechanisms." Ohio : Ohio University, 2007. http://www.ohiolink.edu/etd/view.cgi?ohiou1194628059.

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Krzemien, Joanna. "Control of larval hematopoiesis in Drosophila ; microenvironment, precursors and cell lineage." Toulouse 3, 2008. http://www.theses.fr/2008TOU30206.

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L'hématopoiése larvaire de la drosophile a lieu au sein d'un organe spécialisé, la glande de la lymphe (LG) qui produit des plasmatocytes spécialisés dans la phagocytose et des cellules à cristaux nécessaires à la mélanisation des corps étrangers (4). La LG est aussi à l'origine des lamellocytes nécessaires à l'encapsulation de gros corps étrangers et qui se différencient en réponse à des challenges immuns particuliers tels que le parasitisme par des hyménoptères. En 2004, notre laboratoire a montré que Collier (Col), l'orthologue du facteur de transcription mammifère Early B-Cell Factor est e
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McWhorter, Amy Louise. "Characterization of embryonic hematopoiesis and larval lymph gland development in Drosophila melanogaster." Diss., Restricted to subscribing institutions, 2008. http://proquest.umi.com/pqdweb?did=1779690101&sid=10&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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Huang, Liang. "Functions and Regulatory Mechanisms of the Rel Family Transcription Factors, Dorsal and Dif, and the UBC9 Family SUMO Conjugase, Lesswright, in DrosophilaHematopoiesis." Ohio : Ohio University, 2006. http://www.ohiolink.edu/etd/view.cgi?ohiou1162613472.

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Genais, Thomas. "Caractérisation de la fonction et du mode d'action d'Enok, une histone acétyltransférase de type MOZ, dans le contrôle de la prolifération et de la différenciation des cellules sanguines de drosophile." Thesis, Toulouse 3, 2019. http://www.theses.fr/2019TOU30215.

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L'hématopoïèse est un processus très finement régulé qui mène à la formation de chaque cellule sanguine d'un organisme. Chez les mammifères il existe un nombre important de types cellulaires qui participent à l'établissement des mécanismes de défense du corps. Toutes ces cellules proviennent de la différenciation terminale d'une cellule unique appelée Cellule Souche Hématopoïétique (CSH) qui, par le biais de différenciations successives donnant naissance à des progéniteurs de plus en plus spécifiés, va permettre l'établissement normal de tous les types de cellules sanguines. Les CSH sont parmi
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Rush, Craig Michael. "Crosstalk between the Jak-Stat and Wingless pathways is mediated by Mad in Drosophila melanogaster larval hematopoiesis." Ohio University Art and Sciences Honors Theses / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ouashonors1367508013.

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Abraham, Jinu. "THE ROLE OF DROSOPHILA SUMO CONJUGATING ENZYME LESSWRIGHT IN LARVAL HEMATOPOIESIS: EFFECTS ON CACTUS, DORSAL AND DORSAL-RELATED IMMUNITY FACTOR (DIF)." Ohio : Ohio University, 2007. http://www.ohiolink.edu/etd/view.cgi?ohiou1187275172.

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Book chapters on the topic "Drosophila Hematopoiesis"

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Bodian, D. L., S. Leung, H. Chiu, and S. Govind. "Cytokines in Drosophila Hematopoiesis and Cellular Immunity." In Invertebrate Cytokines and the Phylogeny of Immunity. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-18670-7_2.

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Meister, Marie, and Shubha Govind. "Hematopoietic Development in Drosophila: A Parallel with Vertebrates." In Hematopoietic Stem Cell Development. Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-33535-3_10.

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Evans, Cory J., Sergey A. Sinenko, Lolitika Mandal, Julian A. Martinez‐Agosto, Volker Hartenstein, and Utpal Banerjee. "Genetic Dissection of Hematopoiesis Using Drosophila as a Model System." In Cardiovascular Development. Elsevier, 2007. http://dx.doi.org/10.1016/s1574-3349(07)18011-x.

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