Relevant bibliographies by topics / Drug Resistance, Candida tropicalis

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  2. Dissertations / Theses
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Academic literature on the topic 'Drug Resistance, Candida tropicalis'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "Drug Resistance, Candida tropicalis"

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Forastiero, A., A. C. Mesa-Arango, A. Alastruey-Izquierdo, et al. "Candida tropicalis Antifungal Cross-Resistance Is Related to Different Azole Target (Erg11p) Modifications." Antimicrobial Agents and Chemotherapy 57, no. 10 (2013): 4769–81. http://dx.doi.org/10.1128/aac.00477-13.

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ABSTRACTCandida tropicalisranks between third and fourth amongCandidaspecies most commonly isolated from clinical specimens. Invasive candidiasis and candidemia are treated with amphotericin B or echinocandins as first-line therapy, with extended-spectrum triazoles as acceptable alternatives.Candida tropicalisis usually susceptible to all antifungal agents, although several azole drug-resistant clinical isolates are being reported. However,C. tropicalisresistant to amphotericin B is uncommon, and only a few strains have reliably demonstrated a high level of resistance to this agent. The resist
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Law, D., C. B. Moore, L. A. Joseph, M. G. L. Keaney, and D. W. Denning. "High incidence of antifungal drug resistance in Candida tropicalis." International Journal of Antimicrobial Agents 7, no. 4 (1996): 241–45. http://dx.doi.org/10.1016/s0924-8579(96)00328-7.

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Al-Fattani, Mohammed A., and L. Julia Douglas. "Biofilm matrix of Candida albicans and Candida tropicalis: chemical composition and role in drug resistance." Journal of Medical Microbiology 55, no. 8 (2006): 999–1008. http://dx.doi.org/10.1099/jmm.0.46569-0.

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Matrix material was extracted from biofilms of Candida albicans and Candida tropicalis and analysed chemically. Both preparations contained carbohydrate, protein, hexosamine, phosphorus and uronic acid. However, the major component in C. albicans matrix was glucose (32 %), whereas in C. tropicalis matrix it was hexosamine (27 %). Biofilms of C. albicans were more easily detached from plastic surfaces by treatment with the enzyme lyticase (β-1,3-glucanase) than were those of C. tropicalis. Biofilms of C. albicans were also partially detached by treatment with proteinase K, chitinase, DNase I, o
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Hsieh, Yi-Hsuan, Wen-Ching Chuang, Kun-Hua Yu, Cheng-Ping Jheng, and Cheng-I. Lee. "Sequential Photodynamic Therapy with Phthalocyanine Encapsulated Chitosan-Tripolyphosphate Nanoparticles and Flucytosine Treatment against Candida tropicalis." Pharmaceutics 11, no. 1 (2019): 16. http://dx.doi.org/10.3390/pharmaceutics11010016.

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Antibiotic resistance has become a crisis. Candida tropicalis (C. tropicalis) is one of the most highly virulent and drug-resistant pathogens. An alternative antimicrobial therapy to eradicate C. tropicalis effectively, without the risk of developing drug-resistance, is needed. Photodynamic therapy (PDT) is an alternative therapy that does not carry the risk of undesired drug resistance. To target the pathogens and to enhance the cellular penetration of the applied photosensitizer, we fabricated cationic chitosan/tripolyphosphate nanoparticles to encapsulate phthalocyanine. Our strategy promot
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Kothavade, Rajendra J., M. M. Kura, Arvind G. Valand, and M. H. Panthaki. "Candida tropicalis: its prevalence, pathogenicity and increasing resistance to fluconazole." Journal of Medical Microbiology 59, no. 8 (2010): 873–80. http://dx.doi.org/10.1099/jmm.0.013227-0.

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Candida tropicalis has been identified as the most prevalent pathogenic yeast species of the Candida-non-albicans group. Historically, Candida albicans has been the major species responsible for causing candidiasis in immunocompromised and immunocompetent patients. However, infections (candidiasis) due to C. tropicalis have increased dramatically on a global scale thus proclaiming this organism to be an emerging pathogenic yeast. The reasons for this organism's dominance and its resistance to fluconazole have been difficult to elucidate. In addition, the mechanism of this organism's pathogenic
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Al-Dhaheri, Rawya S., and L. Julia Douglas. "Absence of Amphotericin B-Tolerant Persister Cells in Biofilms of Some Candida Species." Antimicrobial Agents and Chemotherapy 52, no. 5 (2008): 1884–87. http://dx.doi.org/10.1128/aac.01473-07.

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ABSTRACT Biofilms and planktonic cells of five Candida species were surveyed for the presence of persister (drug-tolerant) cell populations after exposure to amphotericin B. None of the planktonic cultures (exponential or stationary phase) contained persister cells. However, persisters were found in biofilms of one of two strains of Candida albicans tested and in biofilms of Candida krusei and Candida parapsilosis, but not in biofilms of Candida glabrata or Candida tropicalis. These results suggest that persister cells cannot solely account for drug resistance in Candida biofilms.
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da Silva, Cecília Rocha, João Batista de Andrade Neto, José Júlio Costa Sidrim, et al. "Synergistic Effects of Amiodarone and Fluconazole on Candida tropicalis Resistant to Fluconazole." Antimicrobial Agents and Chemotherapy 57, no. 4 (2013): 1691–700. http://dx.doi.org/10.1128/aac.00966-12.

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ABSTRACTThere have recently been significant increases in the prevalence of systemic invasive fungal infections. However, the number of antifungal drugs on the market is limited in comparison to the number of available antibacterial drugs. This fact, coupled with the increased frequency of cross-resistance, makes it necessary to develop new therapeutic strategies. Combination drug therapies have become one of the most widely used and effective strategies to alleviate this problem. Amiodarone (AMD) is classically used for the treatment of atrial fibrillation and is the drug of choice for patien
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Kaur, Ravinder, Sahiba Jaggi, Megh Singh Dhakad, and Deepti Rawat. "An etiological and antifungal profile of candidemia in children." International Journal Of Community Medicine And Public Health 6, no. 9 (2019): 3899. http://dx.doi.org/10.18203/2394-6040.ijcmph20193990.

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Background: Candidemia causing increased mortality rates and emergence of antifungal drug resistance needs an urgent intervention to salvage immunocompromised and severely ill patients. This study aimed to isolate and identify Candida species and evaluate their antifungal susceptibility profile from blood stream infections in children.Methods: Fungal cultures from blood recovered positive for yeasts were subcultured on Sabouraud dextrose agar. Suspected purified colonies of Candida were confirmed and identified upto species level by both conventional and automated techniques. Antifungal suscep
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Boonsilp, Siriphan, Anchalee Homkaew, Uraporn Phumisantiphong, Daranee Nutalai, and Thanwa Wongsuk. "Species Distribution, Antifungal Susceptibility, and Molecular Epidemiology of Candida Species Causing Candidemia in a Tertiary Care Hospital in Bangkok, Thailand." Journal of Fungi 7, no. 7 (2021): 577. http://dx.doi.org/10.3390/jof7070577.

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Candida species represent a common cause of bloodstream infection (BSI). Given the emergence of non-albicans Candida (NAC) associated with treatment failure, investigations into the species distribution, fungal susceptibility profile, and molecular epidemiology of pathogens are necessary to optimize the treatment of candidemia and explore the transmission of drug resistance for control management. This study evaluated the prevalence, antifungal susceptibility, and molecular characteristics of Candida species causing BSI in a tertiary-level hospital in Bangkok, Thailand. In total, 54 Candida is
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Mohamed, Nurul Azmawati, Siva Gowri Pathmanathan, Hazilawati Hussin, and Adilahtul Bushro Zaini. "Distribution and Antifungal Susceptibility Pattern of Candida species at a Tertiary Hospital in Malaysia." Journal of Infection in Developing Countries 12, no. 02 (2018): 102–8. http://dx.doi.org/10.3855/jidc.9634.

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Introduction: Invasive Candida infections cause significant mortality and morbidity worldwide. Information on recent trends in species distribution and antifungal resistance in local settings is essential.
 Methodology: Yeast isolates identified through standard culture methods throughout 2014 and 2015 from Hospital Ampang, Malaysia were retrospectively studied. The antifungal susceptibility of Candida species was determined using colorimetric broth microdilution method and MIC values interpreted according to CLSI breakpoints.
 Results: Out of all the 149 yeast cultures collected, mo
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Dissertations / Theses on the topic "Drug Resistance, Candida tropicalis"

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Silva, CecÃlia Rocha da. "Efeito da combinaÃÃo de amiodarona com fluconazol, in vitro, frente a isolados de C.tropicalis resistentes ao fluconazol: novos olhares para antigos fÃrmacos." Universidade Federal do CearÃ, 2012. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=10141.

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Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico<br>Candida tropicalis à uma levedura diplÃide causadora de infecÃÃes superficiais e/ou sistÃmicas, as quais podem ser adquiridas de maneira endÃgena ou exÃgena e podem acometer diversos ÃrgÃos. No Brasil, dentre as espÃcies de Candida spp., a C.tropicalis à a segunda espÃcie mais comumente isolada e no Cearà à pouco estudada. Nos dias atuais, temos vivenciado um aumento significativo das infecÃÃes fÃngicas invasivas. PorÃm as drogas antifÃngicas disponÃveis no mercado sÃo restritas a um pequeno nÃmero quando comparadas as antibacter
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Magri, Marcello Mihailenko Chaves. "Caracterização molecular e perfil de sensibilidade de Candida tropicalis isoladas em corrente sanguínea e cateter de pacientes internados em hospitais de ensino." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/5/5134/tde-07022013-115954/.

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Infecções causadas por Candida tropicalis (C. tropicalis) são associados à elevada morbi-mortalidade, e foram consideradas como importantes causas de infecção de corrente sanguínea no Hospital das Clínicas da Faculdade de Medicina da Universidade de São Paulo (HCFMUSP) de março de 1998 a março de 2001. Adicionalmente, são responsáveis pelo aumento do tempo e dos custos de hospitalização e necessidade de cuidados intensivos. Esse estudo tem como objetivo a caracterização molecular e perfil de sensibilidade de 61 isolados de C. tropicalis a partir de candidemias no HCFMUSP e Universidade Estadua
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Makwana, Amit. "Studies on the N-dealkylation of codeine by Candida tropicalis." Thesis, University of Bath, 1990. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.278237.

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Al-Dhaheri, Rawya. "Drug resistance and apoptosis in Candida biofilms." Thesis, University of Glasgow, 2010. http://theses.gla.ac.uk/1940/.

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Candida species are commonly part of the normal flora in humans; however, they are opportunistic fungal pathogens that are capable of causing a variety of infections in hospitalized and immunocompromised individuals. These infections range from superficial to systemic ones. Many Candida infections involve biofilm formation on the surfaces of implanted devices, such as catheters and prostheses, or host tissues. Candida biofilms are resistant to a range of antifungal agents in current clinical use but the basis of this drug resistance is not clear. The aim of this project was to investigate poss
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Barbosa, Mónica Rodrigues. "Evolution of antifungal drug resistance in Candida albicans." Master's thesis, Universidade de Aveiro, 2014. http://hdl.handle.net/10773/15332.

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Mestrado em Biologia Molecular e Celular<br>The human pathogen Candida albicans is characterized by the presence of a hybrid tRNA (tRNACAGSer) that can be aminoacylated by both LeuRS and SerRS. This tRNA is responsible for the ambiguity of the CUG codon that can be decoded as serine (97%) and Leucine (3%). Previous studies showed that the level of ambiguity is variable depending on the growth condition of the fungus. These studies also revealed that strains mistranslating at a higher level grew better in the presence of azoles, particularly in the presence of fluconazole. To analyse the effec
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Epp, Elias. "Functional genomics in Candida albicans: tackling drug resistance and morphology." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=104697.

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The human fungal pathogen Candida albicans is a diploid organism that lacks a complete sexual cycle, thus making functional genetics challenging. Consequently, linking a function to a gene often relies on predictions from other model organisms. In this thesis, I have explored different genetic strategies to address two important aspects of the biology of C. albicans, drug resistance and morphology, which are associated with virulence. Using a reverse genetics approach based on the model Saccharomyces cerevisiae, I first identify and validate a new drug target in C. albicans. Next, I use forwar
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Alloush, Habib Mahmoud. "The molecular basis of resistance to 5-fluorocytosine in Candida albicans." Thesis, University of Cambridge, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240107.

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Dovigo, Lívia Nordi. "Efetividade da terapia fotodinâmica na inativação de Candida spp /." Araraquara : [s.n.], 2007. http://hdl.handle.net/11449/97303.

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Orientador: Ana Cláudia Pavarina<br>Banca: Carlos Eduardo Vergani<br>Banca: Vanderlei Salvador Bagnato<br>Resumo: O surgimento da resistência antifúngica aos tratamentos convencionais tem proporcionado o desenvolvimento de novas modalidades terapêuticas para o tratamento da candidose bucal. Nesse contexto, a utilização da PDT vem sendo sugerida como método alternativo para a inativação de microrganismos patogênicos. Este estudo avaliou a efetividade da PDT na inativação de C. albicans e C. glabrata, ATCC e resistente a fluconazol, por meio da utilização do e da iluminação com LEDs de agente fo
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Księżopolska, Ewa 1987. "Genomic changes driving the acquisition of multidrug resistance in Candida glabrata." Doctoral thesis, TDX (Tesis Doctorals en Xarxa), 2021. http://hdl.handle.net/10803/672386.

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This project was set out to broaden our knowledge of the processes driving the emergence of antifungal drug resistance in an opportunistic yeast pathogen Candida glabrata. More precisely, we sought to find the mutational signatures of drug resistance and cross-resistance to representatives of two clinically important antifungal drug classes, azoles and echinocandins. By combining in vitro evolution with phenotypic screening and comparative genomics analysis we investigated the relationship between genomic changes, fitness and drug susceptibility to shed light on the cellular mechanisms and evo
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MacPherson, Sarah A. "Roles of zinc cluster protein transcription factors in conferring drug resistance in Saccharomyces cerevisiae and Candida albicans." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=100648.

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Multi-drug or pleiotropic drug resistance (PDR) is a universally acquired mechanism that is evolutionarily conserved. Saccharomyces cerevisiae serves as an excellent model organism for studying this phenomenon in pathogenic fungi, such as Candida albicans. In yeast, PDR is most often mediated by the overexpression of membrane-spanning transporter proteins that act as drug efflux pumps. Their expression is controlled by an intricate network of transcription factors, including many members of the Gal4p superfamily of zinc cluster proteins. Pdr1p, Pdr3p, Pdr8p, Stb5p, Yrm1p, and Yrr1p are all tra
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Books on the topic "Drug Resistance, Candida tropicalis"

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Hube, Bernhard, and Oliver Kurzai. Candida species. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0011.

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Most pathogenic Candida species are members of the microbiota, but also cause superficial or invasive infections. C. albicans is predominant, followed by C. glabrata, C. parapsilosis, and C. tropicalis. C. albicans is polymorphic and grows as yeast, pseudohyphae, or hyphae. The cell wall has multiple functions in pathogenesis. Metabolism and nutrient up-take strategies facilitate growth in multiple niches within the host. Drug resistance is an intrinsic property of C. glabrata and C. krusei, but can be developed by C. albicans and other Candida species during antifungal therapy. Pathogenicity
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Cowen, Leah Elizabeth. Population genomics of drug resistance in the pathogenic yeast Candida Albicans. 2002.

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J, Ernst Erika, and Rogers P. David, eds. Antifungal agents: Methods and protocols. Humana Press, 2005.

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Ernst, Erika J. Antifungal Agents (Methods in Molecular Medicine). Humana Press, 2005.

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Book chapters on the topic "Drug Resistance, Candida tropicalis"

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Akins, Robert A., and Jack D. Sobel. "Antifungal Targets, Mechanisms of Action, and Resistance in Candida albicans." In Antimicrobial Drug Resistance. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-46718-4_30.

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Sobel, Jack D., and R. A. Akins. "The Role of Resistance in Candida Infections: Epidemiology and Treatment." In Antimicrobial Drug Resistance. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47266-9_18.

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Sobel, J. D., and R. A. Akins. "The Role of Resistance in Candida Infections: Epidemiology and Treatment." In Antimicrobial Drug Resistance. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-595-8_18.

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Akins, Robert A., and Jack D. Sobel. "Antifungal Targets, Mechanisms of Action, and Resistance in Candida albicans." In Antimicrobial Drug Resistance. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-180-2_29.

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Lewis, Russell E., and Dimitrios P. Kontoyiannis. "The Impact of Antifungal Drug Resistance in the Clinic." In Candida and Candidiasis. ASM Press, 2014. http://dx.doi.org/10.1128/9781555817176.ch23.

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Sanglard, Dominique. "Mechanisms of Drug Resistance in Candida albicans." In Candida albicans: Cellular and Molecular Biology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-50409-4_15.

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Manzoor, Nikhat. "Candida Pathogenicity and Alternative Therapeutic Strategies." In Pathogenicity and Drug Resistance of Human Pathogens. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-32-9449-3_7.

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Prasad, Rajendra, Remya Nair, and Atanu Banerjee. "Emerging Mechanisms of Drug Resistance in Candida albicans." In Yeasts in Biotechnology and Human Health. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-13035-0_6.

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Prasad, Rajendra, Manpreet Kaur Rawal, and Abdul Haseeb Shah. "Candida Efflux ATPases and Antiporters in Clinical Drug Resistance." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-25304-6_15.

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Ibe, Chibuike, Louise A. Walker, Neil A. R. Gow, and Carol A. Munro. "Unlocking the Therapeutic Potential of the Fungal Cell Wall: Clinical Implications and Drug Resistance." In Candida albicans: Cellular and Molecular Biology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-50409-4_16.

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