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Journal articles on the topic 'Duchenne'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Senatorova, A., and I. Khodun. "ERB-DUCHENNE PALSY (CASE REPORT)." Inter Collegas 5, no. 2 (2018): 80–83. http://dx.doi.org/10.35339/ic.5.2.80-83.

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ERB–DUCHENNE PALSY (case study)Senatorova A.V., Khodun I.I.The frequency of damage to the brachial plexus is 0.5-2 per 1000 live births. Most of them (about 90 % cases) are Erb-Duchesnne palsy. Birth trauma of the brachial plexus occurs mainly in in full-term newborns. Correct diagnosis of Erb-Duchesnne palsy allows to avoid long-term complications as late treatment leads to disability. The article presents a clinical observation of Erb-Duchesnne palsy in a newborn patient, who was diagnosed on the first day of life. Conservative therapy was an effective strategy of baby’s recovery.Key words:

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2

Goldblatt, David. "A Gentleman of Bologna Duchenne, Duchesne, etc." Seminars in Neurology 8, no. 01 (1988): 115–16. http://dx.doi.org/10.1055/s-2008-1041363.

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3

Parent, André. "Duchenne De Boulogne: A Pioneer in Neurology and Medical Photography." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 32, no. 3 (2005): 369–77. http://dx.doi.org/10.1017/s0317167100004315.

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ABSTRACT:Guillaume-Benjamin-Amand Duchenne was born 200 years ago in Boulogne-sur-Mer (Pas-de-Calais, France). He studied medicine in Paris and became a physician in 1831. He practiced general medicine in his native town for about 11 years and then returned to Paris to initiate pioneering studies on electrical stimulation of muscles. Duchenne used electricity not only as a therapeutic agent, as it was commonly the case earlier in the 19th century, but chiefly as a physiological investigation tool to study the anatomy of the living body. Without formal appointment he visited hospital wards acro

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4

Ibrahim Sory, P., T. Sidi, L. Guida, et al. "Dystrophie Musculaire de Duchenne: Aspects cliniques, biologiques et évolutifs à propos de cinq cas dans le service de Rhumatologie au CHU du Point G." Rhumatologie Africaine Francophone 6, no. 2 (2024): 18–23. http://dx.doi.org/10.62455/raf.v6i2.53.

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Résumé  La dystrophie musculaire de Duchenne (DMD) due à la non expression de la dystrophine est liée au chromosome X. Décrite au 19e siècle, est la plus courante dystrophie musculaire de l’enfant [1, 2]. L’incidence est estimée à 30 cas pour 100 000 naissances [1, 2]. But- étudier les caractères cliniques, biologiques et évolutifs de la dystrophie musculaire de Duchenne. Patients et Méthodes : Il s’est agi d’une étude rétrospective portant sur 5 dossiers de DMD, colligés en 7 ans. Résultats Nous rapportons cinq dossiers de garçons colligés entre 2005 et 201

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5

Lwi, Sandy J., James J. Casey, Alice Verstaen, Dyan E. Connelly, Jennifer Merrilees, and Robert W. Levenson. "Genuine Smiles by Patients During Marital Interactions are Associated with Better Caregiver Mental Health." Journals of Gerontology: Series B 74, no. 6 (2018): 975–87. http://dx.doi.org/10.1093/geronb/gbx157.

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Abstract Objective Providing care for a spouse with dementia is associated with an increased risk for poor mental health. To determine whether this vulnerability in caregivers is related to the expression of positive emotion, we examined 57 patients with Alzheimer’s disease and behavioral variant frontotemporal dementia and their spouses as they discussed a marital conflict. Method Facial behavior during the discussion was objectively coded to identify Duchenne (i.e., genuine) smiles and non-Duchenne (i.e., polite) smiles. Caregiver mental health was measured using the Medical Outcomes Survey.

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Thibault, Pascal, Manon Levesque, Pierre Gosselin, and Ursula Hess. "The Duchenne Marker is Not a Universal Signal of Smile Authenticity – But it Can Be Learned!" Social Psychology 43, no. 4 (2012): 215–21. http://dx.doi.org/10.1027/1864-9335/a000122.

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The Duchenne marker has been proposed as a universal marker of smile authenticity. However, Elfenbein, Beaupré, Levesque, and Hess (2007 ) found that, whereas Canadians typically show the Duchenne marker when posing happiness, Gabonese do not. We therefore investigated whether the Duchenne marker is perceived as a marker of smile authenticity by Gabonese and by Mainland Chinese living in Quebec, Canada. The results show that Gabonese do not use the Duchenne marker to assess smile authenticity at all. Mainland Chinese immigrants to Quebec showed sensitivity to the Duchenne marker only when judg

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7

Soim, Aida, Bailey Wallace, Nedra Whitehead, et al. "Health Profile of Preterm Males With Duchenne Muscular Dystrophy." Journal of Child Neurology 36, no. 12 (2021): 1095–102. http://dx.doi.org/10.1177/08830738211047019.

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In this retrospective cohort study, we characterize the health profile of preterm males with Duchenne muscular dystrophy. Major clinical milestones (ambulation cessation, assisted ventilation use, and onset of left ventricular dysfunction) and corticosteroids use in males with Duchenne muscular dystrophy identified through a population-based surveillance system were analyzed using Kaplan-Meier survival curves and Cox proportional hazards modeling. The adjusted risk of receiving any respiratory intervention among preterm males with Duchenne muscular dystrophy was 87% higher than among the corre

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8

Denetclaw, W. F., G. Bi, D. V. Pham, and R. A. Steinhardt. "Heterokaryon myotubes with normal mouse and Duchenne nuclei exhibit sarcolemmal dystrophin staining and efficient intracellular free calcium control." Molecular Biology of the Cell 4, no. 9 (1993): 963–72. http://dx.doi.org/10.1091/mbc.4.9.963.

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Duchenne and mdx muscle tissues lack dystrophin where it normally interacts with glycoproteins in the sarcolemma. Intracellular free calcium ([Ca2+]i) is elevated in Duchenne and mdx myotubes and is correlated with abnormally active calcium-specific leak channels in dystrophic myotubes. We fused Duchenne human and normal mouse myoblasts and identified heterokaryon myotubes by Hoechst 33342 staining to measure the degree to which dystrophin introduced by normal nuclei could incorporate throughout the myotube at the sarcolemma and restore normal calcium homeostasis. Dystrophin expression in myot

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9

Meißner, Thomas. "Duchenne-Muskeldystrophie." MMW - Fortschritte der Medizin 157, no. 13 (2015): 81. http://dx.doi.org/10.1007/s15006-015-3369-7.

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10

Sekelj Fureš, J., and V. Đuranović. "Female Duchenne." European Journal of Paediatric Neurology 21 (June 2017): e229. http://dx.doi.org/10.1016/j.ejpn.2017.04.1244.

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11

Heutinck, Lotte, Nadine van Kampen, Merel Jansen, and Imelda J. M. de Groot. "Physical Activity in Boys With Duchenne Muscular Dystrophy Is Lower and Less Demanding Compared to Healthy Boys." Journal of Child Neurology 32, no. 5 (2017): 450–57. http://dx.doi.org/10.1177/0883073816685506.

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This study describes the amount of physical activity and perception of physical activity in boys with Duchenne muscular dystrophy (DMD) compared to healthy boys. A questionnaire described 6 domains of physical activity. Four Duchenne muscular dystrophy subgroups were made: early and late ambulatory, nonambulatory with relative good, or limited arm function. Eighty-four boys with Duchenne muscular dystrophy (15.0 ± 6.4 years) and 198 healthy boys (14.0 ± 4.3 years) participated. Daily activities were more passive for boys with Duchenne muscular dystrophy. Physical activity was less and low dema

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12

Mwiinga, Fair Banji, Roster Chihwaka Malimba, Mutinta Mirriam Nzima, et al. "Early detection and rehabilitation in Erb-Duchenne paralysis before age 1 year: A Case Report From Lusaka Zambia." Medical Journal of Zambia 50, no. 2 (2023): 192–96. http://dx.doi.org/10.55320/mjz.50.2.353.

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Erb-Duchenne paralysis is a neurological condition characterized by paralysis of the arm which occurs due to injury of the upper trunk of C5-C6 of the brachial plexus and can lead to disturbances in movement and sensation. Erb-Duchenne paralysis commonly presents with a “Waiter’s Tip” deformity characterized by elbow extension, medial rotation of the arm, forearm pronation, and wrist flexion. Management of Erb-Duchenne paralysis may involve strengthening exercises, range of motion exercises, manual therapy, and neuromuscular electrical stimulation. However, in most cases, the diagnosis of Erb-

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13

Robert, D., TN Willig, P. Leger, and J. Paulus. "Long-term nasal ventilation in neuromuscular disorders: report of a consensus conference." European Respiratory Journal 6, no. 4 (1993): 599–606. http://dx.doi.org/10.1183/09031936.93.06040599.

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Following the colloquium entitled "Nasal Ventilation and Neuromuscular Disease", which took place in Lyon, France, October 24, 1991, a group of authorities on neuromuscular disorders met to establish a consensus concerning the application of this technique, in the clinical setting, for patients with Duchenne's muscular dystrophy, non-Duchenne myopathies and the spinal muscular atrophies. This report summarizes recommendations issuing from this conference. The conclusions drawn from this work should not be applied to patients with other diagnoses.

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14

Khatri, Ravi Shankar, Mridul Ranajan, and Shalini . "A COMPARATIVE AYURVEDIC REVIEW OF ETIOPATHOGENESIS OF DUCHENNE MUSCULAR DYSTROPHY (INHERITED DISORDER)." International Journal of Research in Ayurveda and Pharmacy 12, no. 1 (2021): 124–25. http://dx.doi.org/10.7897/2277-4343.120127.

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Duchenne muscular dystrophy (DMD) is an inherited disorder with severe progressive muscle weakness. In Ayurveda, Adibala Pravritta Vyadhi are also known as inherited diseases that caused by Matruja beeja dushti (Shonita) and Pitruja beeja dushti (Shukra). Duchenne muscular dystrophy (DMD) has been classified under Adibala Pravritta Vyadhi as per Ayurveda. The main objective of this article is to describe the various aspect of etiopathogenesis of Duchenne muscular dystrophy (DMD) as per Ayurvedic literature. This article will be helpful to making the Nidana (Diagnosis) as per Ayurveda and also

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15

Sanadhya, Anuradha, Ritvika Jyani, Suresh Goyal, Neha Asora, and Mukesh Kumar Gurjar. "Duchenne muscular dystrophy in a female with x-autosome translocation." International Journal of Contemporary Pediatrics 8, no. 4 (2021): 770. http://dx.doi.org/10.18203/2349-3291.ijcp20211094.

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Duchenne’s muscular dystrophy is the most common hereditary neuromuscular disease, which affects all races. Its classical characteristic clinical features being progressive muscular weakness, intellectual impairment and hypertrophy of the calves with proliferation of connective tissue and progressive fibrosis in muscles. As the disease is inherited as an X-linked recessive trait, thus females not manifesting the disease and acting as carriers only, as second X chromosome prevents the manifestation of disease. We report a case of classical Duchenne muscular dystrophy in 10 year old female with

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16

Melis Gönülal, Melis Gönülal, and Didem Didar Balcı Didem Didar Balcı. "ADALİMUMAB: USAGE İN A CASE WİTH PSORİASİS AND DUCHENNE MUSCULAR DYSTROPHY." INTERNATIONAL JOURNAL OF INNOVATIVE MEDICINE & HEALTHCARE 01, no. 01 (2022): 04–07. http://dx.doi.org/10.55858/ijimh01012022-04.

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There are different biological treatments for psoriasis and one of these treatments is adalimumab. Here we want to present a case with psoriasis and Duchenne muscular dystrophy treated with adalimumab. A 23-year-old male patient with psoriasis and Duchenne muscular dystrophy applied to our dermatology clinic. At the sixth week control of the patient we achieved PASI 90 response. Duchenne muscular dystrophy is a rare disease and its coexistence with psoriasis is a very rare status. No similar case report was found in the literature. We wanted to present both this rare status and psoriasis progr

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17

Ihmsen, Harald, Joachim Schmidt, Helmut Schwilden, Hubert J. Schmitt, and Tino Muenster. "Influence of Disease Progression on the Neuromuscular Blocking Effect of Mivacurium in Children and Adolescents with Duchenne Muscular Dystrophy." Anesthesiology 110, no. 5 (2009): 1016–19. http://dx.doi.org/10.1097/aln.0b013e31819daf31.

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Background Studies with nondepolarizing neuromuscular blocking agents showed a delayed onset and prolonged recovery in patients with Duchenne muscular dystrophy. The objective of this study was to investigate if these alterations depend on disease progression. Methods The authors studied 11 children (6-9 yr) with moderate Duchenne muscular dystrophy, 11 adolescents (12-16 yr) with advanced Duchenne muscular dystrophy, and 2 age-matched control groups of 8 patients each (5-9 and 10-17 yr). Anesthesia was performed with propofol and remifentanil. Patients received a single intravenous dose of 0.

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18

Sarvutiene, Julija, Arunas Ramanavicius, Simonas Ramanavicius, and Urte Prentice. "Advances in Duchenne Muscular Dystrophy: Diagnostic Techniques and Dystrophin Domain Insights." International Journal of Molecular Sciences 26, no. 8 (2025): 3579. https://doi.org/10.3390/ijms26083579.

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Abnormalities in X chromosomes, either numerical or structural, cause X-linked disorders, such as Duchenne muscular dystrophy (DMD). Recent molecular and cytogenetic techniques can help identify DMD gene mutations. The accurate diagnosis of Duchenne is crucial, directly impacting patient treatment management, genetics, and the establishment of effective prevention strategies. This review provides an overview of X chromosomal disorders affecting Duchenne and discusses how mutations in Dystrophin domains can impact detection accuracy. Firstly, the efficiency and use of cytogenetic and molecular

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19

Werneck, Lineu Cesar, and Eduardo Bonilla. "Distrofina na diferenciação das distrofias de duchenne e becker estudo imuno-histoquímico comparado com o estádio clínico, enzimas séricas e biópsia muscular." Arquivos de Neuro-Psiquiatria 48, no. 4 (1990): 454–64. http://dx.doi.org/10.1590/s0004-282x1990000400009.

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Foram estudados 55 casos de distrofia muscular progressiva (34 Duchenne, 12 Duchenne com distrofina residual e 9 Becker), comparando idade, época de início e tempo de sintomas, graduação na escala de Vignos e Archibald, níveis de enzimas séricas e presença de distrofina nas biópsias musculares por imunofluorescência. A intensidade dos sintomas, gravidade do quadro clínico, proliferação de tecido ccnjuntivo endomisial e infiltração por tecido adiposo estão inversamente relacionadas à quantidade de distrofina presente nas biópsias e, diretamente, à presença de fibras hipertróficas e fibras angul

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20

Sheldon, Kennon M., Mike Corcoran, and Melanie Sheldon. "Duchenne Smiles as Honest Signals of Chronic Positive Mood." Perspectives on Psychological Science 16, no. 3 (2021): 654–66. http://dx.doi.org/10.1177/1745691620959831.

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Chronic positive mood (CPM) has been shown to confer a wide variety of social, functional, and health benefits. Some researchers have argued that humans evolved to feel CPM, which explains why most people report better than neutral mood (the “positivity offset bias”) and why particularly happy people have particularly good outcomes. Here, we argue that the Duchenne smile evolved as an honest signal of high levels of CPM, alerting others to the psychological fitness of the smiler. Duchenne smiles are honest because they express felt positive emotion, making it difficult for unhappy people to pr

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21

Lupu-Merca, Violeta, and Sebastian Vaida. "Benefits of the Duchenne Smile and Positive Emotions. A Systematic Review." Educatia 21, no. 27 (May 14, 2024): 93–102. http://dx.doi.org/10.24193/ed21.2024.27.09.

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The Duchenne smile is the genuine smile characterized by the activation of the muscles around the eyes and mouth. It has been associated, in the specialized literature, with the experience of positive emotions. Through the present work, using a qualitative approach, we aimed to identify and systematize the studies carried out in the period 2010-2023 that investigated the relationship between the Duchenne smile and positive emotions. Following the application of specific keywords, 611 studies were identified, from which, after applying the inclusion-exclusion criteria, 8 studies were included i

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22

Khmyzov, Sergij, Anastasiia Hrytsenko, Genadii Kykosh та Anton Hrytsenko. "BIRTH INJURY, DUCHENNE-ERBʼS OBSTETRIC PALSY. DIAGNOSIS AND TREATMENT (LITERATURE REVIEW)". ORTHOPAEDICS TRAUMATOLOGY and PROSTHETICS, № 3 (18 жовтня 2023): 69–78. http://dx.doi.org/10.15674/0030-59872023369-78.

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Obstetric practice dates back thousands of years, providing assistance to women in labor is often complicated by the rapid course of labor, pelvic presentation of the fetus, shoulder dystocia with a possible clavicle fracture. Damage to CV–CVI roots, classic Duchenne–Erb palsy, accounts for 46 % of the total number of obstetric palsies. Objective. To analyze the scientific and medical literature in order to identify historical scientific and practical information about the study of childbirth injuries, and, in particular, Duchenne–Erb's obstetric palsy. Methods. To study and analyze sources of

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23

Timonen, Anne, Michele Lloyd-Puryear, David M. Hougaard, et al. "Duchenne Muscular Dystrophy Newborn Screening: Evaluation of a New GSP® Neonatal Creatine Kinase-MM Kit in a US and Danish Population." International Journal of Neonatal Screening 5, no. 3 (2019): 27. http://dx.doi.org/10.3390/ijns5030027.

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Duchenne muscular dystrophy (DMD/Duchenne) is a progressive X-linked disease and is the most common pediatric-onset form of muscular dystrophy, affecting approximately 1:5000 live male births. DNA testing for mutations in the dystrophin gene confirms the diagnosis of this disorder. This study involves assessment of screening newborns for DMD using an immunoassay for muscle-type (MM) creatine kinase (CK) isoform—the GSP Neonatal CK-MM kit. Comparisons were made with CK activity determination by fluorescence measurement. In addition, the study evaluated the effect of gestational age, age of infa

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24

Jufan, Akhmad Yun, Djayanti Sari, and Karlina Mahardieni. "DUCHENNE MUSCULER DYSTROPHY." Jurnal Komplikasi Anestesi 3, no. 2 (2023): 47–53. http://dx.doi.org/10.22146/jka.v3i2.7242.

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Duchenne muscular dystrophy merupakan suatu kelainan otot yang sering ditemui. Penyakit ini terpaut pada kromosom X yang disebabkan oleh mutasi gen dystrophin. Gejalanya berupa kelemahan otot proksimal yang berat, bersifat degenerasi progresif dan infi ltrasi lemak ke otot. Efek duchenne muscular dystrophy terhadap otot respirasi dan berhubungan dengan kardio-miopati yang dapat mengarah ke kematian.Dilaporkan anak laki-laki usia 12 tahun dengan diagnosa duchenne muscular dystrophy dd/ Baker’s muscular dystrophy dilakukan prosedur biopsi. Pasien dinilai sebagai status fi sik ASA 2 yang dilakuka

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25

Dubey, Abhishek, Gaurav Dubey, Sambodhan Dhawane, and Rishikesh Sharma. "Duchenne Muscular Disease." American Journal of PharmTech Research 8, no. 5 (2018): 33–54. http://dx.doi.org/10.46624/ajptr.2018.v8.i5.003.

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26

Biggar, W. D. "Duchenne Muscular Dystrophy." Pediatrics in Review 27, no. 3 (2006): 83–88. http://dx.doi.org/10.1542/pir.27-3-83.

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27

Juříková, Lenka, Zdeňka Bálintová, and Jana Haberlová. "Duchenne muscular dystrophy." Neurologie pro praxi 20, no. 3 (2019): 180–82. http://dx.doi.org/10.36290/neu.2019.111.

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28

Kornberg, AndrewJ, and EppieM Yiu. "Duchenne muscular dystrophy." Neurology India 56, no. 3 (2008): 236. http://dx.doi.org/10.4103/0028-3886.43441.

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Sussman, Michael. "Duchenne Muscular Dystrophy." Journal of the American Academy of Orthopaedic Surgeons 10, no. 2 (2002): 138–51. http://dx.doi.org/10.5435/00124635-200203000-00009.

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30

Torreggiani, William C., Iain D. Lyburn, Alison C. Harris, and V. A. Rowley. "Duchenne-Landouzy Dystrophy." American Journal of Roentgenology 174, no. 5 (2000): 1467–68. http://dx.doi.org/10.2214/ajr.174.5.1741467a.

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31

Millichap, J. Gordon. "Duchenne Muscular Dystrophy." Pediatric Neurology Briefs 3, no. 5 (1989): 40. http://dx.doi.org/10.15844/pedneurbriefs-3-5-11.

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Biggar, W. Douglas. "Duchenne Muscular Dystrophy." Pediatrics In Review 27, no. 3 (2006): 83–88. http://dx.doi.org/10.1542/pir.27.3.83.

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33

Bushby, K. "Duchenne Muscular Dystrophy." Journal of Medical Genetics 31, no. 6 (1994): 506. http://dx.doi.org/10.1136/jmg.31.6.506.

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Bundey, S. "Duchenne Muscular Dystrophy." Journal of Medical Genetics 25, no. 2 (1988): 140. http://dx.doi.org/10.1136/jmg.25.2.140.

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Bundey, S. "Duchenne Muscular Dystrophy." Journal of Medical Genetics 26, no. 6 (1989): 416. http://dx.doi.org/10.1136/jmg.26.6.416.

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Heckmatt, J. Z. "Duchenne Muscular Dystrophy." Archives of Disease in Childhood 64, no. 5 (1989): 767. http://dx.doi.org/10.1136/adc.64.5.767.

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McDonald, Craig M., Richard T. Abresch, Gregory T. Carter, et al. "Duchenne Muscular Dystrophy." American Journal of Physical Medicine & Rehabilitation 74, Supplement 1 (1995): S70—S92. http://dx.doi.org/10.1097/00002060-199509001-00003.

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Gomez-Merino, Elia, and John R. Bach. "Duchenne Muscular Dystrophy." American Journal of Physical Medicine & Rehabilitation 81, no. 6 (2002): 411–15. http://dx.doi.org/10.1097/00002060-200206000-00003.

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39

Harper, P. "Duchenne Muscular Dystrophy." Journal of Neurology, Neurosurgery & Psychiatry 50, no. 9 (1987): 1249. http://dx.doi.org/10.1136/jnnp.50.9.1249.

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40

Marles, Hugh C. "Duchenne de Boulogne." History of Photography 16, no. 4 (1992): 395–96. http://dx.doi.org/10.1080/03087298.1992.10442576.

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Scott, Oona M. "Duchenne Muscular Dystrophy." Physiotherapy 76, no. 3 (1990): 138. http://dx.doi.org/10.1016/s0031-9406(10)62140-2.

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Villanova, Marcello, Beatrice Brancalion, and Anokhi D. Mehta. "Duchenne Muscular Dystrophy." American Journal of Physical Medicine & Rehabilitation 93, no. 7 (2014): 595–99. http://dx.doi.org/10.1097/phm.0000000000000074.

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Shieh, Perry B. "Duchenne muscular dystrophy." Current Opinion in Neurology 28, no. 5 (2015): 542–46. http://dx.doi.org/10.1097/wco.0000000000000243.

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Roper, Helen P. "Duchenne muscular dystrophy." Neuromuscular Disorders 14, no. 5 (2004): 346–47. http://dx.doi.org/10.1016/j.nmd.2004.02.001.

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45

Davies, Kay E. "Duchenne muscular dystrophy." Trends in Genetics 3 (January 1987): 231–32. http://dx.doi.org/10.1016/0168-9525(87)90244-7.

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46

Topaloglu, Haluk. "Duchenne Muscular Dystrophy." European Journal of Paediatric Neurology 8, no. 5 (2004): 269. http://dx.doi.org/10.1016/j.ejpn.2004.05.004.

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47

Bolhuis, P. A. "Duchenne muscular dystrophy." Journal of the Neurological Sciences 80, no. 1 (1987): 118–19. http://dx.doi.org/10.1016/0022-510x(87)90230-9.

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48

Bolhuis, P. A. "Duchenne muscular dystrophy." Journal of the Neurological Sciences 90, no. 3 (1989): 348. http://dx.doi.org/10.1016/0022-510x(89)90125-1.

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49

Samson, Kurt. "DUCHENNE MUSCULAR DYSTROPHY." Neurology Today 3, no. 9 (2003): 1. http://dx.doi.org/10.1097/00132985-200309000-00001.

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50

Casademont, Jordi. "Duchenne Muscular Dystrophy." Clinical Neurophysiology 115, no. 10 (2004): 2426. http://dx.doi.org/10.1016/j.clinph.2004.05.006.

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