Relevant bibliographies by topics / Durotaxie

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Durotaxie'

Author: Grafiati
Published: 7 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "Durotaxie"

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Sunyer, Raimon, and Xavier Trepat. "Durotaxis." Current Biology 30, no. 9 (2020): R383—R387. http://dx.doi.org/10.1016/j.cub.2020.03.051.

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Huang, Yuxing, Jing Su, Jiayong Liu, et al. "YAP Activation in Promoting Negative Durotaxis and Acral Melanoma Progression." Cells 11, no. 22 (2022): 3543. http://dx.doi.org/10.3390/cells11223543.

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Directed cell migration towards a softer environment is called negative durotaxis. The mechanism and pathological relevance of negative durotaxis in tumor progression still requires in-depth investigation. Here, we report that YAP promotes the negative durotaxis of melanoma. We uncovered that the RhoA-myosin II pathway may underlie the YAP enhanced negative durotaxis of melanoma cells. Acral melanoma is the most common subtype of melanoma in non-Caucasians and tends to develop in a stress-bearing area. We report that acral melanoma patients exhibit YAP amplification and increased YAP activity.
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Puleo, Julieann I., Sara S. Parker, Mackenzie R. Roman, et al. "Mechanosensing during directed cell migration requires dynamic actin polymerization at focal adhesions." Journal of Cell Biology 218, no. 12 (2019): 4215–35. http://dx.doi.org/10.1083/jcb.201902101.

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The mechanical properties of a cell’s microenvironment influence many aspects of cellular behavior, including cell migration. Durotaxis, the migration toward increasing matrix stiffness, has been implicated in processes ranging from development to cancer. During durotaxis, mechanical stimulation by matrix rigidity leads to directed migration. Studies suggest that cells sense mechanical stimuli, or mechanosense, through the acto-myosin cytoskeleton at focal adhesions (FAs); however, FA actin cytoskeletal remodeling and its role in mechanosensing are not fully understood. Here, we show that the
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Style, R. W., Y. Che, S. J. Park, et al. "Patterning droplets with durotaxis." Proceedings of the National Academy of Sciences 110, no. 31 (2013): 12541–44. http://dx.doi.org/10.1073/pnas.1307122110.

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Hartman, Christopher D., Brett C. Isenberg, Samantha G. Chua, and Joyce Y. Wong. "Vascular smooth muscle cell durotaxis depends on extracellular matrix composition." Proceedings of the National Academy of Sciences 113, no. 40 (2016): 11190–95. http://dx.doi.org/10.1073/pnas.1611324113.

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Mechanical compliance has been demonstrated to be a key determinant of cell behavior, directing processes such as spreading, migration, and differentiation. Durotaxis, directional migration from softer to more stiff regions of a substrate, has been observed for a variety of cell types. Recent stiffness mapping experiments have shown that local changes in tissue stiffness in disease are often accompanied by an altered ECM composition in vivo. However, the importance of ECM composition in durotaxis has not yet been explored. To address this question, we have developed and characterized a polyacr
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Yuehua, YANG, and JIANG Hongyuan. "Research Advances in Cell Durotaxis." 应用数学和力学 42, no. 10 (2021): 999–1007. http://dx.doi.org/10.21656/1000-0887.420265.

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Bueno, Jesus, Yuri Bazilevs, Ruben Juanes, and Hector Gomez. "Wettability control of droplet durotaxis." Soft Matter 14, no. 8 (2018): 1417–26. http://dx.doi.org/10.1039/c7sm01917c.

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Doering, Charles R., Xiaoming Mao, and Leonard M. Sander. "Random walker models for durotaxis." Physical Biology 15, no. 6 (2018): 066009. http://dx.doi.org/10.1088/1478-3975/aadc37.

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Stefanoni, Filippo, Maurizio Ventre, Francesco Mollica, and Paolo A. Netti. "A numerical model for durotaxis." Journal of Theoretical Biology 280, no. 1 (2011): 150–58. http://dx.doi.org/10.1016/j.jtbi.2011.04.001.

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Parida, Lipika, and Venkat Padmanabhan. "Durotaxis in Nematode Caenorhabditis elegans." Biophysical Journal 111, no. 3 (2016): 666–74. http://dx.doi.org/10.1016/j.bpj.2016.06.030.

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Dissertations / Theses on the topic "Durotaxie"

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Tettarasar, Samuel. "Régulation du choix de migration par l’histoire environnementale de la cellule." Electronic Thesis or Diss., université Paris-Saclay, 2023. http://www.theses.fr/2023UPASL154.

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Les cellules peuvent migrer de façon aléatoire ou dirigée. La durotaxie est un phénomène de migration dirigée, si les cellules ont le choix, elles vont préférentiellement migrer vers des environnements durs, plutôt que mous. Il a été montré que les cellules pouvaient retenir des informations de leurs environnements passés, ce qui modifie leurs migrations. Ceci est appelé « mémoire mécanique ». Nous avons montré que la mémoire mécanique avait un impact sur la durotaxie des cellules. De plus, nos résultats suggèrent que les cellules sont capables d’associer des stimuli rencontrés dans leur passé
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STEFANONI, Filippo. "DUROTAXIS MODELLING FOR TISSUE ENGINEERING APPLICATIONS." Doctoral thesis, Università degli studi di Ferrara, 2010. http://hdl.handle.net/11392/2389166.

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Tissue Engineering is a very promising research field for the development of natural biological substitutes that restore damaged tissue functions. Cells play a crucial role in tissue regeneration and repair due to their characteristics of proliferation and differentiation, cell-to-cell interaction, biomolecular production and extracellular matrix formation. In particular cell migration is a phenomenon that is involved in different physiological processes such as morphogenesis, wound healing and new tissue deposition. In the absence of external guiding factors it is essentially a phenomen
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Diego, Íñiguez Javier. "On the theory of cell migration: durotaxis and chemotaxis." Doctoral thesis, Universitat Politècnica de Catalunya, 2013. http://hdl.handle.net/10803/129085.

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Cell migration is a fundamental element in a variety of physiological and pathological processes. Alteration of its regulatory mechanisms leads to loss of adhesion and increased motility, critical steps in the initial stages of metastasis. Consequently, cell migration has become the focus of intensive experimental and theoretical studies; however the understanding many of its mechanisms remains elusive. Cell migration is the result of a periodic sequence of protrusion, adhesion remodeling and contraction stages that leads to directed movement towards external stimuli. The spatio-temporal coord
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Márquez, Rosales Susana Belén. "Modelos físicos de migración en células con morfología no polarizadas." Tesis, Universidad de Chile, 2018. http://repositorio.uchile.cl/handle/2250/168174.

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Magíster en Ciencias, Mención Física<br>En esta tesis se estudian migraciones celulares en dos sistemas diferentes cuyos agentes comparten la característica de estar no polarizados. Se proponen modelos físicos que en combinación con datos experimentales y simulaciones numéricas buscan reproducir los fenómenos observados con el fin de entenderlos con mayor profundidad y plantear nuevas preguntas de estudio. En la primera parte, el sistema considerado es un pez anual en su etapa de epibolía donde se estudia la migración de las \textit{Deep Cell Layer } (DCL) hacia los bordes de las \textit{Enve
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Jain, Gaurav. "Cell Migration on Opposing Rigidity Protein Gradients: Single Cell and Co-culture Studies." Diss., Virginia Tech, 2014. http://hdl.handle.net/10919/70847.

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Cell migration is a complex physiological process that is important from embryogenesis to senescence. In vivo, the migration of cells is guided by a complex combination of signals and cues. Directed migration is typically observed when one of these cues is presented to cells as a gradient. Several studies have been conducted into directed migration on gradients that are purely mechanical or chemical. Our goal was to investigate cellular migratory behavior when cells are presented with a choice and have to choose between increasing substrate rigidity or higher protein concentration. We chose
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Castro, Nava Arturo Verfasser], Laporte Laura [Akademischer Betreuer] De, and Martin [Akademischer Betreuer] [Möller. "A light-modulated hydrogel system to analyze cell durotaxic behavior in a dynamic manner / Arturo Castro Nava ; Laura De Laporte, Martin Möller." Aachen : Universitätsbibliothek der RWTH Aachen, 2021. http://d-nb.info/1238603114/34.

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Book chapters on the topic "Durotaxie"

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Kidoaki, Satoru. "Chapter 12. Manipulation of Durotaxis on a Matrix with Cell-scale Stiffness Heterogeneity." In Biomaterials Science Series. Royal Society of Chemistry, 2022. http://dx.doi.org/10.1039/9781839165375-00265.

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Poh, Chieng Ling. "Computational Studies of Cell Durotaxis on Extracellular Matrix Rigidity Gradients as a Model for Wound Healing and Fibrosis." In IRC-SET 2020. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-9472-4_14.

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Conference papers on the topic "Durotaxie"

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Sochol, Ryan D., Adrienne T. Higa, Randall R. R. Janairo, Annie Chou, Song Li, and Liwei Lin. "Unidirectional cellular durotaxis via microfabricated posts of varying anisotropy." In TRANSDUCERS 2009 - 2009 International Solid-State Sensors, Actuators and Microsystems Conference. IEEE, 2009. http://dx.doi.org/10.1109/sensor.2009.5285397.

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Zhou, Y., T. Guo, S. Z. Yang, Y. Zhu, C. S. Jiang, and H. Peng. "Mitochondrial Fission and Bioenergetics Mediate Human Lung Fibroblast Durotaxis." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a5224.

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Reinhardt, James W., Daniel A. Krakauer, and Keith J. Gooch. "Complex Matrix Remodeling and Durotaxis Can Emerge From Simple Rules for Cell-Matrix Interaction in Agent-Based Models." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14295.

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Using a top-down approach, an agent-based model was developed within Netlogo where cells and extracellular matrix (ECM) fibers were composed of multiple agents to create deformable structures capable of exerting, reacting to and transmitting mechanical forces. Simulated cells remodeled the fibrous matrix to change both the density and alignment of the fibers and migrated within the matrix in ways that are consistent with previous experimental work. Cells compacted the matrix in their pericellular regions much more than the average compaction experienced for the entire matrix. Between pairs of
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Ganzleben, I., M. A. Chrysovergi, T. A. Al-Hilal, et al. "Durotaxis in Lung Fibrosis: Transitioning from In Vitro Mechanisms to In Vivo Imaging." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a5551.

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Chang, Wei-Jen, Nadeen Chahine, and Pen-Hsiu Grace Chao. "Effects of Composite Substrate Microstructure on Fibroblast Morphology and Migration." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53859.

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Many studies have focused on the effects of substrate rigidity on cell traction, migration, and differentiation [1–3]. Most cells are known to migrate toward the stiffer substrate, a phenomenon known as durotaxis. Recent reports have also demonstrated the ‘depth-sensing’ ability of cells on soft hydrogels where cell behaviors on thin gels are more similar to those on stiffer substrates [4–5]. Taking advantage of the high fidelity of microfabrication and soft lithography products, we created novel composite substrates composed of a top layer of collagen hydrogel and an underlying microstructure
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Huang, Alvin. "From Bones to Bricks: Design the 3D Printed Durotaxis Chair and La Burbuja Lamp." In ACADIA 2016: Post-Human Frontiers. ACADIA, 2016. http://dx.doi.org/10.52842/conf.acadia.2016.318.

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Huang, Alvin. "From Bones to Bricks: Design the 3D Printed Durotaxis Chair and La Burbuja Lamp." In ACADIA 2016: Post-Human Frontiers. ACADIA, 2016. http://dx.doi.org/10.52842/conf.acadia.2016.318.

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Engler, Adam J. "Probing Mechanisms of Mechano-Sensitive Differentiation in Mesenchymal Stem Cells." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19184.

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Adult mesenchymal stem cells (MSCs) have recently been shown to be responsive to the properties of their adjacent extracellular niche, most notably physical parameters such as topography and elasticity. Elasticity varies dramatically between tissues that MSCs inhibit, which drives elasticity-based differentiation into neurons, muscle, bone, etc. However within tissues, distinct elasticity gradients, brought on by pathological conditions, e.g. myocardial infarction ∼ 8.67 ± 1.50 kPa/mm, or through normal tissue variation, e.g. 0.58 ± 0.88 kPa/mm, could drive MSC migration. In fact, MSCs appear
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