Academic literature on the topic 'Dutch elm disease. Insecticides'

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Journal articles on the topic "Dutch elm disease. Insecticides"

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Mehrabioun Mohammadi, Marzieh, Narges Ahmadi, and Mahdi Arzanlou. "Dutch elm disease." Plant Pathology Science 9, no. 1 (2020): 91–100. http://dx.doi.org/10.29252/pps.9.1.91.

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Hubbes, M. "The American elm and Dutch elm disease." Forestry Chronicle 75, no. 2 (1999): 265–73. http://dx.doi.org/10.5558/tfc75265-2.

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Shortly after World War I, a new disease previously unknown among elms emerged in Holland. It spread rapidly from Europe to Great Britain (1927), United States (1930), and Canada (1945), killing millions of elms. The disease known, as Dutch elm disease (DED) is a wilt disease, caused by the fungus Ophiostoma ulmi. It is transmitted from tree to tree by elm bark beetles (scolytid) vectors. Numerous attempts to control the disease have concentrated on the reduction of insect vector populations, exploitation of natural host resistance, extensive application of fungicides and integrated pest management. In spite of these efforts in Canada, the disease continues to migrate westwards threatening the elm populations in Saskatchewan and Alberta. Today there are approximately 700 000 elm shade trees in cities and towns across Canada and their value exceeds $2.5 billion dollars.With the advance of molecular biology new, powerful tools are now available to study, in greater detail, the molecular and biochemical mechanisms of the DED pathogen, with particular reference to the mechanisms that induce host defenses. A glycoprotein has been isolated and identified such that when injected either in liquid or pellet form into the elm tree, significantly reduced the wilting symptoms of both five-year old elm seedlings and 10 cm diameter trees. The elicitor induces a chain of defensive reactions that prevent the rapid spread of the fungus within the vascular system of the host. Key words: Ophiostoma ulmi, elm bark beetle vectors, induced resistance, chemical control, RFLP, mitochondrial DNA, ribosomal DNA, virus-like DNA, resistance breeding
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Sherald, James L., Frank S. Santamour Jr., Ravindra K. Hajela, Neerja Hajela, and Mariam B. Sticklen. "A Dutch elm disease resistant triploid elm." Canadian Journal of Forest Research 24, no. 4 (1994): 647–53. http://dx.doi.org/10.1139/x94-087.

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A naturally occurring triploid elm hybrid was found in the American elm, Ulmusamericana L., planting on the National Mall in Washington, D.C. Chromosome examinations of mitosis in root tips and meiosis in pollen mother cells showed a chromosome complement of 2n = 3x = 42. The chromosome alignment at meiotic metaphase I was predominantly 14 bivalents and 14 univalents, indicating that one parent was an American elm, which contributed the bivalents through autosyndetic pairing. When DNA underwent restriction digestion with HindIII and probed with a 9.5-kilobase cloned ribosomal DNA fragment from pea, Pisumsativum L., the triploid hybrid showed a band not found in American elms. Seed from the open-pollinated parent tree had low viability and seedlings were highly variable in height, leaf size, and shape. The small leaves of some seedlings suggest that the other parent was a species with leaves smaller than American elm. The hybrid was found to be resistant to Ophiostomaulmi (Buis.) Nannf. When twig crotches were inoculated, only 14% developed wilt compared with 63% in the American elm. None of the 22 hybrid trees inoculated developed systemic wilt compared with 8 of the 18 American elms inoculated. The hybrid, which is easily propagated by softwood cuttings, has rapid growth, good crown structure, and many characteristics of the American elm.
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Moller, Anders Pape. "Elm, Ulmus glabra, Leaf Asymmetry and Dutch Elm Disease." Oikos 85, no. 1 (1999): 109. http://dx.doi.org/10.2307/3546796.

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Scheffer, R. J., J. G. W. F. Voeten, and R. P. Guries. "Biological Control of Dutch Elm Disease." Plant Disease 92, no. 2 (2008): 192–200. http://dx.doi.org/10.1094/pdis-92-2-0192.

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Fox, Jeffrey L. "Assessing the Dutch Elm Disease Backlash." Nature Biotechnology 5, no. 10 (1987): 1002–4. http://dx.doi.org/10.1038/nbt1087-1002.

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Perry, I., and P. D. Moore. "Dutch elm disease as an analogue of Neolithic elm decline." Nature 326, no. 6108 (1987): 72–73. http://dx.doi.org/10.1038/326072a0.

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Santini, A., F. Pecori, A. Pepori, and A. Brookes. "‘Morfeo’ Elm: a new variety resistant to Dutch elm disease." Forest Pathology 42, no. 2 (2011): 171–76. http://dx.doi.org/10.1111/j.1439-0329.2011.00737.x.

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Ďurkovič, Jaroslav, Ingrid Čaňová, Rastislav Lagaňa, et al. "Leaf trait dissimilarities between Dutch elm hybrids with a contrasting tolerance to Dutch elm disease." Annals of Botany 111, no. 2 (2012): 215–27. http://dx.doi.org/10.1093/aob/mcs274.

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Wilson, B. A., J. E. Luther, and T. D. T. Stuart. "Spectral Reflectance Characteristics of Dutch Elm Disease." Canadian Journal of Remote Sensing 24, no. 2 (1998): 200–205. http://dx.doi.org/10.1080/07038992.1998.10855239.

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Dissertations / Theses on the topic "Dutch elm disease. Insecticides"

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Atkins, P. M. "Dutch elm disease : Some aspects of its biology and control." Thesis, University of Salford, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.376861.

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Bates, Malcolm Robert. "DNA polymorphism in the Dutch Elm disease fungus Ophiostoma ulmi." Thesis, Imperial College London, 1990. http://hdl.handle.net/10044/1/47767.

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Mitchell, Andrew George. "Population structures and interaction between the aggressive and non-aggressive subgroups of Ophiostoma ulmi." Thesis, University of Bath, 1988. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234084.

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Blumenstein, Kathrin. "Endophytic fungi in Elms : implications for the integrated management of Dutch Elm Disease." Thesis, Bangor University, 2015. https://research.bangor.ac.uk/portal/en/theses/endophytic-fungi-in-elmsimplications-for-the-integrated-management-of-dutch-elm-disease(3265a1c5-9910-4ed1-9d1e-c569b68d709d).html.

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Integrated pest management calls for new biocontrol solutions in management of forest diseases. Endophytic fungi that are commonly found in tree tissue may have potential in biocontrol. However, the links between endophyte status and disease tolerance are still unclear, and we know little about the mechanisms by which the endophytes can influence tree pathogens. The first goal of the thesis was to compare the endophyte status in elm (Ulmus spp.) trees with low vs. high susceptibility to Dutch elm disease (DED), caused by Ophiostoma fungi, and to find correlations between endophytes and the susceptibility pattern of the trees. The second goal was to investigate the potential mechanisms of antagonism by the endophytes towards the pathogen. Thus, endophytes were isolated from leaves, bark and xylem of elms that differed in DED susceptibility. The isolates were screened for their potential to counteract the pathogen in dual cultures. Selected strains were investigated using Phenotype MicroArrays to obtain the substrate utilization profiles that reflect the endophytes’ ability to compete with the pathogen for a nutritional niche. To test for a protective effect against the disease, promising isolates were injected into young elms. Preliminary analyses were done to identify the extracellular chemicals that some of the endophytes released into the growth medium. The results showed that the frequency and diversity of endophytes was higher in xylem of elms with high susceptibility to DED. Some endophytes deadlocked the pathogen with extracellular chemicals in vitro, while others had a faster growth rate. Several endophytes were able to utilize substrates more effectively than the pathogen. A preventive treatment with endophytes protected elms against DED, but the effect was unstable across years. Bioactive fungal extracts had a complex chemical profile, and the individual compounds in the extracts remain to be identified. Because endophytes antagonized the pathogen through different mechanisms, I suggest that an endophytebased biocontrol of DED could be best achieved through a synergistic effect of several endophyte strains.
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Cole, Thomas Edwin. "Molecular studies of virus-like dsRNAs in a diseased isolate of Ophiostoma novo-ulmi." Thesis, Imperial College London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327031.

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Oghiakhe, Sunday. "Biology and Management of the Dutch Elm Disease Vector, Hylurgopinus rufipes Eichhoff (Coleoptera: Curculionidae) in Manitoba." Entomological Society of America, 2011. http://hdl.handle.net/1993/23811.

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Hylurgopinus rufipes, the native elm bark beetle (NEBB), is the major vector of Dutch elm disease (DED) in Manitoba. Dissections of American elms (Ulmus americana), in the same year as DED symptoms appeared in them, showed that NEBB constructed brood galleries in which a generation completed development, and adult NEBB carrying DED spores would probably leave the newly-symptomatic trees. Rapid removal of freshly diseased trees, completed by mid-August, will prevent spore-bearing NEBB emergence, and is recommended. The relationship between presence of NEBB in stained branch sections and the total number of NEEB per tree could be the basis for methods to prioritize trees for rapid removal. Numbers and densities of overwintering NEBB in elm trees decreased with increasing height, with >70% of the population overwintering above ground doing so in the basal 15 cm. Substantial numbers of NEBB overwinter below the soil surface, and could be unaffected by basal spraying. Mark-recapture studies showed that frequency of spore bearing by overwintering beetles averaged 45% for the wild population and 2% for marked NEBB released from disease-free logs. Most NEBB overwintered close to their emergence site, but some traveled ≥4.8 km before wintering. Studies comparing efficacy of insecticides showed that chlorpyrifos gave 100% control of overwintering NEBB for two years as did bifenthrin: however, permethrin and carbaryl provided transient efficacy. NEBB showed a gradual increase in development rate with increasing constant temperature. Lipid content of overwintering NEBB was higher in late fall than in mid-winter, which might show that depletion of fat reserves could jeopardize survival, but could be a result of conversion to cryoprotectants.
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Betsch, Brody Bly. "A Study of the Impacts of Dutch elm disease, Emerald ash borer, and Amur honeysuckle on the Flora of Rush Run Wildlife Area." Miami University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=miami1564514690185145.

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Björkroth, Jennie. "Effekter av naturvårdsgallring på förekomsten av lunglav på ädellövträd." Thesis, Linköpings universitet, Biologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-120089.

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Sun-exposed broad leaved deciduous trees have a great species diversity of epiphytic lichens. In Europe, these trees have decreased dramatically in number as wooded pastures have become overgrown with trees and bushes, and broad leaved deciduous stands have been replaced by planted coniferous trees. These are the main reasons for many lichens depending on deciduous trees being red-listed. Epiphytic lichens in overgrown areas could benefit from conservation thinning, but few studies have been performed on how this type of cutting affects the lichens. In a previous study, the presence of red-listed epiphytic lichens in a broad leaved deciduous forest was examined. After the study, thinning of trees and bushes was made. Here we study the effects of this thinning on Lobaria pulmonaria. We tested possible factors that may affect the growth of L. pulmonaria, and if there were any differences in incidence and vitality of the lichen between managed and unmanaged stands. Since Dutch elm disease and ash dieback are well spread in the area, we wanted to see if they also affected the growth of L. pulmonaria. There were no differences in incidence and growth between managed and unmanaged stands. The results were unexpected since other studies show that, for instance, increased sun exposure often has a great effect on the growth of lichens. Many elms and ashes were dead or dying and had a significant negative effect on the change of number of lobes and the lobe surface. The diseases of the trees can thus be assumed to be the greatest cause of why the lichens in the managed stand did not benefit from thinning.
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Proctor, Robert Henry. "Phytoalexins and dutch elm disease resistance." 1990. http://catalog.hathitrust.org/api/volumes/oclc/23379309.html.

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Thesis (Ph. D.)--University of Wisconsin--Madison, 1990.
Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 121-132).
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Nasmith, Charles George. "Molecular characteristics associated with the elm response in the Dutch elm disease complex." 2004. http://link.library.utoronto.ca/eir/EIRdetail.cfm?Resources__ID=94672&T=F.

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Books on the topic "Dutch elm disease. Insecticides"

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Sticklen, Mariam B., and James L. Sherald, eds. Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8.

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Brasier, Clive. New horizons in Dutch Elm Disease control. Forestry Commission, 1996.

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Susan, Chapman. Dutch elm disease: January 1979 - August 1990. National Agricultural Library, 1990.

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Haugen, Linda. How to identify and manage Dutch elm disease. The Area, 1998.

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Cannon, William N. Time and materials needed to survey, inject systematic fungicides, and install root-graft barriers for Dutch elm disease management. U.S. Dept. of Agriculture, Forest Service, Northeastern Forest Experiment Station, 1986.

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Atkins, Paul Michael. Dutch elm disease: Some aspects of its biology and control. University of Salford, 1986.

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Aziz, Ammar Y. Biological control of Dutch Elm Disease: "Interactions between Trichoderm spp. and Ophiostoma ulmi". University of Salford, 1988.

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Cao, Chenglong. Biochemical characterization of mutants of Ophiostoma ulmi (Buism.) Nannf., the causal agent of Dutch elm disease. National Library of Canada = Bibliothèque nationale du Canada, 1992.

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(Editor), Mariam B. Sticklen, and James L. Sherald (Editor), eds. Dutch Elm Disease Research: Cellular and Molecular Approaches. Springer, 1993.

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Dutch elm disease research: Cellular and molecular approaches. Springer-Verlag, 1993.

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Book chapters on the topic "Dutch elm disease. Insecticides"

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Cumming, Jeffrey M., Bradley J. Sinclair, Charles A. Triplehorn, et al. "Dutch Elm Disease." In Encyclopedia of Entomology. Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6359-6_1014.

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Heybroek, Hans M. "Why Bother About the Elm?" In Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8_1.

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Domir, Subhash C., M. B. Sticklen, P. M. Pijut, M. G. Bolyard, and R. D. Lineberger. "Micropropagation and Regeneration of DED-Resistant Elms Using Protoplast and Tissue Culture Techniques." In Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8_10.

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Richards, Wayne C. "Cerato-ulmin: A Unique Wilt Toxin of Instrumental Significance in the Development of Dutch Elm Disease." In Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8_11.

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Yaguchi, M., M. Pusztai-Carey, C. Roy, et al. "Amino Acid Sequence and Spectroscopic Studies of Dutch Elm Disease Toxin, Cerato-ulmin." In Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8_12.

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Bolyard, Mark G., and Mariam B. Sticklen. "Strategies for the Production of Disease-Resistant Elms." In Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8_13.

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Domir, Subhash C., Lawrence R. Schreiber, and Paula M. Pijut. "Development of Model Systems to Screen Elms Resistant to Dutch Elm Disease." In Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8_14.

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Elgersma, D. M., T. Roosien, and R. J. Scheffer. "Biological Control of Dutch Elm Disease by Exploiting Resistance in the Host." In Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8_15.

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Hajela, Ravindra K., Lawrence Graham, and Mariam B. Sticklen. "Cloning of Pathogenesis-Related Genes From Ulmus americana." In Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8_16.

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Hubbes, Martin. "Mansonones, Elicitors and Virulence." In Dutch Elm Disease Research. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-6872-8_17.

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Conference papers on the topic "Dutch elm disease. Insecticides"

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Лазебный, Михаил Юрьевич, and Ирина Дмитриевна Самсонова. "EVALUATION OF THE STATE OF ILMOVS AND THE FEATURES OF THEIR HANDS IN GREEN PLANTS OF THE GENERAL USES OF ST. PETERSBURG." In Национальная безопасность России: актуальные аспекты: сборник избранных статей Всероссийской научно-практической конференции (Санкт-Петербург, Май 2020). Crossref, 2020. http://dx.doi.org/10.37539/nb185.2020.33.51.005.

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В настоящее время вязы, пораженные голландской болезнью, выявлены во всех 18 районах Санкт-Петербурга. Проведена оценка состояния ильмовых в зеленых насаждениях общего пользования, выявлена роль факторов их усыхания и проанализированы мероприятия по уходу за ними. Currently, elms affected by the Dutch disease have been identified in all 18 districts of St. Petersburg. The state of elm trees in public green spaces was assessed, the role of their drying factors was revealed, and measures to care for them were analyzed.
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Reports on the topic "Dutch elm disease. Insecticides"

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Cannon, William N. ,. Jr, Jack H. Barger, Charles J. Kostichka, and Charles J. Kostichka. Time and materials needed to survey, inject systemic fungicides, and install root-graft barriers for Dutch elm disease management. U.S. Department of Agriculture, Forest Service, Northeastern Forest Experimental Station, 1986. http://dx.doi.org/10.2737/ne-rp-585.

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Cannon, William N. ,. Jr, Jack H. Barger, Charles J. Kostichka, and Charles J. Kostichka. Time and materials needed to survey, inject systemic fungicides, and install root-graft barriers for Dutch elm disease management. U.S. Department of Agriculture, Forest Service, Northeastern Forest Experimental Station, 1986. http://dx.doi.org/10.2737/ne-rp-585.

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