Relevant bibliographies by topics / Dysbiose buccale

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Academic literature on the topic 'Dysbiose buccale'

Author: Grafiati
Published: 6 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "Dysbiose buccale"

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Côté, Nathalie, and Jean-Charles Pasquier. "La prématurité spontanée et le microbiote maternel." médecine/sciences 34, no. 10 (2018): 799–805. http://dx.doi.org/10.1051/medsci/2018205.

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Des millions d’enfants naissent prématurément chaque année et les cliniciens restent démunis face à la difficulté de maîtriser la physiopathologie associée, ce qui limite les options thérapeutiques. Récemment, il a été suggéré que le microbiote maternel pouvait contribuer au bon déroulement de la grossesse et qu’une dysbiose pourrait entraîner une naissance avant terme. Certaines espèces commensales de Lactobacillus participeraient à une fonction de « filtre vaginal », empêchant une propagation ascendante de pathogènes vers la cavité utérine. Ce compartiment peut par ailleurs être colonisé par
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2

van der Meulen, Taco A., Hermie J. M. Harmsen, Hendrika Bootsma, et al. "Dysbiosis of the buccal mucosa microbiome in primary Sjögren’s syndrome patients." Rheumatology 57, no. 12 (2018): 2225–34. http://dx.doi.org/10.1093/rheumatology/key215.

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3

He, Y., D. Gong, C. Shi, F. Shao, J. Shi, and J. Fei. "Dysbiosis of oral buccal mucosa microbiota in patients with oral lichen planus." Oral Diseases 23, no. 5 (2017): 674–82. http://dx.doi.org/10.1111/odi.12657.

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4

Rodríguez María Lourdes, Rosa Alcira Cristina, Nastri María Lorena, and Jewtuchowicz Virginia Martha. "Oral dysbiosis exacerbates the virulence of Candida parapsilosis sensu stricto via up-regulation of the CPH2 biofilm master gene." World Journal of Advanced Research and Reviews 10, no. 1 (2021): 01–011. http://dx.doi.org/10.30574/wjarr.2021.10.1.0107.

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Candida parapsilosis sensu stricto is the second to third most frequent cause of candidemia. Studies place this yeast as a frequent colonizer of niches of the oral cavity, predominantly in pathological conditions. We hypothesize that a buccal environment in dysbiosis enhances the virulence of C. parapsilosis sensu stricto. Objective: To evaluate at the phenotype and molecular level the production of biofilm in oral isolates of C. parapsilosis sensu stricto and correlate the results with the clinical origin (dysbiosis versus eubiosis). Material and methods: The biofilm-forming ability was compa
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Rodríguez, María Lourdes, Alcira Cristina Rosa, María Lorena Nastri, and Virginia Martha Jewtuchowicz. "Oral dysbiosis exacerbates the virulence of Candida parapsilosis sensu stricto via up-regulation of the CPH2 biofilm master gene." World Journal of Advanced Research and Reviews 10, no. 1 (2021): 01–011. https://doi.org/10.5281/zenodo.4733302.

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<em>Candida parapsilosis</em>&nbsp;sensu stricto is the second to third most frequent cause of candidemia. Studies place this yeast as a frequent colonizer of niches of the oral cavity, predominantly in pathological conditions. We hypothesize that a buccal environment in dysbiosis enhances the virulence of&nbsp;<em>C. parapsilosis</em>&nbsp;sensu stricto.&nbsp; <strong>Objective</strong>: To evaluate at the phenotype and molecular level the production of biofilm in oral isolates of&nbsp;<em>C. parapsilosis</em>&nbsp;sensu stricto and correlate the results with the clinical origin (dysbiosis ve
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6

Na, Hee Sam, Si Yeong Kim, Hyejung Han, et al. "Identification of Potential Oral Microbial Biomarkers for the Diagnosis of Periodontitis." Journal of Clinical Medicine 9, no. 5 (2020): 1549. http://dx.doi.org/10.3390/jcm9051549.

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Periodontitis is a chronic and multifactorial inflammatory disease that can lead to tooth loss. At present, the diagnosis for periodontitis is primarily based on clinical examination and radiographic parameters. Detecting the periodontal pathogens at the subgingival plaque requires skilled professionals to collect samples. Periodontal pathogens are also detected on various mucous membranes in patients with periodontitis. In this study, we characterized the oral microbiome profiles from buccal mucosa and supragingival space in a total of 272 healthy subjects as a control group, and periodontiti
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7

Lavoro, Alessandro, Giovanni Cultrera, Giuseppe Gattuso, et al. "Role of Oral Microbiota Dysbiosis in the Development and Progression of Oral Lichen Planus." Journal of Personalized Medicine 14, no. 4 (2024): 386. http://dx.doi.org/10.3390/jpm14040386.

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Oral lichen planus (OLP) is a chronic inflammatory autoimmune disease of the oral cavity with malignant potential affecting 1.01% of the worldwide population. The clinical patterns of this oral disorder, characterized by relapses and remissions of the lesions, appear on buccal, lingual, gingival, and labial mucosa causing a significant reduction in the quality of life. Currently, there are no specific treatments for this disease, and the available therapies with topical and systemic corticosteroids only reduce symptoms. Although the etiopathogenesis of this pathological condition has not been
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8

Jung, Won, and Sungil Jang. "Oral Microbiome Research on Oral Lichen Planus: Current Findings and Perspectives." Biology 11, no. 5 (2022): 723. http://dx.doi.org/10.3390/biology11050723.

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Oral lichen planus (OLP) is a chronic inflammatory disease of the oral mucosa with an unknown etiology. The role of oral microbes in the development of OLP has gained researchers’ interest. In this review, we summarized the findings of studies focused on the relationship between OLP and oral microbiome, which includes the composition of oral microbiota, molecules produced by oral microbiota or the host, and the oral environment of the host. According to the studies, the oral microbial community in OLP patients undergoes dysbiosis, and the microbial dysbiosis in OLP patients is more prominent i
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9

GIURGIU, Gheorghe, and Manole COJOCARU. "Natural Neuroimunomodulation in Coronavirus Infection." Annals of the Academy of Romanian Scientists Series on Biological Sciences 9, no. 2 (2020): 80–87. http://dx.doi.org/10.56082/annalsarscibio.2020.2.80.

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Dysbiosis of the nasopharyngeal microbiome attracts dysbiosis of the intestinal microbiome and activation of the intestinal microbiome-brain axis. If the first sign of the disease is quickly intervened with the modulation of the activity of the microbiome, implicitly of the immune system (neuroimmunomodulation), the appearance of the disease is eliminated. There is the microbiome: buccal, nasal, intestinal, cardiac, cutaneous and even the microbiome in the brain with which Covid-19 interacts. When the evolution is complicated, it is necessary to intervene with drug treatment to support the aff
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10

Pikuza, O. I., R. A. Faizullina, A. M. Zakirova, et al. "Colonization resistance of buccal epitheliocytes as an indicator of mucosal immune homeostatic resources in children with recurrent respiratory diseases." Rossiyskiy Vestnik Perinatologii i Pediatrii (Russian Bulletin of Perinatology and Pediatrics) 68, no. 5 (2023): 45–52. http://dx.doi.org/10.21508/1027-4065-2023-68-5-45-52.

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Of particular interest is the study of colonization resistance of the oral cavity as a physiological phenomenon that reflects the ability of the microbiota and macroorganism in cooperation to protect the ecosystem of the oral cavity from pathogens. Purpose is to evaluate the significance of indicators of colonization resistance of buccal epithelial cells as a marker of homeostatic resources of mucosal immunity in recurrent respiratory diseases in children. Material and methods. 232 (5–16 years old) children were examined, including 56 children with acute bronchitis, 73 with recurrent bronchiti
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Dissertations / Theses on the topic "Dysbiose buccale"

1

Borsa, Leslie. "Cartographie du microbiote oral et identification de pathogènes oraux associés à la maladie d'Alzheimer." Electronic Thesis or Diss., Université Côte d'Azur, 2022. http://www.theses.fr/2022COAZ6010.

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L'incidence de la maladie d'Alzheimer (MA), qui constitue la première cause de troubles neurocognitifs dans la population adulte, augmente dans le monde entier. A ce jour, et malgré les immenses progrès réalisés en 30 ans concernant la compréhension des mécanismes neuropathologiques, et notamment l'accumulation des protéinesTau et Béta-amyloïdes, la question de l'étiopathogénie et des différents facteurs de risque de la MA reste largement débattue. Parmi les facteurs de risque étudiés, l'inflammation chronique et les pathologies, notamment infectieuses, constituent des voies de recherche parti
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Journal articles
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