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Journal articles on the topic 'Dysbiotic changes'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Bobyr, V. V., V. A. Poniatovskyi, A. P. Chobotar, et al. "Features of structural-morphological changes in cases of experimental intestinal antibiotic-induced dysbiosis." Reports of Morphology 24, no. 3 (2018): 26–31. http://dx.doi.org/10.31393/morphology-journal-2018-24(3)-04.

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Nowadays, scientists often define dysbiosis as a condition of a microbial ecological system, in which there is a simultaneous abnormality of the functions and interaction mechanisms of its key components: macroorganism and indigenous microbiota associated with the mucous membranes of cavities and skin. At the same time, obviously, the basis of all these processes is changes of structural intestinal components that are caused by qualitative and quantitative changes in the normal microflora. Purpose: to study the ultrastructural organization of the mucous membrane of the small intestine of mice

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Nikolaeva, Nina V., Elizaveta V. Afanasievskaya, Eduard S. Gorovitz, and Alexander Ja Perevalov. "Intestinal microbiocinosis status as a criterion for nutritional quality assessment." Вестник Пермского университета. Серия «Биология»=Bulletin of Perm University. Biology, no. 2 (2022): 137–42. http://dx.doi.org/10.17072/1994-9952-2022-2-137-142.

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As a result of dynamic triple bacteriological examination for intestinal dysbiosis performed in 30 generally healthy 1st-year students who came to study from other regions, it was found that changes in the regime and quality of food led to the development of intestinal dysbiosis in 61.54% (out of 28 examined individuals) of nonresident first-year university students six months after the start of the study. The dysbiotic disorders of the intestinal microbiota were found to be primarily associated with a decrease (by 1‒3 orders of magnitude) in the titer of obligate bacteria (bifido- and lactic

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Tarasova, E., E. Yermolenko, V. Donets, et al. "The influence of probiotic Enterococcus faecium strain L5 on the microbiota and cytokines expression in rats with dysbiosis induced by antibiotics." Beneficial Microbes 1, no. 3 (2010): 265–70. http://dx.doi.org/10.3920/bm2010.0008.

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The animal model of intestinal dysbiosis induced by antibiotics was created. Dysbiotic condition was confirmed by the changes in titre of the indigenous microbiota (excessive growth of opportunistic microorganisms and reduced number of lactobacilli, bifidobacteria and enterococci) and the appearance of dyspeptic symptoms. Consumption of the fermented milk product with probiotic strain Enterococcus faecium L5 led to the rapid disappearance of dysbiosis symptoms, normalisation of the microbiota, increase in expression of IL-10 and decrease in IL-8 expression.

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Kamer, Angela R., Smruti Pushalkar, Babak Hamidi, et al. "Periodontal Inflammation and Dysbiosis Relate to Microbial Changes in the Gut." Microorganisms 12, no. 6 (2024): 1225. http://dx.doi.org/10.3390/microorganisms12061225.

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Periodontal disease (PerioD) is a chronic inflammatory disease of dysbiotic etiology. Animal models and few human data showed a relationship between oral bacteria and gut dysbiosis. However, the effect of periodontal inflammation and subgingival dysbiosis on the gut is unknown. We hypothesized that periodontal inflammation and its associated subgingival dysbiosis contribute to gut dysbiosis even in subjects free of known gut disorders. We evaluated and compared elderly subjects with Low and High periodontal inflammation (assessed by Periodontal Inflamed Surface Area (PISA)) for stool and subgi

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Zakharenko, A. A., A. N. Suvorov, I. V. Shlyk, et al. "DISORDERS OF A MICROBIOCENOSIS OF INTESTINES AT PATIENTS WITH A COLORECTAL CANCER AND WAYS OF THEIR CORRECTION (review)." Koloproktologia, no. 2 (June 30, 2016): 48–56. http://dx.doi.org/10.33878/2073-7556-2016-0-2-48-56.

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Patients with colorectal cancer in the perioperative period, revealed pronounced changes of microbiocenosis of colon, which is characterized by changes in the quantitative and qualitative composition of microflora. It showed a significant inhibition of anaerobic microbial component mainly bifidobacteria and lactobacilli. As a result, saprophytic microbes multiply with highly resistant to drugs, acquire pathogenic properties. The most prominent representative of such microorganisms is a pathogenic strain of Clostridium difficile, often provokes the development of pseudomembranous colitis. Depen

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Chervinets, V. M., Yu V. Chervinets, D. V. Ganzya, V. S. Belyaev, and V. S. Zaitseva. "The effect of the intestinal microbiota on the development of diabetes mellitus." Experimental and Clinical Gastroenterology, no. 12 (December 18, 2023): 77–85. http://dx.doi.org/10.31146/1682-8658-ecg-220-12-77-85.

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The aim of the study was to identify the pathogenetic role of intestinal microflora in the development of diabetes mellitus. The review presents data on the peculiarities of the development of the intestinal microflora (MC), factors that determine the occurrence of dysbiosis, as well as the effect of dysbiotic changes in MC on the pathogenesis of various types of diabetes mellitus. The role of MC in the occurrence of insulin resistance, as well as the metabolism of peptides that determine eating behavior, has been studied. The contribution of short-chain fatty acids produced by microorganisms

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Bezmenko, Alexandr A., and Natalya D. Sadovaya. "Vaginal and intestinal microbiocenosis composition in pregnant women." Journal of obstetrics and women's diseases 68, no. 6 (2020): 29–36. http://dx.doi.org/10.17816/jowd68629-36.

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Hypothesis/aims of study. It is assumed that intestinal dysbiosis, as well as bacterial vaginosis, may be one of the risk factors, and in some cases, the direct cause of pregnancy complications. This study was aimed to assess the intestinal and vaginal microbiocenosis composition in women with threatened miscarriage and normal pregnancy. Study design, materials and methods. The study involved 68 women aged 18 to 35 years (mean age 27.6 0.7 years) in pregnancy from 6 to 18 weeks. The main group consisted of 38 women with threatened miscarriage. The control group included women (n = 30) wi

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Barendregt, D., R. Gacesa, N. Plomp, et al. "P0123 The immune system of patients with immune-mediated diseases perceives dysbiotic intestinal microbial species, and IgG reactivity uncovers shared and non-shared responses across diseases." Journal of Crohn's and Colitis 19, Supplement_1 (2025): i505. https://doi.org/10.1093/ecco-jcc/jjae190.0297.

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Abstract Background Immune-mediated diseases (IMIDs), like Crohn’s disease (CD), ulcerative colitis (UC), psoriatic arthritis (PsA) and type I diabetes (T1D), have common features, including intestinal microbial dysbiosis. However, it is unclear whether dysbiosis contributes to IMID pathogenesis and whether the host immune system perceives dysbiotic commensals. In CD, high IgG and T-cell responses to Lachnospiraceae-derived flagellins associate with complex disease, while Lachnospiraceae fecal abundance is decreased in CD, suggesting that individual variation in the host-microbial mutualism is

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Maev, I. V., A. I. Levchenko, and D. N. Andreev. "Changes in the Intestinal Microbiota in Patients with Chronic Pancreatitis: Systematizing Literature Data." Russian Journal of Gastroenterology, Hepatology, Coloproctology 32, no. 4 (2022): 17–26. http://dx.doi.org/10.22416/1382-4376-2022-32-4-17-26.

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The purpose of the review. To systematize literature data on changes in the structure of the intestinal microbiota in patients with chronic pancreatitis (CP).Key findings. The human intestinal microbiota is a dynamically changing system that is constantly undergoing qualitative and quantitative changes, especially in several pathological conditions of the digestive system. At present, the differences in the intestinal microbiota in pancreatic diseases are poorly understood. The severe CP is associated with impaired synthesis of antimicrobial peptides, bicarbonates, and digestive enzymes by the

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Sitkin, S. I., and T. M. Pervunina. "Chronic pancreatitis and the intestinal microbiome in adults and children: Not only SIBO but also gut dysbiosis has clinical significance." Experimental and Clinical Gastroenterology, no. 12 (January 16, 2022): 125–33. http://dx.doi.org/10.31146/1682-8658-ecg-196-12-125-133.

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Chronic pancreatitis is accompanied by both small intestinal bacterial overgrowth and dysbiosis of the gut microbiota. The most typical changes in the gut microbiota in chronic pancreatitis are a decrease in microbial diversity, an increase in the abundance of Proteobacteria, a decrease in the abundance of Bacteroidetes, Actinobacteria, and Firmicutes, especially butyrate-producing bacteria such as Faecalibacterium, a significant association with infectious pathways (KEGG analysis]), concomitant metabolic disorders (diabetes mellitus) and exocrine pancreatic insufficiency. Changes in the gut m

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Ismoilov, Soloi R. "The influence of Fencarole and Zaditen on intestinal microflora of rats in the stages of passive anaphylactic reaction." Reviews on Clinical Pharmacology and Drug Therapy 17, no. 2 (2019): 67–72. http://dx.doi.org/10.17816/rcf17267-72.

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It was experimentally established that in the conditions of passive anaphylactic reaction more distinct dysbiotic shifts occurred in the microflora of the large intestine of rats on the 6th day of sensitization and on the 3rd day of food anaphylaxis. And in the study of the effect of antihistamines on these disorders revealed their ambiguous effect, although fencarole and zaditen did not have sufficient protective effect on intestinal dysbiotic changes.

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Jiménez-Vargas, N. N., M. Guzman Rodriguez, Y. Yu, et al. "A242 STOOL FROM IBS-D PATIENT WITH A HISTORY OF A DYSBIOTIC AND NON-DYSBIOTIC ONSET MODULATE NEURONAL EXCITABILITY VIA DIFFERENT MECHANISMS." Journal of the Canadian Association of Gastroenterology 5, Supplement_1 (2022): 134–35. http://dx.doi.org/10.1093/jcag/gwab049.241.

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Abstract Background We have shown that Irritable Bowel Syndrome diarrhea-predominant (IBS-D) patients with a history of a dysbiotic-like onset have distinct stool metabolomic profiles versus those with a non-dysbiotic-like onset. IBS stool supernatants can sensitize mouse colonic afferent nerves via both histamine and proteases. However, it is unknown if stool supernatants from the two IBS-D subgroups modulate the excitability of nociceptive neurons via different neuroactive mediators Aims To evaluate whether there are differences in neuroactive mediators within stool supernatants from subgrou

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Park, Kiwoong, Suhyeon Park, Arulkumar Nagappan, et al. "Probiotic Escherichia coli Ameliorates Antibiotic-Associated Anxiety Responses in Mice." Nutrients 13, no. 3 (2021): 811. http://dx.doi.org/10.3390/nu13030811.

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Despite the beneficial actions of antibiotics against bacterial infections, the use of antibiotics is a crucial etiological factor influencing microbial dysbiosis-associated adverse outcomes in human health. Based on the assumption that gut microbial dysbiosis can provoke behavioral or psychological disorders, the present study evaluated anxiety-linked behavioral changes in a mouse model of streptomycin-induced dysbiosis. Measuring anxiety-like behavior using the light–dark box and elevated plus maze tests indicated that streptomycin treatment caused acute anxiety in mice. As an intervention f

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Cota-Quintero, Juan, Mercedes Bermúdez, Rosalío Ramos-Payán, et al. "Dysbiotic changes of periodontal pathogens in patients wearing conventional and self-ligating orthodontic appliances." Australasian Orthodontic Journal 39, no. 1 (2023): 61–71. http://dx.doi.org/10.2478/aoj-2023-0006.

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Abstract Objective: This study aimed to analyse dysbiotic changes of periodontal pathogenic bacteria and their relationship with different types of fixed orthodontic appliances in a population located in Northwestern Mexico. Methods: Three groups of patients were identified: a control group without orthodontic appliances (C), a conventional-ligating appliance group (CLA), and a self-ligating appliance group (SLA). Periodontal biofilm samples were collected for DNA extraction to identify the presence and load of Treponema denticola, Prevotella intermedia, and Fusobacterium nucleatum, using a qu

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Tolkachev, Boris Evgenyevich, Evgeny Igorevich Morkovin, Liliya Petrovna Knyshova, Anatoly Trofimovich Yakovlev, and Andrey Valeryevich Strygin. "Changes of intestinal microbiota and ivabradine biotransformation in rats after alcohol introduction." Samara Journal of Science 6, no. 3 (2017): 47–51. http://dx.doi.org/10.17816/snv201763108.

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The role of intestinal microbiota in progress of many pathological processes is discussed in recent publications. It was shown that the continuous intestinal dysbiosis, including the increased bacterial growth syndrome, affects the digestion and the biotransformation of xenobiotics. Intestinal microbiota provokes metabolic failures leading to severe diseases acting via several mechanisms. Alcohol is found to be a common dysbiotic factor and toxic agent affecting the systems of biotransformation. This study was to estimate the associations between the intestinal microbiota and the biotransforma

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Nagpal, Ravinder, Tiffany M. Newman, Shaohua Wang, Shalini Jain, James F. Lovato, and Hariom Yadav. "Obesity-Linked Gut Microbiome Dysbiosis Associated with Derangements in Gut Permeability and Intestinal Cellular Homeostasis Independent of Diet." Journal of Diabetes Research 2018 (September 3, 2018): 1–9. http://dx.doi.org/10.1155/2018/3462092.

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This study aimed to determine the association between non-high-fat diet-induced obesity- (non-DIO-) associated gut microbiome dysbiosis with gut abnormalities like cellular turnover of intestinal cells, tight junctions, and mucin formation that can impact gut permeability. We used leptin-deficient (Lepob/ob) mice in comparison to C57BL/6J control mice, which are fed on identical diets, and performed comparative and correlative analyses of gut microbiome composition, gut permeability, intestinal structural changes, tight junction-mucin formation, cellular turnover, and stemness genes. We found

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Martykanova, Dilyara S., Nailja Ch Davletova, Ilya A. Zemlenuhin, et al. "Characterization of Dysbiotic Changes of Skin Microbiota in Contact Sports Athletes." BioNanoScience 7, no. 1 (2017): 1–3. http://dx.doi.org/10.1007/s12668-017-0396-0.

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Bobyr, V. V., L. О. Stechenko, V. P. Shyrobokov, et al. "Modeling of viral-bacterial infections against antibiotic-induced intestinal dysbiosis." Reports of Morphology 25, no. 2 (2019): 78–84. http://dx.doi.org/10.31393/morphology-journal-2019-25(2)-10.

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The study of the role of viral-bacterial associations in the etiology of acute intestinal infections is considered new to medical microbiology. The purpose of the study was to determine the effect of viral-bacterial associations on the manifestation of virulence of pathogens and the degree of development of structural-morphological disorders of the internal organs in animals with antibiotic-induced dysbiosis. In a study of 210 white laboratory mice, BALB/c lines formed dysbiosis using antibacterial agents (ampicillin, gentamicin, metronidazole) followed by simulation of the experimental infect

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Slazhneva, E. S., E. A. Tikhomirova, and V. G. Atrushkevich. "Periodontopathogens: a new view. Systematic review. Part 1." Стоматология детского возраста и профилактика 20, no. 1 (2020): 70–76. http://dx.doi.org/10.33925/1683-3031-2020-20-1-70-76.

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Relevance. The modern view of periodontitis as a dysbiotic disease that occurs as a result of changes in the microbial composition of the subgingival region is considered in a systematic review. Purpose. To study a new paradigm of development of generalized periodontitis. Materials and methods. Randomized controlled trials (RCTS) were selected for the study, including cluster RCTS, controlled (non-randomized) microbiological and clinical studies of the oral microbiome in adult patients with generalized periodontitis over the past 10 years. Results. The development of periodontal dysbiosis occu

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Chaves, Lee D., Daniel I. McSkimming, Mark A. Bryniarski, et al. "Chronic kidney disease, uremic milieu, and its effects on gut bacterial microbiota dysbiosis." American Journal of Physiology-Renal Physiology 315, no. 3 (2018): F487—F502. http://dx.doi.org/10.1152/ajprenal.00092.2018.

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Several lines of evidence suggest that gut bacterial microbiota is altered in patients with chronic kidney disease (CKD), though the mechanism of which this dysbiosis takes place is not well understood. Recent studies delineated changes in gut microbiota in both CKD patients and experimental animal models using microarray chips. We present 16S ribosomal RNA gene sequencing of both stool pellets and small bowel contents of C57BL/6J mice that underwent a remnant kidney model and establish that changes in microbiota take place in the early gastrointestinal tract. Increased intestinal urea concent

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Hadzevych, O. V. "Formation of intestinal bacteriocenosis in calves associated with bacterial insemination of the uterine canal in high-yielding cows." Journal for Veterinary Medicine, Biotechnology and Biosafety 9, no. 1-2 (2023): 3–7. http://dx.doi.org/10.36016/jvmbbs-2023-9-1-2-1.

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The article provides information on the microbiota composition in the vaginal secretions of cows during the dry period, specifically 30–15 days before calving. The article discusses how endometritis affects the qualitative and quantitative composition of the conditionally pathogenic microflora, as well as how dysbiotic microbiota disorders in the birth canal impact the development of gastrointestinal diseases in calves. The study results establish the quantitative limits of the conditionally pathogenic microbiota, which requires correction when exceeded. When cows have dysbiotic changes, there

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Kuznetsova, Kamalia Yu, Vladimir P. Sergiev, Maria A. Kuznetsova, et al. "Dysbiotic changes in the intestines under the influence of exogenous biological determinants." HEALTH CARE OF THE RUSSIAN FEDERATION 67, no. 2 (2023): 172–81. http://dx.doi.org/10.47470/0044-197x-2023-67-2-172-181.

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Introduction. Data were obtained on the difference in the quantitative and specific composition of intestinal microbiota bacteria in practically healthy individuals with subclinical invasions by pathogenic intestinal protozoa Lamblia intestinalis, Blastocystis hominis. Сhanges in bacterial composition were shown to have characterological features and species-specific «targets» of intestinal microbiota destruction.  Material and methods. Bacteriological and parasitological research methods were used. For a comparative analysis of quantitative and specific changes in the symbiotic microflo

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Plantamura, Emilie, Amiran Dzutsev, Mathias Chamaillard, et al. "MAVS deficiency induces gut dysbiotic microbiota conferring a proallergic phenotype." Proceedings of the National Academy of Sciences 115, no. 41 (2018): 10404–9. http://dx.doi.org/10.1073/pnas.1722372115.

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Prominent changes in the gut microbiota (referred to as “dysbiosis”) play a key role in the development of allergic disorders, but the underlying mechanisms remain unknown. Study of the delayed-type hypersensitivity (DTH) response in mice contributed to our knowledge of the pathophysiology of human allergic contact dermatitis. Here we report a negative regulatory role of the RIG-I–like receptor adaptor mitochondrial antiviral signaling (MAVS) on DTH by modulating gut bacterial ecology. Cohousing and fecal transplantation experiments revealed that the dysbiotic microbiota of Mavs−/− mice confer

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K., Gorobchenko, and Dyachenko A. "Intestinal Dysbiosis in Systemic Inflamation and Possibilities of Its Correction with Probiotics." Microbiology Research Journal International 20, no. 6 (2017): 1–9. https://doi.org/10.9734/MRJI/2017/34534.

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<strong>Aims: </strong>To estimate intestinal microbial changes and study the efficacy of probiotic preparations in systemic inflammation. <strong>Study Design: </strong>Cohort design. <strong>Place and Duration of Study:</strong> Sumy State University, Medical Institute. Department of Microbiology and Clinical Immunology, Kharkiv Medical Academy of Postgraduate Education. <strong>Methodology:</strong> The study involved 162 patient with chronic infections various sites, including 58 (35.8%) patients with respiratory tract infections, 56 (34.6%) patients with infections of the genitourinary sy

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Richard Samuelson, Derrick, Vincent Maffei, Eugene Blanchard, et al. "2262." Journal of Clinical and Translational Science 1, S1 (2017): 4–5. http://dx.doi.org/10.1017/cts.2017.32.

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OBJECTIVES/SPECIFIC AIMS: Alcohol consumption perturbs the normal intestinal microbial communities (alcohol dysbiosis). To begin to investigate the relationship between alcohol-mediated dysbiosis and host defense we developed an alcohol dysbiosis fecal adoptive transfer model, which allows us to isolate the host immune response to a pathogenic challenge at a distal organ (ie, the lung). This model system allowed us to determine whether the host immune responses to Klebsiella pneumoniae are altered by ethanol-associated dysbiosis, independent of alcohol use. We hypothesized that alcohol-induced

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Hurych, J., A. Mascellani Bergo, T. Lerchová та ін. "P911 The changes in the faecal microbiome and metabolome in Crohn's disease after treatment with anti-TNFα antibodies are of secondary origin". Journal of Crohn's and Colitis 17, Supplement_1 (2023): i1023—i1025. http://dx.doi.org/10.1093/ecco-jcc/jjac190.1041.

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Abstract Background Treatment by anti-TNFα antibodies (aTNFα) is known to change the dysbiotic faecal bacteriome in Crohn's disease (CD). However, whether the change is caused by aTNFα itself or by improving the gut inflammation status is unknown. Therefore, we aimed to identify and compare bacterial taxa and faecal metabolite changes upon aTNFα between children with CD and juvenile idiopathic arthritis (JIA), a disease very rarely affecting the gut. Methods Faecal samples were analysed for bacteriome by massively parallel sequencing of the V4 region of the 16S rRNA and for faecal metabolome b

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Lange, Kathleen, Martin Buerger, Andreas Stallmach, and Tony Bruns. "Effects of Antibiotics on Gut Microbiota." Digestive Diseases 34, no. 3 (2016): 260–68. http://dx.doi.org/10.1159/000443360.

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The gut microbiota influences essential human functions including digestion, energy metabolism, and inflammation by modulating multiple endocrine, neural, and immune pathways of the host. Its composition and complexity varies markedly across individuals and across different sites of the gut, but provides a certain level of resilience against external perturbation. Short-term antibiotic treatment is able to shift the gut microbiota to long-term alternative dysbiotic states, which may promote the development and aggravation of disease. Common features of post-antibiotic dysbiosis include a loss

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Marcickiewicz, Justyna, Małgorzata Jamka, and Jarosław Walkowiak. "A Potential Link Between Oral Microbiota and Female Reproductive Health." Microorganisms 13, no. 3 (2025): 619. https://doi.org/10.3390/microorganisms13030619.

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Oral cavity dysbiosis is associated with numerous inflammatory diseases, including diabetes, inflammatory bowel diseases, and periodontal disease. Changes in the oral microenvironment lead to bidirectional interactions between pathogens and individual host systems, which may induce systemic inflammation. There is increasing evidence linking the condition of the oral cavity with the most common causes of female infertility, such as polycystic ovary syndrome and endometriosis, as well as gestational complications, e.g., low birth weight, preterm delivery, and miscarriages. This review highlights

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Ruzhentsova, T. A., A. A. Garbuzov, and A. L. Bayrakova. "Effect of probiotic therapy on the clinical manifestations of neuropsychiatric disorders in patients with dysbiotic changes." RMJ, no. 5 (2025): 22–28. https://doi.org/10.32364/2225-2282-2025-5-5.

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Aim: to assess the duration and changes of psychoneurological and gastrointestinal disorders in patients with psychoneurological conditions following the addition of a probiotic product containing lactobacilli LGG® and bifidobacteria BB-12® to comprehensive therapy. Materials and Methods: a retrospective study included 100 patients (59 female), aged over 18 years (range: 18–95 years; mean age: 49±8.6 years), presenting with psychoneurological symptoms: somatoform disorder (n=37), bipolar disorder (n=9), schizophrenia or schizoaffective disorder (n=12), parkinsonism (n=35, including Parkinson’s

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Kryukov, A. I., N. L. Kunelskaya, A. V. Gurov, G. N. Izotova, A. E. Starostina, and A. S. Lapchenko. "Clinical and microbiological characteristics of dysbiotic changes in the oral and oropharyngeal mucosa." Medical Council, no. 6 (January 1, 2016): 32–35. http://dx.doi.org/10.21518/2079-701x-2016-6-32-35.

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Caenepeel, C., S. Vieira-Silva, J. Sabino, et al. "P854 Quantitative microbiome profiling changes the described dysbiotic state in inflammatory bowel disease." Journal of Crohn's and Colitis 12, supplement_1 (2018): S548—S549. http://dx.doi.org/10.1093/ecco-jcc/jjx180.981.

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Dzhulay, G. S., and T. E. Dzhulay. "Clinical and pathogenic features of COVID-19- associated diarrhea syndrome." Experimental and Clinical Gastroenterology, no. 9 (October 7, 2022): 276–83. http://dx.doi.org/10.31146/1682-8658-ecg-205-9-276-283.

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The article systematizes the results of research in key areas of the problem of diarrhea, associated with COVID-19, analyzes the current ideas about the clinical characteristics, pathogenic mechanisms and relationships with the development of dysbiotic changes and inflammatory bowel diseases, accepted in practice and promising treatment strategies.

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Putelo, Audrey Marie, Tzu-Yu Feng, Sree Kolli, Mitchell McGinty, and Melanie Rutkowski. "Distal inflammatory priming of normal mammary tissue by the gut microbiome drives breast tumor metastasis via metabolic dysregulation." Journal of Immunology 210, no. 1_Supplement (2023): 88.08. http://dx.doi.org/10.4049/jimmunol.210.supp.88.08.

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Abstract Hormone receptor-positive (HR +), HER2 −is the most prevalent metastatic breast cancer subtype, constituting 73.1% of the metastatic disease population in the US. Despite targeted therapies that have increased long-term survival, many patients develop and eventually succumb to metastatic disease. Tumor dissemination occurs early during disease progression and is driven by cellular and molecular changes in the tissue. Though host-intrinsic factors that preferentially predispose certain individuals to metastatic disease are poorly defined, gut microbial health has become increasingly re

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Saettone, Vittorio, Ilaria Biasato, Elisabetta Radice, Achille Schiavone, Domenico Bergero, and Giorgia Meineri. "State-of-the-Art of the Nutritional Alternatives to the Use of Antibiotics in Humans and Monogastric Animals." Animals 10, no. 12 (2020): 2199. http://dx.doi.org/10.3390/ani10122199.

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In recent years, the indiscriminate use of antibiotics has been perpetrated across human medicine, animals destined for zootechnical productions and companion animals. Apart from increasing the resistance rate of numerous microorganisms and generating multi-drug resistance (MDR), the nonrational administration of antibiotics causes sudden changes in the structure of the intestinal microbiota such as dysbiotic phenomena that can have a great clinical significance for both humans and animals. The aim of this review is to describe the state-of-the-art of alternative therapies to the use of antibi

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Slazhneva, E. S., E. A. Tikhomirova, and V. G. Atrushkevich. "Periodontopathogens: a new view. Systematic review. Part 2." Pediatric dentistry and dental profilaxis 20, no. 2 (2020): 160–67. http://dx.doi.org/10.33925/1683-3031-2020-20-2-160-167.

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Relevance. The modern view of periodontitis as a dysbiotic disease that occurs as a result of changes in the microbial composition of the subgingival region is considered in a systematic review.Purpose. To study a new paradigm of development of generalized periodontitis.Materials and methods. Randomized controlled trials (RCTS) were selected for the study, including cluster RCTS, controlled (non-randomized) microbiological and clinical studies of the oral microbiome in adult patients with generalized periodontitis over the past 10 years.Results. The transition from a symbiotic microflora to a

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RUSIN, V. I., YE S. SIRCHAK, and N. YU KURCHAK. "The Dynamics of Immunological Indicants on the Background of Therapy Using Probiotics and Psyllium in Patients with Chronic Pancreatitis after Cholecystectomy." GASTROENTEROLOGY, no. 1.51 (January 15, 2014): 44–48. http://dx.doi.org/10.22141/2308-2097.1.51.2014.81909.

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The results of complex examination and treatment of 64 patients with chronic pancreatitis after cholecystectomy are given. It is ascertained, that complex therapy with the use of probiotics and psyllium in patients with chronic pancreatitis after cholecystectomy has marked positive effect on dysbiotic changes and immunity indicants (IgA, IgG, IgM, C3, C4) compared to monotherapy with corresponding medications.

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Jayasudha, Rajagopalaboopathi, Taraprasad Das, Sama Kalyana Chakravarthy, et al. "Gut mycobiomes are altered in people with type 2 Diabetes Mellitus and Diabetic Retinopathy." PLOS ONE 15, no. 12 (2020): e0243077. http://dx.doi.org/10.1371/journal.pone.0243077.

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Studies have documented dysbiosis in the gut mycobiome in people with Type 2 diabetes mellitus (T2DM). However, it is not known whether dysbiosis in the gut mycobiome of T2DM patients would be reflected in people with diabetic retinopathy (DR) and if so, is the observed mycobiome dysbiosis similar in people with T2DM and DR. Gut mycobiomes were generated from healthy controls (HC), people with T2DM and people with DR through Illumina sequencing of ITS2 region. Data were analysed using QIIME and R software. Dysbiotic changes were observed in people with T2DM and DR compared to HC at the phyla a

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Shnawa, Ibrahim M. S. "An at Glance Insight to Gut Microbiome Driven Autoimmunity." British Journal of Healthcare and Medical Research 12, no. 02 (2025): 110–19. https://doi.org/10.14738/bjhmr.122.18446.

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Autoimmunity is attributed to genetic predisposition [Genome] and environmental factors[exposome] and microbiome[microbiota]. The objective of the present opinion paper was to present an at glance insight to gut microbiome driven autoimmune diseases GMDADs based upon current published literature .The exposome components are; infection ,vaccination ,allergy and stress .Human balanced microbiome is noted in immune homeostasis. While, in its dysbiotic state in which specific microbe linked to induction of auto immune reactions, the infectome. On the onset of autoimmune diseases, leaky gut initiat

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Shnawa, Ibrahim M. S. "An at Glance Insight to Gut Microbiome Driven Autoimmunity." British Journal of Healthcare & Medical Research 12, no. 02 (2025): 110–19. https://doi.org/10.14738/bjhr.1202.18446.

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Autoimmunity is attributed to genetic predisposition [Genome] and environmental factors[exposome] and microbiome[microbiota]. The objective of the present opinion paper was to present an at glance insight to gut microbiome driven autoimmune diseases GMDADs based upon current published literature .The exposome components are; infection ,vaccination ,allergy and stress .Human balanced microbiome is noted in immune homeostasis. While, in its dysbiotic state in which specific microbe linked to induction of auto immune reactions, the infectome. On the onset of autoimmune diseases, leaky gut initiat

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Turaeva, Dilafruz, and Lilia Garifullina. "STATE OF THE GIT IN CHILDREN WITH NON-ALCOLIC FATTY LIVER DISEASE." Eurasian Journal of Medical and Natural Sciences 03, no. 02 (2023): 85–90. http://dx.doi.org/10.37547/ejmns-v03-i02-p1-15.

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Nonalcoholic fatty liver disease (NAFLD) combines with a variety of liver pathologies, including hepatic steatosis, nonalcoholic steatohepatitis, fibrosis and cirrhosis, and acts as hepatic manifestation of metabolic syndrome. Not only the liver is a target organ in the formation of metabolic syndrome: also exist a possibility of gallbladder, pancreas and biliary tract steatosis. Fatty infiltration of the pancreatobiliary system associates with disturbance of digestive processes that promotes dysbiotic changes and intestinal disorders. Changes in intestinal microbiota, in turn, may induce syst

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Turaeva, Dilafruz Kholmurodovna Garifullina Lilia Maratovna. "STATE OF THE GIT IN CHILDREN WITH NON-ALCOLIC FATTY LIVER DISEASE." EURASIAN JOURNAL OF MEDICAL AND NATURAL SCIENCES 3, no. 2 (2023): 85–90. https://doi.org/10.5281/zenodo.7619238.

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Nonalcoholic fatty liver disease (NAFLD) combines with a variety of liver pathologies, including hepatic steatosis, nonalcoholic steatohepatitis, fibrosis and cirrhosis, and acts as hepatic manifestation of metabolic syndrome. Not only the liver is a target organ in the formation of metabolic syndrome: also exist a possibility of gallbladder, pancreas and biliary tract steatosis. Fatty infiltration of the pancreatobiliary system associates with disturbance of digestive processes that promotes dysbiotic changes and intestinal disorders. Changes in intestinal microbiota, in turn, may induce syst

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КУРМАНГАЛИЕВА, С. С., Е. Ш. БАЗАРГАЛИЕВ, Х. И. КУДАБАЕВА, Р. Н. ЖАНАМАНОВА, Ф. С. РАХИМЖАНОВА, and Н. К. КЕНЖИНА. "EFFECT OF THE MICROBIOTA OF THE LARGE INTESTINE ON THE COURSE OF HEPATIC ENCEPHALOPATHY IN CIRRHOSIS." Farmaciâ Kazahstana, no. 2 (June 14, 2022): 55–62. http://dx.doi.org/10.53511/pharmkaz.2022.85.48.009.

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У пациентов с хроническими заболеваниями печени и циррозом печени (ЦП) наблюдается изменение состава и функциональности кишечной микробиоты. Эти изменения прогрессируют по мере увеличения степени цирроза печени. В статье описана взаимосвязь кишечной микробиоты с развитием печеночной энцефалопатии у больных циррозом печени. Эндогенная интоксикация неизбежно влечет за собой печеночную энцефалопатию (ПЭ). По данным нашего исследования при нарастании степени дисбиоза кишечника прогрессирует ПЭ (р<0,05). Была выявлена зависимость между степенью выраженности дисбиотических изменений толстой кишки

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Hashida, Ryoichi, Yasunori Shimozuru, Jessica Chang, Ibis Agosto-Marlin, Takaki Waritani, and Kuniaki Terato. "New Studies of Pathogenesis of Rheumatoid Arthritis with Collagen-Induced and Collagen Antibody-Induced Arthritis Models: New Insight Involving Bacteria Flora." Autoimmune Diseases 2021 (March 25, 2021): 1–13. http://dx.doi.org/10.1155/2021/7385106.

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Much public research suggests that autoimmune diseases such as rheumatoid arthritis (RA) are induced by aberrant “self” immune responses attacking autologous tissues and organ components. However, recent studies have reported that autoimmune diseases may be triggered by dysbiotic composition changes of the intestinal bacteria and an imbalance between these bacteria and intestinal immune systems. However, there are a few solid concepts or methods to study the putative involvement and relationship of these inner environmental factors in RA pathogenesis. Fortunately, Collagen-Induced Arthritis (C

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Rasmussen, Torben Sølbeck, Anna Kirstine Koefoed, Rasmus Riemer Jakobsen, et al. "Bacteriophage-mediated manipulation of the gut microbiome – promises and presents limitations." FEMS Microbiology Reviews 44, no. 4 (2020): 507–21. http://dx.doi.org/10.1093/femsre/fuaa020.

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ABSTRACT Gut microbiome (GM) composition and function are linked to human health and disease, and routes for manipulating the GM have become an area of intense research. Due to its high treatment efficacy, the use of fecal microbiota transplantation (FMT) is generally accepted as a promising experimental treatment for patients suffering from GM imbalances (dysbiosis), e.g. caused by recurrent Clostridioides difficile infections (rCDI). Mounting evidence suggests that bacteriophages (phages) play a key role in successful FMT treatment by restoring the dysbiotic bacterial GM. As a refinement to

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Grellier, Nathan, Marcelino T. Suzuki, Loic Brot, et al. "Impact of IBD-Associated Dysbiosis on Bacterial Quorum Sensing Mediated by Acyl-Homoserine Lactone in Human Gut Microbiota." International Journal of Molecular Sciences 23, no. 23 (2022): 15404. http://dx.doi.org/10.3390/ijms232315404.

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Intestinal dysbiosis is a key feature in the pathogenesis of inflammatory bowel disease (IBD). Acyl-homoserine lactones (AHL) are bacterial quorum-sensing metabolites that may play a role in the changes in host cells-gut microbiota interaction observed during IBD. The objective of our study was to investigate the presence and expression of AHL synthases and receptor genes in the human gut ecosystem during IBD. We used an in silico approach, applied to the Inflammatory Bowel Disease Multi’omics Database comprising bacterial metagenomic and metatranscriptomic data from stools of patients with Cr

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Robles, V., C. Herrera-deGuise, E. Varela, et al. "P880 Dysbiotic changes caused by polyethylene glycol in ulcerative colitis patients: "Lavage" or intrinsic effect?" Journal of Crohn's and Colitis 12, supplement_1 (2018): S562—S563. http://dx.doi.org/10.1093/ecco-jcc/jjx180.1007.

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Jabes, Daniela L., Yara N. L. F. de Maria, David Aciole Barbosa, et al. "Fungal Dysbiosis Correlates with the Development of Tumor-Induced Cachexia in Mice." Journal of Fungi 6, no. 4 (2020): 364. http://dx.doi.org/10.3390/jof6040364.

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Cachexia (CC) is a devastating metabolic syndrome associated with a series of underlying diseases that greatly affects life quality and expectancy among cancer patients. Studies involving mouse models, in which CC was induced through inoculation with tumor cells, originally suggested the existence of a direct correlation between the development of this syndrome and changes in the relative proportions of several bacterial groups present in the digestive tract. However, these analyses have focus solely on the characterization of bacterial dysbiosis, ignoring the possible existence of changes in

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Bobyr, V. V., L. O. Stechenko, V. P. Shirobokov, O. A. Nazarchuk, and O. V. Rymsha. "The role of sorbents and probiotics in prevention of structural and morphological disorders in the small intestine of animals developing in dysbiosis." Reports of Morphology 26, no. 2 (2020): 45–50. http://dx.doi.org/10.31393/morphology-journal-2020-26(2)-07.

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The past decade is characterized by a noticeable increase in the interest of physicians in all areas of activity in the development of new and improvement of existing approaches to the correction of dysbiotic disorders. Among them, the concept of using probiotics occupies a leading position. At the same time, some enterosorbents, the mechanism of action of which is largely due to the sanitation of the intestinal lumen and due to this improvement of conditions for the vital activity of the physiological microbiota, can be attributed to the group of means of improving the normal microflora. In t

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Zhdan, V. M., O. A. Kyrian, M. Yu Babanina, L. M. Shilkina, and O. I. Katerenchuk. "POSSIBILITIES OF DYSBIOTIC CHANGES CORRECTION IN PATIENTS WITH JOINTS AND SPINE INJURIES AND INFLAMMATORY BOWEL DISEASES." Bulletin of Problems Biology and Medicine 4, no. 1 (2020): 131. http://dx.doi.org/10.29254/2077-4214-2020-4-158-131-136.

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Gomez-Ramirez, Uriel, Pedro Valencia-Mayoral, Sandra Mendoza-Elizalde, et al. "Role of Helicobacter pylori and Other Environmental Factors in the Development of Gastric Dysbiosis." Pathogens 10, no. 9 (2021): 1203. http://dx.doi.org/10.3390/pathogens10091203.

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Microbiomes are defined as complex microbial communities, which are mainly composed of bacteria, fungi, and viruses residing in diverse regions of the human body. The human stomach consists of a unique and heterogeneous habitat of microbial communities owing to its anatomical and functional characteristics, that allow the optimal growth of characteristic bacteria in this environment. Gastric dysbiosis, which is defined as compositional and functional alterations of the gastric microbiota, can be induced by multiple environmental factors, such as age, diet, multiple antibiotic therapies, proton

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